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You searched for subject:(apoptosis inhibitor). Showing records 1 – 30 of 74 total matches.

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NSYSU

1. Hou, You-syuan. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 Thyroid carcinoma (TC) is the most common endocrine malignancy and is one of the most rapidly increasing human cancers worldwide. Anaplastic thyroid carcinoma (ATC) is… (more)

Subjects/Keywords: thyroid cancer; BRAF inhibitor; proteomic; apoptosis; resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hou, Y. (2014). Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hou, You-syuan. “Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.” 2014. Thesis, NSYSU. Accessed February 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hou, You-syuan. “Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.” 2014. Web. 25 Feb 2021.

Vancouver:

Hou Y. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Feb 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hou Y. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

2. Shan, Haidong. Neuroprotection gene therapy in retinal degeneration.

Degree: PhD, 2013, University of Oxford

 Retinal degenerative disease, which includes age-related macular degeneration and retinitis pigmentosa, is the main cause of blindness in developed countries. Degeneration of the retinal pigment… (more)

Subjects/Keywords: 617.735; Ophthamology; X-linked inhibitor of apoptosis

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APA (6th Edition):

Shan, H. (2013). Neuroprotection gene therapy in retinal degeneration. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:30f9b5c3-b9a6-4ad0-a952-6840bd73bbc3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581376

Chicago Manual of Style (16th Edition):

Shan, Haidong. “Neuroprotection gene therapy in retinal degeneration.” 2013. Doctoral Dissertation, University of Oxford. Accessed February 25, 2021. http://ora.ox.ac.uk/objects/uuid:30f9b5c3-b9a6-4ad0-a952-6840bd73bbc3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581376.

MLA Handbook (7th Edition):

Shan, Haidong. “Neuroprotection gene therapy in retinal degeneration.” 2013. Web. 25 Feb 2021.

Vancouver:

Shan H. Neuroprotection gene therapy in retinal degeneration. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Feb 25]. Available from: http://ora.ox.ac.uk/objects/uuid:30f9b5c3-b9a6-4ad0-a952-6840bd73bbc3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581376.

Council of Science Editors:

Shan H. Neuroprotection gene therapy in retinal degeneration. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:30f9b5c3-b9a6-4ad0-a952-6840bd73bbc3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581376


University of Ottawa

3. Enwere, Emeka K. Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein .

Degree: 2011, University of Ottawa

 The inhibitor of apoptosis (IAP) proteins traditionally regulate programmed cell death by binding to and inhibiting caspases. Recent studies have uncovered a variety of alternate… (more)

Subjects/Keywords: skeletal muscle; myogenesis; Transcription factor; Apoptosis; Inhibitor of apoptosis; myoblast

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APA (6th Edition):

Enwere, E. K. (2011). Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Enwere, Emeka K. “Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein .” 2011. Thesis, University of Ottawa. Accessed February 25, 2021. http://hdl.handle.net/10393/20048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Enwere, Emeka K. “Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein .” 2011. Web. 25 Feb 2021.

Vancouver:

Enwere EK. Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10393/20048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Enwere EK. Regulation of Skeletal Muscle Formation and Regeneration by the Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

4. Manton, Christa A. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.

Degree: PhD, 2015, Texas Medical Center

  New therapeutic options are needed for glioblastoma, a deadly disease with a median survival of only 14 months with current treatment. The proteasome inhibitor(more)

Subjects/Keywords: bortezomib; marizomib; proteasome; proteasome inhibitor; glioblastoma; caspase; apoptosis; vorinostat; histone deacetylase inhibitor; Cancer Biology

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APA (6th Edition):

Manton, C. A. (2015). Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/545

Chicago Manual of Style (16th Edition):

Manton, Christa A. “Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed February 25, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/545.

MLA Handbook (7th Edition):

Manton, Christa A. “Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.” 2015. Web. 25 Feb 2021.

Vancouver:

Manton CA. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2021 Feb 25]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/545.

Council of Science Editors:

Manton CA. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/545

5. Mehrotra, Swarna. IAP Regulation of Tumor Metastasis: A Dissertation.

Degree: Cancer Biology, Department of Cancer Biology, 2009, U of Massachusetts : Med

  The dissemination of tumor cells to distant organs i.e. metastasis is an exceedingly complex process leading to 90% of all cancer deaths. Despite being… (more)

Subjects/Keywords: Neoplasm Metastasis; Inhibitor of Apoptosis Proteins; Fibronectins; Transcriptional Activation; X-Linked Inhibitor of Apoptosis Protein; Amino Acids, Peptides, and Proteins; Neoplasms

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APA (6th Edition):

Mehrotra, S. (2009). IAP Regulation of Tumor Metastasis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/437

Chicago Manual of Style (16th Edition):

Mehrotra, Swarna. “IAP Regulation of Tumor Metastasis: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed February 25, 2021. https://escholarship.umassmed.edu/gsbs_diss/437.

MLA Handbook (7th Edition):

Mehrotra, Swarna. “IAP Regulation of Tumor Metastasis: A Dissertation.” 2009. Web. 25 Feb 2021.

Vancouver:

Mehrotra S. IAP Regulation of Tumor Metastasis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2021 Feb 25]. Available from: https://escholarship.umassmed.edu/gsbs_diss/437.

Council of Science Editors:

Mehrotra S. IAP Regulation of Tumor Metastasis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/437

6. 甲斐, 健太郎. Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.

Degree: 博士(医学), 2016, Oita University / 大分大学

PROBLEM: The purpose of this study is to evaluate the involvement of death receptor (DR) 6 in the pathogenesis of endometriosis.

METHODS OF STUDY: Endometriotic… (more)

Subjects/Keywords: Apoptosis; death receptor 6; epigenetics; endometriosis; histone deacetylase inhibitor; proliferation

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APA (6th Edition):

甲斐, . (2016). Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

甲斐, 健太郎. “Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.” 2016. Thesis, Oita University / 大分大学. Accessed February 25, 2021. http://hdl.handle.net/10559/15603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

甲斐, 健太郎. “Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis.” 2016. Web. 25 Feb 2021.

Vancouver:

甲斐 . Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10559/15603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

甲斐 . Death Receptor 6 is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bradford

7. Kamala, Ola. A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions.

Degree: PhD, 2014, University of Bradford

 Wounds heal better in skin with terminal hair follicles (large and pigmented) as opposed to those with vellus hair follicles (small and unpigmented), while dermal… (more)

Subjects/Keywords: 616.5; Wound Healing; Inhibitor of Apoptosis Proteins (IAPs); Oestrogen; Hair Follicle

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APA (6th Edition):

Kamala, O. (2014). A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/13841

Chicago Manual of Style (16th Edition):

Kamala, Ola. “A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions.” 2014. Doctoral Dissertation, University of Bradford. Accessed February 25, 2021. http://hdl.handle.net/10454/13841.

MLA Handbook (7th Edition):

Kamala, Ola. “A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions.” 2014. Web. 25 Feb 2021.

Vancouver:

Kamala O. A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions. [Internet] [Doctoral dissertation]. University of Bradford; 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10454/13841.

Council of Science Editors:

Kamala O. A comparison of cultured human dermal fibroblasts derived from terminal and vellus hair bearing skin : differences in the expression of inhibitors of apoptosis proteins, oestrogen receptors, and responses to oestradiol under normal and wound induced conditions. [Doctoral Dissertation]. University of Bradford; 2014. Available from: http://hdl.handle.net/10454/13841

8. COYLE, RACHEL. Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 Malignant rhabdoid tumor (MRT) is a rare paediatric cancer which unfortunately is highly refractive to treatment. Retrospective reviews commonly place the survival rate at approximately… (more)

Subjects/Keywords: Malignant rhabdoid tumour; XIAP; Cancer; Inhibitor of apoptosis proteins

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APA (6th Edition):

COYLE, R. (2019). Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/86089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

COYLE, RACHEL. “Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours.” 2019. Thesis, Trinity College Dublin. Accessed February 25, 2021. http://hdl.handle.net/2262/86089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

COYLE, RACHEL. “Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours.” 2019. Web. 25 Feb 2021.

Vancouver:

COYLE R. Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/2262/86089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

COYLE R. Inhibitor of Apoptosis Proteins as Targets for the Treatment of Malignant Rhabdoid Tumours. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/86089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bath

9. Mafuva, Christopher. Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells.

Degree: Thesis (D.Health), 2019, University of Bath

 Preliminary findings suggest an increase in cervical cancer among sub-Sahara African women on highly active antiretroviral treatment (HAART). There has been a sharp rise in… (more)

Subjects/Keywords: Nucleoside; reverse transcriptase inhibitor; apoptosis; cervical; cancer cells

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APA (6th Edition):

Mafuva, C. (2019). Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/effect-of-ethinylestradiol-and-levonorgestrel-on-nucleoside-reverse-transcriptase-inhibitorinduced-apoptosis-in-human-cervical-epithelial-cancer-cells(9d6b72a6-62d5-4608-bb3e-a0d2b6685f8d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.817805

Chicago Manual of Style (16th Edition):

Mafuva, Christopher. “Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells.” 2019. Doctoral Dissertation, University of Bath. Accessed February 25, 2021. https://researchportal.bath.ac.uk/en/studentthesis/effect-of-ethinylestradiol-and-levonorgestrel-on-nucleoside-reverse-transcriptase-inhibitorinduced-apoptosis-in-human-cervical-epithelial-cancer-cells(9d6b72a6-62d5-4608-bb3e-a0d2b6685f8d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.817805.

MLA Handbook (7th Edition):

Mafuva, Christopher. “Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells.” 2019. Web. 25 Feb 2021.

Vancouver:

Mafuva C. Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells. [Internet] [Doctoral dissertation]. University of Bath; 2019. [cited 2021 Feb 25]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/effect-of-ethinylestradiol-and-levonorgestrel-on-nucleoside-reverse-transcriptase-inhibitorinduced-apoptosis-in-human-cervical-epithelial-cancer-cells(9d6b72a6-62d5-4608-bb3e-a0d2b6685f8d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.817805.

Council of Science Editors:

Mafuva C. Effect of ethinylestradiol and levonorgestrel on nucleoside reverse transcriptase inhibitor-induced apoptosis in human cervical epithelial cancer cells. [Doctoral Dissertation]. University of Bath; 2019. Available from: https://researchportal.bath.ac.uk/en/studentthesis/effect-of-ethinylestradiol-and-levonorgestrel-on-nucleoside-reverse-transcriptase-inhibitorinduced-apoptosis-in-human-cervical-epithelial-cancer-cells(9d6b72a6-62d5-4608-bb3e-a0d2b6685f8d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.817805


University of Southern California

10. Fang, Hua. Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis.

Degree: Doctor of Medicine, Pathobiology, 2012, University of Southern California

 Resistance to cell death, especially to apoptosis, is an important feature of tumor cells, which is also described as one of the hallmarks in cancer.… (more)

Subjects/Keywords: apoptosis; Bcl-2; ABT-737; plasminogen activator inhibitor-1; cancer

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APA (6th Edition):

Fang, H. (2012). Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/33557/rec/6323

Chicago Manual of Style (16th Edition):

Fang, Hua. “Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis.” 2012. Doctoral Dissertation, University of Southern California. Accessed February 25, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/33557/rec/6323.

MLA Handbook (7th Edition):

Fang, Hua. “Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis.” 2012. Web. 25 Feb 2021.

Vancouver:

Fang H. Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Feb 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/33557/rec/6323.

Council of Science Editors:

Fang H. Targeting BCL-2 family proteins and plasminogen activator inhibitor-1 in turmor cell apoptosis. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/33557/rec/6323

11. Wang, Jin. Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors.

Degree: PhD, Pharmaceutical Sciences, 2015, University of Tennessee Health Science Center

  The first part (Chapter 1 and 2) of this dissertation presents a novel combination study of melanoma therapy. Acquired clinical resistance to vemurafenib, a… (more)

Subjects/Keywords: BRAF inhibitor; combination therapy; inhibitor of apoptosis; melanoma; survivin inhibitor; tubulin inhibitor; Medicinal and Pharmaceutical Chemistry; Medicine and Health Sciences; Pharmaceutics and Drug Design; Pharmacy and Pharmaceutical Sciences

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APA (6th Edition):

Wang, J. (2015). Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/282

Chicago Manual of Style (16th Edition):

Wang, Jin. “Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors.” 2015. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed February 25, 2021. https://dc.uthsc.edu/dissertations/282.

MLA Handbook (7th Edition):

Wang, Jin. “Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors.” 2015. Web. 25 Feb 2021.

Vancouver:

Wang J. Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2015. [cited 2021 Feb 25]. Available from: https://dc.uthsc.edu/dissertations/282.

Council of Science Editors:

Wang J. Overcoming Acquired Resistance to BRAF Inhibitors by Novel Synergistic Drug Combination and Discovery of Novel Smac Mimetics as Selective Survivin Inhibitors. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2015. Available from: https://dc.uthsc.edu/dissertations/282


Freie Universität Berlin

12. Quast, Sandra-Annika. Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors.

Degree: 2013, Freie Universität Berlin

 The incidence of malignant melanoma, the most aggressive form of skin cancer, has grown dramatically worldwide in recent decades and is still growing faster than… (more)

Subjects/Keywords: melanoma; TRAIL; apoptosis; overcoming therapy resistance; PI3K inhibitor; mTOR inhibitor; Calcium-dependent potassium channel; 500 Naturwissenschaften und Mathematik

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APA (6th Edition):

Quast, S. (2013). Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quast, Sandra-Annika. “Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors.” 2013. Thesis, Freie Universität Berlin. Accessed February 25, 2021. http://dx.doi.org/10.17169/refubium-8007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quast, Sandra-Annika. “Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors.” 2013. Web. 25 Feb 2021.

Vancouver:

Quast S. Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors. [Internet] [Thesis]. Freie Universität Berlin; 2013. [cited 2021 Feb 25]. Available from: http://dx.doi.org/10.17169/refubium-8007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quast S. Sensitization of melanoma cells for TRAIL-induced apoptosis by signaling pathway inhibitors. [Thesis]. Freie Universität Berlin; 2013. Available from: http://dx.doi.org/10.17169/refubium-8007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

13. Keng, Chun-Lan. The apoptotic mechanism of angiogenesis inhibitor, vasostatin.

Degree: Master, Biological Sciences, 2003, NSYSU

 Abstract Vasostatin, the N-terminal 180 amino acids domain of calreticulin, induces apoptosis in endothelial cells and inhibits angiogenesis. However, the mechanism underlying the apoptosis induce… (more)

Subjects/Keywords: BAEC; vasostatin; angiogenesis inhibitor; apoptosis; ROS

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APA (6th Edition):

Keng, C. (2003). The apoptotic mechanism of angiogenesis inhibitor, vasostatin. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624103-115653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keng, Chun-Lan. “The apoptotic mechanism of angiogenesis inhibitor, vasostatin.” 2003. Thesis, NSYSU. Accessed February 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624103-115653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keng, Chun-Lan. “The apoptotic mechanism of angiogenesis inhibitor, vasostatin.” 2003. Web. 25 Feb 2021.

Vancouver:

Keng C. The apoptotic mechanism of angiogenesis inhibitor, vasostatin. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Feb 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624103-115653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keng C. The apoptotic mechanism of angiogenesis inhibitor, vasostatin. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624103-115653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

14. Lee, Li-ping. Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma.

Degree: Master, Biological Sciences, 2015, NSYSU

 Urothelial carcinoma of urinary bladder (UBUC) and upper tract (UTUC) are common cancer in Taiwan. Most of urothelial carcinoma, especially those of urinary bladder, are… (more)

Subjects/Keywords: P21-activated protein kinase1; apoptosis; PF-3758309; metastasis; PAK inhibitor; urothelial carcinoma

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APA (6th Edition):

Lee, L. (2015). Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612115-111506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Li-ping. “Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma.” 2015. Thesis, NSYSU. Accessed February 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612115-111506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Li-ping. “Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma.” 2015. Web. 25 Feb 2021.

Vancouver:

Lee L. Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Feb 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612115-111506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee L. Exploration of Therapeutic Effects of a Novel p21-activated protein kinase inhibitor, PF-3758309 in Urothelial Carcinoma. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612115-111506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Mezil M'Hidi, Lynda. Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques.

Degree: Docteur es, Pathologie humaine, 2013, Aix Marseille Université

Dans cette étude, nous décrivons pour la première fois les propriétés anti-tumorales de composés de type thiomorpholine sulfonamide hydroxamate dans le cancer du sein. Nous… (more)

Subjects/Keywords: Cancer du sein; Apoptose; Inhibiteur de métalloprotéases; Repositionnement; Breast cancer; Apoptosis; Metalloprteases inhibitor; Repositioning

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APA (6th Edition):

Mezil M'Hidi, L. (2013). Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM5000

Chicago Manual of Style (16th Edition):

Mezil M'Hidi, Lynda. “Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed February 25, 2021. http://www.theses.fr/2013AIXM5000.

MLA Handbook (7th Edition):

Mezil M'Hidi, Lynda. “Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques.” 2013. Web. 25 Feb 2021.

Vancouver:

Mezil M'Hidi L. Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2021 Feb 25]. Available from: http://www.theses.fr/2013AIXM5000.

Council of Science Editors:

Mezil M'Hidi L. Repositionnement d'un inhibiteur de métalloprotéases dans le cancer du sein : Synthèse induite par rayonnement des polymères conducteurs et leurs nanocomposites métalliques. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM5000

16. Oğlakçı İlhan, Ayşegül. Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması .

Degree: ESOGÜ, Tıp Fakültesi, Tıbbi Biyokimya, 2016, Eskisehir Osmangazi University

 Alkol tüketimi ve alkolizm yüksek bir mortalite ve morbiditeye sahip sağlık sorunlarına yol açmaktadır. Aşırı dozda alkol tüketimi önemli bir kalp dokusu hasarı oluşturarak ani… (more)

Subjects/Keywords: Alkol; Miyokart İnfarktüsü (MI); Kalpain İnhibitörü; Apoptoz; Alcohol; Myocard Infarction (MI); Calpain Inhibitor; Apoptosis

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APA (6th Edition):

Oğlakçı İlhan, A. (2016). Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/1057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oğlakçı İlhan, Ayşegül. “Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması .” 2016. Thesis, Eskisehir Osmangazi University. Accessed February 25, 2021. http://hdl.handle.net/11684/1057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oğlakçı İlhan, Ayşegül. “Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması .” 2016. Web. 25 Feb 2021.

Vancouver:

Oğlakçı İlhan A. Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması . [Internet] [Thesis]. Eskisehir Osmangazi University; 2016. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/11684/1057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oğlakçı İlhan A. Ratlarda isoproterenol ile oluşturulan kalp krizi modelinde kronik alkol kullanımının miyokardiyal apoptoz üzerine etkisi ve kalpain inhibitörü 1’in (N-asetil-lösin-lösin-norlösinal) kardiyoprotektif rolünün araştırılması . [Thesis]. Eskisehir Osmangazi University; 2016. Available from: http://hdl.handle.net/11684/1057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

17. Sosin, Angela. Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma.

Degree: PhD, Cancer Biology, 2012, Wayne State University

  Lymphomas frequently retain wild-type (wt) p53 function but overexpress HDM2, compromising p53 activity. Therefore, lymphoma is a suitable model for studying therapeutic value of… (more)

Subjects/Keywords: apoptosis; autoubiquitination; HDM2; lymphoma; p53; Small-molecule inhibitor; Cell Biology; Molecular Biology; Oncology

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APA (6th Edition):

Sosin, A. (2012). Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/622

Chicago Manual of Style (16th Edition):

Sosin, Angela. “Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma.” 2012. Doctoral Dissertation, Wayne State University. Accessed February 25, 2021. https://digitalcommons.wayne.edu/oa_dissertations/622.

MLA Handbook (7th Edition):

Sosin, Angela. “Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma.” 2012. Web. 25 Feb 2021.

Vancouver:

Sosin A. Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma. [Internet] [Doctoral dissertation]. Wayne State University; 2012. [cited 2021 Feb 25]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/622.

Council of Science Editors:

Sosin A. Hdm2 Small-Molecule Inhibitors For Therapeutic Intervention In B-Cell Lymphoma. [Doctoral Dissertation]. Wayne State University; 2012. Available from: https://digitalcommons.wayne.edu/oa_dissertations/622


University of Ottawa

18. Lejmi Mrad, Rim. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .

Degree: 2016, University of Ottawa

 Skeletal muscle atrophy is a debilitating condition caused by pathological conditions including cancer cachexia, disuse and denervation. Disuse atrophy is characterized by reduction in fiber… (more)

Subjects/Keywords: Cellular inhibitor of apoptosis; skeletal muscle atrophy; NF-kB signaling pathway; TWEAK/Fn14 system

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APA (6th Edition):

Lejmi Mrad, R. (2016). Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lejmi Mrad, Rim. “Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .” 2016. Thesis, University of Ottawa. Accessed February 25, 2021. http://hdl.handle.net/10393/34260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lejmi Mrad, Rim. “Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .” 2016. Web. 25 Feb 2021.

Vancouver:

Lejmi Mrad R. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10393/34260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lejmi Mrad R. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

19. Khan, Omar Ali. HR23B, a biomarker for HDAC inhibitors.

Degree: PhD, 2013, University of Oxford

 As our understanding of cancer biology increases and novel therapies are developed, an increasing number of predictive biomarkers are becoming clinically available. Aberrant acetylation has… (more)

Subjects/Keywords: 616.994; Medical Sciences; Clinical laboratory sciences; Oncology; Pharmacology; Tumours; HDAC inhibitor; biomarker; cancer; apoptosis; autophagy

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APA (6th Edition):

Khan, O. A. (2013). HR23B, a biomarker for HDAC inhibitors. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:9cd76c0b-e70e-43f7-a92d-a99f403a077e ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618430

Chicago Manual of Style (16th Edition):

Khan, Omar Ali. “HR23B, a biomarker for HDAC inhibitors.” 2013. Doctoral Dissertation, University of Oxford. Accessed February 25, 2021. http://ora.ox.ac.uk/objects/uuid:9cd76c0b-e70e-43f7-a92d-a99f403a077e ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618430.

MLA Handbook (7th Edition):

Khan, Omar Ali. “HR23B, a biomarker for HDAC inhibitors.” 2013. Web. 25 Feb 2021.

Vancouver:

Khan OA. HR23B, a biomarker for HDAC inhibitors. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Feb 25]. Available from: http://ora.ox.ac.uk/objects/uuid:9cd76c0b-e70e-43f7-a92d-a99f403a077e ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618430.

Council of Science Editors:

Khan OA. HR23B, a biomarker for HDAC inhibitors. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:9cd76c0b-e70e-43f7-a92d-a99f403a077e ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618430


University of Melbourne

20. Anderton, Holly. Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis.

Degree: 2018, University of Melbourne

 The skin is a remarkable organ, a barricade between our vulnerable insides and a constantly changing environment full of physical, chemical, and biological aggressors. Maintenance… (more)

Subjects/Keywords: inhibitor of Apoptosis proteins; Sharpin; cell death; inflammation; skin disease; dermatology; Langerhans cells; microbiome

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APA (6th Edition):

Anderton, H. (2018). Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/221817

Chicago Manual of Style (16th Edition):

Anderton, Holly. “Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis.” 2018. Doctoral Dissertation, University of Melbourne. Accessed February 25, 2021. http://hdl.handle.net/11343/221817.

MLA Handbook (7th Edition):

Anderton, Holly. “Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis.” 2018. Web. 25 Feb 2021.

Vancouver:

Anderton H. Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/11343/221817.

Council of Science Editors:

Anderton H. Inhibitor of APoptosis proteins (IAPs) and SHARPIN regulate the immune response in the skin to limit inflammation and maintain homeostasis. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/221817


University of Melbourne

21. Pollock, Georgina Lauren. Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis.

Degree: 2019, University of Melbourne

 During infection the gastrointestinal pathogen enteropathogenic Escherichia coli (EPEC) forms a characteristic lesion on the surface of infected enterocytes known as an attaching effacing lesion.… (more)

Subjects/Keywords: pathogenic E. coli; EPEC; apoptosis; caspase inhibitor; FAS signalling; bacterial kinase; AE lesion; microvilli effacement

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APA (6th Edition):

Pollock, G. L. (2019). Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/227758

Chicago Manual of Style (16th Edition):

Pollock, Georgina Lauren. “Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis.” 2019. Doctoral Dissertation, University of Melbourne. Accessed February 25, 2021. http://hdl.handle.net/11343/227758.

MLA Handbook (7th Edition):

Pollock, Georgina Lauren. “Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis.” 2019. Web. 25 Feb 2021.

Vancouver:

Pollock GL. Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis. [Internet] [Doctoral dissertation]. University of Melbourne; 2019. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/11343/227758.

Council of Science Editors:

Pollock GL. Investigating the molecular mechanisms of enteropathogenic Escherichia coli pathogenesis. [Doctoral Dissertation]. University of Melbourne; 2019. Available from: http://hdl.handle.net/11343/227758

22. Cheema, Tasbir. Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line .

Degree: 2012, University of Ottawa

 Programmed cell death (apoptosis) is the most common mechanism of cell death in eukaryotes. The ability of cancer cells to evade and inhibit apoptosis has… (more)

Subjects/Keywords: Apoptosis; Smac; XIAP; X-Linked inhibitor of apoptosis protein; Caspase; Inhibitor of apoptosis proteins; BIR domain

apoptosis proteins (IAPs). X-Linked inhibitor of apoptosis protein (XIAP) is… …Figure 3.14 Triglycine linked divalent Smac mimetics show no ability to induce apoptosis at… …Programmed cell death (apoptosis) is the most common mechanism of cell death in… …eukaryotes. The ability of cancer cells to evade and inhibit apoptosis has become a hallmark… …feature of cancer. This is accomplished through a family of proteins known as the inhibitor of… 

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APA (6th Edition):

Cheema, T. (2012). Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheema, Tasbir. “Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line .” 2012. Thesis, University of Ottawa. Accessed February 25, 2021. http://hdl.handle.net/10393/20737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheema, Tasbir. “Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line .” 2012. Web. 25 Feb 2021.

Vancouver:

Cheema T. Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line . [Internet] [Thesis]. University of Ottawa; 2012. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10393/20737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheema T. Rational Design, Synthesis and Evaluation of Novel Second Mitochondrial-Derived Activators of Caspase (Smac) Mimetics That Induce Apoptosis in Human MDA-MB-231 Breast Cancer Cell Line . [Thesis]. University of Ottawa; 2012. Available from: http://hdl.handle.net/10393/20737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Francisco Washington AraÃjo Barros. Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro.

Degree: Master, 2010, Universidade Federal do Ceará

Acridinas sÃo molÃculas policÃclicas aromÃticas planares que possuem a capacidade de intercalar no DNA nuclear. Muitos dos seus representantes apresentam propriedades antibacterianas, antiparasitÃrias e antitumorais.… (more)

Subjects/Keywords: FARMACOLOGIA; Compostos AcridÃnicos; Citotoxicidade; Inibidor de Topoisomerase I; Dano ao DNA; Apoptose; Acridine Compounds; Cytotoxicity; Topoisomerase I inhibitor; DNA Damage; Apoptosis

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APA (6th Edition):

Barros, F. W. A. (2010). Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=4750 ;

Chicago Manual of Style (16th Edition):

Barros, Francisco Washington AraÃjo. “Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro.” 2010. Masters Thesis, Universidade Federal do Ceará. Accessed February 25, 2021. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=4750 ;.

MLA Handbook (7th Edition):

Barros, Francisco Washington AraÃjo. “Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro.” 2010. Web. 25 Feb 2021.

Vancouver:

Barros FWA. Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro. [Internet] [Masters thesis]. Universidade Federal do Ceará 2010. [cited 2021 Feb 25]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=4750 ;.

Council of Science Editors:

Barros FWA. Estudo do potencial anticÃncer de novos derivados acridÃnicos sintÃticos em modelos experimentais in vitro. [Masters Thesis]. Universidade Federal do Ceará 2010. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=4750 ;

24. Panagopoulou, Vasiliki. Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Effect of Angiotensin Converting Enzyme Inhibitors on In-Stent Restenosis andRelation to Plasma Apoptosis Signaling MoleculesTheory: Angiotensin-converting enzyme inhibitors have been reported to inhibit instentrestenosis. To… (more)

Subjects/Keywords: Αναστολείς μετατρεπτικού ενζύμου αγγειοτενσίνης; Απόπτωση; Επαναστένωση εντός της ενδοστεφανιαίας πρόσθεσης; Angiotensin converting enzyme inhibitor; Apoptosis; in- stent restenosis

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APA (6th Edition):

Panagopoulou, V. (2014). Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Panagopoulou, Vasiliki. “Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed February 25, 2021. http://hdl.handle.net/10442/hedi/42537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Panagopoulou, Vasiliki. “Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση.” 2014. Web. 25 Feb 2021.

Vancouver:

Panagopoulou V. Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10442/hedi/42537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Panagopoulou V. Επίδραση της αναστολής του μετατρεπτικού ενζύμου αγγειοτενσίνης σε δείκτες αποπτωτικής δραστηριότητας στη στεφανιαία κυκλοφορία και συσχέτισή τους με την επαναστένωση εντός στεφανιαίων ενδοπροσθέσεων, σε ασθενείς οι οποίοι έχουν υποβληθεί σε διαδερμική στεφανιαία παρέμβαση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/42537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

25. Ramharack, Pritika. HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549).

Degree: 2015, University of KwaZulu-Natal

Abstract available in PDF file. Advisors/Committee Members: Chuturgoon, Anil A. (advisor), Nagiah, Savania. (advisor).

Subjects/Keywords: Lung cancer.; Apoptosis and p53.; HMG-CoA reductase inhibitor.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ramharack, P. (2015). HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549). (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/14680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramharack, Pritika. “HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549).” 2015. Thesis, University of KwaZulu-Natal. Accessed February 25, 2021. http://hdl.handle.net/10413/14680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramharack, Pritika. “HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549).” 2015. Web. 25 Feb 2021.

Vancouver:

Ramharack P. HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549). [Internet] [Thesis]. University of KwaZulu-Natal; 2015. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10413/14680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramharack P. HMG-CoA reductase inhibitor, atorvastatin induces apoptasis in human lung adenocarcinoma cell (A549). [Thesis]. University of KwaZulu-Natal; 2015. Available from: http://hdl.handle.net/10413/14680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. 윤, 종호. Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder.

Degree: 2012, Ajou University

X-linked inhibitor of apoptosis protein (XIAP) is associated with tumor genesis, growth, progression and metastasis, and acts by blocking caspase-mediated apoptosis. In the present study,… (more)

Subjects/Keywords: X-linked inhibitor of apoptosis protein; BRAFV600E mutation; papillary thyroid carcinoma; thyroid neoplasms; lymph nodes; metastasis; recurrence

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APA (6th Edition):

윤, . (2012). Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/7574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012163

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

윤, 종호. “Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder.” 2012. Thesis, Ajou University. Accessed February 25, 2021. http://repository.ajou.ac.kr/handle/201003/7574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012163.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

윤, 종호. “Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder.” 2012. Web. 25 Feb 2021.

Vancouver:

윤 . Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder. [Internet] [Thesis]. Ajou University; 2012. [cited 2021 Feb 25]. Available from: http://repository.ajou.ac.kr/handle/201003/7574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012163.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

윤 . Expression of X-linked Inhibitor of Apoptosis Protein in Neoplastic Thyroid Disorder. [Thesis]. Ajou University; 2012. Available from: http://repository.ajou.ac.kr/handle/201003/7574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012163

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Lau, Rosanna. cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function .

Degree: 2012, University of Ottawa

 The cellular inhibitor of apoptosis protein (cIAP)-2 plays an important role in the protection against apoptosis by inhibiting the endogenous IAP inhibitor Smac, thus allowing… (more)

Subjects/Keywords: Inhibitor of apoptosis; Cancer; Cell signaling; p53

…pathways ........................................................ 3 1.2 Inhibitor of apoptosis… …protein cIAP1 cellular inhibitor of apoptosis protein 1 cIAP2 cellular inhibitor of… …heat shock protein IAP inhibitor of apoptosis protein ID4 inhibitor of DNA binding 4… …phosphatase 1 XIAP X-chromosome-linked inhibitor of apoptosis protein ACKNOWLEDGEMENTS First and… …describe a few pathways governing apoptosis that are commonly deregulated in cancer. Inhibitor of… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lau, R. (2012). cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/22845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lau, Rosanna. “cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function .” 2012. Thesis, University of Ottawa. Accessed February 25, 2021. http://hdl.handle.net/10393/22845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lau, Rosanna. “cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function .” 2012. Web. 25 Feb 2021.

Vancouver:

Lau R. cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function . [Internet] [Thesis]. University of Ottawa; 2012. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10393/22845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lau R. cIAP2 Negatively Regulates Proliferation and Tumourigenesis by Repressing IKK Activity and Maintaining p53 Function . [Thesis]. University of Ottawa; 2012. Available from: http://hdl.handle.net/10393/22845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

28. Toro, Edgardo J. Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor.

Degree: PhD, Medical Sciences - Molecular Cell Biology (IDP), 2012, University of Florida

 The fluoroquinolone antibiotic enoxacin was recently identified as an inhibitor of an interaction between the B2-subunit of vacuolar H+-ATPase (V-ATPase) and microfilaments, and of osteoclast… (more)

Subjects/Keywords: Actins; Adenosine triphosphatases; Apoptosis; Bone resorption; Bones; Cultured cells; Microfilaments; Orthodontics; Osteoclasts; Tooth movement; bisphosphonate  – bone  – inhibitor  – orthodontic  – osteoclast  – vatpase

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APA (6th Edition):

Toro, E. J. (2012). Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0044571

Chicago Manual of Style (16th Edition):

Toro, Edgardo J. “Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor.” 2012. Doctoral Dissertation, University of Florida. Accessed February 25, 2021. https://ufdc.ufl.edu/UFE0044571.

MLA Handbook (7th Edition):

Toro, Edgardo J. “Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor.” 2012. Web. 25 Feb 2021.

Vancouver:

Toro EJ. Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2021 Feb 25]. Available from: https://ufdc.ufl.edu/UFE0044571.

Council of Science Editors:

Toro EJ. Characterization of Enoxacin a Novel Vacuolar H+ ATPase-Directed Osteoclast Inhibitor. [Doctoral Dissertation]. University of Florida; 2012. Available from: https://ufdc.ufl.edu/UFE0044571


University of Georgia

29. Johnson, Melanie E. Survivin expression in canine and feline cancer.

Degree: 2014, University of Georgia

 Survivin is an inhibitor of apoptosis protein that is expressed during embryonic development and several human malignancies but is absent in most adult tissues. Its… (more)

Subjects/Keywords: apoptosis inhibitor; cancer; canine lymphosarcoma; canine mammary tumors; caspase-3; cell division; Ki-67; prognostic indicator; review; survivin

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APA (6th Edition):

Johnson, M. E. (2014). Survivin expression in canine and feline cancer. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/22411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson, Melanie E. “Survivin expression in canine and feline cancer.” 2014. Thesis, University of Georgia. Accessed February 25, 2021. http://hdl.handle.net/10724/22411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson, Melanie E. “Survivin expression in canine and feline cancer.” 2014. Web. 25 Feb 2021.

Vancouver:

Johnson ME. Survivin expression in canine and feline cancer. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10724/22411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson ME. Survivin expression in canine and feline cancer. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/22411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Brancato, Jennifer M. Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers.

Degree: PhD, Pharmacology, 2018, Case Western Reserve University School of Graduate Studies

 Inhibitors of the Bromodomain and Extraterminal (BET) family of epigenetic readers have been extensively studied in a wide range of cancers, and several are being… (more)

Subjects/Keywords: Pharmacology; Oncology; Biomedical Research; Breast cancer; BET protein; BET inhibitor; LIN9; Aurora kinase; apoptosis; senescence; mitotic catastrophe

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APA (6th Edition):

Brancato, J. M. (2018). Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1522842642716411

Chicago Manual of Style (16th Edition):

Brancato, Jennifer M. “Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers.” 2018. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed February 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1522842642716411.

MLA Handbook (7th Edition):

Brancato, Jennifer M. “Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers.” 2018. Web. 25 Feb 2021.

Vancouver:

Brancato JM. Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2018. [cited 2021 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1522842642716411.

Council of Science Editors:

Brancato JM. Defining the Mechanism of Action of Bromodomain and Extraterminal Inhibitors in Triple-Negative Breast Cancers. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1522842642716411

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