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You searched for subject:(caspase 1). Showing records 1 – 30 of 80 total matches.

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University of Alberta

1. Ramadan Elchames, Maison. An investigation of Crohn's Disease in the Oral Cavity.

Degree: MS, Medical Sciences-Oral Biology, 2016, University of Alberta

 Crohn’s disease (CD) is one of the chronic inflammatory bowel diseases (IBD) with a complex etiology involving genetic factors, priming by enteric microflora, environmental factors… (more)

Subjects/Keywords: Crohn's; oral; inflammation; caspase-1

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APA (6th Edition):

Ramadan Elchames, M. (2016). An investigation of Crohn's Disease in the Oral Cavity. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/c0k225b376

Chicago Manual of Style (16th Edition):

Ramadan Elchames, Maison. “An investigation of Crohn's Disease in the Oral Cavity.” 2016. Masters Thesis, University of Alberta. Accessed October 19, 2019. https://era.library.ualberta.ca/files/c0k225b376.

MLA Handbook (7th Edition):

Ramadan Elchames, Maison. “An investigation of Crohn's Disease in the Oral Cavity.” 2016. Web. 19 Oct 2019.

Vancouver:

Ramadan Elchames M. An investigation of Crohn's Disease in the Oral Cavity. [Internet] [Masters thesis]. University of Alberta; 2016. [cited 2019 Oct 19]. Available from: https://era.library.ualberta.ca/files/c0k225b376.

Council of Science Editors:

Ramadan Elchames M. An investigation of Crohn's Disease in the Oral Cavity. [Masters Thesis]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/c0k225b376


University of Illinois – Urbana-Champaign

2. Chiu, Gabriel. Extracellular adenosine as a novel neuroimmune modulator.

Degree: PhD, 0191, 2014, University of Illinois – Urbana-Champaign

 Adenosine is a pleotropic purine nucleoside with actions ranging from neurotransmission to immunomodulation. Pathologic processes that trigger cellular stress and damage also cause significant increases… (more)

Subjects/Keywords: Adenosine; Interleukin-1; Caspase-1; Neuroimmunology

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APA (6th Edition):

Chiu, G. (2014). Extracellular adenosine as a novel neuroimmune modulator. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/49647

Chicago Manual of Style (16th Edition):

Chiu, Gabriel. “Extracellular adenosine as a novel neuroimmune modulator.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 19, 2019. http://hdl.handle.net/2142/49647.

MLA Handbook (7th Edition):

Chiu, Gabriel. “Extracellular adenosine as a novel neuroimmune modulator.” 2014. Web. 19 Oct 2019.

Vancouver:

Chiu G. Extracellular adenosine as a novel neuroimmune modulator. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/2142/49647.

Council of Science Editors:

Chiu G. Extracellular adenosine as a novel neuroimmune modulator. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/49647


Univerzitet u Beogradu

3. Dožić, Branko S., 1968-. Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

Medicina / Medicine

Adenoidni cistični karcinom (ACC), je jedan od najčešćih malignih tumora pljuvačnih žlezda. Učestvuje sa oko 10-15% kod salivarnih neoplazmi. Karakteriše ga spor… (more)

Subjects/Keywords: adenoid cystic carcinoma; apoptosis; Apaf-1; caspase 9; caspase 3.

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APA (6th Edition):

Dožić, Branko S., 1. (2016). Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:11642/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dožić, Branko S., 1968-. “Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda.” 2016. Thesis, Univerzitet u Beogradu. Accessed October 19, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:11642/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dožić, Branko S., 1968-. “Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda.” 2016. Web. 19 Oct 2019.

Vancouver:

Dožić, Branko S. 1. Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Oct 19]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11642/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dožić, Branko S. 1. Imunohistohemijska analiza i prognostički značaj markera unutrašnjeg puta apoptoze u adenoidnom cističnom karcinomu pljuvačnih žlezda. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11642/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

4. Yin, Ying. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.

Degree: PhD, 2013, Temple University

Pharmacology

Atherosclerosis, considered a chronic inflammatory disease, is the underlying mechanism for several cardiovascular diseases. Hyperlipidemia is the number one risk factor for atherogenesis. Caspase-1(more)

Subjects/Keywords: Pharmacology; atherosclerosis, caspase-1, vascular inflammation

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APA (6th Edition):

Yin, Y. (2013). CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,252725

Chicago Manual of Style (16th Edition):

Yin, Ying. “CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.” 2013. Doctoral Dissertation, Temple University. Accessed October 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,252725.

MLA Handbook (7th Edition):

Yin, Ying. “CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.” 2013. Web. 19 Oct 2019.

Vancouver:

Yin Y. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,252725.

Council of Science Editors:

Yin Y. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,252725


University of California – Merced

5. Sater, Ali Abbas Abdul. Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth.

Degree: Quantitative and Systems Biology, 2010, University of California – Merced

 Inflammasomes have been extensively characterized in monocytes and macrophages, but not in epithelial cells, which are the preferred host cells for many pathogens. Here we… (more)

Subjects/Keywords: Biology.; Chlamydia.; Epithelial cells.; Caspase-1.

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APA (6th Edition):

Sater, A. A. A. (2010). Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/6jr544w6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sater, Ali Abbas Abdul. “Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth.” 2010. Thesis, University of California – Merced. Accessed October 19, 2019. http://www.escholarship.org/uc/item/6jr544w6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sater, Ali Abbas Abdul. “Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth.” 2010. Web. 19 Oct 2019.

Vancouver:

Sater AAA. Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth. [Internet] [Thesis]. University of California – Merced; 2010. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/6jr544w6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sater AAA. Chlamydia induced caspase-1 activation is inflammasome dependent: role and sources of reactive oxygen species in chlamydial growth. [Thesis]. University of California – Merced; 2010. Available from: http://www.escholarship.org/uc/item/6jr544w6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

6. Li, Yujin. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.

Degree: MSc, 2016, McMaster University

As a front-line treatment for cardiovascular disease, statins are among some of the most widely prescribed drugs worldwide. Statins are effective at lowering cholesterol, but… (more)

Subjects/Keywords: Inflammasome; Statins; Myopathy; Prenylation; Caspase-1; Atrogenes

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APA (6th Edition):

Li, Y. (2016). Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/20268

Chicago Manual of Style (16th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Masters Thesis, McMaster University. Accessed October 19, 2019. http://hdl.handle.net/11375/20268.

MLA Handbook (7th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Web. 19 Oct 2019.

Vancouver:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Internet] [Masters thesis]. McMaster University; 2016. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/11375/20268.

Council of Science Editors:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Masters Thesis]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20268

7. Silveira, Tatiana Nunes. Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila.

Degree: PhD, Biologia Celular e Molecular, 2010, University of São Paulo

Legionella pneumophila, o agente etiológico da doença dos Legionários, é conhecida por desencadear a formação de poro em membranas de macrófagos derivados de medula óssea… (more)

Subjects/Keywords: caspase-1; caspase-1; formação de poro; inflamassomas; inflammasome; Legionella pneumophila; Legionella pneumophila; pore formation

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APA (6th Edition):

Silveira, T. N. (2010). Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17136/tde-01062010-113434/ ;

Chicago Manual of Style (16th Edition):

Silveira, Tatiana Nunes. “Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila.” 2010. Doctoral Dissertation, University of São Paulo. Accessed October 19, 2019. http://www.teses.usp.br/teses/disponiveis/17/17136/tde-01062010-113434/ ;.

MLA Handbook (7th Edition):

Silveira, Tatiana Nunes. “Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila.” 2010. Web. 19 Oct 2019.

Vancouver:

Silveira TN. Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Oct 19]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17136/tde-01062010-113434/ ;.

Council of Science Editors:

Silveira TN. Ativação de caspase-1 e formação de poros em macrófagos infectados por Legionella pneumophila. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/17/17136/tde-01062010-113434/ ;

8. Lima, Carina Buzzo de. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.

Degree: PhD, Imunologia, 2014, University of São Paulo

O reconhecimento da flagelina é compartilhado pelo receptor transmembrânico TLR5 e citosólico NAIP5/NLRC4. Entretanto, pouco se sabe sobre os mecanismos efetores individuais induzidos a partir… (more)

Subjects/Keywords: Caspase-1; Caspase-1; Inflamassomas; Inflammasome; iNOS; iNOS; NF-kB; NF-kB; PARP-1; PARP-1

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APA (6th Edition):

Lima, C. B. d. (2014). Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;

Chicago Manual of Style (16th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Doctoral Dissertation, University of São Paulo. Accessed October 19, 2019. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

MLA Handbook (7th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Web. 19 Oct 2019.

Vancouver:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2019 Oct 19]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

Council of Science Editors:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;


McGill University

9. Shao, Wei, 1970-. Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway.

Degree: MS, Department of Biochemistry., 2007, McGill University

 Apoptosis is executed by caspase-mediated cleavage of various proteins. Elucidating the consequence of substrate cleavage provides us with insight into cell death and other biological… (more)

Subjects/Keywords: Apoptosis.; Caspase 1.; Caspase 3.; Glycolysis.

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APA (6th Edition):

Shao, Wei, 1. (2007). Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile100248.pdf

Chicago Manual of Style (16th Edition):

Shao, Wei, 1970-. “Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway.” 2007. Masters Thesis, McGill University. Accessed October 19, 2019. http://digitool.library.mcgill.ca/thesisfile100248.pdf.

MLA Handbook (7th Edition):

Shao, Wei, 1970-. “Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway.” 2007. Web. 19 Oct 2019.

Vancouver:

Shao, Wei 1. Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway. [Internet] [Masters thesis]. McGill University; 2007. [cited 2019 Oct 19]. Available from: http://digitool.library.mcgill.ca/thesisfile100248.pdf.

Council of Science Editors:

Shao, Wei 1. Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway. [Masters Thesis]. McGill University; 2007. Available from: http://digitool.library.mcgill.ca/thesisfile100248.pdf

10. Ferreira, Lucas Gonçalves. O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos.

Degree: PhD, Análises Clínicas, 2012, University of São Paulo

Escherichia coli enteroinvasora (EIEC) é um dos agentes etiológicos da disenteria bacilar. Seu processo fisiopatológico é desencadeado pela expressão de fatores de virulência, que proporcionam… (more)

Subjects/Keywords: Caspase-1; Caspase-1; Enteroinvasive Escherichia coli; Escherichia coli enteroinvasora; Flagelina; Flagellin; Inflamação; Inflammation; Macrófagos; Macrophages; Secretion system; Sistema de secreção

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APA (6th Edition):

Ferreira, L. G. (2012). O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9136/tde-13122013-091247/ ;

Chicago Manual of Style (16th Edition):

Ferreira, Lucas Gonçalves. “O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 19, 2019. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-13122013-091247/ ;.

MLA Handbook (7th Edition):

Ferreira, Lucas Gonçalves. “O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos.” 2012. Web. 19 Oct 2019.

Vancouver:

Ferreira LG. O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Oct 19]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-13122013-091247/ ;.

Council of Science Editors:

Ferreira LG. O papel da flagelina e do sistema de secreção de Escherichia coli enteroinvasora na resposta imune inata dos macrófagos. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-13122013-091247/ ;

11. 유, 종균. Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.

Degree: 2015, Ajou University

Naegleria fowleri, also known as the brain-eating amoeba, causes acute primary amoebic meningoencephalitis. During swimming and other recreational water activities, N. fowleri trophozoites penetrate the… (more)

Subjects/Keywords: Naegleriafowleri; PAM; caspase-1; inflammasome; IL-1β; NLRP3; 파울러자유아메바; 아메바성수막뇌염; 비접촉성 병인기전; 염증조절복합체; caspase-1 활성; IL-1β 생성

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APA (6th Edition):

유, . (2015). Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

유, 종균. “Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.” 2015. Thesis, Ajou University. Accessed October 19, 2019. http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

유, 종균. “Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.” 2015. Web. 19 Oct 2019.

Vancouver:

유 . Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. [Internet] [Thesis]. Ajou University; 2015. [cited 2019 Oct 19]. Available from: http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

유 . Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Juruj, Carole. Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis.

Degree: Docteur es, Infection et immunité, 2013, Université Claude Bernard – Lyon I

L'inflammasome est une voie de signalisation du système immunitaire inné impliquée dans la lutte contre les pathogènes et notamment dans la réponse aux infections bactérienne.… (more)

Subjects/Keywords: Inflammasome; AIM2; Francisella tularensis; Régulation; Caspase-1; Péroxynitrites; Inflammasome; AIM2; Francisella tularensis; Regulation; Caspase-1; Peroxynitrite; 616.079

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APA (6th Edition):

Juruj, C. (2013). Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2013LYO10074

Chicago Manual of Style (16th Edition):

Juruj, Carole. “Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis.” 2013. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed October 19, 2019. http://www.theses.fr/2013LYO10074.

MLA Handbook (7th Edition):

Juruj, Carole. “Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis.” 2013. Web. 19 Oct 2019.

Vancouver:

Juruj C. Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2013. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2013LYO10074.

Council of Science Editors:

Juruj C. Étude de la régulation de l'inflammasome AIM2 dans des macrophages infectés par Francisella tularensis : Study of the regulation of AIM2 inflammasome in macrophages infected with Francisella Tularensis. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2013. Available from: http://www.theses.fr/2013LYO10074


University of Alberta

13. Lee, Jason KH. Investigating the Caspase Cleavage of the JunB Transcription Factor.

Degree: MS, Department of Medical Microbiology and Immunology, 2013, University of Alberta

 The activation of caspases is an important step not only in apoptosis induction, but in cellular processes such as proliferation, differentiation, and the immune response.… (more)

Subjects/Keywords: JunB; Apoptosis; AP-1; ALK; Lymphoma; Caspase; ALK+ ALCL

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APA (6th Edition):

Lee, J. K. (2013). Investigating the Caspase Cleavage of the JunB Transcription Factor. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/pn89d7733

Chicago Manual of Style (16th Edition):

Lee, Jason KH. “Investigating the Caspase Cleavage of the JunB Transcription Factor.” 2013. Masters Thesis, University of Alberta. Accessed October 19, 2019. https://era.library.ualberta.ca/files/pn89d7733.

MLA Handbook (7th Edition):

Lee, Jason KH. “Investigating the Caspase Cleavage of the JunB Transcription Factor.” 2013. Web. 19 Oct 2019.

Vancouver:

Lee JK. Investigating the Caspase Cleavage of the JunB Transcription Factor. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2019 Oct 19]. Available from: https://era.library.ualberta.ca/files/pn89d7733.

Council of Science Editors:

Lee JK. Investigating the Caspase Cleavage of the JunB Transcription Factor. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/pn89d7733


NSYSU

14. Tsai, Chia-Chi. Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression.

Degree: Master, Biological Sciences, 2012, NSYSU

 Pseudomonas aeruginosa-induced pneumonia is serious problem that results in severe inflammation response and high mortality in the host. Interleukin-1β (IL-1β) is one of the major… (more)

Subjects/Keywords: MIP-2; IL-1β; Pseudomonas aeruginosa; pneumonia; caspase-1

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APA (6th Edition):

Tsai, C. (2012). Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0807112-175418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsai, Chia-Chi. “Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression.” 2012. Thesis, NSYSU. Accessed October 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0807112-175418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsai, Chia-Chi. “Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression.” 2012. Web. 19 Oct 2019.

Vancouver:

Tsai C. Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression. [Internet] [Thesis]. NSYSU; 2012. [cited 2019 Oct 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0807112-175418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsai C. Pseudomonas aeruginosa induced lung damage is through caspase-1 mediated IL-1β and MIP-2 expression. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0807112-175418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. 심, 문희. Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.

Degree: 2013, Ajou University

Naegleria fowleri, a free living amoeba widespread in moist soil, water and sediment, exist as a virulent pathogen causing fetal primary amoebic meningoencephalitis (PAM) in… (more)

Subjects/Keywords: 파울러자유아메바; inflammasome; U87MG세포; NLRP3; IL-1β; Caspase-1

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APA (6th Edition):

심, . (2013). Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Thesis, Ajou University. Accessed October 19, 2019. http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Web. 19 Oct 2019.

Vancouver:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Internet] [Thesis]. Ajou University; 2013. [cited 2019 Oct 19]. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Thesis]. Ajou University; 2013. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

16. Chavarria-Smith, Joseph Edward. Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes.

Degree: Molecular & Cell Biology, 2015, University of California – Berkeley

 Inflammasomes are cytosolic protein complexes that serve as platforms for the recruitment and activation of the pro-inflammatory CASPASE-1 protease (CASP1). CASP1 activation leads to processing… (more)

Subjects/Keywords: Molecular biology; Immunology; CASPASE-1; Inflammasome; Innate Immunology; NLR; NLRP1; Pathogen

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APA (6th Edition):

Chavarria-Smith, J. E. (2015). Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1n44p7b9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chavarria-Smith, Joseph Edward. “Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes.” 2015. Thesis, University of California – Berkeley. Accessed October 19, 2019. http://www.escholarship.org/uc/item/1n44p7b9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chavarria-Smith, Joseph Edward. “Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes.” 2015. Web. 19 Oct 2019.

Vancouver:

Chavarria-Smith JE. Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes. [Internet] [Thesis]. University of California – Berkeley; 2015. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/1n44p7b9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chavarria-Smith JE. Proteolysis is an evolutionarily conserved mechanism of activating NLRP1 inflammasomes. [Thesis]. University of California – Berkeley; 2015. Available from: http://www.escholarship.org/uc/item/1n44p7b9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade de Lisboa

17. Rodrigues, Ana Rita Cascão, 1983-. Role of IL-1β in rheumatoid arthritis.

Degree: 2012, Universidade de Lisboa

 Rheumatoid arthritis is a chronic inflammatory and immuno-mediated disease with a significant involvement of the synovial membrane of multiple small joints and progressive destruction of… (more)

Subjects/Keywords: Interleucina-1beta; Artrite reumatóide; Caspase 1; Alergia e imunologia

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APA (6th Edition):

Rodrigues, Ana Rita Cascão, 1. (2012). Role of IL-1β in rheumatoid arthritis. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Ana Rita Cascão, 1983-. “Role of IL-1β in rheumatoid arthritis.” 2012. Thesis, Universidade de Lisboa. Accessed October 19, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Ana Rita Cascão, 1983-. “Role of IL-1β in rheumatoid arthritis.” 2012. Web. 19 Oct 2019.

Vancouver:

Rodrigues, Ana Rita Cascão 1. Role of IL-1β in rheumatoid arthritis. [Internet] [Thesis]. Universidade de Lisboa; 2012. [cited 2019 Oct 19]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues, Ana Rita Cascão 1. Role of IL-1β in rheumatoid arthritis. [Thesis]. Universidade de Lisboa; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

18. von Moltke, Jakob Helmuth. Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation.

Degree: Molecular & Cell Biology, 2012, University of California – Berkeley

 To protect against infectious disease, the innate immune system must mount a rapid response that distinguishes between self and non-self, and between pathogen and harmless… (more)

Subjects/Keywords: Immunology; Biology; Microbiology; caspase-1; eicosanoid; inflammasome; leukotriene; nlrc4; prostaglandin

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APA (6th Edition):

von Moltke, J. H. (2012). Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1z80c49q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

von Moltke, Jakob Helmuth. “Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation.” 2012. Thesis, University of California – Berkeley. Accessed October 19, 2019. http://www.escholarship.org/uc/item/1z80c49q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

von Moltke, Jakob Helmuth. “Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation.” 2012. Web. 19 Oct 2019.

Vancouver:

von Moltke JH. Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation. [Internet] [Thesis]. University of California – Berkeley; 2012. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/1z80c49q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

von Moltke JH. Suicidal Cells and Lipid Storms: Mechanisms and Consequences of Cytokine–independent Inflammasome Activation. [Thesis]. University of California – Berkeley; 2012. Available from: http://www.escholarship.org/uc/item/1z80c49q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. LaRock, Christopher. Mechanisms of inflammasome activation and inhibition during Yersinia infection.

Degree: PhD, 2013, University of Washington

 The host inflammatory response is strikingly delayed during the initial stages of infection with Yersinia pestis and Y. pseudotuberculosis, pathogens that can suppress immune defenses.… (more)

Subjects/Keywords: Caspase-1; Inflammasome; Macrophage; Plague; Pyroptosis; Yersinia; Microbiology; Immunology; Biochemistry; microbiology

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APA (6th Edition):

LaRock, C. (2013). Mechanisms of inflammasome activation and inhibition during Yersinia infection. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/22552

Chicago Manual of Style (16th Edition):

LaRock, Christopher. “Mechanisms of inflammasome activation and inhibition during Yersinia infection.” 2013. Doctoral Dissertation, University of Washington. Accessed October 19, 2019. http://hdl.handle.net/1773/22552.

MLA Handbook (7th Edition):

LaRock, Christopher. “Mechanisms of inflammasome activation and inhibition during Yersinia infection.” 2013. Web. 19 Oct 2019.

Vancouver:

LaRock C. Mechanisms of inflammasome activation and inhibition during Yersinia infection. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/1773/22552.

Council of Science Editors:

LaRock C. Mechanisms of inflammasome activation and inhibition during Yersinia infection. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/22552

20. Derangère, Valentin. Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons.

Degree: Docteur es, Sciences de la vie, 2014, Université de Bourgogne

Le cancer du côlon reste un problème majeur de santé publique puisqu’il constitue en France en 2013 le deuxième cancer en termes de mortalité. La… (more)

Subjects/Keywords: Cancer colique; LXR; Oxystérols; CHIP; Pyroptose; Caspase-1; LXR; 571.6; 616.9

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APA (6th Edition):

Derangère, V. (2014). Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons. (Doctoral Dissertation). Université de Bourgogne. Retrieved from http://www.theses.fr/2014DIJOS063

Chicago Manual of Style (16th Edition):

Derangère, Valentin. “Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons.” 2014. Doctoral Dissertation, Université de Bourgogne. Accessed October 19, 2019. http://www.theses.fr/2014DIJOS063.

MLA Handbook (7th Edition):

Derangère, Valentin. “Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons.” 2014. Web. 19 Oct 2019.

Vancouver:

Derangère V. Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons. [Internet] [Doctoral dissertation]. Université de Bourgogne; 2014. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2014DIJOS063.

Council of Science Editors:

Derangère V. Rôle de la caspase-1 dans les phénomènes de mort cellulaire dans le cancer colorectal : Caspase-1 involvement in colorectal cancer cell death phenomenons. [Doctoral Dissertation]. Université de Bourgogne; 2014. Available from: http://www.theses.fr/2014DIJOS063


University of South Florida

21. Basiorka, Ashley. Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype.

Degree: 2017, University of South Florida

 Note: Portions of this abstract have been previously published in the journal Blood, Basiorka et al. Blood. 2016 Oct 13, and has been reproduced in… (more)

Subjects/Keywords: pyroptosis; caspase-1; S100A9; NADPH oxidase; β-catenin; Molecular Biology

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APA (6th Edition):

Basiorka, A. (2017). Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Basiorka, Ashley. “Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype.” 2017. Thesis, University of South Florida. Accessed October 19, 2019. https://scholarcommons.usf.edu/etd/6613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Basiorka, Ashley. “Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype.” 2017. Web. 19 Oct 2019.

Vancouver:

Basiorka A. Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype. [Internet] [Thesis]. University of South Florida; 2017. [cited 2019 Oct 19]. Available from: https://scholarcommons.usf.edu/etd/6613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Basiorka A. Changing the Pathobiological Paradigm in Myelodysplastic Syndromes: The NLRP3 Inflammasome Drives the MDS Phenotype. [Thesis]. University of South Florida; 2017. Available from: https://scholarcommons.usf.edu/etd/6613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

22. Kannan, Harsha. The Inflammasome in Acute Myocarditis.

Degree: MS, Physiology, 2013, Virginia Commonwealth University

 Acute myocarditis is an acute inflammatory syndrome characterized by myocardial damage and dysfunction often due to a viral infection followed by a variable development over… (more)

Subjects/Keywords: Inflammasome; Myocarditis; ASC; Caspase-1; Life Sciences; Physiology

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APA (6th Edition):

Kannan, H. (2013). The Inflammasome in Acute Myocarditis. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/3108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kannan, Harsha. “The Inflammasome in Acute Myocarditis.” 2013. Thesis, Virginia Commonwealth University. Accessed October 19, 2019. https://scholarscompass.vcu.edu/etd/3108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kannan, Harsha. “The Inflammasome in Acute Myocarditis.” 2013. Web. 19 Oct 2019.

Vancouver:

Kannan H. The Inflammasome in Acute Myocarditis. [Internet] [Thesis]. Virginia Commonwealth University; 2013. [cited 2019 Oct 19]. Available from: https://scholarscompass.vcu.edu/etd/3108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kannan H. The Inflammasome in Acute Myocarditis. [Thesis]. Virginia Commonwealth University; 2013. Available from: https://scholarscompass.vcu.edu/etd/3108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

23. Philip, Naomi Hannah. Caspase-8 in Cell Death and Inflammation.

Degree: 2016, University of Pennsylvania

 Pathogenic organisms express virulence factors that can inhibit immune signaling pathways. Thus, the immune system is faced with the challenge of eliciting an effective inflammatory… (more)

Subjects/Keywords: caspase-1; caspase-8; cell death; cytokines; TLR; Yersinia; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Philip, N. H. (2016). Caspase-8 in Cell Death and Inflammation. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Philip, Naomi Hannah. “Caspase-8 in Cell Death and Inflammation.” 2016. Thesis, University of Pennsylvania. Accessed October 19, 2019. https://repository.upenn.edu/edissertations/1945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Philip, Naomi Hannah. “Caspase-8 in Cell Death and Inflammation.” 2016. Web. 19 Oct 2019.

Vancouver:

Philip NH. Caspase-8 in Cell Death and Inflammation. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2019 Oct 19]. Available from: https://repository.upenn.edu/edissertations/1945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Philip NH. Caspase-8 in Cell Death and Inflammation. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/1945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

24. Trueblood, Katherine Eileen. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.

Degree: PhD, Physiology and Biophysics, 2011, Case Western Reserve University

 Diabetic retinopathy, a common complication of diabetes, is the leading cause of blindness in adults in the United States. It is known that high blood… (more)

Subjects/Keywords: Physiology; diabetic retinopathy; inflammation; caspase-1; interleukin-1-beta; NLRP3; purinergic signaling

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APA (6th Edition):

Trueblood, K. E. (2011). Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450

Chicago Manual of Style (16th Edition):

Trueblood, Katherine Eileen. “Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed October 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450.

MLA Handbook (7th Edition):

Trueblood, Katherine Eileen. “Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.” 2011. Web. 19 Oct 2019.

Vancouver:

Trueblood KE. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2019 Oct 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450.

Council of Science Editors:

Trueblood KE. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450


Uppsala University

25. Forsskåhl, Sophia Katarina. Altered expression of inflammasome components in inflammatory bowel disease.

Degree: Biology Education Centre, 2019, Uppsala University

  The inflammasome complex is a multiprotein complex that may play a role in the pathogenesis of inflammatory bowel disease (IBD) by secreting the inflammatory… (more)

Subjects/Keywords: IBD; Inflammatory Bowel Disease; Inflammasome; NLRP1; NLRP3; NLRP6; AIM2; MEFV; Pyrin; Caspase-1; Caspase-4; Caspase-5; IL-1B; IL-18; GSDMD; ASC; Inflammatorisk tarmsjukdom; Inflammasom; Immunology; Immunologi

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APA (6th Edition):

Forsskåhl, S. K. (2019). Altered expression of inflammasome components in inflammatory bowel disease. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Forsskåhl, Sophia Katarina. “Altered expression of inflammasome components in inflammatory bowel disease.” 2019. Thesis, Uppsala University. Accessed October 19, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Forsskåhl, Sophia Katarina. “Altered expression of inflammasome components in inflammatory bowel disease.” 2019. Web. 19 Oct 2019.

Vancouver:

Forsskåhl SK. Altered expression of inflammasome components in inflammatory bowel disease. [Internet] [Thesis]. Uppsala University; 2019. [cited 2019 Oct 19]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Forsskåhl SK. Altered expression of inflammasome components in inflammatory bowel disease. [Thesis]. Uppsala University; 2019. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Παπαδόπουλος, Γεώργιος. Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

INTRODUCTION - OBJECTIVES Autonomic sympathetic innervation of prostatic smooth muscles is mediated by α-adrenergic receptors. Terazosin represents a widely-used α1-adrenergic antagonist which exerts its effect… (more)

Subjects/Keywords: α-αδρενεργικοί ανταγωνιστές; Τεραζοσίνη; Κασπάση-1; Κασπάση-3; Απόπτωση; Αρουραίοι; Προστάτης; Alpha-adrenoreceptor antagonists; Terazosin; Caspase-1; Caspase-3; Apoptosis; Rats; Prostate

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APA (6th Edition):

Παπαδόπουλος, . . (2009). Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/23799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Παπαδόπουλος, Γεώργιος. “Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 19, 2019. http://hdl.handle.net/10442/hedi/23799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Παπαδόπουλος, Γεώργιος. “Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου.” 2009. Web. 19 Oct 2019.

Vancouver:

Παπαδόπουλος . Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10442/hedi/23799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Παπαδόπουλος . Η επίδραση του αποκλεισμού των α1-αδρενεργικών υποδοχέων στην έκφραση των κασπασών στον κοιλιακό προστάτη του αρουραίου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/23799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Fauteux-Daniel, Sébastien. Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes.

Degree: Docteur es, Immunologie, 2018, Lyon

 Le dysfonctionnement de l'exocytose des granules cytotoxiques est responsable d'une susceptibilité accrue aux pathogènes intracellulaires qui s'accompagne de l'activation continue et anarchique des lymphocytes cytotoxiques… (more)

Subjects/Keywords: Syndrome d’activation macrophagique; Lymphohistiocytose hémophagocytique; Auto-inflammation; Lymphocytes cytotoxiques; Perforine; Inflammasome; ASC; Caspase-1; Macrophage activation syndrome; Hemophagocytic lymphohistiocytosis; Auto-inflammatory disorders; Cytotoxic lymphocytes; Perforin; Inflammasome; ASC; Caspase-1; 571.96

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fauteux-Daniel, S. (2018). Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1052

Chicago Manual of Style (16th Edition):

Fauteux-Daniel, Sébastien. “Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes.” 2018. Doctoral Dissertation, Lyon. Accessed October 19, 2019. http://www.theses.fr/2018LYSE1052.

MLA Handbook (7th Edition):

Fauteux-Daniel, Sébastien. “Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes.” 2018. Web. 19 Oct 2019.

Vancouver:

Fauteux-Daniel S. Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2018LYSE1052.

Council of Science Editors:

Fauteux-Daniel S. Étude du rôle de l’inflammasome et de la kinase Styk1 dans la régulation des lymphocytes cytotoxiques : Role of the inflammasome and of Styk1 kinase in the regulation of cytotoxic lymphocytes. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1052

28. Madouri, Fahima. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).

Degree: Docteur es, Biologie des sciences du vivant. Immunologie, 2014, Université d'Orléans

Des études menées au laboratoire avaient démontré un rôle critique de l’inflammasome NLRP3 dans l’asthme allergique en réponse à l’ovalbumine en absence d’adjuvant. Mes travaux… (more)

Subjects/Keywords: Inflammation pulmonaire; Asthme; HDM; NLRP3; Caspase-1; IL-33; PKC-θ; ILC2; Lung inflammation; Asthma; HDM; NLRP3; Caspase-1; IL-33; PKC-θ; ILC2; 571.96

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APA (6th Edition):

Madouri, F. (2014). Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2014ORLE2055

Chicago Manual of Style (16th Edition):

Madouri, Fahima. “Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).” 2014. Doctoral Dissertation, Université d'Orléans. Accessed October 19, 2019. http://www.theses.fr/2014ORLE2055.

MLA Handbook (7th Edition):

Madouri, Fahima. “Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).” 2014. Web. 19 Oct 2019.

Vancouver:

Madouri F. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). [Internet] [Doctoral dissertation]. Université d'Orléans; 2014. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2014ORLE2055.

Council of Science Editors:

Madouri F. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). [Doctoral Dissertation]. Université d'Orléans; 2014. Available from: http://www.theses.fr/2014ORLE2055

29. Guey, Baptiste. Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression.

Degree: Docteur es, Cancérologie, 2016, Lyon

L'inflammasome est une plateforme moléculaire composée d'un récepteur de l'immunité innée tels que NLRP3 ou NLRP1b, de la protéine adaptatrice ASC et de la caspase-1.… (more)

Subjects/Keywords: Inflammasomes; NLRP1b; NLRP3; Caspase-1; Cancer; Cellules NK; Tumeur mammaire; Toxine létale; Inflammasomes; NLRP1b; NLRP3; Caspase-1; Cancer; NK cells; Mammary tumor; Lethal toxin; 616.99

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APA (6th Edition):

Guey, B. (2016). Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1113

Chicago Manual of Style (16th Edition):

Guey, Baptiste. “Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression.” 2016. Doctoral Dissertation, Lyon. Accessed October 19, 2019. http://www.theses.fr/2016LYSE1113.

MLA Handbook (7th Edition):

Guey, Baptiste. “Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression.” 2016. Web. 19 Oct 2019.

Vancouver:

Guey B. Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2016LYSE1113.

Council of Science Editors:

Guey B. Inflammasomes : des mécanismes d’activation de la caspase-1 à la progression tumorale : Inflammasomes : from caspase-1 activation mechanisms to tumor progression. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1113


Universiteit Utrecht

30. Burgh, Robert van der. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.

Degree: 2015, Universiteit Utrecht

 Autoinflammatory disease (AID) is the uncontrolled and recurrent activation of the innate immune response, followed by complete resolution of the inflammation. Patients suffer from recurring… (more)

Subjects/Keywords: Autoinflammation; mitochondria; MKD; autoinflammatory-disease; Serpinb9; isoprenoid; interleukin-1β; inflammasome; caspase-1; GTPase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Burgh, R. v. d. (2015). Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/317827

Chicago Manual of Style (16th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed October 19, 2019. http://dspace.library.uu.nl:8080/handle/1874/317827.

MLA Handbook (7th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Web. 19 Oct 2019.

Vancouver:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Oct 19]. Available from: http://dspace.library.uu.nl:8080/handle/1874/317827.

Council of Science Editors:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/317827

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