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You searched for subject:(cholestatic liver disease). Showing records 1 – 4 of 4 total matches.

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University of Toronto

1. Murillo Perez, Carla Fiorella. A changing natural history of primary biliary cholangitis and its impact on risk stratification.

Degree: 2018, University of Toronto

We sought to describe temporal trends in the presenting characteristics and clinical course of primary biliary cholangitis (PBC) from the 1970s to 2014 in a… (more)

Subjects/Keywords: autoimmune liver disease; bilirubin; cholestatic liver disease; epidemiology; hepatology; 0566

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APA (6th Edition):

Murillo Perez, C. F. (2018). A changing natural history of primary biliary cholangitis and its impact on risk stratification. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91593

Chicago Manual of Style (16th Edition):

Murillo Perez, Carla Fiorella. “A changing natural history of primary biliary cholangitis and its impact on risk stratification.” 2018. Masters Thesis, University of Toronto. Accessed September 19, 2019. http://hdl.handle.net/1807/91593.

MLA Handbook (7th Edition):

Murillo Perez, Carla Fiorella. “A changing natural history of primary biliary cholangitis and its impact on risk stratification.” 2018. Web. 19 Sep 2019.

Vancouver:

Murillo Perez CF. A changing natural history of primary biliary cholangitis and its impact on risk stratification. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Sep 19]. Available from: http://hdl.handle.net/1807/91593.

Council of Science Editors:

Murillo Perez CF. A changing natural history of primary biliary cholangitis and its impact on risk stratification. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91593


Queen Mary, University of London

2. Nicolaou, Michael. Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4.

Degree: PhD, 2012, Queen Mary, University of London

 Mammalian ABC (ATP-binding cassette) transporters are integral membrane proteins that translocate allocrites across biological membranes using ATP as a substrate. ABCB1 is a polyspecific efflux… (more)

Subjects/Keywords: 616.99; Medicine; Cutaneous Research; Cancer; Multidrug resistance; Cholestatic Liver Disease; Membrane Transport Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nicolaou, M. (2012). Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8560 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667063

Chicago Manual of Style (16th Edition):

Nicolaou, Michael. “Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4.” 2012. Doctoral Dissertation, Queen Mary, University of London. Accessed September 19, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8560 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667063.

MLA Handbook (7th Edition):

Nicolaou, Michael. “Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4.” 2012. Web. 19 Sep 2019.

Vancouver:

Nicolaou M. Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2012. [cited 2019 Sep 19]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8560 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667063.

Council of Science Editors:

Nicolaou M. Structure and function analysis of the mammalian ATP-binding cassette transporters, ABCB1 and ABCB4. [Doctoral Dissertation]. Queen Mary, University of London; 2012. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8560 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667063


Kyoto University

3. Jemail, Leila. Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy .

Degree: 2019, Kyoto University

Subjects/Keywords: Experimental Pathology; Cholestatic liver disease; Mechanisms of disease; Mouse model; Macrophage

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jemail, L. (2019). Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/242350

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jemail, Leila. “Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy .” 2019. Thesis, Kyoto University. Accessed September 19, 2019. http://hdl.handle.net/2433/242350.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jemail, Leila. “Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy .” 2019. Web. 19 Sep 2019.

Vancouver:

Jemail L. Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy . [Internet] [Thesis]. Kyoto University; 2019. [cited 2019 Sep 19]. Available from: http://hdl.handle.net/2433/242350.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jemail L. Pivotal roles of Kupffer cells in the progression and regression of DDC-induced chronic choangiopathy . [Thesis]. Kyoto University; 2019. Available from: http://hdl.handle.net/2433/242350

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. González Rubio, Sandra. Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares.

Degree: 2018, Universidad de Córdoba, UCOPress

Subjects/Keywords: Enfermedad hepática colestásica; Óxido nítrico sintasa endotelial; Óxido nítrico; Estrés oxidativo; Ácido glicoquenodesoxicólico; Sp1; cJun; cFos; Ciclina D1; Cholestatic liver disease; Endothelial nitric oxide synthase; Nitric oxide; Oxidative stress; Glycoquenodeoxycholic acid; Cyclin D1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

González Rubio, S. (2018). Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares. (Thesis). Universidad de Córdoba, UCOPress. Retrieved from http://hdl.handle.net/10396/15152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

González Rubio, Sandra. “Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares.” 2018. Thesis, Universidad de Córdoba, UCOPress. Accessed September 19, 2019. http://hdl.handle.net/10396/15152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

González Rubio, Sandra. “Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares.” 2018. Web. 19 Sep 2019.

Vancouver:

González Rubio S. Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares. [Internet] [Thesis]. Universidad de Córdoba, UCOPress; 2018. [cited 2019 Sep 19]. Available from: http://hdl.handle.net/10396/15152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

González Rubio S. Regulación de NOS-3 durante la muerte hepatocelular inducida por ácidos biliares. [Thesis]. Universidad de Córdoba, UCOPress; 2018. Available from: http://hdl.handle.net/10396/15152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.