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You searched for subject:(calcium influx). Showing records 1 – 13 of 13 total matches.

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University of Pretoria

1. Visser, Susanna Salomina. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro.

Degree: Immunology, 2006, University of Pretoria

Please read the abstract in the section 00front of this document Advisors/Committee Members: Prof R Anderson (advisor), Prof A Theron (coadvisor).

Subjects/Keywords: Neutrophils; Adenosine; Calcium influx; Calcium; Superoxide; Elastases; Calcium exflux; Glucocorticoids; UCTD

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APA (6th Edition):

Visser, S. (2006). Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro. (Doctoral Dissertation). University of Pretoria. Retrieved from http://hdl.handle.net/2263/29077

Chicago Manual of Style (16th Edition):

Visser, Susanna. “Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro.” 2006. Doctoral Dissertation, University of Pretoria. Accessed June 20, 2019. http://hdl.handle.net/2263/29077.

MLA Handbook (7th Edition):

Visser, Susanna. “Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro.” 2006. Web. 20 Jun 2019.

Vancouver:

Visser S. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro. [Internet] [Doctoral dissertation]. University of Pretoria; 2006. [cited 2019 Jun 20]. Available from: http://hdl.handle.net/2263/29077.

Council of Science Editors:

Visser S. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro. [Doctoral Dissertation]. University of Pretoria; 2006. Available from: http://hdl.handle.net/2263/29077


University of Pretoria

2. [No author]. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro .

Degree: 2006, University of Pretoria

Please read the abstract in the section 00front of this document Advisors/Committee Members: Prof R Anderson (advisor), Prof A Theron (advisor).

Subjects/Keywords: Neutrophils; Adenosine; Calcium influx; Calcium; Superoxide; Elastases; Calcium exflux; Glucocorticoids; UCTD

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APA (6th Edition):

author], [. (2006). Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro . (Doctoral Dissertation). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-10272005-091524/

Chicago Manual of Style (16th Edition):

author], [No. “Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro .” 2006. Doctoral Dissertation, University of Pretoria. Accessed June 20, 2019. http://upetd.up.ac.za/thesis/available/etd-10272005-091524/.

MLA Handbook (7th Edition):

author], [No. “Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro .” 2006. Web. 20 Jun 2019.

Vancouver:

author] [. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro . [Internet] [Doctoral dissertation]. University of Pretoria; 2006. [cited 2019 Jun 20]. Available from: http://upetd.up.ac.za/thesis/available/etd-10272005-091524/.

Council of Science Editors:

author] [. Effects of adenosine receptor agonists of the A1, A2A and A3 subtypes on the proinflammatory activity of human neutrophils in vitro . [Doctoral Dissertation]. University of Pretoria; 2006. Available from: http://upetd.up.ac.za/thesis/available/etd-10272005-091524/

3. Daisy Maria Bentes de Paula. Estudo do mecanismo molecular de transfecção mediada por ultrassom.

Degree: 2010, Universidade Federal de São Paulo

O ultrassom (US) vem sendo amplamente utilizado para melhorar a eficiência de transfecção de vetores não-virais. No entanto, o mecanismo pelo qual o US promove… (more)

Subjects/Keywords: Ultrassom; Captação de DNA plasmideal; Influxo de cálcio; Endocitose; Clatrina; BIOLOGIA MOLECULAR; Ultrasound; Plasmid uptake; Calcium influx; Endocytosis; Clathrin

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APA (6th Edition):

Paula, D. M. B. d. (2010). Estudo do mecanismo molecular de transfecção mediada por ultrassom. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1543 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1544

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paula, Daisy Maria Bentes de. “Estudo do mecanismo molecular de transfecção mediada por ultrassom.” 2010. Thesis, Universidade Federal de São Paulo. Accessed June 20, 2019. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1543 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1544.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paula, Daisy Maria Bentes de. “Estudo do mecanismo molecular de transfecção mediada por ultrassom.” 2010. Web. 20 Jun 2019.

Vancouver:

Paula DMBd. Estudo do mecanismo molecular de transfecção mediada por ultrassom. [Internet] [Thesis]. Universidade Federal de São Paulo; 2010. [cited 2019 Jun 20]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1543 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1544.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paula DMBd. Estudo do mecanismo molecular de transfecção mediada por ultrassom. [Thesis]. Universidade Federal de São Paulo; 2010. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1543 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1544

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Spasevska, Ivana. The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20.

Degree: Docteur es, Cancérologie, 2017, Lyon

Les anticorps monoclonaux (AcM) anti-CD20 sont essentiels pour le traitement du lymphome non hodgkinien et de la leucémie lymphoïde chronique (LLC). Les AcM agissent soit… (more)

Subjects/Keywords: Anticorps monoclonaux anti-CD20; Rituximab; GA101; EGR-1; Flux calcique; Inhibiteurs des canaux calciques; Anti-CD20 monoclonal antibodies; Rituximab; GA101; EGR-1; Calcium influx; Calcium channel blockers; 616.99

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APA (6th Edition):

Spasevska, I. (2017). The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSE1304

Chicago Manual of Style (16th Edition):

Spasevska, Ivana. “The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20.” 2017. Doctoral Dissertation, Lyon. Accessed June 20, 2019. http://www.theses.fr/2017LYSE1304.

MLA Handbook (7th Edition):

Spasevska, Ivana. “The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20.” 2017. Web. 20 Jun 2019.

Vancouver:

Spasevska I. The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2019 Jun 20]. Available from: http://www.theses.fr/2017LYSE1304.

Council of Science Editors:

Spasevska I. The role of EGR-1 and calcium influx in the antitumor activity of anti-CD20 monoclonal antibodies : Le rôle d'EGR-1 et du flux calcique dans l'activité antitumorale des anticorps monoclonaux anti-CD20. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSE1304

5. Vachel, Laura. Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels.

Degree: Docteur es, Aspects moléculaire et cellulaire de la biologie, 2014, Poitiers

Les canaux TRP (Transient Receptor Potential) sont des acteurs clés de l'homéostasie calcique. Plusieurs de ces canaux interviennent dans l'influx calcique des cellules épithéliales bronchiques,… (more)

Subjects/Keywords: Influx calcique; Cellules épithéliales bronchiques humaines; Mucoviscidose; Trpv6; Trpc6; PLC-Δ1; Cftr-G551d; CFTR-F508del; Calcium influx; Human bronchial epithelial cells; Cf; Trpv6; Trpc6; PLC-Δ1; Cftr-G551d; CFTR-F508del; 571.6

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APA (6th Edition):

Vachel, L. (2014). Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2014POIT2303

Chicago Manual of Style (16th Edition):

Vachel, Laura. “Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels.” 2014. Doctoral Dissertation, Poitiers. Accessed June 20, 2019. http://www.theses.fr/2014POIT2303.

MLA Handbook (7th Edition):

Vachel, Laura. “Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels.” 2014. Web. 20 Jun 2019.

Vancouver:

Vachel L. Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels. [Internet] [Doctoral dissertation]. Poitiers; 2014. [cited 2019 Jun 20]. Available from: http://www.theses.fr/2014POIT2303.

Council of Science Editors:

Vachel L. Étude de l'influx calcique des cellules épithéliales bronchiques mucoviscidosiques : implication des canaux TRP : Ca2+ influx in human bronchial epithelial cells : implication of TRP channels. [Doctoral Dissertation]. Poitiers; 2014. Available from: http://www.theses.fr/2014POIT2303


Universidade Federal de Santa Maria

6. Mateus Fortes Rossato. ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE.

Degree: 2010, Universidade Federal de Santa Maria

O receptor de potencial transiente vanilóide 1 (TRPV1) é um canal iônico permeável a cátions ativado por uma série de estímulos nocivos, como calor, acidificação… (more)

Subjects/Keywords: Binding; calcium influx; eriodictyol; pain; TRPV1; thiol; tiois; TRPV1; dor; eriodictiol; influxo de cálcio; ensaio de ligação específica; 3-NT; BIOQUIMICA; 3-NT

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APA (6th Edition):

Rossato, M. F. (2010). ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3380

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossato, Mateus Fortes. “ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE.” 2010. Thesis, Universidade Federal de Santa Maria. Accessed June 20, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3380.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossato, Mateus Fortes. “ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE.” 2010. Web. 20 Jun 2019.

Vancouver:

Rossato MF. ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2010. [cited 2019 Jun 20]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3380.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossato MF. ERIODICTIOL: UM FLAVONÓIDE ANTAGONISTA DO RECEPTOR TRPV1 COM ATIVIDADE ANTIOXIDANTE. [Thesis]. Universidade Federal de Santa Maria; 2010. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3380

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of the Western Cape

7. Sharma, Rajan. Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds .

Degree: 2017, University of the Western Cape

 Among neurological disorders, many of the most devastating disorders are neurodegenerative. Modern research associates excitotoxicity to a variety of neuropathological conditions, suggesting that the neurodegenerative… (more)

Subjects/Keywords: Neurodegenerative diseases; Excitotoxicity; Apoptosis; Polycyclic cage compounds; Drug design; Neuroprotection; N-methyl-D-aspartate (NMDA) receptors; Calcium influx; Nitric oxide; NO-donating; S-nitrosylation

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APA (6th Edition):

Sharma, R. (2017). Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/6228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Rajan. “Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds .” 2017. Thesis, University of the Western Cape. Accessed June 20, 2019. http://hdl.handle.net/11394/6228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Rajan. “Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds .” 2017. Web. 20 Jun 2019.

Vancouver:

Sharma R. Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds . [Internet] [Thesis]. University of the Western Cape; 2017. [cited 2019 Jun 20]. Available from: http://hdl.handle.net/11394/6228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma R. Synthesis & biological evaluation of neuroprotective molecules with polycyclic scaffolds . [Thesis]. University of the Western Cape; 2017. Available from: http://hdl.handle.net/11394/6228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

8. Yang, Heng. TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique.

Degree: Docteur es, Immunologie, 2012, Université Paris-Sud – Paris XI

TRPM4, un canal cationique non-sélective activé par le Ca2+ intracellulaire, est un acteur moléculaire important impliqué de la régulation du signal calcique et l’activation des… (more)

Subjects/Keywords: Foxp3+ Tregs; TRPM4; Flux calcique; Réponse immunitaire; Réponse anti-tumorale; Immunorégulation; ATP; Mort cellulaire; Foxp3+ Tregs; TRPM4; Calcium influx; Immune response; Immune regulation; Anti-tumor response; ATP; Cell death

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APA (6th Edition):

Yang, H. (2012). TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA114840

Chicago Manual of Style (16th Edition):

Yang, Heng. “TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed June 20, 2019. http://www.theses.fr/2012PA114840.

MLA Handbook (7th Edition):

Yang, Heng. “TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique.” 2012. Web. 20 Jun 2019.

Vancouver:

Yang H. TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2019 Jun 20]. Available from: http://www.theses.fr/2012PA114840.

Council of Science Editors:

Yang H. TRPM4, a non selective cation-permeable channel regulates Foxp3+ regulatory T cells suppressive function and survival trough modulating calcium influx : TRPM4, le canal cationique non-selective régule la fonction suppressive et la survie des lymphocytes T régulateurs Foxp3+ en régulant l'influx calcique. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA114840


Université Paris-Sud – Paris XI

9. Djillani, Alaeddine. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.

Degree: Docteur es, Pharmacologie, Biologie cellulaire et moléculaire, 2013, Université Paris-Sud – Paris XI

 Les polynucléaires neutrophiles représentent 50-70% des leucocytes sanguins et possèdent un rôle majeur dans la défense de l’organisme contre les pathogènes. Le Ca2+ est un… (more)

Subjects/Keywords: Calcium; Influx capacitif; Polynucléaire neutrophile; Lymphocyte; Courant CRAC; Réticulum endoplasmique; Canaux calciques; SOC; SOCE; Thapsigargine (TG); SERCA; Phagocytose; Production de formes réactives de l'oxygène (FRO); Orai; STIM; 2-APB; Calcium; Capacitive calcium entry; Polymorphonuclear neutrophil; Lymphocyte; CRAC courant; Endoplasmic reticulum; Calcium channel; Store-operated calcium channel (SOC); Store-operated calcium entry (SOCE); Thapsigargin (TG); Sarco-endoplasmic calcium ATPase (SERCA); Phagocytosis; Reactive oxygen species (ROS) production; Orai; STIM; 2-APB

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Djillani, A. (2013). Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T056

Chicago Manual of Style (16th Edition):

Djillani, Alaeddine. “Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed June 20, 2019. http://www.theses.fr/2013PA11T056.

MLA Handbook (7th Edition):

Djillani, Alaeddine. “Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.” 2013. Web. 20 Jun 2019.

Vancouver:

Djillani A. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2019 Jun 20]. Available from: http://www.theses.fr/2013PA11T056.

Council of Science Editors:

Djillani A. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T056

10. Shoji, Emi. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第19229号

医博第4028号

Subjects/Keywords: Duchenne muscular dystrophy; Human indcued pluripotent stem cell; Disease modelling; Calcium ion influx; Exon skipping; Dystrophin

Page 1 Page 2

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APA (6th Edition):

Shoji, E. (2015). Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.” 2015. Thesis, Kyoto University / 京都大学. Accessed June 20, 2019. http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.” 2015. Web. 20 Jun 2019.

Vancouver:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2019 Jun 20]. Available from: http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

11. Shoji, Emi. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells.

Degree: 2015, Kyoto University

Subjects/Keywords: Duchenne muscular dystrophy; Human indcued pluripotent stem cell; Disease modelling; Calcium ion influx; Exon skipping; Dystrophin

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APA (6th Edition):

Shoji, E. (2015). Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/200495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. ” 2015. Thesis, Kyoto University. Accessed June 20, 2019. http://hdl.handle.net/2433/200495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. ” 2015. Web. 20 Jun 2019.

Vancouver:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. [Internet] [Thesis]. Kyoto University; 2015. [cited 2019 Jun 20]. Available from: http://hdl.handle.net/2433/200495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/200495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

12. Lin, I-Ju. The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation.

Degree: PhD, Medical Sciences - Genetics (IDP), 2010, University of Florida

 Differentiation of erythroid cells is regulated by cell signaling pathways including those that change the intracellular concentration of calcium. Calcium-dependent proteases have been shown previously… (more)

Subjects/Keywords: Antibodies; Calcium; Chromatin; DNA; Erythroid cells; Gene expression; Genetic loci; Hemoglobins; Promoter regions; Proteins; adult, beta, biotinylation, calcium, calpain, calpeptin, cells, differentiation, efficient, entry, erythroid, erythropoiesis, factors, gene, globin, hemoglobinopathies, high, hsp70, in, influx, inhibition, inhibitor, locus, m, mass, mel, nfkappab, pathways, primary, progenitor, proteomics, regulation, signaling, spectrometry, streptavidin, tfii, throughput, topoisomerase, transcription, usf, vivo

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, I. (2010). The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0042457

Chicago Manual of Style (16th Edition):

Lin, I-Ju. “The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation.” 2010. Doctoral Dissertation, University of Florida. Accessed June 20, 2019. http://ufdc.ufl.edu/UFE0042457.

MLA Handbook (7th Edition):

Lin, I-Ju. “The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation.” 2010. Web. 20 Jun 2019.

Vancouver:

Lin I. The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Jun 20]. Available from: http://ufdc.ufl.edu/UFE0042457.

Council of Science Editors:

Lin I. The Role of Calpain, USF, and TFII-I in Beta-Globin Gene Regulation. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0042457

13. Ampem, Prince Tuffour. The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis.

Degree: PhD, Biomedical Sciences (Molecular Medicine), 2017, University of Toledo Health Science Campus

 Atherosclerosis is the main cause of coronary heart disease, the leading cause of mortality in developed countries. Development of atherosclerotic lesions is in part, associated… (more)

Subjects/Keywords: Biomedical Research; TRPC3; ER stress; Unfolded protein response; apoptosis; Endothelial cells; Atherosclerosis; Calcium influx; CAMKII; STAT1; JNK; Coronary artery disease; lesion; Thapsigargin; Tunicamycin; Pyr10; Apoe knockout; transgenic mouse

…29 3.1 Suppression of constitutive Ca2+ influx reduces ER stress-induced apoptosis in… …29 3.2 Suppression of constitutive Ca2+ influx impairs UPR activation in HCAECs… …stress-induced apoptosis in HCAECs......................................42 3.5 Calcium… …Calcium Measurements to assess effect on BAPTA loading on ATP-induced biphasic response in… …OAG-induced Ba2+ influx in HEK293 cells treated with/without Pyr10, and measurement of… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ampem, P. T. (2017). The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1493230041479167

Chicago Manual of Style (16th Edition):

Ampem, Prince Tuffour. “The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis.” 2017. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed June 20, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1493230041479167.

MLA Handbook (7th Edition):

Ampem, Prince Tuffour. “The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis.” 2017. Web. 20 Jun 2019.

Vancouver:

Ampem PT. The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2017. [cited 2019 Jun 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1493230041479167.

Council of Science Editors:

Ampem PT. The Transient Receptor Potential Canonical 3 (TRPC3) Channel: Novel Role in Endothelial Cell Apoptosis and its Impact on Atherosclerosis. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1493230041479167

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