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You searched for subject:(chemoresistance). Showing records 1 – 30 of 82 total matches.

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Wake Forest University

1. Alexander, Peter M. THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA.

Degree: 2015, Wake Forest University

 Acute leukemias are cancers of the blood and bone marrow that affect thousands of Americans annually. These diseases can be myeloid or lymphoid in origin.… (more)

Subjects/Keywords: Chemoresistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alexander, P. M. (2015). THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/57145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alexander, Peter M. “THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA.” 2015. Thesis, Wake Forest University. Accessed November 19, 2017. http://hdl.handle.net/10339/57145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alexander, Peter M. “THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA.” 2015. Web. 19 Nov 2017.

Vancouver:

Alexander PM. THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA. [Internet] [Thesis]. Wake Forest University; 2015. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10339/57145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alexander PM. THE NOVEL GEMCITABINE PHOSPHOLIPID CONJUGATE KPC34 HAS ACTIVITY AGAINST MULTIPLE MODELS OF RESISTANT ACUTE LYMPHOBLASTIC LEUKEMIA. [Thesis]. Wake Forest University; 2015. Available from: http://hdl.handle.net/10339/57145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

2. Mcdonald, Gail. INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS .

Degree: Anatomy and Cell Biology, 2008, Queens University

 One of the major challenges associated with cancer therapy is the acquisition of chemoresistance by tumour cells. Many novel therapeutic approaches to overcome chemoresistance have… (more)

Subjects/Keywords: PI3K; Chemoresistance

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APA (6th Edition):

Mcdonald, G. (2008). INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/1475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mcdonald, Gail. “INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS .” 2008. Thesis, Queens University. Accessed November 19, 2017. http://hdl.handle.net/1974/1475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mcdonald, Gail. “INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS .” 2008. Web. 19 Nov 2017.

Vancouver:

Mcdonald G. INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS . [Internet] [Thesis]. Queens University; 2008. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/1974/1475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mcdonald G. INHIBITION OF PHOSPHATIDYLINOSITOL 3-KINASE (PI3K) SIGNALLING LEADS TO RESISTANCE TO CHEMOTHERAPEUTIC AGENTS IN HUMAN CANCER CELLS . [Thesis]. Queens University; 2008. Available from: http://hdl.handle.net/1974/1475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

3. Samadi, Nasser. Autotaxin, lysophosphatidate and taxol resistance.

Degree: PhD, Medical Sciences- Laboratory Medicine and Pathology, 2009, University of Alberta

 First-line treatment of breast and other cancers with Taxol is compromised by resistance in up to 40% of patients. To improve chemotherapy, it is vital… (more)

Subjects/Keywords: autotaxin; chemoresistance; lysophosphatidate; taxol

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APA (6th Edition):

Samadi, N. (2009). Autotaxin, lysophosphatidate and taxol resistance. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/1831ck31f

Chicago Manual of Style (16th Edition):

Samadi, Nasser. “Autotaxin, lysophosphatidate and taxol resistance.” 2009. Doctoral Dissertation, University of Alberta. Accessed November 19, 2017. https://era.library.ualberta.ca/files/1831ck31f.

MLA Handbook (7th Edition):

Samadi, Nasser. “Autotaxin, lysophosphatidate and taxol resistance.” 2009. Web. 19 Nov 2017.

Vancouver:

Samadi N. Autotaxin, lysophosphatidate and taxol resistance. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2017 Nov 19]. Available from: https://era.library.ualberta.ca/files/1831ck31f.

Council of Science Editors:

Samadi N. Autotaxin, lysophosphatidate and taxol resistance. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/1831ck31f


Vanderbilt University

4. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed November 19, 2017. http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 19 Nov 2017.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2017 Nov 19]. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;


Euskal Herriko Unibertsitatea / Universidad del País Vasco

5. Rodríguez Pérez, José Antonio. USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy .

Degree: 2013, Euskal Herriko Unibertsitatea / Universidad del País Vasco

 Reversible protein ubiquitination is emerging as a key process for maintaining cell homeostasis, and the enzymes that participate in this process, in particular E3 ubiquitin… (more)

Subjects/Keywords: deubiquitinase; USP1; DNA damage; chemoresistance

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APA (6th Edition):

Rodríguez Pérez, J. A. (2013). USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy . (Thesis). Euskal Herriko Unibertsitatea / Universidad del País Vasco. Retrieved from http://hdl.handle.net/10810/10571

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodríguez Pérez, José Antonio. “USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy .” 2013. Thesis, Euskal Herriko Unibertsitatea / Universidad del País Vasco. Accessed November 19, 2017. http://hdl.handle.net/10810/10571.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodríguez Pérez, José Antonio. “USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy .” 2013. Web. 19 Nov 2017.

Vancouver:

Rodríguez Pérez JA. USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy . [Internet] [Thesis]. Euskal Herriko Unibertsitatea / Universidad del País Vasco; 2013. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10810/10571.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodríguez Pérez JA. USP1 deubiquitinase: cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy . [Thesis]. Euskal Herriko Unibertsitatea / Universidad del País Vasco; 2013. Available from: http://hdl.handle.net/10810/10571

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

6. Black, Madison. The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer .

Degree: Anatomy & Cell Biology, 2015, Queens University

 The ability of tumour cells to avoid immune destruction (immune escape) and their acquired resistance to anti-cancer drugs constitute important barriers to the successful management… (more)

Subjects/Keywords: Chemoresistance; PD-1/PD-L1

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APA (6th Edition):

Black, M. (2015). The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Black, Madison. “The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer .” 2015. Thesis, Queens University. Accessed November 19, 2017. http://hdl.handle.net/1974/13145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Black, Madison. “The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer .” 2015. Web. 19 Nov 2017.

Vancouver:

Black M. The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer . [Internet] [Thesis]. Queens University; 2015. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/1974/13145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Black M. The PD-1/PD-L1 Axis: An Immune-Mediated Mechanism of Chemoresistance in Cancer . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. 三田村, 卓. Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET.

Degree: 博士(医学), 医学, 2013, Hokkaido University

 Ovarian cancer is one of the most aggressive female reproductive tract tumors. Paclitaxel (PTX) is widely used for the treatment of ovarian cancer. However, ovarian… (more)

Subjects/Keywords: ovarian cancer; microRNA; MET; chemoresistance; paclitaxel

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

三田村, . (2013). Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET. (Doctoral Dissertation). Hokkaido University. Retrieved from http://hdl.handle.net/2115/53260

Chicago Manual of Style (16th Edition):

三田村, 卓. “Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET.” 2013. Doctoral Dissertation, Hokkaido University. Accessed November 19, 2017. http://hdl.handle.net/2115/53260.

MLA Handbook (7th Edition):

三田村, 卓. “Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET.” 2013. Web. 19 Nov 2017.

Vancouver:

三田村 . Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET. [Internet] [Doctoral dissertation]. Hokkaido University; 2013. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/2115/53260.

Council of Science Editors:

三田村 . Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET. [Doctoral Dissertation]. Hokkaido University; 2013. Available from: http://hdl.handle.net/2115/53260


Universiteit Utrecht

8. Rontogianni, S. Proteomics and its application to discover chemoresistant biomarkers in human cancer.

Degree: 2013, Universiteit Utrecht

 Primary or acquired resistance to traditional cytotoxic chemotherapy remains a major obstacle to the successful management of patients in clinical oncology. The discovery of reliable… (more)

Subjects/Keywords: proteomics; mass spectrometry; predictive biomarkers; chemoresistance

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APA (6th Edition):

Rontogianni, S. (2013). Proteomics and its application to discover chemoresistant biomarkers in human cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/283412

Chicago Manual of Style (16th Edition):

Rontogianni, S. “Proteomics and its application to discover chemoresistant biomarkers in human cancer.” 2013. Masters Thesis, Universiteit Utrecht. Accessed November 19, 2017. http://dspace.library.uu.nl:8080/handle/1874/283412.

MLA Handbook (7th Edition):

Rontogianni, S. “Proteomics and its application to discover chemoresistant biomarkers in human cancer.” 2013. Web. 19 Nov 2017.

Vancouver:

Rontogianni S. Proteomics and its application to discover chemoresistant biomarkers in human cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2017 Nov 19]. Available from: http://dspace.library.uu.nl:8080/handle/1874/283412.

Council of Science Editors:

Rontogianni S. Proteomics and its application to discover chemoresistant biomarkers in human cancer. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/283412


University of California – San Diego

9. Santosa, Endi Kusuma Pramudya. Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer.

Degree: Biology, 2016, University of California – San Diego

 Cell fusion is an important event that mediates various biological processes. However, the role of cell fusion in cancer has not been very well described… (more)

Subjects/Keywords: Biology; Cancer; Cell Biology; Cell Fusion; Chemoresistance

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APA (6th Edition):

Santosa, E. K. P. (2016). Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/82j251m2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santosa, Endi Kusuma Pramudya. “Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer.” 2016. Thesis, University of California – San Diego. Accessed November 19, 2017. http://www.escholarship.org/uc/item/82j251m2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santosa, Endi Kusuma Pramudya. “Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer.” 2016. Web. 19 Nov 2017.

Vancouver:

Santosa EKP. Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2017 Nov 19]. Available from: http://www.escholarship.org/uc/item/82j251m2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santosa EKP. Heterologous Cell-Cell Fusion as a Mechanism of DNA Exchange and Chemoresistance in Cancer. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/82j251m2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. 三田村, 卓. Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される.

Degree: 博士(医学), 2013, Hokkaido University / 北海道大学

Ovarian cancer is one of the most aggressive female reproductive tract tumors. Paclitaxel (PTX) is widely used for the treatment of ovarian cancer. However, ovarian… (more)

Subjects/Keywords: ovarian cancer; microRNA; MET; chemoresistance; paclitaxel

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APA (6th Edition):

三田村, . (2013). Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される. (Thesis). Hokkaido University / 北海道大学. Retrieved from http://hdl.handle.net/2115/53260 ; http://dx.doi.org/10.14943/doctoral.k11041

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

三田村, 卓. “Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される.” 2013. Thesis, Hokkaido University / 北海道大学. Accessed November 19, 2017. http://hdl.handle.net/2115/53260 ; http://dx.doi.org/10.14943/doctoral.k11041.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

三田村, 卓. “Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される.” 2013. Web. 19 Nov 2017.

Vancouver:

三田村 . Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される. [Internet] [Thesis]. Hokkaido University / 北海道大学; 2013. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/2115/53260 ; http://dx.doi.org/10.14943/doctoral.k11041.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

三田村 . Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET : miRNA-31の発現低下により、レセプターチロシンキナーゼMETの抑制が解除され、卵巣癌のタキサン耐性が誘導される. [Thesis]. Hokkaido University / 北海道大学; 2013. Available from: http://hdl.handle.net/2115/53260 ; http://dx.doi.org/10.14943/doctoral.k11041

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Lee, Yong (Brian). Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer.

Degree: Clinical School - St Vincent's Hospital, 2014, University of New South Wales

 Prostate cancer remains the third leading cause of cancer death in men in the developed world with metastatic castrate resistant prostate cancer (CRPC) being the… (more)

Subjects/Keywords: Focal adhesion kinase; Prostate cancer; Chemoresistance

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APA (6th Edition):

Lee, Y. (. (2014). Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53873

Chicago Manual of Style (16th Edition):

Lee, Yong (Brian). “Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer.” 2014. Doctoral Dissertation, University of New South Wales. Accessed November 19, 2017. http://handle.unsw.edu.au/1959.4/53873.

MLA Handbook (7th Edition):

Lee, Yong (Brian). “Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer.” 2014. Web. 19 Nov 2017.

Vancouver:

Lee Y(. Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2017 Nov 19]. Available from: http://handle.unsw.edu.au/1959.4/53873.

Council of Science Editors:

Lee Y(. Characterisation of Docetaxel-Resistance in Castrate Resistant Prostate Cancer. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53873


University of Miami

12. Escudero, Diogo O. Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer.

Degree: PhD, Molecular Cell and Developmental Biology (Medicine), 2013, University of Miami

 Profound changes in the extracellular matrix (ECM) in the vicinity of tumor cells occur as the tumor establishes its own microenvironment. Among these changes, elevated… (more)

Subjects/Keywords: Hyaluronic Acid Family; Bladder Cancer; Cancer Biomarkers; Chemoresistance

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APA (6th Edition):

Escudero, D. O. (2013). Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer. (Doctoral Dissertation). University of Miami. Retrieved from http://scholarlyrepository.miami.edu/oa_dissertations/1019

Chicago Manual of Style (16th Edition):

Escudero, Diogo O. “Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer.” 2013. Doctoral Dissertation, University of Miami. Accessed November 19, 2017. http://scholarlyrepository.miami.edu/oa_dissertations/1019.

MLA Handbook (7th Edition):

Escudero, Diogo O. “Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer.” 2013. Web. 19 Nov 2017.

Vancouver:

Escudero DO. Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer. [Internet] [Doctoral dissertation]. University of Miami; 2013. [cited 2017 Nov 19]. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/1019.

Council of Science Editors:

Escudero DO. Hyaluronic Acid Family of Molecules: Biomarkers and Functional Studies in Bladder Cancer. [Doctoral Dissertation]. University of Miami; 2013. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/1019


University of Cincinnati

13. LaPensee, Elizabeth W. Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma.

Degree: PhD, Medicine : Cell and Molecular Biology, 2008, University of Cincinnati

  Chemotherapy is mainstay treatment for cancer patients, but unfortunately many tumors are resistant to therapy. Much research now focuses on identifying mechanisms that contribute… (more)

Subjects/Keywords: Cellular Biology; chemoresistance; prolactin; estrogen; bisphenol A; apoptosis

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APA (6th Edition):

LaPensee, E. W. (2008). Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1224593025

Chicago Manual of Style (16th Edition):

LaPensee, Elizabeth W. “Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed November 19, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1224593025.

MLA Handbook (7th Edition):

LaPensee, Elizabeth W. “Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma.” 2008. Web. 19 Nov 2017.

Vancouver:

LaPensee EW. Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2017 Nov 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1224593025.

Council of Science Editors:

LaPensee EW. Mechanisms of Chemoresistance in Breast Cancer and Liposarcoma. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1224593025


NSYSU

14. Weng, Chien-hui. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.

Degree: PhD, Biological Sciences, 2013, NSYSU

 Melanoma, which is the most aggressive form of skin cancer, is notoriously resistant to current cancer therapies. Cisplatin is a first-line chemotherapy drug for melanoma.… (more)

Subjects/Keywords: cisplatin; cancer stem cells; giant cells; chemoresistance; melanoma

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APA (6th Edition):

Weng, C. (2013). Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512

Chicago Manual of Style (16th Edition):

Weng, Chien-hui. “Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.” 2013. Doctoral Dissertation, NSYSU. Accessed November 19, 2017. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512.

MLA Handbook (7th Edition):

Weng, Chien-hui. “Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.” 2013. Web. 19 Nov 2017.

Vancouver:

Weng C. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2017 Nov 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512.

Council of Science Editors:

Weng C. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512


NSYSU

15. Liou, Jhuan-Yi. 2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

 Although cisplatin is among the most potent antitumor agents, cisplatin-based chemotherapy has shown limited effectiveness for treating melanoma. Our previous work showed that cisplatin induces… (more)

Subjects/Keywords: giant cells; energy; chemoresistance; Melanoma; 2-DG; cisplatin

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APA (6th Edition):

Liou, J. (2013). 2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716113-111414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liou, Jhuan-Yi. “2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin.” 2013. Thesis, NSYSU. Accessed November 19, 2017. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716113-111414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liou, Jhuan-Yi. “2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin.” 2013. Web. 19 Nov 2017.

Vancouver:

Liou J. 2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin. [Internet] [Thesis]. NSYSU; 2013. [cited 2017 Nov 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716113-111414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liou J. 2-Deoxyglucose Enhances the Chemosensitivity of B16-F10 Melanoma Cells to Cisplatin. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716113-111414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

16. Roger, Jérôme. Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes .

Degree: 2016, Université de Montréal

 La voie de signalisation Ras/MAPK (Ras/mitogen-activated protein kinase) régule une variété de protéines intracellulaires qui jouent un rôle important dans la croissance et la prolifération… (more)

Subjects/Keywords: MAPK; mélanome; RSK; MDM2; p53; chimiorésistance; melanoma; chemoresistance

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APA (6th Edition):

Roger, J. (2016). Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13402

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roger, Jérôme. “Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes .” 2016. Thesis, Université de Montréal. Accessed November 19, 2017. http://hdl.handle.net/1866/13402.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roger, Jérôme. “Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes .” 2016. Web. 19 Nov 2017.

Vancouver:

Roger J. Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/1866/13402.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roger J. Phosphorylation et régulation de l’E3 ubiquitine ligase MDM2 par la protéine kinase RSK dans les mélanomes . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13402

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

17. Thege, Fredrik Ivar. The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids .

Degree: 2017, Cornell University

 Pancreatic cancer is the third-leading cause of cancer-related death in the US and represents a difficult challenge to modern medicine, with increasing incidence and modest… (more)

Subjects/Keywords: Pancreatic cancer; Chemoresistance; EMT; Heterogeneity; Circulating tumor cells; Spheroids; Biomedical engineering

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APA (6th Edition):

Thege, F. I. (2017). The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/47848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thege, Fredrik Ivar. “The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids .” 2017. Thesis, Cornell University. Accessed November 19, 2017. http://hdl.handle.net/1813/47848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thege, Fredrik Ivar. “The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids .” 2017. Web. 19 Nov 2017.

Vancouver:

Thege FI. The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids . [Internet] [Thesis]. Cornell University; 2017. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/1813/47848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thege FI. The effect of phenotypic heterogeneity on pancreatic circulating tumor cell capture and tumor spheroids . [Thesis]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/47848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

Objective Appropriate cell cycle checkpoints are essential for the maintenance of normal cells and chemosensitivity of cancer cells. Clear cell adenocarcinoma (CCA) of the ovary… (more)

Subjects/Keywords: Transcription factors; DNA damage response; Cell cycle; Checkpoint control; Chemoresistance

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APA (6th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, N. (2014). Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed November 19, 2017. http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Web. 19 Nov 2017.

Vancouver:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. CAZZATO; DENISE. ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS.

Degree: 2013, Università degli Studi di Milano

 Melanoma is the most aggressive form of skin cancer and is notoriously resistant to all current modalities of cancer therapy. Malignant melanomas are composed of… (more)

Subjects/Keywords: acid sphingomyelinase; apoptosis; autophagy; chemoresistance; Settore BIO/14 - Farmacologia

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APA (6th Edition):

DENISE, C. (2013). ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/214971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DENISE, CAZZATO;. “ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS.” 2013. Thesis, Università degli Studi di Milano. Accessed November 19, 2017. http://hdl.handle.net/2434/214971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DENISE, CAZZATO;. “ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS.” 2013. Web. 19 Nov 2017.

Vancouver:

DENISE C. ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS. [Internet] [Thesis]. Università degli Studi di Milano; 2013. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/2434/214971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DENISE C. ACID SPHINGOMYELINASE AND MITOCHONDRIA: A NEW LINKIN CHEMOTHERAPEUTIC DRUGS INDUCED APOPTOSIS. [Thesis]. Università degli Studi di Milano; 2013. Available from: http://hdl.handle.net/2434/214971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

20. Esposito, Mark. CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH .

Degree: PhD, 2017, Princeton University

 Section I: E-selectin binding to Fut3/6-modified Glg1 is essential to bone metastasis via the induction of a Mesenchymal-Epithelial Transition and activation of Wnt signaling. The… (more)

Subjects/Keywords: cancer stem cell; chemoresistance; EMT; e-selectin; MET; metastasis

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APA (6th Edition):

Esposito, M. (2017). CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01rx913s50n

Chicago Manual of Style (16th Edition):

Esposito, Mark. “CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH .” 2017. Doctoral Dissertation, Princeton University. Accessed November 19, 2017. http://arks.princeton.edu/ark:/88435/dsp01rx913s50n.

MLA Handbook (7th Edition):

Esposito, Mark. “CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH .” 2017. Web. 19 Nov 2017.

Vancouver:

Esposito M. CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH . [Internet] [Doctoral dissertation]. Princeton University; 2017. [cited 2017 Nov 19]. Available from: http://arks.princeton.edu/ark:/88435/dsp01rx913s50n.

Council of Science Editors:

Esposito M. CATALYZING CANCER METASTASIS: A STUDY OF THE ENZYMATIC DRIVERS OF TUMOR DISSEMINATION AND GROWTH . [Doctoral Dissertation]. Princeton University; 2017. Available from: http://arks.princeton.edu/ark:/88435/dsp01rx913s50n


University of Iowa

21. Brachova, Pavla. Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas.

Degree: PhD, Molecular and Cellular Biology, 2014, University of Iowa

  The tumor suppressor gene TP53 sits at the crux of response to cellular stresses. This is the most frequently inactivated gene in human tumors,… (more)

Subjects/Keywords: chemoresistance; gain of function; oncomorphic; ovarian cancer; p53; TP53; Cell Biology

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APA (6th Edition):

Brachova, P. (2014). Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas. (Doctoral Dissertation). University of Iowa. Retrieved from http://ir.uiowa.edu/etd/4581

Chicago Manual of Style (16th Edition):

Brachova, Pavla. “Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas.” 2014. Doctoral Dissertation, University of Iowa. Accessed November 19, 2017. http://ir.uiowa.edu/etd/4581.

MLA Handbook (7th Edition):

Brachova, Pavla. “Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas.” 2014. Web. 19 Nov 2017.

Vancouver:

Brachova P. Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2017 Nov 19]. Available from: http://ir.uiowa.edu/etd/4581.

Council of Science Editors:

Brachova P. Oncomorphic Tp53 mutations in advanced serous ovarian carcinomas. [Doctoral Dissertation]. University of Iowa; 2014. Available from: http://ir.uiowa.edu/etd/4581


Universidade Nova

22. Poejo, Joana Margarida de Andrade. Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach.

Degree: 2011, Universidade Nova

Dissertation to obtain a Master Degree in Biotechnology

Recently, prevention of cancer through dietary intervention has received an increasing interest. In particular, dietary polyphenols mainly… (more)

Subjects/Keywords: Colon cancer; Polyphenols; Opuntia spp; Antiproliferative effect; Chemoresistance.

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APA (6th Edition):

Poejo, J. M. d. A. (2011). Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Poejo, Joana Margarida de Andrade. “Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach.” 2011. Thesis, Universidade Nova. Accessed November 19, 2017. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Poejo, Joana Margarida de Andrade. “Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach.” 2011. Web. 19 Nov 2017.

Vancouver:

Poejo JMdA. Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach. [Internet] [Thesis]. Universidade Nova; 2011. [cited 2017 Nov 19]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Poejo JMdA. Evaluation of Opuntia spp. bioactive products as promising natural chemotherapeutical agents- an in vitro approach. [Thesis]. Universidade Nova; 2011. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

23. Haslehurst, Alexandria. The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 The epithelial-mesenchymal transition (EMT) is an evolutionarily conserved developmental process characterized by the loss of intercellular contacts, changes in cell polarity and increased migration and… (more)

Subjects/Keywords: metastasis; chemoresistance; biomarkers; ovarian cancer; prostate cancer; EMT

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APA (6th Edition):

Haslehurst, A. (2014). The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haslehurst, Alexandria. “The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes .” 2014. Thesis, Queens University. Accessed November 19, 2017. http://hdl.handle.net/1974/12599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haslehurst, Alexandria. “The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes .” 2014. Web. 19 Nov 2017.

Vancouver:

Haslehurst A. The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes . [Internet] [Thesis]. Queens University; 2014. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/1974/12599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haslehurst A. The role of the epithelial-mesenchymal transition in aggressive tumour phenotypes . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

24. Huang, Tzu-chuan Jane, MD. Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy.

Degree: MS, 2011, Texas Medical Center

  The plasma membrane xc- cystine/glutamate transporter mediates cellular uptake of cystine in exchange for intracellular glutamate and is highly expressed by pancreatic cancer cells.… (more)

Subjects/Keywords: xCT; SNPs; pancreatic cancer; glutathione; cystine; xc-; gemcitabine; platinum; chemoresistance; immunohistochemistry; Medicine and Health Sciences

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APA (6th Edition):

Huang, Tzu-chuan Jane, M. (2011). Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/219

Chicago Manual of Style (16th Edition):

Huang, Tzu-chuan Jane, MD. “Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy.” 2011. Masters Thesis, Texas Medical Center. Accessed November 19, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/219.

MLA Handbook (7th Edition):

Huang, Tzu-chuan Jane, MD. “Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy.” 2011. Web. 19 Nov 2017.

Vancouver:

Huang, Tzu-chuan Jane M. Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy. [Internet] [Masters thesis]. Texas Medical Center; 2011. [cited 2017 Nov 19]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/219.

Council of Science Editors:

Huang, Tzu-chuan Jane M. Prognostic significance of xCT polymorphisms and expression in patients with advanced pancreatic cancer treated with chemotherapy. [Masters Thesis]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/219


Univerzitet u Beogradu

25. Stojković-Burić, Sonja M., 1985-. Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo.

Degree: Biološki fakultet, 2017, Univerzitet u Beogradu

Molekularna onkologija - Kancerogeneza / Molecular oncology - Cancerogenesis

Uspešnost terapije glioblastoma, najĉešćeg i najagresivnijeg malignog tumora centralnog nervnog sistema, je osujećena usled visokog stepena… (more)

Subjects/Keywords: Glioblastoma; chemoresistance; carmustine (BCNU); temozolomide (TMZ); collateral sensitivity; invasiveness; antiglioma therapy; CoQ10

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stojković-Burić, Sonja M., 1. (2017). Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:15650/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stojković-Burić, Sonja M., 1985-. “Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo.” 2017. Thesis, Univerzitet u Beogradu. Accessed November 19, 2017. https://fedorabg.bg.ac.rs/fedora/get/o:15650/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stojković-Burić, Sonja M., 1985-. “Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo.” 2017. Web. 19 Nov 2017.

Vancouver:

Stojković-Burić, Sonja M. 1. Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo. [Internet] [Thesis]. Univerzitet u Beogradu; 2017. [cited 2017 Nov 19]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:15650/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stojković-Burić, Sonja M. 1. Model ćelija C6 glioma pacova kao oruđe za pronalaženje novih terapeutskih strategija: karakterizacija rezistentnog fenotipa in vitro i in vivo. [Thesis]. Univerzitet u Beogradu; 2017. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:15650/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

26. Yang, Chun-feng. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

 Breast cancer is a leading cause of death in women worldwide, and chemotherapy is one of the primary strategies for breast cancer treatment. However, the… (more)

Subjects/Keywords: DNA double-strand breaks; camptothecin; Phthalate; Bis(2-ethylhexyl) phthalate; DEHP; zebrafish; chemoresistance; breast cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, C. (2016). The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Thesis, NSYSU. Accessed November 19, 2017. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Web. 19 Nov 2017.

Vancouver:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Internet] [Thesis]. NSYSU; 2016. [cited 2017 Nov 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

27. Chang, Insoon. Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis.

Degree: Oral Biology, 2017, UCLA

 Development of chemoresistance, invasive growth, and metastasis remain key challenges in head and neck squamous cell carcinoma (HNSCC) therapy. Recent studies revealed that an activated… (more)

Subjects/Keywords: Molecular biology; Oncology; Dentistry; Chemoresistance; Histone deacetylase; Histone demethylase; HNSCC; Invasion; Metastsis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chang, I. (2017). Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/6pn4c77j

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Insoon. “Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis.” 2017. Thesis, UCLA. Accessed November 19, 2017. http://www.escholarship.org/uc/item/6pn4c77j.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Insoon. “Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis.” 2017. Web. 19 Nov 2017.

Vancouver:

Chang I. Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis. [Internet] [Thesis]. UCLA; 2017. [cited 2017 Nov 19]. Available from: http://www.escholarship.org/uc/item/6pn4c77j.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang I. Epigenetic Regulation of Head and Neck Squamous Cell Carcinoma Chemoresistance, Invasion, and Metastasis. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/6pn4c77j

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Australia

28. Saran, Uttara. Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] Ovarian cancer is the fifth most commonly diagnosed cancer in women in developed countries, with epithelial ovarian carcinomas being the most prevalent type… (more)

Subjects/Keywords: Ovarian cancer; Beta-catenin; sFRPs; sFRP4; Canonical Wnt; Chemoresistance; Cisplatin; Mucinous ovarian tumours

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saran, U. (2011). Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32227&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Saran, Uttara. “Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer.” 2011. Doctoral Dissertation, University of Western Australia. Accessed November 19, 2017. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32227&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Saran, Uttara. “Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer.” 2011. Web. 19 Nov 2017.

Vancouver:

Saran U. Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2017 Nov 19]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32227&local_base=GEN01-INS01.

Council of Science Editors:

Saran U. Interaction of secreted frizzled-related proteins (sFRPs) and the canonical Wnt signalling pathway in human epithelial ovarian cancer. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32227&local_base=GEN01-INS01


University of Michigan

29. Khodadoust, Michael S. Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek.

Degree: PhD, Immunology, 2010, University of Michigan

 The human DEK gene is overexpressed in a number of malignancies, however its potential function in the context of cancer remains unknown. DEK has been… (more)

Subjects/Keywords: DEK; Melanoma; Chemoresistance; Senescence; Interchromatin Granule Clusters; Mcl-1; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Khodadoust, M. S. (2010). Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75982

Chicago Manual of Style (16th Edition):

Khodadoust, Michael S. “Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek.” 2010. Doctoral Dissertation, University of Michigan. Accessed November 19, 2017. http://hdl.handle.net/2027.42/75982.

MLA Handbook (7th Edition):

Khodadoust, Michael S. “Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek.” 2010. Web. 19 Nov 2017.

Vancouver:

Khodadoust MS. Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/2027.42/75982.

Council of Science Editors:

Khodadoust MS. Sub-Nuclear Localization and Tumorigenic Function of the Oncoprotein Dek. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75982


NSYSU

30. Yang, Ho-chun. The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2017, NSYSU

 Di(2-ethylhexyl)phthalate (DEHP), a phthalate derivative, is the most commonly used as plasticizers for industrial and consumer equipment. In recent years, Taiwan scholars had detected that… (more)

Subjects/Keywords: Di(2-ethylhexyl) phthalate; DEHP; doxorubicin; breast cancer; chemoresistance; transporter proteins; epigenetic modulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, H. (2017). The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0520117-130700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Ho-chun. “The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells.” 2017. Thesis, NSYSU. Accessed November 19, 2017. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0520117-130700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Ho-chun. “The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells.” 2017. Web. 19 Nov 2017.

Vancouver:

Yang H. The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells. [Internet] [Thesis]. NSYSU; 2017. [cited 2017 Nov 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0520117-130700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang H. The impacts of prolonged di(2-ethylhexyl)phthalate exposure on doxorubicin-induced anti-breast cancer cells. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0520117-130700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

.