Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

Dept: Cancer Biology

You searched for subject:(breast). Showing records 1 – 30 of 30 total matches.

Search Limiters

Last 2 Years | English Only

▼ Search Limiters


Vanderbilt University

1. Bates, Andreia LaShonne. Roles of Fibroblast MMP2 in Breast to Lung Metastasis.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 CANCER BIOLOGY Roles of Fibroblast MMP2 in Breast to Lung Metastasis Andreia L. Bates Dissertation under the direction of Barbara Fingleton, PhD Breast cancer five-year… (more)

Subjects/Keywords: MMP2; fibroblasts; metastasis; breast cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bates, A. L. (2015). Roles of Fibroblast MMP2 in Breast to Lung Metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03172015-152637/ ;

Chicago Manual of Style (16th Edition):

Bates, Andreia LaShonne. “Roles of Fibroblast MMP2 in Breast to Lung Metastasis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-03172015-152637/ ;.

MLA Handbook (7th Edition):

Bates, Andreia LaShonne. “Roles of Fibroblast MMP2 in Breast to Lung Metastasis.” 2015. Web. 18 Oct 2019.

Vancouver:

Bates AL. Roles of Fibroblast MMP2 in Breast to Lung Metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-03172015-152637/ ;.

Council of Science Editors:

Bates AL. Roles of Fibroblast MMP2 in Breast to Lung Metastasis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-03172015-152637/ ;


Vanderbilt University

2. Johnson, Rachelle Whitney. Molecular mechanisms of Gli2 regulation in osteolytic bone disease.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

Breast cancer frequently metastasizes to the skeleton with severe consequence to patient quality of life. Patients who develop bone metastases suffer from severe bone pain… (more)

Subjects/Keywords: gli2; pthrp; breast cancer; bone; metastasis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnson, R. W. (2011). Molecular mechanisms of Gli2 regulation in osteolytic bone disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08162011-122923/ ;

Chicago Manual of Style (16th Edition):

Johnson, Rachelle Whitney. “Molecular mechanisms of Gli2 regulation in osteolytic bone disease.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu//available/etd-08162011-122923/ ;.

MLA Handbook (7th Edition):

Johnson, Rachelle Whitney. “Molecular mechanisms of Gli2 regulation in osteolytic bone disease.” 2011. Web. 18 Oct 2019.

Vancouver:

Johnson RW. Molecular mechanisms of Gli2 regulation in osteolytic bone disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-08162011-122923/ ;.

Council of Science Editors:

Johnson RW. Molecular mechanisms of Gli2 regulation in osteolytic bone disease. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu//available/etd-08162011-122923/ ;


Vanderbilt University

3. Pickup, Michael William. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

 Dissertation under the direction of Professor Harold L. Moses Transforming Growth Factor Beta (TGF-β) is acts as both a tumor suppressor and promoter in the… (more)

Subjects/Keywords: Breast Cancer; TGF-Beta; Fibroblast; Tumor Microenvironment

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pickup, M. W. (2013). Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10092013-140015/ ;

Chicago Manual of Style (16th Edition):

Pickup, Michael William. “Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-10092013-140015/ ;.

MLA Handbook (7th Edition):

Pickup, Michael William. “Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.” 2013. Web. 18 Oct 2019.

Vancouver:

Pickup MW. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-10092013-140015/ ;.

Council of Science Editors:

Pickup MW. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-10092013-140015/ ;


Wayne State University

4. Irish, Jonathan Curtis. Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells.

Degree: PhD, Cancer Biology, 2016, Wayne State University

Breast cancer is the most common cancer and the second leading cause of cancer death in women. While ER-positive breast cancer subtypes are initially… (more)

Subjects/Keywords: Breast; Cancer; chromatin; ER; SUM44; WHSC1L1; Oncology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Irish, J. C. (2016). Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1641

Chicago Manual of Style (16th Edition):

Irish, Jonathan Curtis. “Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells.” 2016. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/1641.

MLA Handbook (7th Edition):

Irish, Jonathan Curtis. “Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells.” 2016. Web. 18 Oct 2019.

Vancouver:

Irish JC. Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells. [Internet] [Doctoral dissertation]. Wayne State University; 2016. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1641.

Council of Science Editors:

Irish JC. Whsc1l1 Regulates Estrogen Receptor Activity In Sum44 Breast Cancer Cells. [Doctoral Dissertation]. Wayne State University; 2016. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1641


Wayne State University

5. Holowatyj, Andreana Holowatyj. Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions.

Degree: PhD, Cancer Biology, 2017, Wayne State University

  Despite converging incidence rates for breast cancers by race, disparities in mortality persist where black women suffer from poorer prognosis compared to white counterparts.… (more)

Subjects/Keywords: benign breast disease; black; breast cancer; estrogen; inflammation; race; Molecular Biology; Oncology; Public Health

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Holowatyj, A. H. (2017). Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1709

Chicago Manual of Style (16th Edition):

Holowatyj, Andreana Holowatyj. “Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions.” 2017. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/1709.

MLA Handbook (7th Edition):

Holowatyj, Andreana Holowatyj. “Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions.” 2017. Web. 18 Oct 2019.

Vancouver:

Holowatyj AH. Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1709.

Council of Science Editors:

Holowatyj AH. Clinicopathology And Molecular Determinants Underlying Benign Breast And Breast Cancer Lesions. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1709


Vanderbilt University

6. Atwood, Allison Annette. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 Overexpression of activated oncogenes such as Ras(V12) in primary cells induces senescence rather than transformation, thus suppressing tumorigenesis. C/EBPbeta is required for Ras(V12)-induced senescence in… (more)

Subjects/Keywords: Ras; breast cancer; IL6; senescence; C/EBPbeta; sumoylation; degradation; phosphorylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Atwood, A. A. (2010). Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12102010-121704/ ;

Chicago Manual of Style (16th Edition):

Atwood, Allison Annette. “Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-12102010-121704/ ;.

MLA Handbook (7th Edition):

Atwood, Allison Annette. “Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.” 2010. Web. 18 Oct 2019.

Vancouver:

Atwood AA. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-12102010-121704/ ;.

Council of Science Editors:

Atwood AA. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-12102010-121704/ ;


Vanderbilt University

7. Vaught, David Bryan. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Eph receptor tyrosine kinases are membrane bound receptors often expressed by normal epithelial cells but are frequently overexpressed in many human cancers. Of the many… (more)

Subjects/Keywords: Branching Morphogenesis; Mammary Gland; Bone Metastasis; Breast Cancer; EphA2; IL-6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vaught, D. B. (2011). EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03282011-170312/ ;

Chicago Manual of Style (16th Edition):

Vaught, David Bryan. “EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-03282011-170312/ ;.

MLA Handbook (7th Edition):

Vaught, David Bryan. “EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.” 2011. Web. 18 Oct 2019.

Vancouver:

Vaught DB. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-03282011-170312/ ;.

Council of Science Editors:

Vaught DB. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-03282011-170312/ ;


Vanderbilt University

8. Bankaitis, Katherine Venmar. The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Epithelial cancers of the lung, colon, breast, and pancreas comprise the top four most deadly cancers. Progress has been made in treating primary epithelial tumors,… (more)

Subjects/Keywords: metastasis; breast cancer; colon cancer; IL4 receptor; IL4

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bankaitis, K. V. (2016). The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03132016-204718/ ;

Chicago Manual of Style (16th Edition):

Bankaitis, Katherine Venmar. “The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu//available/etd-03132016-204718/ ;.

MLA Handbook (7th Edition):

Bankaitis, Katherine Venmar. “The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis.” 2016. Web. 18 Oct 2019.

Vancouver:

Bankaitis KV. The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-03132016-204718/ ;.

Council of Science Editors:

Bankaitis KV. The epithelial IL4 receptor signaling axis mediates enhanced breast and colon tumor growth and metastasis. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu//available/etd-03132016-204718/ ;


Vanderbilt University

9. Williams, Michelle Marie. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.

Degree: PhD, Cancer Biology, 2017, Vanderbilt University

 Evasion of cell death is essential to every step of tumorigenesis. Anti-apoptotic Bcl-2 family proteins are master inhibitors of cell death, and thus are often… (more)

Subjects/Keywords: Mcl-1; therapeutic resistance; Bcl-2 family proteins; luminal breast cancers

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, M. M. (2017). Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07182017-085645/ ;

Chicago Manual of Style (16th Edition):

Williams, Michelle Marie. “Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-07182017-085645/ ;.

MLA Handbook (7th Edition):

Williams, Michelle Marie. “Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.” 2017. Web. 18 Oct 2019.

Vancouver:

Williams MM. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-07182017-085645/ ;.

Council of Science Editors:

Williams MM. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-07182017-085645/ ;


Vanderbilt University

10. Youngblood, Victoria Marie. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Dysregulation of receptor tyrosine kinases (RTKs) contributes to cellular transformation and cancer progression by disrupting key metabolic signaling pathways. The EPHA2 RTK is overexpressed in… (more)

Subjects/Keywords: breast cancer; ephrin-A1; EPHA2; glutaminolysis; glutamine metabolism; tumor metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Youngblood, V. M. (2016). The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03172016-102302/ ;

Chicago Manual of Style (16th Edition):

Youngblood, Victoria Marie. “The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-03172016-102302/ ;.

MLA Handbook (7th Edition):

Youngblood, Victoria Marie. “The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.” 2016. Web. 18 Oct 2019.

Vancouver:

Youngblood VM. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-03172016-102302/ ;.

Council of Science Editors:

Youngblood VM. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu/available/etd-03172016-102302/ ;


Wayne State University

11. Madden, Julie Marie. Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer.

Degree: PhD, Cancer Biology, 2014, Wayne State University

  Triple negative breast cancer (TNBC) patients suffer from a highly malignant and aggressive cancer that lacks an effective targeted therapeutic. Although many TNBCs, both… (more)

Subjects/Keywords: Breast cancer; EGFR; eIF4B; STAT3; Cell Biology; Oncology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Madden, J. M. (2014). Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1017

Chicago Manual of Style (16th Edition):

Madden, Julie Marie. “Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer.” 2014. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/1017.

MLA Handbook (7th Edition):

Madden, Julie Marie. “Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer.” 2014. Web. 18 Oct 2019.

Vancouver:

Madden JM. Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer. [Internet] [Doctoral dissertation]. Wayne State University; 2014. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1017.

Council of Science Editors:

Madden JM. Combating Resistance To Epidermal Growth Factor Recpetor Inhibitors In Triple Negative Breast Cancer. [Doctoral Dissertation]. Wayne State University; 2014. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1017


Wayne State University

12. Haagenson, Kelly. Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity.

Degree: PhD, Cancer Biology, 2013, Wayne State University

  ABSTRACT EXPRESSION AND REGULATION OF MAP KINASE PHOSPHATASES 1 and 2 IN BREAST CANCER TAMOXIFEN SENSITIVITY by KELLY K. HAAGENSON May 2013 Advisor: Dr.… (more)

Subjects/Keywords: Breast Cancer; MAP Kinase Phosphatases; Tamoxifen Sensitivity; Biology; Oncology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haagenson, K. (2013). Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/659

Chicago Manual of Style (16th Edition):

Haagenson, Kelly. “Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity.” 2013. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/659.

MLA Handbook (7th Edition):

Haagenson, Kelly. “Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity.” 2013. Web. 18 Oct 2019.

Vancouver:

Haagenson K. Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/659.

Council of Science Editors:

Haagenson K. Expression And Regulation Of Map Kinase Phosphatases 1 And 2 In Breast Cancer Tamoxifen Sensitivity. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/659


Wayne State University

13. Ventro, Daniela. The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection.

Degree: PhD, Cancer Biology, 2013, Wayne State University

  THE BCA2 & AMPK PARADIGM: UNRAVELING THE CANCER CONNECTION By DANIELA (BUAC) VENTRO December 2013 Advisor: Dr. Q Ping Dou and Angelika M. Burger… (more)

Subjects/Keywords: Breast Cancer; Cell metabolism; E3 Ligase; Ubiquitin-Proteasome System; Oncology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ventro, D. (2013). The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/873

Chicago Manual of Style (16th Edition):

Ventro, Daniela. “The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection.” 2013. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/873.

MLA Handbook (7th Edition):

Ventro, Daniela. “The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection.” 2013. Web. 18 Oct 2019.

Vancouver:

Ventro D. The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/873.

Council of Science Editors:

Ventro D. The Bca2 And Ampk Paradigm: Unraveling The Cancer Connection. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/873


Wayne State University

14. Victor, Bernadette Caroline. Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer.

Degree: PhD, Cancer Biology, 2011, Wayne State University

  FUNCTIONAL IN VITRO ANALYSES OF LIPID RAFT-ASSOCIATED CATHEPSIN B: IMPLICATION FOR THE INVASIVE PHENOTYPE OF INFLAMMATORY BREAST CANCER by BERNADETTE C. VICTOR December 2011… (more)

Subjects/Keywords: cathepsin B, caveolae, inflammatory breast cancer, invasion, proteolysis; Cell Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Victor, B. C. (2011). Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/400

Chicago Manual of Style (16th Edition):

Victor, Bernadette Caroline. “Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer.” 2011. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/400.

MLA Handbook (7th Edition):

Victor, Bernadette Caroline. “Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer.” 2011. Web. 18 Oct 2019.

Vancouver:

Victor BC. Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/400.

Council of Science Editors:

Victor BC. Functional in vitro analyses of lipid raft-associated cathepsin b: implication for the invasive phenotype of inflammatory breast cancer. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/400


Wayne State University

15. Mcknight, Brooke. Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer.

Degree: PhD, Cancer Biology, 2019, Wayne State University

  With a broad spectrum of therapies available for treating breast cancer, the need for personalized medicine tailoring the cure according to phenotype is evident.… (more)

Subjects/Keywords: breast cancer; EGFR; HER2; immunoPET; immunotherapy; Bioimaging and Biomedical Optics; Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mcknight, B. (2019). Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/2176

Chicago Manual of Style (16th Edition):

Mcknight, Brooke. “Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer.” 2019. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/2176.

MLA Handbook (7th Edition):

Mcknight, Brooke. “Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer.” 2019. Web. 18 Oct 2019.

Vancouver:

Mcknight B. Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer. [Internet] [Doctoral dissertation]. Wayne State University; 2019. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2176.

Council of Science Editors:

Mcknight B. Utilizing Immunopet To Measure Tumor Response To Treatment In Breast Cancer. [Doctoral Dissertation]. Wayne State University; 2019. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2176

16. Jiagge, Evelyn. Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities.

Degree: PhD, Cancer Biology, 2017, University of Michigan

 Triple negative breast cancer is one of the most aggressive breast cancer subtypes, for which there are no approved targeted therapies. In US alone, the… (more)

Subjects/Keywords: breast cancer; triple negative breast cancer; breast cancer stem cells; African ancestry and breast cancer; Genetics; Internal Medicine and Specialties; Medicine (General); Molecular, Cellular and Developmental Biology; Pathology; Science (General); Health Sciences; Science

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jiagge, E. (2017). Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/136936

Chicago Manual of Style (16th Edition):

Jiagge, Evelyn. “Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities.” 2017. Doctoral Dissertation, University of Michigan. Accessed October 18, 2019. http://hdl.handle.net/2027.42/136936.

MLA Handbook (7th Edition):

Jiagge, Evelyn. “Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities.” 2017. Web. 18 Oct 2019.

Vancouver:

Jiagge E. Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2027.42/136936.

Council of Science Editors:

Jiagge E. Understanding the Biology of Triple Negative Breast Cancer and Breast Cancer Stem Cells in Patients of Diverse Ethnicities. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/136936

17. Rao, Meghana Nallamala. The Role of VAPB in Breast Cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 VAPB (VAMP-associated protein B) is an endoplasmic reticulum protein that regulates multiple biological functions. VAPB protein expression is elevated in human breast cancers and correlates… (more)

Subjects/Keywords: cancer; breast; VAPB; secretion; AKT

…7 Table 2. VAPB-expressing human breast cancer cell lines… …5 Figure 1.2 Analysis of VAPB expression in human breast cancer ....................... 19… …breast cancer cells lines and correlates with poor clinical outcome. Despite aberrant… …presented herein investigate how VAPB regulates breast tumor cell proliferation, invasion and in… …invasive potential in breast tumor cells. We also identified novel VAPB interacting proteins that… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rao, M. N. (2012). The Role of VAPB in Breast Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11292012-144810/ ;

Chicago Manual of Style (16th Edition):

Rao, Meghana Nallamala. “The Role of VAPB in Breast Cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-11292012-144810/ ;.

MLA Handbook (7th Edition):

Rao, Meghana Nallamala. “The Role of VAPB in Breast Cancer.” 2012. Web. 18 Oct 2019.

Vancouver:

Rao MN. The Role of VAPB in Breast Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-11292012-144810/ ;.

Council of Science Editors:

Rao MN. The Role of VAPB in Breast Cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-11292012-144810/ ;


University of Michigan

18. Altemus, Megan. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.

Degree: PhD, Cancer Biology, 2019, University of Michigan

Breast cancer has the highest incidence rates of all cancer types among women in the United States, and the second highest mortality. While survival is… (more)

Subjects/Keywords: breast cancer; glycogen metabolism; brain metastasis; microfluidic devices; Oncology and Hematology; Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Altemus, M. (2019). Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/151668

Chicago Manual of Style (16th Edition):

Altemus, Megan. “Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.” 2019. Doctoral Dissertation, University of Michigan. Accessed October 18, 2019. http://hdl.handle.net/2027.42/151668.

MLA Handbook (7th Edition):

Altemus, Megan. “Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.” 2019. Web. 18 Oct 2019.

Vancouver:

Altemus M. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2027.42/151668.

Council of Science Editors:

Altemus M. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/151668

19. Suthar,Tejal. Significance of biochemical markers in the management of breast cancer;.

Degree: Cancer Biology, 2015, Gujarat University

Abstract Not Available

Data not available

Advisors/Committee Members: Bhatavdekar,J M.

Subjects/Keywords: Biochemical; Breast; Cancer; Histopathologically; Lymph; Metastatic; Prolactin; Tumors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Suthar,Tejal. (2015). Significance of biochemical markers in the management of breast cancer;. (Thesis). Gujarat University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/48465

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suthar,Tejal. “Significance of biochemical markers in the management of breast cancer;.” 2015. Thesis, Gujarat University. Accessed October 18, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/48465.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suthar,Tejal. “Significance of biochemical markers in the management of breast cancer;.” 2015. Web. 18 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Suthar,Tejal. Significance of biochemical markers in the management of breast cancer;. [Internet] [Thesis]. Gujarat University; 2015. [cited 2019 Oct 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/48465.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suthar,Tejal. Significance of biochemical markers in the management of breast cancer;. [Thesis]. Gujarat University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/48465

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

20. Karelia,Nilkamal H. Some biochemical studies of human breast cancer;.

Degree: Cancer Biology, 2015, Gujarat University

Abstract Not Available

Data not available

Advisors/Committee Members: Bhatavdekarj,J M.

Subjects/Keywords: Biochemical; Breast; Cancer; Gonadotropins; Human; Postmenopausal; Prognosticators; Steroid

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

H, K. (2015). Some biochemical studies of human breast cancer;. (Thesis). Gujarat University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/48468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

H, Karelia,Nilkamal. “Some biochemical studies of human breast cancer;.” 2015. Thesis, Gujarat University. Accessed October 18, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/48468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

H, Karelia,Nilkamal. “Some biochemical studies of human breast cancer;.” 2015. Web. 18 Oct 2019.

Vancouver:

H K. Some biochemical studies of human breast cancer;. [Internet] [Thesis]. Gujarat University; 2015. [cited 2019 Oct 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/48468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

H K. Some biochemical studies of human breast cancer;. [Thesis]. Gujarat University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/48468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Morrison, Meghan Melinda. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 CANCER BIOLOGY The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis Meghan Melinda Morrison Dissertation under the direction of Professor Rebecca Cook The phosphatidyl… (more)

Subjects/Keywords: mTORC2; mTOR; HER2; lapatinib; breast cancer

…currently in clinical trials for breast cancer . .29 2. Upregulation of… …breast tumors …3 2. mTOR exists in two structurally and functionally distinct complexes… …lactation...19 8. Targeting the PI3K-Akt-mTOR pathway in breast cancer …28 9. Loss of… …in human breast cancer .91 22. Loss of Rictor delays HER2-driven tumor formation… …Rictor loss decreases growth and Akt S473 phosphorylation in established HER2-amplified breast… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Morrison, M. M. (2015). The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07072015-142700/ ;

Chicago Manual of Style (16th Edition):

Morrison, Meghan Melinda. “The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-07072015-142700/ ;.

MLA Handbook (7th Edition):

Morrison, Meghan Melinda. “The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.” 2015. Web. 18 Oct 2019.

Vancouver:

Morrison MM. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-07072015-142700/ ;.

Council of Science Editors:

Morrison MM. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-07072015-142700/ ;

22. Russell, Alisha Joy. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 C/EBPbeta is essential for mammary gland growth and development and has been associated with poor prognosis in breast cancer. Overexpression of C/EBPbeta2 in MCF10A cells,… (more)

Subjects/Keywords: C/EBPbeta; interleukin-1beta; bioinformatics; breast cancer

…68 Figure 13. Immunohistochemical analysis of human breast cancer samples reveals nuclear… …103 Figure 27. Exogenous expression of C/EBPbeta2 in luminal breast cancer cell lines… …INTRODUCTION Breast Cancer Overview According to the American Cancer Society, in 2009 in the United… …States alone it is estimated 200,000 people will be diagnosed with breast cancer and tragically… …40,000 people will die from this disease. In fact breast cancer is so prevalent that 1:8 women… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Russell, A. J. (2010). The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08232010-133130/ ;

Chicago Manual of Style (16th Edition):

Russell, Alisha Joy. “The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-08232010-133130/ ;.

MLA Handbook (7th Edition):

Russell, Alisha Joy. “The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.” 2010. Web. 18 Oct 2019.

Vancouver:

Russell AJ. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-08232010-133130/ ;.

Council of Science Editors:

Russell AJ. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-08232010-133130/ ;

23. Abreu, Maria Mercedes. C/EBPbeta3 (LIP) induces cell death in breast cancer cells.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 C/EBPbeta is a member of a family of basic-leucine zipper transcription factors. It has been shown to be a key regulator of growth and differentiation… (more)

Subjects/Keywords: C/EBPbeta; breast cancer; cell death; autophagy

…expression attenuates proliferation of the MDA-MB-468 breast cancer cell line. ...44… …Figure 7. LIP expression attenuates proliferation of the MDA-MB-231 breast cancer cell line… …46 Figure 8. LIP expression attenuates proliferation of MCF-7 breast cancer cell line… …related genes AV-autophagic acidic vesicles BECN1- Beclin 1 BME- 2-mercaptoethanol BRCA1- breast… …cancer susceptibility gene 1 BRCA2- breast cancer susceptibility gene 2 BSA- bovine serum… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abreu, M. M. (2012). C/EBPbeta3 (LIP) induces cell death in breast cancer cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04162012-131238/ ;

Chicago Manual of Style (16th Edition):

Abreu, Maria Mercedes. “C/EBPbeta3 (LIP) induces cell death in breast cancer cells.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-04162012-131238/ ;.

MLA Handbook (7th Edition):

Abreu, Maria Mercedes. “C/EBPbeta3 (LIP) induces cell death in breast cancer cells.” 2012. Web. 18 Oct 2019.

Vancouver:

Abreu MM. C/EBPbeta3 (LIP) induces cell death in breast cancer cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-04162012-131238/ ;.

Council of Science Editors:

Abreu MM. C/EBPbeta3 (LIP) induces cell death in breast cancer cells. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-04162012-131238/ ;


Wayne State University

24. Rothberg, Jennifer M. Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma.

Degree: PhD, Cancer Biology, 2013, Wayne State University

  Among the non-cellular microenvironmental factors that contribute to malignancy of solid tumors is an acidic peritumoral pH. The first objective was to determine if… (more)

Subjects/Keywords: 3D culture; acidic pH; breast cancer; live-cell imaging; proteolysis; STAT1; Cell Biology; Molecular Biology; Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rothberg, J. M. (2013). Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/694

Chicago Manual of Style (16th Edition):

Rothberg, Jennifer M. “Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma.” 2013. Doctoral Dissertation, Wayne State University. Accessed October 18, 2019. https://digitalcommons.wayne.edu/oa_dissertations/694.

MLA Handbook (7th Edition):

Rothberg, Jennifer M. “Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma.” 2013. Web. 18 Oct 2019.

Vancouver:

Rothberg JM. Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2019 Oct 18]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/694.

Council of Science Editors:

Rothberg JM. Acidic Pericellular Ph: Effects On Proteolysis And Gene Expression As Determined In 3d Models Of Breast Carcinoma. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/694


Vanderbilt University

25. Fang, Wei Bin. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 The EphA2 receptor belongs to the recently cloned Eph family of receptor tyrosine kinase (RTK). High levels of EphA2 RTK have been detected in 60-90%… (more)

Subjects/Keywords: Receptor Tyrosine Kinase; Breast Cancer; EphA2; Neovascularization  – Regulation; Protein-tyrosine kinase  – Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fang, W. B. (2008). The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08222008-132323/ ;

Chicago Manual of Style (16th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-08222008-132323/ ;.

MLA Handbook (7th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Web. 18 Oct 2019.

Vancouver:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-08222008-132323/ ;.

Council of Science Editors:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://etd.library.vanderbilt.edu/available/etd-08222008-132323/ ;


Vanderbilt University

26. Neel, Nicole Fowler. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 Chemokines are a family of small chemotactic cytokines that bind seven transmembrane G protein-coupled receptors. Roles for CXC chemokines and the receptors CXCR2 and CXCR4… (more)

Subjects/Keywords: VASP; breast cancer; proteomics; RhoB; intracellular trafficking; chemokine receptor; CXCR4; CXCR2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Neel, N. F. (2008). Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03052008-172942/ ;

Chicago Manual of Style (16th Edition):

Neel, Nicole Fowler. “Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu//available/etd-03052008-172942/ ;.

MLA Handbook (7th Edition):

Neel, Nicole Fowler. “Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.” 2008. Web. 18 Oct 2019.

Vancouver:

Neel NF. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-03052008-172942/ ;.

Council of Science Editors:

Neel NF. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://etd.library.vanderbilt.edu//available/etd-03052008-172942/ ;

27. Williams, Andrew John. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.

Degree: MS, Cancer Biology, 2014, Vanderbilt University

Breast cancer is the leading cancer diagnosis in pre-menopausal women in the U.S. Of these breast cancers, 25% are diagnosed 2-5 years post-partum. Unfortunately, post-partum… (more)

Subjects/Keywords: breast cancer; wound healing; inflammation; post-partum; TGFbeta

breast cancer [6, 8, 12, 31, 44]. Following weaning, TGFβ has been shown to be… …x28;Fig.5D). 6 6 DISCUSSION Premenopausal breast cancers diagnosed during… …premenopausal breast cancers diagnosed in nulliparous women, pregnant women, or women whose… …there is increased apoptosis in postpartum breast cancers[12]. This could suggest a… …Developmental windows of breast cancer risk provide opportunities for targeted… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, A. J. (2014). TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. (Masters Thesis). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12022014-140155/ ;

Chicago Manual of Style (16th Edition):

Williams, Andrew John. “TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.” 2014. Masters Thesis, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-12022014-140155/ ;.

MLA Handbook (7th Edition):

Williams, Andrew John. “TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.” 2014. Web. 18 Oct 2019.

Vancouver:

Williams AJ. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. [Internet] [Masters thesis]. Vanderbilt University; 2014. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-12022014-140155/ ;.

Council of Science Editors:

Williams AJ. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. [Masters Thesis]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-12022014-140155/ ;

28. Kurley, Sarah Jean. The role of p120 catenin in mammary development and breast cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Cadherins and catenins are important regulators of tissue homeostasis and cancer. Here, we report critical roles for p120 catenin (p120) in mammary development and breast(more)

Subjects/Keywords: mammary gland; cadherin; catenin; p120; metastasis; breast cancer

…26 p120 and its effectors in breast cancer… …27 p120 in breast cancer… …28 p120 effectors in breast cancer… …74 IV. P120 ABLATION POTENTIATES A MOUSE MODEL OF BREAST CANCER, BUT IS PARADOXICALLY… …116 Appendix A. KAISO IN THE MMTV POLYOMA MIDDLE T MOUSE MODEL OF BREAST CANCER… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kurley, S. J. (2012). The role of p120 catenin in mammary development and breast cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03232012-162305/ ;

Chicago Manual of Style (16th Edition):

Kurley, Sarah Jean. “The role of p120 catenin in mammary development and breast cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu//available/etd-03232012-162305/ ;.

MLA Handbook (7th Edition):

Kurley, Sarah Jean. “The role of p120 catenin in mammary development and breast cancer.” 2012. Web. 18 Oct 2019.

Vancouver:

Kurley SJ. The role of p120 catenin in mammary development and breast cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-03232012-162305/ ;.

Council of Science Editors:

Kurley SJ. The role of p120 catenin in mammary development and breast cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu//available/etd-03232012-162305/ ;

29. Matise, Lauren Alicia. Role of TGF-β signaling in carcinoma cell migration and tumor progression.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Transforming growth factor-beta (TGF-β) has a dual role during tumor progression initially as a suppressor and then as a promoter. Much is known about the… (more)

Subjects/Keywords: TGF-beta; tumor-stromal interactions; single cell invasion; collective cell invasion; mouse model; breast cancer; tumor microenvironment

…Department of Defense Breast Cancer Research Program Pre-Doctoral Award (W81XWH-11-10066)… …overexpression has been associated with breast, colon, esophageal, gastric, hepatocellular, lung, and… …associated with a lower risk of breast cancer, suggesting a tumor suppressive role of TGF-β1 in… …invasive breast carcinoma, epithelial TGF-β1 and TGF-β2 were overexpressed in 67% and 76% of… …x29; region (Bacon et al., 2001). In breast cancer, TGFBR2 MSI or mutation… 

Page 1 Page 2 Page 3 Page 4 Page 5

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matise, L. A. (2012). Role of TGF-β signaling in carcinoma cell migration and tumor progression. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07192012-115011/ ;

Chicago Manual of Style (16th Edition):

Matise, Lauren Alicia. “Role of TGF-β signaling in carcinoma cell migration and tumor progression.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-07192012-115011/ ;.

MLA Handbook (7th Edition):

Matise, Lauren Alicia. “Role of TGF-β signaling in carcinoma cell migration and tumor progression.” 2012. Web. 18 Oct 2019.

Vancouver:

Matise LA. Role of TGF-β signaling in carcinoma cell migration and tumor progression. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-07192012-115011/ ;.

Council of Science Editors:

Matise LA. Role of TGF-β signaling in carcinoma cell migration and tumor progression. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-07192012-115011/ ;

30. Bierie, Brian Richard. TGF-â in mammary development and tumorigenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 The transforming growth factor beta (TGF-â) pathway significantly regulates mammary development and tumorigenesis. In human cancer, its signaling pathways are often modified or lost during… (more)

Subjects/Keywords: Transforming growth factors-beta  – Physiological effect; inflammation; transgorming growth factor beta; tumor; mammary; prognosis; chemokine; Breast  – Cancer  – Pathophysiology; Metastasis  – Molecular aspects

…human breast cancer ..................................................... 156 Discussion… …159 Figure 28. TβRII(WKO;PY) and TGF-β signature correlation with human breast… …ER+ or hormone only treated breast cancer relapse-free survival… …specific TGF-β signaling and risk for relapse in human breast cancer. 1 The TGF-β signaling… …incidence of breast cancer (Ziv et al., 2001). This correlation suggests that the TGF-β1… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bierie, B. R. (2008). TGF-â in mammary development and tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08272008-101423/ ;

Chicago Manual of Style (16th Edition):

Bierie, Brian Richard. “TGF-â in mammary development and tumorigenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-08272008-101423/ ;.

MLA Handbook (7th Edition):

Bierie, Brian Richard. “TGF-â in mammary development and tumorigenesis.” 2008. Web. 18 Oct 2019.

Vancouver:

Bierie BR. TGF-â in mammary development and tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-08272008-101423/ ;.

Council of Science Editors:

Bierie BR. TGF-â in mammary development and tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://etd.library.vanderbilt.edu/available/etd-08272008-101423/ ;

.