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You searched for subject:(c Myc). Showing records 1 – 30 of 135 total matches.

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University of Otago

1. Rhodes, Jenny Marie. Linking cohesin-dependent transcription with cell pluripotency .

Degree: 2012, University of Otago

 Embryonic stem cells are pluripotent; they have the ability to form any cell type of the developing embryo. Due to their pluripotent nature there is… (more)

Subjects/Keywords: cohesin; transcription; pluripotency; c-Myc

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APA (6th Edition):

Rhodes, J. M. (2012). Linking cohesin-dependent transcription with cell pluripotency . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/2343

Chicago Manual of Style (16th Edition):

Rhodes, Jenny Marie. “Linking cohesin-dependent transcription with cell pluripotency .” 2012. Doctoral Dissertation, University of Otago. Accessed November 12, 2019. http://hdl.handle.net/10523/2343.

MLA Handbook (7th Edition):

Rhodes, Jenny Marie. “Linking cohesin-dependent transcription with cell pluripotency .” 2012. Web. 12 Nov 2019.

Vancouver:

Rhodes JM. Linking cohesin-dependent transcription with cell pluripotency . [Internet] [Doctoral dissertation]. University of Otago; 2012. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/10523/2343.

Council of Science Editors:

Rhodes JM. Linking cohesin-dependent transcription with cell pluripotency . [Doctoral Dissertation]. University of Otago; 2012. Available from: http://hdl.handle.net/10523/2343


University of Louisville

2. Takwi, Apana Agha L., 1980-. A statin-regulated microRNA represses c-Myc expression and function.

Degree: PhD, 2011, University of Louisville

c-Myc dysregulation is one of the most common abnormalities found in human cancer. MicroRNAs (miRNAs) are functionally intertwined with the c-Myc network as multiple miRNAs… (more)

Subjects/Keywords: micro-RNA; miR-33b; Medulloblastoma; c-Myc; Lovastatin; Myc

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APA (6th Edition):

Takwi, Apana Agha L., 1. (2011). A statin-regulated microRNA represses c-Myc expression and function. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1415 ; https://ir.library.louisville.edu/etd/1415

Chicago Manual of Style (16th Edition):

Takwi, Apana Agha L., 1980-. “A statin-regulated microRNA represses c-Myc expression and function.” 2011. Doctoral Dissertation, University of Louisville. Accessed November 12, 2019. 10.18297/etd/1415 ; https://ir.library.louisville.edu/etd/1415.

MLA Handbook (7th Edition):

Takwi, Apana Agha L., 1980-. “A statin-regulated microRNA represses c-Myc expression and function.” 2011. Web. 12 Nov 2019.

Vancouver:

Takwi, Apana Agha L. 1. A statin-regulated microRNA represses c-Myc expression and function. [Internet] [Doctoral dissertation]. University of Louisville; 2011. [cited 2019 Nov 12]. Available from: 10.18297/etd/1415 ; https://ir.library.louisville.edu/etd/1415.

Council of Science Editors:

Takwi, Apana Agha L. 1. A statin-regulated microRNA represses c-Myc expression and function. [Doctoral Dissertation]. University of Louisville; 2011. Available from: 10.18297/etd/1415 ; https://ir.library.louisville.edu/etd/1415


Universiteit Utrecht

3. Muncan, V. Wnt signaling and c-Myc in intestinal epithelium.

Degree: 2007, Universiteit Utrecht

 constantly produce cells from a stem cell reservoir that give rise to proliferating transit amplifying cells, which subsequently differentiate and are positioned in their proper… (more)

Subjects/Keywords: Biologie; Wnt signaling; intestine; C-myc; regeneration

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APA (6th Edition):

Muncan, V. (2007). Wnt signaling and c-Myc in intestinal epithelium. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/22634

Chicago Manual of Style (16th Edition):

Muncan, V. “Wnt signaling and c-Myc in intestinal epithelium.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed November 12, 2019. http://dspace.library.uu.nl:8080/handle/1874/22634.

MLA Handbook (7th Edition):

Muncan, V. “Wnt signaling and c-Myc in intestinal epithelium.” 2007. Web. 12 Nov 2019.

Vancouver:

Muncan V. Wnt signaling and c-Myc in intestinal epithelium. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Nov 12]. Available from: http://dspace.library.uu.nl:8080/handle/1874/22634.

Council of Science Editors:

Muncan V. Wnt signaling and c-Myc in intestinal epithelium. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/22634


McMaster University

4. MacKenzie, Colleen. ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY.

Degree: MSc, 2018, McMaster University

Breast cancer (BC) is a complex disease with over 25,000 new diagnoses made in Canadian women every year. The disease can be caused by inactivation… (more)

Subjects/Keywords: Breast Cancer; BMI1; ATM; c-Myc

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APA (6th Edition):

MacKenzie, C. (2018). ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/23699

Chicago Manual of Style (16th Edition):

MacKenzie, Colleen. “ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY.” 2018. Masters Thesis, McMaster University. Accessed November 12, 2019. http://hdl.handle.net/11375/23699.

MLA Handbook (7th Edition):

MacKenzie, Colleen. “ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY.” 2018. Web. 12 Nov 2019.

Vancouver:

MacKenzie C. ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY. [Internet] [Masters thesis]. McMaster University; 2018. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/11375/23699.

Council of Science Editors:

MacKenzie C. ROLE OF BMI1 IN PROMOTING BREAST CANCER TUMORIGENESIS THROUGH ATTENUATING THE DNA DAMAGE RESPONSE PATHWAY. [Masters Thesis]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/23699


University of Ottawa

5. Sunohara, Maxwell. Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia .

Degree: 2017, University of Ottawa

 Currently there is no curative therapy for Chronic Myelogenous Leukemia (CML), and patients must remain on the current prescribed treatment, tyrosine kinase inhibitors (TKI), indefinitely.… (more)

Subjects/Keywords: c-MYC; leukemia; OGT; Chronic Myelogenous Leukemia

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APA (6th Edition):

Sunohara, M. (2017). Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/35884

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sunohara, Maxwell. “Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia .” 2017. Thesis, University of Ottawa. Accessed November 12, 2019. http://hdl.handle.net/10393/35884.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sunohara, Maxwell. “Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia .” 2017. Web. 12 Nov 2019.

Vancouver:

Sunohara M. Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/10393/35884.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sunohara M. Targeting the Process of c-MYC Stabilization in Chronic Myelogenous Leukemia . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/35884

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tampere University

6. ÅKERLA, JONNE. cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla .

Degree: 2013, Tampere University

 Ruokatorven adenokarsinooman ilmaantuvuus on nousussa ja taudin ennuste on useimmissa tapauksissa huono. CIP2A-proteiinin esiintyvyyden on todettu suurentuneen monissa syövissä kuten ruokatorven levyepiteelisyövässä. CIP2A-proteiinin esiintyvyys on… (more)

Subjects/Keywords: c-MYC; proteiinifosfataasi 2A; Barrettin ruokatorvi

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APA (6th Edition):

ÅKERLA, J. (2013). cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla . (Masters Thesis). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/94492

Chicago Manual of Style (16th Edition):

ÅKERLA, JONNE. “cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla .” 2013. Masters Thesis, Tampere University. Accessed November 12, 2019. https://trepo.tuni.fi/handle/10024/94492.

MLA Handbook (7th Edition):

ÅKERLA, JONNE. “cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla .” 2013. Web. 12 Nov 2019.

Vancouver:

ÅKERLA J. cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla . [Internet] [Masters thesis]. Tampere University; 2013. [cited 2019 Nov 12]. Available from: https://trepo.tuni.fi/handle/10024/94492.

Council of Science Editors:

ÅKERLA J. cip2a:n esiintyminen ja merkitys ennusteesees ruokatorven adenokarsinoomapotilailla . [Masters Thesis]. Tampere University; 2013. Available from: https://trepo.tuni.fi/handle/10024/94492

7. Arnaud, Nicolas. Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc.

Degree: Docteur es, Sciences de la vie et de la sante, 2017, Limoges

A l’instar du lymphome de Burkitt (LB) avec la translocation de MYC, les lymphomes diffus à grandes cellules B (DLBCL) par d'autres mécanismes (mutation, amplification,… (more)

Subjects/Keywords: NF-κB; C-Myc; DLBCL; TLR9; NF-κB; C-Myc; DLBCL; TLR9; 610

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APA (6th Edition):

Arnaud, N. (2017). Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2017LIMO0105

Chicago Manual of Style (16th Edition):

Arnaud, Nicolas. “Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc.” 2017. Doctoral Dissertation, Limoges. Accessed November 12, 2019. http://www.theses.fr/2017LIMO0105.

MLA Handbook (7th Edition):

Arnaud, Nicolas. “Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc.” 2017. Web. 12 Nov 2019.

Vancouver:

Arnaud N. Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc. [Internet] [Doctoral dissertation]. Limoges; 2017. [cited 2019 Nov 12]. Available from: http://www.theses.fr/2017LIMO0105.

Council of Science Editors:

Arnaud N. Les lymphomes B diffus à grandes cellules de type activé : rôle de NF-κB et c-Myc. : Activated B cell Diffuse Large B cell lymphoma : role of NF-κB and c-Myc. [Doctoral Dissertation]. Limoges; 2017. Available from: http://www.theses.fr/2017LIMO0105


Université Paris-Sud – Paris XI

8. Liu, Qingyuan. Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc.

Degree: Docteur es, Sciences de la vie et de la santé, 2014, Université Paris-Sud – Paris XI

 La régulation de la télomérase s’effectue à de nombreux niveaux dont la transcription de la sous-Unité catalytique (hTERT). Les travaux du laboratoire effectués sur les… (more)

Subjects/Keywords: Télomérase; HTERT; Épigénétique; Méthylation; C-Myc; PKA; Telomerase; HTERT; Epigenetic; Methylation; C-Myc; PKA

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APA (6th Edition):

Liu, Q. (2014). Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T103

Chicago Manual of Style (16th Edition):

Liu, Qingyuan. “Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed November 12, 2019. http://www.theses.fr/2014PA11T103.

MLA Handbook (7th Edition):

Liu, Qingyuan. “Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc.” 2014. Web. 12 Nov 2019.

Vancouver:

Liu Q. Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2019 Nov 12]. Available from: http://www.theses.fr/2014PA11T103.

Council of Science Editors:

Liu Q. Epigenetic Regulation of hTERT in Human Acute Promyelocytic Leukemia Cell Line NB4 and Role of c-Myc : Régulation épigénétique de hTERT dans le modèle de leucémie aiguë promyélocytaire NB4 et rôle de c-Myc. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T103


Université Paris-Sud – Paris XI

9. Penglong, Tipparat. Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide.

Degree: Docteur es, Ascpects moléculaires et cellulaires de la biologie, 2015, Université Paris-Sud – Paris XI

 Pour assurer la production de milliards de globules rouges, l’érythropoièse doit parfaitement contrôler les processus de prolifération et de différenciation. Ces deux processus sont régulés… (more)

Subjects/Keywords: Erythropoïèse; GATA-1; PRB; C-MYC; Bromodomaine; Erythropoiesis; GATA-1; PRB; C-MYC; Bromodomain

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APA (6th Edition):

Penglong, T. (2015). Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA11T022

Chicago Manual of Style (16th Edition):

Penglong, Tipparat. “Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed November 12, 2019. http://www.theses.fr/2015PA11T022.

MLA Handbook (7th Edition):

Penglong, Tipparat. “Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide.” 2015. Web. 12 Nov 2019.

Vancouver:

Penglong T. Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2019 Nov 12]. Available from: http://www.theses.fr/2015PA11T022.

Council of Science Editors:

Penglong T. Molecular Basis of Erythroid Cell Proliferation and Differentiation : Les bases moléculaires de la prolifération et de la différentiation érythroide. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA11T022


Cranfield University

10. Stebbeds, William Joshua David. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.

Degree: MSc by Research, 2011, Cranfield University

 G-quadruplexes (G4-DNA) are a class of secondary structures formed from Guanine rich sequences. In recent years these structures have been implicated in both telomere maintenance… (more)

Subjects/Keywords: G-quadruplexes; G-quadruplexes; oncogene expression; Carcinogenesis; C-Myc; c-Kit

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APA (6th Edition):

Stebbeds, W. J. D. (2011). The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. (Masters Thesis). Cranfield University. Retrieved from http://dspace.lib.cranfield.ac.uk/handle/1826/7179

Chicago Manual of Style (16th Edition):

Stebbeds, William Joshua David. “The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.” 2011. Masters Thesis, Cranfield University. Accessed November 12, 2019. http://dspace.lib.cranfield.ac.uk/handle/1826/7179.

MLA Handbook (7th Edition):

Stebbeds, William Joshua David. “The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.” 2011. Web. 12 Nov 2019.

Vancouver:

Stebbeds WJD. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. [Internet] [Masters thesis]. Cranfield University; 2011. [cited 2019 Nov 12]. Available from: http://dspace.lib.cranfield.ac.uk/handle/1826/7179.

Council of Science Editors:

Stebbeds WJD. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. [Masters Thesis]. Cranfield University; 2011. Available from: http://dspace.lib.cranfield.ac.uk/handle/1826/7179

11. Silva, Flávia Amoroso Matos e. Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC).

Degree: Mestrado, Biociências Aplicadas à Farmácia, 2009, University of São Paulo

As neoplasias de cabeça e pescoço constituem um importante problema de saúde pública devido à alta incidência e alguns tipos estão associados a fatores comportamentais… (more)

Subjects/Keywords: c-Myc; c-Myc; carcinoma oral; CTAK1; CTAK1; HNSCC; I2PP2A; I2PP2A; K protein; prognosis; prognóstico; proteína K

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APA (6th Edition):

Silva, F. A. M. e. (2009). Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC). (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60135/tde-24082009-165141/ ;

Chicago Manual of Style (16th Edition):

Silva, Flávia Amoroso Matos e. “Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC).” 2009. Masters Thesis, University of São Paulo. Accessed November 12, 2019. http://www.teses.usp.br/teses/disponiveis/60/60135/tde-24082009-165141/ ;.

MLA Handbook (7th Edition):

Silva, Flávia Amoroso Matos e. “Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC).” 2009. Web. 12 Nov 2019.

Vancouver:

Silva FAMe. Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC). [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2019 Nov 12]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60135/tde-24082009-165141/ ;.

Council of Science Editors:

Silva FAMe. Estudos das proteínas hnRNP K, SET e MARK3 como potenciais marcadores de prognóstico em câncer epidermóide de cabeça e pescoço (HNSCC). [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/60/60135/tde-24082009-165141/ ;


Université de Montréal

12. Saba, Ingrid. The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages .

Degree: 2012, Université de Montréal

 Les lymphocytes B et T sont issus de cellules progénitrices lymphoïdes de la moelle osseuse qui se différencient grâce à l’action de facteurs de transcription,… (more)

Subjects/Keywords: Miz-1; c-Myc; IL-7R; Differentiation; Apoptosis; STAT5; SOCS1; p53; Miz-1; c-Myc; TCR; Différenciation; Apoptose; Bcl-2

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APA (6th Edition):

Saba, I. (2012). The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saba, Ingrid. “The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages .” 2012. Thesis, Université de Montréal. Accessed November 12, 2019. http://hdl.handle.net/1866/6890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saba, Ingrid. “The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages .” 2012. Web. 12 Nov 2019.

Vancouver:

Saba I. The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/1866/6890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saba I. The BTB/POZ Transcription Factor Miz-1 Is Required To Regulate The Commitment, Survival And Differentiation Of Early B And T Cell Lineages . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/6890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Sobolewski, Cyril. Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2011, Université Henri Poincaré – Nancy I

 Les cyclooxygénases (COXs) sont une famille d'enzymes impliquées dans la biosynthèse des prostaglandines. COX-2 est la forme inductible qui est induite pendant l'inflammation et qui… (more)

Subjects/Keywords: Cox-2; Leucémie; Inflammation; Apoptose; Cycle cellulaire; C-Myc; Cox-2; Leukemia; Inflammation; Apoptosis; Cell cycle; C-Myc; 616.994 1906

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APA (6th Edition):

Sobolewski, C. (2011). Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines. (Doctoral Dissertation). Université Henri Poincaré – Nancy I. Retrieved from http://www.theses.fr/2011NAN10102

Chicago Manual of Style (16th Edition):

Sobolewski, Cyril. “Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines.” 2011. Doctoral Dissertation, Université Henri Poincaré – Nancy I. Accessed November 12, 2019. http://www.theses.fr/2011NAN10102.

MLA Handbook (7th Edition):

Sobolewski, Cyril. “Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines.” 2011. Web. 12 Nov 2019.

Vancouver:

Sobolewski C. Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines. [Internet] [Doctoral dissertation]. Université Henri Poincaré – Nancy I; 2011. [cited 2019 Nov 12]. Available from: http://www.theses.fr/2011NAN10102.

Council of Science Editors:

Sobolewski C. Effets d'inhibiteurs de la cyclooxygénase-2 sur la prolifération et la survie de cellules cancéreuses hématopoïétiques : Effects of cyclooxygenase-2 inhibitors on cell proliferation and cell death in human hematopoietic cancer cell lines. [Doctoral Dissertation]. Université Henri Poincaré – Nancy I; 2011. Available from: http://www.theses.fr/2011NAN10102

14. Beaudoin, Nicolas. L’inhibition de c-MYC : l’approche MAX*.

Degree: M. Sc., Pharmacologie, 2015, Université de Sherbrooke

c-MYC est un facteur de transcription oncogénique dont l’expression est dérégulée dans 78% des gliomes. On observe d’ailleurs une corrélation positive entre sa surexpression et… (more)

Subjects/Keywords: Cancer; Tumeur; Glioblastome; c-MYC; MAX; b-HLH-LZ

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beaudoin, N. (2015). L’inhibition de c-MYC : l’approche MAX*. (Masters Thesis). Université de Sherbrooke. Retrieved from http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_6739.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/6739/5/Beaudoin_Nicolas_MSc_2015.pdf

Chicago Manual of Style (16th Edition):

Beaudoin, Nicolas. “L’inhibition de c-MYC : l’approche MAX*.” 2015. Masters Thesis, Université de Sherbrooke. Accessed November 12, 2019. http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_6739.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/6739/5/Beaudoin_Nicolas_MSc_2015.pdf.

MLA Handbook (7th Edition):

Beaudoin, Nicolas. “L’inhibition de c-MYC : l’approche MAX*.” 2015. Web. 12 Nov 2019.

Vancouver:

Beaudoin N. L’inhibition de c-MYC : l’approche MAX*. [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Nov 12]. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_6739.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/6739/5/Beaudoin_Nicolas_MSc_2015.pdf.

Council of Science Editors:

Beaudoin N. L’inhibition de c-MYC : l’approche MAX*. [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_6739.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/6739/5/Beaudoin_Nicolas_MSc_2015.pdf

15. Noritake, Hidenao. TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する.

Degree: 博士(医学), 2014, Hamamatsu University School of Medicine / 浜松医科大学

Hepatocellular carcinoma (HCC) is the most common hepatic tumor worldwide and is a major cause of death in many countries. Although chronic viral infections and… (more)

Subjects/Keywords: TGFα; C-MYC; mutant CTNNB1; HCC; apoptosis; transdifferentiation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Noritake, H. (2014). TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する. (Thesis). Hamamatsu University School of Medicine / 浜松医科大学. Retrieved from http://hdl.handle.net/10271/2789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Noritake, Hidenao. “TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する.” 2014. Thesis, Hamamatsu University School of Medicine / 浜松医科大学. Accessed November 12, 2019. http://hdl.handle.net/10271/2789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Noritake, Hidenao. “TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する.” 2014. Web. 12 Nov 2019.

Vancouver:

Noritake H. TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する. [Internet] [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2014. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/10271/2789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Noritake H. TGFα, c-MYC, mutated CTNNB1 and their combinations act distinctly on the Hep3B tumors in nude mice : TGFα,c-MYC,変異型CTNNB1遺伝子およびこれらの組み合わせはヌードマウスにおけるHep3B腫瘍形成に明確に作用する. [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2014. Available from: http://hdl.handle.net/10271/2789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

16. Beaudoin, Nicolas. L’inhibition de c-MYC : l’approche MAX* .

Degree: 2015, Université de Sherbrooke

c-MYC est un facteur de transcription oncogénique dont l’expression est dérégulée dans 78% des gliomes. On observe d’ailleurs une corrélation positive entre sa surexpression et… (more)

Subjects/Keywords: Cancer; Tumeur; Glioblastome; c-MYC; MAX; b-HLH-LZ

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beaudoin, N. (2015). L’inhibition de c-MYC : l’approche MAX* . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6739

Chicago Manual of Style (16th Edition):

Beaudoin, Nicolas. “L’inhibition de c-MYC : l’approche MAX* .” 2015. Masters Thesis, Université de Sherbrooke. Accessed November 12, 2019. http://hdl.handle.net/11143/6739.

MLA Handbook (7th Edition):

Beaudoin, Nicolas. “L’inhibition de c-MYC : l’approche MAX* .” 2015. Web. 12 Nov 2019.

Vancouver:

Beaudoin N. L’inhibition de c-MYC : l’approche MAX* . [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/11143/6739.

Council of Science Editors:

Beaudoin N. L’inhibition de c-MYC : l’approche MAX* . [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://hdl.handle.net/11143/6739


Virginia Tech

17. Mercedes-Camacho, Ana Yokayra. Pin1: WW domain ligands, catalytic inhibitors, and the mechanism.

Degree: PhD, Biochemistry, 2011, Virginia Tech

 The peptidyl prolyl cis/trans isomerase, PPIase, has been the focus of numerous studies in the field of cell cycle regulation since proline-directed phosphorylation is an… (more)

Subjects/Keywords: PPIases; Pin1 inhibitors; WW domain ligands; and KIE; c-Myc; ELEBA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mercedes-Camacho, A. Y. (2011). Pin1: WW domain ligands, catalytic inhibitors, and the mechanism. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77093

Chicago Manual of Style (16th Edition):

Mercedes-Camacho, Ana Yokayra. “Pin1: WW domain ligands, catalytic inhibitors, and the mechanism.” 2011. Doctoral Dissertation, Virginia Tech. Accessed November 12, 2019. http://hdl.handle.net/10919/77093.

MLA Handbook (7th Edition):

Mercedes-Camacho, Ana Yokayra. “Pin1: WW domain ligands, catalytic inhibitors, and the mechanism.” 2011. Web. 12 Nov 2019.

Vancouver:

Mercedes-Camacho AY. Pin1: WW domain ligands, catalytic inhibitors, and the mechanism. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/10919/77093.

Council of Science Editors:

Mercedes-Camacho AY. Pin1: WW domain ligands, catalytic inhibitors, and the mechanism. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/77093


Université de Sherbrooke

18. Beaulieu, Marie-Ève. Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc .

Degree: 2011, Université de Sherbrooke

 The transcription factor c-Myc plays a central role in cell growth and proliferation owing to the large number of genes it transactivates or transrepresses and… (more)

Subjects/Keywords: Cancer; Liaison à l'ADN; Hétérodimérisation; Transrépression; C-Myc

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APA (6th Edition):

Beaulieu, M. (2011). Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc . (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/5809

Chicago Manual of Style (16th Edition):

Beaulieu, Marie-Ève. “Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc .” 2011. Doctoral Dissertation, Université de Sherbrooke. Accessed November 12, 2019. http://hdl.handle.net/11143/5809.

MLA Handbook (7th Edition):

Beaulieu, Marie-Ève. “Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc .” 2011. Web. 12 Nov 2019.

Vancouver:

Beaulieu M. Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc . [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2011. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/11143/5809.

Council of Science Editors:

Beaulieu M. Biologie structurale de c-Myc et Max : évidences pour un nouveau mécanisme de transrépression par Myc . [Doctoral Dissertation]. Université de Sherbrooke; 2011. Available from: http://hdl.handle.net/11143/5809


University of Southern California

19. Narayanan, Padmini. Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma.

Degree: MS, Molecular Microbiology and Immunology, 2015, University of Southern California

 RNA-binding protein MSI-2 has been shown to be elevated in several cancers types such as leukemia, hepatocellular carcinoma (HCC) and breast and has been linked… (more)

Subjects/Keywords: hepatocellular carcinoma; self-renewal; MSI-2; c-MYC; IRES

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APA (6th Edition):

Narayanan, P. (2015). Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/605339/rec/4286

Chicago Manual of Style (16th Edition):

Narayanan, Padmini. “Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma.” 2015. Masters Thesis, University of Southern California. Accessed November 12, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/605339/rec/4286.

MLA Handbook (7th Edition):

Narayanan, Padmini. “Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma.” 2015. Web. 12 Nov 2019.

Vancouver:

Narayanan P. Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Nov 12]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/605339/rec/4286.

Council of Science Editors:

Narayanan P. Musashi-2 promotes c-MYC expression through IRES-dependent translation and self-renewal ability in hepatocellular carcinoma. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/605339/rec/4286


Cornell University

20. Jing, Hui. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER .

Degree: 2017, Cornell University

 SIRT2 belongs to the mammalian sirtuin or NAD-dependent lysine deacylase family. Growing evidence suggests that SIRT2 plays important roles in cell cycle regulation, stress response,… (more)

Subjects/Keywords: c-Myc; K-Ras; Lysine fatty acylation; SIRT2; Sirtuin; Chemistry; cancer

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APA (6th Edition):

Jing, H. (2017). MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/59083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jing, Hui. “MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER .” 2017. Thesis, Cornell University. Accessed November 12, 2019. http://hdl.handle.net/1813/59083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jing, Hui. “MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER .” 2017. Web. 12 Nov 2019.

Vancouver:

Jing H. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER . [Internet] [Thesis]. Cornell University; 2017. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/1813/59083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jing H. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER . [Thesis]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/59083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

21. Anderson, Luke Russell. Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer.

Degree: Garvan Institute of Medical Research, 2010, University of New South Wales

 Breast Cancer is the leading cause of cancer deaths in females in Australia. The hormone estrogen is a major aetiological factor in breast carcinogenesis, however,… (more)

Subjects/Keywords: c-MYC; Breast cancer; BAG-1; Tamoxifen; MCF-7

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APA (6th Edition):

Anderson, L. R. (2010). Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50401 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9289/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Anderson, Luke Russell. “Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer.” 2010. Doctoral Dissertation, University of New South Wales. Accessed November 12, 2019. http://handle.unsw.edu.au/1959.4/50401 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9289/SOURCE02?view=true.

MLA Handbook (7th Edition):

Anderson, Luke Russell. “Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer.” 2010. Web. 12 Nov 2019.

Vancouver:

Anderson LR. Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2019 Nov 12]. Available from: http://handle.unsw.edu.au/1959.4/50401 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9289/SOURCE02?view=true.

Council of Science Editors:

Anderson LR. Delineating the role of BCL-2-associated athanogene 1 (BAG-1) in human breast cancer. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/50401 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9289/SOURCE02?view=true


University of Manitoba

22. Chuang, Tony Chih-Yuan. The three-dimensional (3D) organization of telomeres during cellular transformation.

Degree: Physiology, 2010, University of Manitoba

 Statement of Problem Telomere dynamics in the three-dimensional (3D) space of the mammalian nucleus plays an important role in the maintenance of genomic stability. However,… (more)

Subjects/Keywords: telomere; c-myc; deconvolution; molecular imaging; genomic instability; biomarker

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APA (6th Edition):

Chuang, T. C. (2010). The three-dimensional (3D) organization of telomeres during cellular transformation. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4228

Chicago Manual of Style (16th Edition):

Chuang, Tony Chih-Yuan. “The three-dimensional (3D) organization of telomeres during cellular transformation.” 2010. Masters Thesis, University of Manitoba. Accessed November 12, 2019. http://hdl.handle.net/1993/4228.

MLA Handbook (7th Edition):

Chuang, Tony Chih-Yuan. “The three-dimensional (3D) organization of telomeres during cellular transformation.” 2010. Web. 12 Nov 2019.

Vancouver:

Chuang TC. The three-dimensional (3D) organization of telomeres during cellular transformation. [Internet] [Masters thesis]. University of Manitoba; 2010. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/1993/4228.

Council of Science Editors:

Chuang TC. The three-dimensional (3D) organization of telomeres during cellular transformation. [Masters Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4228


University of Texas Southwestern Medical Center

23. Dospoy, Patrick. Characterizing c-Myc Dependent Lung Cancers.

Degree: 2015, University of Texas Southwestern Medical Center

MYC is one of the most commonly deregulated oncogenes in human cancer, including breast, colorectal and lung. While mutations in myc are rare, MYC is… (more)

Subjects/Keywords: Lung Neoplasms; Peptide Fragments; Proto-Oncogene Proteins c-myc

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APA (6th Edition):

Dospoy, P. (2015). Characterizing c-Myc Dependent Lung Cancers. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 12, 2019. http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Web. 12 Nov 2019.

Vancouver:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

24. Gonzalez, Veronica. Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element.

Degree: 2010, University of Arizona

 The activated product of the c-MYC proto-oncogene is one of the strongest known activators of carcinogenesis. It has been estimated that as many as one-seventh… (more)

Subjects/Keywords: c-MYC; C23; G-quadruplex; Nucleolin; Oncogene; Transcription

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APA (6th Edition):

Gonzalez, V. (2010). Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element. (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195898

Chicago Manual of Style (16th Edition):

Gonzalez, Veronica. “Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element. ” 2010. Doctoral Dissertation, University of Arizona. Accessed November 12, 2019. http://hdl.handle.net/10150/195898.

MLA Handbook (7th Edition):

Gonzalez, Veronica. “Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element. ” 2010. Web. 12 Nov 2019.

Vancouver:

Gonzalez V. Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element. [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/10150/195898.

Council of Science Editors:

Gonzalez V. Defining the Role of Nucleolin on the Transcriptional Regulation of c-MYC through Modulation of the c-MYC NHE III1 Element. [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/195898


University of Sydney

25. Williams, Marissa. Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma .

Degree: 2018, University of Sydney

 Malignant Pleural Mesothelioma (MPM) is an aggressive asbestos related malignancy. The global downregulation of microRNA (miRNA) expression is common in MPM, however, the mechanisms driving… (more)

Subjects/Keywords: mesothelioma; microRNA; c-Myc; drug resistance; PD-L1

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APA (6th Edition):

Williams, M. (2018). Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/19754

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Marissa. “Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma .” 2018. Thesis, University of Sydney. Accessed November 12, 2019. http://hdl.handle.net/2123/19754.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Marissa. “Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma .” 2018. Web. 12 Nov 2019.

Vancouver:

Williams M. Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma . [Internet] [Thesis]. University of Sydney; 2018. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/2123/19754.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams M. Aberrant MicroRNA Expression in Malignant Pleural Mesothelioma . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/19754

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Josà Delano Barreto Marinho Filho. ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo.

Degree: PhD, 2012, Universidade Federal do Ceará

As cordiaquinonas sÃo naftoquinonas meroterpenÃides isolados de plantas pertencentes ao gÃnero Cordia com vÃrias atividades biolÃgicas descritas, incluindo atividades antifÃngica, larvicida e citotÃxica. O objetivo… (more)

Subjects/Keywords: FARMACOLOGIA; Estresse Oxidativo; Ensaios de SeleÃÃo de Medicamentos Antitumorais; Genes Myc; Glutationa; Oxidative Stress; Cordiaquinone; Anticancer; C-Myc; Glutathione

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APA (6th Edition):

Filho, J. D. B. M. (2012). ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo. (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8697 ;

Chicago Manual of Style (16th Edition):

Filho, Josà Delano Barreto Marinho. “ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo.” 2012. Doctoral Dissertation, Universidade Federal do Ceará. Accessed November 12, 2019. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8697 ;.

MLA Handbook (7th Edition):

Filho, Josà Delano Barreto Marinho. “ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo.” 2012. Web. 12 Nov 2019.

Vancouver:

Filho JDBM. ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo. [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2012. [cited 2019 Nov 12]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8697 ;.

Council of Science Editors:

Filho JDBM. ParticipaÃÃo das vias atm/atr e c-myc/gsh nos efeitos antitumorais da cordiaquinona J induzidos pelo estresse oxidativo. [Doctoral Dissertation]. Universidade Federal do Ceará 2012. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8697 ;

27. Dahl, Amy Kathleen. The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis.

Degree: Physiology and Pathophysiology, 2016, University of Manitoba

 Murine plasmacytoma models human cancers that involve deregulation of MYC. Overexpression and duplication of the immature colon carcinoma transcript 1 gene, Ict1, along with MYC(more)

Subjects/Keywords: Plasmacytoma; Myc; c-Myc; Ict1; Burkitt Lymphoma

…abbreviation table for c-MYC + Ict1 induced PreBmycER cells… …56 Experiment 2 SKY abbreviation table for c-MYC + Ict1 induced PreBmycER cells… …57 Experiment 3 SKY abbreviation table for c-MYC + Ict1 induced PreBmycER cells… …58 Experiment 1 SKY abbreviation table for c-MYC induced PreBmycER cells. ......59… …Experiment 2 SKY abbreviation table for c-MYC induced PreBmycER cells. ......60 Experiment 3 SKY… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dahl, A. K. (2016). The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31857

Chicago Manual of Style (16th Edition):

Dahl, Amy Kathleen. “The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis.” 2016. Masters Thesis, University of Manitoba. Accessed November 12, 2019. http://hdl.handle.net/1993/31857.

MLA Handbook (7th Edition):

Dahl, Amy Kathleen. “The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis.” 2016. Web. 12 Nov 2019.

Vancouver:

Dahl AK. The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/1993/31857.

Council of Science Editors:

Dahl AK. The role of ICT1 during MYC-deregulated fast-onset mouse plasmacytomagenesis. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31857

28. Saturno, Juvaní Lago. Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral.

Degree: Mestrado, Patologia Bucal, 2014, University of São Paulo

Neste trabalho foram analisados 30 casos de linfomas de células B da região oral, fixados em solução de formaldeído e incluídos em parafina, através da… (more)

Subjects/Keywords: B cells; Bcl-2; Bcl-2; Bcl-6; Bcl-6; c-Myc; c-Myc; Células B; Ciclina D; Cyclin D1; Linfomas; Lymphoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saturno, J. L. (2014). Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-06082014-144841/ ;

Chicago Manual of Style (16th Edition):

Saturno, Juvaní Lago. “Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral.” 2014. Masters Thesis, University of São Paulo. Accessed November 12, 2019. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-06082014-144841/ ;.

MLA Handbook (7th Edition):

Saturno, Juvaní Lago. “Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral.” 2014. Web. 12 Nov 2019.

Vancouver:

Saturno JL. Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Nov 12]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-06082014-144841/ ;.

Council of Science Editors:

Saturno JL. Análise imuno-histoquímica da Bcl-2, Bcl-6, c-Myc e ciclina D1 em linfomas de células B da região oral. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-06082014-144841/ ;


Université de Montréal

29. Kurbegovic, Almira. Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse .

Degree: 2015, Université de Montréal

 La polykystose rénale autosomique dominante (ADPKD) est une des maladies génétiques les plus communes. ADPKD se manifeste le plus souvent au stade adulte par la… (more)

Subjects/Keywords: ADPKD; Polycystine-1; Clivage GPS; Dommages rénaux; Souris; Foie; C-Myc; Pc2; ADPKD; Polycystin-1; GPS cleavage; AKI; Mouse models; Kidney; Liver; C-Myc; Pc2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kurbegovic, A. (2015). Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12095

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kurbegovic, Almira. “Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse .” 2015. Thesis, Université de Montréal. Accessed November 12, 2019. http://hdl.handle.net/1866/12095.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kurbegovic, Almira. “Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse .” 2015. Web. 12 Nov 2019.

Vancouver:

Kurbegovic A. Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse . [Internet] [Thesis]. Université de Montréal; 2015. [cited 2019 Nov 12]. Available from: http://hdl.handle.net/1866/12095.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kurbegovic A. Characterization of polycystin-1 in ADPKD pathogenetic mechanism : biogenesis and functional implications by genetic approaches in mouse . [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/12095

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

30. Zhao, Qiaoli. Molecular mechanisms of shikonin and its derivatives in cancer therapy.

Degree: 2015, Johannes Gutenberg Universität Mainz

The identification of molecular processes involved in cancer development and prognosis opened avenues for targeted therapies, which made treatment more tumor-specific and less toxic than… (more)

Subjects/Keywords: shikonin und derivatives, molekularen Mechanismen, Krebstherapie, EGFR, c-MYC; shikonin and derivatives, molecular mechanisms, cancer therapy, EGFR, c-MYC; Natural sciences and mathematics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, Q. (2015). Molecular mechanisms of shikonin and its derivatives in cancer therapy. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2015/4166/

Chicago Manual of Style (16th Edition):

Zhao, Qiaoli. “Molecular mechanisms of shikonin and its derivatives in cancer therapy.” 2015. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed November 12, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2015/4166/.

MLA Handbook (7th Edition):

Zhao, Qiaoli. “Molecular mechanisms of shikonin and its derivatives in cancer therapy.” 2015. Web. 12 Nov 2019.

Vancouver:

Zhao Q. Molecular mechanisms of shikonin and its derivatives in cancer therapy. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2015. [cited 2019 Nov 12]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2015/4166/.

Council of Science Editors:

Zhao Q. Molecular mechanisms of shikonin and its derivatives in cancer therapy. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2015. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2015/4166/

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