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You searched for subject:(baf57). Showing records 1 – 4 of 4 total matches.

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Cornell University

1. Hah, Nasun. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses.

Degree: PhD, Biochemistry, 2011, Cornell University

 Estrogens play crucial roles in regulating gene expression in physiological and disease states. Estrogens acts through estrogen receptors (ERs) and their binding sites in genomic… (more)

Subjects/Keywords: estrogen; estrogen receptor; GRO-seq; swi/snf; baf57; baf180; silac; proteomic; enhancer; edc; estrogen signaling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hah, N. (2011). Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33589

Chicago Manual of Style (16th Edition):

Hah, Nasun. “Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses.” 2011. Doctoral Dissertation, Cornell University. Accessed January 24, 2021. http://hdl.handle.net/1813/33589.

MLA Handbook (7th Edition):

Hah, Nasun. “Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses.” 2011. Web. 24 Jan 2021.

Vancouver:

Hah N. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1813/33589.

Council of Science Editors:

Hah N. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/33589

2. Faralli, Hervé. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.

Degree: Docteur es, Biologie des eucaryotes, 2010, Aix-Marseille 2

L’unité cellulaire du muscle squelettique est la myofibre, un syncytium hautement spécialisé générant la contraction musculaire. Au cours de la croissance et de la régénération… (more)

Subjects/Keywords: Muscle squelettique; Cellules satellites; Développement; Régénération; Tshz3; MyoD; Myog; Baf57; Swi/snf

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Faralli, H. (2010). Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX22045

Chicago Manual of Style (16th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed January 24, 2021. http://www.theses.fr/2010AIX22045.

MLA Handbook (7th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Web. 24 Jan 2021.

Vancouver:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2021 Jan 24]. Available from: http://www.theses.fr/2010AIX22045.

Council of Science Editors:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX22045

3. LINK, KEVIN A. BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION.

Degree: PhD, Medicine : Cell and Molecular Biology, 2008, University of Cincinnati

 Prostate cancer relies on androgens for growth and survival. Androgens elicit their function primarily through the androgen receptor (AR), a transcription factor that is required… (more)

Subjects/Keywords: Biology, Molecular; prostate cancer; androgen receptor; BAF57; SWI/SNF

BAF57 Modulation of Androgen Receptor Action and Prostate Cancer Progression A… …genes which was dependent on promoter context. Subsequent studies identified the BAF57 subunit… …role in complex recruitment to AR target DNA. BAF57 proved to be critical for AR mediated… …on these findings, the potential for targeting BAF57 as a prostate cancer therapeutic… …option was examined. First, expression of BAF57 was verified in human prostate tissue specimens… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

LINK, K. A. (2008). BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1198854849

Chicago Manual of Style (16th Edition):

LINK, KEVIN A. “BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed January 24, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1198854849.

MLA Handbook (7th Edition):

LINK, KEVIN A. “BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION.” 2008. Web. 24 Jan 2021.

Vancouver:

LINK KA. BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2021 Jan 24]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1198854849.

Council of Science Editors:

LINK KA. BAF57 MODULATION OF ANDROGEN RECEPTOR ACTION AND PROSTATE CANCER PROGRESSION. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1198854849

4. Sethuraman, Aarti. Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis.

Degree: PhD, Biomedical Sciences, 2018, University of Tennessee Health Science Center

Subjects/Keywords: BHLHE40 / DEC1; Breast cancer metastasis; HBEGF; HIF1A; PTK2 / FAK; SMARCE1 / BAF57; Biological Phenomena, Cell Phenomena, and Immunity; Diseases; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences; Neoplasms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sethuraman, A. (2018). Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/454

Chicago Manual of Style (16th Edition):

Sethuraman, Aarti. “Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis.” 2018. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed January 24, 2021. https://dc.uthsc.edu/dissertations/454.

MLA Handbook (7th Edition):

Sethuraman, Aarti. “Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis.” 2018. Web. 24 Jan 2021.

Vancouver:

Sethuraman A. Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2018. [cited 2021 Jan 24]. Available from: https://dc.uthsc.edu/dissertations/454.

Council of Science Editors:

Sethuraman A. Role of SMARCE1 and BHLHE40 in Breast Cancer Metastasis. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2018. Available from: https://dc.uthsc.edu/dissertations/454

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