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You searched for subject:(cell death). Showing records 1 – 30 of 875 total matches.

[1] [2] [3] [4] [5] … [30]

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University of Rochester

1. Hwangbo, Dae-Sung. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.

Degree: PhD, 2012, University of Rochester

 Apoptosis and immune responses are crucial for tissue and organismal homeostasis in metazoans. Loss of proper regulation of these processes can cause serious defects in… (more)

Subjects/Keywords: Cell Death; WD40

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APA (6th Edition):

Hwangbo, D. (2012). Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25510

Chicago Manual of Style (16th Edition):

Hwangbo, Dae-Sung. “Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.” 2012. Doctoral Dissertation, University of Rochester. Accessed July 02, 2020. http://hdl.handle.net/1802/25510.

MLA Handbook (7th Edition):

Hwangbo, Dae-Sung. “Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.” 2012. Web. 02 Jul 2020.

Vancouver:

Hwangbo D. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1802/25510.

Council of Science Editors:

Hwangbo D. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25510


University of Rochester

2. Fornarola, Laura Beth. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.

Degree: PhD, 2014, University of Rochester

 In the developing nervous system, programmed cell death (PCD) is critical for shaping and refining appropriate neuronal connections present in adulthood. Although PCD is regulated… (more)

Subjects/Keywords: Programmed cell death

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APA (6th Edition):

Fornarola, L. B. (2014). The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28436

Chicago Manual of Style (16th Edition):

Fornarola, Laura Beth. “The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.” 2014. Doctoral Dissertation, University of Rochester. Accessed July 02, 2020. http://hdl.handle.net/1802/28436.

MLA Handbook (7th Edition):

Fornarola, Laura Beth. “The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.” 2014. Web. 02 Jul 2020.

Vancouver:

Fornarola LB. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1802/28436.

Council of Science Editors:

Fornarola LB. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28436


University of Waterloo

3. Chong, Stacey. Cell death and proliferation characteristics of the retina after optic nerve section in chickens.

Degree: 2013, University of Waterloo

 Optic nerve section (ONS) is an experimental model for damage of the optic nerve associated with diseases such as glaucoma and optic neuritis. Damage to… (more)

Subjects/Keywords: proliferation; cell death

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APA (6th Edition):

Chong, S. (2013). Cell death and proliferation characteristics of the retina after optic nerve section in chickens. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/7959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chong, Stacey. “Cell death and proliferation characteristics of the retina after optic nerve section in chickens.” 2013. Thesis, University of Waterloo. Accessed July 02, 2020. http://hdl.handle.net/10012/7959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chong, Stacey. “Cell death and proliferation characteristics of the retina after optic nerve section in chickens.” 2013. Web. 02 Jul 2020.

Vancouver:

Chong S. Cell death and proliferation characteristics of the retina after optic nerve section in chickens. [Internet] [Thesis]. University of Waterloo; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10012/7959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chong S. Cell death and proliferation characteristics of the retina after optic nerve section in chickens. [Thesis]. University of Waterloo; 2013. Available from: http://hdl.handle.net/10012/7959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

4. Park, Danielle. The noninvasive imaging of cell death using a Hsp90 ligand.

Degree: Prince of Wales Medical Research Institute, 2012, University of New South Wales

Cell death plays an integral role in physiology, including turnover of cells in the gastrointestinal tract, the menstrual cycle and the immune system. Imbalance of… (more)

Subjects/Keywords: Cell death; Imaging

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APA (6th Edition):

Park, D. (2012). The noninvasive imaging of cell death using a Hsp90 ligand. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Park, Danielle. “The noninvasive imaging of cell death using a Hsp90 ligand.” 2012. Doctoral Dissertation, University of New South Wales. Accessed July 02, 2020. http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true.

MLA Handbook (7th Edition):

Park, Danielle. “The noninvasive imaging of cell death using a Hsp90 ligand.” 2012. Web. 02 Jul 2020.

Vancouver:

Park D. The noninvasive imaging of cell death using a Hsp90 ligand. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Jul 02]. Available from: http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true.

Council of Science Editors:

Park D. The noninvasive imaging of cell death using a Hsp90 ligand. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true


Hong Kong University of Science and Technology

5. Tang, Rui LIFS. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.

Degree: 2016, Hong Kong University of Science and Technology

 Anti-microtubule agents activate the spindle-assembly checkpoint by perturbing spindle formation and trapping cells in mitosis. Whether cells undergo mitotic cell death subsequently is an important… (more)

Subjects/Keywords: Cell cycle ; Mitosis ; Cell death

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APA (6th Edition):

Tang, R. L. (2016). Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Rui LIFS. “Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.” 2016. Thesis, Hong Kong University of Science and Technology. Accessed July 02, 2020. http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Rui LIFS. “Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.” 2016. Web. 02 Jul 2020.

Vancouver:

Tang RL. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2016. [cited 2020 Jul 02]. Available from: http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang RL. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. [Thesis]. Hong Kong University of Science and Technology; 2016. Available from: http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

6. Wang, Lei. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.

Degree: PhD, Human Genetics, 2010, University of Utah

 The steroid hormone ecdysone triggers the stage-specific destruction of Drosophila larval tissues through transcriptional cascades that induce two distinct forms of programmed cell death: caspase-dependent… (more)

Subjects/Keywords: Drosophila; Cell Death; Steroids

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APA (6th Edition):

Wang, L. (2010). Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526

Chicago Manual of Style (16th Edition):

Wang, Lei. “Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.” 2010. Doctoral Dissertation, University of Utah. Accessed July 02, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526.

MLA Handbook (7th Edition):

Wang, Lei. “Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.” 2010. Web. 02 Jul 2020.

Vancouver:

Wang L. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Jul 02]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526.

Council of Science Editors:

Wang L. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526


University of Saskatchewan

7. Bridge, Rylan. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.

Degree: 2016, University of Saskatchewan

 Cells of the Monocyte / Macrophage lineage are key players in innate and adaptive immunity. They eliminate pathogens through their phagocytic and antimicrobial properties, secretion… (more)

Subjects/Keywords: autophagy; cell death; kinome analysis

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APA (6th Edition):

Bridge, R. (2016). INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bridge, Rylan. “INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.” 2016. Thesis, University of Saskatchewan. Accessed July 02, 2020. http://hdl.handle.net/10388/7628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bridge, Rylan. “INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.” 2016. Web. 02 Jul 2020.

Vancouver:

Bridge R. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10388/7628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bridge R. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/7628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Purushothaman, Divya. Regulation of activated T cell death; -.

Degree: Chemical and Biotechnology, 2014, SASTRA University

Abstract included

Reference p116-134, List of publications p114-115, List of abbreviations p135-137, Summary included

Advisors/Committee Members: Sarin, Apurva.

Subjects/Keywords: Immune System; T cell death

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APA (6th Edition):

Purushothaman, D. (2014). Regulation of activated T cell death; -. (Thesis). SASTRA University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Thesis, SASTRA University. Accessed July 02, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Web. 02 Jul 2020.

Vancouver:

Purushothaman D. Regulation of activated T cell death; -. [Internet] [Thesis]. SASTRA University; 2014. [cited 2020 Jul 02]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Purushothaman D. Regulation of activated T cell death; -. [Thesis]. SASTRA University; 2014. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

9. Roche, Meghan C. A study of programmed cell death in cotton (gosypium hirsutum) fiber.

Degree: 2009, Texas A&M University

 Cotton fiber has been postulated to undergo a process of programmed cell death (PCD) during the maturation phase of development. A parallel may exist between… (more)

Subjects/Keywords: programmed cell death; cotton

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APA (6th Edition):

Roche, M. C. (2009). A study of programmed cell death in cotton (gosypium hirsutum) fiber. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roche, Meghan C. “A study of programmed cell death in cotton (gosypium hirsutum) fiber.” 2009. Thesis, Texas A&M University. Accessed July 02, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roche, Meghan C. “A study of programmed cell death in cotton (gosypium hirsutum) fiber.” 2009. Web. 02 Jul 2020.

Vancouver:

Roche MC. A study of programmed cell death in cotton (gosypium hirsutum) fiber. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roche MC. A study of programmed cell death in cotton (gosypium hirsutum) fiber. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

10. Paterson, Scott Ian. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.

Degree: PhD, 2016, University of Edinburgh

 During open orthopaedic surgical procedures, the articular cartilage covering the exposed joint surfaces can be exposed to air for prolonged periods. This exposure facilitates cartilage… (more)

Subjects/Keywords: 616.7; cartilage; drying; cell death

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APA (6th Edition):

Paterson, S. I. (2016). Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/23386

Chicago Manual of Style (16th Edition):

Paterson, Scott Ian. “Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed July 02, 2020. http://hdl.handle.net/1842/23386.

MLA Handbook (7th Edition):

Paterson, Scott Ian. “Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.” 2016. Web. 02 Jul 2020.

Vancouver:

Paterson SI. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1842/23386.

Council of Science Editors:

Paterson SI. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/23386


University of Manitoba

11. Roveimiab, Zeinab. p53 mediates cell death in the heart.

Degree: Physiology and Pathophysiology, 2019, University of Manitoba

 During cellular stress, cells try to survive and overcome the stress by an essential process called autophagy. This process involves degrading and recycling organelles and… (more)

Subjects/Keywords: p53; Cell death; Heart; Autophagy

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APA (6th Edition):

Roveimiab, Z. (2019). p53 mediates cell death in the heart. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34460

Chicago Manual of Style (16th Edition):

Roveimiab, Zeinab. “p53 mediates cell death in the heart.” 2019. Masters Thesis, University of Manitoba. Accessed July 02, 2020. http://hdl.handle.net/1993/34460.

MLA Handbook (7th Edition):

Roveimiab, Zeinab. “p53 mediates cell death in the heart.” 2019. Web. 02 Jul 2020.

Vancouver:

Roveimiab Z. p53 mediates cell death in the heart. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1993/34460.

Council of Science Editors:

Roveimiab Z. p53 mediates cell death in the heart. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34460


University of Victoria

12. Nelson, Kimberlea Lynne. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.

Degree: Department of Biochemistry and Microbiology, 2018, University of Victoria

 Aerolysin is a channel-forming protein toxin secreted by virulent Aeromonas species. The toxin binds to receptors on cells, is proteolytically activated, and then assembles into… (more)

Subjects/Keywords: Aerolysin; Bacterial toxins; Cell death

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APA (6th Edition):

Nelson, K. L. (2018). Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. (Thesis). University of Victoria. Retrieved from https://dspace.library.uvic.ca//handle/1828/9673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Kimberlea Lynne. “Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.” 2018. Thesis, University of Victoria. Accessed July 02, 2020. https://dspace.library.uvic.ca//handle/1828/9673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Kimberlea Lynne. “Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.” 2018. Web. 02 Jul 2020.

Vancouver:

Nelson KL. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. [Internet] [Thesis]. University of Victoria; 2018. [cited 2020 Jul 02]. Available from: https://dspace.library.uvic.ca//handle/1828/9673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson KL. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. [Thesis]. University of Victoria; 2018. Available from: https://dspace.library.uvic.ca//handle/1828/9673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

13. Pakos-Zebrucka, Karolina. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .

Degree: 2017, National University of Ireland – Galway

 Chronic or unresolved stress can lead to cell death, and induction of apoptosis is crucial when the cellular adaptive mechanisms are not able to resolve… (more)

Subjects/Keywords: Autophagy; Cell death; Cell stress; Apoptosis; Biochemistry

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APA (6th Edition):

Pakos-Zebrucka, K. (2017). Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/7114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pakos-Zebrucka, Karolina. “Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .” 2017. Thesis, National University of Ireland – Galway. Accessed July 02, 2020. http://hdl.handle.net/10379/7114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pakos-Zebrucka, Karolina. “Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .” 2017. Web. 02 Jul 2020.

Vancouver:

Pakos-Zebrucka K. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . [Internet] [Thesis]. National University of Ireland – Galway; 2017. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10379/7114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pakos-Zebrucka K. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . [Thesis]. National University of Ireland – Galway; 2017. Available from: http://hdl.handle.net/10379/7114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Notre Dame

14. Randy Jeffrey. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.

Degree: Biological Sciences, 2012, University of Notre Dame

  The use of anti-androgen therapy for the treatment of invasive and metastatic prostate cancer is common practice. However, after an initial response, the tumor… (more)

Subjects/Keywords: cell death; prostate cancer; cell cycle; bicalutamide

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APA (6th Edition):

Jeffrey, R. (2012). Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. (Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/qf85n873c5q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jeffrey, Randy. “Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.” 2012. Thesis, University of Notre Dame. Accessed July 02, 2020. https://curate.nd.edu/show/qf85n873c5q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jeffrey, Randy. “Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.” 2012. Web. 02 Jul 2020.

Vancouver:

Jeffrey R. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. [Internet] [Thesis]. University of Notre Dame; 2012. [cited 2020 Jul 02]. Available from: https://curate.nd.edu/show/qf85n873c5q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jeffrey R. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. [Thesis]. University of Notre Dame; 2012. Available from: https://curate.nd.edu/show/qf85n873c5q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Yang, Chenying. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.

Degree: 2017, University of Melbourne

 Salmonella enterica is a Gram-negative intracellular pathogen, which can cause typhoid fever and non-typhoidal salmonellosis. Every year ~22 million cases and ~200,000 deaths are reported… (more)

Subjects/Keywords: Salmonella; dendritic cell; cell death; IFN-γ

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, C. (2017). Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/194652

Chicago Manual of Style (16th Edition):

Yang, Chenying. “Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.” 2017. Masters Thesis, University of Melbourne. Accessed July 02, 2020. http://hdl.handle.net/11343/194652.

MLA Handbook (7th Edition):

Yang, Chenying. “Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.” 2017. Web. 02 Jul 2020.

Vancouver:

Yang C. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. [Internet] [Masters thesis]. University of Melbourne; 2017. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/11343/194652.

Council of Science Editors:

Yang C. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. [Masters Thesis]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/194652


Texas Medical Center

16. White, Erin. Mechanisms of Adenovirus-Mediated Autophagy.

Degree: PhD, 2011, Texas Medical Center

  A patient diagnosed with a glioma, generally, has an average of 14 months year to live after implementation of conventional therapies such as surgery,… (more)

Subjects/Keywords: Adenovirus; Autophagy; Autophagic Cell Death; GCN2; Cell Death; Cancer Biology; Cell Biology; Virology

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APA (6th Edition):

White, E. (2011). Mechanisms of Adenovirus-Mediated Autophagy. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/180

Chicago Manual of Style (16th Edition):

White, Erin. “Mechanisms of Adenovirus-Mediated Autophagy.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed July 02, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/180.

MLA Handbook (7th Edition):

White, Erin. “Mechanisms of Adenovirus-Mediated Autophagy.” 2011. Web. 02 Jul 2020.

Vancouver:

White E. Mechanisms of Adenovirus-Mediated Autophagy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Jul 02]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/180.

Council of Science Editors:

White E. Mechanisms of Adenovirus-Mediated Autophagy. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/180


Texas A&M University

17. Gillis, David Christopher. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.

Degree: 2015, Texas A&M University

 Inflammation in the developing central nervous system (CNS) can contribute to numerous issues in the fully developed adult. Uncovering pathways responsible for the inflammation is… (more)

Subjects/Keywords: RIP3; neuroinflammation; LPS; programmed cell death

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APA (6th Edition):

Gillis, D. C. (2015). The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Thesis, Texas A&M University. Accessed July 02, 2020. http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Web. 02 Jul 2020.

Vancouver:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

18. Avila Pacheco, Julian Ricardo, 1983-. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.

Degree: 2012, Texas A&M University

 Programmed cell death (PCD) is an active process by which organisms coordinate the controlled destruction of cells. In tomato, the protein kinase Adi3 (AvrPto-dependent Pto-interacting… (more)

Subjects/Keywords: Ubiquitination; Phosphorylation; Tomato; Programmed cell death

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APA (6th Edition):

Avila Pacheco, Julian Ricardo, 1. (2012). Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Thesis, Texas A&M University. Accessed July 02, 2020. http://hdl.handle.net/1969.1/148132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Web. 02 Jul 2020.

Vancouver:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1969.1/148132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

19. Gray, Joel W. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.

Degree: 2013, Texas A&M University

 Programmed cell death (PCD) is a fundamentally important process delicately coordinated throughout an organism?s life cycle. In plants, PCD is an integral part of development,… (more)

Subjects/Keywords: Plant biology; protein kinase; cell-death; metabolism

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APA (6th Edition):

Gray, J. W. (2013). Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gray, Joel W. “Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.” 2013. Thesis, Texas A&M University. Accessed July 02, 2020. http://hdl.handle.net/1969.1/151164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gray, Joel W. “Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.” 2013. Web. 02 Jul 2020.

Vancouver:

Gray JW. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1969.1/151164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gray JW. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Nelson Mandela Metropolitan University

20. Venables, Luanne. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.

Degree: PhD, Faculty of Science, 2014, Nelson Mandela Metropolitan University

 Artemisia afra is one of the oldest, most well known and widely used traditional medicinal plants in South Africa. It is used to treat many… (more)

Subjects/Keywords: Medicinal plants  – South Africa; Cell death

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APA (6th Edition):

Venables, L. (2014). In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. (Doctoral Dissertation). Nelson Mandela Metropolitan University. Retrieved from http://hdl.handle.net/10948/d1021087

Chicago Manual of Style (16th Edition):

Venables, Luanne. “In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.” 2014. Doctoral Dissertation, Nelson Mandela Metropolitan University. Accessed July 02, 2020. http://hdl.handle.net/10948/d1021087.

MLA Handbook (7th Edition):

Venables, Luanne. “In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.” 2014. Web. 02 Jul 2020.

Vancouver:

Venables L. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. [Internet] [Doctoral dissertation]. Nelson Mandela Metropolitan University; 2014. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10948/d1021087.

Council of Science Editors:

Venables L. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. [Doctoral Dissertation]. Nelson Mandela Metropolitan University; 2014. Available from: http://hdl.handle.net/10948/d1021087


University of Johannesburg

21. Mfouo-Tynga, Ivan Sosthene. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).

Degree: 2013, University of Johannesburg

M.Tech. (Biomedical Technology)

The uncontrolled growth of cells in the body is often associated with cancer. It constitutes a major health problem and is one… (more)

Subjects/Keywords: Phthalocyanines; Breast cancer - Photochemotherapy; Anthraquinones; Cell death

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APA (6th Edition):

Mfouo-Tynga, I. S. (2013). Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). (Thesis). University of Johannesburg. Retrieved from http://hdl.handle.net/10210/8735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mfouo-Tynga, Ivan Sosthene. “Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).” 2013. Thesis, University of Johannesburg. Accessed July 02, 2020. http://hdl.handle.net/10210/8735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mfouo-Tynga, Ivan Sosthene. “Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).” 2013. Web. 02 Jul 2020.

Vancouver:

Mfouo-Tynga IS. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). [Internet] [Thesis]. University of Johannesburg; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10210/8735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mfouo-Tynga IS. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). [Thesis]. University of Johannesburg; 2013. Available from: http://hdl.handle.net/10210/8735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

22. Vangeison, Grace Anna. Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia.

Degree: PhD, 2009, University of Rochester

 Following a stroke, rapid brain tissue loss is inevitable in the core of the infarct. However, surrounding regions, termed the penumbra, while metabolically compromised remain… (more)

Subjects/Keywords: Stroke; Hypoxia; Astrocytes; Neuronal Cell Death

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APA (6th Edition):

Vangeison, G. A. (2009). Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8296

Chicago Manual of Style (16th Edition):

Vangeison, Grace Anna. “Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia.” 2009. Doctoral Dissertation, University of Rochester. Accessed July 02, 2020. http://hdl.handle.net/1802/8296.

MLA Handbook (7th Edition):

Vangeison, Grace Anna. “Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia.” 2009. Web. 02 Jul 2020.

Vancouver:

Vangeison GA. Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1802/8296.

Council of Science Editors:

Vangeison GA. Astrocytic hypoxia inducible factor 1 alpha mediates neuronal cell death in hypoxia. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8296


University of Rochester

23. Gundemir, Soner. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.

Degree: PhD, 2011, University of Rochester

 Transglutaminase 2 (TG2) is a multifunctional enzyme that has guanine nucleotide binding and GTP hydrolyzing activity in addition to its transamidating function. Studies show that… (more)

Subjects/Keywords: Hypoxior; Transglutaminase 2; Cell Death; Ischemia

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APA (6th Edition):

Gundemir, S. (2011). Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14598

Chicago Manual of Style (16th Edition):

Gundemir, Soner. “Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.” 2011. Doctoral Dissertation, University of Rochester. Accessed July 02, 2020. http://hdl.handle.net/1802/14598.

MLA Handbook (7th Edition):

Gundemir, Soner. “Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.” 2011. Web. 02 Jul 2020.

Vancouver:

Gundemir S. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1802/14598.

Council of Science Editors:

Gundemir S. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14598


McMaster University

24. Chi, Xiaoke. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.

Degree: PhD, 2016, McMaster University

Apoptosis is a type of programmed cell death which plays a fundamental role in maintaining homeostasis in multi-cellular organisms. The Bcl-2 family has been identified… (more)

Subjects/Keywords: apoptosis; cell death; cancer; Bcl-2 family

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APA (6th Edition):

Chi, X. (2016). EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/20403

Chicago Manual of Style (16th Edition):

Chi, Xiaoke. “EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.” 2016. Doctoral Dissertation, McMaster University. Accessed July 02, 2020. http://hdl.handle.net/11375/20403.

MLA Handbook (7th Edition):

Chi, Xiaoke. “EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.” 2016. Web. 02 Jul 2020.

Vancouver:

Chi X. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. [Internet] [Doctoral dissertation]. McMaster University; 2016. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/11375/20403.

Council of Science Editors:

Chi X. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. [Doctoral Dissertation]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20403


Stellenbosch University

25. Loos, Benjamin. Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process.

Degree: Physiological Sciences, 2009, Stellenbosch University

Thesis (PhD (Physiological Sciences)) – University of Stellenbosch, 2009

ENGLISH ABSTRACT: Three main morphologies of cell death have been described in the diseased myocardium, type I,… (more)

Subjects/Keywords: Physiological sciences; Cell death; Ischemia; Anoxemia

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APA (6th Edition):

Loos, B. (2009). Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process. (Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/1173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loos, Benjamin. “Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process.” 2009. Thesis, Stellenbosch University. Accessed July 02, 2020. http://hdl.handle.net/10019.1/1173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loos, Benjamin. “Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process.” 2009. Web. 02 Jul 2020.

Vancouver:

Loos B. Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process. [Internet] [Thesis]. Stellenbosch University; 2009. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10019.1/1173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loos B. Cell death in hyppxic injury : signaling mechanisms and dynamics in the decision making process. [Thesis]. Stellenbosch University; 2009. Available from: http://hdl.handle.net/10019.1/1173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

26. Wachholz, Kristina Lora Catherine. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .

Degree: 2016, University of Ottawa

 Maternal tolerance during pregnancy increases the risk of infection with certain intracellular pathogens such as Salmonella enterica serovar Typhimurium (S.Tm). Systemic S.Tm infection during pregnancy… (more)

Subjects/Keywords: Pregnancy; Infection; Immunity; Cell Death; Inflammation; Salmonella

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APA (6th Edition):

Wachholz, K. L. C. (2016). Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wachholz, Kristina Lora Catherine. “Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .” 2016. Thesis, University of Ottawa. Accessed July 02, 2020. http://hdl.handle.net/10393/34190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wachholz, Kristina Lora Catherine. “Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .” 2016. Web. 02 Jul 2020.

Vancouver:

Wachholz KLC. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10393/34190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wachholz KLC. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Massey University

27. Hornblow, Susan Christine. Aspects of cell death and autolysis in Saccharomyces cerevisae.

Degree: MS, Microbiology, 1987, Massey University

 The kinetics of cell death and autolysis of twenty two haploid yeast strains were examined over a period of eight months in wine and synthetic… (more)

Subjects/Keywords: Cell death; Yeast

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APA (6th Edition):

Hornblow, S. C. (1987). Aspects of cell death and autolysis in Saccharomyces cerevisae. (Masters Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/14206

Chicago Manual of Style (16th Edition):

Hornblow, Susan Christine. “Aspects of cell death and autolysis in Saccharomyces cerevisae.” 1987. Masters Thesis, Massey University. Accessed July 02, 2020. http://hdl.handle.net/10179/14206.

MLA Handbook (7th Edition):

Hornblow, Susan Christine. “Aspects of cell death and autolysis in Saccharomyces cerevisae.” 1987. Web. 02 Jul 2020.

Vancouver:

Hornblow SC. Aspects of cell death and autolysis in Saccharomyces cerevisae. [Internet] [Masters thesis]. Massey University; 1987. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10179/14206.

Council of Science Editors:

Hornblow SC. Aspects of cell death and autolysis in Saccharomyces cerevisae. [Masters Thesis]. Massey University; 1987. Available from: http://hdl.handle.net/10179/14206


Deakin University

28. Yap, Yann. Deciphering the molecular signaling cascade in rotenone-mediated neuronal death.

Degree: School of Life and Environmental Sciences, 2017, Deakin University

 The study identified a potential neuroprotective protein from neurons. It might be used as a molecular target to develop therapeutics for patients with neurodegenerative diseases. Advisors/Committee Members: Cheung, Steve, Wah Chin Boon.

Subjects/Keywords: neurodegenerative disorders; antioxidant protein; programmed cell death

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APA (6th Edition):

Yap, Y. (2017). Deciphering the molecular signaling cascade in rotenone-mediated neuronal death. (Thesis). Deakin University. Retrieved from http://hdl.handle.net/10536/DRO/DU:30103745

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yap, Yann. “Deciphering the molecular signaling cascade in rotenone-mediated neuronal death.” 2017. Thesis, Deakin University. Accessed July 02, 2020. http://hdl.handle.net/10536/DRO/DU:30103745.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yap, Yann. “Deciphering the molecular signaling cascade in rotenone-mediated neuronal death.” 2017. Web. 02 Jul 2020.

Vancouver:

Yap Y. Deciphering the molecular signaling cascade in rotenone-mediated neuronal death. [Internet] [Thesis]. Deakin University; 2017. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10536/DRO/DU:30103745.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yap Y. Deciphering the molecular signaling cascade in rotenone-mediated neuronal death. [Thesis]. Deakin University; 2017. Available from: http://hdl.handle.net/10536/DRO/DU:30103745

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

29. Drakulić, Dunja R., 1979-. Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Biologija - Neurobiologija / Biology - Neurobiology

Deksametazon, jak sintetski glukokortikoid, se dugi niz godina koristi kao lek u tretmanu različitih bolesti poput psorijaze, adrenalne… (more)

Subjects/Keywords: dexamethasone; brain; cell death/survival; rat

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Drakulić, Dunja R., 1. (2014). Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:7291/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Drakulić, Dunja R., 1979-. “Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom.” 2014. Thesis, Univerzitet u Beogradu. Accessed July 02, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:7291/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Drakulić, Dunja R., 1979-. “Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom.” 2014. Web. 02 Jul 2020.

Vancouver:

Drakulić, Dunja R. 1. Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2020 Jul 02]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7291/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Drakulić, Dunja R. 1. Modulacija apoptotskih signalnih puteva u ćelijama mozga odraslih pacova nakon hroničnog tretmana deksametazonom. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7291/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

30. Viswanathan, Vasanthi. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.

Degree: 2015, Columbia University

 Ferroptosis is a novel non-apoptotic, oxidative form of regulated cell death that can be triggered by diverse small-molecule ferroptosis inducers (FINs) and genetic perturbations. Current… (more)

Subjects/Keywords: Cancer; Enzyme inhibitors; Cell death; Cytology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Viswanathan, V. (2015). Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8H9940V

Chicago Manual of Style (16th Edition):

Viswanathan, Vasanthi. “Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.” 2015. Doctoral Dissertation, Columbia University. Accessed July 02, 2020. https://doi.org/10.7916/D8H9940V.

MLA Handbook (7th Edition):

Viswanathan, Vasanthi. “Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.” 2015. Web. 02 Jul 2020.

Vancouver:

Viswanathan V. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Jul 02]. Available from: https://doi.org/10.7916/D8H9940V.

Council of Science Editors:

Viswanathan V. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8H9940V

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