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You searched for subject:(breast cancer resistance protein). Showing records 1 – 30 of 68535 total matches.

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University of Illinois – Chicago

1. Molloy, Mary E. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.

Degree: 2014, University of Illinois – Chicago

Breast cancer is the most common female malignancy, affecting 1 in 8 women. Resistance to the antiestrogen tamoxifen (TAM), whether de novo or acquired, is… (more)

Subjects/Keywords: PKC alpha (Protein kinase C); breast cancer; endocrine resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Molloy, M. E. (2014). Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Thesis, University of Illinois – Chicago. Accessed April 10, 2021. http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Web. 10 Apr 2021.

Vancouver:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

2. McGrath, Eoghan Patrick. Investigating the role of IRE1α RNase activity in breast cancer .

Degree: 2018, National University of Ireland – Galway

 Metastatic, drug resistant disease poses the greatest threat to the survival of breast cancer patients. Thus, elucidating and targeting mechanisms which govern this phenotype is… (more)

Subjects/Keywords: ER stress; IRE1; breast cancer; unfolded protein response; therapy resistance; JUP; Cell Migration; Unfolded Protein Response in Breast Cancer; Natural Sciences; Biochemistry; Breast cancer; Unfolded protein response

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APA (6th Edition):

McGrath, E. P. (2018). Investigating the role of IRE1α RNase activity in breast cancer . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/14726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McGrath, Eoghan Patrick. “Investigating the role of IRE1α RNase activity in breast cancer .” 2018. Thesis, National University of Ireland – Galway. Accessed April 10, 2021. http://hdl.handle.net/10379/14726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McGrath, Eoghan Patrick. “Investigating the role of IRE1α RNase activity in breast cancer .” 2018. Web. 10 Apr 2021.

Vancouver:

McGrath EP. Investigating the role of IRE1α RNase activity in breast cancer . [Internet] [Thesis]. National University of Ireland – Galway; 2018. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10379/14726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McGrath EP. Investigating the role of IRE1α RNase activity in breast cancer . [Thesis]. National University of Ireland – Galway; 2018. Available from: http://hdl.handle.net/10379/14726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

3. Rainey, Jenna. The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta .

Degree: 2011, University of Ottawa

 Multidrug resistance phosphoglycoprotein (MDR1/P-gp) and breast cancer resistance protein (BCRP) were first isolated in chemoresistant cancer cells and have since been found in a variety… (more)

Subjects/Keywords: Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp); Breast Cancer Resistance Protein (BCRP); Human Placenta

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APA (6th Edition):

Rainey, J. (2011). The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rainey, Jenna. “The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta .” 2011. Thesis, University of Ottawa. Accessed April 10, 2021. http://hdl.handle.net/10393/19961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rainey, Jenna. “The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta .” 2011. Web. 10 Apr 2021.

Vancouver:

Rainey J. The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10393/19961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rainey J. The Regulation of Multidrug Resistance Phosphoglycoprotein (MDR1/P-gp) and Breast Cancer Resistance Protein (BCRP) in the Human Placenta . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. LOWRY, MICHELLE. Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles.

Degree: School of Pharmacy & Pharma. Sciences. Discipline of Pharmacy, 2019, Trinity College Dublin

Breast cancer is the most common female cancer diagnosed worldwide. In 2018 alone, there were over 2 million new breast cancer cases diagnosed. In Ireland,… (more)

Subjects/Keywords: Breast cancer; Cancer drug resistance

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APA (6th Edition):

LOWRY, M. (2019). Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/86081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LOWRY, MICHELLE. “Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles.” 2019. Thesis, Trinity College Dublin. Accessed April 10, 2021. http://hdl.handle.net/2262/86081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LOWRY, MICHELLE. “Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles.” 2019. Web. 10 Apr 2021.

Vancouver:

LOWRY M. Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2262/86081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LOWRY M. Investigating mechanisms of anti-cancer drug resistance in breast cancer cells and extracellular vesicles. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/86081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

5. Eclov, Rachel. Mechanisms Regulating the Expression and Function of MXR.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2013, University of California – San Francisco

 ABCG2 encodes for a multidrug efflux transporter called the mitoxantrone resistance protein (MXR, BCRP) that mediates the efflux of substrates out of the cell and… (more)

Subjects/Keywords: Pharmaceutical sciences; Genetics; ABCG2; ATP-binding cassette transporters; breast cancer resistance protein; epigenetics; mitoxantrone resistance protein; Pharmacogenomics

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APA (6th Edition):

Eclov, R. (2013). Mechanisms Regulating the Expression and Function of MXR. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/8dr5j9nq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eclov, Rachel. “Mechanisms Regulating the Expression and Function of MXR.” 2013. Thesis, University of California – San Francisco. Accessed April 10, 2021. http://www.escholarship.org/uc/item/8dr5j9nq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eclov, Rachel. “Mechanisms Regulating the Expression and Function of MXR.” 2013. Web. 10 Apr 2021.

Vancouver:

Eclov R. Mechanisms Regulating the Expression and Function of MXR. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Apr 10]. Available from: http://www.escholarship.org/uc/item/8dr5j9nq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eclov R. Mechanisms Regulating the Expression and Function of MXR. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/8dr5j9nq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Jahani Aval, Candice Marral. The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells.

Degree: 2018, University of Toronto

Various mechanisms mediate transport of compounds into milk throughout lactation. Breast cancer resistance protein (BCRP) is one of the apically expressed transport proteins in mammary… (more)

Subjects/Keywords: Breast cancer resistance protein; Drug transfer; Lactation; Riboflavin; Riboflavin transporter; Topotecan; 0419

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APA (6th Edition):

Jahani Aval, C. M. (2018). The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89572

Chicago Manual of Style (16th Edition):

Jahani Aval, Candice Marral. “The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells.” 2018. Masters Thesis, University of Toronto. Accessed April 10, 2021. http://hdl.handle.net/1807/89572.

MLA Handbook (7th Edition):

Jahani Aval, Candice Marral. “The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells.” 2018. Web. 10 Apr 2021.

Vancouver:

Jahani Aval CM. The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1807/89572.

Council of Science Editors:

Jahani Aval CM. The Use of High-Dose Riboflavin to Limit Topotecan Efflux in Milk: A Proof-of-Principle Study in Mice and Cells. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89572


Tampere University

7. Haukipää, Mia. Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa .

Degree: 2014, Tampere University

 Sarveiskalvon epiteelin on opittu ilmentävän efflux-proteiineja MRP1-6 (multiresistance protein 1-6), P-gp (P-glykoproteiini) ja BCRP (breast cancer resistance protein), jotka pumppaavat substraattejaan ulos solusta. Tämän tiedon… (more)

Subjects/Keywords: sarveiskalvon epiteeli; hiPSC; HCE; efflux-kuljettajaproteiini; multidrug resistance protein; P-glykoproteiini; breast cancer related proteiini

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APA (6th Edition):

Haukipää, M. (2014). Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa . (Masters Thesis). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/96116

Chicago Manual of Style (16th Edition):

Haukipää, Mia. “Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa .” 2014. Masters Thesis, Tampere University. Accessed April 10, 2021. https://trepo.tuni.fi/handle/10024/96116.

MLA Handbook (7th Edition):

Haukipää, Mia. “Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa .” 2014. Web. 10 Apr 2021.

Vancouver:

Haukipää M. Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa . [Internet] [Masters thesis]. Tampere University; 2014. [cited 2021 Apr 10]. Available from: https://trepo.tuni.fi/handle/10024/96116.

Council of Science Editors:

Haukipää M. Efflux-geenien ilmentyminen hiPS-soluista erilaistetuissa sarveiskalvon epiteelisoluissa . [Masters Thesis]. Tampere University; 2014. Available from: https://trepo.tuni.fi/handle/10024/96116


Univerzitet u Beogradu

8. Abu Rabi, Zaki, 1980-. Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Molekularna biologija kancera dojke / Molekularna biologija kancera dojke

Karcinom dojke, kao najčešći oblik malignih tumora koji se javlja kod žena, spada u grupu heterogenih… (more)

Subjects/Keywords: breast cancer; tamoxifen; resistance

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APA (6th Edition):

Abu Rabi, Zaki, 1. (2014). Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:6759/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abu Rabi, Zaki, 1980-. “Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima.” 2014. Thesis, Univerzitet u Beogradu. Accessed April 10, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:6759/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abu Rabi, Zaki, 1980-. “Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima.” 2014. Web. 10 Apr 2021.

Vancouver:

Abu Rabi, Zaki 1. Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2021 Apr 10]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6759/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abu Rabi, Zaki 1. Značaj biološke heterogenosti karcinoma dojke za odgovor na terapiju antiestrogenima. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6759/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

9. Dedina, Liana. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.

Degree: 2012, University of Toronto

Mother's milk provides multiple benefits to the offspring. However, xenobiotics transferred into breast milk may pose a risk to the nursing infant. The breast cancer(more)

Subjects/Keywords: Pharmacology; Toxicology; Riboflavin; Cimetidine; Breast cancer resistance protein (BCRP); Riboflavin transporters; Breast milk; Mammary Gland; Lactation; 0419; 0383

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APA (6th Edition):

Dedina, L. (2012). Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33644

Chicago Manual of Style (16th Edition):

Dedina, Liana. “Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.” 2012. Masters Thesis, University of Toronto. Accessed April 10, 2021. http://hdl.handle.net/1807/33644.

MLA Handbook (7th Edition):

Dedina, Liana. “Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.” 2012. Web. 10 Apr 2021.

Vancouver:

Dedina L. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1807/33644.

Council of Science Editors:

Dedina L. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33644


University of St. Andrews

10. Fedele, Vita. Polycomb proteins and breast cancer .

Degree: 2012, University of St. Andrews

 In the Western world, breast cancer is the most frequent malignancy in women and still the leading cause of cancer related deaths, therefore, a better… (more)

Subjects/Keywords: Breast cancer; Polycomb protein

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APA (6th Edition):

Fedele, V. (2012). Polycomb proteins and breast cancer . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/3584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fedele, Vita. “Polycomb proteins and breast cancer .” 2012. Thesis, University of St. Andrews. Accessed April 10, 2021. http://hdl.handle.net/10023/3584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fedele, Vita. “Polycomb proteins and breast cancer .” 2012. Web. 10 Apr 2021.

Vancouver:

Fedele V. Polycomb proteins and breast cancer . [Internet] [Thesis]. University of St. Andrews; 2012. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10023/3584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fedele V. Polycomb proteins and breast cancer . [Thesis]. University of St. Andrews; 2012. Available from: http://hdl.handle.net/10023/3584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

11. Rane, Chetan K., 1989-. PAK4 as a drug target in breast cancer therapy.

Degree: PhD, Pharmacology, Cellular and Molecular, 2017, Rutgers University

PAK4, belonging to Group II PAKs, is an important signaling protein with key roles in regulating cell growth, cell survival and cell morphology. PAK4 protein(more)

Subjects/Keywords: Breast – Cancer – Treatment; Protein kinases

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APA (6th Edition):

Rane, Chetan K., 1. (2017). PAK4 as a drug target in breast cancer therapy. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/54913/

Chicago Manual of Style (16th Edition):

Rane, Chetan K., 1989-. “PAK4 as a drug target in breast cancer therapy.” 2017. Doctoral Dissertation, Rutgers University. Accessed April 10, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/54913/.

MLA Handbook (7th Edition):

Rane, Chetan K., 1989-. “PAK4 as a drug target in breast cancer therapy.” 2017. Web. 10 Apr 2021.

Vancouver:

Rane, Chetan K. 1. PAK4 as a drug target in breast cancer therapy. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 Apr 10]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/54913/.

Council of Science Editors:

Rane, Chetan K. 1. PAK4 as a drug target in breast cancer therapy. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/54913/


University of North Carolina – Greensboro

12. Eanes, Lauren A. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.

Degree: 2014, University of North Carolina – Greensboro

 Estrogen receptor (ER) antagonists such as tamoxifen have been used successfully to treat ER+ breast cancers for more than 30 years. Tamoxifen targets the ER… (more)

Subjects/Keywords: Breast – Cancer – Treatment; Tamoxifen; Drug resistance in cancer cells; Estrogen – Receptors – Pathophysiology; Mitogen-activated protein kinases

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APA (6th Edition):

Eanes, L. A. (2014). Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160

Chicago Manual of Style (16th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Masters Thesis, University of North Carolina – Greensboro. Accessed April 10, 2021. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

MLA Handbook (7th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Web. 10 Apr 2021.

Vancouver:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2014. [cited 2021 Apr 10]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

Council of Science Editors:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Masters Thesis]. University of North Carolina – Greensboro; 2014. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160


University of Minnesota

13. Vaidhyanathan, Shruthi. Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases.

Degree: PhD, Pharmaceutics, 2015, University of Minnesota

 The FDA approval of molecularly-targeted drugs that specifically targeted aberrant signaling proteins has brought about new hope for the treatment of advanced melanoma. Historically, metastatic… (more)

Subjects/Keywords: Breast cancer resistance protein; P-glycoprotein; Efflux transporters; brain distribution; Melanoma Brain Metastases; molecularly-targeted drugs; skin cancer; vemurafenib

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APA (6th Edition):

Vaidhyanathan, S. (2015). Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191424

Chicago Manual of Style (16th Edition):

Vaidhyanathan, Shruthi. “Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases.” 2015. Doctoral Dissertation, University of Minnesota. Accessed April 10, 2021. http://hdl.handle.net/11299/191424.

MLA Handbook (7th Edition):

Vaidhyanathan, Shruthi. “Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases.” 2015. Web. 10 Apr 2021.

Vancouver:

Vaidhyanathan S. Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases. [Internet] [Doctoral dissertation]. University of Minnesota; 2015. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11299/191424.

Council of Science Editors:

Vaidhyanathan S. Improving the Delivery of Molecularly-Targeted Agents to Effectively Treat Melanoma Brain Metastases. [Doctoral Dissertation]. University of Minnesota; 2015. Available from: http://hdl.handle.net/11299/191424

14. Eanes, Lauren A. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.

Degree: 2014, NC Docks

 Estrogen receptor (ER) antagonists such as tamoxifen have been used successfully to treat ER+ breast cancers for more than 30 years. Tamoxifen targets the ER… (more)

Subjects/Keywords: Breast $x Cancer $x Treatment; Tamoxifen; Drug resistance in cancer cells; Estrogen $x Receptors $x Pathophysiology; Mitogen-activated protein kinases

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APA (6th Edition):

Eanes, L. A. (2014). Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Thesis, NC Docks. Accessed April 10, 2021. http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Web. 10 Apr 2021.

Vancouver:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Internet] [Thesis]. NC Docks; 2014. [cited 2021 Apr 10]. Available from: http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Thesis]. NC Docks; 2014. Available from: http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

15. Djoukhadar, Ibrahim Khalil. Measuring and Modifying Temozolomide Delivery in Brain Tumours.

Degree: 2017, University of Manchester

Glioblastoma (GBM) is the most common aggressive primary brain tumour in adults. Despite various recent treatment advances, prognosis and survival rates remain dismal. A potential… (more)

Subjects/Keywords: Brain Tumours; Neuro-oncology; Imaging; blood brain barrier; Temozolomide; efflux transporters; P-glycoprotein; Breast Cancer Resistance protein

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APA (6th Edition):

Djoukhadar, I. K. (2017). Measuring and Modifying Temozolomide Delivery in Brain Tumours. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:309634

Chicago Manual of Style (16th Edition):

Djoukhadar, Ibrahim Khalil. “Measuring and Modifying Temozolomide Delivery in Brain Tumours.” 2017. Doctoral Dissertation, University of Manchester. Accessed April 10, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:309634.

MLA Handbook (7th Edition):

Djoukhadar, Ibrahim Khalil. “Measuring and Modifying Temozolomide Delivery in Brain Tumours.” 2017. Web. 10 Apr 2021.

Vancouver:

Djoukhadar IK. Measuring and Modifying Temozolomide Delivery in Brain Tumours. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2021 Apr 10]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:309634.

Council of Science Editors:

Djoukhadar IK. Measuring and Modifying Temozolomide Delivery in Brain Tumours. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:309634


University of Manchester

16. Djoukhadar, Ibrahim. Measuring and modifying Temozolomide delivery in brain tumours.

Degree: PhD, 2017, University of Manchester

 Glioblastoma (GBM) is the most common aggressive primary brain tumour in adults. Despite various recent treatment advances, prognosis and survival rates remain dismal. A potential… (more)

Subjects/Keywords: Breast Cancer Resistance protein; P-glycoprotein; efflux transporters; Temozolomide; Imaging; Neuro-oncology; Brain Tumours; blood brain barrier

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APA (6th Edition):

Djoukhadar, I. (2017). Measuring and modifying Temozolomide delivery in brain tumours. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/measuring-and-modifying-temozolomide-delivery-in-brain-tumours(5ffb33be-c2e9-4c2b-bd40-b3e439b2d8f8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822947

Chicago Manual of Style (16th Edition):

Djoukhadar, Ibrahim. “Measuring and modifying Temozolomide delivery in brain tumours.” 2017. Doctoral Dissertation, University of Manchester. Accessed April 10, 2021. https://www.research.manchester.ac.uk/portal/en/theses/measuring-and-modifying-temozolomide-delivery-in-brain-tumours(5ffb33be-c2e9-4c2b-bd40-b3e439b2d8f8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822947.

MLA Handbook (7th Edition):

Djoukhadar, Ibrahim. “Measuring and modifying Temozolomide delivery in brain tumours.” 2017. Web. 10 Apr 2021.

Vancouver:

Djoukhadar I. Measuring and modifying Temozolomide delivery in brain tumours. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2021 Apr 10]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/measuring-and-modifying-temozolomide-delivery-in-brain-tumours(5ffb33be-c2e9-4c2b-bd40-b3e439b2d8f8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822947.

Council of Science Editors:

Djoukhadar I. Measuring and modifying Temozolomide delivery in brain tumours. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/measuring-and-modifying-temozolomide-delivery-in-brain-tumours(5ffb33be-c2e9-4c2b-bd40-b3e439b2d8f8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822947


Virginia Tech

17. Chen, Chun. Systems Biology Study of Breast Cancer Endocrine Response and Resistance.

Degree: PhD, Genetics, Bioinformatics, and Computational Biology, 2013, Virginia Tech

 As a robust system, cells can wisely choose and switch between different signaling programs according to their differentiation stages and external environments. Cancer cells can… (more)

Subjects/Keywords: Mathematical modeling; breast cancer; endocrine resistance; signaling switch; breast cancer landscape

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APA (6th Edition):

Chen, C. (2013). Systems Biology Study of Breast Cancer Endocrine Response and Resistance. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/51965

Chicago Manual of Style (16th Edition):

Chen, Chun. “Systems Biology Study of Breast Cancer Endocrine Response and Resistance.” 2013. Doctoral Dissertation, Virginia Tech. Accessed April 10, 2021. http://hdl.handle.net/10919/51965.

MLA Handbook (7th Edition):

Chen, Chun. “Systems Biology Study of Breast Cancer Endocrine Response and Resistance.” 2013. Web. 10 Apr 2021.

Vancouver:

Chen C. Systems Biology Study of Breast Cancer Endocrine Response and Resistance. [Internet] [Doctoral dissertation]. Virginia Tech; 2013. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10919/51965.

Council of Science Editors:

Chen C. Systems Biology Study of Breast Cancer Endocrine Response and Resistance. [Doctoral Dissertation]. Virginia Tech; 2013. Available from: http://hdl.handle.net/10919/51965


University of Miami

18. Jegg, Anna-Maria. Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells.

Degree: PhD, Cancer Biology (Medicine), 2013, University of Miami

  Amplification and resulting overexpression of the epidermal growth factor receptor ERBB2 (HER2) is found in ~20% of human breast cancers. Therapies targeting the ERBB2… (more)

Subjects/Keywords: breast cancer; ERBB2 amplified; lapatinib resistance; mTOR

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APA (6th Edition):

Jegg, A. (2013). Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/923

Chicago Manual of Style (16th Edition):

Jegg, Anna-Maria. “Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells.” 2013. Doctoral Dissertation, University of Miami. Accessed April 10, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/923.

MLA Handbook (7th Edition):

Jegg, Anna-Maria. “Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells.” 2013. Web. 10 Apr 2021.

Vancouver:

Jegg A. Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells. [Internet] [Doctoral dissertation]. University of Miami; 2013. [cited 2021 Apr 10]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/923.

Council of Science Editors:

Jegg A. Identification of Molecular Mechanisms of Lapatinib Resistance in ERBB2 Amplified Breast Cancer Cells. [Doctoral Dissertation]. University of Miami; 2013. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/923


Harvard University

19. Luo, Flora. Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer.

Degree: PhD, 2017, Harvard University

Highly specific cyclin-dependent kinase 4 and 6 (CDK4 and CDK6) inhibitors comprise a novel and exciting class of targeted therapeutics in oncology. CDK4/6 inhibitors, such… (more)

Subjects/Keywords: CDK4/6 inhibitors; resistance; breast cancer

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APA (6th Edition):

Luo, F. (2017). Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:41142050

Chicago Manual of Style (16th Edition):

Luo, Flora. “Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer.” 2017. Doctoral Dissertation, Harvard University. Accessed April 10, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:41142050.

MLA Handbook (7th Edition):

Luo, Flora. “Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer.” 2017. Web. 10 Apr 2021.

Vancouver:

Luo F. Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2021 Apr 10]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41142050.

Council of Science Editors:

Luo F. Dissecting Response and Resistance to CDK4/6 inhibition in ER+ Breast Cancer. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41142050


University of Windsor

20. Ferraiuolo, Rosa-Maria. Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance.

Degree: PhD, Biological Sciences, 2015, University of Windsor

  Classification of breast cancer relies on the presence or absence of estrogen receptor alpha (ERα) and progesterone receptor (PR) as well as the overexpression… (more)

Subjects/Keywords: breast cancer; cell cycle regulation; resistance

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APA (6th Edition):

Ferraiuolo, R. (2015). Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance. (Doctoral Dissertation). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/5431

Chicago Manual of Style (16th Edition):

Ferraiuolo, Rosa-Maria. “Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance.” 2015. Doctoral Dissertation, University of Windsor. Accessed April 10, 2021. https://scholar.uwindsor.ca/etd/5431.

MLA Handbook (7th Edition):

Ferraiuolo, Rosa-Maria. “Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance.” 2015. Web. 10 Apr 2021.

Vancouver:

Ferraiuolo R. Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance. [Internet] [Doctoral dissertation]. University of Windsor; 2015. [cited 2021 Apr 10]. Available from: https://scholar.uwindsor.ca/etd/5431.

Council of Science Editors:

Ferraiuolo R. Novel Cell Cycle Regulation in Breast Cancer Treatment Resistance. [Doctoral Dissertation]. University of Windsor; 2015. Available from: https://scholar.uwindsor.ca/etd/5431


University of Louisville

21. Manavalan, Tissa Thomas. Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer.

Degree: PhD, 2012, University of Louisville

 MicroRNAs (miRNAs) regulate gene expression at the post-transcriptional level by repressing translation or stimulating mRNA degradation. In this study, I tested the hypothesis that miRNAs… (more)

Subjects/Keywords: Breast cancer; endocrine; microRNA; resistance; Tamoxifen

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APA (6th Edition):

Manavalan, T. T. (2012). Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/897 ; https://ir.library.louisville.edu/etd/897

Chicago Manual of Style (16th Edition):

Manavalan, Tissa Thomas. “Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer.” 2012. Doctoral Dissertation, University of Louisville. Accessed April 10, 2021. 10.18297/etd/897 ; https://ir.library.louisville.edu/etd/897.

MLA Handbook (7th Edition):

Manavalan, Tissa Thomas. “Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer.” 2012. Web. 10 Apr 2021.

Vancouver:

Manavalan TT. Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer. [Internet] [Doctoral dissertation]. University of Louisville; 2012. [cited 2021 Apr 10]. Available from: 10.18297/etd/897 ; https://ir.library.louisville.edu/etd/897.

Council of Science Editors:

Manavalan TT. Dysregulation of microRNA expression in acquired endocrine-resistant breast cancer. [Doctoral Dissertation]. University of Louisville; 2012. Available from: 10.18297/etd/897 ; https://ir.library.louisville.edu/etd/897


IUPUI

22. Tonsing-Carter, Eva Y. Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin.

Degree: 2014, IUPUI

 Triple-negative breast cancers (TNBCs) are highly refractive to current treatment strategies, and new multi-targeted treatments need to be elucidated. Combination therapy that includes targeting the… (more)

Subjects/Keywords: Mdm2; Breast cancer; Nutlin-3a; Pharmacology; Breast  – Cancer  – Treatment; Breast  – Cancer  – Research; Breast  – Cancer  – Pathophysiology; Protein kinases  – Inhibitors

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APA (6th Edition):

Tonsing-Carter, E. Y. (2014). Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tonsing-Carter, Eva Y. “Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin.” 2014. Thesis, IUPUI. Accessed April 10, 2021. http://hdl.handle.net/1805/6306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tonsing-Carter, Eva Y. “Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin.” 2014. Web. 10 Apr 2021.

Vancouver:

Tonsing-Carter EY. Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1805/6306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tonsing-Carter EY. Modulation of the Mdm2 signaling axis sensitizes triple-negative breast cancer cells to carboplatin. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

23. Schwarz, Theresa. From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity.

Degree: 2017, University of Vienna

 ABC-Transporter sind Membranproteine, die durch Hydrolyse von ATP den Transport einer Vielzahl von Substanzen über Membranen bewerkstelligen. Unter den menschlichen ABC-Transportern zählen P-Glykoprotein (Pgp, ABCB1)… (more)

Subjects/Keywords: 30.00 Naturwissenschaften allgemein: Allgemeines; 35.00 Chemie: Allgemeines; 30.03 Methoden und Techniken in den Naturwissenschaften; Breast Cancer Resistance Protein / Inhibitor / P-glykoprotein / Propafenon / Fumitremorgin C; breast cancer resistance protein / inhibitor / p-glycoprotein / polypharmacology / propafenone / fumitremorgin C

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APA (6th Edition):

Schwarz, T. (2017). From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/48806/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schwarz, Theresa. “From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity.” 2017. Thesis, University of Vienna. Accessed April 10, 2021. http://othes.univie.ac.at/48806/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schwarz, Theresa. “From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity.” 2017. Web. 10 Apr 2021.

Vancouver:

Schwarz T. From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity. [Internet] [Thesis]. University of Vienna; 2017. [cited 2021 Apr 10]. Available from: http://othes.univie.ac.at/48806/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schwarz T. From propafenone to fumitremorgin C – Probing Pgp/BCRP Inhibitor Selectivity. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/48806/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Chaves, Catarina Alexandra da Silva. Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P.

Degree: Docteur es, Toxicologie, 2015, Sorbonne Paris Cité; Universidade do Porto

 La barrière hémato-encéphalique (BHE) représente la principale interface d'échange moléculaire entre la circulation sanguine et le système nerveux central (SNC), où elle joue un rôle… (more)

Subjects/Keywords: Barrière hémato-encéphalique; P-glycoprotéine; Breast cancer resistance protein; Morphine; Régulation; Activation; Blood-brain barrier; P-glycoprotein; Breast cancer resistance protein; Morphine; Regulation; Activation; Barreira-hematoencefálica; Glicoproteína P; Proteína de Resistência ao Cancro da Mama; Morfina; Regulação; Activação; 572.68

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APA (6th Edition):

Chaves, C. A. d. S. (2015). Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P. (Doctoral Dissertation). Sorbonne Paris Cité; Universidade do Porto. Retrieved from http://www.theses.fr/2015USPCB162

Chicago Manual of Style (16th Edition):

Chaves, Catarina Alexandra da Silva. “Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P.” 2015. Doctoral Dissertation, Sorbonne Paris Cité; Universidade do Porto. Accessed April 10, 2021. http://www.theses.fr/2015USPCB162.

MLA Handbook (7th Edition):

Chaves, Catarina Alexandra da Silva. “Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P.” 2015. Web. 10 Apr 2021.

Vancouver:

Chaves CAdS. Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; Universidade do Porto; 2015. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2015USPCB162.

Council of Science Editors:

Chaves CAdS. Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation : Mecanismos de regulação da glicoproteína P e da proteína de resistência ao cancro da mama ao nível da barreira-hematoencefálica : focus no papel da morfina, e na activação da glicoproteína P. [Doctoral Dissertation]. Sorbonne Paris Cité; Universidade do Porto; 2015. Available from: http://www.theses.fr/2015USPCB162


University of KwaZulu-Natal

25. Ndagi, Umar. Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools.

Degree: 2017, University of KwaZulu-Natal

 The alarming rate of varying types of cancer diseases in human remains a global burden requiring drastic treatment in which, a prominent method of combating… (more)

Subjects/Keywords: Breast cancer.; Prostate cancer.; Cancer.; Drug design.; Ligands.; Drug resistance.; Bioinformatics.

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APA (6th Edition):

Ndagi, U. (2017). Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/18521

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ndagi, Umar. “Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools.” 2017. Thesis, University of KwaZulu-Natal. Accessed April 10, 2021. https://researchspace.ukzn.ac.za/handle/10413/18521.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ndagi, Umar. “Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools.” 2017. Web. 10 Apr 2021.

Vancouver:

Ndagi U. Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools. [Internet] [Thesis]. University of KwaZulu-Natal; 2017. [cited 2021 Apr 10]. Available from: https://researchspace.ukzn.ac.za/handle/10413/18521.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ndagi U. Insight into cancer targets and ligand binding landscape using bioinformatics and integrated molecular modeling tools. [Thesis]. University of KwaZulu-Natal; 2017. Available from: https://researchspace.ukzn.ac.za/handle/10413/18521

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

26. Lagas, J.S. Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice.

Degree: 2009, Universiteit Utrecht

 ATP-binding cassette (ABC) multidrug transporters are drug efflux pumps located in the plasma membrane that utilize the energy of ATP hydrolysis to extrude a wide… (more)

Subjects/Keywords: Farmacie; ATP-binding cassette (ABC); ABC multidrug transporter; ABC transporter knockout mice; Combination transporter knockout mice; P-glycoprotein; Multidrug Resistance Protein 2; Breast Cancer Resistance Protein; Multidrug Resistance; Pharmacokinetics; Blood-brain barrier

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APA (6th Edition):

Lagas, J. S. (2009). Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/34065

Chicago Manual of Style (16th Edition):

Lagas, J S. “Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice.” 2009. Doctoral Dissertation, Universiteit Utrecht. Accessed April 10, 2021. http://dspace.library.uu.nl:8080/handle/1874/34065.

MLA Handbook (7th Edition):

Lagas, J S. “Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice.” 2009. Web. 10 Apr 2021.

Vancouver:

Lagas JS. Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2009. [cited 2021 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/34065.

Council of Science Editors:

Lagas JS. Pharmacological functions of multidrug transporters: studies employing combination transporter knockout mice. [Doctoral Dissertation]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/34065

27. Hamy, Anne-Sophie. Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

La chimiothérapie néoadjuvante (CNA) est utilisée dans les cancers du sein agressifs ou localement avancés (CS). Au delà des bénéfices cliniques, elle représente une opportunité… (more)

Subjects/Keywords: Cancer du sein; Chimiothérapie néoadjuvante; Résistance; Breast cancer; Neoadjuvant treatment; Resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hamy, A. (2019). Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS129

Chicago Manual of Style (16th Edition):

Hamy, Anne-Sophie. “Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 10, 2021. http://www.theses.fr/2019SACLS129.

MLA Handbook (7th Edition):

Hamy, Anne-Sophie. “Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein.” 2019. Web. 10 Apr 2021.

Vancouver:

Hamy A. Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2019SACLS129.

Council of Science Editors:

Hamy A. Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer : Identification des facteurs prédictifs de sensibilité ou résistance à la chimiothérapie néoadjuvante dans le cancer du sein. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS129


Universiteit Utrecht

28. Rijnders, S. Protein tyrosine phosphatases and cancer.

Degree: 2009, Universiteit Utrecht

 This thesis describes a complex signaling system that is based on reversible phosphorylation at tyrosine residues mediated by protein tyrosine kinases (PTK) and subsequent dephosphorylation… (more)

Subjects/Keywords: Geneeskunde; protein tyrosien phosphatase; protein tyrosine kinase; signaling; breast cancer; PTP1B

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rijnders, S. (2009). Protein tyrosine phosphatases and cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/32026

Chicago Manual of Style (16th Edition):

Rijnders, S. “Protein tyrosine phosphatases and cancer.” 2009. Masters Thesis, Universiteit Utrecht. Accessed April 10, 2021. http://dspace.library.uu.nl:8080/handle/1874/32026.

MLA Handbook (7th Edition):

Rijnders, S. “Protein tyrosine phosphatases and cancer.” 2009. Web. 10 Apr 2021.

Vancouver:

Rijnders S. Protein tyrosine phosphatases and cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2021 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/32026.

Council of Science Editors:

Rijnders S. Protein tyrosine phosphatases and cancer. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/32026


Univerzitet u Beogradu

29. Kalinić, Marko D., 1988-. Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2.

Degree: Farmaceutski fakultet, 2016, Univerzitet u Beogradu

Farmacija - Farmaceutska–medicinska hemija i strukturna analiza / Pharmacy - Pharmaceutical–medicinal chemistry and structural analysis

Enhancer of Zeste Homolog 2 (EZH2) je epigenetski enzim koji… (more)

Subjects/Keywords: Enhancer of Zeste Homolog 2; epigenetics; anticancer drugs; molecular dynamics; de novo design; P-glycoprotein; breast cancer resistance protein; BCRP; computational classification models

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APA (6th Edition):

Kalinić, Marko D., 1. (2016). Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:10309/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalinić, Marko D., 1988-. “Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2.” 2016. Thesis, Univerzitet u Beogradu. Accessed April 10, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:10309/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalinić, Marko D., 1988-. “Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2.” 2016. Web. 10 Apr 2021.

Vancouver:

Kalinić, Marko D. 1. Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2021 Apr 10]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:10309/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalinić, Marko D. 1. Simulacije milekularne dinamike i računarsko dizajniranje inhibitora protein lizin metiltransferaze EZH2. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:10309/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

30. Dorfman, Elizabeth Howard. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.

Degree: PhD, 2015, University of Washington

 This dissertation is principally comprised of three distinct but related projects. The first is an assessment of the quantity and nature of obstetric pharmacology clinical… (more)

Subjects/Keywords: ABCG2; breast cancer resistance protein; fetal drug exposure; glyburide; obstetric pharmacology; placental drug transport; Genetics; Pharmacology; Obstetrics and gynecology; public health genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dorfman, E. H. (2015). Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34161

Chicago Manual of Style (16th Edition):

Dorfman, Elizabeth Howard. “Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.” 2015. Doctoral Dissertation, University of Washington. Accessed April 10, 2021. http://hdl.handle.net/1773/34161.

MLA Handbook (7th Edition):

Dorfman, Elizabeth Howard. “Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.” 2015. Web. 10 Apr 2021.

Vancouver:

Dorfman EH. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1773/34161.

Council of Science Editors:

Dorfman EH. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34161

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