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You searched for subject:(cofilin). Showing records 1 – 30 of 53 total matches.

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University of Illinois – Urbana-Champaign

1. Nadkarni, Ambika Vithal. A biochemical investigation of actin disassembly mechanisms.

Degree: PhD, Cell and Developmental Biology, 2016, University of Illinois – Urbana-Champaign

 The dynamic nature of the actin cytoskeleton enables the rapid shape changes that are necessary for processes such as wound healing, motility and division of… (more)

Subjects/Keywords: Actin depolymerization; cofilin; Aip1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nadkarni, A. V. (2016). A biochemical investigation of actin disassembly mechanisms. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/92945

Chicago Manual of Style (16th Edition):

Nadkarni, Ambika Vithal. “A biochemical investigation of actin disassembly mechanisms.” 2016. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed August 08, 2020. http://hdl.handle.net/2142/92945.

MLA Handbook (7th Edition):

Nadkarni, Ambika Vithal. “A biochemical investigation of actin disassembly mechanisms.” 2016. Web. 08 Aug 2020.

Vancouver:

Nadkarni AV. A biochemical investigation of actin disassembly mechanisms. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/2142/92945.

Council of Science Editors:

Nadkarni AV. A biochemical investigation of actin disassembly mechanisms. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2016. Available from: http://hdl.handle.net/2142/92945


Penn State University

2. Cichon, Joseph Michael. Cofilin Rods And Neurodegeneration.

Degree: MS, Biology, 2009, Penn State University

 Early stages of neurodegeneration are characterized by synaptic loss and slow cognitive decline. Cofilin/actin rods (rods) have been implicated in neurodegenerative disorders, but the precise… (more)

Subjects/Keywords: cofilin; actin; LIM kinase; cytoskeleton; synapse

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APA (6th Edition):

Cichon, J. M. (2009). Cofilin Rods And Neurodegeneration. (Masters Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/9648

Chicago Manual of Style (16th Edition):

Cichon, Joseph Michael. “Cofilin Rods And Neurodegeneration.” 2009. Masters Thesis, Penn State University. Accessed August 08, 2020. https://etda.libraries.psu.edu/catalog/9648.

MLA Handbook (7th Edition):

Cichon, Joseph Michael. “Cofilin Rods And Neurodegeneration.” 2009. Web. 08 Aug 2020.

Vancouver:

Cichon JM. Cofilin Rods And Neurodegeneration. [Internet] [Masters thesis]. Penn State University; 2009. [cited 2020 Aug 08]. Available from: https://etda.libraries.psu.edu/catalog/9648.

Council of Science Editors:

Cichon JM. Cofilin Rods And Neurodegeneration. [Masters Thesis]. Penn State University; 2009. Available from: https://etda.libraries.psu.edu/catalog/9648


University of Toronto

3. Bent, Russell. Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics.

Degree: 2011, University of Toronto

Transient Receptor Potential ‘Melastatin’ 7 (TRPM7) is a ubiquitously expressed, non-selective divalent cation channel implicated in diverse cellular functions including actomyosin cytoskeletal remodeling, magnesium homeostasis,… (more)

Subjects/Keywords: biology; stroke; TRPM7; cytoskeleton; cofilin; neuron; 0317

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APA (6th Edition):

Bent, R. (2011). Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30181

Chicago Manual of Style (16th Edition):

Bent, Russell. “Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics.” 2011. Masters Thesis, University of Toronto. Accessed August 08, 2020. http://hdl.handle.net/1807/30181.

MLA Handbook (7th Edition):

Bent, Russell. “Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics.” 2011. Web. 08 Aug 2020.

Vancouver:

Bent R. Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1807/30181.

Council of Science Editors:

Bent R. Transient Receptor Potential Melastatin 7 Channels Regulate Neuronal Cytoskeletal Dynamics. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30181


University of Toledo Health Science Campus

4. Madineni, Anusha. Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke.

Degree: MSP, College of Pharmacy, 2013, University of Toledo Health Science Campus

 Cerebral ischemia or stroke is a condition associated with decreased blood supply to brain leading to death of neurons. It is associated with a diverse… (more)

Subjects/Keywords: Cellular Biology; Neurosciences; Pharmacology; Pharmaceuticals; Cofilin; Neurodegeneration; Cerebral ischemia

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APA (6th Edition):

Madineni, A. (2013). Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1364331841

Chicago Manual of Style (16th Edition):

Madineni, Anusha. “Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke.” 2013. Masters Thesis, University of Toledo Health Science Campus. Accessed August 08, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1364331841.

MLA Handbook (7th Edition):

Madineni, Anusha. “Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke.” 2013. Web. 08 Aug 2020.

Vancouver:

Madineni A. Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2013. [cited 2020 Aug 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1364331841.

Council of Science Editors:

Madineni A. Role of Cofilin, an Actin Cytoskeletal Protein, in Ischemic Conditions: Potential Therapeutic Target for Ischemic Stroke. [Masters Thesis]. University of Toledo Health Science Campus; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1364331841

5. 田中, 健之. アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis.

Degree: Chiba University / 千葉大学

研究科: 千葉大学大学院自然科学研究科

千大院自博甲第理253号

修了年: 2005

Advisors/Committee Members: 千葉大学大学院自然科学研究科.

Subjects/Keywords: Slingshot; Cofilin; XAC; Actin

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APA (6th Edition):

田中, . (n.d.). アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis. (Thesis). Chiba University / 千葉大学. Retrieved from http://opac.ll.chiba-u.jp/da/curator/900047513/

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

田中, 健之. “アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis.” Thesis, Chiba University / 千葉大学. Accessed August 08, 2020. http://opac.ll.chiba-u.jp/da/curator/900047513/.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

田中, 健之. “アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis.” Web. 08 Aug 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

田中 . アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis. [Internet] [Thesis]. Chiba University / 千葉大学; [cited 2020 Aug 08]. Available from: http://opac.ll.chiba-u.jp/da/curator/900047513/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

田中 . アフリカツメガエル初期発生におけるSlingshotフォスファターゼの機能に関する研究 : Studies on function of Slingshot phosphatase during early development of Xenopus laevis. [Thesis]. Chiba University / 千葉大学; Available from: http://opac.ll.chiba-u.jp/da/curator/900047513/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Cambridge

6. Jones, Christopher. Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin.

Degree: PhD, University of Cambridge

 The actin cytoskeleton is a complex and dynamic network of protein filaments involved in a great number of cellular processes essential for cellular homeostasis and… (more)

Subjects/Keywords: Shigella; cofilin; PAK; Salmonella; Actin

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APA (6th Edition):

Jones, C. (n.d.). Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/307706

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Jones, Christopher. “Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin.” Doctoral Dissertation, University of Cambridge. Accessed August 08, 2020. https://www.repository.cam.ac.uk/handle/1810/307706.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Jones, Christopher. “Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin.” Web. 08 Aug 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Jones C. Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin. [Internet] [Doctoral dissertation]. University of Cambridge; [cited 2020 Aug 08]. Available from: https://www.repository.cam.ac.uk/handle/1810/307706.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Jones C. Bacterial Manipulation of Actin Dynamics via p21 Activated Kinases and Cofilin. [Doctoral Dissertation]. University of Cambridge; Available from: https://www.repository.cam.ac.uk/handle/1810/307706

Note: this citation may be lacking information needed for this citation format:
No year of publication.

7. E. Giardino. IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA.

Degree: 2015, Università degli Studi di Milano

 Non-functioning pituitary adenomas (NFPAs) are benign in nature, frequently present local invasiveness that strongly reduces neurosurgery success. Medical therapy is still under debate, although evidences… (more)

Subjects/Keywords: NFPA; invasion; DRD2; DRD2 agonist: cofilin; Settore MED/13 - Endocrinologia

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APA (6th Edition):

Giardino, E. (2015). IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/341158

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Giardino, E.. “IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA.” 2015. Thesis, Università degli Studi di Milano. Accessed August 08, 2020. http://hdl.handle.net/2434/341158.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Giardino, E.. “IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA.” 2015. Web. 08 Aug 2020.

Vancouver:

Giardino E. IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/2434/341158.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Giardino E. IL RECETTORE DELLA DOPAMINA DI TIPO 2 (DRD2) INIBISCE LA MIGRAZIONE DI CELLULE DI ADENOMA IPOFISARIO NON SECERNENTE TRAMITE INATTIVAZIONE DI COFILINA. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/341158

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

8. Santiago, Joanne Collazo. COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS.

Degree: 2013, University of Kentucky

 Cofilin’s activity to nucleate actin filament assembly, is regulated by phosphorylation at a single site on the amino terminus, Serine 3. Phosphorylation at this site… (more)

Subjects/Keywords: Prostate Cancer; Cofilin; Actin Cytoskeleton; Filopodia; Metastasis; Medical Sciences

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APA (6th Edition):

Santiago, J. C. (2013). COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/7

Chicago Manual of Style (16th Edition):

Santiago, Joanne Collazo. “COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS.” 2013. Doctoral Dissertation, University of Kentucky. Accessed August 08, 2020. https://uknowledge.uky.edu/toxicology_etds/7.

MLA Handbook (7th Edition):

Santiago, Joanne Collazo. “COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS.” 2013. Web. 08 Aug 2020.

Vancouver:

Santiago JC. COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS. [Internet] [Doctoral dissertation]. University of Kentucky; 2013. [cited 2020 Aug 08]. Available from: https://uknowledge.uky.edu/toxicology_etds/7.

Council of Science Editors:

Santiago JC. COFILIN NAVIGATES CELLULAR CYTOSKELETON AND INVASION RESPONSES TO TGF-β TOWARDS PROSTATE CANCER METASTASIS. [Doctoral Dissertation]. University of Kentucky; 2013. Available from: https://uknowledge.uky.edu/toxicology_etds/7


George Mason University

9. Liang, Huizhi. Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells .

Degree: 2015, George Mason University

 The M-tropic HIV-1 viruses use CCR5 as the co-receptor for entry. The V3 loop in the M-tropic envelope protein, gp120, is largely responsible for the… (more)

Subjects/Keywords: HIV-1; mutagenesis; signal transduction; GP120; cofilin; V3 loop

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APA (6th Edition):

Liang, H. (2015). Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells . (Thesis). George Mason University. Retrieved from http://hdl.handle.net/1920/9774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liang, Huizhi. “Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells .” 2015. Thesis, George Mason University. Accessed August 08, 2020. http://hdl.handle.net/1920/9774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liang, Huizhi. “Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells .” 2015. Web. 08 Aug 2020.

Vancouver:

Liang H. Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells . [Internet] [Thesis]. George Mason University; 2015. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1920/9774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liang H. Mutagenesis of HIV-1 GP120 V3 Loop to Determine Effects on GP120-Mediated CCR5 and CXCR4 Signal Transduction in Resting CD4 T Cells . [Thesis]. George Mason University; 2015. Available from: http://hdl.handle.net/1920/9774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Kanellos, Georgios. Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells.

Degree: PhD, 2017, University of Edinburgh

 Actin cytoskeletal regulation is of critical importance for a number of diverse cellular functions, including cell motility, endocytosis, cell division and transcription. Tight regulation of… (more)

Subjects/Keywords: 572; actin; adf; cofilin; nuclear envelope; LINC complex; tissue homeostasis

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APA (6th Edition):

Kanellos, G. (2017). Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25411

Chicago Manual of Style (16th Edition):

Kanellos, Georgios. “Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed August 08, 2020. http://hdl.handle.net/1842/25411.

MLA Handbook (7th Edition):

Kanellos, Georgios. “Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells.” 2017. Web. 08 Aug 2020.

Vancouver:

Kanellos G. Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1842/25411.

Council of Science Editors:

Kanellos G. Novel roles of ADF/cofilins in maintenance of homeostasis in normal and malignant epithelial cells. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/25411

11. Angelou, Eleni. Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο.

Degree: 2019, University of Patras; Πανεπιστήμιο Πατρών

 Understanding the molecular mechanisms underlying colorectal cancer (CRC) progression is of great importance since invasion and metastasis is the major cause of CRC related morbidity… (more)

Subjects/Keywords: Καρκίνος παχέος εντέρου; Κυτταροσκελετός ακτίνης; Ρυθμιστές κυτταροσκελετου ακτίνης; Χημειοαντίσταση; Επιθήλιο - μεσεγχυματική μετατροπή; EMT; Colon cancer; Chemoresistance; Limk /cofilin; Slingshot; Limk1; Limk2; Cofilin

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APA (6th Edition):

Angelou, E. (2019). Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/45276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Angelou, Eleni. “Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο.” 2019. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed August 08, 2020. http://hdl.handle.net/10442/hedi/45276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Angelou, Eleni. “Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο.” 2019. Web. 08 Aug 2020.

Vancouver:

Angelou E. Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10442/hedi/45276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Angelou E. Ρυθμιστές της δυναμικής του κυτταροσκελετού ακτίνης και επιθήλιο - μεσεγχυματική μετατροπή (ΕΜΤ) στον καρκίνο του παχέος εντέρου στον άνθρωπο. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. Available from: http://hdl.handle.net/10442/hedi/45276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

12. Mikati, Mouna. Modulation of Actin Structure and Dynamics by Actin Binding Proteins.

Degree: Biochemistry & Molecular Biology, 2014, UCLA

 Rapid remodeling of the actin cytoskeleton is essential for many cellular processes including cell growth, differentiation, division and motility. The structure and dynamics of the… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Actin Binding Proteins; Actin Cytoskeleton; Cofilin; Coronin; Drebrin; Protein Interactions

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APA (6th Edition):

Mikati, M. (2014). Modulation of Actin Structure and Dynamics by Actin Binding Proteins. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/58r9c2hr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mikati, Mouna. “Modulation of Actin Structure and Dynamics by Actin Binding Proteins.” 2014. Thesis, UCLA. Accessed August 08, 2020. http://www.escholarship.org/uc/item/58r9c2hr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mikati, Mouna. “Modulation of Actin Structure and Dynamics by Actin Binding Proteins.” 2014. Web. 08 Aug 2020.

Vancouver:

Mikati M. Modulation of Actin Structure and Dynamics by Actin Binding Proteins. [Internet] [Thesis]. UCLA; 2014. [cited 2020 Aug 08]. Available from: http://www.escholarship.org/uc/item/58r9c2hr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mikati M. Modulation of Actin Structure and Dynamics by Actin Binding Proteins. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/58r9c2hr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

13. Brown, Joel. INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS .

Degree: 2018, Cornell University

 Forward genetics allows the identification of novel genes involved in a particular biological process. We performed an ENU forward mutagenesis screen in mice aimed at… (more)

Subjects/Keywords: calcium; Actomyosin; Apical Constriction; Cofilin 1; Neural Tube; SPCA1; Developmental biology; Genetics; Molecular biology

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APA (6th Edition):

Brown, J. (2018). INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/59291

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brown, Joel. “INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS .” 2018. Thesis, Cornell University. Accessed August 08, 2020. http://hdl.handle.net/1813/59291.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brown, Joel. “INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS .” 2018. Web. 08 Aug 2020.

Vancouver:

Brown J. INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS . [Internet] [Thesis]. Cornell University; 2018. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1813/59291.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brown J. INVESTIGATING MAMMALIAN DEVELOPMENT USING MOUSE FORWARD GENETICS . [Thesis]. Cornell University; 2018. Available from: http://hdl.handle.net/1813/59291

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Connecticut

14. Nagarajan, Neerajha. A Depolymerization Based Model of Neuron Growth Cone Shape and Motility.

Degree: MS, Biomedical Engineering, 2012, University of Connecticut

  Cell migration is a ubiquitous process underlying critical biological mechanisms like wound healing, cancer metastasis and even neuron growth cone motility. It is a… (more)

Subjects/Keywords: Cell Motilty; Neurons; Neuron Growth Cones; Actin Filaments; Cofilin; Actin Depolymerization; Quantitative Model

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APA (6th Edition):

Nagarajan, N. (2012). A Depolymerization Based Model of Neuron Growth Cone Shape and Motility. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/363

Chicago Manual of Style (16th Edition):

Nagarajan, Neerajha. “A Depolymerization Based Model of Neuron Growth Cone Shape and Motility.” 2012. Masters Thesis, University of Connecticut. Accessed August 08, 2020. https://opencommons.uconn.edu/gs_theses/363.

MLA Handbook (7th Edition):

Nagarajan, Neerajha. “A Depolymerization Based Model of Neuron Growth Cone Shape and Motility.” 2012. Web. 08 Aug 2020.

Vancouver:

Nagarajan N. A Depolymerization Based Model of Neuron Growth Cone Shape and Motility. [Internet] [Masters thesis]. University of Connecticut; 2012. [cited 2020 Aug 08]. Available from: https://opencommons.uconn.edu/gs_theses/363.

Council of Science Editors:

Nagarajan N. A Depolymerization Based Model of Neuron Growth Cone Shape and Motility. [Masters Thesis]. University of Connecticut; 2012. Available from: https://opencommons.uconn.edu/gs_theses/363

15. Chatzifrangkeskou, Maria. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.

Degree: Docteur es, Biologie, 2016, Université Pierre et Marie Curie – Paris VI

La cardiomyopathie dilatée est l'une des principales causes d'insuffisance cardiaque en Europe. Dans le cadre de la cardiomyopathie liée aux mutations du gène LMNA, en… (more)

Subjects/Keywords: Cardiomyopathie dilatée; LMNA; Cofiline; Actine; ERK 1/2; TGF-β; Dilated cardiomyopathy; LMNA; Cofilin; 573.1

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APA (6th Edition):

Chatzifrangkeskou, M. (2016). Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066380

Chicago Manual of Style (16th Edition):

Chatzifrangkeskou, Maria. “Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed August 08, 2020. http://www.theses.fr/2016PA066380.

MLA Handbook (7th Edition):

Chatzifrangkeskou, Maria. “Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.” 2016. Web. 08 Aug 2020.

Vancouver:

Chatzifrangkeskou M. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2020 Aug 08]. Available from: http://www.theses.fr/2016PA066380.

Council of Science Editors:

Chatzifrangkeskou M. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066380

16. CHUA BOON TIN. Role of calpain and cofilin in apoptosis regulation.

Degree: 2004, National University of Singapore

Subjects/Keywords: calpain; caspase; mitochondria; cofilin; translocation; phosphorylation

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APA (6th Edition):

TIN, C. B. (2004). Role of calpain and cofilin in apoptosis regulation. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/13991

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TIN, CHUA BOON. “Role of calpain and cofilin in apoptosis regulation.” 2004. Thesis, National University of Singapore. Accessed August 08, 2020. http://scholarbank.nus.edu.sg/handle/10635/13991.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TIN, CHUA BOON. “Role of calpain and cofilin in apoptosis regulation.” 2004. Web. 08 Aug 2020.

Vancouver:

TIN CB. Role of calpain and cofilin in apoptosis regulation. [Internet] [Thesis]. National University of Singapore; 2004. [cited 2020 Aug 08]. Available from: http://scholarbank.nus.edu.sg/handle/10635/13991.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TIN CB. Role of calpain and cofilin in apoptosis regulation. [Thesis]. National University of Singapore; 2004. Available from: http://scholarbank.nus.edu.sg/handle/10635/13991

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. LEE CHENG JIE, IRENE. The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics.

Degree: 2010, National University of Singapore

Subjects/Keywords: MRCK; LIMK1; LRAP25; Cofilin; lamellipodia; AlF

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APA (6th Edition):

LEE CHENG JIE, I. (2010). The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/22624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LEE CHENG JIE, IRENE. “The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics.” 2010. Thesis, National University of Singapore. Accessed August 08, 2020. http://scholarbank.nus.edu.sg/handle/10635/22624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LEE CHENG JIE, IRENE. “The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics.” 2010. Web. 08 Aug 2020.

Vancouver:

LEE CHENG JIE I. The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics. [Internet] [Thesis]. National University of Singapore; 2010. [cited 2020 Aug 08]. Available from: http://scholarbank.nus.edu.sg/handle/10635/22624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LEE CHENG JIE I. The role of Leucine Repeat Adaptor Protein 25(LRAP25), a novel interactor of Myotonic dystrophy kinase-related Cdc42-binding kinase (MRCK) in mediating lamellipodial F-actin dynamics. [Thesis]. National University of Singapore; 2010. Available from: http://scholarbank.nus.edu.sg/handle/10635/22624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Vanderpool, Nicole Danielle. Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P.

Degree: MS, Biochemistry, 2012, University of Iowa

  More than 30 missense mutations in the ACTA2 gene, which encodes α–smooth muscle (α–SM) actin, cause thoracic aortic aneurysms and dissections (TAAD). Aortic cell… (more)

Subjects/Keywords: Actin; Aip1p; Cofilin; TAAD; Biochemistry

…11 Yeast Cofilin Expression and Purification… …15 Cofilin Titration - F-actin… …16 Aip1p Enhancement of Cofilin-Mediated Severing - F-actin .............................16… …Effect of the R256H Mutation on the Actin-Cofilin Interaction.........................22 Effect… …Cofilin Activity .............................................................23 CHAPTER IV… 

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APA (6th Edition):

Vanderpool, N. D. (2012). Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/3397

Chicago Manual of Style (16th Edition):

Vanderpool, Nicole Danielle. “Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P.” 2012. Masters Thesis, University of Iowa. Accessed August 08, 2020. https://ir.uiowa.edu/etd/3397.

MLA Handbook (7th Edition):

Vanderpool, Nicole Danielle. “Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P.” 2012. Web. 08 Aug 2020.

Vancouver:

Vanderpool ND. Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P. [Internet] [Masters thesis]. University of Iowa; 2012. [cited 2020 Aug 08]. Available from: https://ir.uiowa.edu/etd/3397.

Council of Science Editors:

Vanderpool ND. Thoracic aortic aneurysm (TAAD)-causing mutation in actin alters regulation by cofilin and AIP1P. [Masters Thesis]. University of Iowa; 2012. Available from: https://ir.uiowa.edu/etd/3397


George Mason University

19. Yi, Fei. The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection .

Degree: 2016, George Mason University

 A dynamic actin cytoskeleton is necessary for viral entry, intracellular migration, and virion release. For HIV-1 infection, during viral entry, the virus triggers early actin… (more)

Subjects/Keywords: Virology; Molecular biology; Microbiology; actin; antiviral drug; cofilin; HIV; kinase inhibitor; LIMK

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APA (6th Edition):

Yi, F. (2016). The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection . (Thesis). George Mason University. Retrieved from http://hdl.handle.net/1920/10450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yi, Fei. “The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection .” 2016. Thesis, George Mason University. Accessed August 08, 2020. http://hdl.handle.net/1920/10450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yi, Fei. “The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection .” 2016. Web. 08 Aug 2020.

Vancouver:

Yi F. The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection . [Internet] [Thesis]. George Mason University; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1920/10450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yi F. The Roles of LIM Kinase (LIMK) Inhibitors in the Regulation of Actin Dynamics During HIV-1 Infection . [Thesis]. George Mason University; 2016. Available from: http://hdl.handle.net/1920/10450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Grenoble

20. Suarez, Cristian. ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility.

Degree: Docteur es, Physique pour les sciences du vivant, 2011, Université de Grenoble

Durant mon travail de thèse, j'ai étudié le rôle central de l'ADF/cofiline, une protéine qui se lie au cytosquelette d'actine, décore spécifiquement les parties ‘âgées'… (more)

Subjects/Keywords: Actine; Cytosquelette; ADF/cofiline; Motilité cellulaire; Actin; Cytoskeleton; ADF/cofilin; Cell motility

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APA (6th Edition):

Suarez, C. (2011). ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2011GRENY033

Chicago Manual of Style (16th Edition):

Suarez, Cristian. “ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility.” 2011. Doctoral Dissertation, Université de Grenoble. Accessed August 08, 2020. http://www.theses.fr/2011GRENY033.

MLA Handbook (7th Edition):

Suarez, Cristian. “ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility.” 2011. Web. 08 Aug 2020.

Vancouver:

Suarez C. ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility. [Internet] [Doctoral dissertation]. Université de Grenoble; 2011. [cited 2020 Aug 08]. Available from: http://www.theses.fr/2011GRENY033.

Council of Science Editors:

Suarez C. ADF/cofiline, un facteur essentiel dans le contrôle de la dynamique de l'actine au cours de la motilité cellulaire : ADF/cofiline, an essential factor that controls actin dynamics during cell motility. [Doctoral Dissertation]. Université de Grenoble; 2011. Available from: http://www.theses.fr/2011GRENY033


University of Southern California

21. Frendo, Michele. The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration.

Degree: PhD, Neuroscience, 2014, University of Southern California

 A well‐known challenge for regenerating axon circuits after injury or disease is the inhibitory environment of the central nervous system (CNS). A largely ignored but… (more)

Subjects/Keywords: cofilin; Limk1; development; motor neurons; regeneration; peripheral nerve injury; peripheral nervous system

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APA (6th Edition):

Frendo, M. (2014). The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/430657/rec/7268

Chicago Manual of Style (16th Edition):

Frendo, Michele. “The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration.” 2014. Doctoral Dissertation, University of Southern California. Accessed August 08, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/430657/rec/7268.

MLA Handbook (7th Edition):

Frendo, Michele. “The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration.” 2014. Web. 08 Aug 2020.

Vancouver:

Frendo M. The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Aug 08]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/430657/rec/7268.

Council of Science Editors:

Frendo M. The role of the cofilin/Limk1 signaling pathway in axon growth during development and regeneration. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/430657/rec/7268


University of Southern California

22. Park, Ko Uoon. The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution.

Degree: MS, Molecular Microbiology & Immunology, 2010, University of Southern California

 The hepatitis C virus (HCV) is a leading causative agent associated with chronic liver disease. The core protein of HCV associates with lipid droplets (LDs)… (more)

Subjects/Keywords: HCV core protein; cofilin; lipid droplet

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APA (6th Edition):

Park, K. U. (2010). The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/429261/rec/6600

Chicago Manual of Style (16th Edition):

Park, Ko Uoon. “The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution.” 2010. Masters Thesis, University of Southern California. Accessed August 08, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/429261/rec/6600.

MLA Handbook (7th Edition):

Park, Ko Uoon. “The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution.” 2010. Web. 08 Aug 2020.

Vancouver:

Park KU. The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2020 Aug 08]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/429261/rec/6600.

Council of Science Editors:

Park KU. The effect of cofilin on HCV core protein association with lipid droplet and on lipid droplet redistribution. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/429261/rec/6600


University of Southern California

23. Yamauchi, Ken. The role of bone morphogenetic protein receptors in commissural axon guidance and growth.

Degree: PhD, Neuroscience, 2012, University of Southern California

 A correctly functioning nervous system requires that neural circuits be precisely wired during development. A growing axon must travel through a constantly changing environment, bypassing… (more)

Subjects/Keywords: commissural; neuron; axon; guidance; growth; Bone Morphogenetic Protein; BMP; cofilin; development; spinal cord; vertebrate; embryonic

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APA (6th Edition):

Yamauchi, K. (2012). The role of bone morphogenetic protein receptors in commissural axon guidance and growth. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7202

Chicago Manual of Style (16th Edition):

Yamauchi, Ken. “The role of bone morphogenetic protein receptors in commissural axon guidance and growth.” 2012. Doctoral Dissertation, University of Southern California. Accessed August 08, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7202.

MLA Handbook (7th Edition):

Yamauchi, Ken. “The role of bone morphogenetic protein receptors in commissural axon guidance and growth.” 2012. Web. 08 Aug 2020.

Vancouver:

Yamauchi K. The role of bone morphogenetic protein receptors in commissural axon guidance and growth. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Aug 08]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7202.

Council of Science Editors:

Yamauchi K. The role of bone morphogenetic protein receptors in commissural axon guidance and growth. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7202

24. Burat, Bastien. Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton.

Degree: Docteur es, Pharmacologie et science du médicament, 2017, Limoges

En transplantation d’organe solide, les Inhibiteurs de la Calcineurine (ICN), Cyclosporine A et Tacrolimus, ont permis un amélioration significative de la survie à court terme… (more)

Subjects/Keywords: Cellules tubulaires proximales; ITRAQ; CiR-C; Cofiline; Tubular proximal cells; ITRAQ; CiR-C; Cofilin; 572.6; 617.95

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APA (6th Edition):

Burat, B. (2017). Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2017LIMO0104

Chicago Manual of Style (16th Edition):

Burat, Bastien. “Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton.” 2017. Doctoral Dissertation, Limoges. Accessed August 08, 2020. http://www.theses.fr/2017LIMO0104.

MLA Handbook (7th Edition):

Burat, Bastien. “Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton.” 2017. Web. 08 Aug 2020.

Vancouver:

Burat B. Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton. [Internet] [Doctoral dissertation]. Limoges; 2017. [cited 2020 Aug 08]. Available from: http://www.theses.fr/2017LIMO0104.

Council of Science Editors:

Burat B. Apports de la protéomique quantitative différentielle haut-débit à l'étude des mécanismes de modification du cytosquelette de cellules tubulaires proximales induits par les Inhibiteurs de la Calcineurine : Contributions of the differential high-throughput quantitative proteomic analysis of tubular proximal cells to the study of Calcineurin Inhibitors-induced modifications of the Actin cytoskeleton. [Doctoral Dissertation]. Limoges; 2017. Available from: http://www.theses.fr/2017LIMO0104

25. Τσινιάς, Γεώργιος Ι. Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα.

Degree: 2014, University of Patras

Η αναδιοργάνωση του κυτταροσκελετού ακτίνης έχει κρίσιμο ρόλο στη διήθηση και τη μετάσταση των καρκινικών κυττάρων. Οι πρωτεΐνες κοφιλίνη και N-WASP συνδέονται με την ακτίνη… (more)

Subjects/Keywords: Κοφιλίνη; β-Παρβίνη; Καρκίνος του λάρυγγα; Ανοσοϊστοχημεία; 616.994 207; Cofilin; b-Parvin; Laryngeal cancer; Immunohistochemistry; N-WASP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Τσινιάς, . . (2014). Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/8190

Chicago Manual of Style (16th Edition):

Τσινιάς, Γεώργιος Ι. “Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα.” 2014. Masters Thesis, University of Patras. Accessed August 08, 2020. http://hdl.handle.net/10889/8190.

MLA Handbook (7th Edition):

Τσινιάς, Γεώργιος Ι. “Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα.” 2014. Web. 08 Aug 2020.

Vancouver:

Τσινιάς . Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα. [Internet] [Masters thesis]. University of Patras; 2014. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10889/8190.

Council of Science Editors:

Τσινιάς . Ο ρόλος πρωτεϊνών που αλληλεπιδρούν με τον κυτταροσκελετό ακτίνης στην παθογένεια και πρόγνωση του καρκίνου του λάρυγγα. [Masters Thesis]. University of Patras; 2014. Available from: http://hdl.handle.net/10889/8190


Jawaharlal Nehru University

26. Satish Tammana T V. Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani.

Degree: Molecular biology, 2009, Jawaharlal Nehru University

None

Bibliography p. 111-126, Publication included

Advisors/Committee Members: Gupta, C M.

Subjects/Keywords: Molecular biology; Leishmania donovani; ADF/cofilin homologue

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

V, S. T. T. (2009). Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/13551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

V, Satish Tammana T. “Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani.” 2009. Thesis, Jawaharlal Nehru University. Accessed August 08, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/13551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

V, Satish Tammana T. “Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani.” 2009. Web. 08 Aug 2020.

Vancouver:

V STT. Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani. [Internet] [Thesis]. Jawaharlal Nehru University; 2009. [cited 2020 Aug 08]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/13551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

V STT. Functional and structural characterization of ADF/cofilin homologue from Leishmania donovani. [Thesis]. Jawaharlal Nehru University; 2009. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/13551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

27. Barros, Rafael Longhi Sampaio de. Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células.

Degree: 2010, Universidade do Rio Grande do Sul

Câncer de Pulmão de Não-pequenas células (CPNPC) é o maior problema de saúde pública atualmente no mundo.Encontramos em nossas pesquisas que os níveis de mRNA… (more)

Subjects/Keywords: Prognosis; Neoplasias pulmonares; Carcinoma pulmonar de células não pequenas; Biomarker; Prognóstico; Nonsmall cell lung cancer; Cofilin; Biomarcadores tumorais; Cofilina 1; Immunohistochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barros, R. L. S. d. (2010). Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/26938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barros, Rafael Longhi Sampaio de. “Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed August 08, 2020. http://hdl.handle.net/10183/26938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barros, Rafael Longhi Sampaio de. “Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células.” 2010. Web. 08 Aug 2020.

Vancouver:

Barros RLSd. Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10183/26938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barros RLSd. Validação do uso da proteína cofilina como biomarcador preditivo do prognóstico de carcinoma de pulmão de não-pequenas células. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/26938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

28. Low, Brian. A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat.

Degree: MS, Physiology, 2009, Virginia Commonwealth University

 Traumatic brain injury is a leading cause of death and disability in the United States. The injury is often composed of two processes: the primary… (more)

Subjects/Keywords: lateral fluid percussion injury; calcineurin; cofilin; Life Sciences; Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Low, B. (2009). A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/Y1XG-DB87 ; https://scholarscompass.vcu.edu/etd/1892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Low, Brian. “A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat.” 2009. Thesis, Virginia Commonwealth University. Accessed August 08, 2020. https://doi.org/10.25772/Y1XG-DB87 ; https://scholarscompass.vcu.edu/etd/1892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Low, Brian. “A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat.” 2009. Web. 08 Aug 2020.

Vancouver:

Low B. A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat. [Internet] [Thesis]. Virginia Commonwealth University; 2009. [cited 2020 Aug 08]. Available from: https://doi.org/10.25772/Y1XG-DB87 ; https://scholarscompass.vcu.edu/etd/1892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Low B. A Cellular Mechanism for Dendritic Spine Loss Following Traumautic Brain Injury in Rat. [Thesis]. Virginia Commonwealth University; 2009. Available from: https://doi.org/10.25772/Y1XG-DB87 ; https://scholarscompass.vcu.edu/etd/1892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Cuberos, Hélène. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2016, Université François-Rabelais de Tours

LIMK1 et LIMK2 sont des sérines/thréonine kinases capables de phosphoryler et d’inactiver la cofiline, un facteur de dépolymérisation de l’actine. Elles sont régulées négativement par… (more)

Subjects/Keywords: Neurofibromatose de type 1; Déficience intellectuelle; LIMK2; LIMK2‐1; Cofiline; Actine; Neurofibromatosis type 1; Intellectual disability; LIMK2‐1; Cofilin; Actin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cuberos, H. (2016). Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2016TOUR3306

Chicago Manual of Style (16th Edition):

Cuberos, Hélène. “Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.” 2016. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed August 08, 2020. http://www.theses.fr/2016TOUR3306.

MLA Handbook (7th Edition):

Cuberos, Hélène. “Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.” 2016. Web. 08 Aug 2020.

Vancouver:

Cuberos H. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2016. [cited 2020 Aug 08]. Available from: http://www.theses.fr/2016TOUR3306.

Council of Science Editors:

Cuberos H. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2016. Available from: http://www.theses.fr/2016TOUR3306

30. Rochelle, Tristan. Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Poitiers

Les oncogènes Bcr-Abl (p190bcr-abl et p210bcr-abl) sont issus d'une translocation chromosomique t(9,22) qui fusionne en phase les gènes bcr et c-abl. p210bcr-abl est généralement responsable… (more)

Subjects/Keywords: Bcr-Abl; Cofiline-1; ADF/destrine; Migration; RhoA; Invadopodes; Bcr-Abl; Cofilin-1; ADF/destrin; Migration; RhoA; Invadopodia; 572.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rochelle, T. (2012). Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2012POIT1401

Chicago Manual of Style (16th Edition):

Rochelle, Tristan. “Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl.” 2012. Doctoral Dissertation, Poitiers. Accessed August 08, 2020. http://www.theses.fr/2012POIT1401.

MLA Handbook (7th Edition):

Rochelle, Tristan. “Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl.” 2012. Web. 08 Aug 2020.

Vancouver:

Rochelle T. Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl. [Internet] [Doctoral dissertation]. Poitiers; 2012. [cited 2020 Aug 08]. Available from: http://www.theses.fr/2012POIT1401.

Council of Science Editors:

Rochelle T. Signalisation des GTPases de la famille Rho dans les phénotypes migratoires induits par les différentes formes de Bcr-Abl : Road marking of the GTPases of the family Rho in the migratory phenotypes led by the various forms of Bcr-Abl. [Doctoral Dissertation]. Poitiers; 2012. Available from: http://www.theses.fr/2012POIT1401

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