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You searched for subject:(aryl hydrocarbon receptor). Showing records 1 – 30 of 120 total matches.

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Oregon State University

1. Phillips, Jessica Lynne. Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression.

Degree: PhD, 2017, Oregon State University

 The aryl hydrocarbon receptor (AhR) is a ligand-activated transcription factor of the basic helix-loop-helix PER/ARNT/SIM (bHLH/PAS) family and regulates a diverse set of genes. The… (more)

Subjects/Keywords: Aryl hydrocarbon receptor

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APA (6th Edition):

Phillips, J. L. (2017). Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/61709

Chicago Manual of Style (16th Edition):

Phillips, Jessica Lynne. “Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression.” 2017. Doctoral Dissertation, Oregon State University. Accessed January 24, 2021. http://hdl.handle.net/1957/61709.

MLA Handbook (7th Edition):

Phillips, Jessica Lynne. “Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression.” 2017. Web. 24 Jan 2021.

Vancouver:

Phillips JL. Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression. [Internet] [Doctoral dissertation]. Oregon State University; 2017. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1957/61709.

Council of Science Editors:

Phillips JL. Roles for the Aryl Hydrocarbon Receptor in Tumor Suppression. [Doctoral Dissertation]. Oregon State University; 2017. Available from: http://hdl.handle.net/1957/61709


Penn State University

2. DiNatale, Brett C. The role of the aryl hydrocarbon receptor in tumor cell phenotype.

Degree: 2011, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor widely associated with its function in xenobiotic metabolism and its ability to bind environmental pollutants… (more)

Subjects/Keywords: ahr; aryl hydrocarbon receptor; cancer

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APA (6th Edition):

DiNatale, B. C. (2011). The role of the aryl hydrocarbon receptor in tumor cell phenotype. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DiNatale, Brett C. “The role of the aryl hydrocarbon receptor in tumor cell phenotype.” 2011. Thesis, Penn State University. Accessed January 24, 2021. https://submit-etda.libraries.psu.edu/catalog/11597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DiNatale, Brett C. “The role of the aryl hydrocarbon receptor in tumor cell phenotype.” 2011. Web. 24 Jan 2021.

Vancouver:

DiNatale BC. The role of the aryl hydrocarbon receptor in tumor cell phenotype. [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Jan 24]. Available from: https://submit-etda.libraries.psu.edu/catalog/11597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DiNatale BC. The role of the aryl hydrocarbon receptor in tumor cell phenotype. [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

3. Mohinta, Sonia. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.

Degree: PhD, Immunology, 2014, Cornell University

 A number of autoimmune and chronic inflammatory diseases are related to environmental stress. Aryl hydrocarbon receptor (AHR) is regarded as an environmental sensor integrating immune… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Th17; Tcell differentiation

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APA (6th Edition):

Mohinta, S. (2014). Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36177

Chicago Manual of Style (16th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Doctoral Dissertation, Cornell University. Accessed January 24, 2021. http://hdl.handle.net/1813/36177.

MLA Handbook (7th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Web. 24 Jan 2021.

Vancouver:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1813/36177.

Council of Science Editors:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36177


Penn State University

4. Smith, Kayla Jo. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.

Degree: 2017, Penn State University

 Barrier tissues such as the skin and intestine are important for the first line of defense against injury and exposure to potentially harmful toxicants or… (more)

Subjects/Keywords: aryl hydrocarbon receptor; skin; intestine; inflammation; keratinocyte

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APA (6th Edition):

Smith, K. J. (2017). THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13833kjs5049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Kayla Jo. “THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.” 2017. Thesis, Penn State University. Accessed January 24, 2021. https://submit-etda.libraries.psu.edu/catalog/13833kjs5049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Kayla Jo. “THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.” 2017. Web. 24 Jan 2021.

Vancouver:

Smith KJ. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Jan 24]. Available from: https://submit-etda.libraries.psu.edu/catalog/13833kjs5049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith KJ. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/13833kjs5049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

5. Narayanan, Gitanjali A. Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression.

Degree: 2012, Penn State University

 Modulation of aryl hydrocarbon receptor (AHR) activity by a class of ligands termed selective AHR modulators (SAhRMs) has been demonstrated to attenuate pro-inflammatory gene expression… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Complement; CD55; DiMNF

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APA (6th Edition):

Narayanan, G. A. (2012). Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Narayanan, Gitanjali A. “Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression.” 2012. Thesis, Penn State University. Accessed January 24, 2021. https://submit-etda.libraries.psu.edu/catalog/13944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Narayanan, Gitanjali A. “Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression.” 2012. Web. 24 Jan 2021.

Vancouver:

Narayanan GA. Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression. [Internet] [Thesis]. Penn State University; 2012. [cited 2021 Jan 24]. Available from: https://submit-etda.libraries.psu.edu/catalog/13944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Narayanan GA. Selective Modulation of the AH Receptor Leads to Repression of Complement Factor Gene Expression. [Thesis]. Penn State University; 2012. Available from: https://submit-etda.libraries.psu.edu/catalog/13944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Houston

6. Butler, Ryan 1986-. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.

Degree: PhD, Biology, 2013, University of Houston

 Ligand-activated transcription factors are a diverse group of proteins that are involved a variety of physiological processes. The purpose of these studies was to investigate… (more)

Subjects/Keywords: Aryl hydrocarbon receptor; Estrogen receptors; Transcription factors

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APA (6th Edition):

Butler, R. 1. (2013). Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/956

Chicago Manual of Style (16th Edition):

Butler, Ryan 1986-. “Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.” 2013. Doctoral Dissertation, University of Houston. Accessed January 24, 2021. http://hdl.handle.net/10657/956.

MLA Handbook (7th Edition):

Butler, Ryan 1986-. “Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.” 2013. Web. 24 Jan 2021.

Vancouver:

Butler R1. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/10657/956.

Council of Science Editors:

Butler R1. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/956


University of Toronto

7. Rajendra, Sharanya. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.

Degree: 2013, University of Toronto

TiPARP is a PARP-like mART that is induced by and negatively regulates AHR transactivation. Despite these insights, not much is known about TiPARP. This study… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Estrogen Receptor Alpha; TCDD; TiPARP; Gene Regulation; 0419

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APA (6th Edition):

Rajendra, S. (2013). The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43310

Chicago Manual of Style (16th Edition):

Rajendra, Sharanya. “The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.” 2013. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/43310.

MLA Handbook (7th Edition):

Rajendra, Sharanya. “The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.” 2013. Web. 24 Jan 2021.

Vancouver:

Rajendra S. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/43310.

Council of Science Editors:

Rajendra S. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43310


University of Alberta

8. El Gendy, Mohamed, A M. Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 Dioxins are widespread environmental contaminants that have been linked to a variety of deleterious effects on human health including increased cancer rates via aryl hydrocarbon(more)

Subjects/Keywords: Cytochrome P450 1A1; harmol; Aryl Hydrocarbon Receptor; harmalol; harmine; Peganum harmala; Aryl Hydrocarbon Receptor antagonists from natural source; harmaline; harman; Chemoprevention

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APA (6th Edition):

El Gendy, Mohamed, A. M. (2012). Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/f7623d21k

Chicago Manual of Style (16th Edition):

El Gendy, Mohamed, A M. “Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source.” 2012. Doctoral Dissertation, University of Alberta. Accessed January 24, 2021. https://era.library.ualberta.ca/files/f7623d21k.

MLA Handbook (7th Edition):

El Gendy, Mohamed, A M. “Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source.” 2012. Web. 24 Jan 2021.

Vancouver:

El Gendy, Mohamed AM. Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2021 Jan 24]. Available from: https://era.library.ualberta.ca/files/f7623d21k.

Council of Science Editors:

El Gendy, Mohamed AM. Identifying Aryl Hydrocarbon Receptor Modulators from a Natural Source. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/f7623d21k


INP Toulouse

9. Malaisé, Yann. Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders.

Degree: Docteur es, Pathologie, Toxicologie, Génétique et Nutrition, 2017, INP Toulouse

 Le bisphénol A (BPA) est un perturbateur endocrinien couramment employé dans l’industrie agroalimentaire, en particulier pour les matériaux en contact des denrées alimentaires. Son utilisation… (more)

Subjects/Keywords: Bisphénols; Système immunitaire; Intestin; Désordres métaboliques; Aryl hydrocarbon receptor; Bisphenols; Immune system; Gut; Metabolic disorders; Aryl hydrocarbon receptor

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APA (6th Edition):

Malaisé, Y. (2017). Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders. (Doctoral Dissertation). INP Toulouse. Retrieved from http://www.theses.fr/2017INPT0119

Chicago Manual of Style (16th Edition):

Malaisé, Yann. “Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders.” 2017. Doctoral Dissertation, INP Toulouse. Accessed January 24, 2021. http://www.theses.fr/2017INPT0119.

MLA Handbook (7th Edition):

Malaisé, Yann. “Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders.” 2017. Web. 24 Jan 2021.

Vancouver:

Malaisé Y. Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders. [Internet] [Doctoral dissertation]. INP Toulouse; 2017. [cited 2021 Jan 24]. Available from: http://www.theses.fr/2017INPT0119.

Council of Science Editors:

Malaisé Y. Caractérisation chez la souris adulte des impacts immuno-modulateurs des bisphénols après exposition périnatale : conséquences sur la fonction "barrière" de l'intestin et la susceptibilité aux désordres métaboliques : Characterization of immuno-modulatory impacts of bisphenols after perinatal exposure in adult mice : consequences on the gut barrier function and the susceptibility to metabolic disorders. [Doctoral Dissertation]. INP Toulouse; 2017. Available from: http://www.theses.fr/2017INPT0119


University of Toronto

10. Yang, Yang. Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells.

Degree: 2015, University of Toronto

The aryl hydrocarbon receptor (AHR) is a ligand activated transcription factor best known for mediating the toxic actions of environmental contaminants, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).… (more)

Subjects/Keywords: Aryl hydrocarbon receptor (AHR); Aryl hydrocarbon receptor repressor (AHRR); Aryl hydrocarbon response element (AHRE); Chromatin immunoprecipitation followed by high-throughput sequencing (ChIP-Seq); dioxin; DNA binding; 0383

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APA (6th Edition):

Yang, Y. (2015). Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/73180

Chicago Manual of Style (16th Edition):

Yang, Yang. “Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells.” 2015. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/73180.

MLA Handbook (7th Edition):

Yang, Yang. “Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells.” 2015. Web. 24 Jan 2021.

Vancouver:

Yang Y. Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/73180.

Council of Science Editors:

Yang Y. Genome-wide Mapping and Analysis of Aryl Hydrocarbon Receptor (AHR) and Aryl Hydrocarbon Receptor Repressor (AHRR) Bound Regions in MCF-7 Human Breast Cancer Cells. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/73180


University of Toronto

11. Hunter, Sarah Rachel. Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids.

Degree: 2014, University of Toronto

The aryl hydrocarbon receptor (AHR) nuclear translocator (ARNT), a partner in the hypoxia and AHR signaling pathways, is induced in rat liver by dexamethasone (DEX),… (more)

Subjects/Keywords: aryl hydrocarbon receptor nuclear translocator; glucocorticoid receptor; glucocorticoids; pregnane X receptor; rat; signaling pathway; 0419

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APA (6th Edition):

Hunter, S. R. (2014). Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/73165

Chicago Manual of Style (16th Edition):

Hunter, Sarah Rachel. “Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids.” 2014. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/73165.

MLA Handbook (7th Edition):

Hunter, Sarah Rachel. “Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids.” 2014. Web. 24 Jan 2021.

Vancouver:

Hunter SR. Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/73165.

Council of Science Editors:

Hunter SR. Induction of the Rat Hepatic Aryl Hydrocarbon Receptor Nuclear Translocator by Glucocorticoids. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/73165


University of Toronto

12. Crosby, Michael. Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice.

Degree: 2017, University of Toronto

Polycyclic aromatic hydrocarbons (PAHs) are environmental pollutants that activate the aryl hydrocarbon receptor, triggering many biological effects typified by induction of cytochrome P450 1A1 (CYP1A1).… (more)

Subjects/Keywords: aryl hydrocarbon receptor; Cyp3a11; drug metabolism; drug metabolizing enzyme; P450; polycyclic aromatic hydrocarbon; 0419

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APA (6th Edition):

Crosby, M. (2017). Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/79379

Chicago Manual of Style (16th Edition):

Crosby, Michael. “Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice.” 2017. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/79379.

MLA Handbook (7th Edition):

Crosby, Michael. “Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice.” 2017. Web. 24 Jan 2021.

Vancouver:

Crosby M. Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/79379.

Council of Science Editors:

Crosby M. Regulation of Hepatic CYP3A4 by 3-Methylcholanthrene in Humanized PXR-CAR-CYP3A4/3A7 Mice. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/79379


University of Otago

13. Kazantseva, Marina Grigorievna. Smoking, genes and inflammation .

Degree: 2012, University of Otago

 Rheumatoid arthritis (RA) is an, autoimmune disease where genetic predisposition and environmental factors increase the risk of developing RA and the severity of the disease.… (more)

Subjects/Keywords: rheumatoid arthritis; inflammation; smoking; aryl hydrocarbon receptor; matrix metalloproteinases

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APA (6th Edition):

Kazantseva, M. G. (2012). Smoking, genes and inflammation . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/2496

Chicago Manual of Style (16th Edition):

Kazantseva, Marina Grigorievna. “Smoking, genes and inflammation .” 2012. Doctoral Dissertation, University of Otago. Accessed January 24, 2021. http://hdl.handle.net/10523/2496.

MLA Handbook (7th Edition):

Kazantseva, Marina Grigorievna. “Smoking, genes and inflammation .” 2012. Web. 24 Jan 2021.

Vancouver:

Kazantseva MG. Smoking, genes and inflammation . [Internet] [Doctoral dissertation]. University of Otago; 2012. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/10523/2496.

Council of Science Editors:

Kazantseva MG. Smoking, genes and inflammation . [Doctoral Dissertation]. University of Otago; 2012. Available from: http://hdl.handle.net/10523/2496


Universiteit Utrecht

14. Mooij, F.A. de. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.

Degree: 2015, Universiteit Utrecht

 Layman’s summary Everybody knows that eating vegetables and overall healthy food is beneficial for your health. In recent years advertisers also started to tell us… (more)

Subjects/Keywords: ahr; aryl hydrocarbon receptor; probiotics; microbiotics; indole-3-carbinol; tryptophan

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APA (6th Edition):

Mooij, F. A. d. (2015). The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/309341

Chicago Manual of Style (16th Edition):

Mooij, F A de. “The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.” 2015. Masters Thesis, Universiteit Utrecht. Accessed January 24, 2021. http://dspace.library.uu.nl:8080/handle/1874/309341.

MLA Handbook (7th Edition):

Mooij, F A de. “The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.” 2015. Web. 24 Jan 2021.

Vancouver:

Mooij FAd. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. [Internet] [Masters thesis]. Universiteit Utrecht; 2015. [cited 2021 Jan 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/309341.

Council of Science Editors:

Mooij FAd. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. [Masters Thesis]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/309341


University of Rochester

15. Pena, Fanny Lys Casado. Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells.

Degree: PhD, 2011, University of Rochester

 The aryl hydrocarbon receptor (Ahr) mediates the toxicity of certain environmental pollutants including dioxins. Accidental dioxin exposure to humans has been correlated with blood malignancies… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Hematopoiesis; Stem Cells; Toxicology; Dioxin

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APA (6th Edition):

Pena, F. L. C. (2011). Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/15793

Chicago Manual of Style (16th Edition):

Pena, Fanny Lys Casado. “Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells.” 2011. Doctoral Dissertation, University of Rochester. Accessed January 24, 2021. http://hdl.handle.net/1802/15793.

MLA Handbook (7th Edition):

Pena, Fanny Lys Casado. “Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells.” 2011. Web. 24 Jan 2021.

Vancouver:

Pena FLC. Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1802/15793.

Council of Science Editors:

Pena FLC. Activation of the Aryl Hydrocarbon Receptor Signaling by 2,3,7,8 Tetra-chlorodibenzo-p-dioxin (TCDD) Alters Cell Function and Pathway-specific Gene Modulation of Hematopoietic Stem/Progenitor Cells. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/15793


University of Rochester

16. Latchney, Sarah Elizabeth. Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis.

Degree: PhD, 2012, University of Rochester

 The ubiquitous environmental contaminant 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD; Dioxin) has been linked to neurotoxicity in humans and experimental animals. TCDD exerts its toxicity by binding to the… (more)

Subjects/Keywords: Hippocampus; Neurotoxicity; Neurogenesis; Neural Stem Cells; Dioxin; Aryl Hydrocarbon Receptor

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APA (6th Edition):

Latchney, S. E. (2012). Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/19821

Chicago Manual of Style (16th Edition):

Latchney, Sarah Elizabeth. “Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis.” 2012. Doctoral Dissertation, University of Rochester. Accessed January 24, 2021. http://hdl.handle.net/1802/19821.

MLA Handbook (7th Edition):

Latchney, Sarah Elizabeth. “Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis.” 2012. Web. 24 Jan 2021.

Vancouver:

Latchney SE. Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1802/19821.

Council of Science Editors:

Latchney SE. Pluripotent Neural Stem Cells are Targets for Dioxin Toxicity: Novel Roles for the Aryl Hydrocarbon Receptor during Neurogenesis. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/19821


University of Rochester

17. Bennett, John A. Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells.

Degree: PhD, 2015, University of Rochester

 All mature cell lineages of the peripheral blood and adaptive immune system are generated from bone marrow stem cells known as hematopoietic stem cells (HSCs).… (more)

Subjects/Keywords: AhR; Aryl Hydrocarbon Receptor; Hematopoiesis; Hematopoietic Stem Cell

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APA (6th Edition):

Bennett, J. A. (2015). Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30118

Chicago Manual of Style (16th Edition):

Bennett, John A. “Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells.” 2015. Doctoral Dissertation, University of Rochester. Accessed January 24, 2021. http://hdl.handle.net/1802/30118.

MLA Handbook (7th Edition):

Bennett, John A. “Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells.” 2015. Web. 24 Jan 2021.

Vancouver:

Bennett JA. Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1802/30118.

Council of Science Editors:

Bennett JA. Lack of Aryl Hydrocarbon Receptor Alters Gene Expression and Functional Capacity of Murine Hematopoietic Stem Cells. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30118


University of Rochester

18. Boule , Lisbeth A. Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life.

Degree: PhD, 2015, University of Rochester

 Developmental exposures have been shown to alter immune-mediated processes later in life, yet the mechanism by which this occurs is unknown. In fact, in most… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; CD4 T Cell; Developmental Exposure

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APA (6th Edition):

Boule , L. A. (2015). Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30128

Chicago Manual of Style (16th Edition):

Boule , Lisbeth A. “Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life.” 2015. Doctoral Dissertation, University of Rochester. Accessed January 24, 2021. http://hdl.handle.net/1802/30128.

MLA Handbook (7th Edition):

Boule , Lisbeth A. “Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life.” 2015. Web. 24 Jan 2021.

Vancouver:

Boule LA. Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1802/30128.

Council of Science Editors:

Boule LA. Characterizing the Effects of Developmental Activation of the Aryl Hydrocarbon Receptor on CD4+ T Cell Responses Later in Life. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30128


Penn State University

19. Girer, Nathaniel Gabriel. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.

Degree: 2016, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor from the basic helix-loop-helix PER/ARNT/SIM family of proteins that is evolutionarily conserved in both vertebrates… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Fibroblast growth factor 21; Liver Metabolism

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APA (6th Edition):

Girer, N. G. (2016). AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13610nug128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Girer, Nathaniel Gabriel. “AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.” 2016. Thesis, Penn State University. Accessed January 24, 2021. https://submit-etda.libraries.psu.edu/catalog/13610nug128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Girer, Nathaniel Gabriel. “AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.” 2016. Web. 24 Jan 2021.

Vancouver:

Girer NG. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Jan 24]. Available from: https://submit-etda.libraries.psu.edu/catalog/13610nug128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Girer NG. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13610nug128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

20. Hubbard, Troy David. Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor.

Degree: 2017, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand activated transcription factor of the basic region helix-loop-helix-PER-ARNT-SIM (bHLH-PAS) homology super family. The AHR was first identified… (more)

Subjects/Keywords: aryl hydrocarbon receptor; AHR; Evolution; Ligand selectivity; PAHs; Toxicity

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APA (6th Edition):

Hubbard, T. D. (2017). Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13677tdh176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hubbard, Troy David. “Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor.” 2017. Thesis, Penn State University. Accessed January 24, 2021. https://submit-etda.libraries.psu.edu/catalog/13677tdh176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hubbard, Troy David. “Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor.” 2017. Web. 24 Jan 2021.

Vancouver:

Hubbard TD. Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Jan 24]. Available from: https://submit-etda.libraries.psu.edu/catalog/13677tdh176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hubbard TD. Ligand Selectivity and Evolutionary Divergence of the Human Aryl Hydrocarbon Receptor. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/13677tdh176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

21. Wiseman, Steve. CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH.

Degree: 2007, University of Waterloo

 Persistent organic pollutants (POPs), including polychlorinated biphenyls (PCBs) are widespread in aquatic systems. These toxicants bioaccumulate in the tissues of aquatic organisms, especially fish as… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Stress Response

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APA (6th Edition):

Wiseman, S. (2007). CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/3234

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wiseman, Steve. “CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH.” 2007. Thesis, University of Waterloo. Accessed January 24, 2021. http://hdl.handle.net/10012/3234.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wiseman, Steve. “CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH.” 2007. Web. 24 Jan 2021.

Vancouver:

Wiseman S. CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH. [Internet] [Thesis]. University of Waterloo; 2007. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/10012/3234.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wiseman S. CHARACTERIZATION OF AHR SIGNALING AND THE IMPACT OF POLYCHLORINATED BIPHENYLS ON THE ADAPTIVE RESPONSES TO STRESS IN FISH. [Thesis]. University of Waterloo; 2007. Available from: http://hdl.handle.net/10012/3234

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

22. Stanford, Elizabeth Ann. The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells.

Degree: PhD, Molecular and Translational Medicine, 2015, Boston University

 Self-renewing, chemoresistant cancer cells that contribute to cancer metastasis and patient relapse have properties similar to those of stem cells, and have been termed "cancer… (more)

Subjects/Keywords: Molecular biology; Aryl hydrocarbon receptor; Breast cancer; Cancer stem cells

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APA (6th Edition):

Stanford, E. A. (2015). The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/16211

Chicago Manual of Style (16th Edition):

Stanford, Elizabeth Ann. “The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells.” 2015. Doctoral Dissertation, Boston University. Accessed January 24, 2021. http://hdl.handle.net/2144/16211.

MLA Handbook (7th Edition):

Stanford, Elizabeth Ann. “The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells.” 2015. Web. 24 Jan 2021.

Vancouver:

Stanford EA. The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/2144/16211.

Council of Science Editors:

Stanford EA. The role of the aryl hydrocarbon receptor in the development of cells with molecular and functional characteristics of breast cancer stem cells. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16211


University of Toronto

23. Cho, Tiffany Elizabeth. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.

Degree: 2015, University of Toronto

The aryl hydrocarbon receptor (AHR) is a ligand-regulated transcription factor that is activated upon binding to various ligands. The activated AHR modulates the expression of… (more)

Subjects/Keywords: 3-Methylcholanthrene; Aryl Hydrocarbon Receptor; Chylous Ascites; Lethality; TIPARP; Toxicity; 0383

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APA (6th Edition):

Cho, T. E. (2015). 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74562

Chicago Manual of Style (16th Edition):

Cho, Tiffany Elizabeth. “3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.” 2015. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/74562.

MLA Handbook (7th Edition):

Cho, Tiffany Elizabeth. “3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.” 2015. Web. 24 Jan 2021.

Vancouver:

Cho TE. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/74562.

Council of Science Editors:

Cho TE. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74562


University of Toronto

24. Powis, Melanie Lynn. Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals.

Degree: 2011, University of Toronto

The aryl hydrocarbon receptor (AHR) mediates the toxic effects of halogenated aromatic hydrocarbons (HAHs), including 2,3,7,8-tetrachlorodibenzo-p-dioxin, 2,3,4,7,8-pentachlorodibenzofuran and 2,3,7,8-tetrachlorodibenzofuran. Y322 is believed to play a… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Coactivators; Dioxin; Omeprazole; Furans; 0419

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APA (6th Edition):

Powis, M. L. (2011). Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/29602

Chicago Manual of Style (16th Edition):

Powis, Melanie Lynn. “Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals.” 2011. Masters Thesis, University of Toronto. Accessed January 24, 2021. http://hdl.handle.net/1807/29602.

MLA Handbook (7th Edition):

Powis, Melanie Lynn. “Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals.” 2011. Web. 24 Jan 2021.

Vancouver:

Powis ML. Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1807/29602.

Council of Science Editors:

Powis ML. Modulation of Aryl Hydrocarbon Receptor-dependent Transcription by Halogenated Compounds and Pharmaceuticals. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/29602


University of Texas Southwestern Medical Center

25. D'Brot, Alejandro. Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations.

Degree: 2014, University of Texas Southwestern Medical Center

 It is now well appreciated that the apoptosome, which governs caspase-dependent cell death, also drives nonapoptotic caspase activation to remodel cells. However, determinants that specify… (more)

Subjects/Keywords: Apoptosomes; Aryl Hydrocarbon Receptor Nuclear Translocator; Caspases; Drosophila melanogaster; Drosophila Proteins

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APA (6th Edition):

D'Brot, A. (2014). Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

D'Brot, Alejandro. “Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 24, 2021. http://hdl.handle.net/2152.5/3311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

D'Brot, Alejandro. “Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations.” 2014. Web. 24 Jan 2021.

Vancouver:

D'Brot A. Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/2152.5/3311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

D'Brot A. Of Apoptosomes and Oncogenes: Repurposing a Death Machine & Deconstructing the Action of P53 Mutations. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

26. Aflorei, Elena Daniela. Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo.

Degree: PhD, 2016, Queen Mary, University of London

 A phenotypically distinct subgroup of familial isolated pituitary adenoma (FIPA) families has mutations in the aryl hydrocarbon receptor-interacting protein (AIP) gene leading to young-onset acromegaly… (more)

Subjects/Keywords: Pituitary adenoma; Aryl hydrocarbon receptor interacting protein gene

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APA (6th Edition):

Aflorei, E. D. (2016). Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/12511 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775181

Chicago Manual of Style (16th Edition):

Aflorei, Elena Daniela. “Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo.” 2016. Doctoral Dissertation, Queen Mary, University of London. Accessed January 24, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/12511 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775181.

MLA Handbook (7th Edition):

Aflorei, Elena Daniela. “Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo.” 2016. Web. 24 Jan 2021.

Vancouver:

Aflorei ED. Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2016. [cited 2021 Jan 24]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12511 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775181.

Council of Science Editors:

Aflorei ED. Application of a Drosophila melanogaster model to study Familial Isolated Pituitary Adenomas syndrome pathogenesis in vivo. [Doctoral Dissertation]. Queen Mary, University of London; 2016. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12511 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775181


University of Illinois – Urbana-Champaign

27. Basavarajappa, Mallikarjuna. Mechanism of methoxychlor toxicity in mouse ovarian antral follicles.

Degree: PhD, 5274, 2012, University of Illinois – Urbana-Champaign

 1,1,1-trichloro-2,2-bis(4-methoxyphenyl)ethane (methoxychlor; MXC) is an organochlorine pesticide used against pests and insects that attack crops, gardens, vegetables, pets, and livestock. MXC targets the ovary and… (more)

Subjects/Keywords: methoxychlor; antral follicles; ovary; mouse; aryl hydrocarbon receptor; atresia; growth

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APA (6th Edition):

Basavarajappa, M. (2012). Mechanism of methoxychlor toxicity in mouse ovarian antral follicles. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29409

Chicago Manual of Style (16th Edition):

Basavarajappa, Mallikarjuna. “Mechanism of methoxychlor toxicity in mouse ovarian antral follicles.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 24, 2021. http://hdl.handle.net/2142/29409.

MLA Handbook (7th Edition):

Basavarajappa, Mallikarjuna. “Mechanism of methoxychlor toxicity in mouse ovarian antral follicles.” 2012. Web. 24 Jan 2021.

Vancouver:

Basavarajappa M. Mechanism of methoxychlor toxicity in mouse ovarian antral follicles. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/2142/29409.

Council of Science Editors:

Basavarajappa M. Mechanism of methoxychlor toxicity in mouse ovarian antral follicles. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29409


Duke University

28. Van Tiem, Lindsey Anne. Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) .

Degree: 2011, Duke University

  Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous environmental contaminants formed from the incomplete combustion of fossil fuels and are found in the environment as complex… (more)

Subjects/Keywords: Toxicology; aryl hydrocarbon receptor; development; morpholino; polycyclic aromatic hydrocarbons; zebrafish

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APA (6th Edition):

Van Tiem, L. A. (2011). Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Van Tiem, Lindsey Anne. “Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) .” 2011. Thesis, Duke University. Accessed January 24, 2021. http://hdl.handle.net/10161/5705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Van Tiem, Lindsey Anne. “Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) .” 2011. Web. 24 Jan 2021.

Vancouver:

Van Tiem LA. Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) . [Internet] [Thesis]. Duke University; 2011. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/10161/5705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Van Tiem LA. Molecular Mechanisms of Polycyclic Aromatic Hydrocarbon-induced Teratogenesis in Zebrafish (Danio rerio) . [Thesis]. Duke University; 2011. Available from: http://hdl.handle.net/10161/5705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Clemson University

29. Scobie, Micaela Ro. The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages.

Degree: PhD, Biological Sciences, 2019, Clemson University

  Glioblastoma Multiforme (GBM) is a highly invasive brain tumor that affects approximately 18,000 people annually in the USA alone yet remains without curative treatment.… (more)

Subjects/Keywords: Aryl hydrocarbon receptor; E804; Glioblastoma Multiforme; Indirubin; Inflammation; Tumor associated macrophage

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Scobie, M. R. (2019). The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/2491

Chicago Manual of Style (16th Edition):

Scobie, Micaela Ro. “The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages.” 2019. Doctoral Dissertation, Clemson University. Accessed January 24, 2021. https://tigerprints.clemson.edu/all_dissertations/2491.

MLA Handbook (7th Edition):

Scobie, Micaela Ro. “The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages.” 2019. Web. 24 Jan 2021.

Vancouver:

Scobie MR. The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages. [Internet] [Doctoral dissertation]. Clemson University; 2019. [cited 2021 Jan 24]. Available from: https://tigerprints.clemson.edu/all_dissertations/2491.

Council of Science Editors:

Scobie MR. The Effects of Indirubin Derivative E804 on Inflammatory Profiles in Glioblastoma Multiforme Cell Lines and Subsequent Crosstalk Effect on Tumor Associated THP-1 Macrophages. [Doctoral Dissertation]. Clemson University; 2019. Available from: https://tigerprints.clemson.edu/all_dissertations/2491


University of Montana

30. Kreitinger, Joanna Michelle. CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES.

Degree: PhD, 2018, University of Montana

  The Aryl hydrocarbon Receptor (AhR) is a ligand-activated transcription factor best known for its role in xenobiotic metabolism of environmental pollutants. Activation of the… (more)

Subjects/Keywords: Aryl hydrocarbon Receptor; Dendritic cells; drug delivery; nanoparticles; Thymus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kreitinger, J. M. (2018). CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES. (Doctoral Dissertation). University of Montana. Retrieved from https://scholarworks.umt.edu/etd/11302

Chicago Manual of Style (16th Edition):

Kreitinger, Joanna Michelle. “CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES.” 2018. Doctoral Dissertation, University of Montana. Accessed January 24, 2021. https://scholarworks.umt.edu/etd/11302.

MLA Handbook (7th Edition):

Kreitinger, Joanna Michelle. “CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES.” 2018. Web. 24 Jan 2021.

Vancouver:

Kreitinger JM. CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES. [Internet] [Doctoral dissertation]. University of Montana; 2018. [cited 2021 Jan 24]. Available from: https://scholarworks.umt.edu/etd/11302.

Council of Science Editors:

Kreitinger JM. CONSEQUENCES OF ARYL HYDROCARBON RECEPTOR ACTIVATION: THYMIC ATROPHY AND THERAPEUTIC LIPOSOMAL NANOPARTICLES. [Doctoral Dissertation]. University of Montana; 2018. Available from: https://scholarworks.umt.edu/etd/11302

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