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You searched for subject:(cancer cells). Showing records 91 – 120 of 1633 total matches.

[1] [2] [3] [4] [5] [6] … [55]

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University of Louisville

91. Bodduluri, Sobha R. Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer.

Degree: PhD, 2013, University of Louisville

 Chronic inflammation promotes a variety of cancers but inflammation also plays an important role in immune surveillance of cancer. Chemokine decoy receptor, D6 scavenges large… (more)

Subjects/Keywords: Mast cells; Intestinal cancer; T cells; D6 receptor; Chemokines; Immune surveillance

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APA (6th Edition):

Bodduluri, S. R. (2013). Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/120 ; https://ir.library.louisville.edu/etd/120

Chicago Manual of Style (16th Edition):

Bodduluri, Sobha R. “Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer.” 2013. Doctoral Dissertation, University of Louisville. Accessed January 23, 2020. 10.18297/etd/120 ; https://ir.library.louisville.edu/etd/120.

MLA Handbook (7th Edition):

Bodduluri, Sobha R. “Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer.” 2013. Web. 23 Jan 2020.

Vancouver:

Bodduluri SR. Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2020 Jan 23]. Available from: 10.18297/etd/120 ; https://ir.library.louisville.edu/etd/120.

Council of Science Editors:

Bodduluri SR. Mast cell-T cell interactions in inflammatory chemokine mediated immune surveillance of intestinal cancer. [Doctoral Dissertation]. University of Louisville; 2013. Available from: 10.18297/etd/120 ; https://ir.library.louisville.edu/etd/120


NSYSU

92. Weng, Chien-hui. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.

Degree: PhD, Biological Sciences, 2013, NSYSU

 Melanoma, which is the most aggressive form of skin cancer, is notoriously resistant to current cancer therapies. Cisplatin is a first-line chemotherapy drug for melanoma.… (more)

Subjects/Keywords: cisplatin; cancer stem cells; giant cells; chemoresistance; melanoma

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APA (6th Edition):

Weng, C. (2013). Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512

Chicago Manual of Style (16th Edition):

Weng, Chien-hui. “Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.” 2013. Doctoral Dissertation, NSYSU. Accessed January 23, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512.

MLA Handbook (7th Edition):

Weng, Chien-hui. “Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo.” 2013. Web. 23 Jan 2020.

Vancouver:

Weng C. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2020 Jan 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512.

Council of Science Editors:

Weng C. Cisplatin Induces the Giant Cells Formation in Melanoma Cells in vitro and in vivo. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0517113-145512


University of Western Australia

93. Kissick, Haydn. Regulatory T-cells in murine mesothelioma.

Degree: PhD, 2010, University of Western Australia

Truncated abstract] Significant morbidity and mortality is associated with cancer. Additionally, treatments for the disease are often highly toxic and associated with adverse side affects.… (more)

Subjects/Keywords: Mesothelioma; Mesothelioma; T cells; Tumors; Cancer; Immunology; Regulatory T-cells

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APA (6th Edition):

Kissick, H. (2010). Regulatory T-cells in murine mesothelioma. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au/R/-?func=dbin-jump-full&local_base=GEN01-INS01&object_id= ; http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30016&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Kissick, Haydn. “Regulatory T-cells in murine mesothelioma.” 2010. Doctoral Dissertation, University of Western Australia. Accessed January 23, 2020. http://repository.uwa.edu.au/R/-?func=dbin-jump-full&local_base=GEN01-INS01&object_id= ; http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30016&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Kissick, Haydn. “Regulatory T-cells in murine mesothelioma.” 2010. Web. 23 Jan 2020.

Vancouver:

Kissick H. Regulatory T-cells in murine mesothelioma. [Internet] [Doctoral dissertation]. University of Western Australia; 2010. [cited 2020 Jan 23]. Available from: http://repository.uwa.edu.au/R/-?func=dbin-jump-full&local_base=GEN01-INS01&object_id= ; http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30016&local_base=GEN01-INS01.

Council of Science Editors:

Kissick H. Regulatory T-cells in murine mesothelioma. [Doctoral Dissertation]. University of Western Australia; 2010. Available from: http://repository.uwa.edu.au/R/-?func=dbin-jump-full&local_base=GEN01-INS01&object_id= ; http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30016&local_base=GEN01-INS01

94. Koshio, Jun. DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である.

Degree: 博士(医学), 2014, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3850号. 学位記番号: 新大院博(医)甲第590号. 学位授与年月日: 平成26年3月24日

Cancer Immunology, Immunotherapy. 2013, 62(10), 1619-1628

Subjects/Keywords: Tumor immunology; Helper T cells; Cancer stem cells; DDX3X

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APA (6th Edition):

Koshio, J. (2014). DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/27354

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koshio, Jun. “DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である.” 2014. Thesis, Niigata University / 新潟大学. Accessed January 23, 2020. http://hdl.handle.net/10191/27354.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koshio, Jun. “DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である.” 2014. Web. 23 Jan 2020.

Vancouver:

Koshio J. DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である. [Internet] [Thesis]. Niigata University / 新潟大学; 2014. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10191/27354.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koshio J. DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 3, X-linked is an immunogenic target of cancer stem cells : DEAD/H box polypeptide 3, X-linked(DDX3X蛋白)は癌幹細胞の免疫学的な標的である. [Thesis]. Niigata University / 新潟大学; 2014. Available from: http://hdl.handle.net/10191/27354

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bradford

95. Ahmedah, Hanadi Talal A. Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples.

Degree: PhD, 2015, University of Bradford

 The integrins play a crucial role in cancer cell proliferation, migration, differentiation, survival and angiogenesis. It has been shown that integrin expression is positively correlated… (more)

Subjects/Keywords: 616.99; Integrins; Protein expression; Migration; Tumour cells survival; Cancer cells; Immunohistochemistry

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APA (6th Edition):

Ahmedah, H. T. A. (2015). Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/14284

Chicago Manual of Style (16th Edition):

Ahmedah, Hanadi Talal A. “Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples.” 2015. Doctoral Dissertation, University of Bradford. Accessed January 23, 2020. http://hdl.handle.net/10454/14284.

MLA Handbook (7th Edition):

Ahmedah, Hanadi Talal A. “Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples.” 2015. Web. 23 Jan 2020.

Vancouver:

Ahmedah HTA. Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples. [Internet] [Doctoral dissertation]. University of Bradford; 2015. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10454/14284.

Council of Science Editors:

Ahmedah HTA. Correlation between the expression of integrins and their role in cancer progression : expression pattern of integrins αvβ3, αvβ5 and α5β1 in clinical and experimental tumour samples. [Doctoral Dissertation]. University of Bradford; 2015. Available from: http://hdl.handle.net/10454/14284


The Ohio State University

96. Scoville, Steven. Human Innate Lymphoid Cell Development.

Degree: PhD, Biomedical Sciences, 2016, The Ohio State University

 Innate lymphoid cells (ILC) were recently discovered as a novel subset of the immune system. While typical adaptive lymphocytes, such as B and T cells,… (more)

Subjects/Keywords: Immunology; cancer; innate lymphoid cells; NK cells; development

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APA (6th Edition):

Scoville, S. (2016). Human Innate Lymphoid Cell Development. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1459952541

Chicago Manual of Style (16th Edition):

Scoville, Steven. “Human Innate Lymphoid Cell Development.” 2016. Doctoral Dissertation, The Ohio State University. Accessed January 23, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1459952541.

MLA Handbook (7th Edition):

Scoville, Steven. “Human Innate Lymphoid Cell Development.” 2016. Web. 23 Jan 2020.

Vancouver:

Scoville S. Human Innate Lymphoid Cell Development. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2020 Jan 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1459952541.

Council of Science Editors:

Scoville S. Human Innate Lymphoid Cell Development. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1459952541


University of Toronto

97. Brandon, Caroline. Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells.

Degree: 2010, University of Toronto

We sought to determine whether activation of the Wnt signaling pathway altered the function of hNSCs in vitro. We took three approaches to activate Wnt… (more)

Subjects/Keywords: Wnt Signaling; Neural Stem Cells; Cancer Stem Cells; Brain Tumour; 0379

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APA (6th Edition):

Brandon, C. (2010). Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25435

Chicago Manual of Style (16th Edition):

Brandon, Caroline. “Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells.” 2010. Masters Thesis, University of Toronto. Accessed January 23, 2020. http://hdl.handle.net/1807/25435.

MLA Handbook (7th Edition):

Brandon, Caroline. “Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells.” 2010. Web. 23 Jan 2020.

Vancouver:

Brandon C. Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/1807/25435.

Council of Science Editors:

Brandon C. Wnt Signaling in Human Neural Stem Cells and Brain Tumour Stem Cells. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25435


Brunel University

98. Biggerstaff, John Patrick. The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells.

Degree: PhD, 2012, Brunel University

 Interleukin-2 activates lymphocytes to become highly cytotoxic for a wide range of tumour cell types in vitro (Iymphokine activated killer or LAK cells), and in… (more)

Subjects/Keywords: 572; Cancer; Melanoma; Tumour cells; Cytoxicity; Kymphokine activated killer (LAK) cells

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APA (6th Edition):

Biggerstaff, J. P. (2012). The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells. (Doctoral Dissertation). Brunel University. Retrieved from http://bura.brunel.ac.uk/handle/2438/6586 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295191

Chicago Manual of Style (16th Edition):

Biggerstaff, John Patrick. “The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells.” 2012. Doctoral Dissertation, Brunel University. Accessed January 23, 2020. http://bura.brunel.ac.uk/handle/2438/6586 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295191.

MLA Handbook (7th Edition):

Biggerstaff, John Patrick. “The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells.” 2012. Web. 23 Jan 2020.

Vancouver:

Biggerstaff JP. The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells. [Internet] [Doctoral dissertation]. Brunel University; 2012. [cited 2020 Jan 23]. Available from: http://bura.brunel.ac.uk/handle/2438/6586 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295191.

Council of Science Editors:

Biggerstaff JP. The role of cytokines, coagulation and fibrinolysis in leucocyte and LAK cell cytotoxicity of tumour cells. [Doctoral Dissertation]. Brunel University; 2012. Available from: http://bura.brunel.ac.uk/handle/2438/6586 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295191


University of Tasmania

99. Qu, Miaomiao. Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development.

Degree: 1995, University of Tasmania

 Application of the chemical carcinogen 7 ,12-dimethylbenz(a)anthracene (DMBA) to murine skin causes tumour development as well as damaging the skin immune system (SIS). Langerhans cells(more)

Subjects/Keywords: Langerhans cells; Cancer cells

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APA (6th Edition):

Qu, M. (1995). Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/21296/1/whole_QuMiaomiao1995_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qu, Miaomiao. “Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development.” 1995. Thesis, University of Tasmania. Accessed January 23, 2020. https://eprints.utas.edu.au/21296/1/whole_QuMiaomiao1995_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qu, Miaomiao. “Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development.” 1995. Web. 23 Jan 2020.

Vancouver:

Qu M. Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development. [Internet] [Thesis]. University of Tasmania; 1995. [cited 2020 Jan 23]. Available from: https://eprints.utas.edu.au/21296/1/whole_QuMiaomiao1995_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qu M. Immunomodulation of Langerhans cells by chemical carcinogens : implications for tumour development. [Thesis]. University of Tasmania; 1995. Available from: https://eprints.utas.edu.au/21296/1/whole_QuMiaomiao1995_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

100. Centuori, Sara Mozelle. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .

Degree: 2011, University of Arizona

 Myeloid-derived suppressor cells (MDSC) and regulatory T cells (Treg) play an essential role in the immunosuppressive networks that contribute to tumor immune evasion. The mechanisms… (more)

Subjects/Keywords: Cancer; immunosuppressive cells; MDSC; Myeloid-derived suppressor cells; Regulatory T cells; Treg

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APA (6th Edition):

Centuori, S. M. (2011). NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145099

Chicago Manual of Style (16th Edition):

Centuori, Sara Mozelle. “NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .” 2011. Doctoral Dissertation, University of Arizona. Accessed January 23, 2020. http://hdl.handle.net/10150/145099.

MLA Handbook (7th Edition):

Centuori, Sara Mozelle. “NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .” 2011. Web. 23 Jan 2020.

Vancouver:

Centuori SM. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10150/145099.

Council of Science Editors:

Centuori SM. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145099

101. Maugeri Sacca', Marcello. Targeting survival pathways in cancer stem cells.

Degree: 2012, Università degli Studi di Catania

 The discovery of tumor-initiating cells endowed with stem-like features, and therefore referred to as cancer stem cells (CSCs), has added a further level of complexity… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; breast cancer, cancer stem cells, deregulated pathways

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APA (6th Edition):

Maugeri Sacca', M. (2012). Targeting survival pathways in cancer stem cells. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/918

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maugeri Sacca', Marcello. “Targeting survival pathways in cancer stem cells.” 2012. Thesis, Università degli Studi di Catania. Accessed January 23, 2020. http://hdl.handle.net/10761/918.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maugeri Sacca', Marcello. “Targeting survival pathways in cancer stem cells.” 2012. Web. 23 Jan 2020.

Vancouver:

Maugeri Sacca' M. Targeting survival pathways in cancer stem cells. [Internet] [Thesis]. Università degli Studi di Catania; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10761/918.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maugeri Sacca' M. Targeting survival pathways in cancer stem cells. [Thesis]. Università degli Studi di Catania; 2012. Available from: http://hdl.handle.net/10761/918

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

102. De Angelis, Maria Laura. Cetuximab effect on human colon cancer stem cells.

Degree: 2014, Università degli Studi di Catania

Cancer Stem cells (CSCs), recently identified in the majority of solid tumors, are thought to drive tumor growth, giving rise to a cascade of differentiated… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; colorectal cancer, cancer stem cells, Cetuximab

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APA (6th Edition):

De Angelis, M. L. (2014). Cetuximab effect on human colon cancer stem cells. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

De Angelis, Maria Laura. “Cetuximab effect on human colon cancer stem cells.” 2014. Thesis, Università degli Studi di Catania. Accessed January 23, 2020. http://hdl.handle.net/10761/1488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

De Angelis, Maria Laura. “Cetuximab effect on human colon cancer stem cells.” 2014. Web. 23 Jan 2020.

Vancouver:

De Angelis ML. Cetuximab effect on human colon cancer stem cells. [Internet] [Thesis]. Università degli Studi di Catania; 2014. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10761/1488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

De Angelis ML. Cetuximab effect on human colon cancer stem cells. [Thesis]. Università degli Studi di Catania; 2014. Available from: http://hdl.handle.net/10761/1488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

103. Salvati, Valentina. Development of effective lung cancer therapies based on lung cancer stem cella targeting.

Degree: 2015, Università degli Studi di Catania

 Il carcinoma polmonare non a piccole cellule (NSCLC) rappresenta circa l 80% di tutti i tumori al polmone ed è il cancro più comune e… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; Cancer stem cells, EGFR phosphorylation, lung cancer

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APA (6th Edition):

Salvati, V. (2015). Development of effective lung cancer therapies based on lung cancer stem cella targeting. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/4035

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salvati, Valentina. “Development of effective lung cancer therapies based on lung cancer stem cella targeting.” 2015. Thesis, Università degli Studi di Catania. Accessed January 23, 2020. http://hdl.handle.net/10761/4035.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salvati, Valentina. “Development of effective lung cancer therapies based on lung cancer stem cella targeting.” 2015. Web. 23 Jan 2020.

Vancouver:

Salvati V. Development of effective lung cancer therapies based on lung cancer stem cella targeting. [Internet] [Thesis]. Università degli Studi di Catania; 2015. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10761/4035.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salvati V. Development of effective lung cancer therapies based on lung cancer stem cella targeting. [Thesis]. Università degli Studi di Catania; 2015. Available from: http://hdl.handle.net/10761/4035

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

104. Gomez-Chou, Sobeyda B. LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER.

Degree: PhD, 2016, Texas Medical Center

  Pancreatic ductal adenocarcinoma (PDAC) is a disease characterized by a dismal prognosis with a 5-year survival rate of 7%. A unique hallmark of this… (more)

Subjects/Keywords: pancreatic cancer; lipocalin 2; inflammation; microenvironment; stellate cells; Cancer Biology

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APA (6th Edition):

Gomez-Chou, S. B. (2016). LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/668

Chicago Manual of Style (16th Edition):

Gomez-Chou, Sobeyda B. “LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed January 23, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/668.

MLA Handbook (7th Edition):

Gomez-Chou, Sobeyda B. “LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER.” 2016. Web. 23 Jan 2020.

Vancouver:

Gomez-Chou SB. LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2020 Jan 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/668.

Council of Science Editors:

Gomez-Chou SB. LIPOCALIN 2 PROMOTES THE ESTABLISHMENT OF A PRO-TUMORIGENIC MICROENVIRONMENT IN PANCREATIC CANCER. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/668


University of Alberta

105. Ghotbi, Zahra. PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2010, University of Alberta

Cancer vaccines have shown little success in clinic. Dendritic cells (DCs) are of particular interest in cancer vaccination due to their role in cell-mediated immunity.… (more)

Subjects/Keywords: cancer vaccines; cancer immunomonitoring; dendritic cells; mannose recepror targeting; PLGA nanoparticles

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APA (6th Edition):

Ghotbi, Z. (2010). PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/2j62s604c

Chicago Manual of Style (16th Edition):

Ghotbi, Zahra. “PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring.” 2010. Masters Thesis, University of Alberta. Accessed January 23, 2020. https://era.library.ualberta.ca/files/2j62s604c.

MLA Handbook (7th Edition):

Ghotbi, Zahra. “PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring.” 2010. Web. 23 Jan 2020.

Vancouver:

Ghotbi Z. PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2020 Jan 23]. Available from: https://era.library.ualberta.ca/files/2j62s604c.

Council of Science Editors:

Ghotbi Z. PLGA-based nanoparticles for targeting of dendritic cells in cancer immunotherapy and immunomonitoring. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/2j62s604c


Durban University of Technology

106. Odayar, Kriya. Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles.

Degree: 2017, Durban University of Technology

Submitted in fulfillment of the requirements of the Degree of Master's in Biotechnology, Durban University of Technology, 2017.

Nanotechnology is explained as the science of… (more)

Subjects/Keywords: Biotechnology; Nanotechnology; Nanoparticles; Drug targeting; Cancer cells; Antineoplastic agents; Cancer – Chemotherapy

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APA (6th Edition):

Odayar, K. (2017). Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles. (Thesis). Durban University of Technology. Retrieved from http://hdl.handle.net/10321/2642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Odayar, Kriya. “Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles.” 2017. Thesis, Durban University of Technology. Accessed January 23, 2020. http://hdl.handle.net/10321/2642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Odayar, Kriya. “Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles.” 2017. Web. 23 Jan 2020.

Vancouver:

Odayar K. Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles. [Internet] [Thesis]. Durban University of Technology; 2017. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10321/2642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Odayar K. Active targeting of cancer cells using gemcitabine conjugated platinum nanoparticles. [Thesis]. Durban University of Technology; 2017. Available from: http://hdl.handle.net/10321/2642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

107. Sharrow, Allison Catherine. Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer.

Degree: 2014, Johns Hopkins University

 Eighty percent of patients with advanced ovarian cancer show an initial clinical response to therapy, but seventy-five percent of these patients eventually relapse. This transient… (more)

Subjects/Keywords: ovarian cancer; cancer stem cells; aldehyde dehydrogenase; Aldefluor

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APA (6th Edition):

Sharrow, A. C. (2014). Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharrow, Allison Catherine. “Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer.” 2014. Thesis, Johns Hopkins University. Accessed January 23, 2020. http://jhir.library.jhu.edu/handle/1774.2/37063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharrow, Allison Catherine. “Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer.” 2014. Web. 23 Jan 2020.

Vancouver:

Sharrow AC. Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2020 Jan 23]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharrow AC. Identification and Characterization of a Stem Cell-Like Population in Ovarian Cancer. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/37063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

108. Liu, Jing. Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling.

Degree: M. Phil., 2015, University of Hong Kong

Ovarian cancer is a lethal malignancy among females worldwide. A combination treatment of surgery and chemotherapy has been adopted in clinical practice, however, many patients… (more)

Subjects/Keywords: Drug resistance in cancer cells; Ovaries - Cancer - Treatment; Cisplatin

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APA (6th Edition):

Liu, J. (2015). Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. (Masters Thesis). University of Hong Kong. Retrieved from Liu, J. [柳競]. (2015). Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719474 ; http://hdl.handle.net/10722/223618

Chicago Manual of Style (16th Edition):

Liu, Jing. “Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling.” 2015. Masters Thesis, University of Hong Kong. Accessed January 23, 2020. Liu, J. [柳競]. (2015). Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719474 ; http://hdl.handle.net/10722/223618.

MLA Handbook (7th Edition):

Liu, Jing. “Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling.” 2015. Web. 23 Jan 2020.

Vancouver:

Liu J. Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2020 Jan 23]. Available from: Liu, J. [柳競]. (2015). Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719474 ; http://hdl.handle.net/10722/223618.

Council of Science Editors:

Liu J. Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. [Masters Thesis]. University of Hong Kong; 2015. Available from: Liu, J. [柳競]. (2015). Enhanced cisplatin resistance of ovarian cancer cells in three-dimensional (3D) culture system through activation of Notch1/HES1 signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719474 ; http://hdl.handle.net/10722/223618


University of Hong Kong

109. Guan, Zhong. Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer.

Degree: M. Phil., 2017, University of Hong Kong

According to the Global Cancer Statistics, breast cancer is the most commonly diagnosed cancer in women. In Hong Kong, breast cancer is the third commonly… (more)

Subjects/Keywords: Drug resistance in cancer cells; Breast - Cancer; Nitrosoamines

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APA (6th Edition):

Guan, Z. (2017). Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/244291

Chicago Manual of Style (16th Edition):

Guan, Zhong. “Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer.” 2017. Masters Thesis, University of Hong Kong. Accessed January 23, 2020. http://hdl.handle.net/10722/244291.

MLA Handbook (7th Edition):

Guan, Zhong. “Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer.” 2017. Web. 23 Jan 2020.

Vancouver:

Guan Z. Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer. [Internet] [Masters thesis]. University of Hong Kong; 2017. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10722/244291.

Council of Science Editors:

Guan Z. Investigation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced chemoresistance in breast cancer. [Masters Thesis]. University of Hong Kong; 2017. Available from: http://hdl.handle.net/10722/244291


University of Hong Kong

110. Gong, Chun. Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance.

Degree: PhD, 2016, University of Hong Kong

 Breast cancer is the most common cancer among women. Breast cancer tumorigenesis could be due to loss/mutation of BRCA1, yet the mechanisms underlying BRCA1-associated tumorigenesis… (more)

Subjects/Keywords: Pathogenesis - Cancer - Breast; Tamoxifen; Drug resistance in cancer cells

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APA (6th Edition):

Gong, C. (2016). Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/249181

Chicago Manual of Style (16th Edition):

Gong, Chun. “Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed January 23, 2020. http://hdl.handle.net/10722/249181.

MLA Handbook (7th Edition):

Gong, Chun. “Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance.” 2016. Web. 23 Jan 2020.

Vancouver:

Gong C. Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10722/249181.

Council of Science Editors:

Gong C. Characterizing novel molecular mechanisms involved in breast cancer tumourigenesis/progression and tamoxifen resistance. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/249181


University of Hong Kong

111. Yung, Ming-ho. Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance.

Degree: PhD, 2013, University of Hong Kong

 Ovarian carcinoma is one of the most leading causes of cancer death among all gynaecologic malignancies worldwide. Although there are advances in cancer treatment for… (more)

Subjects/Keywords: Momordica charantia - Therapeutic use; Ovaries - Cancer; Drug resistance in cancer cells

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APA (6th Edition):

Yung, M. (2013). Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yung, M. [容銘浩]. (2013). Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5137971 ; http://dx.doi.org/10.5353/th_b5137971 ; http://hdl.handle.net/10722/194613

Chicago Manual of Style (16th Edition):

Yung, Ming-ho. “Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed January 23, 2020. Yung, M. [容銘浩]. (2013). Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5137971 ; http://dx.doi.org/10.5353/th_b5137971 ; http://hdl.handle.net/10722/194613.

MLA Handbook (7th Edition):

Yung, Ming-ho. “Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance.” 2013. Web. 23 Jan 2020.

Vancouver:

Yung M. Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2020 Jan 23]. Available from: Yung, M. [容銘浩]. (2013). Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5137971 ; http://dx.doi.org/10.5353/th_b5137971 ; http://hdl.handle.net/10722/194613.

Council of Science Editors:

Yung M. Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Yung, M. [容銘浩]. (2013). Mechanistic and functional characterization of bitter melon extract (BME) and its bioactive component, MAP30, in combating ovarian cancer oncogenesis and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5137971 ; http://dx.doi.org/10.5353/th_b5137971 ; http://hdl.handle.net/10722/194613


University of Hong Kong

112. Xi, Sichuan. Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer.

Degree: PhD, 2000, University of Hong Kong

published_or_final_version

Physiology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Cancer - Cells.; Melatonin.; Prostate - Cancer.

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APA (6th Edition):

Xi, S. (2000). Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xi, S.. (2000). Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124249 ; http://dx.doi.org/10.5353/th_b3124249 ; http://hdl.handle.net/10722/35923

Chicago Manual of Style (16th Edition):

Xi, Sichuan. “Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer.” 2000. Doctoral Dissertation, University of Hong Kong. Accessed January 23, 2020. Xi, S.. (2000). Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124249 ; http://dx.doi.org/10.5353/th_b3124249 ; http://hdl.handle.net/10722/35923.

MLA Handbook (7th Edition):

Xi, Sichuan. “Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer.” 2000. Web. 23 Jan 2020.

Vancouver:

Xi S. Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. [Internet] [Doctoral dissertation]. University of Hong Kong; 2000. [cited 2020 Jan 23]. Available from: Xi, S.. (2000). Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124249 ; http://dx.doi.org/10.5353/th_b3124249 ; http://hdl.handle.net/10722/35923.

Council of Science Editors:

Xi S. Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. [Doctoral Dissertation]. University of Hong Kong; 2000. Available from: Xi, S.. (2000). Oncostatic actions of melatonin on tumor cell growth in the LNCaP model of human prostate cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124249 ; http://dx.doi.org/10.5353/th_b3124249 ; http://hdl.handle.net/10722/35923


University of Hong Kong

113. 王傑民; Wong, Kit-man, Sunny. Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells.

Degree: PhD, 2015, University of Hong Kong

Cancer stem cells are a subpopulation of cells needed for cancer initiation and progression. Previous works have revealed CD26-expressing colorectal cancer (CRC) stem cells are… (more)

Subjects/Keywords: Colon (Anatomy) - Cancer; Rectum - Cancer; Metastasis; Stem cells

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APA (6th Edition):

王傑民; Wong, Kit-man, S. (2015). Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wong, K. S. [王傑民]. (2015). Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435654 ; http://dx.doi.org/10.5353/th_b5435654 ; http://hdl.handle.net/10722/209489

Chicago Manual of Style (16th Edition):

王傑民; Wong, Kit-man, Sunny. “Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed January 23, 2020. Wong, K. S. [王傑民]. (2015). Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435654 ; http://dx.doi.org/10.5353/th_b5435654 ; http://hdl.handle.net/10722/209489.

MLA Handbook (7th Edition):

王傑民; Wong, Kit-man, Sunny. “Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells.” 2015. Web. 23 Jan 2020.

Vancouver:

王傑民; Wong, Kit-man S. Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2020 Jan 23]. Available from: Wong, K. S. [王傑民]. (2015). Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435654 ; http://dx.doi.org/10.5353/th_b5435654 ; http://hdl.handle.net/10722/209489.

Council of Science Editors:

王傑民; Wong, Kit-man S. Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Wong, K. S. [王傑民]. (2015). Identification of the regulatory mechanism for conferring metastasis of CD26-expressing colorectal cancer stem cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435654 ; http://dx.doi.org/10.5353/th_b5435654 ; http://hdl.handle.net/10722/209489


University of Hong Kong

114. Kala, Shashwati. Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells.

Degree: M. Phil., 2014, University of Hong Kong

Resistance to therapy by traditional chemotherapy drugs commonly seen in ovarian cancer has been a major hurdle to developing successful treatment for the disease. Such… (more)

Subjects/Keywords: Ovaries - Cancer; Drug resistance in cancer cells; Ginseng - Therapeutic use

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APA (6th Edition):

Kala, S. (2014). Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. (Masters Thesis). University of Hong Kong. Retrieved from Kala, S.. (2014). Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334847 ; http://dx.doi.org/10.5353/th_b5334847 ; http://hdl.handle.net/10722/221128

Chicago Manual of Style (16th Edition):

Kala, Shashwati. “Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells.” 2014. Masters Thesis, University of Hong Kong. Accessed January 23, 2020. Kala, S.. (2014). Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334847 ; http://dx.doi.org/10.5353/th_b5334847 ; http://hdl.handle.net/10722/221128.

MLA Handbook (7th Edition):

Kala, Shashwati. “Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells.” 2014. Web. 23 Jan 2020.

Vancouver:

Kala S. Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2020 Jan 23]. Available from: Kala, S.. (2014). Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334847 ; http://dx.doi.org/10.5353/th_b5334847 ; http://hdl.handle.net/10722/221128.

Council of Science Editors:

Kala S. Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. [Masters Thesis]. University of Hong Kong; 2014. Available from: Kala, S.. (2014). Role of ginsenoside Rb1 and its metabolite compound K in attenuating chemoresistance and tumour-initiating properties of ovarian cancer cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334847 ; http://dx.doi.org/10.5353/th_b5334847 ; http://hdl.handle.net/10722/221128


University of Hong Kong

115. 盧姵岐; Lo, Jessica. Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma.

Degree: M. Phil., 2014, University of Hong Kong

 Hepatocellular carcinoma (HCC) is often diagnosed at an advanced stage where surgery cannot provide a cure. Sorafenib is currently a new standard treatment for patients… (more)

Subjects/Keywords: CD antigens; Liver - Cancer - Treatment; Drug resistance in cancer cells

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APA (6th Edition):

盧姵岐; Lo, J. (2014). Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Lo, J. [盧姵岐]. (2014). Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5388007 ; http://dx.doi.org/10.5353/th_b5388007 ; http://hdl.handle.net/10722/222196

Chicago Manual of Style (16th Edition):

盧姵岐; Lo, Jessica. “Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma.” 2014. Masters Thesis, University of Hong Kong. Accessed January 23, 2020. Lo, J. [盧姵岐]. (2014). Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5388007 ; http://dx.doi.org/10.5353/th_b5388007 ; http://hdl.handle.net/10722/222196.

MLA Handbook (7th Edition):

盧姵岐; Lo, Jessica. “Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma.” 2014. Web. 23 Jan 2020.

Vancouver:

盧姵岐; Lo J. Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2020 Jan 23]. Available from: Lo, J. [盧姵岐]. (2014). Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5388007 ; http://dx.doi.org/10.5353/th_b5388007 ; http://hdl.handle.net/10722/222196.

Council of Science Editors:

盧姵岐; Lo J. Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2014. Available from: Lo, J. [盧姵岐]. (2014). Functional characterization and therapeutic implication of CD47 in sorafenib resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5388007 ; http://dx.doi.org/10.5353/th_b5388007 ; http://hdl.handle.net/10722/222196


University of Hong Kong

116. Lee, Derek. MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma.

Degree: Master of Medical Sciences, 2014, University of Hong Kong

 Gliomas are the commonest type of primary malignant brain tumours of the central nervous system (CNS). The highly aggressive and infiltrative characteristics of gliomas render… (more)

Subjects/Keywords: Drug resistance in cancer cells; Brain - Cancer - Chemotherapy; Gliioma

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APA (6th Edition):

Lee, D. (2014). MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. (Masters Thesis). University of Hong Kong. Retrieved from Lee, D. [李揚俊]. (2014). MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5318972 ; http://dx.doi.org/10.5353/th_b5318972 ; http://hdl.handle.net/10722/206606

Chicago Manual of Style (16th Edition):

Lee, Derek. “MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma.” 2014. Masters Thesis, University of Hong Kong. Accessed January 23, 2020. Lee, D. [李揚俊]. (2014). MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5318972 ; http://dx.doi.org/10.5353/th_b5318972 ; http://hdl.handle.net/10722/206606.

MLA Handbook (7th Edition):

Lee, Derek. “MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma.” 2014. Web. 23 Jan 2020.

Vancouver:

Lee D. MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2020 Jan 23]. Available from: Lee, D. [李揚俊]. (2014). MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5318972 ; http://dx.doi.org/10.5353/th_b5318972 ; http://hdl.handle.net/10722/206606.

Council of Science Editors:

Lee D. MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. [Masters Thesis]. University of Hong Kong; 2014. Available from: Lee, D. [李揚俊]. (2014). MicroRNA-210 and endoplasmic reticulum chaperones in the regulation of chemoresistance in glioma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5318972 ; http://dx.doi.org/10.5353/th_b5318972 ; http://hdl.handle.net/10722/206606


Nelson Mandela Metropolitan University

117. Du Plessis-Stoman, Debbie. A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines.

Degree: PhD, Faculty of Science, 2010, Nelson Mandela Metropolitan University

 This thesis mainly deals with some biochemical aspects regarding the efficacy of novel platinum anticancer compounds alone and in combination with mangiferin, as part of… (more)

Subjects/Keywords: Cancer  – Chemotherapy.; Antineoplastic Agents.; Platinum compounds  – Therapeutic use; Cancer cells.

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APA (6th Edition):

Du Plessis-Stoman, D. (2010). A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines. (Doctoral Dissertation). Nelson Mandela Metropolitan University. Retrieved from http://hdl.handle.net/10948/d1016160

Chicago Manual of Style (16th Edition):

Du Plessis-Stoman, Debbie. “A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines.” 2010. Doctoral Dissertation, Nelson Mandela Metropolitan University. Accessed January 23, 2020. http://hdl.handle.net/10948/d1016160.

MLA Handbook (7th Edition):

Du Plessis-Stoman, Debbie. “A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines.” 2010. Web. 23 Jan 2020.

Vancouver:

Du Plessis-Stoman D. A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines. [Internet] [Doctoral dissertation]. Nelson Mandela Metropolitan University; 2010. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10948/d1016160.

Council of Science Editors:

Du Plessis-Stoman D. A combination of platinum anticancer drugs and mangiferin causes increased efficacy in cancer cell lines. [Doctoral Dissertation]. Nelson Mandela Metropolitan University; 2010. Available from: http://hdl.handle.net/10948/d1016160


Rhodes University

118. Cooper, Leanne Claire. The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells.

Degree: MS, Faculty of Science, Biochemistry, Microbiology and Biotechnology, 2011, Rhodes University

 Breast cancer is one of the most common forms of cancer in not only South African women, but women all over the world. The molecular… (more)

Subjects/Keywords: Cancer  – Treatment; Heat shock proteins; Cancer cells; Molecular chaperones

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APA (6th Edition):

Cooper, L. C. (2011). The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells. (Masters Thesis). Rhodes University. Retrieved from http://hdl.handle.net/10962/d1004044

Chicago Manual of Style (16th Edition):

Cooper, Leanne Claire. “The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells.” 2011. Masters Thesis, Rhodes University. Accessed January 23, 2020. http://hdl.handle.net/10962/d1004044.

MLA Handbook (7th Edition):

Cooper, Leanne Claire. “The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells.” 2011. Web. 23 Jan 2020.

Vancouver:

Cooper LC. The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells. [Internet] [Masters thesis]. Rhodes University; 2011. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/10962/d1004044.

Council of Science Editors:

Cooper LC. The role of Hsp90 in the Wnt pathway of MCF7 breast cancer cells. [Masters Thesis]. Rhodes University; 2011. Available from: http://hdl.handle.net/10962/d1004044


University of Minnesota

119. Nomura, Alice. Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2016, University of Minnesota

 Pancreatic cancer remains a cancer with the lowest survival rate and shortest median survival. It is predicted to accelerate to the 2nd cause of cancer(more)

Subjects/Keywords: Cancer Stem Cells; CD133; Epithelial-Mesenchymal Transition; Invasion; Metastasis; Pancreatic Cancer

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APA (6th Edition):

Nomura, A. (2016). Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/181788

Chicago Manual of Style (16th Edition):

Nomura, Alice. “Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer.” 2016. Doctoral Dissertation, University of Minnesota. Accessed January 23, 2020. http://hdl.handle.net/11299/181788.

MLA Handbook (7th Edition):

Nomura, Alice. “Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer.” 2016. Web. 23 Jan 2020.

Vancouver:

Nomura A. Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/11299/181788.

Council of Science Editors:

Nomura A. Molecular Mechanisms of Stemness and Invasion in Pancreatic Cancer. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/181788


University of Newcastle

120. Mossman, David. Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells.

Degree: PhD, 2011, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

The role of epigenetics in disease, particularly cancer, has been an emerging issue for the last decade. For… (more)

Subjects/Keywords: epigenetics; cancer; gene expression; cells; epigenetic control; colorectal cancer

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APA (6th Edition):

Mossman, D. (2011). Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/934787

Chicago Manual of Style (16th Edition):

Mossman, David. “Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells.” 2011. Doctoral Dissertation, University of Newcastle. Accessed January 23, 2020. http://hdl.handle.net/1959.13/934787.

MLA Handbook (7th Edition):

Mossman, David. “Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells.” 2011. Web. 23 Jan 2020.

Vancouver:

Mossman D. Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells. [Internet] [Doctoral dissertation]. University of Newcastle; 2011. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/1959.13/934787.

Council of Science Editors:

Mossman D. Mechanisms of epigenetic regulation of gene expression in colorectal cancer cells. [Doctoral Dissertation]. University of Newcastle; 2011. Available from: http://hdl.handle.net/1959.13/934787

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