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You searched for subject:(immune dysfunction). Showing records 1 – 17 of 17 total matches.

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Penn State University

1. Xia, Mingcan. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.

Degree: 2012, Penn State University

 Atherosclerosis (also known as arteriosclerotic vascular disease or ASVD) is a chronic inflammatory disease and characterized by patchy thickening of the inner lining of arterial… (more)

Subjects/Keywords: atherosclerosis; metabolic dysfunction; NKG2D ligand; NKG2D; Immune activation; diabetes; cardiovascular diseases; liver dysfunction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xia, M. (2012). Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xia, Mingcan. “Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.” 2012. Thesis, Penn State University. Accessed September 27, 2020. https://submit-etda.libraries.psu.edu/catalog/13899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xia, Mingcan. “Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.” 2012. Web. 27 Sep 2020.

Vancouver:

Xia M. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. [Internet] [Thesis]. Penn State University; 2012. [cited 2020 Sep 27]. Available from: https://submit-etda.libraries.psu.edu/catalog/13899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xia M. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. [Thesis]. Penn State University; 2012. Available from: https://submit-etda.libraries.psu.edu/catalog/13899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

2. Fu, Juan-juan. Systemic inflammation in obstructive airway disease.

Degree: PhD, 2014, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Obstructive airway disease (OAD) such as asthma and chronic obstructive pulmonary disease (COPD) are common respiratory conditions affecting… (more)

Subjects/Keywords: systemic inflammation; obstructive airway disease; chronic obstructive pulmonary disease; immune dysfunction

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APA (6th Edition):

Fu, J. (2014). Systemic inflammation in obstructive airway disease. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1050186

Chicago Manual of Style (16th Edition):

Fu, Juan-juan. “Systemic inflammation in obstructive airway disease.” 2014. Doctoral Dissertation, University of Newcastle. Accessed September 27, 2020. http://hdl.handle.net/1959.13/1050186.

MLA Handbook (7th Edition):

Fu, Juan-juan. “Systemic inflammation in obstructive airway disease.” 2014. Web. 27 Sep 2020.

Vancouver:

Fu J. Systemic inflammation in obstructive airway disease. [Internet] [Doctoral dissertation]. University of Newcastle; 2014. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/1959.13/1050186.

Council of Science Editors:

Fu J. Systemic inflammation in obstructive airway disease. [Doctoral Dissertation]. University of Newcastle; 2014. Available from: http://hdl.handle.net/1959.13/1050186


University of Southern California

3. Mordwinkin, Nicholas Michael. The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus.

Degree: PhD, Pharmaceutical Sciences, 2012, University of Southern California

 The aim of this dissertation is to evaluate the impact of type 2 diabetes mellitus on oxidative stress and inflammation in the bone marrow and… (more)

Subjects/Keywords: diabetes; angiotensin; oxidative stress; inflammation; immune dysfunction; progenitor cells; stem cells; endothelial dysfunction; cardiovascular disease; bone marrow

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APA (6th Edition):

Mordwinkin, N. M. (2012). The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/9565/rec/7068

Chicago Manual of Style (16th Edition):

Mordwinkin, Nicholas Michael. “The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus.” 2012. Doctoral Dissertation, University of Southern California. Accessed September 27, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/9565/rec/7068.

MLA Handbook (7th Edition):

Mordwinkin, Nicholas Michael. “The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus.” 2012. Web. 27 Sep 2020.

Vancouver:

Mordwinkin NM. The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Sep 27]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/9565/rec/7068.

Council of Science Editors:

Mordwinkin NM. The peptide angiotensin-(1-7) as a novel treatment for complications induced by type 2 diabetes mellitus. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/9565/rec/7068

4. Abdelsalam, Karim. The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis.

Degree: MS, Veterinary and Biomedical Sciences, 2017, South Dakota State University

  BVDV is an important pathogen of cattle that affects both the dairy and beef industry causing severe economic losses. The main problem of BVDV… (more)

Subjects/Keywords: BVDV; immune dysfunction; immune suppression; indirect effect; lymphocyte apoptosis; macrophages; Veterinary Medicine; Veterinary Microbiology and Immunobiology; Veterinary Pathology and Pathobiology

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APA (6th Edition):

Abdelsalam, K. (2017). The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis. (Masters Thesis). South Dakota State University. Retrieved from http://openprairie.sdstate.edu/etd/1129

Chicago Manual of Style (16th Edition):

Abdelsalam, Karim. “The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis.” 2017. Masters Thesis, South Dakota State University. Accessed September 27, 2020. http://openprairie.sdstate.edu/etd/1129.

MLA Handbook (7th Edition):

Abdelsalam, Karim. “The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis.” 2017. Web. 27 Sep 2020.

Vancouver:

Abdelsalam K. The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis. [Internet] [Masters thesis]. South Dakota State University; 2017. [cited 2020 Sep 27]. Available from: http://openprairie.sdstate.edu/etd/1129.

Council of Science Editors:

Abdelsalam K. The Indirect Effect of BVDV on Immune Suppression: The Role of Infected Macrophages in Lymphocyte Apoptosis. [Masters Thesis]. South Dakota State University; 2017. Available from: http://openprairie.sdstate.edu/etd/1129


IUPUI

5. Polanka, Brittanny M. Insomnia and mechanistic pathways to atherosclerotic CVD in HIV.

Degree: 2020, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Study 1: Background: Insomnia may be a risk factor for cardiovascular disease in HIV (HIV-CVD); however, mechanisms have yet to… (more)

Subjects/Keywords: sleep disturbance; insomnia; immune activation; inflammation; endothelial dysfunction; coagulation; human immunodeficiency virus

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APA (6th Edition):

Polanka, B. M. (2020). Insomnia and mechanistic pathways to atherosclerotic CVD in HIV. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/23399

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Polanka, Brittanny M. “Insomnia and mechanistic pathways to atherosclerotic CVD in HIV.” 2020. Thesis, IUPUI. Accessed September 27, 2020. http://hdl.handle.net/1805/23399.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Polanka, Brittanny M. “Insomnia and mechanistic pathways to atherosclerotic CVD in HIV.” 2020. Web. 27 Sep 2020.

Vancouver:

Polanka BM. Insomnia and mechanistic pathways to atherosclerotic CVD in HIV. [Internet] [Thesis]. IUPUI; 2020. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/1805/23399.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Polanka BM. Insomnia and mechanistic pathways to atherosclerotic CVD in HIV. [Thesis]. IUPUI; 2020. Available from: http://hdl.handle.net/1805/23399

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. ZAREEN, ZUNERA. CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy.

Degree: School of Medicine. Discipline of Paediatrics, 2019, Trinity College Dublin

 Neonatal brain injury is an important cause of neonatal death and disability such as cerebral palsy. Perinatal global hypoxic ischaemia associated with neonatal encephalopathy results… (more)

Subjects/Keywords: Neonatal encephalopathy; childhood outcome; multi-organ dysfunction; cytokines; innate immune function; sleep and circadian genes; quality of life

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APA (6th Edition):

ZAREEN, Z. (2019). CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/87268

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZAREEN, ZUNERA. “CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy.” 2019. Thesis, Trinity College Dublin. Accessed September 27, 2020. http://hdl.handle.net/2262/87268.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZAREEN, ZUNERA. “CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy.” 2019. Web. 27 Sep 2020.

Vancouver:

ZAREEN Z. CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/2262/87268.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZAREEN Z. CHAMPION STUDY: Childhood Assessment of Multi-organ dysfunction Post Neonatal encephalopathy. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/87268

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loughborough University

7. Dungey, Maurice. Exercise in haemodialysis patients : impact on markers of inflammation.

Degree: PhD, 2015, Loughborough University

 End-stage renal disease patients have a greatly increased risk of cardiovascular disease partly attributed to the elevated levels of systemic inflammation observed in uraemia. One… (more)

Subjects/Keywords: 616.6; Haemodialysis; End-stage renal disease; Exercise; Inflammation; Monocytes; Cardiovascular disease; Immune dysfunction; Regulatory T cells

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APA (6th Edition):

Dungey, M. (2015). Exercise in haemodialysis patients : impact on markers of inflammation. (Doctoral Dissertation). Loughborough University. Retrieved from http://hdl.handle.net/2134/17157

Chicago Manual of Style (16th Edition):

Dungey, Maurice. “Exercise in haemodialysis patients : impact on markers of inflammation.” 2015. Doctoral Dissertation, Loughborough University. Accessed September 27, 2020. http://hdl.handle.net/2134/17157.

MLA Handbook (7th Edition):

Dungey, Maurice. “Exercise in haemodialysis patients : impact on markers of inflammation.” 2015. Web. 27 Sep 2020.

Vancouver:

Dungey M. Exercise in haemodialysis patients : impact on markers of inflammation. [Internet] [Doctoral dissertation]. Loughborough University; 2015. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/2134/17157.

Council of Science Editors:

Dungey M. Exercise in haemodialysis patients : impact on markers of inflammation. [Doctoral Dissertation]. Loughborough University; 2015. Available from: http://hdl.handle.net/2134/17157


University of Tasmania

8. Eapen, MS. Understanding cellular changes and mediators of airway remodelling in COPD.

Degree: 2018, University of Tasmania

 (Background) (and) (Aims:) Chronic obstructive pulmonary disease (COPD) is emerging as a substantial global health problem, with an estimated annual mortality of over 3 million… (more)

Subjects/Keywords: Chronic Obstructive Pulmonary Disease (COPD); airway wall; cellularity; inflammation; immune dysfunction; myofibroblast; extracellular matrix; airway remodeling

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APA (6th Edition):

Eapen, M. (2018). Understanding cellular changes and mediators of airway remodelling in COPD. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eapen, MS. “Understanding cellular changes and mediators of airway remodelling in COPD.” 2018. Thesis, University of Tasmania. Accessed September 27, 2020. https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eapen, MS. “Understanding cellular changes and mediators of airway remodelling in COPD.” 2018. Web. 27 Sep 2020.

Vancouver:

Eapen M. Understanding cellular changes and mediators of airway remodelling in COPD. [Internet] [Thesis]. University of Tasmania; 2018. [cited 2020 Sep 27]. Available from: https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eapen M. Understanding cellular changes and mediators of airway remodelling in COPD. [Thesis]. University of Tasmania; 2018. Available from: https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

9. Thiel, Johannes Tobias. Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?.

Degree: 2018, Freie Universität Berlin

 Purpose Alveolar macrophages (AM) are the first barrier against respiratory pathogens in the lung. In addition to phagocytic properties, AMs also activate other immune cells… (more)

Subjects/Keywords: alveolar macrophages; chronic alcohol consumption; immune dysfunction; cytokines; lung; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Thiel, J. T. (2018). Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thiel, Johannes Tobias. “Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?.” 2018. Thesis, Freie Universität Berlin. Accessed September 27, 2020. http://dx.doi.org/10.17169/refubium-12751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thiel, Johannes Tobias. “Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?.” 2018. Web. 27 Sep 2020.

Vancouver:

Thiel JT. Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?. [Internet] [Thesis]. Freie Universität Berlin; 2018. [cited 2020 Sep 27]. Available from: http://dx.doi.org/10.17169/refubium-12751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thiel JT. Chronic alcohol consumption – a dangerous cocktail for alveolar macrophages?. [Thesis]. Freie Universität Berlin; 2018. Available from: http://dx.doi.org/10.17169/refubium-12751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

10. Kieswetter, Nathan Scott. Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons.

Degree: 2016, University of KwaZulu-Natal

Abstract available in PDF file. Advisors/Committee Members: Ndung'u, Thumbi. (advisor), Wong, Emily. (advisor).

Subjects/Keywords: Microbial cells.; Tuberculosis.; HIV infected individuals.; Anti-TB immune dysfunction.

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APA (6th Edition):

Kieswetter, N. S. (2016). Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/14799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kieswetter, Nathan Scott. “Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons.” 2016. Thesis, University of KwaZulu-Natal. Accessed September 27, 2020. http://hdl.handle.net/10413/14799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kieswetter, Nathan Scott. “Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons.” 2016. Web. 27 Sep 2020.

Vancouver:

Kieswetter NS. Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons. [Internet] [Thesis]. University of KwaZulu-Natal; 2016. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/10413/14799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kieswetter NS. Development and optimization of real-time PCR assays to detect anti-microbial immune factors and their response to type I and II interferons. [Thesis]. University of KwaZulu-Natal; 2016. Available from: http://hdl.handle.net/10413/14799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waikato

11. McGowan, Jacqueline Ellen. Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function .

Degree: 2014, University of Waikato

 The extent of postpartum negative energy balance in dairy cows is positively associated with body condition score (BCS) at calving. Results of epidemiological studies have… (more)

Subjects/Keywords: Candida albicans; antibody-mediated; cell-mediated; negative energy balance; AMIR; CMIR; delayed-type hypersensitivity; DTH; periparturient immune dysfunction; pasture-based; transition; hen eggwhite lysozyme; human serum albumin; HEWL; HSA

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APA (6th Edition):

McGowan, J. E. (2014). Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function . (Masters Thesis). University of Waikato. Retrieved from http://hdl.handle.net/10289/9214

Chicago Manual of Style (16th Edition):

McGowan, Jacqueline Ellen. “Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function .” 2014. Masters Thesis, University of Waikato. Accessed September 27, 2020. http://hdl.handle.net/10289/9214.

MLA Handbook (7th Edition):

McGowan, Jacqueline Ellen. “Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function .” 2014. Web. 27 Sep 2020.

Vancouver:

McGowan JE. Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function . [Internet] [Masters thesis]. University of Waikato; 2014. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/10289/9214.

Council of Science Editors:

McGowan JE. Effect of Body Condition Score at Calving on Adaptive Immune Function During Early Lactation in New Zealand Dairy Cows: Effect of calving body condition on adaptive immune function . [Masters Thesis]. University of Waikato; 2014. Available from: http://hdl.handle.net/10289/9214


Université Paris-Sud – Paris XI

12. Huertas, Alice. De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets.

Degree: Docteur es, Physiopathologie cellulaire et moléculaire, 2013, Université Paris-Sud – Paris XI

L’hypertension artérielle pulmonaire (HTAP) est une maladie grave caractérisée par une obstruction progressive des artères pulmonaires de petit calibre, conduisant à une augmentation des résistances… (more)

Subjects/Keywords: HTAP; Dysfonction endothéliale; Système immunitaire; Lymphocytes T régulateurs; Leptine; Hypoxie; Modèles animaux; Innovation thérapeutique; PAH; Endothelial dysfunction; Immune system; Regulatory T lymphocytes; Leptin; Hypoxia; Animal models; Novel therapies

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APA (6th Edition):

Huertas, A. (2013). De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA114824

Chicago Manual of Style (16th Edition):

Huertas, Alice. “De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 27, 2020. http://www.theses.fr/2013PA114824.

MLA Handbook (7th Edition):

Huertas, Alice. “De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets.” 2013. Web. 27 Sep 2020.

Vancouver:

Huertas A. De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Sep 27]. Available from: http://www.theses.fr/2013PA114824.

Council of Science Editors:

Huertas A. De la dysfonction endothéliale à la dysfonction immunitaire dans l’hypertension artérielle pulmonaire : nouvelles cibles d’innovation thérapeutique : From endothelial dysfunction to immune dysfunction in pulmonary arterial hypertension : novel therapeutical targets. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA114824


University of Hawaii

13. Clements, Danielle M. REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION.

Degree: 2020, University of Hawaii

Subjects/Keywords: Translation studies; Immunology; HTLV-1; Immune checkpoint blockade; Immunotherapy; T-cell dysfunction

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APA (6th Edition):

Clements, D. M. (2020). REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION. (Thesis). University of Hawaii. Retrieved from http://hdl.handle.net/10125/68913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Clements, Danielle M. “REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION.” 2020. Thesis, University of Hawaii. Accessed September 27, 2020. http://hdl.handle.net/10125/68913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Clements, Danielle M. “REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION.” 2020. Web. 27 Sep 2020.

Vancouver:

Clements DM. REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION. [Internet] [Thesis]. University of Hawaii; 2020. [cited 2020 Sep 27]. Available from: http://hdl.handle.net/10125/68913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Clements DM. REGULATION OF CD8 T-CELL FUNCTION BY MULTIPLE NEGATIVE IMMUNE CHECKPOINT MOLECULES DURING HTLV-1 INFECTION. [Thesis]. University of Hawaii; 2020. Available from: http://hdl.handle.net/10125/68913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Chung, Charlotte Yuk-Yan. Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis.

Degree: PhD, Pharmacology, 2015, Case Western Reserve University School of Graduate Studies

 Systemic inflammation in the HIV-infected patient plays a crucial role in the pathogenesis of CD4+ T cell depletion, and is recently postulated to also drive… (more)

Subjects/Keywords: Virology; Immunology; Cellular Biology; Pharmacology; tight junction; HIV; paracellular permeability; systemic inflammation; immune activation; intestinal epithelial cells; microbial translocation; ART-treated; intestinal barrier dysfunction; transepithelial resistance; IEC - T cell interaction

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APA (6th Edition):

Chung, C. Y. (2015). Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1417822947

Chicago Manual of Style (16th Edition):

Chung, Charlotte Yuk-Yan. “Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis.” 2015. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed September 27, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1417822947.

MLA Handbook (7th Edition):

Chung, Charlotte Yuk-Yan. “Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis.” 2015. Web. 27 Sep 2020.

Vancouver:

Chung CY. Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2015. [cited 2020 Sep 27]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417822947.

Council of Science Editors:

Chung CY. Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417822947


University of Western Ontario

15. Ma, Xin Tong. The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice.

Degree: 2020, University of Western Ontario

 In sepsis, lipopolysaccharide (LPS) activates toll-like receptor 4 to stimulate the release of inflammatory cytokines (e.g. tumor necrosis factor-alpha, TNF-α), leading to cardiac dysfunction. Regulator… (more)

Subjects/Keywords: sepsis; regulator of G protein signaling 2 (RGS2); cardiac dysfunction; endotoxemia; TNF-α; inflammatory cytokine release; Cardiovascular Diseases; Cellular and Molecular Physiology; Immune System Diseases; Physiology

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APA (6th Edition):

Ma, X. T. (2020). The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/7317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ma, Xin Tong. “The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice.” 2020. Thesis, University of Western Ontario. Accessed September 27, 2020. https://ir.lib.uwo.ca/etd/7317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ma, Xin Tong. “The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice.” 2020. Web. 27 Sep 2020.

Vancouver:

Ma XT. The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2020 Sep 27]. Available from: https://ir.lib.uwo.ca/etd/7317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ma XT. The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/7317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Gonçalves, Ana Catarina Martins. Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn.

Degree: 2013, Universidade Fernando Pessoa

Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas

A Doença de Crohn… (more)

Subjects/Keywords: Doença de Crohn; Etiologia; Patogénese; Doenças inflamatórias intestinais; Epidemiologia; Escherichia coli aderente-invasiva; Disbiose; Microflora intestinal; Autofagia; Fatores genéticos e ambientais; Resposta imunitária; Terapia; Crohn's disease; Etiology; Pathogenesis; Epidemiology; Adherent-invasive Escherichia coli; Dysbiosis; Intestinal microflora; Autophagy; genetic and environmental factors; Innate immune dysfunction; Therapeutic

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APA (6th Edition):

Gonçalves, A. C. M. (2013). Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn. (Thesis). Universidade Fernando Pessoa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/4089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonçalves, Ana Catarina Martins. “Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn.” 2013. Thesis, Universidade Fernando Pessoa. Accessed September 27, 2020. http://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/4089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonçalves, Ana Catarina Martins. “Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn.” 2013. Web. 27 Sep 2020.

Vancouver:

Gonçalves ACM. Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn. [Internet] [Thesis]. Universidade Fernando Pessoa; 2013. [cited 2020 Sep 27]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/4089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonçalves ACM. Relação entre a Infeção por Escherichia coli Aderente-Invasina e a Doença de Crohn. [Thesis]. Universidade Fernando Pessoa; 2013. Available from: http://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/4089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Fontaine, Mathieu. Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches.

Degree: Docteur es, Recherche clinique, 2015, Université Claude Bernard – Lyon I

Le choc septique reste une pathologie grave, associée à des taux de mortalité et d'infections nosocomiales (IN) secondaires élevés. La prédiction du pronostic est de… (more)

Subjects/Keywords: Choc septique; Infection nosocomiale; S100A9; ARNm; Marqueur biologique; Dysfonction immunitaire; Tolérance à l’endotoxine; Reprogrammation leucocytaire; Septic shock; Cross infection; Calgranulin B; MRNA; Biologic marker; Immune dysfunction; Endotoxin tolerance; Leukocyte reprogramming; 610

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fontaine, M. (2015). Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2015LYO10038

Chicago Manual of Style (16th Edition):

Fontaine, Mathieu. “Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches.” 2015. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed September 27, 2020. http://www.theses.fr/2015LYO10038.

MLA Handbook (7th Edition):

Fontaine, Mathieu. “Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches.” 2015. Web. 27 Sep 2020.

Vancouver:

Fontaine M. Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2015. [cited 2020 Sep 27]. Available from: http://www.theses.fr/2015LYO10038.

Council of Science Editors:

Fontaine M. Alarmine S100A9 : de la théorie du danger aux infections nosocomiales après un choc septique : approche clinique et expérimentale : S100A9 alarmin : from danger model to nosocomial infections after septic shock : clinical and experimental approaches. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2015. Available from: http://www.theses.fr/2015LYO10038

.