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You searched for subject:(axonal sprouting). Showing records 1 – 6 of 6 total matches.

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University of Tasmania

1. Brizuela, MDV. Injury induced plasticity in primary neuronal culture and the mature brain.

Degree: 2017, University of Tasmania

 The mature central nervous system (CNS) is unable to fully repair after traumatic brain injury (TBI). Following an injury to the adult brain there is… (more)

Subjects/Keywords: Traumatic Brain Injury; Injury-induced plasticity; Primary neuronal culture; Interneurons; Axonal sprouting; Dendritic remodelling.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brizuela, M. (2017). Injury induced plasticity in primary neuronal culture and the mature brain. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/23792/1/Brizuela_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brizuela, MDV. “Injury induced plasticity in primary neuronal culture and the mature brain.” 2017. Thesis, University of Tasmania. Accessed January 22, 2020. https://eprints.utas.edu.au/23792/1/Brizuela_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brizuela, MDV. “Injury induced plasticity in primary neuronal culture and the mature brain.” 2017. Web. 22 Jan 2020.

Vancouver:

Brizuela M. Injury induced plasticity in primary neuronal culture and the mature brain. [Internet] [Thesis]. University of Tasmania; 2017. [cited 2020 Jan 22]. Available from: https://eprints.utas.edu.au/23792/1/Brizuela_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brizuela M. Injury induced plasticity in primary neuronal culture and the mature brain. [Thesis]. University of Tasmania; 2017. Available from: https://eprints.utas.edu.au/23792/1/Brizuela_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

2. Liang, Justine. Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment.

Degree: Biology, 2016, University of California – San Diego

 Several factors contribute to the lack of repair following spinal cord injury, such as the lack of growth-promoting factors, poor intrinsic central nervous system regeneration… (more)

Subjects/Keywords: Neurosciences; Biology; axonal sprouting; chondroitinase ABC; chondroitin sulfate proteoglycan; corticospinal tract; rhesus monkey; spinal cord injury

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APA (6th Edition):

Liang, J. (2016). Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/02t8c2w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liang, Justine. “Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment.” 2016. Thesis, University of California – San Diego. Accessed January 22, 2020. http://www.escholarship.org/uc/item/02t8c2w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liang, Justine. “Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment.” 2016. Web. 22 Jan 2020.

Vancouver:

Liang J. Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Jan 22]. Available from: http://www.escholarship.org/uc/item/02t8c2w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liang J. Chondroitinase ABC Administration in a Non-Human Primate Model of Spinal Cord Injury: Functional and Anatomical Assessment. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/02t8c2w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

3. Ossowski, Natalie M. Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury.

Degree: 2017, University of Western Ontario

 Many individuals who have suffered from spinal cord injury (SCI) have longstanding damage. The molecular environment of the spinal cord is not permissive to axonal(more)

Subjects/Keywords: Spinal cord injury; Sox9; chondroitin sulfate proteoglycans; perineuronal nets; neuroplasticity; axonal sprouting; Molecular and Cellular Neuroscience

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APA (6th Edition):

Ossowski, N. M. (2017). Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ossowski, Natalie M. “Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury.” 2017. Thesis, University of Western Ontario. Accessed January 22, 2020. https://ir.lib.uwo.ca/etd/4868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ossowski, Natalie M. “Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury.” 2017. Web. 22 Jan 2020.

Vancouver:

Ossowski NM. Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Jan 22]. Available from: https://ir.lib.uwo.ca/etd/4868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ossowski NM. Conditional Sox9 ablation 30 days after spinal cord injury: Testing the therapeutic value of a successful acute strategy to increase neuroplasticity in a model of chronic spinal cord injury. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

4. Witzel, Christian. On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior.

Degree: 2018, Freie Universität Berlin

 A variety of molecular and cellular processes influence peripheral nerve regeneration. They can also affect the morphological behaviour of individual regenerating axons. The general behaviour… (more)

Subjects/Keywords: peripheral nervous system; early axonal regeneration; axonal sprouting/branching; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Witzel, C. (2018). On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-826

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Witzel, Christian. “On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior.” 2018. Thesis, Freie Universität Berlin. Accessed January 22, 2020. http://dx.doi.org/10.17169/refubium-826.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Witzel, Christian. “On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior.” 2018. Web. 22 Jan 2020.

Vancouver:

Witzel C. On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior. [Internet] [Thesis]. Freie Universität Berlin; 2018. [cited 2020 Jan 22]. Available from: http://dx.doi.org/10.17169/refubium-826.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Witzel C. On the morphology of peripheral axonal regeneration: Early behaviour of regenerating axons at the repair site after traumatic lesion, and the potential for influencing this behavior. [Thesis]. Freie Universität Berlin; 2018. Available from: http://dx.doi.org/10.17169/refubium-826

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

5. Tanguay, William. Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée .

Degree: 2017, Université de Montréal

 Les neurones dopaminergiques de la substance noire (SNc) sont les plus vulnérables à la dégénérescence dans la maladie de Parkinson et ses modèles animaux. Suite… (more)

Subjects/Keywords: Maladie de Parkinson; Dopamine; Modèle animal; Vulnérabilité; Arborisation axonale; 6-hydroxydopamine; Substance noire; Aire tegmentaire ventrale; Croissance compensatoire; Animal Model; Parkinson's disease; Animal Model; Vulnerability; Axonal Arborisation; Substantia Nigra; Ventral Tegmental Area; Compensatory Sprouting

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tanguay, W. (2017). Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/18909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tanguay, William. “Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée .” 2017. Thesis, Université de Montréal. Accessed January 22, 2020. http://hdl.handle.net/1866/18909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tanguay, William. “Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée .” 2017. Web. 22 Jan 2020.

Vancouver:

Tanguay W. Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1866/18909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tanguay W. Développement d'un modèle murin de la maladie de Parkinson par augmentation compensatoire de l'arborisation axonale dopaminergique-nigrostriée . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/18909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

6. Cassidy, Pamela. Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease .

Degree: 2018, Université de Montréal

Subjects/Keywords: Maladie de Parkinson; Dopamine; Modèle Animal; Néonatal; 6-hydroxydopamine; Arborization Axonale; Croissance Compensatoire; Vulnérabilité; Substance noir; Aire tegmentaire ventrale; Parkinson’s disease; Dopamine; Animal Model; Neonatal; 6-hydroxydopamine; Axonal arborization; Compensatory Sprouting; Vulnerability; Substantia Nigra; Ventral tegmental area

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cassidy, P. (2018). Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/21382

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cassidy, Pamela. “Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease .” 2018. Thesis, Université de Montréal. Accessed January 22, 2020. http://hdl.handle.net/1866/21382.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cassidy, Pamela. “Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease .” 2018. Web. 22 Jan 2020.

Vancouver:

Cassidy P. Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease . [Internet] [Thesis]. Université de Montréal; 2018. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1866/21382.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cassidy P. Increasing Axonal Arborization Size of Dopamine Neurons to Produce a Better Mouse Model of Parkinson's Disease . [Thesis]. Université de Montréal; 2018. Available from: http://hdl.handle.net/1866/21382

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.