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You searched for subject:(clonal expansion). Showing records 1 – 8 of 8 total matches.

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Washington University in St. Louis

1. Xie, Mingchao. Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion.

Degree: PhD, Biology & Biomedical Sciences (Computational & Systems Biology), 2016, Washington University in St. Louis

 Cancers, including hematologic malignancies, arise as a result of the stepwise accumulation of mutations. Some early mutations that potentially initiate clonal expansion might exist in… (more)

Subjects/Keywords: Age; Blood somatic mutation; Clonal expansion; Initial driver mutation; PPM1D; TP53

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APA (6th Edition):

Xie, M. (2016). Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/1015

Chicago Manual of Style (16th Edition):

Xie, Mingchao. “Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion.” 2016. Doctoral Dissertation, Washington University in St. Louis. Accessed December 15, 2019. https://openscholarship.wustl.edu/art_sci_etds/1015.

MLA Handbook (7th Edition):

Xie, Mingchao. “Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion.” 2016. Web. 15 Dec 2019.

Vancouver:

Xie M. Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2016. [cited 2019 Dec 15]. Available from: https://openscholarship.wustl.edu/art_sci_etds/1015.

Council of Science Editors:

Xie M. Discovery and Functional Implication of Genetic Alterations Associated with Clonal Hematopoietic Expansion. [Doctoral Dissertation]. Washington University in St. Louis; 2016. Available from: https://openscholarship.wustl.edu/art_sci_etds/1015

2. CHUA REN JIE, ISAAC. Regulation of T Cell Clonal Expansion.

Degree: 2012, National University of Singapore

Subjects/Keywords: T cell; clonal expansion; regulation

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APA (6th Edition):

CHUA REN JIE, I. (2012). Regulation of T Cell Clonal Expansion. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/35549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHUA REN JIE, ISAAC. “Regulation of T Cell Clonal Expansion.” 2012. Thesis, National University of Singapore. Accessed December 15, 2019. http://scholarbank.nus.edu.sg/handle/10635/35549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHUA REN JIE, ISAAC. “Regulation of T Cell Clonal Expansion.” 2012. Web. 15 Dec 2019.

Vancouver:

CHUA REN JIE I. Regulation of T Cell Clonal Expansion. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2019 Dec 15]. Available from: http://scholarbank.nus.edu.sg/handle/10635/35549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHUA REN JIE I. Regulation of T Cell Clonal Expansion. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/35549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

3. Nwaneshiudu, Adaobi I. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

The human gamma-delta (gd) TCR+ T-cell subset may undergo specific antigen-driven activation and clonal expansion, in the context of systemic sclerosis (SSc)… (more)

Subjects/Keywords: Health Sciences, Immunology; Gamma-delta TCR+ T-cells; Putative antigens; Clonal expansion; Systemic Sclerosis; Autoimmune disease; Fibrosis

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APA (6th Edition):

Nwaneshiudu, A. I. (2008). The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,11843

Chicago Manual of Style (16th Edition):

Nwaneshiudu, Adaobi I. “The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.” 2008. Doctoral Dissertation, Temple University. Accessed December 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,11843.

MLA Handbook (7th Edition):

Nwaneshiudu, Adaobi I. “The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.” 2008. Web. 15 Dec 2019.

Vancouver:

Nwaneshiudu AI. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Dec 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,11843.

Council of Science Editors:

Nwaneshiudu AI. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,11843


Florida International University

4. Abood, Steven. The Effects of Artemisia Derived Natural Products on Adipogenesis.

Degree: PhD, Biology, 2017, Florida International University

  For the first time in human history, more people worldwide suffer from obesity than are undernourished. Numerous health complications are associated with obesity including… (more)

Subjects/Keywords: adipogenesis; obesity; sesquiterpene lactones; mitotic clonal expansion; Biological Psychology; Biology; Cell Biology; Evolution; Health Psychology; Neuroscience and Neurobiology; Nutrition

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APA (6th Edition):

Abood, S. (2017). The Effects of Artemisia Derived Natural Products on Adipogenesis. (Doctoral Dissertation). Florida International University. Retrieved from http://digitalcommons.fiu.edu/etd/3383 ; 10.25148/etd.FIDC001953 ; FIDC001953

Chicago Manual of Style (16th Edition):

Abood, Steven. “The Effects of Artemisia Derived Natural Products on Adipogenesis.” 2017. Doctoral Dissertation, Florida International University. Accessed December 15, 2019. http://digitalcommons.fiu.edu/etd/3383 ; 10.25148/etd.FIDC001953 ; FIDC001953.

MLA Handbook (7th Edition):

Abood, Steven. “The Effects of Artemisia Derived Natural Products on Adipogenesis.” 2017. Web. 15 Dec 2019.

Vancouver:

Abood S. The Effects of Artemisia Derived Natural Products on Adipogenesis. [Internet] [Doctoral dissertation]. Florida International University; 2017. [cited 2019 Dec 15]. Available from: http://digitalcommons.fiu.edu/etd/3383 ; 10.25148/etd.FIDC001953 ; FIDC001953.

Council of Science Editors:

Abood S. The Effects of Artemisia Derived Natural Products on Adipogenesis. [Doctoral Dissertation]. Florida International University; 2017. Available from: http://digitalcommons.fiu.edu/etd/3383 ; 10.25148/etd.FIDC001953 ; FIDC001953


University of Cambridge

5. Chappell, Joel. Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease .

Degree: 2018, University of Cambridge

 Vascular smooth muscle cell (VSMC) accumulation is a hallmark of atherosclerosis and vascular injury. However, fundamental aspects of proliferation and the phenotypic changes within individual… (more)

Subjects/Keywords: vascular smooth muscle cells; single cell RNA sequencing; cellular heterogeneity; heart disease; atherosclerosis; carotid ligation; clonal expansion; brainbow; multi-colour lineage tracing; confetti

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APA (6th Edition):

Chappell, J. (2018). Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274543

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chappell, Joel. “Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease .” 2018. Thesis, University of Cambridge. Accessed December 15, 2019. https://www.repository.cam.ac.uk/handle/1810/274543.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chappell, Joel. “Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease .” 2018. Web. 15 Dec 2019.

Vancouver:

Chappell J. Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Dec 15]. Available from: https://www.repository.cam.ac.uk/handle/1810/274543.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chappell J. Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/274543

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Ide, Jennifer C. Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis .

Degree: 2011, University of Ottawa

 Macrophage-conditioned medium (MacCM) inhibits the differentiation of rodent and human preadipocytes. Previous studies report that murine J774A.1-MacCM inhibits clonal expansion (early required phase of adipogenesis),… (more)

Subjects/Keywords: preadipocyte; adipocyte differentiation; macrophage; cyclin; cyclin-dependent kinase; mitotic clonal expansion

…expression during mitotic clonal expansion in 3T3-L1 preadipocytes 41 Figure 4. J774-A.1 MacCM… …reduces differentiation-induced cyclin D1 and D2 expression during mitotic clonal expansion in… …clonal expansion in 3T3-L1 preadipocytes 44 Figure 6. Differentiation-induced cyclin A… …protein and mRNA expression are inhibited by J774A.1-MacCM during mitotic clonal expansion in… …by J774A.1MacCM during mitotic clonal expansion in 3T3-L1 preadipocytes 47 Figure 8… 

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APA (6th Edition):

Ide, J. C. (2011). Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ide, Jennifer C. “Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis .” 2011. Thesis, University of Ottawa. Accessed December 15, 2019. http://hdl.handle.net/10393/19752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ide, Jennifer C. “Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis .” 2011. Web. 15 Dec 2019.

Vancouver:

Ide JC. Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/10393/19752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ide JC. Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

7. Yokoyama, Akira. Age-related remodelling of oesophageal epithelia by mutated cancer drivers .

Degree: 2019, Kyoto University

Subjects/Keywords: clonal expansion; physiologically normal oesophageal epithelia; mutations in driver genes; NOTCH1; heavy smoking and drinking; ageing; lifestyle risk

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APA (6th Edition):

Yokoyama, A. (2019). Age-related remodelling of oesophageal epithelia by mutated cancer drivers . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/244517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yokoyama, Akira. “Age-related remodelling of oesophageal epithelia by mutated cancer drivers .” 2019. Thesis, Kyoto University. Accessed December 15, 2019. http://hdl.handle.net/2433/244517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yokoyama, Akira. “Age-related remodelling of oesophageal epithelia by mutated cancer drivers .” 2019. Web. 15 Dec 2019.

Vancouver:

Yokoyama A. Age-related remodelling of oesophageal epithelia by mutated cancer drivers . [Internet] [Thesis]. Kyoto University; 2019. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/2433/244517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yokoyama A. Age-related remodelling of oesophageal epithelia by mutated cancer drivers . [Thesis]. Kyoto University; 2019. Available from: http://hdl.handle.net/2433/244517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Brouwer, Andrew Frederick. Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer.

Degree: PhD, Applied and Interdisciplinary Mathematics, 2015, University of Michigan

 The human papillomavirus (HPV) infects multiple sites in the human epithelium and is the etiological agent for over 90% of anogenital cancers and an increasing… (more)

Subjects/Keywords: Human papillomavirus (HPV); Age-period-cohort model; Multisite infectious disease model; Multistage clonal expansion model; Oral cancer; National Health and Nutrition Examination Survey; Public Health; Mathematics; Statistics and Numeric Data; Health Sciences; Science

…the two-stage clonal expansion model . . . . . . . . . Oral HPV prevalence with genital… …Schematic of the two-stage clonal expansion model with period and cohort dependencies… …implications about the underlying biological and epidemiological causes. Multistage clonal expansion… …genetic transformation that leads to clonal expansion, followed by transformations that lead to… …age–period–cohort, and multistage clonal expansion models, including the relevant literature… 

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APA (6th Edition):

Brouwer, A. F. (2015). Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111523

Chicago Manual of Style (16th Edition):

Brouwer, Andrew Frederick. “Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer.” 2015. Doctoral Dissertation, University of Michigan. Accessed December 15, 2019. http://hdl.handle.net/2027.42/111523.

MLA Handbook (7th Edition):

Brouwer, Andrew Frederick. “Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer.” 2015. Web. 15 Dec 2019.

Vancouver:

Brouwer AF. Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/2027.42/111523.

Council of Science Editors:

Brouwer AF. Models of HPV as an Infectious Disease and as an Etiological Agent of Cancer. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111523

.