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You searched for subject:(deubiquitinase). Showing records 1 – 21 of 21 total matches.

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University of California – Berkeley

1. Worden, Evan Josiah. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.

Degree: Molecular & Cell Biology, 2016, University of California – Berkeley

 The 26S proteasome is responsible for selective protein degradation in eukaryotic cells. Polyubiquitin chains mark proteins for degradation by the proteasome, but before degradation can… (more)

Subjects/Keywords: Biochemistry; deubiquitinase; proteasome; ubiquitin

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APA (6th Edition):

Worden, E. J. (2016). Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Thesis, University of California – Berkeley. Accessed March 20, 2019. http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Web. 20 Mar 2019.

Vancouver:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Internet] [Thesis]. University of California – Berkeley; 2016. [cited 2019 Mar 20]. Available from: http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Thesis]. University of California – Berkeley; 2016. Available from: http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Purdue University

2. Wade, Cameron. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.

Degree: MS, PULSe, 2016, Purdue University

 ElaD is a cysteine protease found in Escherichia coli (E. coli) and has been shown to function as a deubiquitinating enzyme (DUB). However, ubiquitin and… (more)

Subjects/Keywords: Bacteria; Deubiquitinase; Escherichia Coli; Ubiquitin

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APA (6th Edition):

Wade, C. (2016). Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. (Thesis). Purdue University. Retrieved from https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Thesis, Purdue University. Accessed March 20, 2019. https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Web. 20 Mar 2019.

Vancouver:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Internet] [Thesis]. Purdue University; 2016. [cited 2019 Mar 20]. Available from: https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Thesis]. Purdue University; 2016. Available from: https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

3. Yates, Peter Christian. The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

 Directed cellular migration, also known as homing, is the hallmark of an efficient and effective immune system. Not only is cellular migration a necessary requirement… (more)

Subjects/Keywords: CCR9; MYSM1; deubiquitinase; trafficking

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APA (6th Edition):

Yates, P. C. (2013). The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/345310/rec/6758

Chicago Manual of Style (16th Edition):

Yates, Peter Christian. “The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes.” 2013. Doctoral Dissertation, University of Southern California. Accessed March 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/345310/rec/6758.

MLA Handbook (7th Edition):

Yates, Peter Christian. “The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes.” 2013. Web. 20 Mar 2019.

Vancouver:

Yates PC. The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2019 Mar 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/345310/rec/6758.

Council of Science Editors:

Yates PC. The histone H2A deubiquitinase MYSM1 regulates CCR9 expression on CD4⁺ T cells and thymocytes. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/345310/rec/6758


University of Washington

4. Whedon, Sam. Chemical strategies for investigation of deubiquitinases.

Degree: PhD, 2019, University of Washington

 Regulation of protein structure and function by post-translational modification is a key mechanism in cellular homeostasis. Among known modifications the small protein ubiquitin is unique… (more)

Subjects/Keywords: dehydroalanine; deubiquitinase; selenocysteine; ubiquitin; Organic chemistry; Chemistry

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APA (6th Edition):

Whedon, S. (2019). Chemical strategies for investigation of deubiquitinases. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/43327

Chicago Manual of Style (16th Edition):

Whedon, Sam. “Chemical strategies for investigation of deubiquitinases.” 2019. Doctoral Dissertation, University of Washington. Accessed March 20, 2019. http://hdl.handle.net/1773/43327.

MLA Handbook (7th Edition):

Whedon, Sam. “Chemical strategies for investigation of deubiquitinases.” 2019. Web. 20 Mar 2019.

Vancouver:

Whedon S. Chemical strategies for investigation of deubiquitinases. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1773/43327.

Council of Science Editors:

Whedon S. Chemical strategies for investigation of deubiquitinases. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/43327


University of Toronto

5. Lui, To-Hang. Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling.

Degree: 2010, University of Toronto

The Wnt pathway is a fundamental signalling pathway conserved in all animals, regulating growth, differentiation, embryonic development, and tissue homeostasis in adults. Wnt signalling is… (more)

Subjects/Keywords: wnt; usp34; dub; axin; ubiquitin specific protease; deubiquitinase; 0307

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APA (6th Edition):

Lui, T. (2010). Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24264

Chicago Manual of Style (16th Edition):

Lui, To-Hang. “Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling.” 2010. Masters Thesis, University of Toronto. Accessed March 20, 2019. http://hdl.handle.net/1807/24264.

MLA Handbook (7th Edition):

Lui, To-Hang. “Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling.” 2010. Web. 20 Mar 2019.

Vancouver:

Lui T. Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1807/24264.

Council of Science Editors:

Lui T. Ubiquitin Specific Protease 34 (USP34), a New Positive Regulator of Canonical Wnt/β-catenin Signalling. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24264


University of Manchester

6. Palazon, Pablo. Mechanisms and consequences of inflammasome activation.

Degree: 2017, University of Manchester

 Inflammation is the response of the body to injury or threats. Immune cells such as macrophages have a crucial role in controlling and regulating this… (more)

Subjects/Keywords: IL-1; IL-18; IL-18BP; USP7; USP47; Inflammation; Inflammasome; Deubiquitinase

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APA (6th Edition):

Palazon, P. (2017). Mechanisms and consequences of inflammasome activation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829

Chicago Manual of Style (16th Edition):

Palazon, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Doctoral Dissertation, University of Manchester. Accessed March 20, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829.

MLA Handbook (7th Edition):

Palazon, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Web. 20 Mar 2019.

Vancouver:

Palazon P. Mechanisms and consequences of inflammasome activation. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2019 Mar 20]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829.

Council of Science Editors:

Palazon P. Mechanisms and consequences of inflammasome activation. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829


Cornell University

7. Akturk, Anil. MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY .

Degree: 2018, Cornell University

 Legionella pneumophila manipulates a wide array of host cellular processes during infection; one that appears to be highly altered is the ubiquitination pathway. The SidE… (more)

Subjects/Keywords: Deubiquitinase; Legionella pneumophila; Phosphodiesterase; SidE family; Ubiquitin; Ubiquitination; Biochemistry; Biophysics

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APA (6th Edition):

Akturk, A. (2018). MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/59276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akturk, Anil. “MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY .” 2018. Thesis, Cornell University. Accessed March 20, 2019. http://hdl.handle.net/1813/59276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akturk, Anil. “MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY .” 2018. Web. 20 Mar 2019.

Vancouver:

Akturk A. MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY . [Internet] [Thesis]. Cornell University; 2018. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1813/59276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akturk A. MODULATION OF UBIQUITINATION PATHWAY BY LEGIONELLA SidE EFFECTOR FAMILY . [Thesis]. Cornell University; 2018. Available from: http://hdl.handle.net/1813/59276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

8. Volk, Aaron Brian. Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  Two coronaviruses (CoVs)—severe acute respiratory syndrome (SARS) virus and Middle East respiratory syndrome (MERS) virus—have emerged in the 21st century from animal reservoirs into… (more)

Subjects/Keywords: coronavirus; deubiquitinase; endoribonuclease; interferon; RNA-seq; vaccine; Virology

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APA (6th Edition):

Volk, A. B. (2018). Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Volk, Aaron Brian. “Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses.” 2018. Thesis, Loyola University Chicago. Accessed March 20, 2019. https://ecommons.luc.edu/luc_theses/3758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Volk, Aaron Brian. “Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses.” 2018. Web. 20 Mar 2019.

Vancouver:

Volk AB. Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2019 Mar 20]. Available from: https://ecommons.luc.edu/luc_theses/3758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Volk AB. Surveying Host Innate Immune Responses To Interferon Antagonist-Deficient Murine Coronaviruses. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

9. Palazón, Pablo. Mechanisms and consequences of inflammasome activation.

Degree: PhD, 2017, University of Manchester

 Inflammation is the response of the body to injury or threats. Immune cells such as macrophages have a crucial role in controlling and regulating this… (more)

Subjects/Keywords: Inflammasome; Inflammation; USP47; Deubiquitinase; IL-18BP; IL-18; IL-1; USP7

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APA (6th Edition):

Palazón, P. (2017). Mechanisms and consequences of inflammasome activation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492

Chicago Manual of Style (16th Edition):

Palazón, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Doctoral Dissertation, University of Manchester. Accessed March 20, 2019. https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492.

MLA Handbook (7th Edition):

Palazón, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Web. 20 Mar 2019.

Vancouver:

Palazón P. Mechanisms and consequences of inflammasome activation. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2019 Mar 20]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492.

Council of Science Editors:

Palazón P. Mechanisms and consequences of inflammasome activation. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492


Princeton University

10. Li, Wenyang. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .

Degree: PhD, 2017, Princeton University

 Abstract Cancer initiation and progression is orchestrated by a complicated web of signaling pathways with numerous effector proteins and RNAs. My studies focus on identifying… (more)

Subjects/Keywords: Cancer; Deubiquitinase; Epithelial to mesenchymal transition; Long non-coding RNA; SLUG; TGF beta

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APA (6th Edition):

Li, W. (2017). Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01st74ct124

Chicago Manual of Style (16th Edition):

Li, Wenyang. “Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .” 2017. Doctoral Dissertation, Princeton University. Accessed March 20, 2019. http://arks.princeton.edu/ark:/88435/dsp01st74ct124.

MLA Handbook (7th Edition):

Li, Wenyang. “Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .” 2017. Web. 20 Mar 2019.

Vancouver:

Li W. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . [Internet] [Doctoral dissertation]. Princeton University; 2017. [cited 2019 Mar 20]. Available from: http://arks.princeton.edu/ark:/88435/dsp01st74ct124.

Council of Science Editors:

Li W. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . [Doctoral Dissertation]. Princeton University; 2017. Available from: http://arks.princeton.edu/ark:/88435/dsp01st74ct124

11. Campagne, Antoine. Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex.

Degree: Docteur es, Biochimie, 2015, Université Pierre et Marie Curie – Paris VI

BAP1 est un suppresseur de tumeurs dont le nombre de partenaires protéiques rend complexe l'appréhension de son rôle dans la cellule. Chez la Drosophile, BAP1… (more)

Subjects/Keywords: Polycomb; PR-DUB; BAP1; ASXL; H2A; Deubiquitinase; Polycomb; PR-DUB; BAP1; 572.8

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APA (6th Edition):

Campagne, A. (2015). Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066624

Chicago Manual of Style (16th Edition):

Campagne, Antoine. “Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed March 20, 2019. http://www.theses.fr/2015PA066624.

MLA Handbook (7th Edition):

Campagne, Antoine. “Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex.” 2015. Web. 20 Mar 2019.

Vancouver:

Campagne A. Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Mar 20]. Available from: http://www.theses.fr/2015PA066624.

Council of Science Editors:

Campagne A. Etude du complexe Polycomb PR-DUB : une approche mécanistique : A mechanistic study of the Polycomb PR-DUB complex. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066624


University of Central Florida

12. Roy, Arpita. Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1).

Degree: 2012, University of Central Florida

 Major diseases such as cardiovascular diseases, diabetes, obesity and tumor growth are known to involve inflammatory angiogenesis. MCP-induced protein 1 (MCPIP1) encoded by ZC3H12A gene,… (more)

Subjects/Keywords: Angiogenesis; mcpip; oxidative stress; er stress; deubiquitinase; Molecular Biology; Dissertations, Academic  – Medicine, Medicine  – Dissertations, Academic

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APA (6th Edition):

Roy, A. (2012). Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1). (Doctoral Dissertation). University of Central Florida. Retrieved from http://stars.library.ucf.edu/etd/2464

Chicago Manual of Style (16th Edition):

Roy, Arpita. “Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1).” 2012. Doctoral Dissertation, University of Central Florida. Accessed March 20, 2019. http://stars.library.ucf.edu/etd/2464.

MLA Handbook (7th Edition):

Roy, Arpita. “Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1).” 2012. Web. 20 Mar 2019.

Vancouver:

Roy A. Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1). [Internet] [Doctoral dissertation]. University of Central Florida; 2012. [cited 2019 Mar 20]. Available from: http://stars.library.ucf.edu/etd/2464.

Council of Science Editors:

Roy A. Molecular Mechanisms Involved In Inflammatory Angiogenesis Induced By Monocyte Chemotactic Protein Induced Protein-1 (mcpip1). [Doctoral Dissertation]. University of Central Florida; 2012. Available from: http://stars.library.ucf.edu/etd/2464

13. Mathis, Bryan J. Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD.

Degree: PhD, Biomedical Science, 2016, University of South Carolina

  The cylindromatosis (CYLD) is a K63-linked deubiquitinase (DUB) that has been linked to the regulation of multiple physiological or pathological processes, such as neural… (more)

Subjects/Keywords: Other Medicine and Health Sciences; Transcriptional; Regulation; NRF2; Deubiquitinase CYLD

…9 1.3 THE DEUBIQUITINASE CYLD… …focus on its possible roles in heart disease. 1.3 THE DEUBIQUITINASE CYLD INTRODUCTION TO… 

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APA (6th Edition):

Mathis, B. J. (2016). Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/3460

Chicago Manual of Style (16th Edition):

Mathis, Bryan J. “Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD.” 2016. Doctoral Dissertation, University of South Carolina. Accessed March 20, 2019. https://scholarcommons.sc.edu/etd/3460.

MLA Handbook (7th Edition):

Mathis, Bryan J. “Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD.” 2016. Web. 20 Mar 2019.

Vancouver:

Mathis BJ. Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD. [Internet] [Doctoral dissertation]. University of South Carolina; 2016. [cited 2019 Mar 20]. Available from: https://scholarcommons.sc.edu/etd/3460.

Council of Science Editors:

Mathis BJ. Transcriptional And Post-Transcriptional Regulation Of NRF2 In The Heart By The Deubiquitinase CYLD. [Doctoral Dissertation]. University of South Carolina; 2016. Available from: https://scholarcommons.sc.edu/etd/3460


Université de Montréal

14. Pak, Helen. The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress .

Degree: 2013, Université de Montréal

 BRCA1 est un suppresseur de tumeur majeur jouant un rôle dans la transcription, la réparation de l’ADN et le maintien de la stabilité génomique. En… (more)

Subjects/Keywords: BRCA1; Dommage à l’ADN; DNA damage; Réparation de l’ADN; DNA repair; Recombinaison Homologue; Homologous recombination; Ubiquitination; Ubiquitin ligase; Déubiquitinase; Deubiquitinase; Dégradation protéasomale; Méthyle méthanesulfonate; Methyl methanesulfonate; Irradiation gamma; Ionizing radiation

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APA (6th Edition):

Pak, H. (2013). The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pak, Helen. “The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress .” 2013. Thesis, Université de Montréal. Accessed March 20, 2019. http://hdl.handle.net/1866/9127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pak, Helen. “The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress .” 2013. Web. 20 Mar 2019.

Vancouver:

Pak H. The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1866/9127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pak H. The role of ubiquitination and deubiquitination in the regulation of BRCA1 function during genotoxic stress . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

15. Yu, Helen. Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 .

Degree: 2015, Université de Montréal

 La déubiquitinase BAP1 (« BRCA1-Associated Protein1 ») a initialement été isolée pour sa capacité de promouvoir la fonction suppressive de tumeurs de BRCA1. BAP1 est… (more)

Subjects/Keywords: Déubiquitinase; Chromatine; Ubiquitine; H2A; Transcription; Dommage à l’ADN; Bris double brin d’ADN; Recombinaison homologue; Deubiquitinase; Ubiquitin; Chromatin; DNA damage; Double strand break; Homologous recombination

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, H. (2015). Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Helen. “Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 .” 2015. Thesis, Université de Montréal. Accessed March 20, 2019. http://hdl.handle.net/1866/12094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Helen. “Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 .” 2015. Web. 20 Mar 2019.

Vancouver:

Yu H. Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 . [Internet] [Thesis]. Université de Montréal; 2015. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1866/12094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu H. Caractérisation fonctionnelle du suppresseur de tumeurs BAP1 . [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/12094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

16. Mashtalir, Nazar. Regulation of BAP1 tumor suppressor complex by post-translational modifications .

Degree: 2014, Université de Montréal

 Le régulateur transcriptionnel BAP1 est une déubiquitinase nucléaire (DUB) dont le substrat est l’histone H2A modifiée par monoubiquitination au niveau des residus lysines 118 et… (more)

Subjects/Keywords: Chromatine; Marque épigénctique; O-GlcNAcylation; Régulation protéolytique; Ubiquitination; Déubiquitinase; Ubiquitine ligase atypique; Activité auto-catalytique; Cancer; O-linked beta-N-acetylglucosamine transferase; Chromatin; Epigenetic mark; Proteolytic processing; Ubiquitination; Deubiquitinase; Atypical ubiquitin ligase; Autocatalytic activity; Host cell factor 1; BRCA1-associated protein 1; UBE2O

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APA (6th Edition):

Mashtalir, N. (2014). Regulation of BAP1 tumor suppressor complex by post-translational modifications . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mashtalir, Nazar. “Regulation of BAP1 tumor suppressor complex by post-translational modifications .” 2014. Thesis, Université de Montréal. Accessed March 20, 2019. http://hdl.handle.net/1866/12772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mashtalir, Nazar. “Regulation of BAP1 tumor suppressor complex by post-translational modifications .” 2014. Web. 20 Mar 2019.

Vancouver:

Mashtalir N. Regulation of BAP1 tumor suppressor complex by post-translational modifications . [Internet] [Thesis]. Université de Montréal; 2014. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1866/12772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mashtalir N. Regulation of BAP1 tumor suppressor complex by post-translational modifications . [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/12772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Reed, Benjamin James. From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis.

Degree: PhD, 2015, University of Washington

 Ribosomes are a key component of cell cycle regulation and protein production. Proper control of ribosome synthesis is necessary for normal cellular functioning. Improper regulation… (more)

Subjects/Keywords: Deubiquitinase; Disorder; Ribosome; Ubiquitin; Ubp10; USP36; Molecular biology; Cellular biology; Biochemistry; pharmacology

…diamidino-2-phenylindole DHR2 DEAH-box RNA helicase 2 DUB Deubiquitinase GAD Gal4 activation… …ubiquitin facilitated by a deubiquitinase (Amerik and Hochstrasser, 2004; Baek, 2003)… …deubiquitinase Ubp10 lead us to identify ubiquitin as a key modifier in RNA Polymerase I turnover. I… …IS REGULATED BY DEUBIQUITINASE UBP10 History of Ubp10: Identifying new roles outside of… …was first identified as a deubiquitinase that associated with the yeast chromatin silencing… 

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APA (6th Edition):

Reed, B. J. (2015). From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/33230

Chicago Manual of Style (16th Edition):

Reed, Benjamin James. “From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis.” 2015. Doctoral Dissertation, University of Washington. Accessed March 20, 2019. http://hdl.handle.net/1773/33230.

MLA Handbook (7th Edition):

Reed, Benjamin James. “From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis.” 2015. Web. 20 Mar 2019.

Vancouver:

Reed BJ. From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1773/33230.

Council of Science Editors:

Reed BJ. From yeast to mammals: the exploration of a conserved, intrinsically disordered deubiquitinase that regulates ribosomal RNA synthesis. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/33230


Université de Montréal

18. Mathien, Simon. Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 .

Degree: 2017, Université de Montréal

Subjects/Keywords: MAP Kinase; ERK3; ubiquitination; stabilité; migration cellulaire; ubiquitine ligase; déubiquitinase; signalisation cellulaire; Protein stability; Cell migration; Ubiquitin ligase; Deubiquitinase; Cell signaling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mathien, S. (2017). Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/19315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mathien, Simon. “Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 .” 2017. Thesis, Université de Montréal. Accessed March 20, 2019. http://hdl.handle.net/1866/19315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mathien, Simon. “Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 .” 2017. Web. 20 Mar 2019.

Vancouver:

Mathien S. Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1866/19315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mathien S. Identification des composantes du système ubiquitine-protéasome régulant la stabilité de la MAPK atypique ERK3 . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/19315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Perry, Jeffrey William. Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis.

Degree: PhD, Microbiology and Immunology, 2012, University of Michigan

 Viruses are obligate intracellular parasites that require their hosts for all steps of the viral life cycle. The mechanism of how an inert particle of… (more)

Subjects/Keywords: Virus; Entry; Endocytosis; Ubiquitin; Deubiquitinase; Unfolded Protein Response; Microbiology and Immunology; Science

…Activity of a Small Molecule Deubiquitinase Inhibitor Occurs via Induction of the Unfolded… …independent. . . . . . . . . . . 207 5.4 Antiviral Activity of a Small Molecule Deubiquitinase… …the host deubiquitinase USP14 in murine macrophages… 

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APA (6th Edition):

Perry, J. W. (2012). Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/93905

Chicago Manual of Style (16th Edition):

Perry, Jeffrey William. “Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis.” 2012. Doctoral Dissertation, University of Michigan. Accessed March 20, 2019. http://hdl.handle.net/2027.42/93905.

MLA Handbook (7th Edition):

Perry, Jeffrey William. “Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis.” 2012. Web. 20 Mar 2019.

Vancouver:

Perry JW. Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/2027.42/93905.

Council of Science Editors:

Perry JW. Murine Norovirus 1 Productively Infects Murine Macrophages in a Dynamin II-, Cholesterol-, and USP14 Dependent Mechanism but is Independent of Endosome Acidification, Clathrin, Caveolin, Flotillin, GRAF 1, and Phagocytosis. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/93905

20. Yang, Wei-Lei. The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation.

Degree: PhD, 2013, Texas Medical Center

  Akt (also known as protein kinase B) serves a central regulator in PI3K/Akt signaling pathways to regulate numerous physiological functions including cell proliferation, survival… (more)

Subjects/Keywords: Ubiquitination; E3 ligase; TRAF6; PKB/Akt; deubiquitination; deubiquitinase; CYLD; tumorigenesis; membrane translocation; protein kinase activation; Cancer Biology; Cell Biology; Medicine and Health Sciences

…reversible by deubiquitinase 25 1-12. The role of DUBs in regulating NF-κB signaling pathways 28… …oncogenic role of TRAF6 in tumorigenesis 128 5-7. The role of deubiquitinase CYLD in regulating… …pathways regulate activity of TRAF6 E3 ligase and CYLD deubiquitinase? 135 6-2. What is the… …working model for Akt activation 78 Figure 4-1. CYLD deubiquitinase promotes deubiquitination… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, W. (2013). The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/392

Chicago Manual of Style (16th Edition):

Yang, Wei-Lei. “The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed March 20, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/392.

MLA Handbook (7th Edition):

Yang, Wei-Lei. “The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation.” 2013. Web. 20 Mar 2019.

Vancouver:

Yang W. The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2019 Mar 20]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/392.

Council of Science Editors:

Yang W. The Role of K63-linked Ubiquitination Cycles in Akt Kinase Activation. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/392

21. Rivière, Gwladys. Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2.

Degree: Docteur es, Chimie, 2011, Université Claude Bernard – Lyon I

Au cours de cette thèse, nous avons étudié deux protéines par RMN : la créatine kinase musculaire (CK-MM) et le domaine UIM-SH3 de la protéine… (more)

Subjects/Keywords: Interaction protéine-protéine et protéine-ligands par RMN; Créatine kinase; Analogue de l'intermédiaire de transition catalytique (TSAC); Motif d'interaction à l'ubiquitine (UIM-SH3); Transduction de l'adaptateur du signal moléculaire 2 (STAM2); Ubiquitine protéase spécifique Y (UBPY); Déubiquitinase; Protein-protein and protein-ligands interaction by NMR; Creatine kinase; Transition state analogue complex (TSAC); Ubiquitin interacting motif (UIM-SH3); Signal transducing adaptator molecular 2 (STAM2); Ubiquitin specific protease Y (UBPY); Deubiquitinase; 572.633

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APA (6th Edition):

Rivière, G. (2011). Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10285

Chicago Manual of Style (16th Edition):

Rivière, Gwladys. “Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed March 20, 2019. http://www.theses.fr/2011LYO10285.

MLA Handbook (7th Edition):

Rivière, Gwladys. “Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2.” 2011. Web. 20 Mar 2019.

Vancouver:

Rivière G. Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2019 Mar 20]. Available from: http://www.theses.fr/2011LYO10285.

Council of Science Editors:

Rivière G. Étude par RMN de la créatine kinase musculaire et d’un nouveau domaine de liaison à l’ubiquitine dans la protéine STAM2 : NMR study of the creatine kinase muscle and a new binding domain in the protein ubiquitin STAM 2. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10285

.