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You searched for subject:(corticostriatal). Showing records 1 – 17 of 17 total matches.

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University of Texas – Austin

1. Yi, Han-Gyol. The role of corticostriatal loops in auditory category learning.

Degree: PhD, Communication Sciences and Disorders, 2017, University of Texas – Austin

 Sounds can signal danger (e.g., roar of a lion), pleasure, (e.g., music), or carry linguistic relevance (e.g. speech). For sounds to guide behavior, the complex… (more)

Subjects/Keywords: Category learning; Auditory; Corticostriatal loops

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APA (6th Edition):

Yi, H. (2017). The role of corticostriatal loops in auditory category learning. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/62983

Chicago Manual of Style (16th Edition):

Yi, Han-Gyol. “The role of corticostriatal loops in auditory category learning.” 2017. Doctoral Dissertation, University of Texas – Austin. Accessed April 16, 2021. http://hdl.handle.net/2152/62983.

MLA Handbook (7th Edition):

Yi, Han-Gyol. “The role of corticostriatal loops in auditory category learning.” 2017. Web. 16 Apr 2021.

Vancouver:

Yi H. The role of corticostriatal loops in auditory category learning. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2017. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2152/62983.

Council of Science Editors:

Yi H. The role of corticostriatal loops in auditory category learning. [Doctoral Dissertation]. University of Texas – Austin; 2017. Available from: http://hdl.handle.net/2152/62983


Universidade do Rio Grande do Norte

2. Brys, Ivani. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal .

Degree: 2014, Universidade do Rio Grande do Norte

 Parkinson disease (PD) is associated with motor symptoms and dopaminergic cell loss in the nigrostriatal pathway. Alpha-synuclein is the major component of the Lewy bodies,… (more)

Subjects/Keywords: Doença de Parkinson; Alfa-sinucleína; circuito corticostriatal

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APA (6th Edition):

Brys, I. (2014). Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal . (Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/19589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brys, Ivani. “Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal .” 2014. Thesis, Universidade do Rio Grande do Norte. Accessed April 16, 2021. http://repositorio.ufrn.br/handle/123456789/19589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brys, Ivani. “Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal .” 2014. Web. 16 Apr 2021.

Vancouver:

Brys I. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal . [Internet] [Thesis]. Universidade do Rio Grande do Norte; 2014. [cited 2021 Apr 16]. Available from: http://repositorio.ufrn.br/handle/123456789/19589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brys I. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal . [Thesis]. Universidade do Rio Grande do Norte; 2014. Available from: http://repositorio.ufrn.br/handle/123456789/19589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

3. Logie, Christopher. Presynaptic control of corticostriatal inputs: role of GABA.

Degree: 2014, University of Manchester

 The basal ganglia (BG) are a group of nuclei in the basal forebrain critical in movement, goal directed behaviour and action selection. Cortical projections to… (more)

Subjects/Keywords: striatum; electrophysiology; GABA; corticostriatal; action selection

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APA (6th Edition):

Logie, C. (2014). Presynaptic control of corticostriatal inputs: role of GABA. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231232

Chicago Manual of Style (16th Edition):

Logie, Christopher. “Presynaptic control of corticostriatal inputs: role of GABA.” 2014. Doctoral Dissertation, University of Manchester. Accessed April 16, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231232.

MLA Handbook (7th Edition):

Logie, Christopher. “Presynaptic control of corticostriatal inputs: role of GABA.” 2014. Web. 16 Apr 2021.

Vancouver:

Logie C. Presynaptic control of corticostriatal inputs: role of GABA. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2021 Apr 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231232.

Council of Science Editors:

Logie C. Presynaptic control of corticostriatal inputs: role of GABA. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231232


University of Manchester

4. Logie, Christopher. Presynaptic control of corticostriatal inputs : role of GABA.

Degree: PhD, 2014, University of Manchester

 The basal ganglia (BG) are a group of nuclei in the basal forebrain critical in movement, goal directed behaviour and action selection. Cortical projections to… (more)

Subjects/Keywords: 612.8; striatum; electrophysiology; GABA; corticostriatal; action selection

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APA (6th Edition):

Logie, C. (2014). Presynaptic control of corticostriatal inputs : role of GABA. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/presynaptic-control-of-corticostriatal-inputs-role-of-gaba(380c8d5b-7dc7-4618-a464-8f7104c4db0c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.626924

Chicago Manual of Style (16th Edition):

Logie, Christopher. “Presynaptic control of corticostriatal inputs : role of GABA.” 2014. Doctoral Dissertation, University of Manchester. Accessed April 16, 2021. https://www.research.manchester.ac.uk/portal/en/theses/presynaptic-control-of-corticostriatal-inputs-role-of-gaba(380c8d5b-7dc7-4618-a464-8f7104c4db0c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.626924.

MLA Handbook (7th Edition):

Logie, Christopher. “Presynaptic control of corticostriatal inputs : role of GABA.” 2014. Web. 16 Apr 2021.

Vancouver:

Logie C. Presynaptic control of corticostriatal inputs : role of GABA. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2021 Apr 16]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/presynaptic-control-of-corticostriatal-inputs-role-of-gaba(380c8d5b-7dc7-4618-a464-8f7104c4db0c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.626924.

Council of Science Editors:

Logie C. Presynaptic control of corticostriatal inputs : role of GABA. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/presynaptic-control-of-corticostriatal-inputs-role-of-gaba(380c8d5b-7dc7-4618-a464-8f7104c4db0c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.626924


Vanderbilt University

5. Eckstrand, Kristen Laurel. Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance.

Degree: PhD, Neuroscience, 2013, Vanderbilt University

 Obesity and obesity-associated disease together are a leading cause of death worldwide. Recent research demonstrates striking similarities between obesity and addiction in their dysregulation of… (more)

Subjects/Keywords: corticostriatal; addiction; impulsivity; visceral adiposity; insulin resistance; obesity; dopamine

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APA (6th Edition):

Eckstrand, K. L. (2013). Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14191

Chicago Manual of Style (16th Edition):

Eckstrand, Kristen Laurel. “Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 16, 2021. http://hdl.handle.net/1803/14191.

MLA Handbook (7th Edition):

Eckstrand, Kristen Laurel. “Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance.” 2013. Web. 16 Apr 2021.

Vancouver:

Eckstrand KL. Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1803/14191.

Council of Science Editors:

Eckstrand KL. Corticostriatal dopamine networks mediate impulsivity in obesity and insulin resistance. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14191


University of California – San Francisco

6. Parker, Philip Ross Lutton. Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease.

Degree: Neuroscience, 2015, University of California – San Francisco

 The basal ganglia are an evolutionarily conserved set of nuclei that are crucial for context-dependent learning and selection of appropriate behavioral outputs. The striatum is… (more)

Subjects/Keywords: Neurosciences; basal ganglia; corticostriatal; electrophysiology; Parkinson's disease; synaptic plasticity; thalamostriatal

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APA (6th Edition):

Parker, P. R. L. (2015). Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/9jr78043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Parker, Philip Ross Lutton. “Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease.” 2015. Thesis, University of California – San Francisco. Accessed April 16, 2021. http://www.escholarship.org/uc/item/9jr78043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Parker, Philip Ross Lutton. “Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease.” 2015. Web. 16 Apr 2021.

Vancouver:

Parker PRL. Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2021 Apr 16]. Available from: http://www.escholarship.org/uc/item/9jr78043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Parker PRL. Acute and Chronic Dopaminergic Modulation of Striatal Circuitry and Function in Health and Disease. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/9jr78043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Brys, Ivani. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal.

Degree: 2014, Federal University of Rio Grande do Norte

Conselho Nacional de Desenvolvimento Científico e Tecnológico - CNPq

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior - CAPES

A Doença de Parkinson (DP) está… (more)

Subjects/Keywords: CNPQ::CIENCIAS BIOLOGICAS; Doença de Parkinson; Alfa-sinucleína; circuito corticostriatal

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brys, I. (2014). Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal. (Doctoral Dissertation). Federal University of Rio Grande do Norte. Retrieved from https://repositorio.ufrn.br/jspui/handle/123456789/19589

Chicago Manual of Style (16th Edition):

Brys, Ivani. “Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal.” 2014. Doctoral Dissertation, Federal University of Rio Grande do Norte. Accessed April 16, 2021. https://repositorio.ufrn.br/jspui/handle/123456789/19589.

MLA Handbook (7th Edition):

Brys, Ivani. “Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal.” 2014. Web. 16 Apr 2021.

Vancouver:

Brys I. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal. [Internet] [Doctoral dissertation]. Federal University of Rio Grande do Norte; 2014. [cited 2021 Apr 16]. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/19589.

Council of Science Editors:

Brys I. Modelo animal da Doença de Parkinson baseado na expressão de alfa - sinucleína: caracterização comportamental, eletrofisiológica e avaliação dos efei tos da estimulação da medula espinhal. [Doctoral Dissertation]. Federal University of Rio Grande do Norte; 2014. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/19589


UCLA

8. Groman, Stephanie Mary. The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction.

Degree: Psychology, 2013, UCLA

 Addictions are multi-dimensional disorders, consisting of several behavioral, affective and cognitive dysfunctions that contribute to the compulsive and persistent drug-seeking and taking that is common… (more)

Subjects/Keywords: Neurosciences; Psychobiology; Addiction; Cognitive control; Corticostriatal circuit; D2-like receptor; Dopamine; Feedback sensitivity

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APA (6th Edition):

Groman, S. M. (2013). The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/82w18059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Groman, Stephanie Mary. “The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction.” 2013. Thesis, UCLA. Accessed April 16, 2021. http://www.escholarship.org/uc/item/82w18059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Groman, Stephanie Mary. “The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction.” 2013. Web. 16 Apr 2021.

Vancouver:

Groman SM. The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction. [Internet] [Thesis]. UCLA; 2013. [cited 2021 Apr 16]. Available from: http://www.escholarship.org/uc/item/82w18059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Groman SM. The Dopamine D2-like Receptor: At the Nexus Between Self-Control and Addiction. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/82w18059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

9. Lee, Joanna Chen. Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study.

Degree: PhD, Speech and Hearing Science, 2012, University of Iowa

  The overall aim of the current research was to investigate the corticostriatal system in developmental language impairment (DLI) at the behavioral and neuroanatomical levels.… (more)

Subjects/Keywords: corticostriatal system; Developmental language impairment; procedural learning; reinforcement learning; Speech and Hearing Science

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APA (6th Edition):

Lee, J. C. (2012). Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2927

Chicago Manual of Style (16th Edition):

Lee, Joanna Chen. “Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study.” 2012. Doctoral Dissertation, University of Iowa. Accessed April 16, 2021. https://ir.uiowa.edu/etd/2927.

MLA Handbook (7th Edition):

Lee, Joanna Chen. “Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study.” 2012. Web. 16 Apr 2021.

Vancouver:

Lee JC. Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study. [Internet] [Doctoral dissertation]. University of Iowa; 2012. [cited 2021 Apr 16]. Available from: https://ir.uiowa.edu/etd/2927.

Council of Science Editors:

Lee JC. Are individual differences in language associated with differences in the corticostriatal system? A behavioral and imaging study. [Doctoral Dissertation]. University of Iowa; 2012. Available from: https://ir.uiowa.edu/etd/2927


University of Texas – Austin

10. Valenzuela, Krystal Schaar. Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion.

Degree: PhD, Psychology, 2018, University of Texas – Austin

 The overarching goal of this dissertation project is to test how behavioral experiences with the paretic and non-paretic forelimbs influence recovery of paretic forelimb strength… (more)

Subjects/Keywords: Forelimb strength; MCAo; Axons; Plasticity; Corticostriatal; Rehabilitative training; Non-paretic limb training; Stroke; Rat

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APA (6th Edition):

Valenzuela, K. S. (2018). Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/72811

Chicago Manual of Style (16th Edition):

Valenzuela, Krystal Schaar. “Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion.” 2018. Doctoral Dissertation, University of Texas – Austin. Accessed April 16, 2021. http://hdl.handle.net/2152/72811.

MLA Handbook (7th Edition):

Valenzuela, Krystal Schaar. “Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion.” 2018. Web. 16 Apr 2021.

Vancouver:

Valenzuela KS. Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2018. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2152/72811.

Council of Science Editors:

Valenzuela KS. Behavioral experience effects on forelimb strength recovery and corticostriatal axonal plasticity after experimental middle cerebral artery occlusion. [Doctoral Dissertation]. University of Texas – Austin; 2018. Available from: http://hdl.handle.net/2152/72811


University of Lund

11. Sjöbom, Joel. The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing.

Degree: 2021, University of Lund

 The cortico-basal ganglia system is instrumental in the construction of movement, through its involvement in aspects such as action selection, initiation/termination and sequencing, though the… (more)

Subjects/Keywords: Neurosciences; Neurophysiology; Electrophysiology; Voluntary movements; In-vivo; Motor Cortex; Corticostriatal; Basal Ganglia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sjöbom, J. (2021). The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/2c49e079-eece-4c6a-940d-ad2422c03279 ; https://portal.research.lu.se/ws/files/89713201/Thesis_Joel_Sj_bom_digital_online_version.pdf

Chicago Manual of Style (16th Edition):

Sjöbom, Joel. “The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing.” 2021. Doctoral Dissertation, University of Lund. Accessed April 16, 2021. https://lup.lub.lu.se/record/2c49e079-eece-4c6a-940d-ad2422c03279 ; https://portal.research.lu.se/ws/files/89713201/Thesis_Joel_Sj_bom_digital_online_version.pdf.

MLA Handbook (7th Edition):

Sjöbom, Joel. “The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing.” 2021. Web. 16 Apr 2021.

Vancouver:

Sjöbom J. The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing. [Internet] [Doctoral dissertation]. University of Lund; 2021. [cited 2021 Apr 16]. Available from: https://lup.lub.lu.se/record/2c49e079-eece-4c6a-940d-ad2422c03279 ; https://portal.research.lu.se/ws/files/89713201/Thesis_Joel_Sj_bom_digital_online_version.pdf.

Council of Science Editors:

Sjöbom J. The role of cortico-basal ganglia systems in the construction of movement: Action selection and sequencing. [Doctoral Dissertation]. University of Lund; 2021. Available from: https://lup.lub.lu.se/record/2c49e079-eece-4c6a-940d-ad2422c03279 ; https://portal.research.lu.se/ws/files/89713201/Thesis_Joel_Sj_bom_digital_online_version.pdf


University of Lund

12. Tamté, Martin. The Role of the Cortico-Basal Ganglia-System in Voluntary Movements.

Degree: 2016, University of Lund

 Bodies with multiple limbs and joints have endless possibilities to move around in their surrounding space. How the nervous system controls this amount of degrees… (more)

Subjects/Keywords: Neurosciences; Neurophysiology; Electrophysiology; Pharmacology; Motor Systems; Voluntary movements; Philosophy; In vivo; Corticostriatal; Parkinson's disease; Dyskinesia; Levodopa; Multiple Electrode Arrays

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APA (6th Edition):

Tamté, M. (2016). The Role of the Cortico-Basal Ganglia-System in Voluntary Movements. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/8776791 ; https://portal.research.lu.se/ws/files/3163803/8776834.pdf

Chicago Manual of Style (16th Edition):

Tamté, Martin. “The Role of the Cortico-Basal Ganglia-System in Voluntary Movements.” 2016. Doctoral Dissertation, University of Lund. Accessed April 16, 2021. https://lup.lub.lu.se/record/8776791 ; https://portal.research.lu.se/ws/files/3163803/8776834.pdf.

MLA Handbook (7th Edition):

Tamté, Martin. “The Role of the Cortico-Basal Ganglia-System in Voluntary Movements.” 2016. Web. 16 Apr 2021.

Vancouver:

Tamté M. The Role of the Cortico-Basal Ganglia-System in Voluntary Movements. [Internet] [Doctoral dissertation]. University of Lund; 2016. [cited 2021 Apr 16]. Available from: https://lup.lub.lu.se/record/8776791 ; https://portal.research.lu.se/ws/files/3163803/8776834.pdf.

Council of Science Editors:

Tamté M. The Role of the Cortico-Basal Ganglia-System in Voluntary Movements. [Doctoral Dissertation]. University of Lund; 2016. Available from: https://lup.lub.lu.se/record/8776791 ; https://portal.research.lu.se/ws/files/3163803/8776834.pdf


University of New Mexico

13. Marquardt, Kristin L. CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE.

Degree: Biomedical Sciences Graduate Program, 2017, University of New Mexico

  Up to 61% of adolescent school aged children with fetal alcohol spectrum disorder (FASD) have been suspended or expelled. Executive function deficits, like dis-inhibition… (more)

Subjects/Keywords: in vivo electrophysiology; prenatal alcohol; reversal; orbital frontal cortex; corticostriatal; Behavioral Neurobiology; Cognitive Neuroscience; Medicine and Health Sciences

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APA (6th Edition):

Marquardt, K. L. (2017). CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/168

Chicago Manual of Style (16th Edition):

Marquardt, Kristin L. “CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE.” 2017. Doctoral Dissertation, University of New Mexico. Accessed April 16, 2021. https://digitalrepository.unm.edu/biom_etds/168.

MLA Handbook (7th Edition):

Marquardt, Kristin L. “CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE.” 2017. Web. 16 Apr 2021.

Vancouver:

Marquardt KL. CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE. [Internet] [Doctoral dissertation]. University of New Mexico; 2017. [cited 2021 Apr 16]. Available from: https://digitalrepository.unm.edu/biom_etds/168.

Council of Science Editors:

Marquardt KL. CORTICO-STRIATAL COORDINATION DISRUPTED IN BEHAVIORALLY INFLEXIBLE MODERATE PRENATAL ALCOHOL EXPOSED MICE. [Doctoral Dissertation]. University of New Mexico; 2017. Available from: https://digitalrepository.unm.edu/biom_etds/168


University of Michigan

14. Simen, Patrick A. Neural mechanisms for control in complex cognition.

Degree: PhD, Psychology, 2004, University of Michigan

 Neural network models of complex cognitive tasks are difficult to build. Most previous work has focused on the difficulty of using structured symbolic representations in… (more)

Subjects/Keywords: Cognition; Complex; Control; Corticostriatal Loops; Mechanisms; Neural Networks; Parkinson's Disease; Prefrontal Cortex

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Simen, P. A. (2004). Neural mechanisms for control in complex cognition. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/124182

Chicago Manual of Style (16th Edition):

Simen, Patrick A. “Neural mechanisms for control in complex cognition.” 2004. Doctoral Dissertation, University of Michigan. Accessed April 16, 2021. http://hdl.handle.net/2027.42/124182.

MLA Handbook (7th Edition):

Simen, Patrick A. “Neural mechanisms for control in complex cognition.” 2004. Web. 16 Apr 2021.

Vancouver:

Simen PA. Neural mechanisms for control in complex cognition. [Internet] [Doctoral dissertation]. University of Michigan; 2004. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2027.42/124182.

Council of Science Editors:

Simen PA. Neural mechanisms for control in complex cognition. [Doctoral Dissertation]. University of Michigan; 2004. Available from: http://hdl.handle.net/2027.42/124182

15. Baker, Phillip M. Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching.

Degree: 2013, University of Illinois – Chicago

 Frontal cortex- basal ganglia circuitry supports behavioral switching when a change in outcome information is used to shift strategies. Less is known about whether specific… (more)

Subjects/Keywords: hyperdirect pathway; prefrontal cortex; corticostriatal; disconnection; cognitive flexibility; proactive switching

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baker, P. M. (2013). Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baker, Phillip M. “Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching.” 2013. Thesis, University of Illinois – Chicago. Accessed April 16, 2021. http://hdl.handle.net/10027/9977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baker, Phillip M. “Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching.” 2013. Web. 16 Apr 2021.

Vancouver:

Baker PM. Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/10027/9977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baker PM. Contributions of the Prelimbic Cortex and Basal Ganglia Circuitry to Proactive Behavioral Switching. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

16. Akrong, James. Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat.

Degree: PhD, 2009, McMaster University

Long-term potentiation (LTP) and depression (LTD) are activity dependent long-lasting changes in synaptic efficacy and have been proposed as mechanisms for learning and memory. Although… (more)

Subjects/Keywords: long term potentiation; long term depression; learning and memory; neural plasticity; basal ganglia motor loop; corticostriatal pathway; thalamocortical

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Akrong, J. (2009). Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/17238

Chicago Manual of Style (16th Edition):

Akrong, James. “Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat.” 2009. Doctoral Dissertation, McMaster University. Accessed April 16, 2021. http://hdl.handle.net/11375/17238.

MLA Handbook (7th Edition):

Akrong, James. “Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat.” 2009. Web. 16 Apr 2021.

Vancouver:

Akrong J. Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat. [Internet] [Doctoral dissertation]. McMaster University; 2009. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/11375/17238.

Council of Science Editors:

Akrong J. Long-Term Potentiation and Long-Term Depression in the Corticostriatal Motor System of the Non-Anesthetized Rat. [Doctoral Dissertation]. McMaster University; 2009. Available from: http://hdl.handle.net/11375/17238


Université de Montréal

17. Maheux, Jérôme. Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques.

Degree: 2012, Université de Montréal

Subjects/Keywords: Schizophrénie; antipsychotique; striatum; dopamine; sérotonine; D2; mGluR5; A2A; 5-HT2A; 5-HT1A; voie corticostriée; Schizophrenia; Antipsychotic drugs; serotonin; corticostriatal pathway; Health Sciences - Pharmacology / Sciences de la santé - Pharmacologie (UMI : 0419)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maheux, J. (2012). Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/8559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maheux, Jérôme. “Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques.” 2012. Thesis, Université de Montréal. Accessed April 16, 2021. http://hdl.handle.net/1866/8559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maheux, Jérôme. “Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques.” 2012. Web. 16 Apr 2021.

Vancouver:

Maheux J. Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques. [Internet] [Thesis]. Université de Montréal; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1866/8559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maheux J. Mécanismes cellulaires de l'induction du facteur de transcription Nur77 après un traitement aux antipsychotiques. [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/8559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.