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You searched for subject:(autoimmunity). Showing records 1 – 30 of 547 total matches.

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University of Georgia

1. Rowley, Sean Michael. Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response.

Degree: MS, Veterinary and Biomedical Sciences, 2012, University of Georgia

 The protein kinase Tpl2 (MAP3K8) regulates innate inflammatory responses and is being actively pursued for therapeutic inhibition during chronic autoimmunity. Herein, we addressed the contribution… (more)

Subjects/Keywords: autoimmunity

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APA (6th Edition):

Rowley, S. M. (2012). Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response. (Masters Thesis). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/rowley_sean_m_201212_ms

Chicago Manual of Style (16th Edition):

Rowley, Sean Michael. “Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response.” 2012. Masters Thesis, University of Georgia. Accessed April 02, 2020. http://purl.galileo.usg.edu/uga_etd/rowley_sean_m_201212_ms.

MLA Handbook (7th Edition):

Rowley, Sean Michael. “Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response.” 2012. Web. 02 Apr 2020.

Vancouver:

Rowley SM. Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response. [Internet] [Masters thesis]. University of Georgia; 2012. [cited 2020 Apr 02]. Available from: http://purl.galileo.usg.edu/uga_etd/rowley_sean_m_201212_ms.

Council of Science Editors:

Rowley SM. Tpl2 (MAP3K8) regulates the migration, differentiation, and function of critical innate immune cells during the inflammatory response. [Masters Thesis]. University of Georgia; 2012. Available from: http://purl.galileo.usg.edu/uga_etd/rowley_sean_m_201212_ms

2. Fye, Jason. Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity.

Degree: 2013, Wake Forest University

 TREX1 is a 3’ exonuclease that degrades both single– and double–stranded polynucleotides in order to facilitate orderly cell death and prevent immune dysfunction. Mutations in… (more)

Subjects/Keywords: Autoimmunity

…framework for understanding dysfunctional dsDNA metabolism in TREX1-associated autoimmunity. xi… …phenotypes associated with human autoimmunity correlate with an intact dsDNA binding mechanism… …syndrome and related phenotypes: linking nucleic acid metabolism with autoimmunity. Hum Mol Genet… …82. Stetson, D.B., et al., Trex1 prevents cell-intrinsic initiation of autoimmunity. Cell… …signals that trigger the innate immune response and autoimmunity. We tested this concept using… 

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APA (6th Edition):

Fye, J. (2013). Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/39134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fye, Jason. “Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity.” 2013. Thesis, Wake Forest University. Accessed April 02, 2020. http://hdl.handle.net/10339/39134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fye, Jason. “Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity.” 2013. Web. 02 Apr 2020.

Vancouver:

Fye J. Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity. [Internet] [Thesis]. Wake Forest University; 2013. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10339/39134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fye J. Structural and Biochemical Examination of Aberrant dsDNA Metabolism in TREX1-associated Autoimmunity. [Thesis]. Wake Forest University; 2013. Available from: http://hdl.handle.net/10339/39134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

3. Alharshawi, Khaled Ahmed. PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation.

Degree: 2017, University of Illinois – Chicago

 GM-CSF-induced bone marrow derived dendritic cells (G-BMDCs) have been shown by our lab to be able to selectively cause Treg proliferation when co-cultured with CD4+… (more)

Subjects/Keywords: Tregs; T1D; Autoimmunity

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APA (6th Edition):

Alharshawi, K. A. (2017). PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alharshawi, Khaled Ahmed. “PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation.” 2017. Thesis, University of Illinois – Chicago. Accessed April 02, 2020. http://hdl.handle.net/10027/21959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alharshawi, Khaled Ahmed. “PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation.” 2017. Web. 02 Apr 2020.

Vancouver:

Alharshawi KA. PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10027/21959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alharshawi KA. PKC-ϴ is Dispensable for G-BMDC-Induced TCR-Independent Treg Proliferation. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

4. Malm, Karina. Lifestyle habits and quality of life in established rheumatoid arthritis.

Degree: 2017, University of Lund

 Rheumatoid Arthritis (RA) is a chronic, inflammatory, and systemic disease of unknown aetiology that affects 0.5‒1.0% of the population in Europe, more women than men.… (more)

Subjects/Keywords: Rheumatology and Autoimmunity

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APA (6th Edition):

Malm, K. (2017). Lifestyle habits and quality of life in established rheumatoid arthritis. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/2b6f2c47-038f-46dc-a358-815c6b57f152 ; https://portal.research.lu.se/ws/files/33807223/Karina_Malm_inkl._omslag.pdf

Chicago Manual of Style (16th Edition):

Malm, Karina. “Lifestyle habits and quality of life in established rheumatoid arthritis.” 2017. Doctoral Dissertation, University of Lund. Accessed April 02, 2020. https://lup.lub.lu.se/record/2b6f2c47-038f-46dc-a358-815c6b57f152 ; https://portal.research.lu.se/ws/files/33807223/Karina_Malm_inkl._omslag.pdf.

MLA Handbook (7th Edition):

Malm, Karina. “Lifestyle habits and quality of life in established rheumatoid arthritis.” 2017. Web. 02 Apr 2020.

Vancouver:

Malm K. Lifestyle habits and quality of life in established rheumatoid arthritis. [Internet] [Doctoral dissertation]. University of Lund; 2017. [cited 2020 Apr 02]. Available from: https://lup.lub.lu.se/record/2b6f2c47-038f-46dc-a358-815c6b57f152 ; https://portal.research.lu.se/ws/files/33807223/Karina_Malm_inkl._omslag.pdf.

Council of Science Editors:

Malm K. Lifestyle habits and quality of life in established rheumatoid arthritis. [Doctoral Dissertation]. University of Lund; 2017. Available from: https://lup.lub.lu.se/record/2b6f2c47-038f-46dc-a358-815c6b57f152 ; https://portal.research.lu.se/ws/files/33807223/Karina_Malm_inkl._omslag.pdf


Temple University

5. Schiraldi, Michael. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

There is a role for environmental factors in the pathogenesis of autoimmune diseases in humans and animals. Correlations have been made between… (more)

Subjects/Keywords: Immunology; autoimmunity; mercury; murine

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APA (6th Edition):

Schiraldi, M. (2011). Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,136653

Chicago Manual of Style (16th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,136653.

MLA Handbook (7th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Web. 02 Apr 2020.

Vancouver:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653.

Council of Science Editors:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653


University of Alberta

6. Thangavelu, Govindarajan. Pivotal role of co-inhibitory molecules in immune tolerance.

Degree: PhD, Department of Surgery, 2011, University of Alberta

 The main function of co-inhibitory molecules is to regulate T cell immune responses by providing negative signals to those cells. Homeostatic activation of T cells… (more)

Subjects/Keywords: Tolerance; Programmed Death-1; Autoimmunity

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APA (6th Edition):

Thangavelu, G. (2011). Pivotal role of co-inhibitory molecules in immune tolerance. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/bv73c0666

Chicago Manual of Style (16th Edition):

Thangavelu, Govindarajan. “Pivotal role of co-inhibitory molecules in immune tolerance.” 2011. Doctoral Dissertation, University of Alberta. Accessed April 02, 2020. https://era.library.ualberta.ca/files/bv73c0666.

MLA Handbook (7th Edition):

Thangavelu, Govindarajan. “Pivotal role of co-inhibitory molecules in immune tolerance.” 2011. Web. 02 Apr 2020.

Vancouver:

Thangavelu G. Pivotal role of co-inhibitory molecules in immune tolerance. [Internet] [Doctoral dissertation]. University of Alberta; 2011. [cited 2020 Apr 02]. Available from: https://era.library.ualberta.ca/files/bv73c0666.

Council of Science Editors:

Thangavelu G. Pivotal role of co-inhibitory molecules in immune tolerance. [Doctoral Dissertation]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/bv73c0666


University of Aberdeen

7. Parikh, Khyati. Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE).

Degree: 2009, University of Aberdeen

 To examine the pathophysiological consequences of autoimmunity to TAG-1, CD4+ T cells and autoantibodies specific for TAG-1 were generated and their potential to induce EAE… (more)

Subjects/Keywords: 616.8; Multiple sclerosis : Autoimmunity

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APA (6th Edition):

Parikh, K. (2009). Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE). (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25214 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499706

Chicago Manual of Style (16th Edition):

Parikh, Khyati. “Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE).” 2009. Doctoral Dissertation, University of Aberdeen. Accessed April 02, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25214 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499706.

MLA Handbook (7th Edition):

Parikh, Khyati. “Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE).” 2009. Web. 02 Apr 2020.

Vancouver:

Parikh K. Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE). [Internet] [Doctoral dissertation]. University of Aberdeen; 2009. [cited 2020 Apr 02]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25214 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499706.

Council of Science Editors:

Parikh K. Investigating the role of auto-immune responses to transient axonal glycoprotein-1 (TAG-1) in experimental autoimmune encephalomyelitis (EAE). [Doctoral Dissertation]. University of Aberdeen; 2009. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25214 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499706


University of Aberdeen

8. Minas, Konstantinos. New approaches to autoimmune therapy through gene analysis.

Degree: 2008, University of Aberdeen

 Experimental Autoimmune Uveitis (EAU) is the murine and rat model of the equivalent chronic inflammatory condition in humans. Tolerance to EAU can be induced via… (more)

Subjects/Keywords: 616.079; Uveitis : Autoimmunity : Autoimmune diseases

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APA (6th Edition):

Minas, K. (2008). New approaches to autoimmune therapy through gene analysis. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25620 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499655

Chicago Manual of Style (16th Edition):

Minas, Konstantinos. “New approaches to autoimmune therapy through gene analysis.” 2008. Doctoral Dissertation, University of Aberdeen. Accessed April 02, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25620 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499655.

MLA Handbook (7th Edition):

Minas, Konstantinos. “New approaches to autoimmune therapy through gene analysis.” 2008. Web. 02 Apr 2020.

Vancouver:

Minas K. New approaches to autoimmune therapy through gene analysis. [Internet] [Doctoral dissertation]. University of Aberdeen; 2008. [cited 2020 Apr 02]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25620 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499655.

Council of Science Editors:

Minas K. New approaches to autoimmune therapy through gene analysis. [Doctoral Dissertation]. University of Aberdeen; 2008. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25620 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499655


University of Rochester

9. Roberts, Mustimbo Eli Pollard. Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis.

Degree: PhD, 2012, University of Rochester

 A paucity of identified causative mechanisms in primary Sjogren’s Syndrome (pSS) contributes to inadequate classification criteria utilized for diagnosis. However, known memory-phenotype B cell aberrations… (more)

Subjects/Keywords: Sjögren’s; Autoimmunity; B Cells

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APA (6th Edition):

Roberts, M. E. P. (2012). Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/19710

Chicago Manual of Style (16th Edition):

Roberts, Mustimbo Eli Pollard. “Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis.” 2012. Doctoral Dissertation, University of Rochester. Accessed April 02, 2020. http://hdl.handle.net/1802/19710.

MLA Handbook (7th Edition):

Roberts, Mustimbo Eli Pollard. “Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis.” 2012. Web. 02 Apr 2020.

Vancouver:

Roberts MEP. Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1802/19710.

Council of Science Editors:

Roberts MEP. Memory B cell Phenotypic and Gene Expression Profiling in Primary Sjögren’s Syndrome: Implications for Disease Pathogenesis and Diagnosis. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/19710


Louisiana State University

10. Zimmer, Jacqueline Nicole. The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence.

Degree: MA, Arts and Humanities, 2014, Louisiana State University

 Corruption, unfettered violence, and racist enforcement tactics have historically defined the operations of the New Orleans Police Department, and consequently, many New Orleanians do not… (more)

Subjects/Keywords: street code; narcoeconomy; autoimmunity

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APA (6th Edition):

Zimmer, J. N. (2014). The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence. (Masters Thesis). Louisiana State University. Retrieved from etd-03202014-083129 ; https://digitalcommons.lsu.edu/gradschool_theses/966

Chicago Manual of Style (16th Edition):

Zimmer, Jacqueline Nicole. “The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence.” 2014. Masters Thesis, Louisiana State University. Accessed April 02, 2020. etd-03202014-083129 ; https://digitalcommons.lsu.edu/gradschool_theses/966.

MLA Handbook (7th Edition):

Zimmer, Jacqueline Nicole. “The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence.” 2014. Web. 02 Apr 2020.

Vancouver:

Zimmer JN. The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence. [Internet] [Masters thesis]. Louisiana State University; 2014. [cited 2020 Apr 02]. Available from: etd-03202014-083129 ; https://digitalcommons.lsu.edu/gradschool_theses/966.

Council of Science Editors:

Zimmer JN. The New Orleans murder epidemic: Emmanuel Levinas and Jacques Derrida on the irresponsibility of violence. [Masters Thesis]. Louisiana State University; 2014. Available from: etd-03202014-083129 ; https://digitalcommons.lsu.edu/gradschool_theses/966


University of Debrecen

11. Krümmel, Donia. Autoimmunity, immune dysregulations and their role in encephalitides .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 The aim of this thesis is to review our current understanding of noninfectious immune system responses that may lead to brain damage and neurological deficits.… (more)

Subjects/Keywords: Autoimmunity; encephalitis

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APA (6th Edition):

Krümmel, D. (n.d.). Autoimmunity, immune dysregulations and their role in encephalitides . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/246701

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Krümmel, Donia. “Autoimmunity, immune dysregulations and their role in encephalitides .” Thesis, University of Debrecen. Accessed April 02, 2020. http://hdl.handle.net/2437/246701.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Krümmel, Donia. “Autoimmunity, immune dysregulations and their role in encephalitides .” Web. 02 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Krümmel D. Autoimmunity, immune dysregulations and their role in encephalitides . [Internet] [Thesis]. University of Debrecen; [cited 2020 Apr 02]. Available from: http://hdl.handle.net/2437/246701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Krümmel D. Autoimmunity, immune dysregulations and their role in encephalitides . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/246701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Brigham Young University

12. Clark, Daniel N. Promoter Polymorphisms in Interferon Regulatory Factor 5.

Degree: PhD, 2013, Brigham Young University

  The promoter region of interferon regulatory factor 5 (IRF5) contains the rs2004640 T or G single nucleotide polymorphism (SNP) and a CGGGG indel. Both… (more)

Subjects/Keywords: autoimmunity; cytokines; apoptosis; Microbiology

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APA (6th Edition):

Clark, D. N. (2013). Promoter Polymorphisms in Interferon Regulatory Factor 5. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5057&context=etd

Chicago Manual of Style (16th Edition):

Clark, Daniel N. “Promoter Polymorphisms in Interferon Regulatory Factor 5.” 2013. Doctoral Dissertation, Brigham Young University. Accessed April 02, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5057&context=etd.

MLA Handbook (7th Edition):

Clark, Daniel N. “Promoter Polymorphisms in Interferon Regulatory Factor 5.” 2013. Web. 02 Apr 2020.

Vancouver:

Clark DN. Promoter Polymorphisms in Interferon Regulatory Factor 5. [Internet] [Doctoral dissertation]. Brigham Young University; 2013. [cited 2020 Apr 02]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5057&context=etd.

Council of Science Editors:

Clark DN. Promoter Polymorphisms in Interferon Regulatory Factor 5. [Doctoral Dissertation]. Brigham Young University; 2013. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5057&context=etd


Columbia University

13. Platt, Maryann P. Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice.

Degree: 2019, Columbia University

 Autoimmune encephalitic (AE) syndromes represent a unique manifestation of autoimmunity: the immune system recognizes the brain as foreign, and interferes with neuronal function. AE syndromes… (more)

Subjects/Keywords: Neurosciences; Immunology; Autoimmunity; Streptococcus pyogenes

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APA (6th Edition):

Platt, M. P. (2019). Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-hd2p-gr43

Chicago Manual of Style (16th Edition):

Platt, Maryann P. “Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice.” 2019. Doctoral Dissertation, Columbia University. Accessed April 02, 2020. https://doi.org/10.7916/d8-hd2p-gr43.

MLA Handbook (7th Edition):

Platt, Maryann P. “Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice.” 2019. Web. 02 Apr 2020.

Vancouver:

Platt MP. Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2020 Apr 02]. Available from: https://doi.org/10.7916/d8-hd2p-gr43.

Council of Science Editors:

Platt MP. Cellular Mechanisms of Neurovascular Breakdown and Neuronal Dysfunction Following Recurrent Group A Streptococcus Infections in Mice. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-hd2p-gr43


University of Toronto

14. Minty, Gillian Eleanor Summersgill. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.

Degree: 2013, University of Toronto

The New Zealand Black (NZB) mouse chromosome 13 (c13) is linked to development of autoimmunity. B6 mice containing a portion of NZBc13 (B6.NZBc13 (c13)) develop… (more)

Subjects/Keywords: Autoimmunity; mouse model; SLE; 0982

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APA (6th Edition):

Minty, G. E. S. (2013). The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43245

Chicago Manual of Style (16th Edition):

Minty, Gillian Eleanor Summersgill. “The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.” 2013. Masters Thesis, University of Toronto. Accessed April 02, 2020. http://hdl.handle.net/1807/43245.

MLA Handbook (7th Edition):

Minty, Gillian Eleanor Summersgill. “The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.” 2013. Web. 02 Apr 2020.

Vancouver:

Minty GES. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1807/43245.

Council of Science Editors:

Minty GES. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43245


Victoria University of Wellington

15. O'Sullivan, David. Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis.

Degree: 2012, Victoria University of Wellington

 Multiple sclerosis (MS) is an inflammatory disease, mediated by immune cells attacking the myelin sheaths that surround nerve axons. The autoimmune nature of this disease… (more)

Subjects/Keywords: Autoimmunity; Multiple sclerosis; MS; Immunology

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APA (6th Edition):

O'Sullivan, D. (2012). Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/2126

Chicago Manual of Style (16th Edition):

O'Sullivan, David. “Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis.” 2012. Doctoral Dissertation, Victoria University of Wellington. Accessed April 02, 2020. http://hdl.handle.net/10063/2126.

MLA Handbook (7th Edition):

O'Sullivan, David. “Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis.” 2012. Web. 02 Apr 2020.

Vancouver:

O'Sullivan D. Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2012. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10063/2126.

Council of Science Editors:

O'Sullivan D. Using Experimental Autoimmune Encephalomyelitis to Identify Prospective Treatments for Multiple Sclerosis. [Doctoral Dissertation]. Victoria University of Wellington; 2012. Available from: http://hdl.handle.net/10063/2126

16. White, Madeleine P. J. Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis.

Degree: 2015, Victoria University of Wellington

 Multiple sclerosis (MS) is an immune-mediated disease in which self-reacting T lymphocytes enter the central nervous system (CNS) and direct the damage of the myelin… (more)

Subjects/Keywords: Multiple sclerosis; Innate immunology; Autoimmunity

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APA (6th Edition):

White, M. P. J. (2015). Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/4655

Chicago Manual of Style (16th Edition):

White, Madeleine P J. “Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis.” 2015. Doctoral Dissertation, Victoria University of Wellington. Accessed April 02, 2020. http://hdl.handle.net/10063/4655.

MLA Handbook (7th Edition):

White, Madeleine P J. “Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis.” 2015. Web. 02 Apr 2020.

Vancouver:

White MPJ. Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2015. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10063/4655.

Council of Science Editors:

White MPJ. Innate immunomodulation with MIS416: mechanism of action in experimental autoimmune encephalomyelitis. [Doctoral Dissertation]. Victoria University of Wellington; 2015. Available from: http://hdl.handle.net/10063/4655


Harvard University

17. Ganguli, Sangrag. Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways.

Degree: Master of Medical Sciences, 2019, Harvard University

The vertebrate immune system consists of cytosolic receptors that recognize a wide range of viral nucleic acids during an infection and elicit a robust antiviral… (more)

Subjects/Keywords: Lupus; autoimmunity; viral immunology; screening

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APA (6th Edition):

Ganguli, S. (2019). Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways. (Masters Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057395

Chicago Manual of Style (16th Edition):

Ganguli, Sangrag. “Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways.” 2019. Masters Thesis, Harvard University. Accessed April 02, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057395.

MLA Handbook (7th Edition):

Ganguli, Sangrag. “Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways.” 2019. Web. 02 Apr 2020.

Vancouver:

Ganguli S. Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways. [Internet] [Masters thesis]. Harvard University; 2019. [cited 2020 Apr 02]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057395.

Council of Science Editors:

Ganguli S. Identification of Small Molecule Modulators of the RIG-I and MDA5 Pathways. [Masters Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057395

18. Miller, Katy Jo Coyle. Autoimmunity of Periodontitis.

Degree: 1963, North Texas State University

The purpose of this investigation is to determine if auto-antibodies are demonstrable in inflammatory periodontal disease using methods other than those of Novotny. Advisors/Committee Members: Redden, David R., Guthrie, Rufus K..

Subjects/Keywords: periodontitis; autoimmunity

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APA (6th Edition):

Miller, K. J. C. (1963). Autoimmunity of Periodontitis. (Thesis). North Texas State University. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc108240/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miller, Katy Jo Coyle. “Autoimmunity of Periodontitis.” 1963. Thesis, North Texas State University. Accessed April 02, 2020. https://digital.library.unt.edu/ark:/67531/metadc108240/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miller, Katy Jo Coyle. “Autoimmunity of Periodontitis.” 1963. Web. 02 Apr 2020.

Vancouver:

Miller KJC. Autoimmunity of Periodontitis. [Internet] [Thesis]. North Texas State University; 1963. [cited 2020 Apr 02]. Available from: https://digital.library.unt.edu/ark:/67531/metadc108240/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miller KJC. Autoimmunity of Periodontitis. [Thesis]. North Texas State University; 1963. Available from: https://digital.library.unt.edu/ark:/67531/metadc108240/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

19. Vogelzang, Alexis. Interleukin 21 in immunity and autoimmunity.

Degree: Garvan Institute of Medical Research, 2010, University of New South Wales

 T cell help to B cells is a fundamental property of adaptive immunity, yet only recentlyhave many of the cellular and molecular mechanisms of T… (more)

Subjects/Keywords: Autoimmunity; Interleukin-21; Germinal centers

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APA (6th Edition):

Vogelzang, A. (2010). Interleukin 21 in immunity and autoimmunity. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/45608 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8888/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Vogelzang, Alexis. “Interleukin 21 in immunity and autoimmunity.” 2010. Doctoral Dissertation, University of New South Wales. Accessed April 02, 2020. http://handle.unsw.edu.au/1959.4/45608 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8888/SOURCE02?view=true.

MLA Handbook (7th Edition):

Vogelzang, Alexis. “Interleukin 21 in immunity and autoimmunity.” 2010. Web. 02 Apr 2020.

Vancouver:

Vogelzang A. Interleukin 21 in immunity and autoimmunity. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2020 Apr 02]. Available from: http://handle.unsw.edu.au/1959.4/45608 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8888/SOURCE02?view=true.

Council of Science Editors:

Vogelzang A. Interleukin 21 in immunity and autoimmunity. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/45608 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8888/SOURCE02?view=true


University of Texas Southwestern Medical Center

20. Paek, So Yeon. Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity.

Degree: 2011, University of Texas Southwestern Medical Center

Autoimmunity is a complex process that involves recognition of self antigens by autoreactive T cells or tissue targeting by autoantibodies produced by B cells. Specific… (more)

Subjects/Keywords: Peptide Fragments; T-Lymphocytes; Autoimmunity

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APA (6th Edition):

Paek, S. Y. (2011). Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paek, So Yeon. “Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed April 02, 2020. http://hdl.handle.net/2152.5/927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paek, So Yeon. “Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity.” 2011. Web. 02 Apr 2020.

Vancouver:

Paek SY. Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/2152.5/927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paek SY. Soluble Peptide Treatment Reverses CD8 T Cell-Induced Disease in a Mouse Model of Spontaneous Tissue-Selective Autoimmunity. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

21. Burren, Oliver Simon. Integrative statistical methods for the genomic analysis of immune-mediated disease.

Degree: PhD, 2020, University of Cambridge

 Genome-wide association studies (GWAS) have proved to be a successful method in cataloguing loci influencing thousands of complex human disease phenotypes. However, elucidating the causal… (more)

Subjects/Keywords: Genetics; genomics; statistics; autoimmunity; immunology

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APA (6th Edition):

Burren, O. S. (2020). Integrative statistical methods for the genomic analysis of immune-mediated disease. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301259

Chicago Manual of Style (16th Edition):

Burren, Oliver Simon. “Integrative statistical methods for the genomic analysis of immune-mediated disease.” 2020. Doctoral Dissertation, University of Cambridge. Accessed April 02, 2020. https://www.repository.cam.ac.uk/handle/1810/301259.

MLA Handbook (7th Edition):

Burren, Oliver Simon. “Integrative statistical methods for the genomic analysis of immune-mediated disease.” 2020. Web. 02 Apr 2020.

Vancouver:

Burren OS. Integrative statistical methods for the genomic analysis of immune-mediated disease. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Apr 02]. Available from: https://www.repository.cam.ac.uk/handle/1810/301259.

Council of Science Editors:

Burren OS. Integrative statistical methods for the genomic analysis of immune-mediated disease. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301259


University of Melbourne

22. Ross, Ellen Margaret. Autoantigen-specific regulatory T cells in autoimmunity.

Degree: 2012, University of Melbourne

 In order to protect the host from disease, the immune system is equipped with the capacity to recognise and respond to a plethora of pathogens.… (more)

Subjects/Keywords: regulatory T cells; autoimmunity; tolerance

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APA (6th Edition):

Ross, E. M. (2012). Autoantigen-specific regulatory T cells in autoimmunity. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38044

Chicago Manual of Style (16th Edition):

Ross, Ellen Margaret. “Autoantigen-specific regulatory T cells in autoimmunity.” 2012. Doctoral Dissertation, University of Melbourne. Accessed April 02, 2020. http://hdl.handle.net/11343/38044.

MLA Handbook (7th Edition):

Ross, Ellen Margaret. “Autoantigen-specific regulatory T cells in autoimmunity.” 2012. Web. 02 Apr 2020.

Vancouver:

Ross EM. Autoantigen-specific regulatory T cells in autoimmunity. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/11343/38044.

Council of Science Editors:

Ross EM. Autoantigen-specific regulatory T cells in autoimmunity. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/38044


University of Lund

23. Lood, Christian. On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets.

Degree: 2012, University of Lund

 Systemic lupus erythematosus (SLE) is an autoimmune rheumatic disorder characterized by inflammation in several organ systems. SLE patients have an impaired ability to clear dying… (more)

Subjects/Keywords: Rheumatology and Autoimmunity; systemic lupus erythematosus; autoimmunity; rheumatology; immunology; cardiovascular disease

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APA (6th Edition):

Lood, C. (2012). On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/2440350 ; https://portal.research.lu.se/ws/files/3651543/2440361.pdf

Chicago Manual of Style (16th Edition):

Lood, Christian. “On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets.” 2012. Doctoral Dissertation, University of Lund. Accessed April 02, 2020. https://lup.lub.lu.se/record/2440350 ; https://portal.research.lu.se/ws/files/3651543/2440361.pdf.

MLA Handbook (7th Edition):

Lood, Christian. “On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets.” 2012. Web. 02 Apr 2020.

Vancouver:

Lood C. On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets. [Internet] [Doctoral dissertation]. University of Lund; 2012. [cited 2020 Apr 02]. Available from: https://lup.lub.lu.se/record/2440350 ; https://portal.research.lu.se/ws/files/3651543/2440361.pdf.

Council of Science Editors:

Lood C. On the immunopathogenesis of systemic lupus erythematosus - Immune complexes, type I interferon system, complement system and platelets. [Doctoral Dissertation]. University of Lund; 2012. Available from: https://lup.lub.lu.se/record/2440350 ; https://portal.research.lu.se/ws/files/3651543/2440361.pdf


University of Utah

24. Bolz, Devin Dean. MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;.

Degree: PhD, Pathology;, University of Utah

 Borrelia burgdorferi and Borrelia hermsii represent two vector-borne bacterial pathogens that are causative agents for Lyme disease and relapsing fever, respectively. Though related, these pathogens… (more)

Subjects/Keywords: Autoimmunity; Receptor Signaling

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APA (6th Edition):

Bolz, D. D. (n.d.). MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/91/rec/863

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Bolz, Devin Dean. “MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;.” Doctoral Dissertation, University of Utah. Accessed April 02, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/91/rec/863.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Bolz, Devin Dean. “MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;.” Web. 02 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bolz DD. MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;. [Internet] [Doctoral dissertation]. University of Utah; [cited 2020 Apr 02]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/91/rec/863.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Bolz DD. MyD88 play a pivotal role in host defense to lyme disease and relapsing fever Borrelia;. [Doctoral Dissertation]. University of Utah; Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/91/rec/863

Note: this citation may be lacking information needed for this citation format:
No year of publication.


Texas Medical Center

25. Martinez, Gustavo Javier. MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS.

Degree: PhD, 2011, Texas Medical Center

  CD4+ T helper (Th) lymphocytes are vital for integrating immune responses by orchestrating the function of other immune cell types. Naïve Th cells can… (more)

Subjects/Keywords: immunology; T helper; inflammation; autoimmunity; Immunity

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APA (6th Edition):

Martinez, G. J. (2011). MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/151

Chicago Manual of Style (16th Edition):

Martinez, Gustavo Javier. “MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed April 02, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/151.

MLA Handbook (7th Edition):

Martinez, Gustavo Javier. “MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS.” 2011. Web. 02 Apr 2020.

Vancouver:

Martinez GJ. MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Apr 02]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/151.

Council of Science Editors:

Martinez GJ. MOLECULAR MECHANISMS UNDERLYING THE TRANSCRIPTIONAL REGULATION OF T HELPER 17 AND REGULATORY T CELLS. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/151


Temple University

26. Piaggio, Eduardo. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

The development of autoimmune diseases is frequently linked to exposure to environmental factors such as chemicals, drugs or infections. In the experimental… (more)

Subjects/Keywords: Health Sciences, Immunology; Autoimmunity; Mercury; PD-1

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APA (6th Edition):

Piaggio, E. (2009). The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,52042

Chicago Manual of Style (16th Edition):

Piaggio, Eduardo. “The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,52042.

MLA Handbook (7th Edition):

Piaggio, Eduardo. “The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.” 2009. Web. 02 Apr 2020.

Vancouver:

Piaggio E. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,52042.

Council of Science Editors:

Piaggio E. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,52042


Temple University

27. Gallo, Paul Matthew. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Systemic lupus erythematosus (SLE) is complex autoimmune disease in which autoantibodies form against double stranded DNA (dsDNA) and nuclear antigens. Autoantigen immune… (more)

Subjects/Keywords: Biomechanics; Accounting;

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APA (6th Edition):

Gallo, P. M. (2017). The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,452916

Chicago Manual of Style (16th Edition):

Gallo, Paul Matthew. “The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,452916.

MLA Handbook (7th Edition):

Gallo, Paul Matthew. “The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.” 2017. Web. 02 Apr 2020.

Vancouver:

Gallo PM. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,452916.

Council of Science Editors:

Gallo PM. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,452916

28. Zamoiski, Rachel D. The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure.

Degree: 2014, Johns Hopkins University

 1,25(OH)2D, the biologically active form of vitamin D, is not commonly measured, as it is tightly regulated and does not change with supplementation or sun… (more)

Subjects/Keywords: Vitamin D; metals; inflammation; autoimmunity; blood pressure

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APA (6th Edition):

Zamoiski, R. D. (2014). The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zamoiski, Rachel D. “The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure.” 2014. Thesis, Johns Hopkins University. Accessed April 02, 2020. http://jhir.library.jhu.edu/handle/1774.2/37960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zamoiski, Rachel D. “The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure.” 2014. Web. 02 Apr 2020.

Vancouver:

Zamoiski RD. The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2020 Apr 02]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zamoiski RD. The associations of vitamin D and metal exposures with inflammation, autoimmunity, and blood pressure. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/37960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

29. Smith, Casey. Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis.

Degree: PhD, 2009, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Background: Autoimmune diseases arise from the breakdown of central tolerance resulting in the escape of self reactive T-lymphocytes… (more)

Subjects/Keywords: autoimmune diseases; lymphocytic hypophysitis; autoantigens; pituitary autoimmunity

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APA (6th Edition):

Smith, C. (2009). Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/802688

Chicago Manual of Style (16th Edition):

Smith, Casey. “Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis.” 2009. Doctoral Dissertation, University of Newcastle. Accessed April 02, 2020. http://hdl.handle.net/1959.13/802688.

MLA Handbook (7th Edition):

Smith, Casey. “Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis.” 2009. Web. 02 Apr 2020.

Vancouver:

Smith C. Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis. [Internet] [Doctoral dissertation]. University of Newcastle; 2009. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1959.13/802688.

Council of Science Editors:

Smith C. Autoantibody targets in autoimmune polyendocrine syndrome type 1 and lymphocytic hypophysitis. [Doctoral Dissertation]. University of Newcastle; 2009. Available from: http://hdl.handle.net/1959.13/802688


University of Rochester

30. King, Irah. The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis.

Degree: PhD, 2009, University of Rochester

 Multiple Sclerosis (MS) and its prototypical animal model, Experimental Autoimmune Encephalomyelitis (EAE), are CNS demyelinating diseases believed to be mediated by activation of myelin-reactive CD4+… (more)

Subjects/Keywords: EAE; GM-CSF; Autoimmunity; Dendritic cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

King, I. (2009). The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6614

Chicago Manual of Style (16th Edition):

King, Irah. “The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 02, 2020. http://hdl.handle.net/1802/6614.

MLA Handbook (7th Edition):

King, Irah. “The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis.” 2009. Web. 02 Apr 2020.

Vancouver:

King I. The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1802/6614.

Council of Science Editors:

King I. The Role of granulocyte/macrophage colony-stimulating factor during the priming and effector phases of experimental autoimmune encephalomyelitis. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6614

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