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You searched for subject:(hepatotoxicity). Showing records 1 – 30 of 101 total matches.

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Penn State University

1. James, Karma Delores. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism .

Degree: 2016, Penn State University

 A growing body of evidence suggests the hepatotoxic potential of high doses of EGCG. In vitro several mechanisms of EGCG cytotoxicity have been examined; however… (more)

Subjects/Keywords: EGCG; hepatotoxicity; bioavailability; mitochondria; antioxidant

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

James, K. D. (2016). Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/28937

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

James, Karma Delores. “Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism .” 2016. Thesis, Penn State University. Accessed January 27, 2021. https://submit-etda.libraries.psu.edu/catalog/28937.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

James, Karma Delores. “Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism .” 2016. Web. 27 Jan 2021.

Vancouver:

James KD. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism . [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Jan 27]. Available from: https://submit-etda.libraries.psu.edu/catalog/28937.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

James KD. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism . [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/28937

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boise State University

2. Lamb, Cheri Lou. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.

Degree: 2016, Boise State University

 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) is a persistent environmental pollutant and high-affinity ligand for the aryl hydrocarbon receptor (AhR). Exposure to TCDD elicits a spectrum of toxic effects,… (more)

Subjects/Keywords: Wound Healing; Inflammation; Hepatotoxicity; Toxicology

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APA (6th Edition):

Lamb, C. L. (2016). Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. (Thesis). Boise State University. Retrieved from https://scholarworks.boisestate.edu/td/1114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lamb, Cheri Lou. “Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.” 2016. Thesis, Boise State University. Accessed January 27, 2021. https://scholarworks.boisestate.edu/td/1114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lamb, Cheri Lou. “Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.” 2016. Web. 27 Jan 2021.

Vancouver:

Lamb CL. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. [Internet] [Thesis]. Boise State University; 2016. [cited 2021 Jan 27]. Available from: https://scholarworks.boisestate.edu/td/1114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lamb CL. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. [Thesis]. Boise State University; 2016. Available from: https://scholarworks.boisestate.edu/td/1114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

3. García Cañaveras, Juan Carlos. Metabolomics as a tool for the study of drug-induced hepatotoxicity .

Degree: 2015, Universitat de Valencia

 Drug-induced liver injury (DILI) is a major health and economic problem and the leading cause of hepatic dysfunction, drug failure during clinical testing and post-market… (more)

Subjects/Keywords: metabolomics; hepatotoxicity; lc-ms

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APA (6th Edition):

García Cañaveras, J. C. (2015). Metabolomics as a tool for the study of drug-induced hepatotoxicity . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/42776

Chicago Manual of Style (16th Edition):

García Cañaveras, Juan Carlos. “Metabolomics as a tool for the study of drug-induced hepatotoxicity .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed January 27, 2021. http://hdl.handle.net/10550/42776.

MLA Handbook (7th Edition):

García Cañaveras, Juan Carlos. “Metabolomics as a tool for the study of drug-induced hepatotoxicity .” 2015. Web. 27 Jan 2021.

Vancouver:

García Cañaveras JC. Metabolomics as a tool for the study of drug-induced hepatotoxicity . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10550/42776.

Council of Science Editors:

García Cañaveras JC. Metabolomics as a tool for the study of drug-induced hepatotoxicity . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/42776


Addis Ababa University

4. Getnet, Yimer. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .

Degree: 2005, Addis Ababa University

 Anti-tuberculosis drug induced hepatotoxicity (DIH) is a common problem in the management of tuberculosis. This study was intended to identify possible risk factors for development… (more)

Subjects/Keywords: Tuberculosis; HIV; Hepatotoxicity

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APA (6th Edition):

Getnet, Y. (2005). Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . (Thesis). Addis Ababa University. Retrieved from http://etd.aau.edu.et/dspace/handle/123456789/5674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Getnet, Yimer. “Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .” 2005. Thesis, Addis Ababa University. Accessed January 27, 2021. http://etd.aau.edu.et/dspace/handle/123456789/5674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Getnet, Yimer. “Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .” 2005. Web. 27 Jan 2021.

Vancouver:

Getnet Y. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . [Internet] [Thesis]. Addis Ababa University; 2005. [cited 2021 Jan 27]. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Getnet Y. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . [Thesis]. Addis Ababa University; 2005. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

5. Petronijević, Marija M., 1978-. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.

Degree: Farmaceutski fakultet, 2014, Univerzitet u Beogradu

Farmakologija - Farmakoepidemiologija / Pharmacology - Pharmacoepidemiology

Usled mogućih veoma ozbiljnih kliničkih posledica i velikih poteškoća pri otkrivanju i dijagnostikovanju, hepatotoksičnost lekova ili lekovima izazvano… (more)

Subjects/Keywords: hepatotoxicity; adverse reactions; spontaneous reporting; pharmacovigilance

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APA (6th Edition):

Petronijević, Marija M., 1. (2014). Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petronijević, Marija M., 1978-. “Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.” 2014. Thesis, Univerzitet u Beogradu. Accessed January 27, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petronijević, Marija M., 1978-. “Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.” 2014. Web. 27 Jan 2021.

Vancouver:

Petronijević, Marija M. 1. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2021 Jan 27]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petronijević, Marija M. 1. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

6. Chang, Shiow-Huey. Adherence Issues of Isoniazid Therapy Among Children.

Degree: Nursing, 2013, University of California – San Francisco

 An estimated 8.7 million individuals worldwide are infected with the bacillus mycobacterium tuberculosis, with 1.4 million deaths from tuberculosis (TB) in 2011. A 9-month course… (more)

Subjects/Keywords: Nursing; hepatotoxicity; LTBI; stigma; transaminase; tuberculosis

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APA (6th Edition):

Chang, S. (2013). Adherence Issues of Isoniazid Therapy Among Children. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/1tt2t0t4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Shiow-Huey. “Adherence Issues of Isoniazid Therapy Among Children.” 2013. Thesis, University of California – San Francisco. Accessed January 27, 2021. http://www.escholarship.org/uc/item/1tt2t0t4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Shiow-Huey. “Adherence Issues of Isoniazid Therapy Among Children.” 2013. Web. 27 Jan 2021.

Vancouver:

Chang S. Adherence Issues of Isoniazid Therapy Among Children. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Jan 27]. Available from: http://www.escholarship.org/uc/item/1tt2t0t4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang S. Adherence Issues of Isoniazid Therapy Among Children. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/1tt2t0t4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

7. Aung, Aung. Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis.

Degree: MPH, Public Health, 2018, Georgia State University

  BACKGROUND Multidrug-resistant (MDR) tuberculosis (TB) is a public health emergency that causes substantial morbidity and mortality. Treatment of MDR-TB requires 9-24 months of second-line… (more)

Subjects/Keywords: MDR-TB; HCV; HIV; Treatment outcome; Hepatotoxicity

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APA (6th Edition):

Aung, A. (2018). Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/iph_theses/616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aung, Aung. “Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis.” 2018. Thesis, Georgia State University. Accessed January 27, 2021. https://scholarworks.gsu.edu/iph_theses/616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aung, Aung. “Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis.” 2018. Web. 27 Jan 2021.

Vancouver:

Aung A. Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis. [Internet] [Thesis]. Georgia State University; 2018. [cited 2021 Jan 27]. Available from: https://scholarworks.gsu.edu/iph_theses/616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aung A. Hepatitis C and Risk of Adverse Treatment Outcomes among Patients with HIV and Multidrug-resistant Tuberculosis. [Thesis]. Georgia State University; 2018. Available from: https://scholarworks.gsu.edu/iph_theses/616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

8. Van Tonder, Jacob John. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .

Degree: 2012, University of Pretoria

 In vitro testing includes both cell-based and cell-free systems that can be used to detect toxicity induced by xenobiotics. In vitro methods are especially useful… (more)

Subjects/Keywords: Apoptosis; Cyp1a1; Ddd; Dde; Ddt; Hepatotoxicity; Mechanistic profiling; UCTD

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APA (6th Edition):

Van Tonder, J. J. (2012). Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . (Doctoral Dissertation). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-04262012-112100/

Chicago Manual of Style (16th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .” 2012. Doctoral Dissertation, University of Pretoria. Accessed January 27, 2021. http://upetd.up.ac.za/thesis/available/etd-04262012-112100/.

MLA Handbook (7th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .” 2012. Web. 27 Jan 2021.

Vancouver:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . [Internet] [Doctoral dissertation]. University of Pretoria; 2012. [cited 2021 Jan 27]. Available from: http://upetd.up.ac.za/thesis/available/etd-04262012-112100/.

Council of Science Editors:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . [Doctoral Dissertation]. University of Pretoria; 2012. Available from: http://upetd.up.ac.za/thesis/available/etd-04262012-112100/


Universidade do Rio Grande do Sul

9. Rodrigues, Daniela. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.

Degree: 2012, Universidade do Rio Grande do Sul

O transplante de hepatócitos é uma modalidade terapêutica atrativa para as doenças hepáticas, assim como uma alternativa para o transplante hepático. O objetivo do presente… (more)

Subjects/Keywords: Acetaminophen; Acetaminofen; Hepatotoxicity; Gastroenterologia; Toxicidade; Rat hepatocyte transplantation; Hepatócitos; Ratos

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APA (6th Edition):

Rodrigues, D. (2012). Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/66678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Daniela. “Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed January 27, 2021. http://hdl.handle.net/10183/66678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Daniela. “Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.” 2012. Web. 27 Jan 2021.

Vancouver:

Rodrigues D. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10183/66678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues D. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/66678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Martins, Nádia Maria. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.

Degree: PhD, Toxicologia, 2007, University of São Paulo

A cisplatina ainda é um dos agentes quimioterápicos mais efetivos. No entanto, em elevadas doses pode ocorrer hepatotoxicidade. Alguns antioxidantes têm sido mostrado amenizar a… (more)

Subjects/Keywords: cisplatin; cisplatina; citoproteção; cytoprotection; hepatotoxicidade; hepatotoxicity; mitochondria; mitocôndria e EROs; ROS

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APA (6th Edition):

Martins, N. M. (2007). Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;

Chicago Manual of Style (16th Edition):

Martins, Nádia Maria. “Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.” 2007. Doctoral Dissertation, University of São Paulo. Accessed January 27, 2021. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;.

MLA Handbook (7th Edition):

Martins, Nádia Maria. “Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.” 2007. Web. 27 Jan 2021.

Vancouver:

Martins NM. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2021 Jan 27]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;.

Council of Science Editors:

Martins NM. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;

11. Kyriakou, Loukas. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Cadmium is a known industrial and environmental pollutant. It causes hepatotoxicity uponacute administration.Features of cadmium-induced acute hepatoxicity encompass necrosis,apoptosis, peliosis and inflammatory infiltration.Gadolinium chloride (GdCl3)… (more)

Subjects/Keywords: Ηπατοτοξικότητα; Κάδμιο; Γαδολίνιο; Νέκρωση; Ηπατοκύτταρα; Hepatotoxicity; Cadmium; Gadolinium; Necrosis; Hepatocytes

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APA (6th Edition):

Kyriakou, L. (2015). Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kyriakou, Loukas. “Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 27, 2021. http://hdl.handle.net/10442/hedi/42861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kyriakou, Loukas. “Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.” 2015. Web. 27 Jan 2021.

Vancouver:

Kyriakou L. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10442/hedi/42861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kyriakou L. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/42861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Dimou, Pantelis. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

In this paper, it has been studies the hepatotoxicity og danazol after a partial hepatectomy, as well as after an intramuscular drug administration, which both… (more)

Subjects/Keywords: Δαναζόλη; Ηπατοτοξικότητα; Ηπατεκτομή; Φάρμακα; Θειοκεταμίδη; Danazol; Hepatotoxicity; Hepatectomy; Drugs; Thioakeatmidi

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dimou, P. (2014). Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/43669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dimou, Pantelis. “Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 27, 2021. http://hdl.handle.net/10442/hedi/43669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dimou, Pantelis. “Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.” 2014. Web. 27 Jan 2021.

Vancouver:

Dimou P. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10442/hedi/43669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dimou P. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/43669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

13. Ghazi, Terisha. Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity.

Degree: 2019, University of KwaZulu-Natal

 The Fusarium-produced mycotoxin, Fusaric acid (FA), is a frequent contaminant of agricultural foods that exhibits toxicity in plants and animals with little information on its… (more)

Subjects/Keywords: Fusaric acid (FA).; Hepatotoxicity.; HepG2 cells.; DNA methylation.; Fusarium.

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APA (6th Edition):

Ghazi, T. (2019). Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/18611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghazi, Terisha. “Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity.” 2019. Thesis, University of KwaZulu-Natal. Accessed January 27, 2021. https://researchspace.ukzn.ac.za/handle/10413/18611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghazi, Terisha. “Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity.” 2019. Web. 27 Jan 2021.

Vancouver:

Ghazi T. Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity. [Internet] [Thesis]. University of KwaZulu-Natal; 2019. [cited 2021 Jan 27]. Available from: https://researchspace.ukzn.ac.za/handle/10413/18611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghazi T. Fusaric acid-induced epigenetic modifications in vitro and in vivo: alternative mechanisms of hepatotoxicity. [Thesis]. University of KwaZulu-Natal; 2019. Available from: https://researchspace.ukzn.ac.za/handle/10413/18611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Umar, Shahid. Studies on some aspects of acute phase protein metabolism; -.

Degree: Biochemistry, 1992, Aligarh Muslim University

Abstract not available newline newline

Summary 107-112, Bibliography p. 93-106

Advisors/Committee Members: Sahib, M K.

Subjects/Keywords: Metabolism; Immunologic; Biochemical; Mechanism; Hepatotoxicity

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APA (6th Edition):

Umar, S. (1992). Studies on some aspects of acute phase protein metabolism; -. (Thesis). Aligarh Muslim University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/54043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Umar, Shahid. “Studies on some aspects of acute phase protein metabolism; -.” 1992. Thesis, Aligarh Muslim University. Accessed January 27, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/54043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Umar, Shahid. “Studies on some aspects of acute phase protein metabolism; -.” 1992. Web. 27 Jan 2021.

Vancouver:

Umar S. Studies on some aspects of acute phase protein metabolism; -. [Internet] [Thesis]. Aligarh Muslim University; 1992. [cited 2021 Jan 27]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Umar S. Studies on some aspects of acute phase protein metabolism; -. [Thesis]. Aligarh Muslim University; 1992. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

15. Van Tonder, Jacob John. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.

Degree: Pharmacology, 2011, University of Pretoria

 In vitro testing includes both cell-based and cell-free systems that can be used to detect toxicity induced by xenobiotics. In vitro methods are especially useful… (more)

Subjects/Keywords: Apoptosis; Cyp1a1; Ddd; Dde; Ddt; Hepatotoxicity; Mechanistic profiling; UCTD

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APA (6th Edition):

Van Tonder, J. (2011). Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. (Doctoral Dissertation). University of Pretoria. Retrieved from http://hdl.handle.net/2263/24162

Chicago Manual of Style (16th Edition):

Van Tonder, Jacob. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.” 2011. Doctoral Dissertation, University of Pretoria. Accessed January 27, 2021. http://hdl.handle.net/2263/24162.

MLA Handbook (7th Edition):

Van Tonder, Jacob. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.” 2011. Web. 27 Jan 2021.

Vancouver:

Van Tonder J. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. [Internet] [Doctoral dissertation]. University of Pretoria; 2011. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/2263/24162.

Council of Science Editors:

Van Tonder J. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. [Doctoral Dissertation]. University of Pretoria; 2011. Available from: http://hdl.handle.net/2263/24162


University of Connecticut

16. Rohrer, Philip. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.

Degree: MS, Pharmaceutical Science, 2014, University of Connecticut

Subjects/Keywords: Nrf2; APAP; liver; gender; hepatotoxicity

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APA (6th Edition):

Rohrer, P. (2014). The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/574

Chicago Manual of Style (16th Edition):

Rohrer, Philip. “The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.” 2014. Masters Thesis, University of Connecticut. Accessed January 27, 2021. https://opencommons.uconn.edu/gs_theses/574.

MLA Handbook (7th Edition):

Rohrer, Philip. “The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.” 2014. Web. 27 Jan 2021.

Vancouver:

Rohrer P. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. [Internet] [Masters thesis]. University of Connecticut; 2014. [cited 2021 Jan 27]. Available from: https://opencommons.uconn.edu/gs_theses/574.

Council of Science Editors:

Rohrer P. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. [Masters Thesis]. University of Connecticut; 2014. Available from: https://opencommons.uconn.edu/gs_theses/574


University of Vienna

17. Strobl, Jennifer Maria. Hepatotoxicity related target side effect association.

Degree: 2019, University of Vienna

Die Leber stellt eines der wichtigsten Organe des Körpers dar und ist für die Gesundheit des Menschen von außerordentlicher Bedeutung. Da die Leber oft mit… (more)

Subjects/Keywords: 44.40 Pharmazie, Pharmazeutika; 44.42 Pharmazeutische Chemie; Lebertoxizität; hepatotoxicity

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APA (6th Edition):

Strobl, J. M. (2019). Hepatotoxicity related target side effect association. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/59503/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Strobl, Jennifer Maria. “Hepatotoxicity related target side effect association.” 2019. Thesis, University of Vienna. Accessed January 27, 2021. http://othes.univie.ac.at/59503/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Strobl, Jennifer Maria. “Hepatotoxicity related target side effect association.” 2019. Web. 27 Jan 2021.

Vancouver:

Strobl JM. Hepatotoxicity related target side effect association. [Internet] [Thesis]. University of Vienna; 2019. [cited 2021 Jan 27]. Available from: http://othes.univie.ac.at/59503/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Strobl JM. Hepatotoxicity related target side effect association. [Thesis]. University of Vienna; 2019. Available from: http://othes.univie.ac.at/59503/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

18. Dinić, Miroslav R., 1988-. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.

Degree: Farmaceutski fakultet, 2018, Univerzitet u Beogradu

Farmacija - Farmaceutska mikrobiologija / Pharmacy - Pharmaceutical Microbiology

Izolacija i karakterizacija bakterijskih biomolekula koji interaguju sa receptorima ćelija domaćina predstavlja kljuĉ za razumevanje mehanizama… (more)

Subjects/Keywords: lactobacilli; exopolysaccharide; postbiotics; autophagy; hepatotoxicity; hyperalgesia; inflammatory mediators; immunosuppression

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APA (6th Edition):

Dinić, Miroslav R., 1. (2018). Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dinić, Miroslav R., 1988-. “Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.” 2018. Thesis, Univerzitet u Beogradu. Accessed January 27, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dinić, Miroslav R., 1988-. “Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.” 2018. Web. 27 Jan 2021.

Vancouver:

Dinić, Miroslav R. 1. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2021 Jan 27]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dinić, Miroslav R. 1. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Kirk, Natalie M. The use of glutathione in canine itraconazole-associated hepatotoxicity.

Degree: MS, VMS-Veterinary Clinical Medcne, 2020, University of Illinois – Urbana-Champaign

 Azole antifungals are essential and effective drugs in treating a variety of superficial and systemic mycoses in veterinary species. Their activity is attributable to inhibition… (more)

Subjects/Keywords: itraconazole; hepatotoxicity; glutathione; canine

…with hepatotoxicity in man (Lewis, et al., 1984; Duarte et al., 1984), in which the… …incidence of KTZ-associated hepatotoxicity is similar to that of ITZ (Lo Re et al., 2016)… …hepatotoxicity, which is often asymptomatic but can lead to serious illness. Hepatotoxicity… …Hepatotoxicity, as measured by elevated serum alanine transferase (ALT) activity, is a… …of these cases are asymptomatic, although the overall incidence of clinical hepatotoxicity… 

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APA (6th Edition):

Kirk, N. M. (2020). The use of glutathione in canine itraconazole-associated hepatotoxicity. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/108015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kirk, Natalie M. “The use of glutathione in canine itraconazole-associated hepatotoxicity.” 2020. Thesis, University of Illinois – Urbana-Champaign. Accessed January 27, 2021. http://hdl.handle.net/2142/108015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kirk, Natalie M. “The use of glutathione in canine itraconazole-associated hepatotoxicity.” 2020. Web. 27 Jan 2021.

Vancouver:

Kirk NM. The use of glutathione in canine itraconazole-associated hepatotoxicity. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2020. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/2142/108015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kirk NM. The use of glutathione in canine itraconazole-associated hepatotoxicity. [Thesis]. University of Illinois – Urbana-Champaign; 2020. Available from: http://hdl.handle.net/2142/108015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

20. Chan, Katherine. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.

Degree: 2008, University of Toronto

Hepatotoxicity is a serious adverse health effect caused by drugs and other chemical toxins generally detected in the later stages of drug development or in… (more)

Subjects/Keywords: xenobiotic metabolism; hepatotoxicity; QSAR; 0383

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APA (6th Edition):

Chan, K. (2008). Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17290

Chicago Manual of Style (16th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Doctoral Dissertation, University of Toronto. Accessed January 27, 2021. http://hdl.handle.net/1807/17290.

MLA Handbook (7th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Web. 27 Jan 2021.

Vancouver:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1807/17290.

Council of Science Editors:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17290


University of Toronto

21. Hanna, Daniel. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.

Degree: PhD, 2018, University of Toronto

 Liver cancer is the third most common cause of cancer-related death in humans and is 2-4 times more common in men than in women for… (more)

Subjects/Keywords: Hepatotoxicity; Inflammation; Liver cancer; Mouse models; Procarcinogens; Sex differences; 0419

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APA (6th Edition):

Hanna, D. (2018). Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91754

Chicago Manual of Style (16th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Doctoral Dissertation, University of Toronto. Accessed January 27, 2021. http://hdl.handle.net/1807/91754.

MLA Handbook (7th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Web. 27 Jan 2021.

Vancouver:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1807/91754.

Council of Science Editors:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91754

22. Orbach, Sophia Michelle. Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis.

Degree: PhD, Chemical Engineering, 2018, Virginia Tech

 The liver is responsible for lipid and glucose metabolism, protein and bile synthesis and the biotransformation of xenobiotics. These functions, performed by hepatocytes, are dependent… (more)

Subjects/Keywords: organotypic culture models; liver; multi-cellular; hepatotoxicity; acetaminophen; high-throughput; fibrosis

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APA (6th Edition):

Orbach, S. M. (2018). Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/93313

Chicago Manual of Style (16th Edition):

Orbach, Sophia Michelle. “Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis.” 2018. Doctoral Dissertation, Virginia Tech. Accessed January 27, 2021. http://hdl.handle.net/10919/93313.

MLA Handbook (7th Edition):

Orbach, Sophia Michelle. “Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis.” 2018. Web. 27 Jan 2021.

Vancouver:

Orbach SM. Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis. [Internet] [Doctoral dissertation]. Virginia Tech; 2018. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10919/93313.

Council of Science Editors:

Orbach SM. Multi-Cellular Organotypic Liver Models for the Investigation of Chemical Toxicity and Liver Fibrosis. [Doctoral Dissertation]. Virginia Tech; 2018. Available from: http://hdl.handle.net/10919/93313


University of Sydney

23. Mach, John. The effect of ageing on drug induced liver injury: insights from animal models .

Degree: 2015, University of Sydney

 Historically, older people (>65 years) were thought to be at increased risk of drug induced liver injury (DILI). This has not been well established empirically.… (more)

Subjects/Keywords: Drug induced liver injury; paracetamol; isoniazid; hepatotoxicity; ageing

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APA (6th Edition):

Mach, J. (2015). The effect of ageing on drug induced liver injury: insights from animal models . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/14689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mach, John. “The effect of ageing on drug induced liver injury: insights from animal models .” 2015. Thesis, University of Sydney. Accessed January 27, 2021. http://hdl.handle.net/2123/14689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mach, John. “The effect of ageing on drug induced liver injury: insights from animal models .” 2015. Web. 27 Jan 2021.

Vancouver:

Mach J. The effect of ageing on drug induced liver injury: insights from animal models . [Internet] [Thesis]. University of Sydney; 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/2123/14689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mach J. The effect of ageing on drug induced liver injury: insights from animal models . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/14689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Paludetto, Marie-Noëlle. Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects.

Degree: Docteur es, Pharmacologie, 2019, Université Toulouse III – Paul Sabatier

Le pazopanib et le sunitinib sont des inhibiteurs de tyrosine kinases (ITK) indiqués dans le traitement de différents cancers. Ils s'administrent par voie orale au… (more)

Subjects/Keywords: Inhibiteurs de tyrosine kinases; Métabolites réactifs; Hépatotoxicité; Interactions médicamenteuses; Méthode UPLC-MS/MS; Tyrosine kinase inhibitors; Reactive metabolites; Hepatotoxicity; Drug-drug interactions; Hepatotoxicity, drug-drug interactions

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APA (6th Edition):

Paludetto, M. (2019). Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2019TOU30029

Chicago Manual of Style (16th Edition):

Paludetto, Marie-Noëlle. “Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects.” 2019. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 27, 2021. http://www.theses.fr/2019TOU30029.

MLA Handbook (7th Edition):

Paludetto, Marie-Noëlle. “Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects.” 2019. Web. 27 Jan 2021.

Vancouver:

Paludetto M. Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2019. [cited 2021 Jan 27]. Available from: http://www.theses.fr/2019TOU30029.

Council of Science Editors:

Paludetto M. Etude du métabolisme d'inhibiteurs de tyrosine kinases, caractérisation de nouveaux métabolites réactifs du pazopanib et du sunitinib par une approche de catalyse biomimétique et implication potentielle dans leurs effets indésirables : Metabolism study of tyrosine kinase inhibitors, characterization of new reactive metabolites of pazopanib and sunitinib by a biomimetic catalytic approach and potential involvement in their adverse effects. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2019. Available from: http://www.theses.fr/2019TOU30029

25. Driessen, Marja. Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing.

Degree: 2014, Leiden Academic Centre for Drug Research (LACDR), Deparment Toxicology, Faculty of Science, Leiden University

 In this thesis we showed the applicability of the zebrafish embryo as an alternative model for hepatotoxicity testing using analysis of mechanisms through toxicogenomics. By… (more)

Subjects/Keywords: Zebrafish embryo model; Hepatotoxicity; Toxicogenomics; Zebrafish embryo model; Hepatotoxicity; Toxicogenomics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Driessen, M. (2014). Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing. (Doctoral Dissertation). Leiden Academic Centre for Drug Research (LACDR), Deparment Toxicology, Faculty of Science, Leiden University. Retrieved from http://hdl.handle.net/1887/30138

Chicago Manual of Style (16th Edition):

Driessen, Marja. “Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing.” 2014. Doctoral Dissertation, Leiden Academic Centre for Drug Research (LACDR), Deparment Toxicology, Faculty of Science, Leiden University. Accessed January 27, 2021. http://hdl.handle.net/1887/30138.

MLA Handbook (7th Edition):

Driessen, Marja. “Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing.” 2014. Web. 27 Jan 2021.

Vancouver:

Driessen M. Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing. [Internet] [Doctoral dissertation]. Leiden Academic Centre for Drug Research (LACDR), Deparment Toxicology, Faculty of Science, Leiden University; 2014. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1887/30138.

Council of Science Editors:

Driessen M. Evaluation of the zebrafish embryo as an alternative model for hepatotoxicity testing. [Doctoral Dissertation]. Leiden Academic Centre for Drug Research (LACDR), Deparment Toxicology, Faculty of Science, Leiden University; 2014. Available from: http://hdl.handle.net/1887/30138


Universidade do Rio Grande do Sul

26. Schemitt, Elizângela Gonçalves. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.

Degree: 2014, Universidade do Rio Grande do Sul

Introdução: A Insuficiência Hepática Aguda Grave (IHAG) é uma síndrome clínica rara, caracterizada por uma disfunção grave e súbita das células do fígado. A tioacetamida… (more)

Subjects/Keywords: Fígado; Hepatotoxicity; Lipid peroxidation; Toxicidade; Free radicals; Falência hepática aguda; Antioxidants; Estresse oxidativo; Glutamina; Liver

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schemitt, E. G. (2014). Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/110198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schemitt, Elizângela Gonçalves. “Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.” 2014. Thesis, Universidade do Rio Grande do Sul. Accessed January 27, 2021. http://hdl.handle.net/10183/110198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schemitt, Elizângela Gonçalves. “Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.” 2014. Web. 27 Jan 2021.

Vancouver:

Schemitt EG. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2014. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10183/110198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schemitt EG. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. [Thesis]. Universidade do Rio Grande do Sul; 2014. Available from: http://hdl.handle.net/10183/110198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. 馬渡, 力. A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.

Degree: 博士(薬学), 2016, Nagasaki University / 長崎大学

 Background: To identify certain genes related to anti-tuberculosis drug-induced hepatotoxicity (ATDH) for Japanese patients with pulmonary tuberculosis (TB), we examined an association study of single… (more)

Subjects/Keywords: Tuberculosis; Anti-tuberculosis drug-induced hepatotoxicity; Single nucleotide polymorphism; Glutathione synthesis; Case-control association study

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

馬渡, . (2016). A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

馬渡, 力. “A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed January 27, 2021. http://hdl.handle.net/10069/36832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

馬渡, 力. “A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.” 2016. Web. 27 Jan 2021.

Vancouver:

馬渡 . A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10069/36832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

馬渡 . A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/36832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Singh, Padam. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.

Degree: Zoology, 2012, Chaudhary Charan Singh University

None newline

Summary p. 51-65, Refrences p. 66-70

Advisors/Committee Members: Malhotra, N M.

Subjects/Keywords: Hepatotoxicity; Haemato Biochemical; Teleost Clarias Batrachus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Singh, P. (2012). Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. (Thesis). Chaudhary Charan Singh University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/40371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Singh, Padam. “Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.” 2012. Thesis, Chaudhary Charan Singh University. Accessed January 27, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/40371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Singh, Padam. “Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.” 2012. Web. 27 Jan 2021.

Vancouver:

Singh P. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. [Internet] [Thesis]. Chaudhary Charan Singh University; 2012. [cited 2021 Jan 27]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/40371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Singh P. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. [Thesis]. Chaudhary Charan Singh University; 2012. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/40371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Fernandes, Fábio Henrique [UNESP]. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.

Degree: 2017, Universidade Estadual Paulista

As consequências da poluição ambiental não se limitam a danos ao ambiente, mas também atingem diretamente a saúde humana. Recentemente, a Organização Mundial de Saúde… (more)

Subjects/Keywords: Azo corantes; Genotoxicidade; Hepatotoxicidade; Mutagênese ambiental; Azo dyes; Environmental mutagenesis; Genotoxicity; Hepatotoxicity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fernandes, F. H. [. (2017). Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/148643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fernandes, Fábio Henrique [UNESP]. “Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.” 2017. Thesis, Universidade Estadual Paulista. Accessed January 27, 2021. http://hdl.handle.net/11449/148643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fernandes, Fábio Henrique [UNESP]. “Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.” 2017. Web. 27 Jan 2021.

Vancouver:

Fernandes FH[. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. [Internet] [Thesis]. Universidade Estadual Paulista; 2017. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/11449/148643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fernandes FH[. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. [Thesis]. Universidade Estadual Paulista; 2017. Available from: http://hdl.handle.net/11449/148643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Acar, Özge. Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .

Degree: ESOGÜ, Fen Edebiyat Fakültesi, Biyoloji, 2015, Eskisehir Osmangazi University

 Siklofosfamid (CP) klinikte kanser ve non-malignant hastalıkların tedavisinde yaygın olarak kullanılan, yüksek derecede etkili, alkilleyici sitotoksik bir ilaçtır. CP’nin antitümoral etkinliği yüksek dozda kullanılmasına bağlıdır.… (more)

Subjects/Keywords: Siklofosfamid; Oksidatif Stres; Hepatotoksisite; Selenyum; Sitoprotektivite; Sıçan; Cyclophosphamide; Oxidative Stress; Hepatotoxicity; Selenium; Cytoprotectivity; Rat

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APA (6th Edition):

Acar, . (2015). Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Acar, Özge. “Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .” 2015. Thesis, Eskisehir Osmangazi University. Accessed January 27, 2021. http://hdl.handle.net/11684/444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Acar, Özge. “Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .” 2015. Web. 27 Jan 2021.

Vancouver:

Acar . Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . [Internet] [Thesis]. Eskisehir Osmangazi University; 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/11684/444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Acar . Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . [Thesis]. Eskisehir Osmangazi University; 2015. Available from: http://hdl.handle.net/11684/444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4]

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