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You searched for subject:(glial scar). Showing records 1 – 26 of 26 total matches.

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University of Arizona

1. Zbesko, Jacob C. Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes .

Degree: 2020, University of Arizona

 Stroke affects nearly 800,000 Americans every year and, of survivors, around one third will develop dementia. Not only personally devastating to sufferers, this disease places… (more)

Subjects/Keywords: B-lymphocyte; Glial scar; Stoke

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zbesko, J. C. (2020). Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/645775

Chicago Manual of Style (16th Edition):

Zbesko, Jacob C. “Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes .” 2020. Doctoral Dissertation, University of Arizona. Accessed February 25, 2021. http://hdl.handle.net/10150/645775.

MLA Handbook (7th Edition):

Zbesko, Jacob C. “Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes .” 2020. Web. 25 Feb 2021.

Vancouver:

Zbesko JC. Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes . [Internet] [Doctoral dissertation]. University of Arizona; 2020. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10150/645775.

Council of Science Editors:

Zbesko JC. Investigating the Chronic Inflammatory Response to Stroke: The Role of Glial Scar Astrocytes and B-Lymphocytes . [Doctoral Dissertation]. University of Arizona; 2020. Available from: http://hdl.handle.net/10150/645775


University of Miami

2. Hackett, Amber. The Role of NG2 Cells after Contusive Spinal Cord Injury.

Degree: PhD, Neuroscience (Medicine), 2016, University of Miami

  NG2 cells, also known as oligodendrocyte progenitor cells (OPCs), are tiled throughout the adult central nervous system (CNS) and proliferate and differentiate into oligodendrocytes… (more)

Subjects/Keywords: Spinal Cord Injury; Glial Scar; Oligodendrocytes; Cytokines

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APA (6th Edition):

Hackett, A. (2016). The Role of NG2 Cells after Contusive Spinal Cord Injury. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1767

Chicago Manual of Style (16th Edition):

Hackett, Amber. “The Role of NG2 Cells after Contusive Spinal Cord Injury.” 2016. Doctoral Dissertation, University of Miami. Accessed February 25, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/1767.

MLA Handbook (7th Edition):

Hackett, Amber. “The Role of NG2 Cells after Contusive Spinal Cord Injury.” 2016. Web. 25 Feb 2021.

Vancouver:

Hackett A. The Role of NG2 Cells after Contusive Spinal Cord Injury. [Internet] [Doctoral dissertation]. University of Miami; 2016. [cited 2021 Feb 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1767.

Council of Science Editors:

Hackett A. The Role of NG2 Cells after Contusive Spinal Cord Injury. [Doctoral Dissertation]. University of Miami; 2016. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1767


University of Miami

3. Freer, Lucy. Macrophage Depletion as a Therapeutic Method After SCI.

Degree: PhD, Neuroscience (Medicine), 2018, University of Miami

 Developing successful therapies for spinal cord injury (SCI) is a formidable medical challenge in part because of the axonal growth-inhibitory environment that develops following injury… (more)

Subjects/Keywords: Macrophage; Glial Scar; Spinal cord injury; Fibrotic Scar

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APA (6th Edition):

Freer, L. (2018). Macrophage Depletion as a Therapeutic Method After SCI. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/2019

Chicago Manual of Style (16th Edition):

Freer, Lucy. “Macrophage Depletion as a Therapeutic Method After SCI.” 2018. Doctoral Dissertation, University of Miami. Accessed February 25, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/2019.

MLA Handbook (7th Edition):

Freer, Lucy. “Macrophage Depletion as a Therapeutic Method After SCI.” 2018. Web. 25 Feb 2021.

Vancouver:

Freer L. Macrophage Depletion as a Therapeutic Method After SCI. [Internet] [Doctoral dissertation]. University of Miami; 2018. [cited 2021 Feb 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2019.

Council of Science Editors:

Freer L. Macrophage Depletion as a Therapeutic Method After SCI. [Doctoral Dissertation]. University of Miami; 2018. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2019


Wayne State University

4. Ereifej, Evon S. Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface.

Degree: PhD, Biomedical Engineering, 2012, Wayne State University

  Neural electrode devices hold great promise to help people with the restoration of lost functions, however, research is lacking in the biomaterial design of… (more)

Subjects/Keywords: astrocytes, biocompatibility, biomaterials, glial scar, nanopatterns, neural electrodes; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Ereifej, E. S. (2012). Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/436

Chicago Manual of Style (16th Edition):

Ereifej, Evon S. “Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface.” 2012. Doctoral Dissertation, Wayne State University. Accessed February 25, 2021. https://digitalcommons.wayne.edu/oa_dissertations/436.

MLA Handbook (7th Edition):

Ereifej, Evon S. “Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface.” 2012. Web. 25 Feb 2021.

Vancouver:

Ereifej ES. Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface. [Internet] [Doctoral dissertation]. Wayne State University; 2012. [cited 2021 Feb 25]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/436.

Council of Science Editors:

Ereifej ES. Studying the glial cell response to biomaterials and surface topography for improving the neural electrode interface. [Doctoral Dissertation]. Wayne State University; 2012. Available from: https://digitalcommons.wayne.edu/oa_dissertations/436

5. Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi, Mayumi. Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。.

Degree: 博士(医学), 2016, Nara Medical University / 奈良県立医科大学

OBJECTIVE Transplantation of bone marrow stromal cells (BMSCs) is a theoretical potential as a therapeutic strategy in the treatment of spinal cord injury (SCI). Although… (more)

Subjects/Keywords: bone marrow stromal cell; cell sheet; axonal regeneration; spinal cord injury; ascorbic acid; glial scar

Page 1

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APA (6th Edition):

Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi, M. (2016). Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi, Mayumi. “Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。.” 2016. Thesis, Nara Medical University / 奈良県立医科大学. Accessed February 25, 2021. http://hdl.handle.net/10564/3282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi, Mayumi. “Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。.” 2016. Web. 25 Feb 2021.

Vancouver:

Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi M. Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2016. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10564/3282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Okuda, Akinori; Horii-Hayashi, Noriko; Sasagawa, Takayo; Shimizu, Takamasa; Shigematsu, Hideki; Iwata, Eiichiro; Morimoto, Yasuhiko; Masuda, Keisuke; Koizumi, Munehisa; Akahane, Manabu; Nishi M. Bone marrow stromal cell sheets may promote axonal regeneration and functional recovery with suppression of glial scar formation after spinal cord transection injury in rats. : 骨髄間葉系細胞シートはラット脊髄離断損傷後にグリア瘢痕形成を抑制し、軸索再生と後肢運動機能改善を促進する。. [Thesis]. Nara Medical University / 奈良県立医科大学; 2016. Available from: http://hdl.handle.net/10564/3282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

6. Ab Patar, Mohd Nor Azim. Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies .

Degree: 2018, National University of Ireland – Galway

 There are several detrimental events that occur in the aftermath of a spinal cord injury (SCI); these include glial scar formation, accumulation of axon growth… (more)

Subjects/Keywords: Spinal cord injury; Glial scar; Ex vivo slice cultures; Chondroitinase ABC; Lentivirus; Medicine; Anatomy

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APA (6th Edition):

Ab Patar, M. N. A. (2018). Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/7282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ab Patar, Mohd Nor Azim. “Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies .” 2018. Thesis, National University of Ireland – Galway. Accessed February 25, 2021. http://hdl.handle.net/10379/7282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ab Patar, Mohd Nor Azim. “Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies .” 2018. Web. 25 Feb 2021.

Vancouver:

Ab Patar MNA. Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies . [Internet] [Thesis]. National University of Ireland – Galway; 2018. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10379/7282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ab Patar MNA. Ex Vivo slice culture model of rat spinal cord injury as a platform for testing proposed treatment strategies . [Thesis]. National University of Ireland – Galway; 2018. Available from: http://hdl.handle.net/10379/7282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

7. Pakulska, Malgorzata Maria. Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration.

Degree: PhD, 2016, University of Toronto

Spinal cord injury (SCI) is a devastating condition that affects millions of people worldwide. Successful drug therapies for SCI have been difficult to achieve due… (more)

Subjects/Keywords: adult stem cells; chondroitinase abc; CXCL12; drug delivery; glial scar; spinal cord; 0541

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APA (6th Edition):

Pakulska, M. M. (2016). Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89261

Chicago Manual of Style (16th Edition):

Pakulska, Malgorzata Maria. “Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration.” 2016. Doctoral Dissertation, University of Toronto. Accessed February 25, 2021. http://hdl.handle.net/1807/89261.

MLA Handbook (7th Edition):

Pakulska, Malgorzata Maria. “Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration.” 2016. Web. 25 Feb 2021.

Vancouver:

Pakulska MM. Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1807/89261.

Council of Science Editors:

Pakulska MM. Combined Delivery of Chondroitinase ABC (ChABC) and Stromal Cell Derived Factor 1α (SDF1α) for Spinal Cord Regeneration. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89261


University of Toronto

8. Dragas, Rachel. Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth.

Degree: 2018, University of Toronto

The astroglial scar is a defining hallmark of secondary pathology following central nervous system (CNS) injury that significantly obstructs neuroregeneration. Neural progenitor cell (NPC) therapies… (more)

Subjects/Keywords: central nervous system injury; glial scar; hepatocyte growth factor; neural progenitor cells; reactive astrocytes; regeneration; 0564

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APA (6th Edition):

Dragas, R. (2018). Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89622

Chicago Manual of Style (16th Edition):

Dragas, Rachel. “Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth.” 2018. Masters Thesis, University of Toronto. Accessed February 25, 2021. http://hdl.handle.net/1807/89622.

MLA Handbook (7th Edition):

Dragas, Rachel. “Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth.” 2018. Web. 25 Feb 2021.

Vancouver:

Dragas R. Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1807/89622.

Council of Science Editors:

Dragas R. Hepatocyte Growth Factor-Preconditioned Neural Progenitor Cells Attenuate Astrocyte Reactivity and Promote Neurite Outgrowth. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89622


Univerzitet u Beogradu

9. Dacić, Sanja A., 1973-. Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Biologija - Neurobiologija / Biology - Neurobiology

Traumatska povreda mozga (TPM) predstavlja veliki zdravstveni problem savremenog društva jer je vodeći uzrok smrti i invaliditeta kod… (more)

Subjects/Keywords: rat; brain injury; locomotor coordination; reactive astrogliosis; glial scar; neuronal sprouting and branching; synaptic plasticity; chondroitin sulfate proteoglycans; B vitamins; ribavirin

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APA (6th Edition):

Dacić, Sanja A., 1. (2014). Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:7805/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dacić, Sanja A., 1973-. “Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova.” 2014. Thesis, Univerzitet u Beogradu. Accessed February 25, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:7805/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dacić, Sanja A., 1973-. “Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova.” 2014. Web. 25 Feb 2021.

Vancouver:

Dacić, Sanja A. 1. Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2021 Feb 25]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7805/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dacić, Sanja A. 1. Uticaj B vitaminskih kompleksa i ribavirina na odgovor nervnih i glijskih ćelija i oporavak motornih funkcija nakon povrede mozga pacova. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7805/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

10. Dyck, Scott. Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury.

Degree: Physiology and Pathophysiology, 2015, University of Manitoba

 Populations of oligodendrocytes are susceptible to cell death following spinal cord injury (SCI), which results in axon demyelination. Multipotent resident neural precursor cells (NPCs) and… (more)

Subjects/Keywords: Spinal cord injury; Glial scar; Astrogliosis; CSPGs; Chondroitin sulfate proteoglycans; Cell replacement; Oligodendrocyte replacement; Oligodendrogenesis; Neuroinflammation

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APA (6th Edition):

Dyck, S. (2015). Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dyck, Scott. “Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury.” 2015. Thesis, University of Manitoba. Accessed February 25, 2021. http://hdl.handle.net/1993/32952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dyck, Scott. “Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury.” 2015. Web. 25 Feb 2021.

Vancouver:

Dyck S. Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury. [Internet] [Thesis]. University of Manitoba; 2015. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1993/32952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dyck S. Elucidating the role and mechanisms of CSPGs in modulating endogenous repair processes in spinal cord injury. [Thesis]. University of Manitoba; 2015. Available from: http://hdl.handle.net/1993/32952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Fuller, Molly Lynn. The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions.

Degree: PhD, Neurosciences, 2007, Case Western Reserve University School of Graduate Studies

 The response of glial cells to injury of the adult central nervous system (CNS) leads to formation of a glial scar, a dense network of… (more)

Subjects/Keywords: Biology, Neuroscience; BMP; CSPG; glial scar; demyelination; CNS

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APA (6th Edition):

Fuller, M. L. (2007). The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1183990990

Chicago Manual of Style (16th Edition):

Fuller, Molly Lynn. “The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions.” 2007. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed February 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1183990990.

MLA Handbook (7th Edition):

Fuller, Molly Lynn. “The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions.” 2007. Web. 25 Feb 2021.

Vancouver:

Fuller ML. The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2007. [cited 2021 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183990990.

Council of Science Editors:

Fuller ML. The Role of Bone Morphogenetic Proteins in Reactive Gliosis after Demyelinating Spinal Cord Lesions. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183990990


NSYSU

12. Chen, Chun-Hong. The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury.

Degree: PhD, Doctoral Degree Program in Marine Biotechnology, 2016, NSYSU

 In the present study intrathecal pretreatment 11-dehydrosinulariolide attenuated spinal cord injury (SCI)-induced cell apoptosis by upregulating the antiapoptotic protein Bcl-2 and cell survival-related pathway proteins… (more)

Subjects/Keywords: microglia; marine natural product; 11-dehydrosinulariolide; neuroprotection; spinal cord injury; anti-inflammation; phosphatase and tensin homologue deleted on chromosome 10; astrocyte; glial scar; chondroitin sulfate proteoglycans

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APA (6th Edition):

Chen, C. (2016). The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730114-150126

Chicago Manual of Style (16th Edition):

Chen, Chun-Hong. “The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury.” 2016. Doctoral Dissertation, NSYSU. Accessed February 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730114-150126.

MLA Handbook (7th Edition):

Chen, Chun-Hong. “The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury.” 2016. Web. 25 Feb 2021.

Vancouver:

Chen C. The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury. [Internet] [Doctoral dissertation]. NSYSU; 2016. [cited 2021 Feb 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730114-150126.

Council of Science Editors:

Chen C. The therapeutic effects of coral-compound and PTEN in rats with traumatic spinal cord injury. [Doctoral Dissertation]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730114-150126


Duke University

13. Polikov, Vadim Steven. An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring .

Degree: 2009, Duke University

  Chronically implanted recording electrode arrays linked to prosthetics have the potential to make positive impacts on patients suffering from full or partial paralysis [1;2].… (more)

Subjects/Keywords: Biomedical Engineering; Neurosciences; astrocyte; cortical electrodes; glial scar; in vitro; neural precursor cell

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APA (6th Edition):

Polikov, V. S. (2009). An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/3202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Polikov, Vadim Steven. “An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring .” 2009. Thesis, Duke University. Accessed February 25, 2021. http://hdl.handle.net/10161/3202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Polikov, Vadim Steven. “An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring .” 2009. Web. 25 Feb 2021.

Vancouver:

Polikov VS. An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring . [Internet] [Thesis]. Duke University; 2009. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10161/3202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Polikov VS. An in vitro model of the brain tissue reaction to chronically implanted recording electrodes reveals essential roles for serum and bFGF in glial scarring . [Thesis]. Duke University; 2009. Available from: http://hdl.handle.net/10161/3202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arkansas

14. Walker, Addison. Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury.

Degree: MSBME, 2016, University of Arkansas

  Following injury to the central nervous system, extracellular modulations are apparent at the site of injury, often resulting in a glial scar. Astrocytes are… (more)

Subjects/Keywords: Biological sciences; Applied sciences; Astrocytes; Extracellular matrix; Glial scar; Repeat injury; Traumatic brain injury; Bioelectrical and Neuroengineering; Cell Biology; Molecular and Cellular Neuroscience

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APA (6th Edition):

Walker, A. (2016). Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury. (Masters Thesis). University of Arkansas. Retrieved from https://scholarworks.uark.edu/etd/1754

Chicago Manual of Style (16th Edition):

Walker, Addison. “Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury.” 2016. Masters Thesis, University of Arkansas. Accessed February 25, 2021. https://scholarworks.uark.edu/etd/1754.

MLA Handbook (7th Edition):

Walker, Addison. “Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury.” 2016. Web. 25 Feb 2021.

Vancouver:

Walker A. Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury. [Internet] [Masters thesis]. University of Arkansas; 2016. [cited 2021 Feb 25]. Available from: https://scholarworks.uark.edu/etd/1754.

Council of Science Editors:

Walker A. Exploring the Production of Extracellular Matrix by Astrocytes in Response to Mimetic Traumatic Brain Injury. [Masters Thesis]. University of Arkansas; 2016. Available from: https://scholarworks.uark.edu/etd/1754

15. Hagerty, Kailyn M. Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke.

Degree: MS, Physiology and Neuroscience, 2012, Wright State University

 Stroke is one of the leading causes of death and the top cause of long term disability. Currently there exists no standardized treatment of care… (more)

Subjects/Keywords: Neurosciences; ischemic stroke; rats; fluoxetine; angiogenesis; glial scar

…infarct (indicative of a glial scar) or spread out 24 and in diffuse areas away… …These animals were used to compare differences in angiogenesis, glial scarring, and infarct… …oxide synthase (eNOS, 5 μg/mL, AbCAM antibody) and glial fibrillary acidic protein… 

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APA (6th Edition):

Hagerty, K. M. (2012). Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1341957043

Chicago Manual of Style (16th Edition):

Hagerty, Kailyn M. “Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke.” 2012. Masters Thesis, Wright State University. Accessed February 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1341957043.

MLA Handbook (7th Edition):

Hagerty, Kailyn M. “Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke.” 2012. Web. 25 Feb 2021.

Vancouver:

Hagerty KM. Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke. [Internet] [Masters thesis]. Wright State University; 2012. [cited 2021 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1341957043.

Council of Science Editors:

Hagerty KM. Comparing Two Different Statins in a Delayed Pharmacological Treatment for Ischemic Stroke. [Masters Thesis]. Wright State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1341957043

16. Thomaty, Sandie. Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat.

Degree: Docteur es, Neurosciences, 2015, Aix Marseille Université

Les lésions spinales conduisent à des altérations majeures des fonctions sensorimotrices. Les récupérations fonctionnelles consécutives à ces atteintes sont très limitées, notamment en raison des… (more)

Subjects/Keywords: Gm-Csf; Neuroinflammation; Neuroplasticité; Microglie; Mastocytes; Cicatrice gliale; Lésion spinale; Récupérations fonctionnelles; Réorganisations corticales; Rat adulte; Gm-Csf; Neuroinflammation; Neuroplasticity; Microglia; Mast cells; Glial scar; Spinal cord injury; Functional recovery; Cortical reorganization; Adult rat

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APA (6th Edition):

Thomaty, S. (2015). Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2015AIXM4772

Chicago Manual of Style (16th Edition):

Thomaty, Sandie. “Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat.” 2015. Doctoral Dissertation, Aix Marseille Université. Accessed February 25, 2021. http://www.theses.fr/2015AIXM4772.

MLA Handbook (7th Edition):

Thomaty, Sandie. “Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat.” 2015. Web. 25 Feb 2021.

Vancouver:

Thomaty S. Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat. [Internet] [Doctoral dissertation]. Aix Marseille Université 2015. [cited 2021 Feb 25]. Available from: http://www.theses.fr/2015AIXM4772.

Council of Science Editors:

Thomaty S. Influence des processus inflammatoires sur la neuroplasticité et sur les récupérations fonctionnelles après lésion spinale chez le rat adulte : Influence of inflammatory processes on neuroplasticity and functional recovery after spinal cord injury in the adult rat. [Doctoral Dissertation]. Aix Marseille Université 2015. Available from: http://www.theses.fr/2015AIXM4772

17. Manns, Richard Peter Charles. Repulsive cues and signalling cascades of the axon growth cone.

Degree: PhD, 2013, University of Cambridge

 The aim of the work described in this thesis is to investigate the nature and mechanisms of action of repellent cues for growing axons. In… (more)

Subjects/Keywords: axon repulsion; axon guidance; neuron; glial scar; astrocyte; gliosis; spinal cord; prosaposin; nitrosylation; dorsal root ganglion; Semaphorin-3A; astroglioma; rapamycin; mTOR; Nogo-66; amino-Nogo; Nogo-A; reticulon-4A; nitric oxide; cGMP; proteasome; concentration dependence; growth cone; quantitative immunofluorescence; protein disulphide isomerase; protein disulfide isomerase

Page 1

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APA (6th Edition):

Manns, R. P. C. (2013). Repulsive cues and signalling cascades of the axon growth cone. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/245741https://www.repository.cam.ac.uk/bitstream/1810/245741/10/thesis.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/245741/11/thesis.pdf.jpg

Chicago Manual of Style (16th Edition):

Manns, Richard Peter Charles. “Repulsive cues and signalling cascades of the axon growth cone.” 2013. Doctoral Dissertation, University of Cambridge. Accessed February 25, 2021. https://www.repository.cam.ac.uk/handle/1810/245741https://www.repository.cam.ac.uk/bitstream/1810/245741/10/thesis.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/245741/11/thesis.pdf.jpg.

MLA Handbook (7th Edition):

Manns, Richard Peter Charles. “Repulsive cues and signalling cascades of the axon growth cone.” 2013. Web. 25 Feb 2021.

Vancouver:

Manns RPC. Repulsive cues and signalling cascades of the axon growth cone. [Internet] [Doctoral dissertation]. University of Cambridge; 2013. [cited 2021 Feb 25]. Available from: https://www.repository.cam.ac.uk/handle/1810/245741https://www.repository.cam.ac.uk/bitstream/1810/245741/10/thesis.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/245741/11/thesis.pdf.jpg.

Council of Science Editors:

Manns RPC. Repulsive cues and signalling cascades of the axon growth cone. [Doctoral Dissertation]. University of Cambridge; 2013. Available from: https://www.repository.cam.ac.uk/handle/1810/245741https://www.repository.cam.ac.uk/bitstream/1810/245741/10/thesis.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/245741/11/thesis.pdf.jpg

18. Alizadeh, Arsalan. Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury.

Degree: Physiology and Pathophysiology, 2018, University of Manitoba

 Spinal cord injury (SCI) elicits a robust glial and neuroinflammatory response that governs secondary injury processes and causes progressive neural degeneration and loss of neurological… (more)

Subjects/Keywords: Spinal Cord Injury; Glial Scar; Neuroinflammation; Neuregulin-1; T cells; B cells; Macrophages; Microglia; Astrocytes

…B Lymphocytes 27 1.2.2.6 Glial Scar Formation 33 1.3 Neuregulin-1 40 1.3.1… …Neuroinflammation and glial scar formation in SCI. 38 Figure 1.5 Structural diversity of Nrg-1. 42… …activation Demyelination Glial scar formation Oligodendrocyte loss Fig. 1.2: Secondary injury… …lead to progressive neural tissue damage. Glial cell activation, scar formation and… …Objectives 59 1.5 References 61 Chapter 2: Neuregulin-1 Positively Modulates Glial Response and… 

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APA (6th Edition):

Alizadeh, A. (2018). Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33364

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alizadeh, Arsalan. “Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury.” 2018. Thesis, University of Manitoba. Accessed February 25, 2021. http://hdl.handle.net/1993/33364.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alizadeh, Arsalan. “Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury.” 2018. Web. 25 Feb 2021.

Vancouver:

Alizadeh A. Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury. [Internet] [Thesis]. University of Manitoba; 2018. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1993/33364.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alizadeh A. Elucidating the role of neuregulin-1 in glial and immune response following traumatic spinal cord injury. [Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33364

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

19. Manns, Richard Peter Charles. Repulsive cues and signalling cascades of the axon growth cone.

Degree: PhD, 2013, University of Cambridge

 The aim of the work described in this thesis is to investigate the nature and mechanisms of action of repellent cues for growing axons. In… (more)

Subjects/Keywords: 612.8; axon repulsion; axon guidance; neuron; glial scar; astrocyte; gliosis; spinal cord; prosaposin; nitrosylation; dorsal root ganglion; Semaphorin-3A; astroglioma; rapamycin; mTOR; Nogo-66; amino-Nogo; Nogo-A; reticulon-4A; nitric oxide; cGMP; proteasome; concentration dependence; growth cone; quantitative immunofluorescence; protein disulphide isomerase; protein disulfide isomerase

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APA (6th Edition):

Manns, R. P. C. (2013). Repulsive cues and signalling cascades of the axon growth cone. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.16341 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607707

Chicago Manual of Style (16th Edition):

Manns, Richard Peter Charles. “Repulsive cues and signalling cascades of the axon growth cone.” 2013. Doctoral Dissertation, University of Cambridge. Accessed February 25, 2021. https://doi.org/10.17863/CAM.16341 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607707.

MLA Handbook (7th Edition):

Manns, Richard Peter Charles. “Repulsive cues and signalling cascades of the axon growth cone.” 2013. Web. 25 Feb 2021.

Vancouver:

Manns RPC. Repulsive cues and signalling cascades of the axon growth cone. [Internet] [Doctoral dissertation]. University of Cambridge; 2013. [cited 2021 Feb 25]. Available from: https://doi.org/10.17863/CAM.16341 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607707.

Council of Science Editors:

Manns RPC. Repulsive cues and signalling cascades of the axon growth cone. [Doctoral Dissertation]. University of Cambridge; 2013. Available from: https://doi.org/10.17863/CAM.16341 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607707

20. Pillet, Laure-Elise. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.

Degree: Docteur es, Neurobiologie, 2018, Sorbonne Paris Cité

La synapse est le lieu de communication entre les neurones à l'origine de nos capacités cognitives. Les mutations des gènes codant pour des protéines synaptiques… (more)

Subjects/Keywords: Déficience intellectuelle; Oligophrénine-1; RhoGAP; Myosin light chain; Astrocytes; Cicarisation gliale corticale; Migration; Morphologie; KO conditionnel astrocytaire; Intellectual disability; Oligophrenin-1; RhoGAP; Myosin light chain; Astrocytes; Cotical glial scar; Migration; Morphology; Astrocytic conditional KO; 616.858 8

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APA (6th Edition):

Pillet, L. (2018). Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB246

Chicago Manual of Style (16th Edition):

Pillet, Laure-Elise. “Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed February 25, 2021. http://www.theses.fr/2018USPCB246.

MLA Handbook (7th Edition):

Pillet, Laure-Elise. “Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.” 2018. Web. 25 Feb 2021.

Vancouver:

Pillet L. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2021 Feb 25]. Available from: http://www.theses.fr/2018USPCB246.

Council of Science Editors:

Pillet L. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB246


Georgia Tech

21. McConnell, George Charles. Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state.

Degree: PhD, Biomedical Engineering, 2008, Georgia Tech

 Thanks to pioneering scientists and clinicians, prosthetic devices that are controlled by intra-cortical electrodes recording one's 'thoughts' are a reality today, and no longer merely… (more)

Subjects/Keywords: Immunohistochemistry; Neural recording; Chronic implants; Brain; Silicon; Impedance spectroscopy; Neurodegeneration; Neuroinflammation; Laminin; Neuroprosthesis; Microelectrode; Cole; Bioimpedance; Biosensor; Astroglial scar; Biocompatibility; Glial scar; Implants, Artificial; Microelectrode; Foreign-body reaction; Inflammation

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APA (6th Edition):

McConnell, G. C. (2008). Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/26615

Chicago Manual of Style (16th Edition):

McConnell, George Charles. “Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state.” 2008. Doctoral Dissertation, Georgia Tech. Accessed February 25, 2021. http://hdl.handle.net/1853/26615.

MLA Handbook (7th Edition):

McConnell, George Charles. “Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state.” 2008. Web. 25 Feb 2021.

Vancouver:

McConnell GC. Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state. [Internet] [Doctoral dissertation]. Georgia Tech; 2008. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1853/26615.

Council of Science Editors:

McConnell GC. Chronic inflammation surrounding intra-cortical electrodes is correlated with a local, neurodegenerative state. [Doctoral Dissertation]. Georgia Tech; 2008. Available from: http://hdl.handle.net/1853/26615


Georgia Tech

22. Zhong, Yinghui. Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes.

Degree: PhD, Biomedical Engineering, 2006, Georgia Tech

 Stable single-unit recordings from the nervous system using microelectrode arrays can have significant implications for the treatment of a wide variety of sensory and movement… (more)

Subjects/Keywords: Neural implant; Drug delivery; Coatings; Inflammation; Glial scar; Nervous system; Electrodes; Implants, Artificial; Foreign-body reaction; Wound healing; Neuroglia; Coatings; Controlled release preparations; Anti-inflammatory agents

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APA (6th Edition):

Zhong, Y. (2006). Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/19760

Chicago Manual of Style (16th Edition):

Zhong, Yinghui. “Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes.” 2006. Doctoral Dissertation, Georgia Tech. Accessed February 25, 2021. http://hdl.handle.net/1853/19760.

MLA Handbook (7th Edition):

Zhong, Yinghui. “Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes.” 2006. Web. 25 Feb 2021.

Vancouver:

Zhong Y. Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes. [Internet] [Doctoral dissertation]. Georgia Tech; 2006. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1853/19760.

Council of Science Editors:

Zhong Y. Development and Characterization of Anti-Inflammatory Coatings for Implanted Neural Probes. [Doctoral Dissertation]. Georgia Tech; 2006. Available from: http://hdl.handle.net/1853/19760

23. Millner, Mary Angela. Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration.

Degree: MS, Biomedical Engineering, 2008, Georgia Tech

 Injury to the central nervous system (CNS) results in the formation of a highly inhibitory glial scar consisting mainly of chondroitin sulfate proteoglycans (CSPGs). CSPGs… (more)

Subjects/Keywords: Nerve regeneration; Glycosaminoglycan; Chondroitin sulfate; Astrocyte; Glial scar; Central nervous system wounds and injuries.; Chondroitin sulfates; Proteoglycans

…CNS) results in the formation of a highly inhibitory glial scar consisting mainly of… …inhibitory glial scar, which presents both biochemical and physical barriers. Disruption of the… …recovery after injury. Hence, the glial scar acts as both a physical and chemical barrier to… …forming the highly inhibitory glial scar. CSPGs often function through their interactions with… …injured spinal cord must overcome the inhibitory effects of the glial scar while maintaining its… 

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APA (6th Edition):

Millner, M. A. (2008). Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/24647

Chicago Manual of Style (16th Edition):

Millner, Mary Angela. “Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration.” 2008. Masters Thesis, Georgia Tech. Accessed February 25, 2021. http://hdl.handle.net/1853/24647.

MLA Handbook (7th Edition):

Millner, Mary Angela. “Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration.” 2008. Web. 25 Feb 2021.

Vancouver:

Millner MA. Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration. [Internet] [Masters thesis]. Georgia Tech; 2008. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/1853/24647.

Council of Science Editors:

Millner MA. Modulation of CSPG sulfation patterns through siRNA silencing of sulfotransferase expression to promote CNS regeneration. [Masters Thesis]. Georgia Tech; 2008. Available from: http://hdl.handle.net/1853/24647

24. Filous, Angela R, Ph.D. The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury.

Degree: PhD, Neurosciences, 2014, Case Western Reserve University School of Graduate Studies

 Macrophage infiltration into the lesion core after spinal cord injury causes injured dystrophic axons to dieback from the lesion center. Although it was once thought… (more)

Subjects/Keywords: Neurosciences; NG2; chondroitin sulfate proteoglycans; spinal cord injury, axonal dieback; regeneration; glial scar; spinal cord; entrapment; neuroscience; oligodendrocyte precursor cells

…the rostral end of the glial scar, as has been shown previously. The fibers that reach… …to cross the inhibitory rim of an in vitro model of the glial scar through an MMP-2… …is often referred to as the glial scar, because it has long been thought to be made up… …recruited to the site of injury. The glial scar provides both a physical and chemical barrier to… …have also been shown to inhibit neurite outgrowth. Glial scar formation Injury leads to the… 

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APA (6th Edition):

Filous, Angela R, P. D. (2014). The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1396605992

Chicago Manual of Style (16th Edition):

Filous, Angela R, Ph D. “The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury.” 2014. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed February 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1396605992.

MLA Handbook (7th Edition):

Filous, Angela R, Ph D. “The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury.” 2014. Web. 25 Feb 2021.

Vancouver:

Filous, Angela R PD. The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2014. [cited 2021 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396605992.

Council of Science Editors:

Filous, Angela R PD. The Role of NG2+ Cells in Regeneration Failure After Spinal Cord Injury. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396605992

25. Lang, Bradley Thomas. THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY.

Degree: PhD, Neurosciences, 2015, Case Western Reserve University School of Graduate Studies

 Contusive spinal cord injury (SCI) is a devastating condition that leads to permanent disability due to the lack of neuronal regeneration and functional plasticity. It… (more)

Subjects/Keywords: Biomedical Research; Biology; Neurobiology; Neurology; Neurosciences; Spinal Cord Injury; Regenerative Medicine; Chondroitin Sulfate Proteoglycans; PTPsigma; Protein Tyrosine Phosphatase Sigma; Receptor Protein Tyrosine Phosphatase Sigma; Axonal Regeneration; Axonal Plasticity; Glial Scar

glial scar Figure 44 171 Correlation between lesion size and behavioral recovery Figure 45… …upregulation of glial derived chondroitin sulfate proteoglycans (CSPGs) within the scar… …A.P. Tran, and J. Silver (2014). Functional regeneration beyond the glial scar… …characterization of the glial scar The primary barrier to regenerating axons in the CNS is the glial scar… …glial scar are reactive astrocytes, which become hypertrophic as they increase expression of… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lang, B. T. (2015). THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1417619755

Chicago Manual of Style (16th Edition):

Lang, Bradley Thomas. “THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY.” 2015. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed February 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1417619755.

MLA Handbook (7th Edition):

Lang, Bradley Thomas. “THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY.” 2015. Web. 25 Feb 2021.

Vancouver:

Lang BT. THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2015. [cited 2021 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417619755.

Council of Science Editors:

Lang BT. THE ROLE OF PTPs IN REGENERATION FAILURE FOLLOWING SPINAL CORD INJURY. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417619755

26. Nicola, Fabrício do Couto. Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular.

Degree: 2017, Universidade do Rio Grande do Sul

A lesão medular (LM) é uma patologia incapacitante que resulta em déficits sensoriais e motores. No Brasil, a incidência anual é de 30 novos casos… (more)

Subjects/Keywords: Traumatismos da medula espinal; Spinal cord injury; Transplante de células-tronco; Mesenchymal stem cell; Células-tronco mesenquimais; Dental pulp stem cell; Dente decíduo; Tissue protection; Neuroprotection; Neuroproteção; Regeneração da medula espinal; Cell death; Apoptosis; Neurônios; Astrogliosis; Apoptose; Proteína glial fibrilar ácida; Glial scar; Proteínas S100; Progenitor cells; Functional recovery

…transplantation on the progenitor cells number, glial scar formation and astrocytic modifications after… …subacute phase, reducing glial scar formation. Key-words: Spinal cord injury, mesenchymal stem… …astrogliosis, glial scar, progenitor cells, functional recovery. XI 1. INTRODUÇÃO A lesão medular… …cicatriz glial e modificações astrocitárias após o modelo de contusão medular. Observou-se a… …proteína fibrilar glial ácida (GFAP) e aumentam a expressão do canal retificador de… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nicola, F. d. C. (2017). Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nicola, Fabrício do Couto. “Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 25, 2021. http://hdl.handle.net/10183/170284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nicola, Fabrício do Couto. “Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular.” 2017. Web. 25 Feb 2021.

Vancouver:

Nicola FdC. Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10183/170284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nicola FdC. Efeito neuroprotetor do transplante de células-tronco mesenquimais derivadas de dente decíduo humano em ratos Wistar submetidos à lesão medular. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.