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You searched for subject:(breast cancer metastasis). Showing records 1 – 30 of 316 total matches.

[1] [2] [3] [4] [5] … [11]

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Vanderbilt University

1. Bates, Andreia LaShonne. Roles of Fibroblast MMP2 in Breast to Lung Metastasis.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

CANCER BIOLOGY Roles of Fibroblast MMP2 in Breast to Lung Metastasis Andreia L. Bates Dissertation under the direction of Barbara Fingleton, PhD Breast cancer five-year… (more)

Subjects/Keywords: MMP2; fibroblasts; metastasis; breast cancer

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APA (6th Edition):

Bates, A. L. (2015). Roles of Fibroblast MMP2 in Breast to Lung Metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10839

Chicago Manual of Style (16th Edition):

Bates, Andreia LaShonne. “Roles of Fibroblast MMP2 in Breast to Lung Metastasis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 06, 2021. http://hdl.handle.net/1803/10839.

MLA Handbook (7th Edition):

Bates, Andreia LaShonne. “Roles of Fibroblast MMP2 in Breast to Lung Metastasis.” 2015. Web. 06 Mar 2021.

Vancouver:

Bates AL. Roles of Fibroblast MMP2 in Breast to Lung Metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1803/10839.

Council of Science Editors:

Bates AL. Roles of Fibroblast MMP2 in Breast to Lung Metastasis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10839


Texas A&M University

2. Xu, Yixiang. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.

Degree: PhD, Medical Sciences, 2016, Texas A&M University

 Twist1 is a basic helix-loop-helix transcription factor family that serves as one of the master regulators promoting epithelial-mesenchymal transition (EMT). Twist1 has been shown in… (more)

Subjects/Keywords: Twist1; Breast cancer metastasis; Foxa1

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APA (6th Edition):

Xu, Y. (2016). Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158963

Chicago Manual of Style (16th Edition):

Xu, Yixiang. “Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.” 2016. Doctoral Dissertation, Texas A&M University. Accessed March 06, 2021. http://hdl.handle.net/1969.1/158963.

MLA Handbook (7th Edition):

Xu, Yixiang. “Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.” 2016. Web. 06 Mar 2021.

Vancouver:

Xu Y. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1969.1/158963.

Council of Science Editors:

Xu Y. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158963


Queens University

3. Carefoot, Esther. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 Met has been found to be over-expressed in human breast cancer, correlating with disease progression and poor prognosis. Src and Stat3 have also been found… (more)

Subjects/Keywords: Breast cancer ; xenografts ; Therapeutics ; Metastasis

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APA (6th Edition):

Carefoot, E. (2014). The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carefoot, Esther. “The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .” 2014. Thesis, Queens University. Accessed March 06, 2021. http://hdl.handle.net/1974/8582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carefoot, Esther. “The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .” 2014. Web. 06 Mar 2021.

Vancouver:

Carefoot E. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . [Internet] [Thesis]. Queens University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1974/8582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carefoot E. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Nairobi

4. Gatete, Grace N. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .

Degree: 2014, University of Nairobi

 Introduction The presence of bone metastasis in patients with breast cancer impacts on the management options. The diagnosis of metastatic bone disease is important in… (more)

Subjects/Keywords: Breast Cancer; Bone metastasis

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APA (6th Edition):

Gatete, G. N. (2014). Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . (Thesis). University of Nairobi. Retrieved from http://hdl.handle.net/11295/76164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gatete, Grace N. “Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .” 2014. Thesis, University of Nairobi. Accessed March 06, 2021. http://hdl.handle.net/11295/76164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gatete, Grace N. “Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .” 2014. Web. 06 Mar 2021.

Vancouver:

Gatete GN. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . [Internet] [Thesis]. University of Nairobi; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11295/76164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gatete GN. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . [Thesis]. University of Nairobi; 2014. Available from: http://hdl.handle.net/11295/76164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

5. Henckels, Eric Patrick. Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis.

Degree: 2013, Columbia University

 Matrix Metallopeptidase 1 (MMP-1) expression has repeatedly been correlated to tumorigenesis and metastasis. Yet, MMP-1 regulation in a metastatic context remains largely unknown. Here we… (more)

Subjects/Keywords: Oncology; Metastasis; Carcinogenesis; Breast – Cancer

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APA (6th Edition):

Henckels, E. P. (2013). Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8TT4Z9W

Chicago Manual of Style (16th Edition):

Henckels, Eric Patrick. “Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis.” 2013. Doctoral Dissertation, Columbia University. Accessed March 06, 2021. https://doi.org/10.7916/D8TT4Z9W.

MLA Handbook (7th Edition):

Henckels, Eric Patrick. “Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis.” 2013. Web. 06 Mar 2021.

Vancouver:

Henckels EP. Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2021 Mar 06]. Available from: https://doi.org/10.7916/D8TT4Z9W.

Council of Science Editors:

Henckels EP. Regulation of Matrix Metallopeptidase-1 in Breast Cancer Metastasis. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8TT4Z9W


University of Illinois – Chicago

6. Chen, Xinyu. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.

Degree: 2018, University of Illinois – Chicago

 Our previous results suggest that pan-Akt inhibition could exert toxicity indicating that in order to move forward with Akt inhibition for cancer therapy selective Akt… (more)

Subjects/Keywords: Akt; Breast Cancer; Metastasis

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APA (6th Edition):

Chen, X. (2018). The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Xinyu. “The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.” 2018. Thesis, University of Illinois – Chicago. Accessed March 06, 2021. http://hdl.handle.net/10027/23330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Xinyu. “The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.” 2018. Web. 06 Mar 2021.

Vancouver:

Chen X. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10027/23330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen X. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

7. Cao, Yuan. BMP4: a metastasis suppressor gene in breast cancer.

Degree: 2011, University of Melbourne

Breast cancer is the leading cause of cancer related death among Australian women, and results from metastatic disease that develops in vital organs. To reduce… (more)

Subjects/Keywords: breast cancer; metastasis; BMP4; MDSC

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APA (6th Edition):

Cao, Y. (2011). BMP4: a metastasis suppressor gene in breast cancer. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36899

Chicago Manual of Style (16th Edition):

Cao, Yuan. “BMP4: a metastasis suppressor gene in breast cancer.” 2011. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/36899.

MLA Handbook (7th Edition):

Cao, Yuan. “BMP4: a metastasis suppressor gene in breast cancer.” 2011. Web. 06 Mar 2021.

Vancouver:

Cao Y. BMP4: a metastasis suppressor gene in breast cancer. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/36899.

Council of Science Editors:

Cao Y. BMP4: a metastasis suppressor gene in breast cancer. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36899


University of Melbourne

8. Carter, Rachel Zoe. Integrin β3 as a therapeutic target for breast cancer metastasis to bone.

Degree: 2011, University of Melbourne

Breast cancer is the most common cancer in women, with patients currently having high survival rates if the disease is confined to the breast. However,… (more)

Subjects/Keywords: breast cancer; metastasis; bone; integrin

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APA (6th Edition):

Carter, R. Z. (2011). Integrin β3 as a therapeutic target for breast cancer metastasis to bone. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37019

Chicago Manual of Style (16th Edition):

Carter, Rachel Zoe. “Integrin β3 as a therapeutic target for breast cancer metastasis to bone.” 2011. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/37019.

MLA Handbook (7th Edition):

Carter, Rachel Zoe. “Integrin β3 as a therapeutic target for breast cancer metastasis to bone.” 2011. Web. 06 Mar 2021.

Vancouver:

Carter RZ. Integrin β3 as a therapeutic target for breast cancer metastasis to bone. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/37019.

Council of Science Editors:

Carter RZ. Integrin β3 as a therapeutic target for breast cancer metastasis to bone. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/37019


University of Melbourne

9. SWIERCZAK, AGNIESZKA. The role of myeloid cells in breast cancer metastasis.

Degree: 2012, University of Melbourne

Metastasis is the leading cause of death in breast cancer patients. The current methods for predicting likelihood of metastasis are insufficient and treatment options are… (more)

Subjects/Keywords: breast cancer; metastasis; inflammation

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APA (6th Edition):

SWIERCZAK, A. (2012). The role of myeloid cells in breast cancer metastasis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37730

Chicago Manual of Style (16th Edition):

SWIERCZAK, AGNIESZKA. “The role of myeloid cells in breast cancer metastasis.” 2012. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/37730.

MLA Handbook (7th Edition):

SWIERCZAK, AGNIESZKA. “The role of myeloid cells in breast cancer metastasis.” 2012. Web. 06 Mar 2021.

Vancouver:

SWIERCZAK A. The role of myeloid cells in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/37730.

Council of Science Editors:

SWIERCZAK A. The role of myeloid cells in breast cancer metastasis. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37730


Duke University

10. Wang, Jun. Role of ABL Family Kinases in Breast Cancer .

Degree: 2016, Duke University

  The ABL family of non-receptor tyrosine kinases, ABL1 (also known as c-ABL) and ABL2 (also known as Arg), links diverse extracellular stimuli to signaling… (more)

Subjects/Keywords: Pharmacology; ABL; Breast Cancer; Metastasis

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APA (6th Edition):

Wang, J. (2016). Role of ABL Family Kinases in Breast Cancer . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Jun. “Role of ABL Family Kinases in Breast Cancer .” 2016. Thesis, Duke University. Accessed March 06, 2021. http://hdl.handle.net/10161/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Jun. “Role of ABL Family Kinases in Breast Cancer .” 2016. Web. 06 Mar 2021.

Vancouver:

Wang J. Role of ABL Family Kinases in Breast Cancer . [Internet] [Thesis]. Duke University; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10161/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang J. Role of ABL Family Kinases in Breast Cancer . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Mourad, Nancy. Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton.

Degree: Garvan Institute of Medical Research, 2017, University of New South Wales

Breast cancer cells disseminate to the skeleton where they can reside in a dormant state for prolonged periods. These dormant disseminated tumour cells (DTCs) are… (more)

Subjects/Keywords: Bone metastasis; Dormancy; Breast cancer

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APA (6th Edition):

Mourad, N. (2017). Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/58626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:46470/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Mourad, Nancy. “Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton.” 2017. Doctoral Dissertation, University of New South Wales. Accessed March 06, 2021. http://handle.unsw.edu.au/1959.4/58626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:46470/SOURCE02?view=true.

MLA Handbook (7th Edition):

Mourad, Nancy. “Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton.” 2017. Web. 06 Mar 2021.

Vancouver:

Mourad N. Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2021 Mar 06]. Available from: http://handle.unsw.edu.au/1959.4/58626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:46470/SOURCE02?view=true.

Council of Science Editors:

Mourad N. Defining the Transcriptomic Profile of Dormant Breast Cancer Cells in the Skeleton. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/58626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:46470/SOURCE02?view=true


University of Arizona

12. Cheng, Julia N. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .

Degree: 2020, University of Arizona

 Tumors that express the estrogen receptor alpha (ERa) are the most commonly diagnosed breast cancer subtype (>70%), and also have the highest predilection to form… (more)

Subjects/Keywords: breast cancer; estrogen; metastasis

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APA (6th Edition):

Cheng, J. N. (2020). The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/648593

Chicago Manual of Style (16th Edition):

Cheng, Julia N. “The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .” 2020. Doctoral Dissertation, University of Arizona. Accessed March 06, 2021. http://hdl.handle.net/10150/648593.

MLA Handbook (7th Edition):

Cheng, Julia N. “The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .” 2020. Web. 06 Mar 2021.

Vancouver:

Cheng JN. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . [Internet] [Doctoral dissertation]. University of Arizona; 2020. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10150/648593.

Council of Science Editors:

Cheng JN. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . [Doctoral Dissertation]. University of Arizona; 2020. Available from: http://hdl.handle.net/10150/648593


Columbia University

13. Horowicz-Mehler, Nathalie Cecilia. Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer.

Degree: 2017, Columbia University

Metastasis is the most fearsome aspect of breast cancer (BC) a common disease in women, because it drives mortality. Although BC can invade almost any… (more)

Subjects/Keywords: Liver metastasis; Breast – Cancer; Breast – Cancer – Epidemiology; Mastectomy; Metastasis; Epidemiology

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APA (6th Edition):

Horowicz-Mehler, N. C. (2017). Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8HD81C6

Chicago Manual of Style (16th Edition):

Horowicz-Mehler, Nathalie Cecilia. “Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer.” 2017. Doctoral Dissertation, Columbia University. Accessed March 06, 2021. https://doi.org/10.7916/D8HD81C6.

MLA Handbook (7th Edition):

Horowicz-Mehler, Nathalie Cecilia. “Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer.” 2017. Web. 06 Mar 2021.

Vancouver:

Horowicz-Mehler NC. Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2021 Mar 06]. Available from: https://doi.org/10.7916/D8HD81C6.

Council of Science Editors:

Horowicz-Mehler NC. Risk for Lung or Liver Metastasis in Women with Metastatic Breast Cancer. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D8HD81C6

14. Jupp, Lara. The role of Pim-1 in breast cancer metastasis.

Degree: 2017, University of Melbourne

Breast cancer is the most common cancer in women. Despite advances in treatment options, the spread of breast cancer to distant organs (metastasis) remains the… (more)

Subjects/Keywords: cancer; breast cancer; Pim-1; metastasis; kinase

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APA (6th Edition):

Jupp, L. (2017). The role of Pim-1 in breast cancer metastasis. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/191777

Chicago Manual of Style (16th Edition):

Jupp, Lara. “The role of Pim-1 in breast cancer metastasis.” 2017. Masters Thesis, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/191777.

MLA Handbook (7th Edition):

Jupp, Lara. “The role of Pim-1 in breast cancer metastasis.” 2017. Web. 06 Mar 2021.

Vancouver:

Jupp L. The role of Pim-1 in breast cancer metastasis. [Internet] [Masters thesis]. University of Melbourne; 2017. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/191777.

Council of Science Editors:

Jupp L. The role of Pim-1 in breast cancer metastasis. [Masters Thesis]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/191777


Boise State University

15. Tawara, Ken. Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M.

Degree: 2011, Boise State University

Breast cancer is the most diagnosed cancer in women and is the second most common cancer-related death for women worldwide. While the primary tumor itself… (more)

Subjects/Keywords: Breast Cancer; Metastasis; Bone; Cancer Biology

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APA (6th Edition):

Tawara, K. (2011). Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M. (Thesis). Boise State University. Retrieved from https://scholarworks.boisestate.edu/td/196

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tawara, Ken. “Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M.” 2011. Thesis, Boise State University. Accessed March 06, 2021. https://scholarworks.boisestate.edu/td/196.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tawara, Ken. “Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M.” 2011. Web. 06 Mar 2021.

Vancouver:

Tawara K. Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M. [Internet] [Thesis]. Boise State University; 2011. [cited 2021 Mar 06]. Available from: https://scholarworks.boisestate.edu/td/196.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tawara K. Progression of Breast Cancer Metastatic Disease and Subsequent Osteolytic Bone Degradation Mediated by Oncostatin M. [Thesis]. Boise State University; 2011. Available from: https://scholarworks.boisestate.edu/td/196

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

16. Erzinger, Stephanie. Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells.

Degree: MS, 2011, Texas Medical Center

  EphA2, also known as ECK (epithelial cell kinase), is a transmembrane receptor tyrosine kinase that is commonly over-expressed in cancers such as those of… (more)

Subjects/Keywords: EphA2; Breast Cancer; Metastatic Breast Cancer; Metastasis; Breast Cancer Cells; Biology; Cancer Biology

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APA (6th Edition):

Erzinger, S. (2011). Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/116

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Erzinger, Stephanie. “Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells.” 2011. Thesis, Texas Medical Center. Accessed March 06, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/116.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Erzinger, Stephanie. “Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells.” 2011. Web. 06 Mar 2021.

Vancouver:

Erzinger S. Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells. [Internet] [Thesis]. Texas Medical Center; 2011. [cited 2021 Mar 06]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/116.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Erzinger S. Investigating the Effects of Silencing EphA2 in Metastatic Breast Cancer Cells. [Thesis]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/116

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

17. Su, Mei-lin. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Sialylation is catalyzed by sialyltransferases (STs) that adding sialic acids to the terminal positions of oligosaccharide of glycoproteins and glycolipids. This process is frequently enhanced… (more)

Subjects/Keywords: sialylation; CCR7; metastasis; AL10; breast cancer

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APA (6th Edition):

Su, M. (2012). Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Mei-lin. “Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.” 2012. Thesis, NSYSU. Accessed March 06, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Mei-lin. “Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.” 2012. Web. 06 Mar 2021.

Vancouver:

Su M. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Mar 06]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su M. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

18. Sanders, A. BRCA1 breast cancer: metastasis en mouse models.

Degree: 2012, Universiteit Utrecht

 The BRCA1 gene is a tumour suppressor gene frequently mutated in heritable forms of breast cancers. BRCA1 deficient tumours show a basal-like phenotype. Attempts have… (more)

Subjects/Keywords: BRCA1; breast cancer; metastasis; mouse models

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APA (6th Edition):

Sanders, A. (2012). BRCA1 breast cancer: metastasis en mouse models. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/256356

Chicago Manual of Style (16th Edition):

Sanders, A. “BRCA1 breast cancer: metastasis en mouse models.” 2012. Masters Thesis, Universiteit Utrecht. Accessed March 06, 2021. http://dspace.library.uu.nl:8080/handle/1874/256356.

MLA Handbook (7th Edition):

Sanders, A. “BRCA1 breast cancer: metastasis en mouse models.” 2012. Web. 06 Mar 2021.

Vancouver:

Sanders A. BRCA1 breast cancer: metastasis en mouse models. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2021 Mar 06]. Available from: http://dspace.library.uu.nl:8080/handle/1874/256356.

Council of Science Editors:

Sanders A. BRCA1 breast cancer: metastasis en mouse models. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/256356


University of California – San Diego

19. Plunkett, Christopher Michael. Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness.

Degree: Bioengineering, 2018, University of California – San Diego

 The progression of mammary carcinoma involves a variety of cellular and environmental factors that facilitate invasion of the stroma. Stiffening of the extracellular matrix is… (more)

Subjects/Keywords: Bioengineering; Biomaterials; Breast Cancer; Mechanobiology; Metastasis

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APA (6th Edition):

Plunkett, C. M. (2018). Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8854z28x

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Plunkett, Christopher Michael. “Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness.” 2018. Thesis, University of California – San Diego. Accessed March 06, 2021. http://www.escholarship.org/uc/item/8854z28x.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Plunkett, Christopher Michael. “Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness.” 2018. Web. 06 Mar 2021.

Vancouver:

Plunkett CM. Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 06]. Available from: http://www.escholarship.org/uc/item/8854z28x.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Plunkett CM. Modeling Mammary Tumor Invasion in Response to Changes in Matrix Stiffness. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/8854z28x

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

20. Johnson, Rachelle Whitney. Molecular mechanisms of Gli2 regulation in osteolytic bone disease.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

Breast cancer frequently metastasizes to the skeleton with severe consequence to patient quality of life. Patients who develop bone metastases suffer from severe bone pain… (more)

Subjects/Keywords: gli2; pthrp; breast cancer; bone; metastasis

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APA (6th Edition):

Johnson, R. W. (2011). Molecular mechanisms of Gli2 regulation in osteolytic bone disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13912

Chicago Manual of Style (16th Edition):

Johnson, Rachelle Whitney. “Molecular mechanisms of Gli2 regulation in osteolytic bone disease.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed March 06, 2021. http://hdl.handle.net/1803/13912.

MLA Handbook (7th Edition):

Johnson, Rachelle Whitney. “Molecular mechanisms of Gli2 regulation in osteolytic bone disease.” 2011. Web. 06 Mar 2021.

Vancouver:

Johnson RW. Molecular mechanisms of Gli2 regulation in osteolytic bone disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1803/13912.

Council of Science Editors:

Johnson RW. Molecular mechanisms of Gli2 regulation in osteolytic bone disease. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/13912


Penn State University

21. Chandler, Paige Nicole. In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes.

Degree: 2013, Penn State University

 Approximately 80% of women with advanced breast cancer will experience metastasis to bone. Bone metastases often result in skeletal fractures, spinal compression, hypercalcemia, aplastic anemia… (more)

Subjects/Keywords: breast cancer; metastasis; megakaryocytes; osteoblasts; cytokines

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APA (6th Edition):

Chandler, P. N. (2013). In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/18771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chandler, Paige Nicole. “In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes.” 2013. Thesis, Penn State University. Accessed March 06, 2021. https://submit-etda.libraries.psu.edu/catalog/18771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chandler, Paige Nicole. “In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes.” 2013. Web. 06 Mar 2021.

Vancouver:

Chandler PN. In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes. [Internet] [Thesis]. Penn State University; 2013. [cited 2021 Mar 06]. Available from: https://submit-etda.libraries.psu.edu/catalog/18771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chandler PN. In Vitro Interactions Among Breast Cancer Cells, Osteoblasts and Megakaryocytes. [Thesis]. Penn State University; 2013. Available from: https://submit-etda.libraries.psu.edu/catalog/18771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Nairobi

22. Omondi, Marilynn A. Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital .

Degree: 2016, University of Nairobi

 Background: Breast cancer is the second most common malignancy in Kenyan women. Breast cancer survival is linked to early detection, timely and appropriate treatment. Furthermore,… (more)

Subjects/Keywords: Lymph Node Metastasis In Breast Cancer

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APA (6th Edition):

Omondi, M. A. (2016). Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital . (Thesis). University of Nairobi. Retrieved from http://hdl.handle.net/11295/97684

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Omondi, Marilynn A. “Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital .” 2016. Thesis, University of Nairobi. Accessed March 06, 2021. http://hdl.handle.net/11295/97684.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Omondi, Marilynn A. “Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital .” 2016. Web. 06 Mar 2021.

Vancouver:

Omondi MA. Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital . [Internet] [Thesis]. University of Nairobi; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11295/97684.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Omondi MA. Correlation Between Receptor Status And The Presence Of Axillary Lymph Node Metastasis In Breast Cancer In Kenyatta National Hospital . [Thesis]. University of Nairobi; 2016. Available from: http://hdl.handle.net/11295/97684

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

23. Divelbiss, Michelle. Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms.

Degree: MS, Medical Sciences, 2016, Boston University

Metastasis is the leading cause of poor prognosis for individuals diagnosed with cancer. Breast cancer is particularly prevalent with 1 in 10 women receiving a… (more)

Subjects/Keywords: Medicine; Breast cancer; Mena; Metastasis; Neoplasm

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APA (6th Edition):

Divelbiss, M. (2016). Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16749

Chicago Manual of Style (16th Edition):

Divelbiss, Michelle. “Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms.” 2016. Masters Thesis, Boston University. Accessed March 06, 2021. http://hdl.handle.net/2144/16749.

MLA Handbook (7th Edition):

Divelbiss, Michelle. “Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms.” 2016. Web. 06 Mar 2021.

Vancouver:

Divelbiss M. Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/2144/16749.

Council of Science Editors:

Divelbiss M. Developing MenaCalc: an assay to predict risk of breast cancer tumor metastasis through quantification of Mena protein isoforms. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16749


University of Guelph

24. Delroba, Alham. Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer.

Degree: MS, Department of Biomedical Sciences, 2016, University of Guelph

 Transforming growth factor β (TGFβ) signalling promotes breast cancer metastasis, but also inhibits tumour growth during the early stages of cancer by promoting apoptosis. We… (more)

Subjects/Keywords: Potential; therapeutic; options; breast; cancer; metastasis

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APA (6th Edition):

Delroba, A. (2016). Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10140

Chicago Manual of Style (16th Edition):

Delroba, Alham. “Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer.” 2016. Masters Thesis, University of Guelph. Accessed March 06, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10140.

MLA Handbook (7th Edition):

Delroba, Alham. “Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer.” 2016. Web. 06 Mar 2021.

Vancouver:

Delroba A. Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer. [Internet] [Masters thesis]. University of Guelph; 2016. [cited 2021 Mar 06]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10140.

Council of Science Editors:

Delroba A. Par6 links PI3K to the TGFbeta receptor complex: potential implications for PI3K targeting in advanced TGFbeta-dependent breast cancer. [Masters Thesis]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10140


University of Adelaide

25. Akekawatchai, Chareeporn. Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line.

Degree: 2007, University of Adelaide

"The present study investigated the expression and function of IGF-1R, CXCR4 and CCR7, in metastatic MDA-MB-231 and non-metastatic MCF-7 cells." Advisors/Committee Members: School of Molecular and Biomedical Science : Microbiology and Immunology (school).

Subjects/Keywords: Breast cancer; metastasis

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APA (6th Edition):

Akekawatchai, C. (2007). Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/56618

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akekawatchai, Chareeporn. “Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line.” 2007. Thesis, University of Adelaide. Accessed March 06, 2021. http://hdl.handle.net/2440/56618.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akekawatchai, Chareeporn. “Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line.” 2007. Web. 06 Mar 2021.

Vancouver:

Akekawatchai C. Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line. [Internet] [Thesis]. University of Adelaide; 2007. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/2440/56618.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akekawatchai C. Cross-talk between CXCR4 and IGF-1R signal transduction pathways in a metastatic breast cancer cell line. [Thesis]. University of Adelaide; 2007. Available from: http://hdl.handle.net/2440/56618

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

26. Thai, Le Minh. Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance.

Degree: 2007, University of Adelaide

 This thesis describes two studies, one in vitro and one in vivo, which show that Apo2L/TRAIL can prevent breast cancer-induced bone destruction, and highlight the… (more)

Subjects/Keywords: breast cancer; metastasis

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APA (6th Edition):

Thai, L. M. (2007). Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/57334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thai, Le Minh. “Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance.” 2007. Thesis, University of Adelaide. Accessed March 06, 2021. http://hdl.handle.net/2440/57334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thai, Le Minh. “Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance.” 2007. Web. 06 Mar 2021.

Vancouver:

Thai LM. Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance. [Internet] [Thesis]. University of Adelaide; 2007. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/2440/57334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thai LM. Apo2L/TRAIL in breast cancer bone metastasis: in vitro and in vivo studies into molecular mechanisms of action and resistance. [Thesis]. University of Adelaide; 2007. Available from: http://hdl.handle.net/2440/57334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

27. Withana, Nimali P. Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis.

Degree: 2012, University of Melbourne

 The major cause of mortality associated with breast cancer is the development of distant meatastases to sites such as lung and bone. Approximately 70% of… (more)

Subjects/Keywords: breast cancer; metastasis; cysteine cathepsin B

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APA (6th Edition):

Withana, N. P. (2012). Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37012

Chicago Manual of Style (16th Edition):

Withana, Nimali P. “Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis.” 2012. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/37012.

MLA Handbook (7th Edition):

Withana, Nimali P. “Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis.” 2012. Web. 06 Mar 2021.

Vancouver:

Withana NP. Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/37012.

Council of Science Editors:

Withana NP. Contribution of tumour derived cysteine cathepsin B to breast cancer metastasis. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37012


University of Melbourne

28. BURROWS, ALLAN. The contribution of stromal caveolin-1 to breast cancer metastasis.

Degree: 2012, University of Melbourne

 In a previous study, our group has shown that the expression of caveolin-1 (CAV1) in normal tissue surrounding a primary breast cancer is a powerful… (more)

Subjects/Keywords: caveolin; breast cancer; metastasis; fibroblast; stroma

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APA (6th Edition):

BURROWS, A. (2012). The contribution of stromal caveolin-1 to breast cancer metastasis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37974

Chicago Manual of Style (16th Edition):

BURROWS, ALLAN. “The contribution of stromal caveolin-1 to breast cancer metastasis.” 2012. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/37974.

MLA Handbook (7th Edition):

BURROWS, ALLAN. “The contribution of stromal caveolin-1 to breast cancer metastasis.” 2012. Web. 06 Mar 2021.

Vancouver:

BURROWS A. The contribution of stromal caveolin-1 to breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/37974.

Council of Science Editors:

BURROWS A. The contribution of stromal caveolin-1 to breast cancer metastasis. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37974


University of Windsor

29. Crozier, Martin Vance. The pleiotropic effects of dexamethasone on breast cancer cell behaviour.

Degree: PhD, Biological Sciences, 2014, University of Windsor

  Endogenous glucocorticoid (GC) steroids are lipophilic hormones secreted in response to the hypothalamic-pituitary-adrenal axis. Their anti-inflammatory and immunosuppressive potency is the basis for their… (more)

Subjects/Keywords: Breast Cancer; Dexamethasone; Glucocorticoids; Metastasis; Paclitaxel

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APA (6th Edition):

Crozier, M. V. (2014). The pleiotropic effects of dexamethasone on breast cancer cell behaviour. (Doctoral Dissertation). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/5211

Chicago Manual of Style (16th Edition):

Crozier, Martin Vance. “The pleiotropic effects of dexamethasone on breast cancer cell behaviour.” 2014. Doctoral Dissertation, University of Windsor. Accessed March 06, 2021. https://scholar.uwindsor.ca/etd/5211.

MLA Handbook (7th Edition):

Crozier, Martin Vance. “The pleiotropic effects of dexamethasone on breast cancer cell behaviour.” 2014. Web. 06 Mar 2021.

Vancouver:

Crozier MV. The pleiotropic effects of dexamethasone on breast cancer cell behaviour. [Internet] [Doctoral dissertation]. University of Windsor; 2014. [cited 2021 Mar 06]. Available from: https://scholar.uwindsor.ca/etd/5211.

Council of Science Editors:

Crozier MV. The pleiotropic effects of dexamethasone on breast cancer cell behaviour. [Doctoral Dissertation]. University of Windsor; 2014. Available from: https://scholar.uwindsor.ca/etd/5211


University of Melbourne

30. Rautela, Jai. An investigation of type-1 interferon and the immune response against breast cancer metastasis.

Degree: 2016, University of Melbourne

Breast cancer is a highly prevalent disease that, like many cancers, lacks effective therapies aimed at treating and preventing metastasis. Harnessing the host immune system… (more)

Subjects/Keywords: oncology; breast cancer; immunotherapy; interferon; metastasis

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APA (6th Edition):

Rautela, J. (2016). An investigation of type-1 interferon and the immune response against breast cancer metastasis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/119606

Chicago Manual of Style (16th Edition):

Rautela, Jai. “An investigation of type-1 interferon and the immune response against breast cancer metastasis.” 2016. Doctoral Dissertation, University of Melbourne. Accessed March 06, 2021. http://hdl.handle.net/11343/119606.

MLA Handbook (7th Edition):

Rautela, Jai. “An investigation of type-1 interferon and the immune response against breast cancer metastasis.” 2016. Web. 06 Mar 2021.

Vancouver:

Rautela J. An investigation of type-1 interferon and the immune response against breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/11343/119606.

Council of Science Editors:

Rautela J. An investigation of type-1 interferon and the immune response against breast cancer metastasis. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/119606

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