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You searched for subject:(c Jun N terminal kinase). Showing records 1 – 30 of 26940 total matches.

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1. Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.

Degree: 博士(医学), 2015, Nara Medical University / 奈良県立医科大学

Acute aortic dissection (AAD) is a life-threating disease; however, there is almost no effective pharmacotherapy for it. An increase in c-Jun N-terminal kinase (JNK) phosphorylation… (more)

Subjects/Keywords: Stretch; c-Jun N-terminal kinase; p38; Acute aortic dissection; Olmesartan

Page 1

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APA (6th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, K. (2015). Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. “Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.” 2015. Thesis, Nara Medical University / 奈良県立医科大学. Accessed May 09, 2021. http://hdl.handle.net/10564/2939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. “Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.” 2015. Web. 09 May 2021.

Vancouver:

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama K. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. [cited 2021 May 09]. Available from: http://hdl.handle.net/10564/2939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama K. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. Available from: http://hdl.handle.net/10564/2939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern Mississippi

2. Chen, Qichuan. The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.

Degree: MS, Biological Sciences, 2014, University of Southern Mississippi

  From a genetic and allelic modifier screen, we report that the Drosophila melanogaster T-box transcription factor midline (mid), a homolog to the human TBX20… (more)

Subjects/Keywords: mid; midline; Tbx20; dFOXO; C-Jun-N-terminal kinase; insulin receptor; Developmental Biology

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APA (6th Edition):

Chen, Q. (2014). The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. (Masters Thesis). University of Southern Mississippi. Retrieved from https://aquila.usm.edu/masters_theses/76

Chicago Manual of Style (16th Edition):

Chen, Qichuan. “The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.” 2014. Masters Thesis, University of Southern Mississippi. Accessed May 09, 2021. https://aquila.usm.edu/masters_theses/76.

MLA Handbook (7th Edition):

Chen, Qichuan. “The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.” 2014. Web. 09 May 2021.

Vancouver:

Chen Q. The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. [Internet] [Masters thesis]. University of Southern Mississippi; 2014. [cited 2021 May 09]. Available from: https://aquila.usm.edu/masters_theses/76.

Council of Science Editors:

Chen Q. The <i>Drosophila</i> T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. [Masters Thesis]. University of Southern Mississippi; 2014. Available from: https://aquila.usm.edu/masters_theses/76

3. Yu, Lola. Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization.

Degree: Cancer Biology, Molecular, Cell, and Cancer Biology, 2020, U of Massachusetts : Med

  Breast cancer is the second most common malignancy in the world, accounting for over 1.7 million new diagnoses and an estimated 500,000 deaths per… (more)

Subjects/Keywords: JNK; c-Jun N terminal kinase; IL-6; IL6; macrophage; MAPK; Cancer Biology

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APA (6th Edition):

Yu, L. (2020). Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization. (Masters Thesis). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/1094

Chicago Manual of Style (16th Edition):

Yu, Lola. “Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization.” 2020. Masters Thesis, U of Massachusetts : Med. Accessed May 09, 2021. https://escholarship.umassmed.edu/gsbs_diss/1094.

MLA Handbook (7th Edition):

Yu, Lola. “Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization.” 2020. Web. 09 May 2021.

Vancouver:

Yu L. Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization. [Internet] [Masters thesis]. U of Massachusetts : Med; 2020. [cited 2021 May 09]. Available from: https://escholarship.umassmed.edu/gsbs_diss/1094.

Council of Science Editors:

Yu L. Investigating the role of the c-Jun NH2-terminal kinase pathway in ErbB2-driven breast cancer and macrophage polarization. [Masters Thesis]. U of Massachusetts : Med; 2020. Available from: https://escholarship.umassmed.edu/gsbs_diss/1094


University of Washington

4. Kuhar, Jamie Rose. Mechanisms of Opioid Receptor Desensitization.

Degree: PhD, 2015, University of Washington

 Agonists targeting MOR are effective analgesics, but their clinical use is hindered by side effects, including tolerance and addiction. KOR agonists also produce analgesia, but… (more)

Subjects/Keywords: Arrestin; c-Jun N-terminal kinase; Fentanyl; Morphine; Opioid; p38; Pharmacology; pharmacology

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APA (6th Edition):

Kuhar, J. R. (2015). Mechanisms of Opioid Receptor Desensitization. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34122

Chicago Manual of Style (16th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Doctoral Dissertation, University of Washington. Accessed May 09, 2021. http://hdl.handle.net/1773/34122.

MLA Handbook (7th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Web. 09 May 2021.

Vancouver:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2021 May 09]. Available from: http://hdl.handle.net/1773/34122.

Council of Science Editors:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34122


University of Notre Dame

5. Anthony E Clemons. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.

Degree: Biological Sciences, 2014, University of Notre Dame

  Gene expression during embryogenesis impacts adult mosquito fitness, the ability of an organism to survive and transmit its genotype to offspring as compared to… (more)

Subjects/Keywords: Aeded aegypti; Puckered; C-Jun N-terminal Kinase; insulin signaling pathway; stress signaling pathway

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APA (6th Edition):

Clemons, A. E. (2014). Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. (Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/w089280462m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Clemons, Anthony E. “Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.” 2014. Thesis, University of Notre Dame. Accessed May 09, 2021. https://curate.nd.edu/show/w089280462m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Clemons, Anthony E. “Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.” 2014. Web. 09 May 2021.

Vancouver:

Clemons AE. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. [Internet] [Thesis]. University of Notre Dame; 2014. [cited 2021 May 09]. Available from: https://curate.nd.edu/show/w089280462m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Clemons AE. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. [Thesis]. University of Notre Dame; 2014. Available from: https://curate.nd.edu/show/w089280462m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

6. Jessica L. Lowry (7946000). A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.

Degree: 2013, University of Illinois – Chicago

 High levels of nitric oxide (NO) generated in the vasculature under inflammatory conditions are usually attributed to inducible nitric oxide synthase (iNOS), but the role… (more)

Subjects/Keywords: Uncategorized; Nitric Oxide; c-Jun-N-Terminal Kinase; endothelial nitric oxide synthase; inflammation; migration

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APA (6th Edition):

(7946000), J. L. L. (2013). A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/10054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

(7946000), Jessica L. Lowry. “A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.” 2013. Thesis, University of Illinois – Chicago. Accessed May 09, 2021. http://hdl.handle.net/10027/10054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

(7946000), Jessica L. Lowry. “A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.” 2013. Web. 09 May 2021.

Vancouver:

(7946000) JLL. A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2021 May 09]. Available from: http://hdl.handle.net/10027/10054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

(7946000) JLL. A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/10054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. Spurlock III, Charles Floyd. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.

Degree: PhD, Microbiology and Immunology, 2014, Vanderbilt University

 Rheumatoid arthritis is the most common serious autoimmune disease affecting almost one percent of the human population worldwide. Methotrexate is the most commonly used disease-modifying… (more)

Subjects/Keywords: methotrexate; autoimmune disease; inflammation; rheumatoid arthritis; cell cycle checkpoints; c-Jun-N-terminal kinase; p53; tetrahydrobiopterin; long non-coding RNA

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APA (6th Edition):

Spurlock III, C. F. (2014). Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10993

Chicago Manual of Style (16th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed May 09, 2021. http://hdl.handle.net/1803/10993.

MLA Handbook (7th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Web. 09 May 2021.

Vancouver:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 May 09]. Available from: http://hdl.handle.net/1803/10993.

Council of Science Editors:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/10993


University of Illinois – Chicago

8. Thomas Hanigan (7986395). Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery.

Degree: 2018, University of Illinois – Chicago

 Histone deacetylase (HDAC) activity is regulated by formation of co-regulator complexes and post-translational modification. Whether these mechanisms are transformed in cancer and how this affects… (more)

Subjects/Keywords: Uncategorized; Histone Deacetylase; HDAC; Chemical Biology; Photoreactive Probes; Target Engagement; Breast Cancer; C-Jun N-terminal Kinase; JNK

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APA (6th Edition):

(7986395), T. H. (2018). Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

(7986395), Thomas Hanigan. “Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery.” 2018. Thesis, University of Illinois – Chicago. Accessed May 09, 2021. http://hdl.handle.net/10027/22605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

(7986395), Thomas Hanigan. “Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery.” 2018. Web. 09 May 2021.

Vancouver:

(7986395) TH. Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2021 May 09]. Available from: http://hdl.handle.net/10027/22605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

(7986395) TH. Dynamic Post-Translational Control of HDAC Activity in Breast Cancer and Implications for Drug Discovery. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/22605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

9. Prasad, Karothu Durga. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 Pyrazoloanthrone and its analogues form the central core of the thesis and the work is focused on the evaluation of chemical and biological applications of… (more)

Subjects/Keywords: Pyrazoloanthrones; Pyrazoloanthrone; Anthrapyrazolones; C-Jun N-Terminal Kinase (JNK); Protein Kinase Inhibition; Cyanide/Fluoride Ion Detection; Picric Acid; Fluoremetric Chemosensor; Septic Shock; JNK Signals; 1, 9-pyrazoloanthrone; Organic Chemistry

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APA (6th Edition):

Prasad, K. D. (2018). Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/2980

Chicago Manual of Style (16th Edition):

Prasad, Karothu Durga. “Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed May 09, 2021. http://etd.iisc.ac.in/handle/2005/2980.

MLA Handbook (7th Edition):

Prasad, Karothu Durga. “Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.” 2018. Web. 09 May 2021.

Vancouver:

Prasad KD. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2021 May 09]. Available from: http://etd.iisc.ac.in/handle/2005/2980.

Council of Science Editors:

Prasad KD. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/2980


University of Kansas

10. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed May 09, 2021. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 09 May 2021.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 May 09]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Adelaide

11. Melino, Michelle. The role of c-jun N-terminal kinase (JNK) in human T cell function.

Degree: 2009, University of Adelaide

 T cells are involved in cellular pathways which enable the immune system to protect us against infection and cancer. However, the same mechanisms also allow… (more)

Subjects/Keywords: T cells; cytokines; c-jun N-terminal kinase; mitogen-activated protein kinases

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APA (6th Edition):

Melino, M. (2009). The role of c-jun N-terminal kinase (JNK) in human T cell function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Thesis, University of Adelaide. Accessed May 09, 2021. http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Web. 09 May 2021.

Vancouver:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Internet] [Thesis]. University of Adelaide; 2009. [cited 2021 May 09]. Available from: http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Thesis]. University of Adelaide; 2009. Available from: http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

12. Olausson, Josefin. Studies on microcirculation in insulin resistance.

Degree: 2015, University of Gothenburg / Göteborgs Universitet

 The overall aim of this thesis was to investigate the microcirculation in insulin resistance, with focus on the expression of endothelin-1, through a translational approach.… (more)

Subjects/Keywords: endothelin-1; type 2 diabetes; coronary heart disease; phosphodiesterase-5 inhibition; tadalafil; insulin resistance; endothelial dysfunction; c-Jun N-terminal kinase; nitric oxide; microdialysis

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APA (6th Edition):

Olausson, J. (2015). Studies on microcirculation in insulin resistance. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/39546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Olausson, Josefin. “Studies on microcirculation in insulin resistance.” 2015. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed May 09, 2021. http://hdl.handle.net/2077/39546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Olausson, Josefin. “Studies on microcirculation in insulin resistance.” 2015. Web. 09 May 2021.

Vancouver:

Olausson J. Studies on microcirculation in insulin resistance. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. [cited 2021 May 09]. Available from: http://hdl.handle.net/2077/39546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Olausson J. Studies on microcirculation in insulin resistance. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. Available from: http://hdl.handle.net/2077/39546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. 鈴木, 彰子. c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.

Degree: 医学博士, 2017, Iwate Medical University / 岩手医科大学

2016

Subjects/Keywords: Ballooned hepatocyte; Caspase 9; c-Jun N-terminal kinase; Rubicon; SP600125; Ballooned hepatocyte; Caspase 9; c-Jun N-terminal kinase; Rubicon; SP600125

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APA (6th Edition):

鈴木, . (2017). c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. (Thesis). Iwate Medical University / 岩手医科大学. Retrieved from http://id.nii.ac.jp/1181/00008785/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

鈴木, 彰子. “c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.” 2017. Thesis, Iwate Medical University / 岩手医科大学. Accessed May 09, 2021. http://id.nii.ac.jp/1181/00008785/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

鈴木, 彰子. “c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.” 2017. Web. 09 May 2021.

Vancouver:

鈴木 . c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. [Internet] [Thesis]. Iwate Medical University / 岩手医科大学; 2017. [cited 2021 May 09]. Available from: http://id.nii.ac.jp/1181/00008785/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

鈴木 . c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. [Thesis]. Iwate Medical University / 岩手医科大学; 2017. Available from: http://id.nii.ac.jp/1181/00008785/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

14. Nash, Rodney James. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.

Degree: 2014, University of Georgia

 CD9, a member of the tetraspanin superfamily, is expressed in several cell lines and is associated with cellular activities such as migration, proliferation and metastasis… (more)

Subjects/Keywords: Cluster of differentiation 9; Forssman antigen; Dolichos biflorus agglutinin; Mitogen activated protein kinase; Stress activated protein kinase/ C-Jun N terminal kinase; Extracellular signal regulated kinase; Apoptosis; Rescue and Juxtacrine Organization

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APA (6th Edition):

Nash, R. J. (2014). Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/23477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nash, Rodney James. “Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.” 2014. Thesis, University of Georgia. Accessed May 09, 2021. http://hdl.handle.net/10724/23477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nash, Rodney James. “Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.” 2014. Web. 09 May 2021.

Vancouver:

Nash RJ. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 May 09]. Available from: http://hdl.handle.net/10724/23477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nash RJ. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/23477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

15. Sharma, Neelesh. Mechanism of resistance to fumonisin B1-induced cytotoxicity.

Degree: 2014, University of Georgia

 Fumonisin B1 (FB1) is a mycotoxin produced by Fusarium verticillioides present on corn worldwide. Fumonisin B1 inhibits ceramide synthase and induces expression of cytokines in… (more)

Subjects/Keywords: Fumonisin B1; Resistance; Hepatotoxicity; Tumor necrosis factor ‡, Sphingosine kinase; c-Jun NH2-terminal kinase; T cell depletion

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APA (6th Edition):

Sharma, N. (2014). Mechanism of resistance to fumonisin B1-induced cytotoxicity. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/22792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Neelesh. “Mechanism of resistance to fumonisin B1-induced cytotoxicity.” 2014. Thesis, University of Georgia. Accessed May 09, 2021. http://hdl.handle.net/10724/22792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Neelesh. “Mechanism of resistance to fumonisin B1-induced cytotoxicity.” 2014. Web. 09 May 2021.

Vancouver:

Sharma N. Mechanism of resistance to fumonisin B1-induced cytotoxicity. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 May 09]. Available from: http://hdl.handle.net/10724/22792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma N. Mechanism of resistance to fumonisin B1-induced cytotoxicity. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/22792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. WOO SZE KWANG. VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS.

Degree: 2015, National University of Singapore

Subjects/Keywords: Virtual screening; Molecular modeling; c-jun N-terminal kinase; Pregnane X receptor; Mitogen-activated protein kinase enzymes; Nuclear receptors

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APA (6th Edition):

KWANG, W. S. (2015). VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/129129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KWANG, WOO SZE. “VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS.” 2015. Thesis, National University of Singapore. Accessed May 09, 2021. http://scholarbank.nus.edu.sg/handle/10635/129129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KWANG, WOO SZE. “VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS.” 2015. Web. 09 May 2021.

Vancouver:

KWANG WS. VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS. [Internet] [Thesis]. National University of Singapore; 2015. [cited 2021 May 09]. Available from: http://scholarbank.nus.edu.sg/handle/10635/129129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KWANG WS. VIRTUAL SCREENING FOR JNK INHIBITORS AND PREDICTION OF PXR ACTIVITY USING COMPUTATIONAL MODELS. [Thesis]. National University of Singapore; 2015. Available from: http://scholarbank.nus.edu.sg/handle/10635/129129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Gkirtzimanaki, Aikaterini. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Cancer can be perceived as a disease of communication between and within cells. The aberrations are pleiotropic, but mitogen‐activated protein kinase (MAPK) pathways feature prominently.… (more)

Subjects/Keywords: Καρκινογένεση πνεύμονα; Σηματοδότηση; Πρωτο-ογκογονίδια; Ογκοκατασταλτικό γονίδιο; Κινάση ενεργοποιούμενη από μιττογόνα; Απώλεια ετεροζυγωτίας; Επιγενετική ρύθμιση; Επιθηλιακά κύτταρα; MAP3K8 (Mittogen activated protein 3 kinase 8); TPL2 (Tumor progression locus 2); COT (Cancer Osaka Thyroid); p53; JNK (C-Jun n-Terminal kinase); ras; NPM (Nucleophosmin); MIR370 (Micro-RNA 370)

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APA (6th Edition):

Gkirtzimanaki, A. (2012). Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/29220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gkirtzimanaki, Aikaterini. “Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/29220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gkirtzimanaki, Aikaterini. “Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.” 2012. Web. 09 May 2021.

Vancouver:

Gkirtzimanaki A. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/29220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gkirtzimanaki A. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/29220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

18. Tang, Christine. Mechanisms of High Glucose-induced Decrease in β-cell Function.

Degree: 2010, University of Toronto

Chronic hyperglycemia, a hallmark of type 2 diabetes, can decrease β-cell function and mass (β-cell glucotoxicity); however, the mechanisms are incompletely understood. The objective was… (more)

Subjects/Keywords: Glucotoxicity; Oxidative Stress; Type 2 Diabetes; Endoplasmic Reticulum Stress; c-jun N-terminal kinase; 0719

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APA (6th Edition):

Tang, C. (2010). Mechanisms of High Glucose-induced Decrease in β-cell Function. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/26331

Chicago Manual of Style (16th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Doctoral Dissertation, University of Toronto. Accessed May 09, 2021. http://hdl.handle.net/1807/26331.

MLA Handbook (7th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Web. 09 May 2021.

Vancouver:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2021 May 09]. Available from: http://hdl.handle.net/1807/26331.

Council of Science Editors:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/26331


University of Arizona

19. Orton, Christopher R. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .

Degree: 2006, University of Arizona

 Defining the mechanics and consequences of protein adduction is crucial to understanding the toxicity of reactive electrophiles. Application of tandem mass spectrometry and data analysis… (more)

Subjects/Keywords: Proteomics; Protein Adduction Kinetics; Quantitative Mass Spectrometry; Glutathione S-transferase; c-Jun N-terminal Kinase Signaling

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APA (6th Edition):

Orton, C. R. (2006). Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194247

Chicago Manual of Style (16th Edition):

Orton, Christopher R. “Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .” 2006. Doctoral Dissertation, University of Arizona. Accessed May 09, 2021. http://hdl.handle.net/10150/194247.

MLA Handbook (7th Edition):

Orton, Christopher R. “Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .” 2006. Web. 09 May 2021.

Vancouver:

Orton CR. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2021 May 09]. Available from: http://hdl.handle.net/10150/194247.

Council of Science Editors:

Orton CR. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/194247


University of New South Wales

20. Wang, Fang. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.

Degree: Clinical School - St George Hospital, 2006, University of New South Wales

 To explore the potential mechanisms of tendon degeneration, we investigated the role of c-Jun N-terminal Kinase (JNK) activation and the regulation of matrix metalloproteinase 1… (more)

Subjects/Keywords: supraspinatus tendon; c-Jun N-terminal kinase (JNK); matrix metalloproteinase 1 (MMP1); rotator cuff tear; oxidative stress; hydrogen peroxide

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APA (6th Edition):

Wang, F. (2006). Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Wang, Fang. “Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.” 2006. Masters Thesis, University of New South Wales. Accessed May 09, 2021. http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true.

MLA Handbook (7th Edition):

Wang, Fang. “Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.” 2006. Web. 09 May 2021.

Vancouver:

Wang F. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. [Internet] [Masters thesis]. University of New South Wales; 2006. [cited 2021 May 09]. Available from: http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true.

Council of Science Editors:

Wang F. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. [Masters Thesis]. University of New South Wales; 2006. Available from: http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true

21. Καρκαλή, Αικατερίνη. Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 MAPK (Mitogen Activated Protein Kinases) signaling pathways have been extensively studied and are known to control multiple biological processes, such as proliferation, apoptosis, differentiation and… (more)

Subjects/Keywords: Φωσφατάσες διπλής εξειδίκευσης της Drosophila melanogaster; Ενεργοποιούµενες από µιτογόνα ερεθίσµατα κινάσες; Οξειδωτικό στρες; ΦΩΣΦΟΡΥΛΙΩΣΗ ΠΡΩΤΕΙΝΩΝ; Εµβρυϊκό Νευρικό Σύστηµα της Drosophila melanogaster; Φωσφατάση Puckered; Κινάση του N-τελικού άκρου του µεταγραφικού παράγοντα c-Jun; Κινάση p38; Dual Specificity Phosphatase’s of Drosophila melanogaster; Mitogen – Activated Protein Kinases (MAPKs); Oxidative stress; PROTEIN PHOSPHORYLATION; Embryonic Nervous System of Drosophila melanogaster; Puckered phosphatase; c-Jun N-terminal kinase (JNK); p38 kinase

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APA (6th Edition):

Καρκαλή, . . (2012). Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Web. 09 May 2021.

Vancouver:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

22. Gopalan, Archana. Targeting breast cancer with natural forms of vitamin E and simvastatin.

Degree: PhD, Nutritional Sciences, 2012, University of Texas – Austin

 Breast cancer is the second leading cause of death due to cancer in women. A number of effective therapeutic strategies have been implemented in clinics… (more)

Subjects/Keywords: Breast cancer; Vitamin E; Simvastatin; Apoptosis; Stem cells; TIC; Mevalonate pathway; Ceramide synthesis pathways; Death receptor 5 (DR5); c-Jun N-terminal kinase/C/EBP homologous protein (JNK/CHOP); FLICE inhibitory protein (c-FLIP); B-cell lymphoma 2 (Bcl-2); Survivin; Signal transducer and activator of transcription 3 (Stat3); Bcl-xL; Cyclin D1; c-Myc

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APA (6th Edition):

Gopalan, A. (2012). Targeting breast cancer with natural forms of vitamin E and simvastatin. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2012-05-5520

Chicago Manual of Style (16th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Doctoral Dissertation, University of Texas – Austin. Accessed May 09, 2021. http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

MLA Handbook (7th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Web. 09 May 2021.

Vancouver:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2012. [cited 2021 May 09]. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

Council of Science Editors:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Doctoral Dissertation]. University of Texas – Austin; 2012. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520

23. Henrie, Hélène. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.

Degree: Docteur es, Physiologie, physiopathologie, 2017, Université Paris-Saclay (ComUE)

Les microtubules sont des éléments dynamiques du cytosquelette qui contrôlent à la fois l’organisation du cytoplasme, la polarité, la migration et la division cellulaire. Notre… (more)

Subjects/Keywords: Microtubules; JNK (c-Jun NH2-Terminal Kinase); CLIP-170 (Cytoplasmic Linker Protein of 170 kDa); Beta-Tubuline; Sauvetage microtubulaire; Cellules épithéliales; Microtubules; JNK (c-Jun NH2-Terminal Kinase); CLIP-170 (Cytoplasmic Linker Protein of 170 kDa); Beta-Tubulin; Microtubule rescue; Epithelial cells

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APA (6th Edition):

Henrie, H. (2017). Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2017SACLS461

Chicago Manual of Style (16th Edition):

Henrie, Hélène. “Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed May 09, 2021. http://www.theses.fr/2017SACLS461.

MLA Handbook (7th Edition):

Henrie, Hélène. “Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.” 2017. Web. 09 May 2021.

Vancouver:

Henrie H. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2017. [cited 2021 May 09]. Available from: http://www.theses.fr/2017SACLS461.

Council of Science Editors:

Henrie H. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2017. Available from: http://www.theses.fr/2017SACLS461


Stellenbosch University

24. Fan, WenJun. Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats.

Degree: PhD, Biomedical Sciences, 2017, Stellenbosch University

 ENGLISH ABSTRACT : Obesity is an important risk factor for the development of insulin resistance, the metabolic syndrome and diabetes and has also been implicated… (more)

Subjects/Keywords: Ischaemia; Myocardial protection; Jun N-terminal Kinase; Substrate metabolism; Rats as laboratory animals; UCTD; Obesity in animals; Myocardial reperfusion; Insulin resistance  – Animal models

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fan, W. (2017). Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/100943

Chicago Manual of Style (16th Edition):

Fan, WenJun. “Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats.” 2017. Doctoral Dissertation, Stellenbosch University. Accessed May 09, 2021. http://hdl.handle.net/10019.1/100943.

MLA Handbook (7th Edition):

Fan, WenJun. “Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats.” 2017. Web. 09 May 2021.

Vancouver:

Fan W. Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats. [Internet] [Doctoral dissertation]. Stellenbosch University; 2017. [cited 2021 May 09]. Available from: http://hdl.handle.net/10019.1/100943.

Council of Science Editors:

Fan W. Effect of different substrates on functional performance and kinase activation during reperfusion after ischaemia in hearts from obese insulin resistant rats. [Doctoral Dissertation]. Stellenbosch University; 2017. Available from: http://hdl.handle.net/10019.1/100943

25. Λαγκαδινού, Ελένη. Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.

Degree: 2008, University of Patras

Η θεραπεία της Οξείας Μυελογενούς Λευχαιμίας (ΟΜΛ) είναι συχνά ανεπιτυχής λόγω ανάπτυξης κυτταρικής αντίστασης στα αντιλευχαιμικά φάρμακα. Εκτός από την έκφραση Ρ-γλυκοπρωτείνης στα λευχαιμικά κύτταρα,… (more)

Subjects/Keywords: Οξεία μυελογενής λευχαιμία (ΟΜΛ); Χημειοαντίσταση; Μηχανισμοί μεταγωγής σήματος; 616.994 190 61; Acute myeloid leukemia (AML); Drug resistance; Jun N-terminal Kinase (JNK)

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APA (6th Edition):

Λαγκαδινού, . (2008). Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. (Doctoral Dissertation). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/2208

Chicago Manual of Style (16th Edition):

Λαγκαδινού, Ελένη. “Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.” 2008. Doctoral Dissertation, University of Patras. Accessed May 09, 2021. http://nemertes.lis.upatras.gr/jspui/handle/10889/2208.

MLA Handbook (7th Edition):

Λαγκαδινού, Ελένη. “Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.” 2008. Web. 09 May 2021.

Vancouver:

Λαγκαδινού . Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. [Internet] [Doctoral dissertation]. University of Patras; 2008. [cited 2021 May 09]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/2208.

Council of Science Editors:

Λαγκαδινού . Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. [Doctoral Dissertation]. University of Patras; 2008. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/2208

26. NGUYEN, THI KIM OANH. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.

Degree: 2018, Ajou University

Mitochondria are dynamic organelles essential for the life and death of the cells. The dynamic nature of mitochondria allows them to organize a network of… (more)

Subjects/Keywords: DNA damage signaling; Mitochondria fission; Nuclear; JNK, Jun N-terminal Kinase; SIRT1, Sirtuin 1; ATM, ataxia-telangiectasia mutated; DNA 손상신호; 미토콘드리아 분열;

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APA (6th Edition):

NGUYEN, T. K. O. (2018). Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

NGUYEN, THI KIM OANH. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Thesis, Ajou University. Accessed May 09, 2021. http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

NGUYEN, THI KIM OANH. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Web. 09 May 2021.

Vancouver:

NGUYEN TKO. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Internet] [Thesis]. Ajou University; 2018. [cited 2021 May 09]. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

NGUYEN TKO. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Thesis]. Ajou University; 2018. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Zamani, Marzieh. The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells.

Degree: PhD, 2013, University of Hertfordshire

 Nitric oxide (NO) is an important biological molecule within the body, which over production of this molecule in response to different stimulations can cause various… (more)

Subjects/Keywords: 616.0473; Nitric Oxide (NO); Nitric Oxide Synthase (NOS); Inducible Nitric Oxide Synthase (iNOS); c-Jun N-terminal Kinase (JNK); Activator Protein-1 (AP-1); Mitogen activated Protein Kinase (MAPK); Cationic Amino Acid Transporters (CAT); Vascular Smooth Muscle Cells (VSMCs); J774 macrophages; Cardiovascular disease; Inflammation; soluble Guanylate Cyclase (sGC); L-arginine; Bacterial Lipopolysaccharide (LPS); Interferon-gamma (IFN-?); TAM-67; a-Fos; SP600125; MAP kinase kinases (MKK4)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zamani, M. (2013). The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells. (Doctoral Dissertation). University of Hertfordshire. Retrieved from http://hdl.handle.net/2299/10626

Chicago Manual of Style (16th Edition):

Zamani, Marzieh. “The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells.” 2013. Doctoral Dissertation, University of Hertfordshire. Accessed May 09, 2021. http://hdl.handle.net/2299/10626.

MLA Handbook (7th Edition):

Zamani, Marzieh. “The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells.” 2013. Web. 09 May 2021.

Vancouver:

Zamani M. The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells. [Internet] [Doctoral dissertation]. University of Hertfordshire; 2013. [cited 2021 May 09]. Available from: http://hdl.handle.net/2299/10626.

Council of Science Editors:

Zamani M. The role of the JNK/AP-1 pathway in the induction of iNOS and CATs in vascular cells. [Doctoral Dissertation]. University of Hertfordshire; 2013. Available from: http://hdl.handle.net/2299/10626

28. Οικονομίδου, Όλγα. Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.

Degree: 2005, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Subjects/Keywords: Υποδοχέας γλυκοκορτικοειδών GR; Πρωτεΐνη ενεργοποίησης-1; Πυρηνικός παράγοντας κΒ; c-jun N-terminal κινάση; Πρωτεΐνη αναστολέας του πυρηνικού παράγοντα; Συστηματικός ερυθηματώδης λύκος ( ΣΕΛ); Μονοπάτι μεταγωγής σήματος; Λεμφοκύτταρα; Glucocorticoid receptor (GR); Activating protein-1; Nuclear factor kB; c-jun N-terminal kinase; IkBa; Systemic lupus erythematosus ( SLE); Signal transduction; Lymphocytes

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APA (6th Edition):

Οικονομίδου, . . (2005). Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/20025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Οικονομίδου, Όλγα. “Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.” 2005. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/20025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Οικονομίδου, Όλγα. “Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.” 2005. Web. 09 May 2021.

Vancouver:

Οικονομίδου . Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/20025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Οικονομίδου . Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. Available from: http://hdl.handle.net/10442/hedi/20025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of St. Andrews

29. Borger, Eva. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia.

Degree: 2012, University of St. Andrews

 Dementia causes an increasing social and economic burden worldwide, demanding action regarding its diagnosis, treatment and everyday management. Recent years have seen many advances in… (more)

Subjects/Keywords: Alzheimer's disease; Frontotemporal dementia; Endophilin; Peroxiredoxin; EFHD2; C-Jun N-tmerinal kinase; QP552.A45B7; Alzheimer's disease – Molecular aspects; Presenile dementia; Amyloid beta-protein; Cellular signal transduction

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APA (6th Edition):

Borger, E. (2012). New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia. (Doctoral Dissertation). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/3092

Chicago Manual of Style (16th Edition):

Borger, Eva. “New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia.” 2012. Doctoral Dissertation, University of St. Andrews. Accessed May 09, 2021. http://hdl.handle.net/10023/3092.

MLA Handbook (7th Edition):

Borger, Eva. “New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia.” 2012. Web. 09 May 2021.

Vancouver:

Borger E. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia. [Internet] [Doctoral dissertation]. University of St. Andrews; 2012. [cited 2021 May 09]. Available from: http://hdl.handle.net/10023/3092.

Council of Science Editors:

Borger E. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia. [Doctoral Dissertation]. University of St. Andrews; 2012. Available from: http://hdl.handle.net/10023/3092


University of Southern California

30. Tao, Litao. Investigation of the molecular mechanisms of ototoxicity.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2014, University of Southern California

 Sensory hair cells are essential for transforming the mechanical vibrations of sound into electric signals that our nervous system can interpret. However, sensory hair cells… (more)

Subjects/Keywords: aminoglycoside antibiotics; ototoxicity; cyclin-dependent kinase 2; c-Jun; RNA sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tao, L. (2014). Investigation of the molecular mechanisms of ototoxicity. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651

Chicago Manual of Style (16th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Doctoral Dissertation, University of Southern California. Accessed May 09, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651.

MLA Handbook (7th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Web. 09 May 2021.

Vancouver:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2021 May 09]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651.

Council of Science Editors:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651

[1] [2] [3] [4] [5] … [898]

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