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You searched for subject:(cell cycle checkpoints). Showing records 1 – 24 of 24 total matches.

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1. Krenning, L. Cell cycle checkpoints: reversible when possible, irreversible when needed.

Degree: 2015, Universiteit Utrecht

Cell cycle checkpoints are reversible in nature, and can prevent progression into the next cell cycle phase if needed. In the case of DNA damage,… (more)

Subjects/Keywords: Cell cycle checkpoints; DNA damage; Checkpoint Recovery; Cell fate; Mitosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Krenning, L. (2015). Cell cycle checkpoints: reversible when possible, irreversible when needed. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/322216

Chicago Manual of Style (16th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed April 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/322216.

MLA Handbook (7th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Web. 21 Apr 2019.

Vancouver:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Apr 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216.

Council of Science Editors:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216


University of Texas Southwestern Medical Center

2. Kim, Soonjoung. Spindle Checkpoint at Kinetochores.

Degree: 2014, University of Texas Southwestern Medical Center

 The kinetochore—a large protein assembly on centromeric chromatin—functions as the docking site for spindle microtubules and as a signaling hub for the spindle checkpoint. The… (more)

Subjects/Keywords: Cell Cycle Proteins; Kinetochores; M Phase Cell Cycle Checkpoints; Mad2 Proteins; Phosphorylation; Spindle Apparatus

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APA (6th Edition):

Kim, S. (2014). Spindle Checkpoint at Kinetochores. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Soonjoung. “Spindle Checkpoint at Kinetochores.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed April 21, 2019. http://hdl.handle.net/2152.5/3590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Soonjoung. “Spindle Checkpoint at Kinetochores.” 2014. Web. 21 Apr 2019.

Vancouver:

Kim S. Spindle Checkpoint at Kinetochores. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/2152.5/3590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim S. Spindle Checkpoint at Kinetochores. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

3. Bristow, Sara Lynn. Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator .

Degree: 2013, Duke University

  The cell division cycle is the process in which the entirety of a cell's contents is duplicated completely and then equally segregated into two… (more)

Subjects/Keywords: Genetics; Molecular biology; CDK; cell cycle; checkpoints; periodic; Saccharomyces cerevisiae; transcription

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APA (6th Edition):

Bristow, S. L. (2013). Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/8036

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bristow, Sara Lynn. “Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator .” 2013. Thesis, Duke University. Accessed April 21, 2019. http://hdl.handle.net/10161/8036.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bristow, Sara Lynn. “Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator .” 2013. Web. 21 Apr 2019.

Vancouver:

Bristow SL. Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator . [Internet] [Thesis]. Duke University; 2013. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/10161/8036.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bristow SL. Characterizing the Relationship Between Cell-Cycle Progression and a Transcriptional Oscillator . [Thesis]. Duke University; 2013. Available from: http://hdl.handle.net/10161/8036

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Keifenheim, Daniel L. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2015, U of Massachusetts : Med

  The coordination between cell growth and division is a highly regulated process that is intimately linked to the cell cycle. Efforts to identify an… (more)

Subjects/Keywords: Cell Size; Cell Cycle; Cell Cycle Checkpoints; Cell Division; Schizosaccharomyces; Cell Biology; Cellular and Molecular Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Keifenheim, D. L. (2015). Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/784

Chicago Manual of Style (16th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed April 21, 2019. http://escholarship.umassmed.edu/gsbs_diss/784.

MLA Handbook (7th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Web. 21 Apr 2019.

Vancouver:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Apr 21]. Available from: http://escholarship.umassmed.edu/gsbs_diss/784.

Council of Science Editors:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/784


Cornell University

5. Balmus, Gabriel. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo .

Degree: 2013, Cornell University

 The DNA damage response (DDR) represents the primary line of defense against exogenous and endogenous genotoxic agents that threaten the stability of our genomes. The… (more)

Subjects/Keywords: DNA damage; ATM; ATR; HUS1; cell cycle checkpoints; cancer; development; genomic instability

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APA (6th Edition):

Balmus, G. (2013). Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/33855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balmus, Gabriel. “Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo .” 2013. Thesis, Cornell University. Accessed April 21, 2019. http://hdl.handle.net/1813/33855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balmus, Gabriel. “Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo .” 2013. Web. 21 Apr 2019.

Vancouver:

Balmus G. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo . [Internet] [Thesis]. Cornell University; 2013. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/1813/33855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balmus G. Roles For The Dna Damage Checkpoint Gene Hus1 In Responding To Endogenous And Exogenous Stresses In Vivo . [Thesis]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Riverside

6. Sjogren, Caroline Alice. A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints.

Degree: Genetics, Genomics and Bioinformatics, 2016, University of California – Riverside

 Aluminum (Al) toxicity is a serious global problem that reduces crop production due to severe root growth inhibition. While Al toxic regions are considered to… (more)

Subjects/Keywords: Genetics; Plant sciences; Molecular biology; Aluminum; ATR; ATRIP/SUV2; cell cycle checkpoints; endoreduplication; SOG1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sjogren, C. A. (2016). A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/8gn3k88s

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sjogren, Caroline Alice. “A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints.” 2016. Thesis, University of California – Riverside. Accessed April 21, 2019. http://www.escholarship.org/uc/item/8gn3k88s.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sjogren, Caroline Alice. “A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints.” 2016. Web. 21 Apr 2019.

Vancouver:

Sjogren CA. A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints. [Internet] [Thesis]. University of California – Riverside; 2016. [cited 2019 Apr 21]. Available from: http://www.escholarship.org/uc/item/8gn3k88s.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sjogren CA. A Study of Aluminum Dependent Root Growth Inhibition in Arabidopsis thaliana as Mediated by DNA Damage Checkpoints. [Thesis]. University of California – Riverside; 2016. Available from: http://www.escholarship.org/uc/item/8gn3k88s

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat Autònoma de Barcelona

7. Marquina Rodríguez, María Isabel. Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7.

Degree: Departament de Bioquímica i Biologia Molecular, 2010, Universitat Autònoma de Barcelona

 Phospho/dephosphorilation in serine, threonine and tyrosine residues is one of the most widely mechanisms of protein regulation in higher organisms. These processes are strictly coordinated… (more)

Subjects/Keywords: Sacharomyces cerevisiae; Ciclo celular; Cicle cel·lular; Cell cycle; Checkpoints; Ciències Humanes; 577

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APA (6th Edition):

Marquina Rodríguez, M. I. (2010). Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/402488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marquina Rodríguez, María Isabel. “Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7.” 2010. Thesis, Universitat Autònoma de Barcelona. Accessed April 21, 2019. http://hdl.handle.net/10803/402488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marquina Rodríguez, María Isabel. “Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7.” 2010. Web. 21 Apr 2019.

Vancouver:

Marquina Rodríguez MI. Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2010. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/10803/402488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marquina Rodríguez MI. Estudio funcional de Ypi1, una subunidad reguladora esencial de la proteína fosfatasa Glc7. [Thesis]. Universitat Autònoma de Barcelona; 2010. Available from: http://hdl.handle.net/10803/402488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Jia, Luying. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.

Degree: 2015, University of Texas Southwestern Medical Center

 The spindle checkpoint is an essential mechanism to ensure accurate chromosome segregation during mitosis. The checkpoint signal originates from the kinetochore, which is a huge… (more)

Subjects/Keywords: Anaphase-Promoting Complex-Cyclosome; Cdc20 Proteins; Cell Cycle Proteins; M Phase Cell Cycle Checkpoints; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins

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APA (6th Edition):

Jia, L. (2015). The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed April 21, 2019. http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Web. 21 Apr 2019.

Vancouver:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Pope, Patricia A. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2013, U of Massachusetts : Med

  Progression through the cell cycle is tightly controlled, and the decision whether or not to enter a new cell cycle can be influenced by… (more)

Subjects/Keywords: G1 Phase Cell Cycle Checkpoints; Pheromones; Saccharomyces cerevisiae; Cell Biology; Cellular and Molecular Physiology; Molecular Biology

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APA (6th Edition):

Pope, P. A. (2013). Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/680

Chicago Manual of Style (16th Edition):

Pope, Patricia A. “Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed April 21, 2019. http://escholarship.umassmed.edu/gsbs_diss/680.

MLA Handbook (7th Edition):

Pope, Patricia A. “Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.” 2013. Web. 21 Apr 2019.

Vancouver:

Pope PA. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Apr 21]. Available from: http://escholarship.umassmed.edu/gsbs_diss/680.

Council of Science Editors:

Pope PA. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/680


Vanderbilt University

10. Spurlock III, Charles Floyd. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.

Degree: PhD, Microbiology and Immunology, 2014, Vanderbilt University

 Rheumatoid arthritis is the most common serious autoimmune disease affecting almost one percent of the human population worldwide. Methotrexate is the most commonly used disease-modifying… (more)

Subjects/Keywords: methotrexate; autoimmune disease; inflammation; rheumatoid arthritis; cell cycle checkpoints; c-Jun-N-terminal kinase; p53; tetrahydrobiopterin; long non-coding RNA

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APA (6th Edition):

Spurlock III, C. F. (2014). Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;

Chicago Manual of Style (16th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 21, 2019. http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;.

MLA Handbook (7th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Web. 21 Apr 2019.

Vancouver:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Apr 21]. Available from: http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;.

Council of Science Editors:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;


University of North Texas

11. Hajeri, Vinita A. Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans.

Degree: 2008, University of North Texas

 The soil-nematode Caenorhabditis elegans survives oxygen deprivation (anoxia < 0.001 kPa of O2, 0% O2) by entering into a state of suspended animation during which… (more)

Subjects/Keywords: oxygen deprivation; Cell cycle; checkpoints; Caenorhabditis elegans.; Anoxemia.; Cell cycle.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hajeri, V. A. (2008). Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc9776/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hajeri, Vinita A. “Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans.” 2008. Thesis, University of North Texas. Accessed April 21, 2019. https://digital.library.unt.edu/ark:/67531/metadc9776/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hajeri, Vinita A. “Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans.” 2008. Web. 21 Apr 2019.

Vancouver:

Hajeri VA. Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans. [Internet] [Thesis]. University of North Texas; 2008. [cited 2019 Apr 21]. Available from: https://digital.library.unt.edu/ark:/67531/metadc9776/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hajeri VA. Genetic and Cellular Analysis of Anoxia-Induced Cell Cycle Arrest in Caenorhabditis elegans. [Thesis]. University of North Texas; 2008. Available from: https://digital.library.unt.edu/ark:/67531/metadc9776/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Ji, Zhejian. Safeguard of Mitosis: The Spindle Checkpoint.

Degree: 2016, University of Texas Southwestern Medical Center

 In mitosis, the kinetochore-microtubule attachment is under surveillance by the spindle checkpoint to ensure the fidelity of chromosome segregation. Defects in the checkpoint could lead… (more)

Subjects/Keywords: Cell Cycle Checkpoints; Cell Cycle Proteins; Microtubule-Associated Proteins; Nuclear Proteins; Protein Processing, Post-Translational; Protein-Serine-Threonine Kinases; Protein-Tyrosine Kinases; Signal Transduction

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APA (6th Edition):

Ji, Z. (2016). Safeguard of Mitosis: The Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ji, Zhejian. “Safeguard of Mitosis: The Spindle Checkpoint.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed April 21, 2019. http://hdl.handle.net/2152.5/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ji, Zhejian. “Safeguard of Mitosis: The Spindle Checkpoint.” 2016. Web. 21 Apr 2019.

Vancouver:

Ji Z. Safeguard of Mitosis: The Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/2152.5/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ji Z. Safeguard of Mitosis: The Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Krenning, L. Cell cycle checkpoints: reversible when possible, irreversible when needed.

Degree: 2015, University Utrecht

Cell cycle checkpoints are reversible in nature, and can prevent progression into the next cell cycle phase if needed. In the case of DNA damage,… (more)

Subjects/Keywords: Cell cycle checkpoints; DNA damage; Checkpoint Recovery; Cell fate; Mitosis

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APA (6th Edition):

Krenning, L. (2015). Cell cycle checkpoints: reversible when possible, irreversible when needed. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/322216 ; URN:NBN:NL:UI:10-1874-322216 ; urn:isbn:978-90-393-6422-2 ; URN:NBN:NL:UI:10-1874-322216 ; http://dspace.library.uu.nl/handle/1874/322216

Chicago Manual of Style (16th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Doctoral Dissertation, University Utrecht. Accessed April 21, 2019. http://dspace.library.uu.nl/handle/1874/322216 ; URN:NBN:NL:UI:10-1874-322216 ; urn:isbn:978-90-393-6422-2 ; URN:NBN:NL:UI:10-1874-322216 ; http://dspace.library.uu.nl/handle/1874/322216.

MLA Handbook (7th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Web. 21 Apr 2019.

Vancouver:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Internet] [Doctoral dissertation]. University Utrecht; 2015. [cited 2019 Apr 21]. Available from: http://dspace.library.uu.nl/handle/1874/322216 ; URN:NBN:NL:UI:10-1874-322216 ; urn:isbn:978-90-393-6422-2 ; URN:NBN:NL:UI:10-1874-322216 ; http://dspace.library.uu.nl/handle/1874/322216.

Council of Science Editors:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Doctoral Dissertation]. University Utrecht; 2015. Available from: http://dspace.library.uu.nl/handle/1874/322216 ; URN:NBN:NL:UI:10-1874-322216 ; urn:isbn:978-90-393-6422-2 ; URN:NBN:NL:UI:10-1874-322216 ; http://dspace.library.uu.nl/handle/1874/322216


University of Pennsylvania

14. Demicco, Amy. Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation.

Degree: 2015, University of Pennsylvania

 Coordinated orchestration of gene expression programs at the transcriptional, post-transcriptional, and post-translational levels is essential for development and function of all cells, including lymphocytes. Normal… (more)

Subjects/Keywords: cell cycle checkpoints; cyclin D3; genome stability; HuR; lymphocyte development; p53; Allergy and Immunology; Cell Biology; Immunology and Infectious Disease; Medical Immunology; Molecular Biology

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APA (6th Edition):

Demicco, A. (2015). Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Demicco, Amy. “Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation.” 2015. Thesis, University of Pennsylvania. Accessed April 21, 2019. https://repository.upenn.edu/edissertations/1682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Demicco, Amy. “Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation.” 2015. Web. 21 Apr 2019.

Vancouver:

Demicco A. Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2019 Apr 21]. Available from: https://repository.upenn.edu/edissertations/1682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Demicco A. Context- Dependent Gene Expression Programs Promote Lymphocyte Development and Function and Suppress Transformation. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/1682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Lincoln University

15. Ling, Hong. Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach.

Degree: 2011, Lincoln University

 The control of cell cycle checkpoints in cell cycle regulation is an extremely important function in living organisms. Mutation of the checkpoint regulators can cause… (more)

Subjects/Keywords: artificial neural networks; biomarkers; cell cycle; cell cycle checkpoints; cell cycle regulation; cell division; chemical kinetic equations; cellular senescence; DNA-damage signal transduction pathway; G1/S checkpoint pathway; nonlinear ordinary differential equations; p53-Mdm2 oscillation system; parameter sensitivity analysis; recurrent neural networks; biological robustness; Type II error

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APA (6th Edition):

Ling, H. (2011). Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach. (Thesis). Lincoln University. Retrieved from http://hdl.handle.net/10182/4008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ling, Hong. “Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach.” 2011. Thesis, Lincoln University. Accessed April 21, 2019. http://hdl.handle.net/10182/4008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ling, Hong. “Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach.” 2011. Web. 21 Apr 2019.

Vancouver:

Ling H. Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach. [Internet] [Thesis]. Lincoln University; 2011. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/10182/4008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ling H. Investigation of robustness and dynamic behaviour of G1/S checkpoint/DNA-damage signal transduction pathway based on mathematical modelling and a novel neural network approach. [Thesis]. Lincoln University; 2011. Available from: http://hdl.handle.net/10182/4008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

16. Hamer, Geert. DNA double-strand breaks & poptosis in the testis.

Degree: 2003, Universiteit Utrecht

 During spermatogenesis, DNA damage is a naturally occurring event. At a certain stage, during the first meiotic prophase, DNA breaks are endogenously induced and even… (more)

Subjects/Keywords: Biologie; spermatogenesis; spermatogonia; ionizing irradiation; DNA double-strand breaks; apoptosis; cell cycle arrest; cell cycle checkpoints; meiosis; spermatocytes; meiotic recombination

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APA (6th Edition):

Hamer, G. (2003). DNA double-strand breaks & poptosis in the testis. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/236

Chicago Manual of Style (16th Edition):

Hamer, Geert. “DNA double-strand breaks & poptosis in the testis.” 2003. Doctoral Dissertation, Universiteit Utrecht. Accessed April 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/236.

MLA Handbook (7th Edition):

Hamer, Geert. “DNA double-strand breaks & poptosis in the testis.” 2003. Web. 21 Apr 2019.

Vancouver:

Hamer G. DNA double-strand breaks & poptosis in the testis. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2003. [cited 2019 Apr 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/236.

Council of Science Editors:

Hamer G. DNA double-strand breaks & poptosis in the testis. [Doctoral Dissertation]. Universiteit Utrecht; 2003. Available from: http://dspace.library.uu.nl:8080/handle/1874/236

17. Hamer, Geert. DNA double-strand breaks & poptosis in the testis.

Degree: 2003, University Utrecht

 During spermatogenesis, DNA damage is a naturally occurring event. At a certain stage, during the first meiotic prophase, DNA breaks are endogenously induced and even… (more)

Subjects/Keywords: spermatogenesis; spermatogonia; ionizing irradiation; DNA double-strand breaks; apoptosis; cell cycle arrest; cell cycle checkpoints; meiosis; spermatocytes; meiotic recombination

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APA (6th Edition):

Hamer, G. (2003). DNA double-strand breaks & poptosis in the testis. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/236 ; URN:NBN:NL:UI:10-1874-236 ; URN:NBN:NL:UI:10-1874-236 ; http://dspace.library.uu.nl/handle/1874/236

Chicago Manual of Style (16th Edition):

Hamer, Geert. “DNA double-strand breaks & poptosis in the testis.” 2003. Doctoral Dissertation, University Utrecht. Accessed April 21, 2019. http://dspace.library.uu.nl/handle/1874/236 ; URN:NBN:NL:UI:10-1874-236 ; URN:NBN:NL:UI:10-1874-236 ; http://dspace.library.uu.nl/handle/1874/236.

MLA Handbook (7th Edition):

Hamer, Geert. “DNA double-strand breaks & poptosis in the testis.” 2003. Web. 21 Apr 2019.

Vancouver:

Hamer G. DNA double-strand breaks & poptosis in the testis. [Internet] [Doctoral dissertation]. University Utrecht; 2003. [cited 2019 Apr 21]. Available from: http://dspace.library.uu.nl/handle/1874/236 ; URN:NBN:NL:UI:10-1874-236 ; URN:NBN:NL:UI:10-1874-236 ; http://dspace.library.uu.nl/handle/1874/236.

Council of Science Editors:

Hamer G. DNA double-strand breaks & poptosis in the testis. [Doctoral Dissertation]. University Utrecht; 2003. Available from: http://dspace.library.uu.nl/handle/1874/236 ; URN:NBN:NL:UI:10-1874-236 ; URN:NBN:NL:UI:10-1874-236 ; http://dspace.library.uu.nl/handle/1874/236

18. Thompson, Elizabeth Ellen Anderson. CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis.

Degree: PhD, Toxicology, 2010, North Carolina State University

 Human epidermis is routinely subjected to DNA damage induced by solar radiation and keratinocytes have developed intricate mechanisms to respond to UVB-induced DNA damage. Despite… (more)

Subjects/Keywords: squamous cell carcinoma; cell cycle checkpoints; CCAAT/Enhancer Binding Protein alpha

…activate cell cycle checkpoints (7, 8), induce permanent growth arrest, or undergo… …understand the role of the transcription factor C/EBPα in cell cycle checkpoints and skin… …x29;. Cell Cycle Checkpoints Interestingly, many of the same biochemical pathways that… …These DNA damage checkpoints result in a delay in cell cycle progression allowing time for the… …could be functioning in UVB induced cell cycle checkpoints in vivo, and 4) to determine… 

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APA (6th Edition):

Thompson, E. E. A. (2010). CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/6257

Chicago Manual of Style (16th Edition):

Thompson, Elizabeth Ellen Anderson. “CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis.” 2010. Doctoral Dissertation, North Carolina State University. Accessed April 21, 2019. http://www.lib.ncsu.edu/resolver/1840.16/6257.

MLA Handbook (7th Edition):

Thompson, Elizabeth Ellen Anderson. “CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis.” 2010. Web. 21 Apr 2019.

Vancouver:

Thompson EEA. CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis. [Internet] [Doctoral dissertation]. North Carolina State University; 2010. [cited 2019 Apr 21]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6257.

Council of Science Editors:

Thompson EEA. CCAAT/Enhancer Binding Protein Alpha in UVB Responses in Human and Mouse Skin and Mouse Skin Tumorigenesis. [Doctoral Dissertation]. North Carolina State University; 2010. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6257

19. Rodríguez-Gabriel, Miguel Angel. The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe.

Degree: 2018, Public Library of Science

Subjects/Keywords: G1 Phase Cell Cycle Checkpoints; Nitrogen; Phenotype; Schizosaccharomyces pombe Proteins; RNA-Binding Proteins; Biología y Biomedicina / Biología

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APA (6th Edition):

Rodríguez-Gabriel, M. A. (2018). The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe. (Thesis). Public Library of Science. Retrieved from http://hdl.handle.net/10486/666236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodríguez-Gabriel, Miguel Angel. “The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe.” 2018. Thesis, Public Library of Science. Accessed April 21, 2019. http://hdl.handle.net/10486/666236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodríguez-Gabriel, Miguel Angel. “The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe.” 2018. Web. 21 Apr 2019.

Vancouver:

Rodríguez-Gabriel MA. The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe. [Internet] [Thesis]. Public Library of Science; 2018. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/10486/666236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodríguez-Gabriel MA. The RNA binding protein Csx1 promotes sexual differentiation in schizosaccharomyces pombe. [Thesis]. Public Library of Science; 2018. Available from: http://hdl.handle.net/10486/666236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

20. Ragland, Ryan L. Mechanisms of Common Fragile Site Instability and Cancer.

Degree: PhD, Human Genetics, 2009, University of Michigan

 An increase in DNA damage is an “enabling characteristic” in tumorigenesis, and normally occurs at loci called common fragile sites (CFSs), or by the loss… (more)

Subjects/Keywords: Common Fragile Sites; Genomic Instability; Seckel Syndrome; ATR; Cancer; Cell Cycle Checkpoints; Genetics; Health Sciences

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APA (6th Edition):

Ragland, R. L. (2009). Mechanisms of Common Fragile Site Instability and Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/63666

Chicago Manual of Style (16th Edition):

Ragland, Ryan L. “Mechanisms of Common Fragile Site Instability and Cancer.” 2009. Doctoral Dissertation, University of Michigan. Accessed April 21, 2019. http://hdl.handle.net/2027.42/63666.

MLA Handbook (7th Edition):

Ragland, Ryan L. “Mechanisms of Common Fragile Site Instability and Cancer.” 2009. Web. 21 Apr 2019.

Vancouver:

Ragland RL. Mechanisms of Common Fragile Site Instability and Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/2027.42/63666.

Council of Science Editors:

Ragland RL. Mechanisms of Common Fragile Site Instability and Cancer. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/63666

21. Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe, Koji. Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.).

Degree: 博士(歯学), 2015, Fukuoka Dental College / 福岡歯科大学

Reactive oxygen species (ROS) can cause severe damage to DNA, proteins and lipids in normal cells, contributing to carcinogenesis and various pathological conditions. While cellular… (more)

Subjects/Keywords: Cellar senescence; Reactive oxygen species; Cyclin-dependent kinase inhibitors; Normal human epidermal keratinocyte; Biological Markers; Cell Aging; Cell Line, Tumor; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase Inhibitor p16; DNA Methylation; Epidermis; Epigenesis, Genetic; G1 Phase Cell Cycle Checkpoints; Humans; Hydrogen Peroxide; Keratinocytes; Organ Specificity; Promoter Regions, Genetic; Reactive Oxygen Species; Retinoblastoma Protein; Signal Transduction; beta-Galactosidase

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APA (6th Edition):

Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe, K. (2015). Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000033/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe, Koji. “Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.).” 2015. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed April 21, 2019. http://id.nii.ac.jp/1167/00000033/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe, Koji. “Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.).” 2015. Web. 21 Apr 2019.

Vancouver:

Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe K. Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. [cited 2019 Apr 21]. Available from: http://id.nii.ac.jp/1167/00000033/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sasaki, Mina; Kajiya, Hiroshi; Ozeki, Satoru; Okabe K. Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). : Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. Available from: http://id.nii.ac.jp/1167/00000033/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

22. Simoneau, Antoine. Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique .

Degree: 2013, Université de Montréal

 L'ADN de chaque cellule est constamment soumis à des stress pouvant compromettre son intégrité. Les bris double-brins sont probablement les dommages les plus nocifs pour… (more)

Subjects/Keywords: Complexe Mre11-Rad50-Xrs2; bris double-brins; réponse aux dommages à l'ADN; points de contrôle des dommages à l'ADN; recombinaison homologue; JENH; syndrome de Nimègue; Tel1/ATM; résection; CDK; Mre11-Rad50-Xrs2 complex; double-strand break; cell cycle; DNA damage response; DNA damage checkpoints; homologous recombination; NHEJ; Nijmegen breakage sundrome; Tel1/ATM; CDK

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APA (6th Edition):

Simoneau, A. (2013). Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simoneau, Antoine. “Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique .” 2013. Thesis, Université de Montréal. Accessed April 21, 2019. http://hdl.handle.net/1866/9229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simoneau, Antoine. “Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique .” 2013. Web. 21 Apr 2019.

Vancouver:

Simoneau A. Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/1866/9229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simoneau A. Étude du rôle de la phosphorylation du complexe Mre11-Rad50-Xrs2 dans le maintien de l'intégrité génomique . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Bozdarov, Johny. Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers.

Degree: 2011, University of Waterloo

Cell-cycle checkpoint proteins help maintain genomic integrity by sensing damaged DNA and initiating DNA repair or apoptosis. Checkpoint protein activation to cell-cycle damaging agents can… (more)

Subjects/Keywords: genotoxicity biomarker; Rad1; Hus1; rainbow trout; alternative splicing; 9-1-1 complex; aquatic toxicology; cell-cycle checkpoints

…2 Figure 1.2: A summary of the cell-cycle checkpoints: G1/S, intra-S and G2/M… …abbreviations used when explaining the cell-cycle and cell-cycle checkpoints and a list of all… …details of each checkpoint will be discussed in section 1.3. Cell-cycle checkpoints operate via… …checkpoints The cell-cycle checkpoints are important in preserving the integrity of the DNA from DNA… …8 Figure 1.2: A summary of the cell-cycle checkpoints: G1/S, intra-S and G2/M To the top… 

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APA (6th Edition):

Bozdarov, J. (2011). Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/5712

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bozdarov, Johny. “Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers.” 2011. Thesis, University of Waterloo. Accessed April 21, 2019. http://hdl.handle.net/10012/5712.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bozdarov, Johny. “Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers.” 2011. Web. 21 Apr 2019.

Vancouver:

Bozdarov J. Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers. [Internet] [Thesis]. University of Waterloo; 2011. [cited 2019 Apr 21]. Available from: http://hdl.handle.net/10012/5712.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bozdarov J. Potential use of the Oncorhynchus mykiss checkpoint proteins Rad1 and Hus1 as genotoxicity biomarkers. [Thesis]. University of Waterloo; 2011. Available from: http://hdl.handle.net/10012/5712

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Keirsey, Jeremy K. Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase.

Degree: MS, Pathology, 2012, The Ohio State University

  Bloom’s syndrome (BS) is a genetic instability disorder resulting from loss of function of the BLM helicase. It confers a marked predisposition to most… (more)

Subjects/Keywords: Biochemistry; Biology; Cellular Biology; Genetics; Health Sciences; Molecular Biology; Oncology; BLM; Phosphorylation; DNA damage; Cell-cycle checkpoints; DNA Replication Repair; CHK1; Topoisomerase; cancer Biology

…22 rDNA transcription .. ...23 Cell cycle regulation and localization of… …106 xi Replication fork binding assays …...106 Cell-cycle synchronization… …chromosomes or acentric chromosome fragments from the prior cell division (Fenech, 2007)… …BS cell extracts (Nicotera, 1991). Although the reported biochemical disturbances… …an enzyme involved in the regulation of SOD enzymes directly. Cell fusion studies… 

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APA (6th Edition):

Keirsey, J. K. (2012). Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1343348330

Chicago Manual of Style (16th Edition):

Keirsey, Jeremy K. “Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase.” 2012. Masters Thesis, The Ohio State University. Accessed April 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1343348330.

MLA Handbook (7th Edition):

Keirsey, Jeremy K. “Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase.” 2012. Web. 21 Apr 2019.

Vancouver:

Keirsey JK. Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase. [Internet] [Masters thesis]. The Ohio State University; 2012. [cited 2019 Apr 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1343348330.

Council of Science Editors:

Keirsey JK. Phosphorylation regulation of the function, localization and protein interactions of the BLM helicase. [Masters Thesis]. The Ohio State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1343348330

.