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You searched for subject:(epithelial to mesenchymal transition). Showing records 1 – 30 of 42193 total matches.

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University of Melbourne

1. Gunasinghe, N. P. A. Devika. Epithelial to mesenchymal transition in human breast cancer cells.

Degree: 2011, University of Melbourne

 Breast cancer is the most frequently diagnosed malignancy in females and accounts for the highest cancer related mortality worldwide. According to the available statistics, more… (more)

Subjects/Keywords: epithelial to mesenchymal transition (EMT); mesenchymal to epithelial transition (MET); breast cancer; metastasis; e-cadherin

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APA (6th Edition):

Gunasinghe, N. P. A. D. (2011). Epithelial to mesenchymal transition in human breast cancer cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37325

Chicago Manual of Style (16th Edition):

Gunasinghe, N P A Devika. “Epithelial to mesenchymal transition in human breast cancer cells.” 2011. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/37325.

MLA Handbook (7th Edition):

Gunasinghe, N P A Devika. “Epithelial to mesenchymal transition in human breast cancer cells.” 2011. Web. 10 Apr 2021.

Vancouver:

Gunasinghe NPAD. Epithelial to mesenchymal transition in human breast cancer cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/37325.

Council of Science Editors:

Gunasinghe NPAD. Epithelial to mesenchymal transition in human breast cancer cells. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/37325


Georgia Tech

2. Zhang, Mengnan. Analysis of the role of miRNAs in ovarian cancer metastasis.

Degree: PhD, Biology, 2019, Georgia Tech

 Cancer mortality is primarily due to metastasis. Metastasis is a complex multi-step process involving, on the molecular level, regulatory control of two key development pathways:… (more)

Subjects/Keywords: miRNA; Gene expression; Ovarian cancer; Epithelial-to-mesenchymal mesenchymal-to-epithelial transition

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APA (6th Edition):

Zhang, M. (2019). Analysis of the role of miRNAs in ovarian cancer metastasis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/62647

Chicago Manual of Style (16th Edition):

Zhang, Mengnan. “Analysis of the role of miRNAs in ovarian cancer metastasis.” 2019. Doctoral Dissertation, Georgia Tech. Accessed April 10, 2021. http://hdl.handle.net/1853/62647.

MLA Handbook (7th Edition):

Zhang, Mengnan. “Analysis of the role of miRNAs in ovarian cancer metastasis.” 2019. Web. 10 Apr 2021.

Vancouver:

Zhang M. Analysis of the role of miRNAs in ovarian cancer metastasis. [Internet] [Doctoral dissertation]. Georgia Tech; 2019. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1853/62647.

Council of Science Editors:

Zhang M. Analysis of the role of miRNAs in ovarian cancer metastasis. [Doctoral Dissertation]. Georgia Tech; 2019. Available from: http://hdl.handle.net/1853/62647


Universiteit Utrecht

3. Pilzecker, B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.

Degree: 2013, Universiteit Utrecht

 Carcinomas are most prevalent type of cancers and arise from an epithelial layer. Epithelial layers have a strict organization; cells are tightly linked through different… (more)

Subjects/Keywords: Carcinomas; Epithelial to Mesenchymal Transition (EMT); E-cadherin; N-cadherin

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APA (6th Edition):

Pilzecker, B. (2013). The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/287101

Chicago Manual of Style (16th Edition):

Pilzecker, B. “The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.” 2013. Masters Thesis, Universiteit Utrecht. Accessed April 10, 2021. http://dspace.library.uu.nl:8080/handle/1874/287101.

MLA Handbook (7th Edition):

Pilzecker, B. “The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.” 2013. Web. 10 Apr 2021.

Vancouver:

Pilzecker B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2021 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/287101.

Council of Science Editors:

Pilzecker B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/287101

4. Sultana, Shamima. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Endometriosis is defined as the presence of endometrial glandular and stromal cells outside of the uterine cavity. A previous study reported that microRNA (miR)-542-3p plays… (more)

Subjects/Keywords: decidualization; endometriosis; mesenchymal‐to‐epithelial transition; microRNA; migration

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APA (6th Edition):

Sultana, S. (2018). Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000611/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sultana, Shamima. “Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed April 10, 2021. http://id.nii.ac.jp/1386/00000611/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sultana, Shamima. “Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.” 2018. Web. 10 Apr 2021.

Vancouver:

Sultana S. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Apr 10]. Available from: http://id.nii.ac.jp/1386/00000611/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sultana S. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000611/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Kao, Yu-Chen. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.

Degree: Master, Biological Sciences, 2017, NSYSU

 The Transforming growth factor β1 (TGF-β1) is belong to transforming growth factor superfamily. Many tumor lesions process are related to TGF-β1, such as: cell proliferation,… (more)

Subjects/Keywords: TGF-β; bromotyrosine derivative; small molecular inhibitors; epithelial-to-mesenchymal transition

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APA (6th Edition):

Kao, Y. (2017). Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kao, Yu-Chen. “Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.” 2017. Thesis, NSYSU. Accessed April 10, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kao, Yu-Chen. “Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.” 2017. Web. 10 Apr 2021.

Vancouver:

Kao Y. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. [Internet] [Thesis]. NSYSU; 2017. [cited 2021 Apr 10]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kao Y. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

6. Dang, Hien T. The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma.

Degree: 2012, Penn State University

 Hepatocellular carcinoma (HCC) is one of the most common malignancies worldwide and is the third leading cause of cancer deaths. Unfortunately, patients with HCC present… (more)

Subjects/Keywords: liver cancer; cancer stem cells; epithelial to mesenchymal transition

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APA (6th Edition):

Dang, H. T. (2012). The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/15868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dang, Hien T. “The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma.” 2012. Thesis, Penn State University. Accessed April 10, 2021. https://submit-etda.libraries.psu.edu/catalog/15868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dang, Hien T. “The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma.” 2012. Web. 10 Apr 2021.

Vancouver:

Dang HT. The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma. [Internet] [Thesis]. Penn State University; 2012. [cited 2021 Apr 10]. Available from: https://submit-etda.libraries.psu.edu/catalog/15868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dang HT. The Regulatioon of Mesenchymal and cancer stem cell phenotypes in hepatocellular carcinoma. [Thesis]. Penn State University; 2012. Available from: https://submit-etda.libraries.psu.edu/catalog/15868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

7. Brethour, Dylan Edward. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.

Degree: 2016, University of Toronto

The prion protein (PrP) was recently found to be evolutionarily linked to a subfamily of ZIP transporters which possess a PrP-like domain. A member of… (more)

Subjects/Keywords: epithelial-to-mesenchymal transition; mass spectrometry; ncam; prion protein; ZIP6; 0317

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APA (6th Edition):

Brethour, D. E. (2016). PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/80225

Chicago Manual of Style (16th Edition):

Brethour, Dylan Edward. “PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.” 2016. Masters Thesis, University of Toronto. Accessed April 10, 2021. http://hdl.handle.net/1807/80225.

MLA Handbook (7th Edition):

Brethour, Dylan Edward. “PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.” 2016. Web. 10 Apr 2021.

Vancouver:

Brethour DE. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1807/80225.

Council of Science Editors:

Brethour DE. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/80225


University of Iowa

8. Nauseef, Jones Trevor. An investigation of the molecular and biophysical properties of metastatic cells.

Degree: PhD, Molecular Physiology and Biophysics, 2015, University of Iowa

  Prostate cancer presents a significant paradox: it is very common, yet rarely fatal. To wit, the prostate is the most common non-skin tissue for… (more)

Subjects/Keywords: publicabstract; Biophysics; Cancer; Epithelial to mesenchymal transition; Metastasis; Microfluidics; Biophysics

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APA (6th Edition):

Nauseef, J. T. (2015). An investigation of the molecular and biophysical properties of metastatic cells. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/3150

Chicago Manual of Style (16th Edition):

Nauseef, Jones Trevor. “An investigation of the molecular and biophysical properties of metastatic cells.” 2015. Doctoral Dissertation, University of Iowa. Accessed April 10, 2021. https://ir.uiowa.edu/etd/3150.

MLA Handbook (7th Edition):

Nauseef, Jones Trevor. “An investigation of the molecular and biophysical properties of metastatic cells.” 2015. Web. 10 Apr 2021.

Vancouver:

Nauseef JT. An investigation of the molecular and biophysical properties of metastatic cells. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2021 Apr 10]. Available from: https://ir.uiowa.edu/etd/3150.

Council of Science Editors:

Nauseef JT. An investigation of the molecular and biophysical properties of metastatic cells. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/3150


University of Illinois – Chicago

9. Li, Yanyang. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.

Degree: 2017, University of Illinois – Chicago

 The communication between the epicardium and the underlying myocardium is crucial not only for proper heart development but also for homeostasis and response to injury… (more)

Subjects/Keywords: Cardiovascular development; NF-κB; Epithelial-to-Mesenchymal Transition; Mass spectrometry

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APA (6th Edition):

Li, Y. (2017). Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yanyang. “Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.” 2017. Thesis, University of Illinois – Chicago. Accessed April 10, 2021. http://hdl.handle.net/10027/21930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yanyang. “Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.” 2017. Web. 10 Apr 2021.

Vancouver:

Li Y. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10027/21930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

10. Ell, Brian James. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .

Degree: PhD, 2013, Princeton University

 In breast cancer, mortality is predominantly associated with metastasis, cancerous spread to distant organs. Understanding tumor dissemination is vitally important to continued therapeutic advancement, but… (more)

Subjects/Keywords: Breast Cancer; Epithelial-to-mesenchymal transition; Metastasis; MicroRNA; Osteoclast

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APA (6th Edition):

Ell, B. J. (2013). Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp015x21tf54k

Chicago Manual of Style (16th Edition):

Ell, Brian James. “Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .” 2013. Doctoral Dissertation, Princeton University. Accessed April 10, 2021. http://arks.princeton.edu/ark:/88435/dsp015x21tf54k.

MLA Handbook (7th Edition):

Ell, Brian James. “Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .” 2013. Web. 10 Apr 2021.

Vancouver:

Ell BJ. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . [Internet] [Doctoral dissertation]. Princeton University; 2013. [cited 2021 Apr 10]. Available from: http://arks.princeton.edu/ark:/88435/dsp015x21tf54k.

Council of Science Editors:

Ell BJ. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . [Doctoral Dissertation]. Princeton University; 2013. Available from: http://arks.princeton.edu/ark:/88435/dsp015x21tf54k


University of Minnesota

11. Fairchild, Corinne Leigh Alinea. Restricting cell movement: the role of Tspan18 in neural crest migration.

Degree: PhD, 2013, University of Minnesota

 The neural crest is a unique population of stem cells that arise from the developing central nervous system of vertebrate embryos. Unlike surrounding neuroepithelial cells,… (more)

Subjects/Keywords: Cadherin; Epithelial-to-mesenchymal transition (EMT); FoxD3; Neural crest; Tetraspanin

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APA (6th Edition):

Fairchild, C. L. A. (2013). Restricting cell movement: the role of Tspan18 in neural crest migration. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/157977

Chicago Manual of Style (16th Edition):

Fairchild, Corinne Leigh Alinea. “Restricting cell movement: the role of Tspan18 in neural crest migration.” 2013. Doctoral Dissertation, University of Minnesota. Accessed April 10, 2021. http://purl.umn.edu/157977.

MLA Handbook (7th Edition):

Fairchild, Corinne Leigh Alinea. “Restricting cell movement: the role of Tspan18 in neural crest migration.” 2013. Web. 10 Apr 2021.

Vancouver:

Fairchild CLA. Restricting cell movement: the role of Tspan18 in neural crest migration. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2021 Apr 10]. Available from: http://purl.umn.edu/157977.

Council of Science Editors:

Fairchild CLA. Restricting cell movement: the role of Tspan18 in neural crest migration. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://purl.umn.edu/157977


University of Melbourne

12. Nguyen, Linh My. Effects of chemotherapy on colorectal liver metastases.

Degree: 2012, University of Melbourne

 Background: Colorectal cancer (CRC) is the fourth most frequently occurring cancer in the world. Despite optimum surgical endeavours, many patients will develop disease recurrence. Treatments… (more)

Subjects/Keywords: OXi4503; Sunitinib; epithelial to mesenchymal transition; colorectal liver metastases

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APA (6th Edition):

Nguyen, L. M. (2012). Effects of chemotherapy on colorectal liver metastases. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37303

Chicago Manual of Style (16th Edition):

Nguyen, Linh My. “Effects of chemotherapy on colorectal liver metastases.” 2012. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/37303.

MLA Handbook (7th Edition):

Nguyen, Linh My. “Effects of chemotherapy on colorectal liver metastases.” 2012. Web. 10 Apr 2021.

Vancouver:

Nguyen LM. Effects of chemotherapy on colorectal liver metastases. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/37303.

Council of Science Editors:

Nguyen LM. Effects of chemotherapy on colorectal liver metastases. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37303


University of Melbourne

13. Chen, Anna. Investigating the role of hypoxia in tumour progression in breast cancer.

Degree: 2015, University of Melbourne

 Metastasis is a major cause of morbidity and mortality in breast cancer patients. The molecular processes and mediators that underpin this process have yet to… (more)

Subjects/Keywords: breast cancer; hypoxia; metastasis; epithelial-to-mesenchymal transition; tumour microenvironment

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APA (6th Edition):

Chen, A. (2015). Investigating the role of hypoxia in tumour progression in breast cancer. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/92001

Chicago Manual of Style (16th Edition):

Chen, Anna. “Investigating the role of hypoxia in tumour progression in breast cancer.” 2015. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/92001.

MLA Handbook (7th Edition):

Chen, Anna. “Investigating the role of hypoxia in tumour progression in breast cancer.” 2015. Web. 10 Apr 2021.

Vancouver:

Chen A. Investigating the role of hypoxia in tumour progression in breast cancer. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/92001.

Council of Science Editors:

Chen A. Investigating the role of hypoxia in tumour progression in breast cancer. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/92001


University of South Florida

14. Richards, Edward J. Function of Long Noncoding RNAs in Breast Cancer.

Degree: 2015, University of South Florida

 Breast cancer is a disease that will be diagnosed in about 1 in 10 women throughout their lifetime. The majority of breast cancers are originated… (more)

Subjects/Keywords: TGFβ; epithelial to mesenchymal transition; metastasis; Cell Biology; Molecular Biology

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APA (6th Edition):

Richards, E. J. (2015). Function of Long Noncoding RNAs in Breast Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richards, Edward J. “Function of Long Noncoding RNAs in Breast Cancer.” 2015. Thesis, University of South Florida. Accessed April 10, 2021. https://scholarcommons.usf.edu/etd/5767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richards, Edward J. “Function of Long Noncoding RNAs in Breast Cancer.” 2015. Web. 10 Apr 2021.

Vancouver:

Richards EJ. Function of Long Noncoding RNAs in Breast Cancer. [Internet] [Thesis]. University of South Florida; 2015. [cited 2021 Apr 10]. Available from: https://scholarcommons.usf.edu/etd/5767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richards EJ. Function of Long Noncoding RNAs in Breast Cancer. [Thesis]. University of South Florida; 2015. Available from: https://scholarcommons.usf.edu/etd/5767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

15. Petrovich, Giulia. Investigating the differential instructive roles of WT1's isoforms.

Degree: PhD, 2016, University of Edinburgh

 The Wilms' tumour suppressor gene Wt1 is a key regulator of embryonic development and tissue homeostasis. In humans, mutation in the gene may lead to… (more)

Subjects/Keywords: 616.99; roles of WT1 isoforms; WT1; epithelial to mesenchymal transition; EMT

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APA (6th Edition):

Petrovich, G. (2016). Investigating the differential instructive roles of WT1's isoforms. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/23423

Chicago Manual of Style (16th Edition):

Petrovich, Giulia. “Investigating the differential instructive roles of WT1's isoforms.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed April 10, 2021. http://hdl.handle.net/1842/23423.

MLA Handbook (7th Edition):

Petrovich, Giulia. “Investigating the differential instructive roles of WT1's isoforms.” 2016. Web. 10 Apr 2021.

Vancouver:

Petrovich G. Investigating the differential instructive roles of WT1's isoforms. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1842/23423.

Council of Science Editors:

Petrovich G. Investigating the differential instructive roles of WT1's isoforms. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/23423

16. Limeta, Angelo. Tackling Metastatic Cancer: From Systems Biology to Therapeutics .

Degree: Chalmers tekniska högskola / Institutionen för biologi och bioteknik, 2019, Chalmers University of Technology

 Most cancer associated deaths are due to metastasis, the process in which a primary tumor migrates into neighboring tissues and forms secondary metastases. This project… (more)

Subjects/Keywords: Cancer; Metastasis; RNA-seq; Mesenchymal-to-Epithelial Transition; CRISPRCas13,; base editing

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APA (6th Edition):

Limeta, A. (2019). Tackling Metastatic Cancer: From Systems Biology to Therapeutics . (Thesis). Chalmers University of Technology. Retrieved from http://hdl.handle.net/20.500.12380/300654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Limeta, Angelo. “Tackling Metastatic Cancer: From Systems Biology to Therapeutics .” 2019. Thesis, Chalmers University of Technology. Accessed April 10, 2021. http://hdl.handle.net/20.500.12380/300654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Limeta, Angelo. “Tackling Metastatic Cancer: From Systems Biology to Therapeutics .” 2019. Web. 10 Apr 2021.

Vancouver:

Limeta A. Tackling Metastatic Cancer: From Systems Biology to Therapeutics . [Internet] [Thesis]. Chalmers University of Technology; 2019. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/20.500.12380/300654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Limeta A. Tackling Metastatic Cancer: From Systems Biology to Therapeutics . [Thesis]. Chalmers University of Technology; 2019. Available from: http://hdl.handle.net/20.500.12380/300654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. García-Escolano, Marta. The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study .

Degree: 2019, University of Alicante

 Inhibitor of Differentiation (ID) proteins are a family of four (ID1-4) bHLH transcription factors that lack the DNA binding domain. They act by forming dimers… (more)

Subjects/Keywords: Inhibitor of differentiation genes; Breast cancer; Epithelial to mesenchymal transition; Immunophenotype

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APA (6th Edition):

García-Escolano, M. (2019). The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study . (Thesis). University of Alicante. Retrieved from http://hdl.handle.net/10045/113562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

García-Escolano, Marta. “The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study .” 2019. Thesis, University of Alicante. Accessed April 10, 2021. http://hdl.handle.net/10045/113562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

García-Escolano, Marta. “The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study .” 2019. Web. 10 Apr 2021.

Vancouver:

García-Escolano M. The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study . [Internet] [Thesis]. University of Alicante; 2019. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10045/113562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

García-Escolano M. The inhibitor of differentiation genes expression and association with epithelial-to-mesenchymal markers in phenotypes of breast cancer: an in vitro and clinicopathological study . [Thesis]. University of Alicante; 2019. Available from: http://hdl.handle.net/10045/113562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

18. Villarreal Ponce, Alvaro P. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.

Degree: Biomedical Sciences, 2017, University of California – Irvine

 The capacity of epithelial cells to acquire enhanced lineage plasticity could depend on their ability to undergo EMT. Investigations performed on cultured epithelial cells support… (more)

Subjects/Keywords: Biology; Biochemistry; EMT; Epithelial Stem Cells; Epithelial-to-Mesenchymal Transition; Mammary gland; Ovol2; Zeb1

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APA (6th Edition):

Villarreal Ponce, A. P. (2017). An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/5b8693wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Villarreal Ponce, Alvaro P. “An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.” 2017. Thesis, University of California – Irvine. Accessed April 10, 2021. http://www.escholarship.org/uc/item/5b8693wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Villarreal Ponce, Alvaro P. “An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.” 2017. Web. 10 Apr 2021.

Vancouver:

Villarreal Ponce AP. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2021 Apr 10]. Available from: http://www.escholarship.org/uc/item/5b8693wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Villarreal Ponce AP. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/5b8693wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

19. Rasmussen, Jeffrey Philip. Epithelial morphogenesis of the Caenorhabditis elegans pharynx.

Degree: PhD, 2012, University of Washington

 The assembly of cells into functional organs requires the coordination of cell shape and polarity with organ architecture. Although defects in cell shape and polarity… (more)

Subjects/Keywords: Caenorhabditis elegans; epithelial polarity; fusogen; laminin; mesenchymal to epithelial transition; tubulogenesis; Molecular and cellular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rasmussen, J. P. (2012). Epithelial morphogenesis of the Caenorhabditis elegans pharynx. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/19742

Chicago Manual of Style (16th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Doctoral Dissertation, University of Washington. Accessed April 10, 2021. http://hdl.handle.net/1773/19742.

MLA Handbook (7th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Web. 10 Apr 2021.

Vancouver:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Internet] [Doctoral dissertation]. University of Washington; 2012. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1773/19742.

Council of Science Editors:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Doctoral Dissertation]. University of Washington; 2012. Available from: http://hdl.handle.net/1773/19742


Universiteit Utrecht

20. Wiebrands, K. A role of epithelial-mesenchymal transitions in carcinogenic progression.

Degree: 2010, Universiteit Utrecht

 The epithelial-mesenchymal transition plays an important role in several developmental processes, tissue repair, but is also associated with fibrosis and cancer. During tumorigenic progression, EMT… (more)

Subjects/Keywords: epithelial-mesenchymal transition; EMT; metastasis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wiebrands, K. (2010). A role of epithelial-mesenchymal transitions in carcinogenic progression. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/190315

Chicago Manual of Style (16th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Masters Thesis, Universiteit Utrecht. Accessed April 10, 2021. http://dspace.library.uu.nl:8080/handle/1874/190315.

MLA Handbook (7th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Web. 10 Apr 2021.

Vancouver:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2021 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315.

Council of Science Editors:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315

21. Mourareau, Céline. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.

Degree: Docteur es, Biologie céllulaire, 2016, Reims

Chaque année 610 000 cancers sont diagnostiqués dans le monde induits par une infection à papillomavirus humains à haut-risque (HPV-HR). Bien que les carcinomes des… (more)

Subjects/Keywords: Carcinome; Oropharynx; Papillomavirus Humain; Transition Epithélio-Mésenchymateuse; Carcinoma; Oropharynx; Human Papillomavirus; Epithelial-To-Mesenchymal Transition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mourareau, C. (2016). Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2016REIMS029

Chicago Manual of Style (16th Edition):

Mourareau, Céline. “Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.” 2016. Doctoral Dissertation, Reims. Accessed April 10, 2021. http://www.theses.fr/2016REIMS029.

MLA Handbook (7th Edition):

Mourareau, Céline. “Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.” 2016. Web. 10 Apr 2021.

Vancouver:

Mourareau C. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. [Internet] [Doctoral dissertation]. Reims; 2016. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2016REIMS029.

Council of Science Editors:

Mourareau C. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. [Doctoral Dissertation]. Reims; 2016. Available from: http://www.theses.fr/2016REIMS029

22. Molina-Castro, Silvia. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.

Degree: Docteur es, Microbiologie Immunologie, 2017, Bordeaux

 Le cancer gastrique (CG) est une maladie multifactorielle, fréquemment associée à l’infection chronique par des souches CagA+ d’Helicobacter pylori. La transition épithélio-mésenchymateuse (EMT) est un… (more)

Subjects/Keywords: Cellules souches gastriques; Transition épithélio-mésenchymateuse; CD44; Cancer stem cells; Epithelial to mesenchymal transition; CD44

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Molina-Castro, S. (2017). Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2017BORD0623

Chicago Manual of Style (16th Edition):

Molina-Castro, Silvia. “Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.” 2017. Doctoral Dissertation, Bordeaux. Accessed April 10, 2021. http://www.theses.fr/2017BORD0623.

MLA Handbook (7th Edition):

Molina-Castro, Silvia. “Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.” 2017. Web. 10 Apr 2021.

Vancouver:

Molina-Castro S. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. [Internet] [Doctoral dissertation]. Bordeaux; 2017. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2017BORD0623.

Council of Science Editors:

Molina-Castro S. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. [Doctoral Dissertation]. Bordeaux; 2017. Available from: http://www.theses.fr/2017BORD0623


University of Vienna

23. Martinez Turtos, Adriana. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.

Degree: 2018, University of Vienna

 Leberkrebs ist der weltweit sechsthäufigste maligne Tumor, der zur zweithäufigsten Mortalität aller Krebserkrankten führt. Das hepatozelluläre Karzinom (HCC) ist der vorherrschende primäre Leberkrebs. Die schlechte… (more)

Subjects/Keywords: 42.13 Molekularbiologie; hepatozelluläre Karzinom / epithelial zu mesenchymalen Transition / extrazelluläre Vesikel; hepatocellular carcinoma / epithelial-to-mesenchymal transition / extracellular vesicles

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APA (6th Edition):

Martinez Turtos, A. (2018). Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/51143/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martinez Turtos, Adriana. “Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.” 2018. Thesis, University of Vienna. Accessed April 10, 2021. http://othes.univie.ac.at/51143/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martinez Turtos, Adriana. “Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.” 2018. Web. 10 Apr 2021.

Vancouver:

Martinez Turtos A. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. [Internet] [Thesis]. University of Vienna; 2018. [cited 2021 Apr 10]. Available from: http://othes.univie.ac.at/51143/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martinez Turtos A. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. [Thesis]. University of Vienna; 2018. Available from: http://othes.univie.ac.at/51143/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Carter, Lauren. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .

Degree: 2018, University of Ottawa

 The ovarian surface epithelium (OSE) is a monolayer of cells surrounding the ovary that is ruptured during ovulation. After ovulation the wound is repaired, however… (more)

Subjects/Keywords: ovarian surface epithelium; ovulation; stemness; epithelial-to-mesenchymal transition; wound repair; Cox2; Brca1; Snai1

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APA (6th Edition):

Carter, L. (2018). Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/38491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carter, Lauren. “Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .” 2018. Thesis, University of Ottawa. Accessed April 10, 2021. http://hdl.handle.net/10393/38491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carter, Lauren. “Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .” 2018. Web. 10 Apr 2021.

Vancouver:

Carter L. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10393/38491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carter L. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/38491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tulane University

25. Hoang, Van. Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer.

Degree: 2016, Tulane University

Triple-negative breast cancer (TNBC) presents a clinical challenge due to the aggressive nature of the disease and a lack of targeted therapies. Constitutive activation of… (more)

Subjects/Keywords: MAPK signaling (MEK1/2, MEK5); triple-negative breast cancer (TNBC); epithelial-to-mesenchymal transition (EMT)

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APA (6th Edition):

Hoang, V. (2016). Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:74409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoang, Van. “Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer.” 2016. Thesis, Tulane University. Accessed April 10, 2021. https://digitallibrary.tulane.edu/islandora/object/tulane:74409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoang, Van. “Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer.” 2016. Web. 10 Apr 2021.

Vancouver:

Hoang V. Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer. [Internet] [Thesis]. Tulane University; 2016. [cited 2021 Apr 10]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:74409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoang V. Differential regulation of the EMT axis by MEK1/2 and MEK5 in triple-negative breast cancer. [Thesis]. Tulane University; 2016. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:74409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Akrida, Ioanna. Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού.

Degree: 2018, University of Patras; Πανεπιστήμιο Πατρών

Phyllodes breast tumors (PTs) are rare fibroepithelial neoplasms, currently classified as benign, borderline and malignant. PTs can recur locally and may have metastatic potential. They… (more)

Subjects/Keywords: Επιθηλιομεσενχυματική μετατροπή (ΕΜΤ); Φυλλοειδείς όγκοι μαστού; Epithelial to mesenchymal transition; Phyllodes breast tumors

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APA (6th Edition):

Akrida, I. (2018). Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/45271

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akrida, Ioanna. “Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού.” 2018. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed April 10, 2021. http://hdl.handle.net/10442/hedi/45271.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akrida, Ioanna. “Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού.” 2018. Web. 10 Apr 2021.

Vancouver:

Akrida I. Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2018. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10442/hedi/45271.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akrida I. Μελέτη σηματοδοτικών οδών που εμπλέκονται στην επιθηλιο-μεσεγχυματική μετατροπή στους φυλλοειδείς όγκους μαστού. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2018. Available from: http://hdl.handle.net/10442/hedi/45271

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

27. Wokpetah, Joseph Mawumenyo. Regulation of cancer stem cells by the tumor microenvironment.

Degree: 2014, Penn State University

 Cancer is the abnormal growth of cells that tend to multiply in an uncontrolled way and, in some cases, metastasize to other locations in the… (more)

Subjects/Keywords: Epithelial to mesenchymal transition; metastasis; transforming growth factor beta; Young's modulus; upregulation; downregulation

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APA (6th Edition):

Wokpetah, J. M. (2014). Regulation of cancer stem cells by the tumor microenvironment. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/21336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wokpetah, Joseph Mawumenyo. “Regulation of cancer stem cells by the tumor microenvironment.” 2014. Thesis, Penn State University. Accessed April 10, 2021. https://submit-etda.libraries.psu.edu/catalog/21336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wokpetah, Joseph Mawumenyo. “Regulation of cancer stem cells by the tumor microenvironment.” 2014. Web. 10 Apr 2021.

Vancouver:

Wokpetah JM. Regulation of cancer stem cells by the tumor microenvironment. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Apr 10]. Available from: https://submit-etda.libraries.psu.edu/catalog/21336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wokpetah JM. Regulation of cancer stem cells by the tumor microenvironment. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/21336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. G. Celesti. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.

Degree: 2011, Università degli Studi di Milano

 Background. Colorectal cancer (CRC) is a major cause of death for cancer in western countries, ranking third in both sexes. Therapeutic developments in the past… (more)

Subjects/Keywords: epithelial to mesenchymal transition; colorectal cancer; metastasis; twist1; Settore BIO/10 - Biochimica

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APA (6th Edition):

Celesti, G. (2011). MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/158084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Celesti, G.. “MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.” 2011. Thesis, Università degli Studi di Milano. Accessed April 10, 2021. http://hdl.handle.net/2434/158084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Celesti, G.. “MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.” 2011. Web. 10 Apr 2021.

Vancouver:

Celesti G. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2434/158084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Celesti G. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/158084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

29. Maitah, Ma'in Yehya. Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer.

Degree: PhD, Pathology, 2011, Wayne State University

  The American Cancer Society estimated that 222,520 Americans were diagnosed with lung cancer and 157,300 died of lung cancer in 2010 (Jemal et al.… (more)

Subjects/Keywords: Cancer; Epithelial to mesenchymal transition; Lung Cancer; Sonic Hedgehog; TGF-beta; Molecular Biology; Oncology; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maitah, M. Y. (2011). Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/320

Chicago Manual of Style (16th Edition):

Maitah, Ma'in Yehya. “Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer.” 2011. Doctoral Dissertation, Wayne State University. Accessed April 10, 2021. https://digitalcommons.wayne.edu/oa_dissertations/320.

MLA Handbook (7th Edition):

Maitah, Ma'in Yehya. “Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer.” 2011. Web. 10 Apr 2021.

Vancouver:

Maitah MY. Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2021 Apr 10]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/320.

Council of Science Editors:

Maitah MY. Hedgehog signaling: a potential therapeutic target for non-small cell lung cancer. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/320


Washington State University

30. [No author]. Mechanisms of Endometrial Regeneration .

Degree: 2013, Washington State University

 The uterus is one of the most plastic adult organs with tremendous regenerative capacity. The endometrium (innermost lining of the uterus) undergoes regular cycles of… (more)

Subjects/Keywords: Biology; Endometrial regeneration; Endometrium; Mesenchymal-to-epithelial transition; Stem Cells; Transdifferentiation; Uterus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (2013). Mechanisms of Endometrial Regeneration . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/4771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Mechanisms of Endometrial Regeneration .” 2013. Thesis, Washington State University. Accessed April 10, 2021. http://hdl.handle.net/2376/4771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Mechanisms of Endometrial Regeneration .” 2013. Web. 10 Apr 2021.

Vancouver:

author] [. Mechanisms of Endometrial Regeneration . [Internet] [Thesis]. Washington State University; 2013. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2376/4771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Mechanisms of Endometrial Regeneration . [Thesis]. Washington State University; 2013. Available from: http://hdl.handle.net/2376/4771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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