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You searched for subject:(glioblastoma). Showing records 1 – 30 of 723 total matches.

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Oregon State University

1. Lal, Sangeet Kumar. Calpain 2 proteolysis regulates glioblastoma cell invasion.

Degree: PhD, Biochemistry and Biophysics, 2011, Oregon State University

Glioblastoma is the most malignant primary brain tumor with the average patients surviving only one year after diagnosis, even with aggressive therapy. The formation of… (more)

Subjects/Keywords: Glioblastoma

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APA (6th Edition):

Lal, S. K. (2011). Calpain 2 proteolysis regulates glioblastoma cell invasion. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/19988

Chicago Manual of Style (16th Edition):

Lal, Sangeet Kumar. “Calpain 2 proteolysis regulates glioblastoma cell invasion.” 2011. Doctoral Dissertation, Oregon State University. Accessed February 26, 2020. http://hdl.handle.net/1957/19988.

MLA Handbook (7th Edition):

Lal, Sangeet Kumar. “Calpain 2 proteolysis regulates glioblastoma cell invasion.” 2011. Web. 26 Feb 2020.

Vancouver:

Lal SK. Calpain 2 proteolysis regulates glioblastoma cell invasion. [Internet] [Doctoral dissertation]. Oregon State University; 2011. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/1957/19988.

Council of Science Editors:

Lal SK. Calpain 2 proteolysis regulates glioblastoma cell invasion. [Doctoral Dissertation]. Oregon State University; 2011. Available from: http://hdl.handle.net/1957/19988


Universidad de Cantabria

2. Fernández Fuente, Gonzalo. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.

Degree: 2017, Universidad de Cantabria

 En este trabajo, hemos estudiado la biología de los Glioblastomas, el tumor cerebral primario más frecuente en adultos y que representa uno de los tipos… (more)

Subjects/Keywords: Glioblastoma

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APA (6th Edition):

Fernández Fuente, G. (2017). Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. (Doctoral Dissertation). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/11393

Chicago Manual of Style (16th Edition):

Fernández Fuente, Gonzalo. “Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.” 2017. Doctoral Dissertation, Universidad de Cantabria. Accessed February 26, 2020. http://hdl.handle.net/10902/11393.

MLA Handbook (7th Edition):

Fernández Fuente, Gonzalo. “Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.” 2017. Web. 26 Feb 2020.

Vancouver:

Fernández Fuente G. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. [Internet] [Doctoral dissertation]. Universidad de Cantabria; 2017. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10902/11393.

Council of Science Editors:

Fernández Fuente G. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. [Doctoral Dissertation]. Universidad de Cantabria; 2017. Available from: http://hdl.handle.net/10902/11393


University of Manitoba

3. Yuan, Haynes (Shek Hei). Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.

Degree: Pharmacology and Therapeutics, 2019, University of Manitoba

 Background Glioblastoma Multiforme (GBM) is an aggressive cerebral cancer. Standard chemotherapeutic of GBM is Temozolomide; a DNA alkylating agent. Patients relapse mostly due to recurrence… (more)

Subjects/Keywords: Glioblastoma Multiforme; Glioblastoma Multiforme

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APA (6th Edition):

Yuan, H. (. H. (2019). Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34166

Chicago Manual of Style (16th Edition):

Yuan, Haynes (Shek Hei). “Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.” 2019. Masters Thesis, University of Manitoba. Accessed February 26, 2020. http://hdl.handle.net/1993/34166.

MLA Handbook (7th Edition):

Yuan, Haynes (Shek Hei). “Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.” 2019. Web. 26 Feb 2020.

Vancouver:

Yuan H(H. Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/1993/34166.

Council of Science Editors:

Yuan H(H. Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34166


University of Georgia

4. Huang, Yan. Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines.

Degree: MS, Pharmacy (Pharmaceutics), 2003, University of Georgia

 Low-density lipoprotein (LDL) is the major cholesterol carrier in human plasma. To utilize the elevated expression of LDL receptor on many types of cancer cells,… (more)

Subjects/Keywords: Glioblastoma

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APA (6th Edition):

Huang, Y. (2003). Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines. (Masters Thesis). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/huang_yan_200312_ms

Chicago Manual of Style (16th Edition):

Huang, Yan. “Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines.” 2003. Masters Thesis, University of Georgia. Accessed February 26, 2020. http://purl.galileo.usg.edu/uga_etd/huang_yan_200312_ms.

MLA Handbook (7th Edition):

Huang, Yan. “Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines.” 2003. Web. 26 Feb 2020.

Vancouver:

Huang Y. Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines. [Internet] [Masters thesis]. University of Georgia; 2003. [cited 2020 Feb 26]. Available from: http://purl.galileo.usg.edu/uga_etd/huang_yan_200312_ms.

Council of Science Editors:

Huang Y. Comparative cellular uptake studies of a carborane cholesteryl ester by human glioma cell lines. [Masters Thesis]. University of Georgia; 2003. Available from: http://purl.galileo.usg.edu/uga_etd/huang_yan_200312_ms


Universidade do Rio Grande do Sul

5. Chiela, Eduardo Cremonese Filippi. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.

Degree: 2011, Universidade do Rio Grande do Sul

Glioblastomas (GBMs) são os tumores primários (gliomas) mais comuns e agressivos do Sistema Nervosos Central, classificados pelos oncologistas como um dos maiores desafios da oncoterapia.… (more)

Subjects/Keywords: Glioblastoma; Resveratrol

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APA (6th Edition):

Chiela, E. C. F. (2011). A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/28430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiela, Eduardo Cremonese Filippi. “A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.” 2011. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/28430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiela, Eduardo Cremonese Filippi. “A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.” 2011. Web. 26 Feb 2020.

Vancouver:

Chiela ECF. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2011. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/28430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiela ECF. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. [Thesis]. Universidade do Rio Grande do Sul; 2011. Available from: http://hdl.handle.net/10183/28430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

6. Silva, Mardja Mansur Bueno e. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.

Degree: 2016, Universidade do Rio Grande do Sul

Mitocôndrias desempenham funções celulares vitais. O funcionamento dessas organelas e seu papel no metabolismo celular, não surpreendentemente, têm sido implicados no desenvolvimento de diferentes cânceres.… (more)

Subjects/Keywords: Glioblastoma; Temozolamida

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APA (6th Edition):

Silva, M. M. B. e. (2016). Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Mardja Mansur Bueno e. “Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/150664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Mardja Mansur Bueno e. “Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.” 2016. Web. 26 Feb 2020.

Vancouver:

Silva MMBe. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/150664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva MMBe. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/150664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

7. Folck, Anthony F. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.

Degree: 2016, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Secondary gliomas are an incurable form of brain cancer that are diagnosed in people at a median age of 45… (more)

Subjects/Keywords: Astrocytoma; Glioblastoma

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APA (6th Edition):

Folck, A. F. (2016). A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/11053

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Folck, Anthony F. “A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.” 2016. Thesis, IUPUI. Accessed February 26, 2020. http://hdl.handle.net/1805/11053.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Folck, Anthony F. “A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.” 2016. Web. 26 Feb 2020.

Vancouver:

Folck AF. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. [Internet] [Thesis]. IUPUI; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/1805/11053.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Folck AF. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. [Thesis]. IUPUI; 2016. Available from: http://hdl.handle.net/1805/11053

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Cantabria

8. López López, Carlos. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).

Degree: 2016, Universidad de Cantabria

 RESUMEN: El glioblastoma multiforme (GBM) es una neoplasia relativamente frecuente entre las del sistema nervioso central, que se caracteriza por su excepcional agresividad y por… (more)

Subjects/Keywords: Glioblastoma multiforme

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APA (6th Edition):

López López, C. (2016). Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). (Doctoral Dissertation). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/8433

Chicago Manual of Style (16th Edition):

López López, Carlos. “Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).” 2016. Doctoral Dissertation, Universidad de Cantabria. Accessed February 26, 2020. http://hdl.handle.net/10902/8433.

MLA Handbook (7th Edition):

López López, Carlos. “Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).” 2016. Web. 26 Feb 2020.

Vancouver:

López López C. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). [Internet] [Doctoral dissertation]. Universidad de Cantabria; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10902/8433.

Council of Science Editors:

López López C. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). [Doctoral Dissertation]. Universidad de Cantabria; 2016. Available from: http://hdl.handle.net/10902/8433


Harvard University

9. Sastry, Rahul. The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population.

Degree: Doctor of Medicine, 2017, Harvard University

Background: Despite updated management of glioblastoma (GB), progression is virtually inevitable. Previous data suggest a survival benefit from resection at progression; however, relatively few studies… (more)

Subjects/Keywords: glioblastoma; neurosurgery; neuro-oncology; bevacizumab; progressive glioblastoma

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APA (6th Edition):

Sastry, R. (2017). The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621373

Chicago Manual of Style (16th Edition):

Sastry, Rahul. “The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population.” 2017. Doctoral Dissertation, Harvard University. Accessed February 26, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621373.

MLA Handbook (7th Edition):

Sastry, Rahul. “The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population.” 2017. Web. 26 Feb 2020.

Vancouver:

Sastry R. The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2020 Feb 26]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621373.

Council of Science Editors:

Sastry R. The Impact of Surgery on Survival After Progression of Glioblastoma: A Retrospective Cohort Analysis of a Contemporary Patient Population. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621373


Universidade do Rio Grande do Sul

10. Tamajusuku, Alessandra Sayuri Kikuchi. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.

Degree: 2010, Universidade do Rio Grande do Sul

O nucleotídeo de purina ATP, no meio extracelular, participa de diversos processos fisiológicos e patológicos como vasodilatação/constrição, proliferação, diferenciação, modulação sináptica, dor, inflamação e morte… (more)

Subjects/Keywords: Glioma; Glioblastoma; Camundongos

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APA (6th Edition):

Tamajusuku, A. S. K. (2010). Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Web. 26 Feb 2020.

Vancouver:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

11. Kipper, Franciele Cristina. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.

Degree: 2017, Universidade do Rio Grande do Sul

 Gliomas são tumores do sistema nervoso central caracterizados por alta invasibilidade e mortalidade. Inúmeros esforços foram feitos nas últimas décadas para melhorar a sobrevida dos… (more)

Subjects/Keywords: Glioblastoma; Glioma; Temozolomida

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APA (6th Edition):

Kipper, F. C. (2017). Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Web. 26 Feb 2020.

Vancouver:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

12. Pereira, Mariana Brutschin. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.

Degree: 2017, Universidade do Rio Grande do Sul

Introdução: A complexidade das populações de células do sistema imunológico infiltrando tumores humanos com seus efeitos sinérgicos ou antagônicos pode influenciar os tumores de forma… (more)

Subjects/Keywords: Glioblastoma; Sistema imunológico

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APA (6th Edition):

Pereira, M. B. (2017). Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pereira, Mariana Brutschin. “Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/170192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pereira, Mariana Brutschin. “Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.” 2017. Web. 26 Feb 2020.

Vancouver:

Pereira MB. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/170192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pereira MB. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Victoria University of Wellington

13. Brow, Susanna. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.

Degree: 2015, Victoria University of Wellington

 Objective Glioblastomas (GBMs) are the most prevalent primary brain tumours in adults and the outcome for this disease remains very poor. With treatment options limited,… (more)

Subjects/Keywords: SIRT1; Sirtuin; Glioblastoma

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APA (6th Edition):

Brow, S. (2015). Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/4879

Chicago Manual of Style (16th Edition):

Brow, Susanna. “Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.” 2015. Masters Thesis, Victoria University of Wellington. Accessed February 26, 2020. http://hdl.handle.net/10063/4879.

MLA Handbook (7th Edition):

Brow, Susanna. “Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.” 2015. Web. 26 Feb 2020.

Vancouver:

Brow S. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. [Internet] [Masters thesis]. Victoria University of Wellington; 2015. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10063/4879.

Council of Science Editors:

Brow S. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. [Masters Thesis]. Victoria University of Wellington; 2015. Available from: http://hdl.handle.net/10063/4879


University of Minnesota

14. Becker, Chani. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.

Degree: PhD, Neuroscience, 2016, University of Minnesota

Glioblastoma multiforme (GBM) is a lethal cancer. Without treatment, patients diagnosed with this disease survive nine months. With the best therapeutics science has to offer,… (more)

Subjects/Keywords: BBB; Glioblastoma; Glioma

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APA (6th Edition):

Becker, C. (2016). Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/182319

Chicago Manual of Style (16th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Doctoral Dissertation, University of Minnesota. Accessed February 26, 2020. http://hdl.handle.net/11299/182319.

MLA Handbook (7th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Web. 26 Feb 2020.

Vancouver:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/11299/182319.

Council of Science Editors:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/182319


Universidade do Rio Grande do Sul

15. Silva, Andrew Oliveira. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.

Degree: 2016, Universidade do Rio Grande do Sul

Apesar dos progressos na compreensão da biologia dos Glioblastomas (GBM), poucos avanços terapêuticos foram obtidos, desde que a Temozolomida (TMZ) foi implementada, em 2005, como… (more)

Subjects/Keywords: Glioblastoma; Temozolomida; Vimblastina

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APA (6th Edition):

Silva, A. O. (2016). Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Andrew Oliveira. “Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/150589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Andrew Oliveira. “Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.” 2016. Web. 26 Feb 2020.

Vancouver:

Silva AO. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/150589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva AO. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/150589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

16. Jindani, Rajika. Glioblastoma multiforme: etiology, progression, and treatment.

Degree: MS, Medical Sciences, 2017, Boston University

Glioblastoma multiforme is the most common and malignant brain tumor, accounting for more than 52% of all primary brain tumors. The molecular heterogeneity of the… (more)

Subjects/Keywords: Medicine; GBM; Glioblastoma

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APA (6th Edition):

Jindani, R. (2017). Glioblastoma multiforme: etiology, progression, and treatment. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/26730

Chicago Manual of Style (16th Edition):

Jindani, Rajika. “Glioblastoma multiforme: etiology, progression, and treatment.” 2017. Masters Thesis, Boston University. Accessed February 26, 2020. http://hdl.handle.net/2144/26730.

MLA Handbook (7th Edition):

Jindani, Rajika. “Glioblastoma multiforme: etiology, progression, and treatment.” 2017. Web. 26 Feb 2020.

Vancouver:

Jindani R. Glioblastoma multiforme: etiology, progression, and treatment. [Internet] [Masters thesis]. Boston University; 2017. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/2144/26730.

Council of Science Editors:

Jindani R. Glioblastoma multiforme: etiology, progression, and treatment. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/26730


University of Ottawa

17. Gont, Alexander. Inactivation of Lgl1 in Glioblastoma .

Degree: 2016, University of Ottawa

Glioblastoma is the most aggressive and invasive adult brain cancer. In glioblastoma, the loss of the tumour suppressor PTEN is the most common genetic alteration… (more)

Subjects/Keywords: cancer; glioblastoma; Lgl

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APA (6th Edition):

Gont, A. (2016). Inactivation of Lgl1 in Glioblastoma . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gont, Alexander. “Inactivation of Lgl1 in Glioblastoma .” 2016. Thesis, University of Ottawa. Accessed February 26, 2020. http://hdl.handle.net/10393/34965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gont, Alexander. “Inactivation of Lgl1 in Glioblastoma .” 2016. Web. 26 Feb 2020.

Vancouver:

Gont A. Inactivation of Lgl1 in Glioblastoma . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10393/34965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gont A. Inactivation of Lgl1 in Glioblastoma . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

18. Daniel, Paul Marcel. Identification of CREB as a transcriptional regulator of glioblastoma biology.

Degree: 2015, University of Melbourne

Glioblastoma (GBM) is both the most common and malignant type of brain tumour with a median survival of 7.4 months from the date of diagnosis;… (more)

Subjects/Keywords: glioblastoma; CREB; transcription

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APA (6th Edition):

Daniel, P. M. (2015). Identification of CREB as a transcriptional regulator of glioblastoma biology. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/58851

Chicago Manual of Style (16th Edition):

Daniel, Paul Marcel. “Identification of CREB as a transcriptional regulator of glioblastoma biology.” 2015. Doctoral Dissertation, University of Melbourne. Accessed February 26, 2020. http://hdl.handle.net/11343/58851.

MLA Handbook (7th Edition):

Daniel, Paul Marcel. “Identification of CREB as a transcriptional regulator of glioblastoma biology.” 2015. Web. 26 Feb 2020.

Vancouver:

Daniel PM. Identification of CREB as a transcriptional regulator of glioblastoma biology. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/11343/58851.

Council of Science Editors:

Daniel PM. Identification of CREB as a transcriptional regulator of glioblastoma biology. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/58851


University of Toronto

19. Albert-Vartanian, Alenoush. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.

Degree: 2013, University of Toronto

Glioblastoma (GBM), similar to many other cancers, exhibits enhanced aerobic glycolysis with concomitant lactate production, a phenomenon known as the Warburg effect. We have demonstrated… (more)

Subjects/Keywords: Metabolism; Glioblastoma; 0992

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APA (6th Edition):

Albert-Vartanian, A. (2013). Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42661

Chicago Manual of Style (16th Edition):

Albert-Vartanian, Alenoush. “Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.” 2013. Masters Thesis, University of Toronto. Accessed February 26, 2020. http://hdl.handle.net/1807/42661.

MLA Handbook (7th Edition):

Albert-Vartanian, Alenoush. “Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.” 2013. Web. 26 Feb 2020.

Vancouver:

Albert-Vartanian A. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/1807/42661.

Council of Science Editors:

Albert-Vartanian A. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42661


University of Houston

20. Avci, Naze Gul. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.

Degree: PhD, Biomedical Engineering, 2015, University of Houston

Glioblastoma multiforme (GBM) is the most malignant primary brain tumor with a high infiltrative capacity and increased vascularity. Despite current therapies overall patient survival rate… (more)

Subjects/Keywords: Glioblastoma; 3D spheroids

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APA (6th Edition):

Avci, N. G. (2015). The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2005

Chicago Manual of Style (16th Edition):

Avci, Naze Gul. “The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.” 2015. Doctoral Dissertation, University of Houston. Accessed February 26, 2020. http://hdl.handle.net/10657/2005.

MLA Handbook (7th Edition):

Avci, Naze Gul. “The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.” 2015. Web. 26 Feb 2020.

Vancouver:

Avci NG. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10657/2005.

Council of Science Editors:

Avci NG. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/2005


Universidade do Rio Grande do Sul

21. Kagami, Luciano Porto. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.

Degree: 2017, Universidade do Rio Grande do Sul

Os tumores cerebrais primários mais comuns são os gliomas. Os gliomas representam 31% de todos os tumores cerebrais diagnosticados nos Estados Unidos sendo 81% destes… (more)

Subjects/Keywords: Medicamentos; Glioblastoma; Modelagem molecular

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APA (6th Edition):

Kagami, L. P. (2017). Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kagami, Luciano Porto. “Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/170643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kagami, Luciano Porto. “Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.” 2017. Web. 26 Feb 2020.

Vancouver:

Kagami LP. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/170643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kagami LP. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

22. Sankaranarayanan, Preethi. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.

Degree: MS, Bioengineering, 2013, University of Utah

 Despite recent large-scale profiling efforts, the best prognostic predictor ofglioblastoma multiforme (GBM) remains the patient’s age at diagnosis. Wedescribe a global pattern of tumor-exclusive co-occurring… (more)

Subjects/Keywords: Copy number; Glioblastoma multiforme; GSVD

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APA (6th Edition):

Sankaranarayanan, P. (2013). Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101

Chicago Manual of Style (16th Edition):

Sankaranarayanan, Preethi. “Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.” 2013. Masters Thesis, University of Utah. Accessed February 26, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101.

MLA Handbook (7th Edition):

Sankaranarayanan, Preethi. “Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.” 2013. Web. 26 Feb 2020.

Vancouver:

Sankaranarayanan P. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. [Internet] [Masters thesis]. University of Utah; 2013. [cited 2020 Feb 26]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101.

Council of Science Editors:

Sankaranarayanan P. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. [Masters Thesis]. University of Utah; 2013. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101


Victoria University of Wellington

23. Carson, Georgia. The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation.

Degree: 2016, Victoria University of Wellington

 Although not recommended by mainstream oncologists, intravenous injections of pharmacological ascorbate are currently an alternative therapy option for cancer patients. Research has not yet determined… (more)

Subjects/Keywords: Glioblastoma; Ascorbate; DNA damage

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APA (6th Edition):

Carson, G. (2016). The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/5196

Chicago Manual of Style (16th Edition):

Carson, Georgia. “The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation.” 2016. Masters Thesis, Victoria University of Wellington. Accessed February 26, 2020. http://hdl.handle.net/10063/5196.

MLA Handbook (7th Edition):

Carson, Georgia. “The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation.” 2016. Web. 26 Feb 2020.

Vancouver:

Carson G. The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation. [Internet] [Masters thesis]. Victoria University of Wellington; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10063/5196.

Council of Science Editors:

Carson G. The Two Faces of Ascorbate: Prooxidant Activity and Radio-Sensitisation. [Masters Thesis]. Victoria University of Wellington; 2016. Available from: http://hdl.handle.net/10063/5196


Victoria University of Wellington

24. Petley, Emma Victoria. Assessment of combined vaccination and immune modulation as an anti-tumour therapy.

Degree: 2016, Victoria University of Wellington

Glioblastoma multiforme (GBM) is a common and lethal type of brain cancer, with a very poor prognosis. Current therapy consisting of surgical resection, radiation and… (more)

Subjects/Keywords: Glioblastoma multiforme; Vaccination; Checkpoint blockade

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APA (6th Edition):

Petley, E. V. (2016). Assessment of combined vaccination and immune modulation as an anti-tumour therapy. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/6172

Chicago Manual of Style (16th Edition):

Petley, Emma Victoria. “Assessment of combined vaccination and immune modulation as an anti-tumour therapy.” 2016. Masters Thesis, Victoria University of Wellington. Accessed February 26, 2020. http://hdl.handle.net/10063/6172.

MLA Handbook (7th Edition):

Petley, Emma Victoria. “Assessment of combined vaccination and immune modulation as an anti-tumour therapy.” 2016. Web. 26 Feb 2020.

Vancouver:

Petley EV. Assessment of combined vaccination and immune modulation as an anti-tumour therapy. [Internet] [Masters thesis]. Victoria University of Wellington; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10063/6172.

Council of Science Editors:

Petley EV. Assessment of combined vaccination and immune modulation as an anti-tumour therapy. [Masters Thesis]. Victoria University of Wellington; 2016. Available from: http://hdl.handle.net/10063/6172


Universidade Estadual de Campinas

25. Ruas, Juliana Silveira, 1989-. Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura .

Degree: 2015, Universidade Estadual de Campinas

 Resumo: A maioria das células tumorais depende da glicólise para a ressíntese de ATP durante um processo de rápida proliferação, mesmo que haja disponibilidade de… (more)

Subjects/Keywords: Glicólise; Glioblastoma; Mitocôndria; Fosforilação oxidativa

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APA (6th Edition):

Ruas, Juliana Silveira, 1. (2015). Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura . (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/313030

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ruas, Juliana Silveira, 1989-. “Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura .” 2015. Thesis, Universidade Estadual de Campinas. Accessed February 26, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313030.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ruas, Juliana Silveira, 1989-. “Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura .” 2015. Web. 26 Feb 2020.

Vancouver:

Ruas, Juliana Silveira 1. Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura . [Internet] [Thesis]. Universidade Estadual de Campinas; 2015. [cited 2020 Feb 26]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313030.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ruas, Juliana Silveira 1. Metabolismo energético mitocondrial na proliferação de células de glioblastoma U-87MG e T98G em cultura . [Thesis]. Universidade Estadual de Campinas; 2015. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313030

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

26. Klank, Rebecca. Physical Determinants of Glioma Cell Migration and Disease Progression.

Degree: PhD, Biomedical Engineering, 2015, University of Minnesota

Glioblastoma (GBM) is a highly aggressive brain cancer (generally, “glioma”) with poor patient prognosis, even with current standard treatments. In order to rationally develop novel… (more)

Subjects/Keywords: CD44; Glioblastoma; Glioma; Tumor Modeling

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APA (6th Edition):

Klank, R. (2015). Physical Determinants of Glioma Cell Migration and Disease Progression. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191324

Chicago Manual of Style (16th Edition):

Klank, Rebecca. “Physical Determinants of Glioma Cell Migration and Disease Progression.” 2015. Doctoral Dissertation, University of Minnesota. Accessed February 26, 2020. http://hdl.handle.net/11299/191324.

MLA Handbook (7th Edition):

Klank, Rebecca. “Physical Determinants of Glioma Cell Migration and Disease Progression.” 2015. Web. 26 Feb 2020.

Vancouver:

Klank R. Physical Determinants of Glioma Cell Migration and Disease Progression. [Internet] [Doctoral dissertation]. University of Minnesota; 2015. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/11299/191324.

Council of Science Editors:

Klank R. Physical Determinants of Glioma Cell Migration and Disease Progression. [Doctoral Dissertation]. University of Minnesota; 2015. Available from: http://hdl.handle.net/11299/191324


University of Rochester

27. Stripay, Jennifer L. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.

Degree: PhD, 2016, University of Rochester

Glioblastoma multiforme is the most common primary brain tumor in adults, the most malignant of all intracranial tumors, and is associated with inevitable recurrence and… (more)

Subjects/Keywords: Cancer; cbl; Glioblastoma; Redox; Therapy

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APA (6th Edition):

Stripay, J. L. (2016). Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/31636

Chicago Manual of Style (16th Edition):

Stripay, Jennifer L. “Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.” 2016. Doctoral Dissertation, University of Rochester. Accessed February 26, 2020. http://hdl.handle.net/1802/31636.

MLA Handbook (7th Edition):

Stripay, Jennifer L. “Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.” 2016. Web. 26 Feb 2020.

Vancouver:

Stripay JL. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/1802/31636.

Council of Science Editors:

Stripay JL. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/31636


Boston University

28. Handley, Meghan Victoria. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.

Degree: MS, Medical Sciences, 2015, Boston University

Glioblastoma multiforme (GBM) is the most malignant form of brain cancer. Therapies targeting glioblastoma have not consistently been able to give those diagnosed the best… (more)

Subjects/Keywords: Medicine; Glioblastoma; GSK-3 inhibitors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Handley, M. V. (2015). GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16063

Chicago Manual of Style (16th Edition):

Handley, Meghan Victoria. “GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.” 2015. Masters Thesis, Boston University. Accessed February 26, 2020. http://hdl.handle.net/2144/16063.

MLA Handbook (7th Edition):

Handley, Meghan Victoria. “GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.” 2015. Web. 26 Feb 2020.

Vancouver:

Handley MV. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/2144/16063.

Council of Science Editors:

Handley MV. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16063


NSYSU

29. Lee, Ya-che. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.

Degree: Master, Biological Sciences, 2016, NSYSU

 Pluchea indica (L.) Less. is a perennial plant known for its versatile uses in traditional oriental medicine. In our previous study, we demonstrate the in… (more)

Subjects/Keywords: Glioblastoma Multiforme; Autophagy; Pluchea indica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, Y. (2016). Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Ya-che. “Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.” 2016. Thesis, NSYSU. Accessed February 26, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Ya-che. “Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.” 2016. Web. 26 Feb 2020.

Vancouver:

Lee Y. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. [Internet] [Thesis]. NSYSU; 2016. [cited 2020 Feb 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee Y. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

30. Silva, Andrew Oliveira. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.

Degree: 2012, Universidade do Rio Grande do Sul

 Gliomas malignos compreendem o subtipo mais comum e devastador de tumores primários do sistema nervoso central (SNC), sendo o Glioblastoma Multiforme (GBM) a forma mais… (more)

Subjects/Keywords: Glioblastoma; Glioma; Apoptose; Autofagia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva, A. O. (2012). Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2020. http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Web. 26 Feb 2020.

Vancouver:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2020 Feb 26]. Available from: http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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