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You searched for subject:(anoikis). Showing records 1 – 30 of 35 total matches.

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Dalhousie University

1. Arsenault, Daniel. The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells.

Degree: MS, Department of Biochemistry & Molecular Biology, 2012, Dalhousie University

 Phosphatidylcholine (PC) is an essential component of biological membranes and is synthesized by the CDP-choline pathway under the control of the rate-limiting enzyme CTP:phosphocholine cytidylyltransferase-alpha… (more)

Subjects/Keywords: H-ras; Anoikis; Phosphatidylcholine; Cancer

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APA (6th Edition):

Arsenault, D. (2012). The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15710

Chicago Manual of Style (16th Edition):

Arsenault, Daniel. “The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells.” 2012. Masters Thesis, Dalhousie University. Accessed September 25, 2018. http://hdl.handle.net/10222/15710.

MLA Handbook (7th Edition):

Arsenault, Daniel. “The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells.” 2012. Web. 25 Sep 2018.

Vancouver:

Arsenault D. The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells. [Internet] [Masters thesis]. Dalhousie University; 2012. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/10222/15710.

Council of Science Editors:

Arsenault D. The role of the CDP-choline pathway in the anoikis resitance of Ras transformed intestinal epithelial cells. [Masters Thesis]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15710


University of Notre Dame

2. Cassandra Buchheit. Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells.

Degree: PhD, Biological Sciences, 2015, University of Notre Dame

  When mammary epithelial cells become detached from the extracellular matrix (ECM), they undergo programmed cell death. Over the past twenty years, our knowledge regarding… (more)

Subjects/Keywords: inflammatory breast cancer; necrosis; anoikis

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APA (6th Edition):

Buchheit, C. (2015). Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/wp988g87473

Chicago Manual of Style (16th Edition):

Buchheit, Cassandra. “Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells.” 2015. Doctoral Dissertation, University of Notre Dame. Accessed September 25, 2018. https://curate.nd.edu/show/wp988g87473.

MLA Handbook (7th Edition):

Buchheit, Cassandra. “Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells.” 2015. Web. 25 Sep 2018.

Vancouver:

Buchheit C. Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells. [Internet] [Doctoral dissertation]. University of Notre Dame; 2015. [cited 2018 Sep 25]. Available from: https://curate.nd.edu/show/wp988g87473.

Council of Science Editors:

Buchheit C. Elucidating cell death mechanisms in extracellular matrix-detached epithelial cells. [Doctoral Dissertation]. University of Notre Dame; 2015. Available from: https://curate.nd.edu/show/wp988g87473


West Virginia University

3. Kumar, Sanjeev, 1973-. Regulation of anoikis by ankyrin complexes.

Degree: 2010, West Virginia University

Anoikis is a subset of apoptosis, suppressed by cell-extracellular matrix association. It is a safeguard mechanism against tumor metastasis. The present study was designed to… (more)

Subjects/Keywords: Membrane proteins.; Cadherins.; Anoikis.; Ankyrins.; Cadherins.

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APA (6th Edition):

Kumar, Sanjeev, 1. (2010). Regulation of anoikis by ankyrin complexes. (Thesis). West Virginia University. Retrieved from http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=23542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kumar, Sanjeev, 1973-. “Regulation of anoikis by ankyrin complexes.” 2010. Thesis, West Virginia University. Accessed September 25, 2018. http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=23542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kumar, Sanjeev, 1973-. “Regulation of anoikis by ankyrin complexes.” 2010. Web. 25 Sep 2018.

Vancouver:

Kumar, Sanjeev 1. Regulation of anoikis by ankyrin complexes. [Internet] [Thesis]. West Virginia University; 2010. [cited 2018 Sep 25]. Available from: http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=23542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kumar, Sanjeev 1. Regulation of anoikis by ankyrin complexes. [Thesis]. West Virginia University; 2010. Available from: http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=23542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Silva, Enny Fernandes. Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico.

Degree: PhD, Bioquímica, 2003, University of São Paulo

Células epiteliais, endoteliais e fibroblastos podem ser induzidos a apoptose (anoikis) quando se destacam da matriz extracelular. O processo de anoikis tem a função de… (more)

Subjects/Keywords: Anoikis; Anoikis; Apoptose; Apoptosis; Nitric oxide; Óxido nítrico

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APA (6th Edition):

Silva, E. F. (2003). Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102008-092816/ ;

Chicago Manual of Style (16th Edition):

Silva, Enny Fernandes. “Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico.” 2003. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2018. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102008-092816/ ;.

MLA Handbook (7th Edition):

Silva, Enny Fernandes. “Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico.” 2003. Web. 25 Sep 2018.

Vancouver:

Silva EF. Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico. [Internet] [Doctoral dissertation]. University of São Paulo; 2003. [cited 2018 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102008-092816/ ;.

Council of Science Editors:

Silva EF. Indução de apoptose relacionada com perda de adesão (anoikis) em células A431 por óxido nítrico. [Doctoral Dissertation]. University of São Paulo; 2003. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102008-092816/ ;


Virginia Commonwealth University

5. Griffin, Brian P. The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer.

Degree: MS, Biochemistry, 2015, Virginia Commonwealth University

  In the presented study, we identified that SRSF3 controls the alternative splicing of CPEB2 and consequently promotes a metastatic phenotype in triple negative breast… (more)

Subjects/Keywords: proteomics; siRNA; anoikis; metastasis; Biochemistry; Cancer Biology; Molecular Biology

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APA (6th Edition):

Griffin, B. P. (2015). The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/3976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Griffin, Brian P. “The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer.” 2015. Thesis, Virginia Commonwealth University. Accessed September 25, 2018. https://scholarscompass.vcu.edu/etd/3976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Griffin, Brian P. “The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer.” 2015. Web. 25 Sep 2018.

Vancouver:

Griffin BP. The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer. [Internet] [Thesis]. Virginia Commonwealth University; 2015. [cited 2018 Sep 25]. Available from: https://scholarscompass.vcu.edu/etd/3976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Griffin BP. The Role of SRSF3 in Control of Alternative Splicing of CPEB2 in Triple Negative Breast Cancer. [Thesis]. Virginia Commonwealth University; 2015. Available from: https://scholarscompass.vcu.edu/etd/3976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rice University

6. Caneba, Christine. Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration.

Degree: 2013, Rice University

 Ovarian cancer remains the most lethal gynecological malignancy worldwide, with most of the disease detected at later stages. Elucidating pathways based on upregulation of proteins… (more)

Subjects/Keywords: Cancer; Metabolism; Migration; Anoikis; Nitric oxide; Invasion; Glycolysis; Oxidative phosphorylation

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APA (6th Edition):

Caneba, C. (2013). Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration. (Thesis). Rice University. Retrieved from http://hdl.handle.net/1911/76401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Caneba, Christine. “Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration.” 2013. Thesis, Rice University. Accessed September 25, 2018. http://hdl.handle.net/1911/76401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Caneba, Christine. “Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration.” 2013. Web. 25 Sep 2018.

Vancouver:

Caneba C. Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration. [Internet] [Thesis]. Rice University; 2013. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/1911/76401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Caneba C. Ovarian Cancer Metabolism: Effect of Anoikis Condition and Nitric Oxide on Ovarian Cancer Metabolism, and Effect of Metabolites on Ovarian Cancer Migration. [Thesis]. Rice University; 2013. Available from: http://hdl.handle.net/1911/76401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

7. Kamarajugadda, Sushamadevi. Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species.

Degree: Medical Sciences
Biochemistry and Molecular Biology (IDP), 2011, University of Florida

 Cancer cells commonly exhibit aberrant glucose metabolism characterized by a preference for aerobic glycolysis rather than mitochondrial oxidation. However, the significance of this phenomenon, known… (more)

Subjects/Keywords: anoikis  – metabolism  – metastasis; Biochemistry and Molecular Biology (IDP)

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APA (6th Edition):

Kamarajugadda, S. (2011). Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043595

Chicago Manual of Style (16th Edition):

Kamarajugadda, Sushamadevi. “Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species.” 2011. Doctoral Dissertation, University of Florida. Accessed September 25, 2018. http://ufdc.ufl.edu/UFE0043595.

MLA Handbook (7th Edition):

Kamarajugadda, Sushamadevi. “Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species.” 2011. Web. 25 Sep 2018.

Vancouver:

Kamarajugadda S. Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2018 Sep 25]. Available from: http://ufdc.ufl.edu/UFE0043595.

Council of Science Editors:

Kamarajugadda S. Acquisition of Anoikis Resistance by Attenuation of Mitochondrial Respiration and Reactive Oxygen Species. [Doctoral Dissertation]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043595


Universidade do Estado do Rio de Janeiro

8. Laila Ribeiro Fernandes. Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C.

Degree: Master, 2013, Universidade do Estado do Rio de Janeiro

 Recentemente, nosso grupo demonstrou que a matriz extracelular de astrocitomas promove a seleçãode células endoteliais altamente proliferativas, porém com reduzida capacidade tubulogênica, além de determinar… (more)

Subjects/Keywords: Angiogênese; Tenascina-C; Tubulogênese; Adesão; Anoikis; Glioma; Angiogenesis; Tenascin-C; Tubulogenesis; Adhesion; Anoikis; Glioma; MORFOLOGIA; Endotélio - Teses; Neovascularização - Teses; Tenascina; Células - Adesão - Teses; Matriz Extracelular - Teses

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APA (6th Edition):

Fernandes, L. R. (2013). Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7254 ;

Chicago Manual of Style (16th Edition):

Fernandes, Laila Ribeiro. “Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C.” 2013. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed September 25, 2018. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7254 ;.

MLA Handbook (7th Edition):

Fernandes, Laila Ribeiro. “Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C.” 2013. Web. 25 Sep 2018.

Vancouver:

Fernandes LR. Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2013. [cited 2018 Sep 25]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7254 ;.

Council of Science Editors:

Fernandes LR. Estudo da adesão, sobrevivência e tubulogênese endotelial em modelo de células de glioma silenciadas para a expressão de Tenascina-C. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2013. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7254 ;


University of South Florida

9. Woods, Nicholas Taylor. Regulation of bax activation and apoptosis by src and acetylated mutant p53.

Degree: 2008, University of South Florida

 Apoptosis is an inherent suicide mechanism that cells invoke for a variety of reasons including embryo cavitation, tissue homeostasis, excessive DNA damage and aberrant oncogene… (more)

Subjects/Keywords: Anoikis; Bif-1; Cancer; Ku70; Mcl-1; American Studies; Arts and Humanities

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APA (6th Edition):

Woods, N. T. (2008). Regulation of bax activation and apoptosis by src and acetylated mutant p53. (Thesis). University of South Florida. Retrieved from http://scholarcommons.usf.edu/etd/572

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woods, Nicholas Taylor. “Regulation of bax activation and apoptosis by src and acetylated mutant p53.” 2008. Thesis, University of South Florida. Accessed September 25, 2018. http://scholarcommons.usf.edu/etd/572.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woods, Nicholas Taylor. “Regulation of bax activation and apoptosis by src and acetylated mutant p53.” 2008. Web. 25 Sep 2018.

Vancouver:

Woods NT. Regulation of bax activation and apoptosis by src and acetylated mutant p53. [Internet] [Thesis]. University of South Florida; 2008. [cited 2018 Sep 25]. Available from: http://scholarcommons.usf.edu/etd/572.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woods NT. Regulation of bax activation and apoptosis by src and acetylated mutant p53. [Thesis]. University of South Florida; 2008. Available from: http://scholarcommons.usf.edu/etd/572

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Mariana Toricelli Pinto. Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma.

Degree: 2010, Universidade Federal de São Paulo

Although malignant melanoma is the less frequently diagnosed skin cancer, it shows a poor prognosis due its chemoresistance and metastasis development. One of the adquired… (more)

Subjects/Keywords: Timp1; β; 1 integrinas; tetraspaninas; transformação melanócitos; Anoikis; N-glicosilação aberrante de tumores; FARMACOLOGIA GERAL

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APA (6th Edition):

Pinto, M. T. (2010). Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pinto, Mariana Toricelli. “Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma.” 2010. Thesis, Universidade Federal de São Paulo. Accessed September 25, 2018. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pinto, Mariana Toricelli. “Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma.” 2010. Web. 25 Sep 2018.

Vancouver:

Pinto MT. Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma. [Internet] [Thesis]. Universidade Federal de São Paulo; 2010. [cited 2018 Sep 25]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pinto MT. Associação entre Timp1, β1-integrinas e CD63 ao longo da gênese do melanoma. [Thesis]. Universidade Federal de São Paulo; 2010. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Vrazeli, Paraskevi. HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων.

Degree: 2016, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

 The heat shock protein Hsp70 (HSPA1A: heat shock protein family A (Hsp70) member 1A) is the most thoroughly studied and analyzed protein in the A-family… (more)

Subjects/Keywords: Hsp70; MCF; P53; EMT; Ανέστια; Μετανάστευση; HSP70; MCF; p53; EMT; Anoikis; Metastasis

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APA (6th Edition):

Vrazeli, P. (2016). HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/38540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vrazeli, Paraskevi. “HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων.” 2016. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed September 25, 2018. http://hdl.handle.net/10442/hedi/38540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vrazeli, Paraskevi. “HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων.” 2016. Web. 25 Sep 2018.

Vancouver:

Vrazeli P. HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2016. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/10442/hedi/38540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vrazeli P. HSP70: μηχανισμός ρύθμισης της απόπτωσης και μετανάστευσης των MCF7 καρκινικών κυττάρων. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2016. Available from: http://hdl.handle.net/10442/hedi/38540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Pedanou, Victoria E. Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens.

Degree: Cancer Biology, Molecular, Cell, and Cancer Biology, 2016, U of Massachusetts : Med

  Epithelial cells that lose attachment to the extracellular matrix (ECM) undergo a specialized form of apoptosis called anoikis. Anoikis has an important role in… (more)

Subjects/Keywords: cancer biology; breast cancer; anoikis; histone demethylase; jmjd1a; kdm3a; Biology; Cancer Biology

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APA (6th Edition):

Pedanou, V. E. (2016). Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/889

Chicago Manual of Style (16th Edition):

Pedanou, Victoria E. “Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed September 25, 2018. http://escholarship.umassmed.edu/gsbs_diss/889.

MLA Handbook (7th Edition):

Pedanou, Victoria E. “Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens.” 2016. Web. 25 Sep 2018.

Vancouver:

Pedanou VE. Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2018 Sep 25]. Available from: http://escholarship.umassmed.edu/gsbs_diss/889.

Council of Science Editors:

Pedanou VE. Identification of Novel Pathways that Promote Anoikis through Genome-wide Screens. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/889


Université de Sherbrooke

13. Beauséjour, Marco. Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines .

Degree: 2017, Université de Sherbrooke

 La phosphatidylinositol-3 kinase (PI3-K) est un complexe comportant une sous-unité catalytique (C) et régulatrice (R). En ce qui concerne la classe I, cinq isoformes R… (more)

Subjects/Keywords: PI3-K; Isoformes; Cancer colorectal; Anoïkose; Survie cellulaire; Anoikis; Cell survival; Isoforms; Colorectal cancer

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APA (6th Edition):

Beauséjour, M. (2017). Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines . (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/11565

Chicago Manual of Style (16th Edition):

Beauséjour, Marco. “Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines .” 2017. Doctoral Dissertation, Université de Sherbrooke. Accessed September 25, 2018. http://hdl.handle.net/11143/11565.

MLA Handbook (7th Edition):

Beauséjour, Marco. “Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines .” 2017. Web. 25 Sep 2018.

Vancouver:

Beauséjour M. Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines . [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2017. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/11143/11565.

Council of Science Editors:

Beauséjour M. Rôles des complexes isoformes de la PI3-K dans la survie et la résistance à l’anoïkose chez les cellules carcinomateuses colorectales humaines . [Doctoral Dissertation]. Université de Sherbrooke; 2017. Available from: http://hdl.handle.net/11143/11565

14. 佐藤, 正法. Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害.

Degree: 博士(医学), 2014, Hokkaido University / 北海道大学

The transcription factor nuclear factor-κB (NF-κB) plays a crucial role in pancreatic cancer (PC) progression. NF-κB is also involved in resistance to anoikis, a special… (more)

Subjects/Keywords: Anoikis; Dehydroxymethylepoxyquinomicin (DHMEQ); Nuclear factor-κB (NF-κB); Pancreatic cancer (PC); Peritoneal metastasis

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APA (6th Edition):

佐藤, . (2014). Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害. (Thesis). Hokkaido University / 北海道大学. Retrieved from http://hdl.handle.net/2115/57776 ; http://dx.doi.org/10.14943/doctoral.k11596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐藤, 正法. “Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害.” 2014. Thesis, Hokkaido University / 北海道大学. Accessed September 25, 2018. http://hdl.handle.net/2115/57776 ; http://dx.doi.org/10.14943/doctoral.k11596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐藤, 正法. “Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害.” 2014. Web. 25 Sep 2018.

Vancouver:

佐藤 . Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害. [Internet] [Thesis]. Hokkaido University / 北海道大学; 2014. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2115/57776 ; http://dx.doi.org/10.14943/doctoral.k11596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

佐藤 . Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ : NF-κB阻害剤 (-)-DHMEQによる膵癌細胞のアノイキス誘導及び腹膜転移阻害. [Thesis]. Hokkaido University / 北海道大学; 2014. Available from: http://hdl.handle.net/2115/57776 ; http://dx.doi.org/10.14943/doctoral.k11596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Simpson, Craig Darryl. Determination of Molecular Regulators of Anoikis Resistance.

Degree: 2012, University of Toronto

As a barrier to metastases, cells normally undergo apoptosis after they lose contact with their extra cellular matrix or their neighbouring cells. This cell death… (more)

Subjects/Keywords: Anoikis; Chemical Biology; Metastasis; Screening; 0379

…4.4.2 ABHD4 knockdown confers resistance to anoikis… …93 4.4.3 Mechanism of anoikis resistance after ABHD4 knockdown… …97 4.4.4 ABHD4 knockdown imparts anoikis resistance through… …107 5.1 Chemical biology approach to identify novel anoikis… …5.2 Genetic approach to determine anoikis regulators… 

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APA (6th Edition):

Simpson, C. D. (2012). Determination of Molecular Regulators of Anoikis Resistance. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34925

Chicago Manual of Style (16th Edition):

Simpson, Craig Darryl. “Determination of Molecular Regulators of Anoikis Resistance.” 2012. Doctoral Dissertation, University of Toronto. Accessed September 25, 2018. http://hdl.handle.net/1807/34925.

MLA Handbook (7th Edition):

Simpson, Craig Darryl. “Determination of Molecular Regulators of Anoikis Resistance.” 2012. Web. 25 Sep 2018.

Vancouver:

Simpson CD. Determination of Molecular Regulators of Anoikis Resistance. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/1807/34925.

Council of Science Editors:

Simpson CD. Determination of Molecular Regulators of Anoikis Resistance. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34925


Kyoto University

16. FRISCO, HEIDIE LAYA. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation.

Degree: 2015, Kyoto University

Subjects/Keywords: chemical biology; adhesamine; anoikis; cell transplantation; self-assembly; peptides

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APA (6th Edition):

FRISCO, H. L. (2015). Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/199211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

FRISCO, HEIDIE LAYA. “Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. ” 2015. Thesis, Kyoto University. Accessed September 25, 2018. http://hdl.handle.net/2433/199211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

FRISCO, HEIDIE LAYA. “Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. ” 2015. Web. 25 Sep 2018.

Vancouver:

FRISCO HL. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. [Internet] [Thesis]. Kyoto University; 2015. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/199211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

FRISCO HL. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/199211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

17. FRISCO, HEIDIE LAYA. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用.

Degree: 博士(医科学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第18902号

医科博第58号

Subjects/Keywords: chemical biology; adhesamine; anoikis; cell transplantation; self-assembly; peptides

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APA (6th Edition):

FRISCO, H. L. (2015). Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/199211 ; http://dx.doi.org/10.14989/doctor.k18902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

FRISCO, HEIDIE LAYA. “Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用.” 2015. Thesis, Kyoto University / 京都大学. Accessed September 25, 2018. http://hdl.handle.net/2433/199211 ; http://dx.doi.org/10.14989/doctor.k18902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

FRISCO, HEIDIE LAYA. “Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用.” 2015. Web. 25 Sep 2018.

Vancouver:

FRISCO HL. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/199211 ; http://dx.doi.org/10.14989/doctor.k18902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

FRISCO HL. Synthetic Molecules that Protect Cells from Anoikis and Their Use in Cell Transplantation. : アノイキスから細胞を保護する合成分子と細胞移植での利用. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/199211 ; http://dx.doi.org/10.14989/doctor.k18902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

18. Alhazzazi, Turki Yousef. Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer.

Degree: PhD, Oral Health Sciences, 2012, University of Michigan

 Head and neck cancer is the eighth most common cancer worldwide, and squamous cell carcinoma represents the majority of the head and neck cancer cases.… (more)

Subjects/Keywords: Head and Neck Cancer; Oral Cancer; Sirtuins; SIRT3; Sirtuin-3; Anoikis; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Alhazzazi, T. Y. (2012). Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91596

Chicago Manual of Style (16th Edition):

Alhazzazi, Turki Yousef. “Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer.” 2012. Doctoral Dissertation, University of Michigan. Accessed September 25, 2018. http://hdl.handle.net/2027.42/91596.

MLA Handbook (7th Edition):

Alhazzazi, Turki Yousef. “Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer.” 2012. Web. 25 Sep 2018.

Vancouver:

Alhazzazi TY. Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2027.42/91596.

Council of Science Editors:

Alhazzazi TY. Sirtuin-3 (SIRT3), A Novel Potential Therapeutic Target for Head and Neck Cancer. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91596


Indian Institute of Science

19. Sundararaman, Ananthalakshmy. Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation.

Degree: 2016, Indian Institute of Science

 AMP-activated protein kinase (AMPK) is a key regulator of energy homeostasis in cells. It has been implicated as a therapeutic target for various metabolic diseases… (more)

Subjects/Keywords: AMP Activated Protein Kinase; AMPK Matrix Deprivation; Anoikis-Detachment Induced Apoptosis; Anchorage-Deprivation; Reactive Oxygen Species (ROS); Calcium Signalling; Mechanotransduction; Cell Adhesion Complexes; Calcium-Oxidant Signaling Network; Upstream Kinases; Apoptosis; Anoikis; Integrin Signaling; Molecular Biology

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APA (6th Edition):

Sundararaman, A. (2016). Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation. (Thesis). Indian Institute of Science. Retrieved from http://etd.iisc.ernet.in/handle/2005/2976 ; http://etd.ncsi.iisc.ernet.in/abstracts/3838/G28251-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sundararaman, Ananthalakshmy. “Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation.” 2016. Thesis, Indian Institute of Science. Accessed September 25, 2018. http://etd.iisc.ernet.in/handle/2005/2976 ; http://etd.ncsi.iisc.ernet.in/abstracts/3838/G28251-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sundararaman, Ananthalakshmy. “Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation.” 2016. Web. 25 Sep 2018.

Vancouver:

Sundararaman A. Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation. [Internet] [Thesis]. Indian Institute of Science; 2016. [cited 2018 Sep 25]. Available from: http://etd.iisc.ernet.in/handle/2005/2976 ; http://etd.ncsi.iisc.ernet.in/abstracts/3838/G28251-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sundararaman A. Deciphering the Mechanisms of AMPK Activation upon Anchorage- Deprivation. [Thesis]. Indian Institute of Science; 2016. Available from: http://etd.iisc.ernet.in/handle/2005/2976 ; http://etd.ncsi.iisc.ernet.in/abstracts/3838/G28251-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Sweetwyne, Mariya T. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.

Degree: PhD, 2009, University of Alabama – Birmingham

Thrombospondin-1 (TSP1) is a multifunctional matricellular protein released by platelets in response to injury and secreted by cells under stress. TSP1 is cleaved into functional… (more)

Subjects/Keywords: Anoikis  – physiology<; br>; Calreticulin  – metabolism<; br>; Fibroplasts  – metabolism<; br>; Mice, Knockout<; br>; Receptors, LDL  – agonists<; br>; Thrombospondin 1  – pharmacology

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APA (6th Edition):

Sweetwyne, M. T. (2009). Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,747

Chicago Manual of Style (16th Edition):

Sweetwyne, Mariya T. “Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 25, 2018. http://contentdm.mhsl.uab.edu/u?/etd,747.

MLA Handbook (7th Edition):

Sweetwyne, Mariya T. “Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.” 2009. Web. 25 Sep 2018.

Vancouver:

Sweetwyne MT. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2018 Sep 25]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,747.

Council of Science Editors:

Sweetwyne MT. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,747

21. Yamanoi, Kouji. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる.

Degree: 博士(医学), 2017, Kyoto University / 京都大学

新制・課程博士

甲第20662号

医博第4272号

Subjects/Keywords: functional genomics screen; shRNA library; Anoikis resistance; High grade serous ovarian cancer

Page 1 Page 2

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APA (6th Edition):

Yamanoi, K. (2017). Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/227585 ; http://dx.doi.org/10.14989/doctor.k20662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamanoi, Kouji. “Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる.” 2017. Thesis, Kyoto University / 京都大学. Accessed September 25, 2018. http://hdl.handle.net/2433/227585 ; http://dx.doi.org/10.14989/doctor.k20662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamanoi, Kouji. “Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる.” 2017. Web. 25 Sep 2018.

Vancouver:

Yamanoi K. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる. [Internet] [Thesis]. Kyoto University / 京都大学; 2017. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/227585 ; http://dx.doi.org/10.14989/doctor.k20662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamanoi K. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. : 機能的ゲノミクススクリーンにより同定した因子ABHD2の発現低下は、卵巣癌のアノイキス抵抗性、化学療法抵抗性をもたらし、予後不良につながる. [Thesis]. Kyoto University / 京都大学; 2017. Available from: http://hdl.handle.net/2433/227585 ; http://dx.doi.org/10.14989/doctor.k20662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

22. Lee, Yishan. Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis .

Degree: 2008, Duke University

  Reversible acetylation has emerged as an important post-translational modification that rivals phosphorylation in regulating chromatin structure and gene expression. Acetylation of histone is associated… (more)

Subjects/Keywords: Biology, Molecular; Biology, Physiology; Biology, Cell; HDAC; deacetylation; transformation; anoikis

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APA (6th Edition):

Lee, Y. (2008). Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Yishan. “Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis .” 2008. Thesis, Duke University. Accessed September 25, 2018. http://hdl.handle.net/10161/654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Yishan. “Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis .” 2008. Web. 25 Sep 2018.

Vancouver:

Lee Y. Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis . [Internet] [Thesis]. Duke University; 2008. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/10161/654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee Y. Roles of Cytoplasmic Deacetylase Hdac6 in Oncogenic Tumorigenesis . [Thesis]. Duke University; 2008. Available from: http://hdl.handle.net/10161/654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

23. Maamer - Azzabi, Aida. Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737.

Degree: Docteur es, Biologie, Médecine et Santé, 2013, Université Paris-Sud – Paris XI

La progression tumorale est la conséquence de multiples altérations génotypiques et phénotypiques; L’une d’entre elles, nécessaire à la formation de métastases, est l’acquisition d’une résistance… (more)

Subjects/Keywords: Anoïkis; Cancer du côlon; Les protéines de la famille Bcl-2; Métastases; ABT-737; Anoikis; Colon cancer; Bcl-2 family; Metastasis; ABT-737

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APA (6th Edition):

Maamer - Azzabi, A. (2013). Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T054

Chicago Manual of Style (16th Edition):

Maamer - Azzabi, Aida. “Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 25, 2018. http://www.theses.fr/2013PA11T054.

MLA Handbook (7th Edition):

Maamer - Azzabi, Aida. “Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737.” 2013. Web. 25 Sep 2018.

Vancouver:

Maamer - Azzabi A. Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2018 Sep 25]. Available from: http://www.theses.fr/2013PA11T054.

Council of Science Editors:

Maamer - Azzabi A. Mécanismes moléculaires de l'acquisition d'une sensibilité à l'apoptose induite par l'ABT-737 et d'une résistance à l'anoïkis de cellules coliques métastatiques : Metastatic SW620 colon cancer cells are primed for death when detached and can be sensitized to anoikis by the BH3-mimetic ABT-737. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T054


Kyoto University

24. Yamanoi, Kouji. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer.

Degree: 2017, Kyoto University

Subjects/Keywords: functional genomics screen; shRNA library; Anoikis resistance; High grade serous ovarian cancer

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APA (6th Edition):

Yamanoi, K. (2017). Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/227585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamanoi, Kouji. “Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. ” 2017. Thesis, Kyoto University. Accessed September 25, 2018. http://hdl.handle.net/2433/227585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamanoi, Kouji. “Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. ” 2017. Web. 25 Sep 2018.

Vancouver:

Yamanoi K. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. [Internet] [Thesis]. Kyoto University; 2017. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/227585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamanoi K. Suppression of ABHD2, identified through a functional genomics screen, causes anoikis resistance, chemoresistance and poor prognosis in ovarian cancer. [Thesis]. Kyoto University; 2017. Available from: http://hdl.handle.net/2433/227585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

25. Takahashi, Ryo. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers.

Degree: 2014, Kyoto University

Subjects/Keywords: AFAP1L1; Colorectal cancer; vinculin; cell shape and mortility; invadopodia; anoikis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Takahashi, R. (2014). AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/189659

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takahashi, Ryo. “AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. ” 2014. Thesis, Kyoto University. Accessed September 25, 2018. http://hdl.handle.net/2433/189659.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takahashi, Ryo. “AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. ” 2014. Web. 25 Sep 2018.

Vancouver:

Takahashi R. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. [Internet] [Thesis]. Kyoto University; 2014. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/189659.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takahashi R. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. [Thesis]. Kyoto University; 2014. Available from: http://hdl.handle.net/2433/189659

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

26. Muñoz, Nina Margarita. Contribution of transforming growth factor-β signaling to intestinal cancer development.

Degree: PhD, Cancer Biology, 2006, Vanderbilt University

 Transforming Growth Factor-β (TGF-β) is a cytokine involved in the regulation of multiple cellular responses, and it is accepted that the TGF-β signaling pathway is… (more)

Subjects/Keywords: Transforming Growth Factor-beta; Anoikis; Colon cancer; Signaling network deregulation

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APA (6th Edition):

Muñoz, N. M. (2006). Contribution of transforming growth factor-β signaling to intestinal cancer development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11292006-125524/ ;

Chicago Manual of Style (16th Edition):

Muñoz, Nina Margarita. “Contribution of transforming growth factor-β signaling to intestinal cancer development.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed September 25, 2018. http://etd.library.vanderbilt.edu/available/etd-11292006-125524/ ;.

MLA Handbook (7th Edition):

Muñoz, Nina Margarita. “Contribution of transforming growth factor-β signaling to intestinal cancer development.” 2006. Web. 25 Sep 2018.

Vancouver:

Muñoz NM. Contribution of transforming growth factor-β signaling to intestinal cancer development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2018 Sep 25]. Available from: http://etd.library.vanderbilt.edu/available/etd-11292006-125524/ ;.

Council of Science Editors:

Muñoz NM. Contribution of transforming growth factor-β signaling to intestinal cancer development. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://etd.library.vanderbilt.edu/available/etd-11292006-125524/ ;


Kyoto University / 京都大学

27. Takahashi, Ryo. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

新制・課程博士

甲第18502号

医博第3922号

Subjects/Keywords: AFAP1L1; Colorectal cancer; vinculin; cell shape and mortility; invadopodia; anoikis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Takahashi, R. (2014). AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/189659 ; http://dx.doi.org/10.14989/doctor.k18502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takahashi, Ryo. “AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する.” 2014. Thesis, Kyoto University / 京都大学. Accessed September 25, 2018. http://hdl.handle.net/2433/189659 ; http://dx.doi.org/10.14989/doctor.k18502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takahashi, Ryo. “AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する.” 2014. Web. 25 Sep 2018.

Vancouver:

Takahashi R. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2433/189659 ; http://dx.doi.org/10.14989/doctor.k18502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takahashi R. AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers. : ビンキュリンの新規相互作用因子 AFAP1L1は細胞形態及び遊走能を変化させ、大腸癌進展を促進する. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/189659 ; http://dx.doi.org/10.14989/doctor.k18502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

28. Widau, Ryan Cole. PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS.

Degree: 2010, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Modulation of sphingolipid-induced apoptosis is a potential mechanism to enhance the effectiveness of chemotherapeutic drugs. Ceramide is a pleiotropic, sphingolipid… (more)

Subjects/Keywords: SPHINGOLIPID; ADHESION; CERAMIDE; APOPTOSIS; ANOIKIS; PP2A; PROTEIN PHOSPHATASE 2A; DAPK; DEATH ASSOCIATED PROTEIN KINASE; Apoptosis; Ceramides; Sphingolipids; Phosphoprotein phosphatases; Protein kinases; Chemotherapy  – Effectiveness

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APA (6th Edition):

Widau, R. C. (2010). PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Widau, Ryan Cole. “PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS.” 2010. Thesis, IUPUI. Accessed September 25, 2018. http://hdl.handle.net/1805/2131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Widau, Ryan Cole. “PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS.” 2010. Web. 25 Sep 2018.

Vancouver:

Widau RC. PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS. [Internet] [Thesis]. IUPUI; 2010. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/1805/2131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Widau RC. PROTEIN PHOSPHATASE 2A (PP2A) HOLOENZYMES REGULATE DEATH ASSOCIATED PROTEIN KINASE (DAPK) IN CERAMIDE-INDUCED ANOIKIS. [Thesis]. IUPUI; 2010. Available from: http://hdl.handle.net/1805/2131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Spaeth-Cook, Douglas M, Jr. Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer.

Degree: MS, Public Health, 2017, The Ohio State University

 Epithelial ovarian cancer (EOC) is most commonly diagnosed at advanced stages, resulting in poor prognoses. Unlike most cancers, EOC does not typically spread hematogenously or… (more)

Subjects/Keywords: Bioinformatics; Public Health; Oncology; Ovarian cancer; anoikis; live cell imaging; peritoneal cancer; TXNIP; Thioredoxin-Interacting Protein

…further. We hypothesized that these outlying genes would play a modulatory role in anoikis… …methods to identify potential drivers of anoikis-resistance in ovarian cancer, 3) develop… …upon detachment, would immediately undergo a form of pre-programmed cell death termed anoikis… …KD cell line exhibiting greater resistance to anoikis. This was concluded based on the… …methods to identify TXNIP as a potential driver of anoikis-resistance in ovarian cancer… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Spaeth-Cook, Douglas M, J. (2017). Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1494314758133333

Chicago Manual of Style (16th Edition):

Spaeth-Cook, Douglas M, Jr. “Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer.” 2017. Masters Thesis, The Ohio State University. Accessed September 25, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1494314758133333.

MLA Handbook (7th Edition):

Spaeth-Cook, Douglas M, Jr. “Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer.” 2017. Web. 25 Sep 2018.

Vancouver:

Spaeth-Cook, Douglas M J. Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer. [Internet] [Masters thesis]. The Ohio State University; 2017. [cited 2018 Sep 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1494314758133333.

Council of Science Editors:

Spaeth-Cook, Douglas M J. Identification of Thioredoxin-Interacting Protein as a Potential Mediator of Anoikis-Resistance in Ovarian Cancer. [Masters Thesis]. The Ohio State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1494314758133333

30. 佐藤, 正法. Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ.

Degree: 博士(医学), 医学, 2014, Hokkaido University

 The transcription factor nuclear factor-κB (NF-κB) plays a crucial role in pancreatic cancer (PC) progression. NF-κB is also involved in resistance to anoikis, a special… (more)

Subjects/Keywords: Anoikis; Dehydroxymethylepoxyquinomicin (DHMEQ); Nuclear factor-κB (NF-κB); Pancreatic cancer (PC); Peritoneal metastasis

…338 sato ET AL. Anoikis Induction in PC Cells by (−)-DHMEQ Although (… …both. Cyclin D1, also reported to be involved in anoikis resistance (28), was… …XL and Bax was not affected by either suspension culture or (−)-DHMEQ. Anoikis… …These results suggest that AsPC-1Gluc and MPanc96 cells were anoikis resistant, whereas BxPC-3… …and HPAC cells were anoikis sensitive (Fig. 5A). Furthermore, the anoikis… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

佐藤, . (2014). Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ. (Doctoral Dissertation). Hokkaido University. Retrieved from http://hdl.handle.net/2115/57776

Chicago Manual of Style (16th Edition):

佐藤, 正法. “Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ.” 2014. Doctoral Dissertation, Hokkaido University. Accessed September 25, 2018. http://hdl.handle.net/2115/57776.

MLA Handbook (7th Edition):

佐藤, 正法. “Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ.” 2014. Web. 25 Sep 2018.

Vancouver:

佐藤 . Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ. [Internet] [Doctoral dissertation]. Hokkaido University; 2014. [cited 2018 Sep 25]. Available from: http://hdl.handle.net/2115/57776.

Council of Science Editors:

佐藤 . Anoikis Induction and Inhibition of Peritoneal Metastasis of Pancreatic Cancer Cells by a Nuclear Factor-κB inhibitor, (-)-DHMEQ. [Doctoral Dissertation]. Hokkaido University; 2014. Available from: http://hdl.handle.net/2115/57776

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