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You searched for subject:(chemokines). Showing records 1 – 30 of 426 total matches.

[1] [2] [3] [4] [5] … [15]

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University of Melbourne

1. WAUGH, MICHELLE. The role of chemokines in the retina.

Degree: 2013, University of Melbourne

 This thesis investigates mice with genetic knock-outs in certain chemokines or their receptors which have shown signs of Age Related Macula Degeneration (AMD) by causing… (more)

Subjects/Keywords: chemokines; inflammation; retina

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APA (6th Edition):

WAUGH, M. (2013). The role of chemokines in the retina. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38187

Chicago Manual of Style (16th Edition):

WAUGH, MICHELLE. “The role of chemokines in the retina.” 2013. Doctoral Dissertation, University of Melbourne. Accessed April 14, 2021. http://hdl.handle.net/11343/38187.

MLA Handbook (7th Edition):

WAUGH, MICHELLE. “The role of chemokines in the retina.” 2013. Web. 14 Apr 2021.

Vancouver:

WAUGH M. The role of chemokines in the retina. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11343/38187.

Council of Science Editors:

WAUGH M. The role of chemokines in the retina. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38187


University of Aberdeen

2. Yu, Tian. Role of atypical chemokine receptor-2 in ocular inflammation.

Degree: PhD, 2015, University of Aberdeen

 The atypical chemokine receptor-2 (ACKR2) is a chemokine decoy receptor that recognises pro-inflammatory CC chemokines. Many studies showed up-regulated inflammation and delayed resolution of inflammatory… (more)

Subjects/Keywords: 617.7; Eye; Chemokines

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APA (6th Edition):

Yu, T. (2015). Role of atypical chemokine receptor-2 in ocular inflammation. (Doctoral Dissertation). University of Aberdeen. Retrieved from https://abdn.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153024900005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996

Chicago Manual of Style (16th Edition):

Yu, Tian. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed April 14, 2021. https://abdn.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153024900005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996.

MLA Handbook (7th Edition):

Yu, Tian. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Web. 14 Apr 2021.

Vancouver:

Yu T. Role of atypical chemokine receptor-2 in ocular inflammation. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2021 Apr 14]. Available from: https://abdn.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153024900005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996.

Council of Science Editors:

Yu T. Role of atypical chemokine receptor-2 in ocular inflammation. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: https://abdn.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153024900005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996


University of Hong Kong

3. Sung, Lan, Fion. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).

Degree: 1999, University of Hong Kong

Subjects/Keywords: Chemokines.; Homocysteine.

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APA (6th Edition):

Sung, Lan, F. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/56679

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sung, Lan, Fion. “Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).” 1999. Thesis, University of Hong Kong. Accessed April 14, 2021. http://hdl.handle.net/10722/56679.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sung, Lan, Fion. “Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).” 1999. Web. 14 Apr 2021.

Vancouver:

Sung, Lan F. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). [Internet] [Thesis]. University of Hong Kong; 1999. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10722/56679.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sung, Lan F. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). [Thesis]. University of Hong Kong; 1999. Available from: http://hdl.handle.net/10722/56679

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

4. Thach, Chia Tha. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.

Degree: PhD, 2013, University of Rochester

 With the increase in the production of engineered nanomaterials, researchers are discovering that there is a direct impact of these nanomaterials on the cell membrane.… (more)

Subjects/Keywords: Nanoparticles; Cellular signaling; Chemokines; Cholesterol

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APA (6th Edition):

Thach, C. T. (2013). Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26782

Chicago Manual of Style (16th Edition):

Thach, Chia Tha. “Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.” 2013. Doctoral Dissertation, University of Rochester. Accessed April 14, 2021. http://hdl.handle.net/1802/26782.

MLA Handbook (7th Edition):

Thach, Chia Tha. “Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.” 2013. Web. 14 Apr 2021.

Vancouver:

Thach CT. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1802/26782.

Council of Science Editors:

Thach CT. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26782


North Carolina State University

5. Bost, Phillip Chapman. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.

Degree: MS, Toxicology, 2009, North Carolina State University

 The purpose of this research was to investigate the potential of a therapeutic vanadium compound in the modification and regulation of common repair mechanisms associated… (more)

Subjects/Keywords: STAT; chemokines; fibrosis; vanadium

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APA (6th Edition):

Bost, P. C. (2009). Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bost, Phillip Chapman. “Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.” 2009. Thesis, North Carolina State University. Accessed April 14, 2021. http://www.lib.ncsu.edu/resolver/1840.16/851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bost, Phillip Chapman. “Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.” 2009. Web. 14 Apr 2021.

Vancouver:

Bost PC. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. [Internet] [Thesis]. North Carolina State University; 2009. [cited 2021 Apr 14]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bost PC. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. [Thesis]. North Carolina State University; 2009. Available from: http://www.lib.ncsu.edu/resolver/1840.16/851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

6. Guo, Michael. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.

Degree: Neurological Sciences, 2017, University of Vermont

  Interstitial cystitis is a serious chronic condition that causes bladder pain and increased voiding frequency in millions of adults in the US, most of… (more)

Subjects/Keywords: chemokines; cystitis; urinary bladder; inflammation

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APA (6th Edition):

Guo, M. (2017). The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Michael. “The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.” 2017. Thesis, University of Vermont. Accessed April 14, 2021. https://scholarworks.uvm.edu/hcoltheses/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Michael. “The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.” 2017. Web. 14 Apr 2021.

Vancouver:

Guo M. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. [Internet] [Thesis]. University of Vermont; 2017. [cited 2021 Apr 14]. Available from: https://scholarworks.uvm.edu/hcoltheses/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo M. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/hcoltheses/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

7. Arms, Lauren. Inflammation-Induced Plasticity of Micturition Reflex Pathways.

Degree: PhD, Neuroscience, 2011, University of Vermont

 Although a seemingly basic and simple behavior, micturition necessitates precise integration and coordination of multiple divisions of the nervous system: visceral sensory, somatic motor, sympathetic,… (more)

Subjects/Keywords: Cyclophosphamide; Cystometry; pAKT; Chemokines

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APA (6th Edition):

Arms, L. (2011). Inflammation-Induced Plasticity of Micturition Reflex Pathways. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/9

Chicago Manual of Style (16th Edition):

Arms, Lauren. “Inflammation-Induced Plasticity of Micturition Reflex Pathways.” 2011. Doctoral Dissertation, University of Vermont. Accessed April 14, 2021. https://scholarworks.uvm.edu/graddis/9.

MLA Handbook (7th Edition):

Arms, Lauren. “Inflammation-Induced Plasticity of Micturition Reflex Pathways.” 2011. Web. 14 Apr 2021.

Vancouver:

Arms L. Inflammation-Induced Plasticity of Micturition Reflex Pathways. [Internet] [Doctoral dissertation]. University of Vermont; 2011. [cited 2021 Apr 14]. Available from: https://scholarworks.uvm.edu/graddis/9.

Council of Science Editors:

Arms L. Inflammation-Induced Plasticity of Micturition Reflex Pathways. [Doctoral Dissertation]. University of Vermont; 2011. Available from: https://scholarworks.uvm.edu/graddis/9


University of California – San Francisco

8. Kelly, Lisa. EBI2 positions naïve and activated B cells.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 The immune system is organized to allow lymphocytes to survey for antigen and rapidly respond to an infection. B lymphocytes reside in B cell follicles… (more)

Subjects/Keywords: Immunology; B cells; chemokines; GPCR

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APA (6th Edition):

Kelly, L. (2011). EBI2 positions naïve and activated B cells. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0cf2v6js

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kelly, Lisa. “EBI2 positions naïve and activated B cells.” 2011. Thesis, University of California – San Francisco. Accessed April 14, 2021. http://www.escholarship.org/uc/item/0cf2v6js.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kelly, Lisa. “EBI2 positions naïve and activated B cells.” 2011. Web. 14 Apr 2021.

Vancouver:

Kelly L. EBI2 positions naïve and activated B cells. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/0cf2v6js.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kelly L. EBI2 positions naïve and activated B cells. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/0cf2v6js

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

9. Patel, Ankur S. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.

Degree: 2016, University of Illinois – Chicago

 This study investigated the role of chemokines in Cyclosporine (CsA) induced gingival overgrowth (GO). 14 patients with renal transplants undergoing CsA treatment and 3 healthy… (more)

Subjects/Keywords: gingival overgrowth; cyclosporine; chemokines

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APA (6th Edition):

Patel, A. S. (2016). The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Ankur S. “The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.” 2016. Thesis, University of Illinois – Chicago. Accessed April 14, 2021. http://hdl.handle.net/10027/21278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Ankur S. “The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.” 2016. Web. 14 Apr 2021.

Vancouver:

Patel AS. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10027/21278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel AS. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina – Greensboro

10. Paila, Hari Srinivas Kalyan. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.

Degree: 2016, University of North Carolina – Greensboro

 Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization by mass spectrometry, and phosphoproteomic methods… (more)

Subjects/Keywords: Chemokines – Receptors; Cellular signal transduction

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APA (6th Edition):

Paila, H. S. K. (2016). Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. (Doctoral Dissertation). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370

Chicago Manual of Style (16th Edition):

Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Doctoral Dissertation, University of North Carolina – Greensboro. Accessed April 14, 2021. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370.

MLA Handbook (7th Edition):

Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Web. 14 Apr 2021.

Vancouver:

Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Internet] [Doctoral dissertation]. University of North Carolina – Greensboro; 2016. [cited 2021 Apr 14]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370.

Council of Science Editors:

Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Doctoral Dissertation]. University of North Carolina – Greensboro; 2016. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370


University of Aberdeen

11. Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation.; University of Aberdeen.Development Trust. Role of atypical chemokine receptor-2 in ocular inflammation.

Degree: Dept. of Immunity, Infection and Inflammation., 2015, University of Aberdeen

Subjects/Keywords: Eye; Chemokines

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APA (6th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, I. a. I. ;. U. o. A. D. T. (2015). Role of atypical chemokine receptor-2 in ocular inflammation. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation ; University of Aberdeen Development Trust. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed April 14, 2021. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation ; University of Aberdeen Development Trust. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Web. 14 Apr 2021.

Vancouver:

Yu, Tian.; University of Aberdeen.Dept. of Immunity IaI;UoADT. Role of atypical chemokine receptor-2 in ocular inflammation. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2021 Apr 14]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer.

Council of Science Editors:

Yu, Tian.; University of Aberdeen.Dept. of Immunity IaI;UoADT. Role of atypical chemokine receptor-2 in ocular inflammation. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer


University of Southern California

12. Tam, Yvonne. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.

Degree: MS, Cranio-Facial Biology, 2012, University of Southern California

 Background: Some investigators have detected edema, swelling and histological changes in the soft tissue under pontics, while others have demonstrated that exceptional plaque control can… (more)

Subjects/Keywords: cytokines; chemokines; implants; pontics

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APA (6th Edition):

Tam, Y. (2012). Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1300

Chicago Manual of Style (16th Edition):

Tam, Yvonne. “Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.” 2012. Masters Thesis, University of Southern California. Accessed April 14, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1300.

MLA Handbook (7th Edition):

Tam, Yvonne. “Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.” 2012. Web. 14 Apr 2021.

Vancouver:

Tam Y. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2021 Apr 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1300.

Council of Science Editors:

Tam Y. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1300


University of Tennessee – Knoxville

13. Dogra, Pranay. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.

Degree: 2015, University of Tennessee – Knoxville

 Human Cytomegalovirus (HCMV) is the leading cause of both non-hereditary mental retardation and hearing loss, and CMV infection/reactivation causes serious complications in transplant and immune… (more)

Subjects/Keywords: Cytomegalovirus; Chemokines; Co-infection; Viral Chemokines; Antiviral Peptides; Other Microbiology; Virology

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APA (6th Edition):

Dogra, P. (2015). CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. (Doctoral Dissertation). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_graddiss/3497

Chicago Manual of Style (16th Edition):

Dogra, Pranay. “CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.” 2015. Doctoral Dissertation, University of Tennessee – Knoxville. Accessed April 14, 2021. https://trace.tennessee.edu/utk_graddiss/3497.

MLA Handbook (7th Edition):

Dogra, Pranay. “CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.” 2015. Web. 14 Apr 2021.

Vancouver:

Dogra P. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. [Internet] [Doctoral dissertation]. University of Tennessee – Knoxville; 2015. [cited 2021 Apr 14]. Available from: https://trace.tennessee.edu/utk_graddiss/3497.

Council of Science Editors:

Dogra P. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. [Doctoral Dissertation]. University of Tennessee – Knoxville; 2015. Available from: https://trace.tennessee.edu/utk_graddiss/3497

14. 足立, 育子. Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Previous studies have demonstrated that the activation of peroxisome proliferator-activated receptors (PPARs) not only has positive effects on permeability barrier homoeostasis but also has anti-inflammatory… (more)

Subjects/Keywords: barrier abrogation; chemokines; IL-4; PPARa

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APA (6th Edition):

足立, . (2016). Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

足立, 育子. “Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.” 2016. Thesis, Oita University / 大分大学. Accessed April 14, 2021. http://hdl.handle.net/10559/15621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

足立, 育子. “Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.” 2016. Web. 14 Apr 2021.

Vancouver:

足立 . Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10559/15621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

足立 . Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Merced

15. Tian, Wei. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.

Degree: Quantitative and Systems Biology, 2009, University of California – Merced

 Eotaxin belongs to the chemokine family of proteins, which are involved in inflammation and in the development of the immune system, regulating activation and chemotaxis… (more)

Subjects/Keywords: Biology; chemokines; eotaxin; CC-chemokine inhibitor

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APA (6th Edition):

Tian, W. (2009). Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/0fs1p96r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tian, Wei. “Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.” 2009. Thesis, University of California – Merced. Accessed April 14, 2021. http://www.escholarship.org/uc/item/0fs1p96r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tian, Wei. “Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.” 2009. Web. 14 Apr 2021.

Vancouver:

Tian W. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. [Internet] [Thesis]. University of California – Merced; 2009. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/0fs1p96r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tian W. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. [Thesis]. University of California – Merced; 2009. Available from: http://www.escholarship.org/uc/item/0fs1p96r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

16. Ellison, Stephen Patrick. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.

Degree: PhD, 2015, Temple University

Physiology

BACKGROUND: Despite aggressive dietary modification, lipid lowering medications, and other medical therapy, vascular proliferative diseases continue to account for 50% of all mortality in… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Ellison, S. P. (2015). THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253270

Chicago Manual of Style (16th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Doctoral Dissertation, Temple University. Accessed April 14, 2021. http://digital.library.temple.edu/u?/p245801coll10,253270.

MLA Handbook (7th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Web. 14 Apr 2021.

Vancouver:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2021 Apr 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270.

Council of Science Editors:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270

17. 石川, 桂二郎. Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

 Recent clinical observations have indicated that vascular endothelial growth factor (VEGF) is a key factor that stimulates the development of preretinal pathological neovascularization (NV). However,… (more)

Subjects/Keywords: angiogenesis; chemokines; inflammation; ischemic retinopathy; macrophage/microglia

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APA (6th Edition):

石川, . (2013). Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

石川, 桂二郎. “Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.” 2013. Thesis, Kyushu University / 九州大学. Accessed April 14, 2021. http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

石川, 桂二郎. “Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.” 2013. Web. 14 Apr 2021.

Vancouver:

石川 . Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

石川 . Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Emmanouil, Georgios. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.

Degree: 2018, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Colorectal cancer is the second leading etiology of cancer death in Western countries as almost half of the patients die of metastatic disease after curative… (more)

Subjects/Keywords: Αγγειογένεση όγκου; Χημειοκίνες; Angiogenens index; Chemokines

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APA (6th Edition):

Emmanouil, G. (2018). Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/43650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Emmanouil, Georgios. “Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.” 2018. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed April 14, 2021. http://hdl.handle.net/10442/hedi/43650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Emmanouil, Georgios. “Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.” 2018. Web. 14 Apr 2021.

Vancouver:

Emmanouil G. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10442/hedi/43650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Emmanouil G. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. Available from: http://hdl.handle.net/10442/hedi/43650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brigham Young University

19. Lew, Cynthia S. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.

Degree: MS, 2012, Brigham Young University

  In order to better understand the mechanism of antimicrobial chemokine activity, including binding to and killing of bacteria, random transposon mutagenesis was performed in… (more)

Subjects/Keywords: antimicrobial chemokines; Y. pseudotuberculosis; rfaD; Microbiology

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APA (6th Edition):

Lew, C. S. (2012). Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd

Chicago Manual of Style (16th Edition):

Lew, Cynthia S. “Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.” 2012. Masters Thesis, Brigham Young University. Accessed April 14, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd.

MLA Handbook (7th Edition):

Lew, Cynthia S. “Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.” 2012. Web. 14 Apr 2021.

Vancouver:

Lew CS. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. [Internet] [Masters thesis]. Brigham Young University; 2012. [cited 2021 Apr 14]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd.

Council of Science Editors:

Lew CS. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. [Masters Thesis]. Brigham Young University; 2012. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd


Université Paris-Sud – Paris XI

20. Poupel, Lucie. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.

Degree: Docteur es, Immunologie, 2013, Université Paris-Sud – Paris XI

: L’athérosclérose est une maladie inflammatoire chronique des grosses artères à localisation intimale. Elle est probablement la résultante d’une réaction inflammatoire mal contrôlée ayant pour… (more)

Subjects/Keywords: Chimiokine; Monocytes; Atherosclerose; Chemokines; Monocytes; Atherosclerosis

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APA (6th Edition):

Poupel, L. (2013). Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T032

Chicago Manual of Style (16th Edition):

Poupel, Lucie. “Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 14, 2021. http://www.theses.fr/2013PA11T032.

MLA Handbook (7th Edition):

Poupel, Lucie. “Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.” 2013. Web. 14 Apr 2021.

Vancouver:

Poupel L. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2013PA11T032.

Council of Science Editors:

Poupel L. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T032


Université Paris-Sud – Paris XI

21. Weiss, Julia Miriam. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.

Degree: Docteur es, Immunologie et biothérapies, 2011, Université Paris-Sud – Paris XI

 La myasthénie (Myasthenia Gravis) est une maladie neuromusculaire impliquant des auto-anticorps dirigés majoritairement contre le récepteur à l’acétylcholine (RACh) et entrainant une fatigabilité musculaire. Ces… (more)

Subjects/Keywords: Auto-immunité; Autoimmunity; Myasthenia Gravis; Thymus; Chemokines

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APA (6th Edition):

Weiss, J. M. (2011). Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114831

Chicago Manual of Style (16th Edition):

Weiss, Julia Miriam. “Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 14, 2021. http://www.theses.fr/2011PA114831.

MLA Handbook (7th Edition):

Weiss, Julia Miriam. “Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.” 2011. Web. 14 Apr 2021.

Vancouver:

Weiss JM. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2011PA114831.

Council of Science Editors:

Weiss JM. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114831


University of KwaZulu-Natal

22. Mdleleni, Yanga. Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA.

Degree: 2017, University of KwaZulu-Natal

 Trichinellosis is an important re-emerging parasitic zoonosis caused by nematode species of the genus Trichinella and Plasmodium falciparum malaria is among the leading causes of… (more)

Subjects/Keywords: Trichinellosis.; Plasmodium falciparum.; Malaria.; Chemokines.; Haematological profile.

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APA (6th Edition):

Mdleleni, Y. (2017). Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/17987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mdleleni, Yanga. “Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA.” 2017. Thesis, University of KwaZulu-Natal. Accessed April 14, 2021. https://researchspace.ukzn.ac.za/handle/10413/17987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mdleleni, Yanga. “Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA.” 2017. Web. 14 Apr 2021.

Vancouver:

Mdleleni Y. Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA. [Internet] [Thesis]. University of KwaZulu-Natal; 2017. [cited 2021 Apr 14]. Available from: https://researchspace.ukzn.ac.za/handle/10413/17987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mdleleni Y. Chemokines and haematological profile in Sprague-Dawley rats infected with trichinella Zimbabwensis and Plasmodium berghei ANKA. [Thesis]. University of KwaZulu-Natal; 2017. Available from: https://researchspace.ukzn.ac.za/handle/10413/17987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


George Mason University

23. Teunis, Allison Leigh. DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES .

Degree: 2017, George Mason University

 A confounding obstacle faced by the medical field is the ability of many disease agents to suppress the body’s immune system which allows them to… (more)

Subjects/Keywords: Microbiology; anthrax; chemokines; hydrogel; mice; microparticles; survival

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APA (6th Edition):

Teunis, A. L. (2017). DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES . (Thesis). George Mason University. Retrieved from http://hdl.handle.net/1920/11236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teunis, Allison Leigh. “DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES .” 2017. Thesis, George Mason University. Accessed April 14, 2021. http://hdl.handle.net/1920/11236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teunis, Allison Leigh. “DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES .” 2017. Web. 14 Apr 2021.

Vancouver:

Teunis AL. DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES . [Internet] [Thesis]. George Mason University; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1920/11236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teunis AL. DEVELOPMENT OF CHEMOKINE-RELEASING MICROPARTICLES FOR THE MANIPULATION OF IMMUNE CELL RESPONSES . [Thesis]. George Mason University; 2017. Available from: http://hdl.handle.net/1920/11236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Azzaoui, Imane. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.

Degree: Docteur es, Immunologie (Médecine), 2011, Université Lille II – Droit et Santé

 Les chimiokines sont un élément essentiel du trafic cellulaire aussi bien homéostatique que dans des situations pathologiques. Outre cette fonction chimiotactique spécifique à ce type… (more)

Subjects/Keywords: Chimiokine CCL18; Allergie; Chemokines; Allergy; Immunology

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APA (6th Edition):

Azzaoui, I. (2011). CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2011LIL2S019

Chicago Manual of Style (16th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed April 14, 2021. http://www.theses.fr/2011LIL2S019.

MLA Handbook (7th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Web. 14 Apr 2021.

Vancouver:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2011. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2011LIL2S019.

Council of Science Editors:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2011. Available from: http://www.theses.fr/2011LIL2S019

25. Williamson, Lauren Leshen. Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience .

Degree: 2014, Duke University

  The interactions between the brain and the immune system are extensive and each has a profound influence on the other. The hippocampus is a… (more)

Subjects/Keywords: Neurosciences; Psychology; chemokines; cytokines; enrichment; infection; microglia

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APA (6th Edition):

Williamson, L. L. (2014). Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/8714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williamson, Lauren Leshen. “Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience .” 2014. Thesis, Duke University. Accessed April 14, 2021. http://hdl.handle.net/10161/8714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williamson, Lauren Leshen. “Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience .” 2014. Web. 14 Apr 2021.

Vancouver:

Williamson LL. Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience . [Internet] [Thesis]. Duke University; 2014. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10161/8714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williamson LL. Neuroimmune Signaling in the Hippocampus: Mechanisms of Risk and Resilience . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/8714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Regenass, Pierre. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.

Degree: Docteur es, Chimie biologique et thérapeutique, 2015, Université de Strasbourg

Les chimiokines forment une vaste famille de cytokines chimioattractantes surtout connues pour leur implication dans des phénomènes pro-inflammatoires. Ainsi, l’obtention de composés modulant l’action de… (more)

Subjects/Keywords: Chimiokines; Neutraligands; Chemokines; Neutraligands; 547.7; 615.19

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APA (6th Edition):

Regenass, P. (2015). Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2015STRAF057

Chicago Manual of Style (16th Edition):

Regenass, Pierre. “Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.” 2015. Doctoral Dissertation, Université de Strasbourg. Accessed April 14, 2021. http://www.theses.fr/2015STRAF057.

MLA Handbook (7th Edition):

Regenass, Pierre. “Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.” 2015. Web. 14 Apr 2021.

Vancouver:

Regenass P. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2015. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2015STRAF057.

Council of Science Editors:

Regenass P. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. [Doctoral Dissertation]. Université de Strasbourg; 2015. Available from: http://www.theses.fr/2015STRAF057


University of Adelaide

27. Massy-Westropp, Madeleine Louisa. Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration.

Degree: 2020, University of Adelaide

 The mitogen-associated protein kinase (MAPK) pathway regulates cell growth and proliferation; activating mutations in constituent kinases Ras and BRAF promote excessive MAPK signalling and tumourigenesis.… (more)

Subjects/Keywords: BRAF; melanoma; colorectal cancer; tumour immunology; chemokines

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APA (6th Edition):

Massy-Westropp, M. L. (2020). Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/127699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Massy-Westropp, Madeleine Louisa. “Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration.” 2020. Thesis, University of Adelaide. Accessed April 14, 2021. http://hdl.handle.net/2440/127699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Massy-Westropp, Madeleine Louisa. “Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration.” 2020. Web. 14 Apr 2021.

Vancouver:

Massy-Westropp ML. Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration. [Internet] [Thesis]. University of Adelaide; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2440/127699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Massy-Westropp ML. Modulation of BRAFV600E Signalling in Melanoma and Colorectal Cancer: Consequences for Tumour Immune Cell Infiltration. [Thesis]. University of Adelaide; 2020. Available from: http://hdl.handle.net/2440/127699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

28. Festa, Lindsay Kathryne. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.

Degree: 2018, Drexel University

Despite the introduction of antiretroviral therapy (ART), approximately 50% of HIV+ patients still experience some degree of neurological dysfunction. The loss of dendritic spines, the… (more)

Subjects/Keywords: Pharmacology; Physiology; Adaptation (Biology); Chemokines; Dendritic cells

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APA (6th Edition):

Festa, L. K. (2018). Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A7966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Festa, Lindsay Kathryne. “Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.” 2018. Thesis, Drexel University. Accessed April 14, 2021. https://idea.library.drexel.edu/islandora/object/idea%3A7966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Festa, Lindsay Kathryne. “Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.” 2018. Web. 14 Apr 2021.

Vancouver:

Festa LK. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. [Internet] [Thesis]. Drexel University; 2018. [cited 2021 Apr 14]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A7966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Festa LK. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. [Thesis]. Drexel University; 2018. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A7966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

29. Franck, Charlotte Kristel M. Influence of Posttranslational Modifications on Peptide and Protein Activity .

Degree: 2020, University of Sydney

 Peptides and proteins represent a class of biomolecules that play a crucial role in almost all processes of life. The modification of proteins following translation… (more)

Subjects/Keywords: Tyrosine; Sulfation; Evasin; Ticks; Immunomodulatory; Chemokines

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APA (6th Edition):

Franck, C. K. M. (2020). Influence of Posttranslational Modifications on Peptide and Protein Activity . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/24287

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Franck, Charlotte Kristel M. “Influence of Posttranslational Modifications on Peptide and Protein Activity .” 2020. Thesis, University of Sydney. Accessed April 14, 2021. http://hdl.handle.net/2123/24287.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Franck, Charlotte Kristel M. “Influence of Posttranslational Modifications on Peptide and Protein Activity .” 2020. Web. 14 Apr 2021.

Vancouver:

Franck CKM. Influence of Posttranslational Modifications on Peptide and Protein Activity . [Internet] [Thesis]. University of Sydney; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2123/24287.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Franck CKM. Influence of Posttranslational Modifications on Peptide and Protein Activity . [Thesis]. University of Sydney; 2020. Available from: http://hdl.handle.net/2123/24287

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. NC DOCKS at The University of North Carolina at Greensboro; Paila, Hari Srinivas Kalyan. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.

Degree: 2016, NC Docks

 Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization by mass spectrometry, and phosphoproteomic methods… (more)

Subjects/Keywords: Chemokines $x Receptors; Cellular signal transduction

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APA (6th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Paila, H. S. K. (2016). Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/uncg/f/Paila_uncg_0154D_12120.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Thesis, NC Docks. Accessed April 14, 2021. http://libres.uncg.edu/ir/uncg/f/Paila_uncg_0154D_12120.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Web. 14 Apr 2021.

Vancouver:

NC DOCKS at The University of North Carolina at Greensboro; Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Internet] [Thesis]. NC Docks; 2016. [cited 2021 Apr 14]. Available from: http://libres.uncg.edu/ir/uncg/f/Paila_uncg_0154D_12120.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

NC DOCKS at The University of North Carolina at Greensboro; Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Thesis]. NC Docks; 2016. Available from: http://libres.uncg.edu/ir/uncg/f/Paila_uncg_0154D_12120.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [15]

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