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You searched for subject:(histone deacetylase). Showing records 1 – 30 of 186 total matches.

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University of Melbourne

1. NEWBOLD, ANDREA. Mechanisms of action of histone deacetylase inhibitors.

Degree: 2011, University of Melbourne

 The opposing activities of histone acetyltransferases (HATs) and histone deacetylases (HDACs) play important roles in tightly regulating gene expression through regulated chromatin remodelling. It is… (more)

Subjects/Keywords: histone deacetylase inhibitor

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APA (6th Edition):

NEWBOLD, A. (2011). Mechanisms of action of histone deacetylase inhibitors. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36731

Chicago Manual of Style (16th Edition):

NEWBOLD, ANDREA. “Mechanisms of action of histone deacetylase inhibitors.” 2011. Doctoral Dissertation, University of Melbourne. Accessed October 25, 2020. http://hdl.handle.net/11343/36731.

MLA Handbook (7th Edition):

NEWBOLD, ANDREA. “Mechanisms of action of histone deacetylase inhibitors.” 2011. Web. 25 Oct 2020.

Vancouver:

NEWBOLD A. Mechanisms of action of histone deacetylase inhibitors. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/11343/36731.

Council of Science Editors:

NEWBOLD A. Mechanisms of action of histone deacetylase inhibitors. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36731


Oregon State University

2. Nian, Hui. Dietary organosulfur and organoselenium compounds as HDAC inhibitors.

Degree: PhD, Biochemistry and Biophysics, 2010, Oregon State University

Histone deacetylase (HDAC) inhibitors have the potential to de-repress epigenetically silenced genes in cancer cells, leading to cell cycle arrest and apoptosis. Dietary HDAC inhibitors… (more)

Subjects/Keywords: HDAC; Histone deacetylase  – Inhibitors

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APA (6th Edition):

Nian, H. (2010). Dietary organosulfur and organoselenium compounds as HDAC inhibitors. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/15093

Chicago Manual of Style (16th Edition):

Nian, Hui. “Dietary organosulfur and organoselenium compounds as HDAC inhibitors.” 2010. Doctoral Dissertation, Oregon State University. Accessed October 25, 2020. http://hdl.handle.net/1957/15093.

MLA Handbook (7th Edition):

Nian, Hui. “Dietary organosulfur and organoselenium compounds as HDAC inhibitors.” 2010. Web. 25 Oct 2020.

Vancouver:

Nian H. Dietary organosulfur and organoselenium compounds as HDAC inhibitors. [Internet] [Doctoral dissertation]. Oregon State University; 2010. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1957/15093.

Council of Science Editors:

Nian H. Dietary organosulfur and organoselenium compounds as HDAC inhibitors. [Doctoral Dissertation]. Oregon State University; 2010. Available from: http://hdl.handle.net/1957/15093


Harvard University

3. Sinapius, Ryan. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.

Degree: ALM, 2019, Harvard University

An estimated 15.5 million Americans have or are currently in remission from cancer and this number is likely to rise to 20.3 million by 2026… (more)

Subjects/Keywords: HDAC3; HDAC8; Histone Deacetylase

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APA (6th Edition):

Sinapius, R. (2019). Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. (Masters Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004173

Chicago Manual of Style (16th Edition):

Sinapius, Ryan. “Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.” 2019. Masters Thesis, Harvard University. Accessed October 25, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004173.

MLA Handbook (7th Edition):

Sinapius, Ryan. “Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.” 2019. Web. 25 Oct 2020.

Vancouver:

Sinapius R. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. [Internet] [Masters thesis]. Harvard University; 2019. [cited 2020 Oct 25]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004173.

Council of Science Editors:

Sinapius R. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. [Masters Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004173


Harvard University

4. Sinapius, Ryan. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.

Degree: 2019, Harvard University

An estimated 15.5 million Americans have or are currently in remission from cancer and this number is likely to rise to 20.3 million by 2026… (more)

Subjects/Keywords: HDAC3; HDAC8; Histone Deacetylase

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APA (6th Edition):

Sinapius, R. (2019). Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. (Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sinapius, Ryan. “Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.” 2019. Thesis, Harvard University. Accessed October 25, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sinapius, Ryan. “Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival.” 2019. Web. 25 Oct 2020.

Vancouver:

Sinapius R. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. [Internet] [Thesis]. Harvard University; 2019. [cited 2020 Oct 25]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sinapius R. Effect of Histone Deacetylase Inhibition on Ovarian Cancer Cell Survival. [Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

5. Bai, He. Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes.

Degree: 2013, University of Illinois – Chicago

Histone deacetylases (HDACs) regulate chromatin structure and function. Since much of the literature has reported aberrant expression and recruitment of HDAC in malignant tissues, the… (more)

Subjects/Keywords: Histone Deacetylase; Photolabeling; BeProFL

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APA (6th Edition):

Bai, H. (2013). Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bai, He. “Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes.” 2013. Thesis, University of Illinois – Chicago. Accessed October 25, 2020. http://hdl.handle.net/10027/9803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bai, He. “Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes.” 2013. Web. 25 Oct 2020.

Vancouver:

Bai H. Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10027/9803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bai H. Exploration of Histone Deacetylase Ligand Binding Modes by Photoaffinity Probes. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Morales Medina, Cyndi Raquel. Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression.

Degree: 2014, University of Texas Southwestern Medical Center

 Stress-induced pathological hypertrophy is observed in most forms of heart disease. If left unchecked, pathological remodeling can lead to heart failure. Histone deacetylases (HDACs) participate… (more)

Subjects/Keywords: Cardiomegaly; Histone Deacetylase Inhibitors; Histone Deacetylases

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APA (6th Edition):

Morales Medina, C. R. (2014). Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morales Medina, Cyndi Raquel. “Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed October 25, 2020. http://hdl.handle.net/2152.5/3952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morales Medina, Cyndi Raquel. “Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression.” 2014. Web. 25 Oct 2020.

Vancouver:

Morales Medina CR. Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2152.5/3952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morales Medina CR. Inhibition of Class I HDACs Blunts Cardiac Hypertrophy via TSC2-Dependent mTOR Repression. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queensland University of Technology

7. Tuttle, Camilla Susannah Laura. The expression of HAT and HDAC enzymes in asthma airways.

Degree: 2013, Queensland University of Technology

Asthma is chronic inflammatory disease of the lower airways that is both, genetically inherited and environmentally influenced. This project investigated how molecular mechanisms known to be influenced both genetically and environmentally, contribute to the onset of asthma.

Subjects/Keywords: Asthma; Histone acetylation; Histone acetyltransferase; Histone deacetylase; Sputum

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APA (6th Edition):

Tuttle, C. S. L. (2013). The expression of HAT and HDAC enzymes in asthma airways. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/62873/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tuttle, Camilla Susannah Laura. “The expression of HAT and HDAC enzymes in asthma airways.” 2013. Thesis, Queensland University of Technology. Accessed October 25, 2020. https://eprints.qut.edu.au/62873/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tuttle, Camilla Susannah Laura. “The expression of HAT and HDAC enzymes in asthma airways.” 2013. Web. 25 Oct 2020.

Vancouver:

Tuttle CSL. The expression of HAT and HDAC enzymes in asthma airways. [Internet] [Thesis]. Queensland University of Technology; 2013. [cited 2020 Oct 25]. Available from: https://eprints.qut.edu.au/62873/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tuttle CSL. The expression of HAT and HDAC enzymes in asthma airways. [Thesis]. Queensland University of Technology; 2013. Available from: https://eprints.qut.edu.au/62873/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

8. Simic, Damir. Targeting HDAC5 to Improve Cancer Chemotherapy.

Degree: 2019, Drexel University

 Chemotherapy resistance and dose-limiting adverse effects remain major challenges for tumor management. It has been shown that cytoplasmic translocation of HDAC5 plays a critical role… (more)

Subjects/Keywords: Biology; Cancer; Cancer – Chemotherapy; Histone deacetylase

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APA (6th Edition):

Simic, D. (2019). Targeting HDAC5 to Improve Cancer Chemotherapy. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A9458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simic, Damir. “Targeting HDAC5 to Improve Cancer Chemotherapy.” 2019. Thesis, Drexel University. Accessed October 25, 2020. https://idea.library.drexel.edu/islandora/object/idea%3A9458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simic, Damir. “Targeting HDAC5 to Improve Cancer Chemotherapy.” 2019. Web. 25 Oct 2020.

Vancouver:

Simic D. Targeting HDAC5 to Improve Cancer Chemotherapy. [Internet] [Thesis]. Drexel University; 2019. [cited 2020 Oct 25]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simic D. Targeting HDAC5 to Improve Cancer Chemotherapy. [Thesis]. Drexel University; 2019. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

9. Fong, Man Kim. Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana.

Degree: MS, Molecular and Enviromental Plant Sciences, 2005, Texas A&M University

 The reversible process of histone acetylation and deacetylation is an important mechanism of epigenetic regulation in the control of gene expression and chromatin structure. In… (more)

Subjects/Keywords: histone; deacetylase; chromatin

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APA (6th Edition):

Fong, M. K. (2005). Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/2365

Chicago Manual of Style (16th Edition):

Fong, Man Kim. “Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana.” 2005. Masters Thesis, Texas A&M University. Accessed October 25, 2020. http://hdl.handle.net/1969.1/2365.

MLA Handbook (7th Edition):

Fong, Man Kim. “Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana.” 2005. Web. 25 Oct 2020.

Vancouver:

Fong MK. Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana. [Internet] [Masters thesis]. Texas A&M University; 2005. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1969.1/2365.

Council of Science Editors:

Fong MK. Characterization and expression of histone deacetylase 1 (athd1) in Arabidopsis thaliana. [Masters Thesis]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/2365


University of Adelaide

10. Algate, Kent. Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition.

Degree: 2018, University of Adelaide

 Periodontitis (PD) is one of the most common bone loss pathologies in adults and currently affects more than 60% of the population in its destructive… (more)

Subjects/Keywords: Bone metabolism; epigenetics; histone deacetylase; inflammation; periodontics

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APA (6th Edition):

Algate, K. (2018). Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/127113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Algate, Kent. “Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition.” 2018. Thesis, University of Adelaide. Accessed October 25, 2020. http://hdl.handle.net/2440/127113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Algate, Kent. “Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition.” 2018. Web. 25 Oct 2020.

Vancouver:

Algate K. Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition. [Internet] [Thesis]. University of Adelaide; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2440/127113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Algate K. Epigenetic Regulation of Cells Involved in Periodontal Bone Destruction through Targeted Histone Deacetylase Inhibition. [Thesis]. University of Adelaide; 2018. Available from: http://hdl.handle.net/2440/127113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

11. Quach, Khong Tri. Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy .

Degree: DE – Gyógyszerésztudományi Kar, University of Debrecen

 Nowadays, it became evident that the onset of cancer and its progression may not occur only due to genetic mutations but also because of changes… (more)

Subjects/Keywords: histone deacetylase inhibitors

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APA (6th Edition):

Quach, K. T. (n.d.). Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/263457

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quach, Khong Tri. “Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy .” Thesis, University of Debrecen. Accessed October 25, 2020. http://hdl.handle.net/2437/263457.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quach, Khong Tri. “Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy .” Web. 25 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Quach KT. Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy . [Internet] [Thesis]. University of Debrecen; [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2437/263457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Quach KT. Histone Deacetylase Inhibitors: Novel advances in Cancer Therapy . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/263457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Melbourne

12. West, Alison Clare. Immunomodulatory properties of the histone deacetylase inhibitor vorinostat.

Degree: 2012, University of Melbourne

Histone deacetylase inhibitors (HDACi) are an exciting class of anti-cancer agents that are particularly efficacious against hematological malignancies, however have limited efficacy against solid tumours.… (more)

Subjects/Keywords: histone deacetylase inhibitor; vorinostat; tumour immunology

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APA (6th Edition):

West, A. C. (2012). Immunomodulatory properties of the histone deacetylase inhibitor vorinostat. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37995

Chicago Manual of Style (16th Edition):

West, Alison Clare. “Immunomodulatory properties of the histone deacetylase inhibitor vorinostat.” 2012. Doctoral Dissertation, University of Melbourne. Accessed October 25, 2020. http://hdl.handle.net/11343/37995.

MLA Handbook (7th Edition):

West, Alison Clare. “Immunomodulatory properties of the histone deacetylase inhibitor vorinostat.” 2012. Web. 25 Oct 2020.

Vancouver:

West AC. Immunomodulatory properties of the histone deacetylase inhibitor vorinostat. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/11343/37995.

Council of Science Editors:

West AC. Immunomodulatory properties of the histone deacetylase inhibitor vorinostat. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37995

13. Wolfson, Noah Ariel. Determining HDAC8 Substrate Specificity.

Degree: PhD, Biological Chemistry, 2014, University of Michigan

Histone deacetylases (HDACs) are a group of 18 enzymes that catalyze the deacetylation of acetyl lysine residues in proteins. Acetyl lysine residues are present within… (more)

Subjects/Keywords: HDAC; Histone Deacetylase; Lysine Deacetylase; Biological Chemistry; Health Sciences

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APA (6th Edition):

Wolfson, N. A. (2014). Determining HDAC8 Substrate Specificity. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/108746

Chicago Manual of Style (16th Edition):

Wolfson, Noah Ariel. “Determining HDAC8 Substrate Specificity.” 2014. Doctoral Dissertation, University of Michigan. Accessed October 25, 2020. http://hdl.handle.net/2027.42/108746.

MLA Handbook (7th Edition):

Wolfson, Noah Ariel. “Determining HDAC8 Substrate Specificity.” 2014. Web. 25 Oct 2020.

Vancouver:

Wolfson NA. Determining HDAC8 Substrate Specificity. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2027.42/108746.

Council of Science Editors:

Wolfson NA. Determining HDAC8 Substrate Specificity. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/108746


University of Pennsylvania

14. Hai, Yang. Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family.

Degree: 2016, University of Pennsylvania

 Arginases and deacetylases are metallohydrolases that catalyze two distinct chemical transformations. The arginases catalyze the hydrolysis of the guanidinium group of arginine by using a… (more)

Subjects/Keywords: Arginase; Crystallography; Histone deacetylase; Metallohydrolase; polyamine deacetylase; Biochemistry

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APA (6th Edition):

Hai, Y. (2016). Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hai, Yang. “Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family.” 2016. Thesis, University of Pennsylvania. Accessed October 25, 2020. https://repository.upenn.edu/edissertations/1753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hai, Yang. “Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family.” 2016. Web. 25 Oct 2020.

Vancouver:

Hai Y. Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2020 Oct 25]. Available from: https://repository.upenn.edu/edissertations/1753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hai Y. Structure and Function of Metallohydrolases in the Arginase-Deacetylase Family. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/1753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Bryan, Erin E. Expression of Class I Histone Deacetylases in Insect Cells.

Degree: MS, 2006, Worcester Polytechnic Institute

 "Histone deacetylases (HDACs) have become one of the leading areas of research for cancer, neurodegenerative diseases, diabetes, obesity, and inflammation. Although HDACs are currently expressible… (more)

Subjects/Keywords: histone deacetylase; Histone deacetylase; Transcription factors

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APA (6th Edition):

Bryan, E. E. (2006). Expression of Class I Histone Deacetylases in Insect Cells. (Thesis). Worcester Polytechnic Institute. Retrieved from etd-053006-130545 ; https://digitalcommons.wpi.edu/etd-theses/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bryan, Erin E. “Expression of Class I Histone Deacetylases in Insect Cells.” 2006. Thesis, Worcester Polytechnic Institute. Accessed October 25, 2020. etd-053006-130545 ; https://digitalcommons.wpi.edu/etd-theses/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bryan, Erin E. “Expression of Class I Histone Deacetylases in Insect Cells.” 2006. Web. 25 Oct 2020.

Vancouver:

Bryan EE. Expression of Class I Histone Deacetylases in Insect Cells. [Internet] [Thesis]. Worcester Polytechnic Institute; 2006. [cited 2020 Oct 25]. Available from: etd-053006-130545 ; https://digitalcommons.wpi.edu/etd-theses/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bryan EE. Expression of Class I Histone Deacetylases in Insect Cells. [Thesis]. Worcester Polytechnic Institute; 2006. Available from: etd-053006-130545 ; https://digitalcommons.wpi.edu/etd-theses/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

16. Fathi, Shaghayegh. Targeted delivery of histone deacetylase inhibitors for use in cancer therapy.

Degree: PhD, Chemistry and Biochemistry, 2016, Georgia Tech

 Cancer is one of the leading causes of death around the world, with lung, breast and prostate cancer being the most common cancers. Most of… (more)

Subjects/Keywords: Cancer; Histone deacetylase; Histone deacetylase inhibitors; Targeted delivery; Azithromycin; Tamoxifen; SAHA; Pyrimethamine; Liposome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fathi, S. (2016). Targeted delivery of histone deacetylase inhibitors for use in cancer therapy. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/61091

Chicago Manual of Style (16th Edition):

Fathi, Shaghayegh. “Targeted delivery of histone deacetylase inhibitors for use in cancer therapy.” 2016. Doctoral Dissertation, Georgia Tech. Accessed October 25, 2020. http://hdl.handle.net/1853/61091.

MLA Handbook (7th Edition):

Fathi, Shaghayegh. “Targeted delivery of histone deacetylase inhibitors for use in cancer therapy.” 2016. Web. 25 Oct 2020.

Vancouver:

Fathi S. Targeted delivery of histone deacetylase inhibitors for use in cancer therapy. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1853/61091.

Council of Science Editors:

Fathi S. Targeted delivery of histone deacetylase inhibitors for use in cancer therapy. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/61091


University of Miami

17. Plotkin, Amy Jayne. The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling.

Degree: PhD, Cancer Biology (Medicine), 2014, University of Miami

  Estrogen receptor negative (ER-) breast cancer is more aggressive and associated with both shorter disease-free and overall survival than ER positive breast cancer. Anti-estrogen… (more)

Subjects/Keywords: breast cancer; estrogen receptor; histone deacetylase; histone acetylation; transcriptional repression

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APA (6th Edition):

Plotkin, A. J. (2014). The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1268

Chicago Manual of Style (16th Edition):

Plotkin, Amy Jayne. “The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling.” 2014. Doctoral Dissertation, University of Miami. Accessed October 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1268.

MLA Handbook (7th Edition):

Plotkin, Amy Jayne. “The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling.” 2014. Web. 25 Oct 2020.

Vancouver:

Plotkin AJ. The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling. [Internet] [Doctoral dissertation]. University of Miami; 2014. [cited 2020 Oct 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1268.

Council of Science Editors:

Plotkin AJ. The Role of Class I Histone Deacetylases in the Transcriptional Repression of Estrogen Receptor in Response to hMAPK Signaling. [Doctoral Dissertation]. University of Miami; 2014. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1268

18. Lancelot, Julien. Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets.

Degree: Docteur es, Parasitologie et mycologie, 2013, Université Lille II – Droit et Santé

 La schistosomiase représente actuellement la seconde endémie parasitaire mondiale après le paludisme. Annuellement, cette pathologie est responsable de 280 000 décès et 700 millions d’individus… (more)

Subjects/Keywords: Schistosoma mansoni; Sirtuines; Epigénomique; Histone deacetylases; Schistosomiasis; Histone Deacetylase; Mansoni sirtuin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lancelot, J. (2013). Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2013LIL2S027

Chicago Manual of Style (16th Edition):

Lancelot, Julien. “Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets.” 2013. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed October 25, 2020. http://www.theses.fr/2013LIL2S027.

MLA Handbook (7th Edition):

Lancelot, Julien. “Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets.” 2013. Web. 25 Oct 2020.

Vancouver:

Lancelot J. Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2013. [cited 2020 Oct 25]. Available from: http://www.theses.fr/2013LIL2S027.

Council of Science Editors:

Lancelot J. Caractérisation des sirtuines de Schistosoma mansoni : cibles thérapeutiques potentielles : Charaterization of Schistosoma mansoni sirtuins : potential therapeutic targets. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2013. Available from: http://www.theses.fr/2013LIL2S027

19. 甲斐, 健太郎. Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.

Degree: 博士(医学), 2016, Oita University / 大分大学

PROBLEM: The purpose of this study is to evaluate the involvement of death receptor (DR) 6 in the pathogenesis of endometriosis.

METHODS OF STUDY: Endometriotic… (more)

Subjects/Keywords: Apoptosis; death receptor 6; epigenetics; endometriosis; histone deacetylase inhibitor; proliferation

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APA (6th Edition):

甲斐, . (2016). Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

甲斐, 健太郎. “Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.” 2016. Thesis, Oita University / 大分大学. Accessed October 25, 2020. http://hdl.handle.net/10559/15603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

甲斐, 健太郎. “Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.” 2016. Web. 25 Oct 2020.

Vancouver:

甲斐 . Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10559/15603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

甲斐 . Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 牟田(井生), 久美子. SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する.

Degree: 博士(医学), 2015, Nagasaki University / 長崎大学

 Objective: Long-term peritoneal dialysis causes peritoneal fibrosis in submesothelial areas. However, the mechanism of peritoneal fibrosis is unclear. Epigenetics is the mechanism to induce heritable… (more)

Subjects/Keywords: Chlorhexidine gluconate; Histone deacetylase inhibitor; Peritoneal fibrosis; SAHA; Suberoylanilide hydroxamic acid

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

牟田(井生), . (2015). SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

牟田(井生), 久美子. “SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed October 25, 2020. http://hdl.handle.net/10069/35978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

牟田(井生), 久美子. “SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する.” 2015. Web. 25 Oct 2020.

Vancouver:

牟田(井生) . SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10069/35978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

牟田(井生) . SAHA Suppresses Peritoneal Fibrosis in Mice : SAHAがマウスの腹膜線維化を抑制する. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/35978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

21. Doyle, Kelly. Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites.

Degree: PhD, Medicinal Chemistry;, 2010, University of Utah

 Redox signaling is a mechanism that facilitates homeostasis during redox insult resulting from cellular respiration, defense, and inflammation. Cellular perception of, and adaptation to redox… (more)

Subjects/Keywords: Cyclooxygenase; Cyclopentenone; Histone deacetylase; Lipoxygenase; Peroxiredoxin; Redox Biochemistry

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APA (6th Edition):

Doyle, K. (2010). Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/930/rec/853

Chicago Manual of Style (16th Edition):

Doyle, Kelly. “Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites.” 2010. Doctoral Dissertation, University of Utah. Accessed October 25, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/930/rec/853.

MLA Handbook (7th Edition):

Doyle, Kelly. “Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites.” 2010. Web. 25 Oct 2020.

Vancouver:

Doyle K. Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Oct 25]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/930/rec/853.

Council of Science Editors:

Doyle K. Oxidation of peroxiredoxins and carbonylation of class I histone deacetylases by arachidonic acid metabolites. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/930/rec/853


University of Otago

22. Nutsford, Ashley. The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection .

Degree: University of Otago

 Influenza A virus (IAV) continues to have a major impact on global public health by causing regular seasonal epidemics, unpredictable pandemics and zoonotic outbreaks. These… (more)

Subjects/Keywords: Influenza; Histone Deacetylase 11; HDAC11

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nutsford, A. (n.d.). The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/7494

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Nutsford, Ashley. “The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection .” Masters Thesis, University of Otago. Accessed October 25, 2020. http://hdl.handle.net/10523/7494.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Nutsford, Ashley. “The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection .” Web. 25 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Nutsford A. The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection . [Internet] [Masters thesis]. University of Otago; [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10523/7494.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Nutsford A. The Antiviral Potential of Host Histone Deacetylase 11 during Influenza Virus Infection . [Masters Thesis]. University of Otago; Available from: http://hdl.handle.net/10523/7494

Note: this citation may be lacking information needed for this citation format:
No year of publication.

23. Lopez, Jeffrey. Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases.

Degree: PhD, Chemical Biology, 2017, University of Michigan

 Lysine acetylation is a dynamic post-translational modification occurring ubiquitously in cells. The histone deacetylase (HDAC) family catalyzes the removal of an acetyl group from the… (more)

Subjects/Keywords: Histone deacetylase 8; protein substrates; Chemical Biology; Acetylation; Biological Chemistry; Science

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APA (6th Edition):

Lopez, J. (2017). Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138676

Chicago Manual of Style (16th Edition):

Lopez, Jeffrey. “Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases.” 2017. Doctoral Dissertation, University of Michigan. Accessed October 25, 2020. http://hdl.handle.net/2027.42/138676.

MLA Handbook (7th Edition):

Lopez, Jeffrey. “Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases.” 2017. Web. 25 Oct 2020.

Vancouver:

Lopez J. Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2027.42/138676.

Council of Science Editors:

Lopez J. Understanding HDAC8-Catalyzed Deacetylation: From New Substrates to Diseases. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138676


Drexel University

24. Kalinski, Ashley Lauren. Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons.

Degree: 2015, Drexel University

Neurons are a distinct population of cells that communicate information allowing us to think, move and respond to our environment. A typical neuron has a… (more)

Subjects/Keywords: Neurosciences; Axon; Messenger RNA; Histone deacetylase; Genetic translation; Regeneration (Biology)

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APA (6th Edition):

Kalinski, A. L. (2015). Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalinski, Ashley Lauren. “Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons.” 2015. Thesis, Drexel University. Accessed October 25, 2020. http://hdl.handle.net/1860/idea:7474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalinski, Ashley Lauren. “Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons.” 2015. Web. 25 Oct 2020.

Vancouver:

Kalinski AL. Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons. [Internet] [Thesis]. Drexel University; 2015. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1860/idea:7474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalinski AL. Alterations in the Local Environment of Axons Converge on Intrinsic Pathways that Support Growth of Adult Neurons. [Thesis]. Drexel University; 2015. Available from: http://hdl.handle.net/1860/idea:7474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

25. Wang, Mengqiao. A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints.

Degree: PhD, Molecular and Cell Biology, 2013, Cornell University

 An increasing number of cellular activities are under the regulation of lysine acetylation. This post-translational modification (PTM) is reversibly catalyzed by histone acetyltransferases (HATs) and… (more)

Subjects/Keywords: Histone deacetylase Hos3; Morphogenesis checkpoint; Spindle positioning checkpoint

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APA (6th Edition):

Wang, M. (2013). A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33908

Chicago Manual of Style (16th Edition):

Wang, Mengqiao. “A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints.” 2013. Doctoral Dissertation, Cornell University. Accessed October 25, 2020. http://hdl.handle.net/1813/33908.

MLA Handbook (7th Edition):

Wang, Mengqiao. “A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints.” 2013. Web. 25 Oct 2020.

Vancouver:

Wang M. A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1813/33908.

Council of Science Editors:

Wang M. A Histone Deacetylase Hos3 Establishes Crosslink Between The Morphogenesis And Spindle Positioning Checkpoints. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33908


University of Saskatchewan

26. Sander, Michelle. The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines.

Degree: 2016, University of Saskatchewan

Histone deacetylase inhibitors (HDIs) are a novel class of chemotherapeutics that have potent anti-proliferative and cytotoxic properties in many cancer-derived cell lines. Research has demonstrated… (more)

Subjects/Keywords: histone deacetylase inhibitors; breast cancer; SRC; MYC; miRNA

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APA (6th Edition):

Sander, M. (2016). The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2016-03-2531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sander, Michelle. “The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines.” 2016. Thesis, University of Saskatchewan. Accessed October 25, 2020. http://hdl.handle.net/10388/ETD-2016-03-2531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sander, Michelle. “The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines.” 2016. Web. 25 Oct 2020.

Vancouver:

Sander M. The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10388/ETD-2016-03-2531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sander M. The effect of histone deacetylase inhibitors on gene expression in breast cancer cell lines. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/ETD-2016-03-2531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Tech University

27. Chakraborty, Arup. A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model.

Degree: PhD, Biology, 2011, Texas Tech University

 Acquired resistance to histone deacetylase inhibitors (HDIs), a new group of anti-cancer agents, limits their clinical efficacy. A detailed understanding of the mechanisms of resistance… (more)

Subjects/Keywords: biology; romidepsin; cutaneous T-cell; MAPK pathway; Bim; histone deacetylase inhibitors

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APA (6th Edition):

Chakraborty, A. (2011). A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model. (Doctoral Dissertation). Texas Tech University. Retrieved from http://hdl.handle.net/2346/67746

Chicago Manual of Style (16th Edition):

Chakraborty, Arup. “A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model.” 2011. Doctoral Dissertation, Texas Tech University. Accessed October 25, 2020. http://hdl.handle.net/2346/67746.

MLA Handbook (7th Edition):

Chakraborty, Arup. “A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model.” 2011. Web. 25 Oct 2020.

Vancouver:

Chakraborty A. A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model. [Internet] [Doctoral dissertation]. Texas Tech University; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2346/67746.

Council of Science Editors:

Chakraborty A. A study of mechanisms of resistance to the histone deacetylase inhibitor romidepsin in a cutaneous T-cell lymphoma model. [Doctoral Dissertation]. Texas Tech University; 2011. Available from: http://hdl.handle.net/2346/67746


Wayne State University

28. Aubie, Emily Lynn. Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors.

Degree: PhD, Chemistry, 2011, Wayne State University

Histone Deacetylase (HDAC) proteins are transcriptional regulators that affect histone proteins, which are involved in packaging of DNA into chromosomes. HDACs have been linked… (more)

Subjects/Keywords: ELISA; HDACi; Histone Deacetylase; yeast three hybrid; Biochemistry; Chemistry; Organic Chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aubie, E. L. (2011). Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/301

Chicago Manual of Style (16th Edition):

Aubie, Emily Lynn. “Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors.” 2011. Doctoral Dissertation, Wayne State University. Accessed October 25, 2020. https://digitalcommons.wayne.edu/oa_dissertations/301.

MLA Handbook (7th Edition):

Aubie, Emily Lynn. “Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors.” 2011. Web. 25 Oct 2020.

Vancouver:

Aubie EL. Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2020 Oct 25]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/301.

Council of Science Editors:

Aubie EL. Development of chemical inducers of dimerization for screening competitive histone deactelyase inhibitors. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/301


University of Guelph

29. Syed, Zafir. The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas.

Degree: MS, Department of Pathobiology, 2014, University of Guelph

 Survival of patients with high-grade gliomas is dismal. Oncolytic viruses (OV) and histone deacetylase inhibitors (HDIs) represent revolutionary approaches to treating cancers, with minimal toxic… (more)

Subjects/Keywords: oncolytic virotherapy; histone deacetylase inhibitor; brain cancer; glioma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Syed, Z. (2014). The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8291

Chicago Manual of Style (16th Edition):

Syed, Zafir. “The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas.” 2014. Masters Thesis, University of Guelph. Accessed October 25, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8291.

MLA Handbook (7th Edition):

Syed, Zafir. “The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas.” 2014. Web. 25 Oct 2020.

Vancouver:

Syed Z. The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas. [Internet] [Masters thesis]. University of Guelph; 2014. [cited 2020 Oct 25]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8291.

Council of Science Editors:

Syed Z. The Combination of Histone Deacetylase Inhibitors and Oncolytic Viruses for the treatment of High-grade Gliomas. [Masters Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8291


University of Guelph

30. Ternamian, Christian. Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition.

Degree: MS, Department of Pathobiology, 2014, University of Guelph

 B-cell acute lymphoblastic leukemia (B-ALL) is a hematological disease characterized by the proliferation and extramedullary distribution of malignant B lymphoblasts. Though conventional treatment can be… (more)

Subjects/Keywords: Oncolytic Virotherapy; Histone deacetylase inhibitor; L1210; CD45; Leukemia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ternamian, C. (2014). Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8293

Chicago Manual of Style (16th Edition):

Ternamian, Christian. “Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition.” 2014. Masters Thesis, University of Guelph. Accessed October 25, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8293.

MLA Handbook (7th Edition):

Ternamian, Christian. “Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition.” 2014. Web. 25 Oct 2020.

Vancouver:

Ternamian C. Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition. [Internet] [Masters thesis]. University of Guelph; 2014. [cited 2020 Oct 25]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8293.

Council of Science Editors:

Ternamian C. Targeting B Cell Acute Lymphoblastic Leukemia with Oncolytic Virotherapy and Histone Deacetylase Inhibition. [Masters Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8293

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