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You searched for subject:(genomic instability). Showing records 1 – 30 of 136 total matches.

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University of Oxford

1. Lopez-Martinez, David. Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway.

Degree: PhD, 2018, University of Oxford

 Interstrand crosslinks (ICLs) are a highly deleterious form of DNA damage because they link the two strands of DNA, blocking replication, transcription and chromosome segregation.… (more)

Subjects/Keywords: 572; DNA repair; Genomic instability

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APA (6th Edition):

Lopez-Martinez, D. (2018). Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:1a749320-e434-41e3-8b5d-32c5481c0dee ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770584

Chicago Manual of Style (16th Edition):

Lopez-Martinez, David. “Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway.” 2018. Doctoral Dissertation, University of Oxford. Accessed January 15, 2021. http://ora.ox.ac.uk/objects/uuid:1a749320-e434-41e3-8b5d-32c5481c0dee ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770584.

MLA Handbook (7th Edition):

Lopez-Martinez, David. “Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway.” 2018. Web. 15 Jan 2021.

Vancouver:

Lopez-Martinez D. Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway. [Internet] [Doctoral dissertation]. University of Oxford; 2018. [cited 2021 Jan 15]. Available from: http://ora.ox.ac.uk/objects/uuid:1a749320-e434-41e3-8b5d-32c5481c0dee ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770584.

Council of Science Editors:

Lopez-Martinez D. Characterisation of novel phosphorylation sites on FANCD2 and their role in the Fanconi anaemia pathway. [Doctoral Dissertation]. University of Oxford; 2018. Available from: http://ora.ox.ac.uk/objects/uuid:1a749320-e434-41e3-8b5d-32c5481c0dee ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770584


Universiteit Utrecht

2. Peeters, J.G.C. Genetic instability: its causes and its consequences.

Degree: 2012, Universiteit Utrecht

 Alterations within the genome, referred to as genetic instability, can occur at the whole chromosome level, i.e. whole chromosomal instability (W-CIN), as well as at… (more)

Subjects/Keywords: Whole chromosomal instability; genomic instability; structural chromosomal instability; cancer; tumorigenesis

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APA (6th Edition):

Peeters, J. G. C. (2012). Genetic instability: its causes and its consequences. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/257555

Chicago Manual of Style (16th Edition):

Peeters, J G C. “Genetic instability: its causes and its consequences.” 2012. Masters Thesis, Universiteit Utrecht. Accessed January 15, 2021. http://dspace.library.uu.nl:8080/handle/1874/257555.

MLA Handbook (7th Edition):

Peeters, J G C. “Genetic instability: its causes and its consequences.” 2012. Web. 15 Jan 2021.

Vancouver:

Peeters JGC. Genetic instability: its causes and its consequences. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2021 Jan 15]. Available from: http://dspace.library.uu.nl:8080/handle/1874/257555.

Council of Science Editors:

Peeters JGC. Genetic instability: its causes and its consequences. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/257555


University of California – Riverside

3. Wang, Yehong. Analysis of Heterochromatin-Associated Genomic Instability.

Degree: Environmental Toxicology, 2009, University of California – Riverside

 Heterochromatin represents special regions in the genome that have been found to be prone to breakage and rearrangements in cancer cells. Through two separate but… (more)

Subjects/Keywords: Environmental Health; Biology, Genetics; Genomic Instability; Heterochromatin

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APA (6th Edition):

Wang, Y. (2009). Analysis of Heterochromatin-Associated Genomic Instability. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/7kr0h1ds

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Yehong. “Analysis of Heterochromatin-Associated Genomic Instability.” 2009. Thesis, University of California – Riverside. Accessed January 15, 2021. http://www.escholarship.org/uc/item/7kr0h1ds.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Yehong. “Analysis of Heterochromatin-Associated Genomic Instability.” 2009. Web. 15 Jan 2021.

Vancouver:

Wang Y. Analysis of Heterochromatin-Associated Genomic Instability. [Internet] [Thesis]. University of California – Riverside; 2009. [cited 2021 Jan 15]. Available from: http://www.escholarship.org/uc/item/7kr0h1ds.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang Y. Analysis of Heterochromatin-Associated Genomic Instability. [Thesis]. University of California – Riverside; 2009. Available from: http://www.escholarship.org/uc/item/7kr0h1ds

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington State University

4. [No author]. Formation and Bypass of APOBEC-mediated DNA lesions .

Degree: 2019, Washington State University

 AID/APOBEC family cytidine deaminases are established as endogenous DNA mutators that drive genomic instability in a wide array of human cancers. Many members of this… (more)

Subjects/Keywords: Molecular biology; APOBEC; Genomic instability; Replication Fork

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APA (6th Edition):

author], [. (2019). Formation and Bypass of APOBEC-mediated DNA lesions . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/17869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Formation and Bypass of APOBEC-mediated DNA lesions .” 2019. Thesis, Washington State University. Accessed January 15, 2021. http://hdl.handle.net/2376/17869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Formation and Bypass of APOBEC-mediated DNA lesions .” 2019. Web. 15 Jan 2021.

Vancouver:

author] [. Formation and Bypass of APOBEC-mediated DNA lesions . [Internet] [Thesis]. Washington State University; 2019. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/2376/17869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Formation and Bypass of APOBEC-mediated DNA lesions . [Thesis]. Washington State University; 2019. Available from: http://hdl.handle.net/2376/17869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Torres, Michael Jason 1982-. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.

Degree: 2014, University of Texas Southwestern Medical Center

 The exocyst complex, first described in yeast, is a heterooctomeric complex that serves as a signaling platform to mediate cellular responses to diverse spatial and… (more)

Subjects/Keywords: DNA Repair; Genomic Instability; Vesicular Transport Proteins

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APA (6th Edition):

Torres, M. J. 1. (2014). The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Torres, Michael Jason 1982-. “The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 15, 2021. http://hdl.handle.net/2152.5/3327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Torres, Michael Jason 1982-. “The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.” 2014. Web. 15 Jan 2021.

Vancouver:

Torres MJ1. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/2152.5/3327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Torres MJ1. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

6. Worrall, Joseph Thomas. Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells.

Degree: PhD, 2018, Queen Mary, University of London

Genomic instability and aneuploidy, which are ubiquitous hallmarks of cancer cells, encompass both structural and numerical chromosome aberrations. Strikingly, cancer cells often display recurrent patterns… (more)

Subjects/Keywords: Genomic instability; aneuploidy; cancer cells; chromosome missegregation

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APA (6th Edition):

Worrall, J. T. (2018). Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/31873 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766098

Chicago Manual of Style (16th Edition):

Worrall, Joseph Thomas. “Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells.” 2018. Doctoral Dissertation, Queen Mary, University of London. Accessed January 15, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/31873 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766098.

MLA Handbook (7th Edition):

Worrall, Joseph Thomas. “Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells.” 2018. Web. 15 Jan 2021.

Vancouver:

Worrall JT. Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2018. [cited 2021 Jan 15]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/31873 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766098.

Council of Science Editors:

Worrall JT. Determining individual chromosome missegregation rates and the responses to aneuploidy in human cells. [Doctoral Dissertation]. Queen Mary, University of London; 2018. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/31873 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766098


Texas State University – San Marcos

7. Banks, Kathryn Primrose. The Function of ALYREF on UAP56-Mediated R-Loop Resolution.

Degree: MS, Biochemistry, 2020, Texas State University – San Marcos

 R-loops arise during transcription, and serve important roles including class switch recombination, protection of genes from DNA methylation at GC rich promoters, and promotion of… (more)

Subjects/Keywords: R-loops; Genomic instability; ALYREF; UAP56; Genomics

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APA (6th Edition):

Banks, K. P. (2020). The Function of ALYREF on UAP56-Mediated R-Loop Resolution. (Masters Thesis). Texas State University – San Marcos. Retrieved from https://digital.library.txstate.edu/handle/10877/9886

Chicago Manual of Style (16th Edition):

Banks, Kathryn Primrose. “The Function of ALYREF on UAP56-Mediated R-Loop Resolution.” 2020. Masters Thesis, Texas State University – San Marcos. Accessed January 15, 2021. https://digital.library.txstate.edu/handle/10877/9886.

MLA Handbook (7th Edition):

Banks, Kathryn Primrose. “The Function of ALYREF on UAP56-Mediated R-Loop Resolution.” 2020. Web. 15 Jan 2021.

Vancouver:

Banks KP. The Function of ALYREF on UAP56-Mediated R-Loop Resolution. [Internet] [Masters thesis]. Texas State University – San Marcos; 2020. [cited 2021 Jan 15]. Available from: https://digital.library.txstate.edu/handle/10877/9886.

Council of Science Editors:

Banks KP. The Function of ALYREF on UAP56-Mediated R-Loop Resolution. [Masters Thesis]. Texas State University – San Marcos; 2020. Available from: https://digital.library.txstate.edu/handle/10877/9886


NSYSU

8. Yeh, Yi-Jan. Genomic instability in NSCLC detected by RAPD.

Degree: Master, Institute of Biomedical Sciences, 2001, NSYSU

 Abstract Lung cancer is one of the most common cancer death in Taiwan. The dead population of lung cancer are over five thousands per year.… (more)

Subjects/Keywords: NSCLC; Genomic instability; RAPD

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APA (6th Edition):

Yeh, Y. (2001). Genomic instability in NSCLC detected by RAPD. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727101-155454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeh, Yi-Jan. “Genomic instability in NSCLC detected by RAPD.” 2001. Thesis, NSYSU. Accessed January 15, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727101-155454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeh, Yi-Jan. “Genomic instability in NSCLC detected by RAPD.” 2001. Web. 15 Jan 2021.

Vancouver:

Yeh Y. Genomic instability in NSCLC detected by RAPD. [Internet] [Thesis]. NSYSU; 2001. [cited 2021 Jan 15]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727101-155454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeh Y. Genomic instability in NSCLC detected by RAPD. [Thesis]. NSYSU; 2001. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727101-155454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

9. Idica, Adam K. miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication.

Degree: Biological Sciences, 2016, University of California – Irvine

 microRNAs (miRs) are small endogenously encoded RNAs that post-transcriptionally repress gene expression by degradation or translational repression of target mRNAs. miRs are involved in normal… (more)

Subjects/Keywords: Molecular biology; Biochemistry; genomic instability; HIV-1; LINE-1; microRNA; Retrotransposon

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APA (6th Edition):

Idica, A. K. (2016). miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/80x871t6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Idica, Adam K. “miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication.” 2016. Thesis, University of California – Irvine. Accessed January 15, 2021. http://www.escholarship.org/uc/item/80x871t6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Idica, Adam K. “miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication.” 2016. Web. 15 Jan 2021.

Vancouver:

Idica AK. miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2021 Jan 15]. Available from: http://www.escholarship.org/uc/item/80x871t6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Idica AK. miR-128 Directly Represses L1 Retrotransposition, Cellular Targets in L1 Retrotransposition and HIV-1 Replication. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/80x871t6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

10. Puccetti, Matthew Vincent. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.

Degree: PhD, Pathology, 2018, Vanderbilt University

 Genome maintenance is essential for the viability of eukaryotic cells and for the prevention of tumorigenesis. DNA replication stress is a significant contributor to genomic(more)

Subjects/Keywords: DNA translocases; DNA damage response; genomic instability; replication fork remodeling; tumorigenesis

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APA (6th Edition):

Puccetti, M. V. (2018). The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15446

Chicago Manual of Style (16th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 15, 2021. http://hdl.handle.net/1803/15446.

MLA Handbook (7th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Web. 15 Jan 2021.

Vancouver:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1803/15446.

Council of Science Editors:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/15446


Penn State University

11. Choe, Katherine Naeun. HUWE1 Interacts with PCNA to Alleviate Replication Stress.

Degree: 2016, Penn State University

 The integrity of the genome relies on the accurate and faithful duplication of genetic information from a parental cell to its progeny during each cellular… (more)

Subjects/Keywords: DNA replication; Genomic instability; H2AX; DNA Damage; HUWE1; PCNA; Replication stress

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APA (6th Edition):

Choe, K. N. (2016). HUWE1 Interacts with PCNA to Alleviate Replication Stress. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13492kzc152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choe, Katherine Naeun. “HUWE1 Interacts with PCNA to Alleviate Replication Stress.” 2016. Thesis, Penn State University. Accessed January 15, 2021. https://submit-etda.libraries.psu.edu/catalog/13492kzc152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choe, Katherine Naeun. “HUWE1 Interacts with PCNA to Alleviate Replication Stress.” 2016. Web. 15 Jan 2021.

Vancouver:

Choe KN. HUWE1 Interacts with PCNA to Alleviate Replication Stress. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Jan 15]. Available from: https://submit-etda.libraries.psu.edu/catalog/13492kzc152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choe KN. HUWE1 Interacts with PCNA to Alleviate Replication Stress. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13492kzc152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. A. Colosio. MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS.

Degree: 2014, Università degli Studi di Milano

 ABSTRACT An accurate DNA replication is essential to prevent genome instability events, such as mutations and chromosomal rearrangements that are hallmarks of neoplastic transformation and… (more)

Subjects/Keywords: genomic instability; cell cycle checkpoint; DNA repair; Settore BIO/10 - Biochimica

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APA (6th Edition):

Colosio, A. (2014). MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/234147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Colosio, A.. “MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS.” 2014. Thesis, Università degli Studi di Milano. Accessed January 15, 2021. http://hdl.handle.net/2434/234147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Colosio, A.. “MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS.” 2014. Web. 15 Jan 2021.

Vancouver:

Colosio A. MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/2434/234147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Colosio A. MECHANISMS MEDIATING REPLICATION FORK COLLAPSE AND PROCESSING IN CHECKPOINT DEFECTIVE CELLS. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/234147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Bianco, Julien. Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication.

Degree: Docteur es, Biologie Santé, 2010, Université Montpellier I

Il a été montré récemment que l'instabilité génétique joue un rôle central dans les étapes précoces de la tumorigenèse. Celle-ci provoquerait une activation chronique des… (more)

Subjects/Keywords: Replication; Checkpoint; Instabilité génomique; Replication; Checkpoint; Genomic instability

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APA (6th Edition):

Bianco, J. (2010). Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2010MON1T009

Chicago Manual of Style (16th Edition):

Bianco, Julien. “Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication.” 2010. Doctoral Dissertation, Université Montpellier I. Accessed January 15, 2021. http://www.theses.fr/2010MON1T009.

MLA Handbook (7th Edition):

Bianco, Julien. “Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication.” 2010. Web. 15 Jan 2021.

Vancouver:

Bianco J. Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication. [Internet] [Doctoral dissertation]. Université Montpellier I; 2010. [cited 2021 Jan 15]. Available from: http://www.theses.fr/2010MON1T009.

Council of Science Editors:

Bianco J. Rôle du complexe Claspine-Timeless-Tipin dans le maintien de la stabilité du génome au cours de la réplication : Role of Claspine-Timeless-Tipin complex in genome stability maintenance during replication. [Doctoral Dissertation]. Université Montpellier I; 2010. Available from: http://www.theses.fr/2010MON1T009


University of Adelaide

14. Puccini, Joseph. Characterisation of caspase-2 function in the DNA damage response and tumour suppression.

Degree: 2014, University of Adelaide

 Caspases are a family of cysteine proteases that have essential functions in the regulation of apoptosis and inflammation. Despite being the most evolutionarily conserved caspase,… (more)

Subjects/Keywords: cancer; tumour suppression; DNA damage response; caspases; genomic instability; cell cycle

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APA (6th Edition):

Puccini, J. (2014). Characterisation of caspase-2 function in the DNA damage response and tumour suppression. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/93503

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Puccini, Joseph. “Characterisation of caspase-2 function in the DNA damage response and tumour suppression.” 2014. Thesis, University of Adelaide. Accessed January 15, 2021. http://hdl.handle.net/2440/93503.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Puccini, Joseph. “Characterisation of caspase-2 function in the DNA damage response and tumour suppression.” 2014. Web. 15 Jan 2021.

Vancouver:

Puccini J. Characterisation of caspase-2 function in the DNA damage response and tumour suppression. [Internet] [Thesis]. University of Adelaide; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/2440/93503.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Puccini J. Characterisation of caspase-2 function in the DNA damage response and tumour suppression. [Thesis]. University of Adelaide; 2014. Available from: http://hdl.handle.net/2440/93503

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

15. Čabarkapa, Andrea M., 1985-. Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom.

Degree: Biološki fakultet, 2017, Univerzitet u Beogradu

Biologija - Genetika / Biology - Genetics

Reumatoidni artritis (RA) predstavlja autoimunsko inflamatorno oboljenje, u čijem razvoju ključnu ulogu imaju inflamatorni medijatori i oksidativni stres,… (more)

Subjects/Keywords: rheumatoid arthritis; olive leaf extract; oxidative stress; genomic instability; inflammation; antioxidants

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APA (6th Edition):

Čabarkapa, Andrea M., 1. (2017). Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:14398/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Čabarkapa, Andrea M., 1985-. “Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom.” 2017. Thesis, Univerzitet u Beogradu. Accessed January 15, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:14398/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Čabarkapa, Andrea M., 1985-. “Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom.” 2017. Web. 15 Jan 2021.

Vancouver:

Čabarkapa, Andrea M. 1. Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom. [Internet] [Thesis]. Univerzitet u Beogradu; 2017. [cited 2021 Jan 15]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:14398/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Čabarkapa, Andrea M. 1. Uticaj etanolnog ekstrakta lista masline (Olea europaea L.) na genomsku nestabilnost, parametre oksidativnog stresa i inflamacije kod pacijenata sa reumatoidnim artritisom. [Thesis]. Univerzitet u Beogradu; 2017. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:14398/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

16. Finn, Kenneth John. Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance.

Degree: Biochemistry and Molecular Biology, 2013, University of California – San Francisco

 Eukaryotic cells employ a battery of overlapping control mechanisms to ensure that each segment of their genome is replicated once, and only once, per cell… (more)

Subjects/Keywords: Molecular biology; Evolution; Gene Amplification; Genomic Instability; Oncogenesis; Replication; Re-Replication

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APA (6th Edition):

Finn, K. J. (2013). Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/85w067p6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Finn, Kenneth John. “Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance.” 2013. Thesis, University of California – San Francisco. Accessed January 15, 2021. http://www.escholarship.org/uc/item/85w067p6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Finn, Kenneth John. “Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance.” 2013. Web. 15 Jan 2021.

Vancouver:

Finn KJ. Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Jan 15]. Available from: http://www.escholarship.org/uc/item/85w067p6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Finn KJ. Re-Replication Induced Gene Amplification: Phenomenon, Mechanism, and Significance. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/85w067p6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

17. Richardson, Christopher Douglas. Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins.

Degree: Biochemistry and Molecular Biology, 2014, University of California – San Francisco

 Replication control is fundamental to genomic stability as aberrant replication within a single cell cycle can induce high rates of segmental amplification, chromosomal aneuploidy, and… (more)

Subjects/Keywords: Biochemistry; Molecular biology; DNA; Genomic Instability; Local Control; Replication; Re-replication

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APA (6th Edition):

Richardson, C. D. (2014). Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2qg774vw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richardson, Christopher Douglas. “Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins.” 2014. Thesis, University of California – San Francisco. Accessed January 15, 2021. http://www.escholarship.org/uc/item/2qg774vw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richardson, Christopher Douglas. “Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins.” 2014. Web. 15 Jan 2021.

Vancouver:

Richardson CD. Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2021 Jan 15]. Available from: http://www.escholarship.org/uc/item/2qg774vw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richardson CD. Re-Initiation Promoters: Genetic Elements that Modify Cell Cycle Control of Adjacent DNA Replication Origins. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/2qg774vw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

18. Chuang, Tony Chih-Yuan. The three-dimensional (3D) organization of telomeres during cellular transformation.

Degree: Physiology, 2010, University of Manitoba

 Statement of Problem Telomere dynamics in the three-dimensional (3D) space of the mammalian nucleus plays an important role in the maintenance of genomic stability. However,… (more)

Subjects/Keywords: telomere; c-myc; deconvolution; molecular imaging; genomic instability; biomarker

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APA (6th Edition):

Chuang, T. C. (2010). The three-dimensional (3D) organization of telomeres during cellular transformation. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4228

Chicago Manual of Style (16th Edition):

Chuang, Tony Chih-Yuan. “The three-dimensional (3D) organization of telomeres during cellular transformation.” 2010. Masters Thesis, University of Manitoba. Accessed January 15, 2021. http://hdl.handle.net/1993/4228.

MLA Handbook (7th Edition):

Chuang, Tony Chih-Yuan. “The three-dimensional (3D) organization of telomeres during cellular transformation.” 2010. Web. 15 Jan 2021.

Vancouver:

Chuang TC. The three-dimensional (3D) organization of telomeres during cellular transformation. [Internet] [Masters thesis]. University of Manitoba; 2010. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1993/4228.

Council of Science Editors:

Chuang TC. The three-dimensional (3D) organization of telomeres during cellular transformation. [Masters Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4228


University of Toronto

19. Lalonde, Emilie Rose. Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer.

Degree: PhD, 2016, University of Toronto

Prostate cancer represents a major global challenge, with 1.1 million new cases in 2012. According to the Public Health Agency of Canada, by 2028 prostate… (more)

Subjects/Keywords: Biomarker; Gene signature; Genomic instability; Precision medicine; Prognosis; Prostate cancer; 0564

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APA (6th Edition):

Lalonde, E. R. (2016). Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75324

Chicago Manual of Style (16th Edition):

Lalonde, Emilie Rose. “Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer.” 2016. Doctoral Dissertation, University of Toronto. Accessed January 15, 2021. http://hdl.handle.net/1807/75324.

MLA Handbook (7th Edition):

Lalonde, Emilie Rose. “Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer.” 2016. Web. 15 Jan 2021.

Vancouver:

Lalonde ER. Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1807/75324.

Council of Science Editors:

Lalonde ER. Integrative Modelling of Clinical, Genomic, and Microenvironmental Factors for Improved Prediction of Patient Outcome in Localized Prostate Cancer. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75324


Duke University

20. Dulmage, Keely. Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea.

Degree: 2015, Duke University

  Like most Archaea, the hypersaline-adapted organism Halobacterium salinarum exhibits characteristics from all three domains of life, including a eukaryotic histone protein, a universal propensity… (more)

Subjects/Keywords: Microbiology; Genetics; Molecular biology; Archaea; Cell division; Genomic instability; Halophiles; Histone

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APA (6th Edition):

Dulmage, K. (2015). Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/11341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dulmage, Keely. “Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea. ” 2015. Thesis, Duke University. Accessed January 15, 2021. http://hdl.handle.net/10161/11341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dulmage, Keely. “Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea. ” 2015. Web. 15 Jan 2021.

Vancouver:

Dulmage K. Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea. [Internet] [Thesis]. Duke University; 2015. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/10161/11341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dulmage K. Large-scale Effectors of Gene Expression and New Models of Cell Division in the Haloarchaea. [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/11341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

21. Boteva, Lora. Investigating transcription, replication and chromatin structure in determining common fragile site instability.

Degree: PhD, 2017, University of Edinburgh

 Common fragile sites are a set of genomic locations with a propensity to form lesions, breaks and gaps on mitotic chromosomes upon induction of replication… (more)

Subjects/Keywords: common fragile sites; CFS fragility; mitotic compaction; cytogenetic lesions; genomic instability

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APA (6th Edition):

Boteva, L. (2017). Investigating transcription, replication and chromatin structure in determining common fragile site instability. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28803

Chicago Manual of Style (16th Edition):

Boteva, Lora. “Investigating transcription, replication and chromatin structure in determining common fragile site instability.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed January 15, 2021. http://hdl.handle.net/1842/28803.

MLA Handbook (7th Edition):

Boteva, Lora. “Investigating transcription, replication and chromatin structure in determining common fragile site instability.” 2017. Web. 15 Jan 2021.

Vancouver:

Boteva L. Investigating transcription, replication and chromatin structure in determining common fragile site instability. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1842/28803.

Council of Science Editors:

Boteva L. Investigating transcription, replication and chromatin structure in determining common fragile site instability. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28803


University of Lethbridge

22. Sidler, Corinne. A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation .

Degree: 2014, University of Lethbridge

 Aging is associated with the functional decline of organs, and results in age-dependent differences in stress-sensitivity. Younger animals are more sensitive to mutagenic insults than… (more)

Subjects/Keywords: aging; genomic instability; histone methyltransferases; human fibroblasts; ionizing radiation; life cyces

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APA (6th Edition):

Sidler, C. (2014). A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation . (Thesis). University of Lethbridge. Retrieved from http://hdl.handle.net/10133/3508

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sidler, Corinne. “A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation .” 2014. Thesis, University of Lethbridge. Accessed January 15, 2021. http://hdl.handle.net/10133/3508.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sidler, Corinne. “A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation .” 2014. Web. 15 Jan 2021.

Vancouver:

Sidler C. A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation . [Internet] [Thesis]. University of Lethbridge; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/10133/3508.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sidler C. A Role of Epigenetics in Aging and the Age-Dependent Response to Ionizing Radiation . [Thesis]. University of Lethbridge; 2014. Available from: http://hdl.handle.net/10133/3508

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

23. Jarvis, Morgan L. Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe .

Degree: Pathology and Molecular Medicine, 2008, Queens University

 Replication fork stalling is a source of potentially tumourigenic genomic instability. The RecQ family helicase, Rqh1, is critical for the prevention of replication fork collapse… (more)

Subjects/Keywords: Genomic instability ; DNA replication

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APA (6th Edition):

Jarvis, M. L. (2008). Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/1227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jarvis, Morgan L. “Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe .” 2008. Thesis, Queens University. Accessed January 15, 2021. http://hdl.handle.net/1974/1227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jarvis, Morgan L. “Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe .” 2008. Web. 15 Jan 2021.

Vancouver:

Jarvis ML. Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe . [Internet] [Thesis]. Queens University; 2008. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1974/1227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jarvis ML. Development of a novel screen protocol for the identification of genes causing replication associated genomic instability in Schizosaccharomyces pombe . [Thesis]. Queens University; 2008. Available from: http://hdl.handle.net/1974/1227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

24. Han, Kyudong. Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages.

Degree: PhD, 2006, Louisiana State University

 LINE-1s (Long interspersed elements or L1s) and Alus are highly successful non-long terminal repeat retrotransposons with copy numbers of ~520,000 and >1 million within the… (more)

Subjects/Keywords: genomic deletion; comparative genomics; genomic instability; primate evolution; retrotransposons

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APA (6th Edition):

Han, K. (2006). Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-08302006-100849 ; https://digitalcommons.lsu.edu/gradschool_dissertations/807

Chicago Manual of Style (16th Edition):

Han, Kyudong. “Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages.” 2006. Doctoral Dissertation, Louisiana State University. Accessed January 15, 2021. etd-08302006-100849 ; https://digitalcommons.lsu.edu/gradschool_dissertations/807.

MLA Handbook (7th Edition):

Han, Kyudong. “Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages.” 2006. Web. 15 Jan 2021.

Vancouver:

Han K. Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages. [Internet] [Doctoral dissertation]. Louisiana State University; 2006. [cited 2021 Jan 15]. Available from: etd-08302006-100849 ; https://digitalcommons.lsu.edu/gradschool_dissertations/807.

Council of Science Editors:

Han K. Retrotransposon mediated genomic fluidity in the human and chimpanzee lineages. [Doctoral Dissertation]. Louisiana State University; 2006. Available from: etd-08302006-100849 ; https://digitalcommons.lsu.edu/gradschool_dissertations/807


Texas Medical Center

25. Pal, Sangita. UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST.

Degree: PhD, 2017, Texas Medical Center

  Aging brings a gradual decline in molecular fidelity and biological functionality, resulting in age related phenotypes and diseases. Despite continued efforts to uncover the… (more)

Subjects/Keywords: Replicative aging; Genomic instability; rDNA instability; Double-strand break repair; Cohesin; Biochemistry; Cell Biology; Molecular Biology; Molecular Genetics

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APA (6th Edition):

Pal, S. (2017). UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/770

Chicago Manual of Style (16th Edition):

Pal, Sangita. “UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed January 15, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/770.

MLA Handbook (7th Edition):

Pal, Sangita. “UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST.” 2017. Web. 15 Jan 2021.

Vancouver:

Pal S. UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Jan 15]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/770.

Council of Science Editors:

Pal S. UNDERSTANDING THE MECHANISM OF GENOMIC INSTABILITY DURING REPLICATIVE AGING IN BUDDING YEAST. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/770


Mississippi State University

26. Esteban-Perez, Clara Ines. GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION.

Degree: PhD, Biological Sciences, 2011, Mississippi State University

  Embryonic stem (ES) cells have the ability to maintain pluripotency and self-renewal during <i>in vitro</i> maintenance, which is a key to their clinical applications.… (more)

Subjects/Keywords: genomic instability; cell lineage commitment; cell differentiation; pluripotency; Embryonic stem cell; repetitive sequences; tumor transformation.

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APA (6th Edition):

Esteban-Perez, C. I. (2011). GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION. (Doctoral Dissertation). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-04082011-115340/ ;

Chicago Manual of Style (16th Edition):

Esteban-Perez, Clara Ines. “GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION.” 2011. Doctoral Dissertation, Mississippi State University. Accessed January 15, 2021. http://sun.library.msstate.edu/ETD-db/theses/available/etd-04082011-115340/ ;.

MLA Handbook (7th Edition):

Esteban-Perez, Clara Ines. “GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION.” 2011. Web. 15 Jan 2021.

Vancouver:

Esteban-Perez CI. GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION. [Internet] [Doctoral dissertation]. Mississippi State University; 2011. [cited 2021 Jan 15]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-04082011-115340/ ;.

Council of Science Editors:

Esteban-Perez CI. GENOMIC INSTABILITY MAY BE A SIGNAL OF HUMAN EMBRYONIC STEM CELL DIFFERENTIATION. [Doctoral Dissertation]. Mississippi State University; 2011. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-04082011-115340/ ;

27. Bodnar-Wachtel, Mélanie. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.

Degree: Docteur es, Immunologie, 2015, Université Claude Bernard – Lyon I

Mon travail de thèse s'intéresse au rôle du récepteur à l'immunité innée NLRP3, composant essentiel de l'inflammasome, dans le développement tumoral pulmonaire. Nos résultats révèlent… (more)

Subjects/Keywords: NLRP3; H2AFX; CBNPC; Réparation; Instabilité génomique; NLRP3; H2AFX; NSCLC; DNA repair; Genomic instability; 571.96

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APA (6th Edition):

Bodnar-Wachtel, M. (2015). Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2015LYO10184

Chicago Manual of Style (16th Edition):

Bodnar-Wachtel, Mélanie. “Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.” 2015. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed January 15, 2021. http://www.theses.fr/2015LYO10184.

MLA Handbook (7th Edition):

Bodnar-Wachtel, Mélanie. “Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.” 2015. Web. 15 Jan 2021.

Vancouver:

Bodnar-Wachtel M. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2015. [cited 2021 Jan 15]. Available from: http://www.theses.fr/2015LYO10184.

Council of Science Editors:

Bodnar-Wachtel M. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2015. Available from: http://www.theses.fr/2015LYO10184


Cornell University

28. Balmus, Gabriel. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo.

Degree: PhD, Physiology, 2013, Cornell University

 The DNA damage response (DDR) represents the primary line of defense against exogenous and endogenous genotoxic agents that threaten the stability of our genomes. The… (more)

Subjects/Keywords: DNA damage; ATM; ATR; HUS1; cell cycle checkpoints; cancer; development; genomic instability

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Balmus, G. (2013). Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33855

Chicago Manual of Style (16th Edition):

Balmus, Gabriel. “Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo.” 2013. Doctoral Dissertation, Cornell University. Accessed January 15, 2021. http://hdl.handle.net/1813/33855.

MLA Handbook (7th Edition):

Balmus, Gabriel. “Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo.” 2013. Web. 15 Jan 2021.

Vancouver:

Balmus G. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1813/33855.

Council of Science Editors:

Balmus G. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33855


Vanderbilt University

29. Tidball, Andrew Martin. A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 The essential micronutrient manganese is enriched in brain, especially the basal ganglia. We sought to identify neuronal signaling pathways responsive to neurologically relevant manganese levels,… (more)

Subjects/Keywords: manganese; induced-pluripotent stem cells; ATM; p53; cell signaling; cytotoxicity; genomic instability; Huntingtons disease

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APA (6th Edition):

Tidball, A. M. (2014). A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14230

Chicago Manual of Style (16th Edition):

Tidball, Andrew Martin. “A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed January 15, 2021. http://hdl.handle.net/1803/14230.

MLA Handbook (7th Edition):

Tidball, Andrew Martin. “A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling.” 2014. Web. 15 Jan 2021.

Vancouver:

Tidball AM. A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1803/14230.

Council of Science Editors:

Tidball AM. A Manganese-Handling Deficit in Huntington’s Disease Selectively Impairs ATM-p53 Signaling. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14230


Queen Mary, University of London

30. Patani, Hemalvi. Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency.

Degree: PhD, 2019, Queen Mary, University of London

 Aberrant DNA hypermethylation of promoter CpG islands in the context of a hypomethylated genome is a hallmark of cancer. DNA hypomethylation is typically associated with… (more)

Subjects/Keywords: Medicine and Dentistry; DNA hypermethylation; genomic instability; gene repression; cancer initiation; oncogenic transformation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Patani, H. (2019). Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/60605 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791864

Chicago Manual of Style (16th Edition):

Patani, Hemalvi. “Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency.” 2019. Doctoral Dissertation, Queen Mary, University of London. Accessed January 15, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/60605 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791864.

MLA Handbook (7th Edition):

Patani, Hemalvi. “Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency.” 2019. Web. 15 Jan 2021.

Vancouver:

Patani H. Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2019. [cited 2021 Jan 15]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/60605 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791864.

Council of Science Editors:

Patani H. Investigating cancer-like DNA methylation patterns and genome stability upon reprogramming to naïve human pluripotency. [Doctoral Dissertation]. Queen Mary, University of London; 2019. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/60605 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791864

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