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You searched for subject:(beta catenin). Showing records 1 – 30 of 298 total matches.

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1. Marcato, Vasco. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.

Degree: Docteur es, Génétique, 2015, Sorbonne Paris Cité

La réponse antivirale innée constitue la première réaction d’une cellule à une infection virale. Il s’agit d’une réponse rapide, transitoire, non-spécifique, ubiquitaire qui a été… (more)

Subjects/Keywords: Β-caténine; Β-catenin; 616.042

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APA (6th Edition):

Marcato, V. (2015). La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB103

Chicago Manual of Style (16th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed February 28, 2021. http://www.theses.fr/2015USPCB103.

MLA Handbook (7th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Web. 28 Feb 2021.

Vancouver:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2015USPCB103.

Council of Science Editors:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB103

2. 神宮司, 健太郎. DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

我々はこれまでに細胞性粘菌Dictyostelium discoideumが分泌する分化誘導因子Differentiation-inducing factor-1(DIF-1)がβ-cateninタンパク質の分解を誘導することでWnt/β-cateninシグナル伝達経路を阻害し、ヒトがん細胞株の増殖を抑制することを報告してきた。β-cateninタンパク質の分解がDIF-1の効果に不可欠であるか否かを明らかにするために、我々はWnt/β-cateninシグナル伝達経路が恒常的に活性化されているヒト由来大腸がん細胞株(HCT-116、SW-620、DLD-1)に対するDIF-1の効果を検討した。DIF-1はβ-cateninタンパク質の分解非依存性にcyclin D1の発現をmRNA、タンパク質レベル共に減少させ、G_0/G_1期における細胞周期拘束を起こすことにより、強力に細胞増殖を抑制した。DIF-1によるtranscription factor 7-like 2(TCF7L2)発現量の抑制によって、TCF依存性転写活性及びcyclin D1プロモーター活性が抑制されることが明らかとなった。ルシフェラーゼレポーター活性測定及びTCF7L2プロモーター断片を用いたEMSAにより、翻訳開始点を起点として-609から-601塩基上流に位置する転写因子early growth response-1(Egr-1)結合部位が、DIF-1の効果に関与していることが示された。さらに、RNAi法による内在性TCF7L2の欠失はcyclin D1プロモーター活性及びタンパク質量の減少につながり、そして、TCF7L2の強制発現はDIF-1によるTCF依存性転写活性及びcyclin D1プロモーター活性の低下効果を減弱させた。したがって、DIF-1は大腸がん細胞株において、Egr-1依存性のTCF7L2転写活性を減少させることによりTCF7L2発現を抑制し、Wnt/β-cateninシグナル伝達経路を阻害していることが示唆された。我々の結果は、DIF-1によるWnt/β-cateninシグナル伝達経路の阻害機構に新たな知見をもたらすものである。

We previously reported that differentiation-inducing factor-1 (DIF-1), a morphogen in Dictyostelium discoideum, inhibits… (more)

Subjects/Keywords: DIF-1; Wnt/β-catenin

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APA (6th Edition):

神宮司, . (2013). DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Thesis, Kyushu University / 九州大学. Accessed February 28, 2021. http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Web. 28 Feb 2021.

Vancouver:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

3. Abdulla, Solen. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.

Degree: MSc, 2017, McMaster University

The Wnt/β-catenin pathway is a fundamental regulator of embryonic development and adult tissue homeostasis. The key effector, β-catenin, is a multifunctional protein that occupies dual… (more)

Subjects/Keywords: β-catenin; Stem Cells

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APA (6th Edition):

Abdulla, S. (2017). INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/23461

Chicago Manual of Style (16th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/23461.

MLA Handbook (7th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Web. 28 Feb 2021.

Vancouver:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/23461.

Council of Science Editors:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/23461


University of Melbourne

4. Liu, He. Interaction between p21-activated kinase 1 and beta-catenin.

Degree: 2012, University of Melbourne

 Colorectal cancer (CRC) was the second most frequently occurring cancer and the second leading cause of cancer death in Australia in 2007 (AIHW2010). Hyper-activation of… (more)

Subjects/Keywords: PAK1; beta-catenin; coloractal cancer

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APA (6th Edition):

Liu, H. (2012). Interaction between p21-activated kinase 1 and beta-catenin. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38242

Chicago Manual of Style (16th Edition):

Liu, He. “Interaction between p21-activated kinase 1 and beta-catenin.” 2012. Doctoral Dissertation, University of Melbourne. Accessed February 28, 2021. http://hdl.handle.net/11343/38242.

MLA Handbook (7th Edition):

Liu, He. “Interaction between p21-activated kinase 1 and beta-catenin.” 2012. Web. 28 Feb 2021.

Vancouver:

Liu H. Interaction between p21-activated kinase 1 and beta-catenin. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11343/38242.

Council of Science Editors:

Liu H. Interaction between p21-activated kinase 1 and beta-catenin. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/38242


University of Toronto

5. Shipstone, Arun. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.

Degree: 2015, University of Toronto

Dynamic linkage between the cadherin-catenin complex (CCC) and the actin cytoskeleton at Adherens Junctions is essential for cell-cell adhesion in epithelial cells. Alpha-catenin is a… (more)

Subjects/Keywords: alpha-catenin; Beta-catenin; catenin; cell adhesion; Drosophila; E-Cadherin; 0379

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APA (6th Edition):

Shipstone, A. (2015). Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/88582

Chicago Manual of Style (16th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Masters Thesis, University of Toronto. Accessed February 28, 2021. http://hdl.handle.net/1807/88582.

MLA Handbook (7th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Web. 28 Feb 2021.

Vancouver:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1807/88582.

Council of Science Editors:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/88582


University of Alberta

6. Ha, Jacqueline R. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.

Degree: MS, Medical Sciences-Paediatrics, 2012, University of Alberta

 β-catenin is a potent oncoprotein that serves as a structural anchor at the adherens junctions and as a transcriptional co-activator of the Wnt Signaling pathway.… (more)

Subjects/Keywords: β-catenin; β-catenin is O-GlcNAc modified at Serine 23; O-GlcNAcylation

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APA (6th Edition):

Ha, J. R. (2012). β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jd472x88t

Chicago Manual of Style (16th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Masters Thesis, University of Alberta. Accessed February 28, 2021. https://era.library.ualberta.ca/files/jd472x88t.

MLA Handbook (7th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Web. 28 Feb 2021.

Vancouver:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2021 Feb 28]. Available from: https://era.library.ualberta.ca/files/jd472x88t.

Council of Science Editors:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/jd472x88t

7. Sacco, Sonia. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université Côte d'Azur (ComUE)

La glande surrénale est un organe endocrinien d’une importance vitale de par son rôle dans le maintien de l’homéostasie corporelle. Pour assurer cette fonction, le… (more)

Subjects/Keywords: Glande surrénale; Rspondin; Β-catenin; Zonation; Homéostasie; Adrenal gland; Rspondin; Β-catenin; Zonation; Homeostasis

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APA (6th Edition):

Sacco, S. (2016). Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2016AZUR4125

Chicago Manual of Style (16th Edition):

Sacco, Sonia. “Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.” 2016. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed February 28, 2021. http://www.theses.fr/2016AZUR4125.

MLA Handbook (7th Edition):

Sacco, Sonia. “Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.” 2016. Web. 28 Feb 2021.

Vancouver:

Sacco S. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2016. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2016AZUR4125.

Council of Science Editors:

Sacco S. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2016. Available from: http://www.theses.fr/2016AZUR4125


York University

8. Chimenti, Michael Alfredo. The Role of a -Catenin - FMRP Complex In Skeletal Muscle.

Degree: MSc -MS, Biology, 2019, York University

 Skeletal muscle consists of multinucleated myofibers which generate contractile force and regulate glucose levels in the human body. Over time, skeletal muscle can become damaged… (more)

Subjects/Keywords: Skeletal muscle; Biology; Fragile X Mental Retardation Protein; FMRP; β-catenin; Beta-catenin; translation; post-transcriptional; myofiber; C2C12 myoblast

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APA (6th Edition):

Chimenti, M. A. (2019). The Role of a -Catenin - FMRP Complex In Skeletal Muscle. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/36313

Chicago Manual of Style (16th Edition):

Chimenti, Michael Alfredo. “The Role of a -Catenin - FMRP Complex In Skeletal Muscle.” 2019. Masters Thesis, York University. Accessed February 28, 2021. http://hdl.handle.net/10315/36313.

MLA Handbook (7th Edition):

Chimenti, Michael Alfredo. “The Role of a -Catenin - FMRP Complex In Skeletal Muscle.” 2019. Web. 28 Feb 2021.

Vancouver:

Chimenti MA. The Role of a -Catenin - FMRP Complex In Skeletal Muscle. [Internet] [Masters thesis]. York University; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10315/36313.

Council of Science Editors:

Chimenti MA. The Role of a -Catenin - FMRP Complex In Skeletal Muscle. [Masters Thesis]. York University; 2019. Available from: http://hdl.handle.net/10315/36313


University of Rochester

9. Zhang, Yongchun. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.

Degree: PhD, 2015, University of Rochester

 This work addresses the central question of how B-CATENIN signaling regulates chondrocyte differentiation during endochondral bone formation and cartilage development through integration with BMP and… (more)

Subjects/Keywords: beta-CATENIN; CCN1; BMP2; Chondrocyte; Cartilage

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APA (6th Edition):

Zhang, Y. (2015). B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30113

Chicago Manual of Style (16th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Doctoral Dissertation, University of Rochester. Accessed February 28, 2021. http://hdl.handle.net/1802/30113.

MLA Handbook (7th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Web. 28 Feb 2021.

Vancouver:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1802/30113.

Council of Science Editors:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30113


University of Manchester

10. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: 2011, University of Manchester

 The University of ManchesterAdam GilesDoctor of Philosophy – PhDWnt signalling in oestrogen-induced lactotroph hyperplasia2011The anterior pituitary gland is the major hormonal regulator in the body.… (more)

Subjects/Keywords: Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed February 28, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 28 Feb 2021.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2021 Feb 28]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389


Penn State University

11. Rennoll, Sherri Ann. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.

Degree: 2015, Penn State University

 Over 90% of sporadic colorectal cancers (CRCs) are associated with initiating mutations in components of the Wnt/β-catenin signaling pathway. These mutations result in elevated nuclear… (more)

Subjects/Keywords: MYC; beta-catenin; AXIN2; colorectal cancer

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APA (6th Edition):

Rennoll, S. A. (2015). Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/26902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rennoll, Sherri Ann. “Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.” 2015. Thesis, Penn State University. Accessed February 28, 2021. https://submit-etda.libraries.psu.edu/catalog/26902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rennoll, Sherri Ann. “Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.” 2015. Web. 28 Feb 2021.

Vancouver:

Rennoll SA. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Feb 28]. Available from: https://submit-etda.libraries.psu.edu/catalog/26902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rennoll SA. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/26902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

12. Cunanan, Joanna. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.

Degree: MSMS, 2019, McMaster University

M.Sc. Thesis Dissertation, August 2019, McMaster University

Renal dysplasia, defined as the abnormal development of kidney tissue, is the leading cause of kidney disease in… (more)

Subjects/Keywords: kidney development; beta-catenin; renal dysplasia; quercetin

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APA (6th Edition):

Cunanan, J. (2019). Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24793

Chicago Manual of Style (16th Edition):

Cunanan, Joanna. “Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.” 2019. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/24793.

MLA Handbook (7th Edition):

Cunanan, Joanna. “Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.” 2019. Web. 28 Feb 2021.

Vancouver:

Cunanan J. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/24793.

Council of Science Editors:

Cunanan J. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24793

13. Manring, Heather Renee. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.

Degree: 2012, Wake Forest University

 The Wnt/Beta-catenin signaling pathway has many roles including regulation of developmental pathways, cell proliferation, and homeostasis of adult tissues. Wnt/Beta-catenin signaling activity depends on the… (more)

Subjects/Keywords: Beta-catenin

…contribution of Wnt/β-catenin signaling to key pancreatic functions including beta-cell proliferation… …Wnt/β-catenin pathway genes on beta-cell function will be discussed in greater detail in… …blot WRE Wnt response element β-trcp Beta-transducin repeat containing protein x LIST… …OF FIGURES Page Figure 1.1. Schematic of the Wnt/β-catenin pathway in the inactive (A… …3 Figure 1.2. β-catenin function is partially mediated by its location within the cell… 

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APA (6th Edition):

Manring, H. R. (2012). COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Thesis, Wake Forest University. Accessed February 28, 2021. http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Web. 28 Feb 2021.

Vancouver:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

14. Gowda, Asha. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.

Degree: MS, Medical Sciences, 2014, Boston University

 PURPOSE: Uveal melanoma (UM) is the most common intraocular malignancy in adults with an incidence of six per one million individuals each year. Globe conserving… (more)

Subjects/Keywords: Ophthalmology; Beta-catenin; Melanoma; Protein kinase C

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APA (6th Edition):

Gowda, A. (2014). The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15046

Chicago Manual of Style (16th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Masters Thesis, Boston University. Accessed February 28, 2021. http://hdl.handle.net/2144/15046.

MLA Handbook (7th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Web. 28 Feb 2021.

Vancouver:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Internet] [Masters thesis]. Boston University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2144/15046.

Council of Science Editors:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15046


University of Manchester

15. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: PhD, 2011, University of Manchester

 The anterior pituitary gland is the major hormonal regulator in the body. The gland contains five secretory cell types whose emergence during development is defined… (more)

Subjects/Keywords: 616.4; Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed February 28, 2021. https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 28 Feb 2021.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2021 Feb 28]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396


University of Toronto

16. Al-Jazrawe, Mushriq. Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin.

Degree: PhD, 2019, University of Toronto

 Despite substantial advances in the genomic characterization of neoplasia, effective therapy of many diseases remains elusive. Beyond genomic alterations, several signalling processes maintain the neoplastic… (more)

Subjects/Keywords: beta-catenin; desmoid; fibromatosis; stroma; tumour; 0992

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APA (6th Edition):

Al-Jazrawe, M. (2019). Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/97316

Chicago Manual of Style (16th Edition):

Al-Jazrawe, Mushriq. “Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin.” 2019. Doctoral Dissertation, University of Toronto. Accessed February 28, 2021. http://hdl.handle.net/1807/97316.

MLA Handbook (7th Edition):

Al-Jazrawe, Mushriq. “Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin.” 2019. Web. 28 Feb 2021.

Vancouver:

Al-Jazrawe M. Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin. [Internet] [Doctoral dissertation]. University of Toronto; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1807/97316.

Council of Science Editors:

Al-Jazrawe M. Investigating the Maintenance of Desmoid Tumors beyond Beta-catenin. [Doctoral Dissertation]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/97316


University of Southern California

17. Wu, Jia. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Our lab's previous studies have demonstrated that Wnt/β‐catenin/p300 transcription initiates cell differentiation, whereas Wnt/β‐catenin/cyclic AMP‐responsive element binding protein‐binding protein (CBP) transcription mediates cell proliferation/maintenance of… (more)

Subjects/Keywords: Wnt; β ‐Catenin/p300; Paneth cell

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APA (6th Edition):

Wu, J. (2014). Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958

Chicago Manual of Style (16th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Masters Thesis, University of Southern California. Accessed February 28, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958.

MLA Handbook (7th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Web. 28 Feb 2021.

Vancouver:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2021 Feb 28]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958.

Council of Science Editors:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958


University of Illinois – Urbana-Champaign

18. Xu, Guanying. Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development.

Degree: MS, Food Science & Human Nutrition, 2018, University of Illinois – Urbana-Champaign

 Diet induced obesity (DIO), resulting from long-term consumption of a high fat diet, modifies multiple signaling pathways. These response pathways are closely associated with cell… (more)

Subjects/Keywords: diet-induced-obesity; wnt; beta-catenin; proliferation

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APA (6th Edition):

Xu, G. (2018). Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/101719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Guanying. “Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development.” 2018. Thesis, University of Illinois – Urbana-Champaign. Accessed February 28, 2021. http://hdl.handle.net/2142/101719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Guanying. “Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development.” 2018. Web. 28 Feb 2021.

Vancouver:

Xu G. Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2018. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2142/101719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu G. Diet-induced-obesity suppresses beta-catenin and promotes cell proliferation during colon development. [Thesis]. University of Illinois – Urbana-Champaign; 2018. Available from: http://hdl.handle.net/2142/101719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

19. Hobson, Katherine. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.

Degree: MS, Nutrition, 2018, Georgia State University

  Background: Lung cancer is the leading cause of cancer related death for both men and women. Non-small cell lung cancer (NSCLC) accounts for 80%… (more)

Subjects/Keywords: Lung Cancer; Methionine Restriction; Beta-catenin

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APA (6th Edition):

Hobson, K. (2018). METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/nutrition_theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hobson, Katherine. “METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.” 2018. Thesis, Georgia State University. Accessed February 28, 2021. https://scholarworks.gsu.edu/nutrition_theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hobson, Katherine. “METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.” 2018. Web. 28 Feb 2021.

Vancouver:

Hobson K. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. [Internet] [Thesis]. Georgia State University; 2018. [cited 2021 Feb 28]. Available from: https://scholarworks.gsu.edu/nutrition_theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hobson K. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. [Thesis]. Georgia State University; 2018. Available from: https://scholarworks.gsu.edu/nutrition_theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

20. Dietrich, Philipp. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.

Degree: Women's & Children's Health, 2015, University of New South Wales

 Acute myeloid leukaemia (AML) remains the leading cause of leukaemia-related death with a relative five-year survival rate of only 24% in Australia. This poor prognosis… (more)

Subjects/Keywords: Acute Myeloid Leukaemia; GPR84; MLLAF9; beta-catenin

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APA (6th Edition):

Dietrich, P. (2015). Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Doctoral Dissertation, University of New South Wales. Accessed February 28, 2021. http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

MLA Handbook (7th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Web. 28 Feb 2021.

Vancouver:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2015. [cited 2021 Feb 28]. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

Council of Science Editors:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Doctoral Dissertation]. University of New South Wales; 2015. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true


Université Montpellier II

21. Flacelière, Maud. La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis.

Degree: Docteur es, Biologie Santé, 2012, Université Montpellier II

Les β-arrestines (Arrbs) régulent diverses voies de signalisation dont la voie Wnt/β-caténine (Wnt), un acteur clé dans le cancer colorectal. Le but de mon projet… (more)

Subjects/Keywords: Β-arrestine2; Cancer colorectal; Voie Wnt/β-caténine; Tumorigenèse intestinale; Β-arrestin2; Colorectal cancer; Wnt/β-catenin pathway; Intestinal tumorigenesis

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APA (6th Edition):

Flacelière, M. (2012). La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2012MON20012

Chicago Manual of Style (16th Edition):

Flacelière, Maud. “La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis.” 2012. Doctoral Dissertation, Université Montpellier II. Accessed February 28, 2021. http://www.theses.fr/2012MON20012.

MLA Handbook (7th Edition):

Flacelière, Maud. “La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis.” 2012. Web. 28 Feb 2021.

Vancouver:

Flacelière M. La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis. [Internet] [Doctoral dissertation]. Université Montpellier II; 2012. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2012MON20012.

Council of Science Editors:

Flacelière M. La β-arrestine2, un acteur majeur de la tumorigenèse intestinale dépendante de la voie Wnt/β-caténine. : β-arrestine2, a major actor of the Wnt/β-catenin-dependent intestinal tumorigenesis. [Doctoral Dissertation]. Université Montpellier II; 2012. Available from: http://www.theses.fr/2012MON20012


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

22. Sklavos, Argyrios. Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες.

Degree: 2018, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Recent evidence has suggested that downregulation of the Wnt/βcatenin signaling pathway may contribute to the development and growth of HCC. Consequently, elements of this pathway… (more)

Subjects/Keywords: Μοριακό μονοπάτι Wnt/β-catenin; Μειορρύθμιση; Πολλαπλασιασμός; Απόπτωση; Wnt/β-catenin molecular pathway; Downregulation; Proliferation; Apoptosis

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APA (6th Edition):

Sklavos, A. (2018). Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/43345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sklavos, Argyrios. “Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες.” 2018. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed February 28, 2021. http://hdl.handle.net/10442/hedi/43345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sklavos, Argyrios. “Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες.” 2018. Web. 28 Feb 2021.

Vancouver:

Sklavos A. Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2018. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10442/hedi/43345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sklavos A. Ο ρόλος του Wnt/β-catenin και του εκλεκτικού αναστολέα του στη μετάδοση του σήματος του ηπατοκυτταρικού καρκίνου: πειραματική μελέτη σε μύες. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2018. Available from: http://hdl.handle.net/10442/hedi/43345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

23. Vonbrüll, Matthias. Manipulation of Wnt signaling in tumorcells with antisense molecules.

Degree: 2018, University of Vienna

Seit der Erfindung von Antisense Molekülen gab es zahlreiche Versuche diese therapeutisch anzuwenden. Die FDA Zulassungen von Fomivirsen, Pegaptanib und Mipomersen heben das große Potential… (more)

Subjects/Keywords: 42.13 Molekularbiologie; Antisense Molekül / β-catenin / Morpholino / PNA / Wnt Signalweg; Antisense / β-catenin / Morpholino / PNA / Wnt signaling

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APA (6th Edition):

Vonbrüll, M. (2018). Manipulation of Wnt signaling in tumorcells with antisense molecules. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/53305/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vonbrüll, Matthias. “Manipulation of Wnt signaling in tumorcells with antisense molecules.” 2018. Thesis, University of Vienna. Accessed February 28, 2021. http://othes.univie.ac.at/53305/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vonbrüll, Matthias. “Manipulation of Wnt signaling in tumorcells with antisense molecules.” 2018. Web. 28 Feb 2021.

Vancouver:

Vonbrüll M. Manipulation of Wnt signaling in tumorcells with antisense molecules. [Internet] [Thesis]. University of Vienna; 2018. [cited 2021 Feb 28]. Available from: http://othes.univie.ac.at/53305/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vonbrüll M. Manipulation of Wnt signaling in tumorcells with antisense molecules. [Thesis]. University of Vienna; 2018. Available from: http://othes.univie.ac.at/53305/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Le Rolle, Morgane. Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Côte d'Azur (ComUE)

 La préservation de la fertilité suscite une inquiétude croissante. Depuis les dernières décennies, la fertilité a diminué dans les pays industrialisés. En conséquence, un nombre… (more)

Subjects/Keywords: Cellules germinales; Ovaire; WNT/β-catenin; POU5F1; Fertilité; Germ cells; Ovary; WNT/β-catenin; POU5F1; Pluripotency; Fertility

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Le Rolle, M. (2019). Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2019AZUR6014

Chicago Manual of Style (16th Edition):

Le Rolle, Morgane. “Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals.” 2019. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed February 28, 2021. http://www.theses.fr/2019AZUR6014.

MLA Handbook (7th Edition):

Le Rolle, Morgane. “Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals.” 2019. Web. 28 Feb 2021.

Vancouver:

Le Rolle M. Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2019. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2019AZUR6014.

Council of Science Editors:

Le Rolle M. Voie de signalisation WNT/ β-catenin et différenciation des cellules germinales chez les mammifères : WNT/β-catenin signaling pathway and germ cell differentiation in mammals. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2019. Available from: http://www.theses.fr/2019AZUR6014


University of Helsinki

25. Diaz, Heli. Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models.

Degree: Department of Food and Environmental Sciences; Helsingfors universitet, Agrikultur- och forstvetenskapliga fakulteten, Institutionen för livsmedels- och miljövetenskaper, 2014, University of Helsinki

 Background Protein called ?-catenin has the key role in the Wnt signaling pathway which induces cell division and growth. The disruption of the ?-catenin degradation… (more)

Subjects/Keywords: Ser675; Ser552; β-catenin; phosphorylation; colon cancer; β-kateniini; fosforylaatio; suolistosyöpä; Näringslära; Nutrition; Ravitsemustiede

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APA (6th Edition):

Diaz, H. (2014). Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/45423

Chicago Manual of Style (16th Edition):

Diaz, Heli. “Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models.” 2014. Masters Thesis, University of Helsinki. Accessed February 28, 2021. http://hdl.handle.net/10138/45423.

MLA Handbook (7th Edition):

Diaz, Heli. “Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models.” 2014. Web. 28 Feb 2021.

Vancouver:

Diaz H. Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models. [Internet] [Masters thesis]. University of Helsinki; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10138/45423.

Council of Science Editors:

Diaz H. Ravintotekijöiden vaikutus β-kateniinin aminohappotähteiden fosforylaatioon paksusuolisyöpämalleissa: The effect of dietary components on phosphorylation of β-catenin in colon cancer models. [Masters Thesis]. University of Helsinki; 2014. Available from: http://hdl.handle.net/10138/45423


Université Paris-Sud – Paris XI

26. Sartor, Chiara. Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Université Paris-Sud – Paris XI

 La voie de signalisation WNT/β-caténine est impliquée dans de nombreuses processi cellulaires, du développement à la physiologie. Dans le foie adulte, elle est nécessaire pour… (more)

Subjects/Keywords: Foie; Carcinome hépatocellulaire; Β-caténine; Hnf4α; Liver; Hepatocellular carcinoma; Β-catenin; Hnf4α

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APA (6th Edition):

Sartor, C. (2015). Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA11T046

Chicago Manual of Style (16th Edition):

Sartor, Chiara. “Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed February 28, 2021. http://www.theses.fr/2015PA11T046.

MLA Handbook (7th Edition):

Sartor, Chiara. “Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine.” 2015. Web. 28 Feb 2021.

Vancouver:

Sartor C. Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2015PA11T046.

Council of Science Editors:

Sartor C. Hnf4α and Choline Metabolism Role in β-catenin Activated Liver Carcinogenesis : Le rôle d’Hnfα et du métabolisme de la choline dans les carcinomes hépatocellulaires activés pas la β-caténine. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA11T046

27. Bertrand, Juliette. Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2017, Sorbonne Paris Cité

Les mélanocytes, cellules responsables de la pigmentation de la peau et des poils, protègent les cellules des stress environnementaux, en particulier des rayonnements ultra-violets (UV)… (more)

Subjects/Keywords: Mélanocyte; Pigmentation; UVB; Β-caténine; Dicer; Melanocyte; Pigmentation; UVB; Β-catenin; Dicer; 616.989

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APA (6th Edition):

Bertrand, J. (2017). Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB019

Chicago Manual of Style (16th Edition):

Bertrand, Juliette. “Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed February 28, 2021. http://www.theses.fr/2017USPCB019.

MLA Handbook (7th Edition):

Bertrand, Juliette. “Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage.” 2017. Web. 28 Feb 2021.

Vancouver:

Bertrand J. Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2017USPCB019.

Council of Science Editors:

Bertrand J. Rôle de Dicer dans la pigmentation et sa régulation par les UVB dans le lignage mélanocytaire : Role of Dicer in pigmentation and its regulation by UVB in the melanocyte lineage. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB019

28. Parisi, Alice. Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte.

Degree: Docteur es, Biologie cellulaire, 2014, Université Paris Descartes – Paris V

 La voie de signalisation Wnt/β-caténine (Wnt canonique) est impliquée dans une grande variété de fonctions biologiques, entre autres dans l’établissement des axes embryonnaires, l’organogenèse et… (more)

Subjects/Keywords: Muscle squelettique; APC; WNT; Β-caténine; Skeletal muscle; APC; WNT; Β-catenin; 571.6

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parisi, A. (2014). Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T033

Chicago Manual of Style (16th Edition):

Parisi, Alice. “Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed February 28, 2021. http://www.theses.fr/2014PA05T033.

MLA Handbook (7th Edition):

Parisi, Alice. “Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte.” 2014. Web. 28 Feb 2021.

Vancouver:

Parisi A. Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2014PA05T033.

Council of Science Editors:

Parisi A. Defining the role of APC and canonical WNT signaling in embryonic and adult myogenesis : Etude du rôle d'APC et de la voie de signalisation WNT au cours de la myogenèse embryonnaire et adulte. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T033


University of North Texas

29. Subedi, Ashok. Roles of Primary Cilia in the Oligodendrocyte Lineage.

Degree: 2018, University of North Texas

 Primary cilia are nonmotile, hair-shaped organelles that extend from the basal body in the centrosome. The present study is the first investigation of this organelle… (more)

Subjects/Keywords: Oligodendrocyte; Primary Cilia; Wnt/β-catenin; Myelination; Centrosome

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APA (6th Edition):

Subedi, A. (2018). Roles of Primary Cilia in the Oligodendrocyte Lineage. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc1404594/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Subedi, Ashok. “Roles of Primary Cilia in the Oligodendrocyte Lineage.” 2018. Thesis, University of North Texas. Accessed February 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc1404594/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Subedi, Ashok. “Roles of Primary Cilia in the Oligodendrocyte Lineage.” 2018. Web. 28 Feb 2021.

Vancouver:

Subedi A. Roles of Primary Cilia in the Oligodendrocyte Lineage. [Internet] [Thesis]. University of North Texas; 2018. [cited 2021 Feb 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc1404594/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Subedi A. Roles of Primary Cilia in the Oligodendrocyte Lineage. [Thesis]. University of North Texas; 2018. Available from: https://digital.library.unt.edu/ark:/67531/metadc1404594/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. 吉田, 直裕. Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析.

Degree: 博士(医学), 2018, Kurume University / 久留米大学

2015年度

Subjects/Keywords: Wnt; colorectal cancer; β-catenin

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APA (6th Edition):

吉田, . (2018). Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析. (Thesis). Kurume University / 久留米大学. Retrieved from http://hdl.handle.net/11316/710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

吉田, 直裕. “Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析.” 2018. Thesis, Kurume University / 久留米大学. Accessed February 28, 2021. http://hdl.handle.net/11316/710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

吉田, 直裕. “Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析.” 2018. Web. 28 Feb 2021.

Vancouver:

吉田 . Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析. [Internet] [Thesis]. Kurume University / 久留米大学; 2018. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11316/710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

吉田 . Analysis of Wnt and β-catenin Expression in Advanced Colorectal Cancer : 進行大腸直腸癌におけるWntおよびβカテニン発現の解析. [Thesis]. Kurume University / 久留米大学; 2018. Available from: http://hdl.handle.net/11316/710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] [6] [7] [8] [9] [10]

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