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You searched for subject:(all trans retinoic acid). Showing records 1 – 30 of 15711 total matches.

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1. Dovijarski, Nemanja. The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells.

Degree: MSc -MS, Kinesiology & Health Science, 2019, York University

 Mitochondrial biogenesis is a process by which the cell increases its mitochondrial content, involving nuclear-derived and mitochondrial -derived protein synthesis and assembly. Previous literature has… (more)

Subjects/Keywords: Physiology; Retinoic Acid; Muscle Cells; Muscle Physiology; Nicotinamide Riboside; 9-cis retinoic acid; All-trans retinoic acid; Mitochondria; Mitochondrial biogenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dovijarski, N. (2019). The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells. (Masters Thesis). York University. Retrieved from https://yorkspace.library.yorku.ca/xmlui/handle/10315/36654

Chicago Manual of Style (16th Edition):

Dovijarski, Nemanja. “The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells.” 2019. Masters Thesis, York University. Accessed September 20, 2020. https://yorkspace.library.yorku.ca/xmlui/handle/10315/36654.

MLA Handbook (7th Edition):

Dovijarski, Nemanja. “The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells.” 2019. Web. 20 Sep 2020.

Vancouver:

Dovijarski N. The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells. [Internet] [Masters thesis]. York University; 2019. [cited 2020 Sep 20]. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/36654.

Council of Science Editors:

Dovijarski N. The Effects of Retinoic Acid, Nicotinamide Riboside and Chronic Contractile Activity on Mitochondrial Biogenesis in Skeletal Muscle Cells. [Masters Thesis]. York University; 2019. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/36654


Cornell University

2. Congleton, Johanna. Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells.

Degree: PhD, Environmental Toxicology, 2012, Cornell University

All-trans retinoic acid (ATRA) is successful in treating acute promyelocytic leukemia but has shown limited success with other cancers. Identifying signaling pathways that confer ATRA… (more)

Subjects/Keywords: All-trans retinoic acid; cd38; Src family kinases

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APA (6th Edition):

Congleton, J. (2012). Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29336

Chicago Manual of Style (16th Edition):

Congleton, Johanna. “Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells.” 2012. Doctoral Dissertation, Cornell University. Accessed September 20, 2020. http://hdl.handle.net/1813/29336.

MLA Handbook (7th Edition):

Congleton, Johanna. “Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells.” 2012. Web. 20 Sep 2020.

Vancouver:

Congleton J. Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1813/29336.

Council of Science Editors:

Congleton J. Signal Regulation By Cd38, Lyn, And Mapk-Associated Molecules During All-Trans Retinoic Acid-Induced Differentiation Of Acute Myelogenous Leukemia Cells. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29336


University of Maryland

3. Perry, Trinity Lynn. ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION.

Degree: Cell Biology & Molecular Genetics, 2011, University of Maryland

 Ascaris suum is known to cause ascariasis of both pigs and humans. Ascariasis is associated with a reduced control of intracellular pathogens and decreased vaccine… (more)

Subjects/Keywords: Immunology; All-trans Retinoic Acid; Ascaris suum; Macrophage; Porcine

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APA (6th Edition):

Perry, T. L. (2011). ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION. (Thesis). University of Maryland. Retrieved from http://hdl.handle.net/1903/12343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perry, Trinity Lynn. “ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION.” 2011. Thesis, University of Maryland. Accessed September 20, 2020. http://hdl.handle.net/1903/12343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perry, Trinity Lynn. “ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION.” 2011. Web. 20 Sep 2020.

Vancouver:

Perry TL. ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION. [Internet] [Thesis]. University of Maryland; 2011. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1903/12343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perry TL. ALTERED PORCINE ALVEOLAR MACROPHAGE PHENOTYPE BY ALL-TRANS RETINOIC ACID AND ASCARIS SUUM INFECTION. [Thesis]. University of Maryland; 2011. Available from: http://hdl.handle.net/1903/12343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

4. Maciel, Bruna Leal Lima. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .

Degree: 2013, Universidade do Rio Grande do Norte

 Nutritional status is an important determinant to the response against Leishmania infection, although few studies have characterized the molecular basis for the association found between… (more)

Subjects/Keywords: Leishmaniose visceral. Ácido all-trans retinóico. IL-10; Visceral leishmaniasis. All-trans retinoic acid. IL-10

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APA (6th Edition):

Maciel, B. L. L. (2013). Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . (Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/12575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Thesis, Universidade do Rio Grande do Norte. Accessed September 20, 2020. http://repositorio.ufrn.br/handle/123456789/12575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Web. 20 Sep 2020.

Vancouver:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Internet] [Thesis]. Universidade do Rio Grande do Norte; 2013. [cited 2020 Sep 20]. Available from: http://repositorio.ufrn.br/handle/123456789/12575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Thesis]. Universidade do Rio Grande do Norte; 2013. Available from: http://repositorio.ufrn.br/handle/123456789/12575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

5. Maciel, Bruna Leal Lima. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .

Degree: 2013, Universidade do Rio Grande do Norte

 Nutritional status is an important determinant to the response against Leishmania infection, although few studies have characterized the molecular basis for the association found between… (more)

Subjects/Keywords: Leishmaniose visceral. Ácido all-trans retinóico. IL-10; Visceral leishmaniasis. All-trans retinoic acid. IL-10

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APA (6th Edition):

Maciel, B. L. L. (2013). Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . (Doctoral Dissertation). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/12575

Chicago Manual of Style (16th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Doctoral Dissertation, Universidade do Rio Grande do Norte. Accessed September 20, 2020. http://repositorio.ufrn.br/handle/123456789/12575.

MLA Handbook (7th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Web. 20 Sep 2020.

Vancouver:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Internet] [Doctoral dissertation]. Universidade do Rio Grande do Norte; 2013. [cited 2020 Sep 20]. Available from: http://repositorio.ufrn.br/handle/123456789/12575.

Council of Science Editors:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Doctoral Dissertation]. Universidade do Rio Grande do Norte; 2013. Available from: http://repositorio.ufrn.br/handle/123456789/12575


Universidade do Rio Grande do Norte

6. Maciel, Bruna Leal Lima. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .

Degree: 2013, Universidade do Rio Grande do Norte

 Nutritional status is an important determinant to the response against Leishmania infection, although few studies have characterized the molecular basis for the association found between… (more)

Subjects/Keywords: Leishmaniose visceral. Ácido all-trans retinóico. IL-10; Visceral leishmaniasis. All-trans retinoic acid. IL-10

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maciel, B. L. L. (2013). Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . (Doctoral Dissertation). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/12575

Chicago Manual of Style (16th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Doctoral Dissertation, Universidade do Rio Grande do Norte. Accessed September 20, 2020. http://repositorio.ufrn.br/handle/123456789/12575.

MLA Handbook (7th Edition):

Maciel, Bruna Leal Lima. “Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum .” 2013. Web. 20 Sep 2020.

Vancouver:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Internet] [Doctoral dissertation]. Universidade do Rio Grande do Norte; 2013. [cited 2020 Sep 20]. Available from: http://repositorio.ufrn.br/handle/123456789/12575.

Council of Science Editors:

Maciel BLL. Estado nutricional e efeito da vitamina A na resposta imune frente à infecção por Leishmania Infantum . [Doctoral Dissertation]. Universidade do Rio Grande do Norte; 2013. Available from: http://repositorio.ufrn.br/handle/123456789/12575

7. 村上, 真史. ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.

Degree: 2018, Nagoya University / 名古屋大学

Ceramide is the central lipid in the sphingolipid metabolism. Ceramide kinase (CERK) and its product, ceramide 1-phosphate (C1P), have been implicated in various cellular functions.… (more)

Subjects/Keywords: All-trans retinoic acid; neuronal differentiation; SH-SY5Y cell; ceramide kinase; promoter analysis; COUP-TFI

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APA (6th Edition):

村上, . (2018). ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/14433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

村上, 真史. “ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed September 20, 2020. http://hdl.handle.net/2237/14433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

村上, 真史. “ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.” 2018. Web. 20 Sep 2020.

Vancouver:

村上 . ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2237/14433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

村上 . ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/14433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

8. Singh, Neeraj. The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia.

Degree: PhD, Veterinary Medical Sciences, 2010, Virginia Tech

 Oxidative stress plays a vital role in the pathogenesis of many chronic and acute inflammatory diseases. Reactive oxygen species (ROS) and reactive nitrogen species (RNS)… (more)

Subjects/Keywords: All-trans Retinoic Acid; Free radicals; Macrophages; IRAK-1; Lipopolysaccharide; Sepsis; NADPH oxidase; iNOS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Singh, N. (2010). The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77145

Chicago Manual of Style (16th Edition):

Singh, Neeraj. “The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia.” 2010. Doctoral Dissertation, Virginia Tech. Accessed September 20, 2020. http://hdl.handle.net/10919/77145.

MLA Handbook (7th Edition):

Singh, Neeraj. “The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia.” 2010. Web. 20 Sep 2020.

Vancouver:

Singh N. The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia. [Internet] [Doctoral dissertation]. Virginia Tech; 2010. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10919/77145.

Council of Science Editors:

Singh N. The Role of IRAK-1 in the Regulation of Free Radicals and Oxidative Stress during Endotoxemia. [Doctoral Dissertation]. Virginia Tech; 2010. Available from: http://hdl.handle.net/10919/77145

9. Liao, Xiaofeng. Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting.

Degree: PhD, Veterinary Medicine, 2017, Virginia Tech

 Systemic lupus erythematosus (SLE) is an autoimmune disease involving the inflammatory damages of multiple organs. Lupus nephritis (LN) as the manifestation in the kidney occurs… (more)

Subjects/Keywords: systemic lupus erythematosus; lupus nephritis; vitamin A; all-trans-retinoic acid; IFNα; dendritic cells

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APA (6th Edition):

Liao, X. (2017). Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/78696

Chicago Manual of Style (16th Edition):

Liao, Xiaofeng. “Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting.” 2017. Doctoral Dissertation, Virginia Tech. Accessed September 20, 2020. http://hdl.handle.net/10919/78696.

MLA Handbook (7th Edition):

Liao, Xiaofeng. “Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting.” 2017. Web. 20 Sep 2020.

Vancouver:

Liao X. Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting. [Internet] [Doctoral dissertation]. Virginia Tech; 2017. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10919/78696.

Council of Science Editors:

Liao X. Treatment of Systemic Lupus Erythematosus by Nutrition and Dendritic Cell Targeting. [Doctoral Dissertation]. Virginia Tech; 2017. Available from: http://hdl.handle.net/10919/78696


Virginia Tech

10. Surace, Michael Joseph. Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages.

Degree: PhD, Biology, 2010, Virginia Tech

 Macrophages can be activated by a variety of extracellular signals to polarize to either the M1 (inflammatory and antimicrobial) or to the M2 (wound repair… (more)

Subjects/Keywords: LPS; Lipopolysaccharide; ATRA; Interleukin 4; IL-4; All-trans Retinoic Acid; Arginase; Macrophage; M2; Alternative

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Surace, M. J. (2010). Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/26704

Chicago Manual of Style (16th Edition):

Surace, Michael Joseph. “Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages.” 2010. Doctoral Dissertation, Virginia Tech. Accessed September 20, 2020. http://hdl.handle.net/10919/26704.

MLA Handbook (7th Edition):

Surace, Michael Joseph. “Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages.” 2010. Web. 20 Sep 2020.

Vancouver:

Surace MJ. Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages. [Internet] [Doctoral dissertation]. Virginia Tech; 2010. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10919/26704.

Council of Science Editors:

Surace MJ. Signaling Cross-Talk Regulating the Expression of Arginase 1 in Murine Macrophages. [Doctoral Dissertation]. Virginia Tech; 2010. Available from: http://hdl.handle.net/10919/26704


University of Georgia

11. Campbell, Jerry Lamar. Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice.

Degree: 2014, University of Georgia

 The teratogenicity of all-trans retinoic acid (RA) has been known for decades, however to date no extensive investigation of the dose-response and kinetics across a… (more)

Subjects/Keywords: all-trans retinoic acid; fetal limb malformation; cleft palate; benchmark dose; physiologically based pharmacokinetic modeling

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APA (6th Edition):

Campbell, J. L. (2014). Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/22072

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Campbell, Jerry Lamar. “Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice.” 2014. Thesis, University of Georgia. Accessed September 20, 2020. http://hdl.handle.net/10724/22072.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Campbell, Jerry Lamar. “Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice.” 2014. Web. 20 Sep 2020.

Vancouver:

Campbell JL. Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice. [Internet] [Thesis]. University of Georgia; 2014. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10724/22072.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campbell JL. Physiologically-based pharmacokinetic modeling and dose-response for fetal skeletal malformations induced by ingestion of all-trans retinoic acid in CD-1 mice. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/22072

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

12. Sarkar, Suparna Ajoy. Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development.

Degree: 2014, University of Georgia

 Tumor-suppressor p53 regulates cell cycle, differentiation and apoptosis. The effect of all-trans-retinoic acid (RA) on p53, other protooncogenes and downstream signaling molecules were investigated during… (more)

Subjects/Keywords: p53; c-Myc; all-trans-retinoic acid; murine embryonic stem cells; differentiation

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APA (6th Edition):

Sarkar, S. A. (2014). Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/20429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sarkar, Suparna Ajoy. “Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development.” 2014. Thesis, University of Georgia. Accessed September 20, 2020. http://hdl.handle.net/10724/20429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sarkar, Suparna Ajoy. “Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development.” 2014. Web. 20 Sep 2020.

Vancouver:

Sarkar SA. Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development. [Internet] [Thesis]. University of Georgia; 2014. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10724/20429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sarkar SA. Involvement of p53 and selected proto-oncogenes in all-trans-retinoic acid-mediated murine embryonic development. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/20429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Lima, Ildercílio Mota de Souza. Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes.

Degree: Mestrado, Genética, 2013, University of São Paulo

 Células pluripotentes são aquelas que possuem a capacidade de dar origem às células dos três folhetos embrionários (ectoderma, mesoderma e endoderma), bem como também às… (more)

Subjects/Keywords: All-trans-retinoic Acid.; Ácido Trans-retinóico; Cell differentiation; Células-tronco; Diferenciação celular; NTera-2; NTera-2; Pluripotência; Pluripotency; Stem cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lima, I. M. d. S. (2013). Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17135/tde-29082013-155854/ ;

Chicago Manual of Style (16th Edition):

Lima, Ildercílio Mota de Souza. “Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes.” 2013. Masters Thesis, University of São Paulo. Accessed September 20, 2020. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-29082013-155854/ ;.

MLA Handbook (7th Edition):

Lima, Ildercílio Mota de Souza. “Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes.” 2013. Web. 20 Sep 2020.

Vancouver:

Lima IMdS. Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Sep 20]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-29082013-155854/ ;.

Council of Science Editors:

Lima IMdS. Um Modelo para Estudos de Modulação da Pluripotência e Diferenciação Celular em Células-Tronco Pluripotentes. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-29082013-155854/ ;


Temple University

14. Sirisani, Evelyn. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

All-trans retinoic acid (atRA) mediated growth inhibition results in the arrest of the cell cycle during the G1 phase in CAOV3 cells… (more)

Subjects/Keywords: Microbiology; Immunology; Molecular biology; all-trans retinoic acid; full length HOXA1; HOXA1; HOXB4; p16INK4a; truncated form HOXA1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sirisani, E. (2012). The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210886

Chicago Manual of Style (16th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Doctoral Dissertation, Temple University. Accessed September 20, 2020. http://digital.library.temple.edu/u?/p245801coll10,210886.

MLA Handbook (7th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Web. 20 Sep 2020.

Vancouver:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Sep 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886.

Council of Science Editors:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886


Temple University

15. Narvekar, Mayuri. DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA).

Degree: PhD, 2014, Temple University

Pharmaceutical Sciences

The commonly used PLGA-based delivery systems are often limited by their inadequate drug loading and release properties. This study reports the integration of… (more)

Subjects/Keywords: Pharmaceutical sciences; Nanotechnology;

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APA (6th Edition):

Narvekar, M. (2014). DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA). (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,245651

Chicago Manual of Style (16th Edition):

Narvekar, Mayuri. “DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA).” 2014. Doctoral Dissertation, Temple University. Accessed September 20, 2020. http://digital.library.temple.edu/u?/p245801coll10,245651.

MLA Handbook (7th Edition):

Narvekar, Mayuri. “DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA).” 2014. Web. 20 Sep 2020.

Vancouver:

Narvekar M. DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA). [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Sep 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,245651.

Council of Science Editors:

Narvekar M. DEVELOPMENT AND CHARACTERIZATION OF POLYMER-OIL NANOSTRUCTURED CARRIER (PONC) FOR CONTROLLED DELIVERY OF ALL-TRANS RETINOIC ACID (ATRA). [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,245651


Penn State University

16. Zhang, Yao. IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER .

Degree: 2008, Penn State University

All-trans retinoic acid (ATRA) is considered as the major active metabolite of vitamin A. ATRA regulates many physiological processes, such as embryonic development, cellular proliferation… (more)

Subjects/Keywords: RARE; CYP26A1; all-trans retinoic acid; promoter

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Y. (2008). IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/9026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Yao. “IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER .” 2008. Thesis, Penn State University. Accessed September 20, 2020. https://submit-etda.libraries.psu.edu/catalog/9026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Yao. “IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER .” 2008. Web. 20 Sep 2020.

Vancouver:

Zhang Y. IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER . [Internet] [Thesis]. Penn State University; 2008. [cited 2020 Sep 20]. Available from: https://submit-etda.libraries.psu.edu/catalog/9026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Y. IDENTIFICATION OF ADDITIONAL RETINOIC ACID RESPONSE ELEMENTS INVOLVED IN THE INDUCIBILITY OF CYP26A1 IN LIVER . [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/9026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

17. Naylor, Elizabeth. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.

Degree: 2014, University of Manchester

 Photoageing of human skin by cumulative exposure to ultraviolet radiation (UVR) is characterised by extensive remodelling of the long-lived dermal elastic fibre network. Fibrillin microfibrils… (more)

Subjects/Keywords: fibrillin microfibrils; extracellular matrix; skin; ageing; photoageing; ultraviolet radiation; all trans retinoic acid; retinoid; anti-ageing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Naylor, E. (2014). Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:219248

Chicago Manual of Style (16th Edition):

Naylor, Elizabeth. “Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.” 2014. Doctoral Dissertation, University of Manchester. Accessed September 20, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:219248.

MLA Handbook (7th Edition):

Naylor, Elizabeth. “Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.” 2014. Web. 20 Sep 2020.

Vancouver:

Naylor E. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2020 Sep 20]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:219248.

Council of Science Editors:

Naylor E. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:219248


University of Manchester

18. Naylor, Elizabeth. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.

Degree: PhD, 2014, University of Manchester

 Photoageing of human skin by cumulative exposure to ultraviolet radiation (UVR) is characterised by extensive remodelling of the long-lived dermal elastic fibre network. Fibrillin microfibrils… (more)

Subjects/Keywords: anti-ageing; fibrillin microfibrils; extracellular matrix; skin; ageing; photoageing; ultraviolet radiation; all trans retinoic acid; retinoid

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Naylor, E. (2014). Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/ultrastructural-characterisation-of-fibrillin-microfibrils-from-photoaged-and-alltrans-retinoic-acidtreated-skin(8a2be1a1-3678-4d0a-9b0c-51b4ab17771a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771308

Chicago Manual of Style (16th Edition):

Naylor, Elizabeth. “Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.” 2014. Doctoral Dissertation, University of Manchester. Accessed September 20, 2020. https://www.research.manchester.ac.uk/portal/en/theses/ultrastructural-characterisation-of-fibrillin-microfibrils-from-photoaged-and-alltrans-retinoic-acidtreated-skin(8a2be1a1-3678-4d0a-9b0c-51b4ab17771a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771308.

MLA Handbook (7th Edition):

Naylor, Elizabeth. “Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin.” 2014. Web. 20 Sep 2020.

Vancouver:

Naylor E. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2020 Sep 20]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/ultrastructural-characterisation-of-fibrillin-microfibrils-from-photoaged-and-alltrans-retinoic-acidtreated-skin(8a2be1a1-3678-4d0a-9b0c-51b4ab17771a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771308.

Council of Science Editors:

Naylor E. Ultrastructural characterisation of fibrillin microfibrils from photoaged and all-trans retinoic acid-treated skin. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/ultrastructural-characterisation-of-fibrillin-microfibrils-from-photoaged-and-alltrans-retinoic-acidtreated-skin(8a2be1a1-3678-4d0a-9b0c-51b4ab17771a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771308


University of Washington

19. Topletz, Ariel Rebecca. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.

Degree: PhD, 2013, University of Washington

All-trans-retinoic acid (atRA), the active metabolite of Vitamin A (retinol), is an essential nutrient during both fetal development and adult life. The concentration of atRA… (more)

Subjects/Keywords: 18-OH-RA; 4-oxo-RA; all-trans-retinoic acid; CYP26A1; CYP26B1; fetal liver; Pharmaceutical sciences; pharmaceutics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Topletz, A. R. (2013). The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/23714

Chicago Manual of Style (16th Edition):

Topletz, Ariel Rebecca. “The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.” 2013. Doctoral Dissertation, University of Washington. Accessed September 20, 2020. http://hdl.handle.net/1773/23714.

MLA Handbook (7th Edition):

Topletz, Ariel Rebecca. “The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.” 2013. Web. 20 Sep 2020.

Vancouver:

Topletz AR. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1773/23714.

Council of Science Editors:

Topletz AR. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/23714

20. D. Belperio. EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT.

Degree: 2017, Università degli Studi di Milano

 PHOX2B (Paired-like homeobox2B) and PHOX2A (Paired-like homeobox2A), are two paired like homeodomain proteins required for the correct autonomic nervous system development and for the specification… (more)

Subjects/Keywords: PHOX2B; PHOX2A; Congenital Central Hypoventilation Syndrom; Neuroblastoma; 3-keto-desogestrel; All-trans retinoic acid; Settore BIO/11 - Biologia Molecolare; Settore BIO/14 - Farmacologia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Belperio, D. (2017). EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/488326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Belperio, D.. “EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT.” 2017. Thesis, Università degli Studi di Milano. Accessed September 20, 2020. http://hdl.handle.net/2434/488326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Belperio, D.. “EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT.” 2017. Web. 20 Sep 2020.

Vancouver:

Belperio D. EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2434/488326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Belperio D. EFFECTS OF 3-KETODESOGESTREL AND ALL-TRANS RETINOIC ACID ON PHOX2A AND PHOX2B EXPRESSION: A COMMON STRATEGY AS NEW THERAPEUTIC PERSPECTIVE IN CONGENITAL CENTRAL HYPOVENTILATION SYNDROME (CCHS) AND NEUROBLASTOMA (NB) TREATMENT. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/488326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Maryland

21. Rajawat, Yogendra Singh. REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS.

Degree: Veterinary Medical Science, 2013, University of Maryland

Retinoic acids (RAs) have diverse biologic effects and regulate several cellular functions. Here, we investigated the role of RA on autophagy by studying its effects… (more)

Subjects/Keywords: Veterinary medicine; Cellular biology; Molecular biology; All trans retinoic acid; Autophagosome maturation; Autophagy; Cation-independent Mannose-6-Phosphate Receptor; LC3; Vitamin A

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rajawat, Y. S. (2013). REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS. (Thesis). University of Maryland. Retrieved from http://hdl.handle.net/1903/14646

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rajawat, Yogendra Singh. “REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS.” 2013. Thesis, University of Maryland. Accessed September 20, 2020. http://hdl.handle.net/1903/14646.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rajawat, Yogendra Singh. “REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS.” 2013. Web. 20 Sep 2020.

Vancouver:

Rajawat YS. REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS. [Internet] [Thesis]. University of Maryland; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1903/14646.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rajawat YS. REGULATION OF MACROAUTOPHAGY BY VITAMIN A/ RETINOIDS. [Thesis]. University of Maryland; 2013. Available from: http://hdl.handle.net/1903/14646

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Φακιολάς, Στέφανος. Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA.

Degree: 2012, University of Patras

 Στην παρούσα εργασία πραγματοποιήθηκε γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA. Σε καλλιέργειες κυττάρων HaCaT χορηγήθηκαν τα… (more)

Subjects/Keywords: Μικροσυστοιχίες; Ρετινοειδή; All-trans ρετινοϊκό οξύ; Ασιτρετίνη; Γονιδιωματική ανάλυση; Ψωρίαση; Κερατινοκύτταρα; Γονιδιακή έκφραση; 572.865; Microarrays; Retinoids; All-trans retinoic acid; Acitretin; Genome analysis; Psoriasis; Keratinocytes; Gene expression; Real-time PCR; HaCaT

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Φακιολάς, . (2012). Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/5720

Chicago Manual of Style (16th Edition):

Φακιολάς, Στέφανος. “Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA.” 2012. Masters Thesis, University of Patras. Accessed September 20, 2020. http://hdl.handle.net/10889/5720.

MLA Handbook (7th Edition):

Φακιολάς, Στέφανος. “Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA.” 2012. Web. 20 Sep 2020.

Vancouver:

Φακιολάς . Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA. [Internet] [Masters thesis]. University of Patras; 2012. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10889/5720.

Council of Science Editors:

Φακιολάς . Γονιδιωματική ανάλυση της επίδρασης αντιψωριασικών φαρμάκων σε καλλιέργειες ανθρώπινων κερατινοκυττάρων με τη χρήση μικροσυστοιχιών DNA. [Masters Thesis]. University of Patras; 2012. Available from: http://hdl.handle.net/10889/5720

23. Alakhras, Raghda Said H. Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4.

Degree: 2011, University of Patras

 min A and its metabolites retinal and retinoic acid are important molecules for the regulation of normal cellular growth, differentiation and other important functions. Retinoids… (more)

Subjects/Keywords: Retinoids - all-trans retinoic acid; Micronucleation; Chromosome breakage; Chromosome delay; Multinucleation; Mitotic spindle defect; DNA fragmentation; COMET assay; 616.994 061; Ρετινοειδή - all-trans ρετινοϊκό οξύ; Μικροπυρήνωση; Χρωμοσωματική θραύση; Χρωμοσωματική καθυστέρηση; Πολυπυρήνωση; Ανωμαλίες μιτωτικής ατράκτου; Κατάτμηση DNA; Μέθοδος COMET

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APA (6th Edition):

Alakhras, R. S. H. (2011). Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4. (Doctoral Dissertation). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/4762

Chicago Manual of Style (16th Edition):

Alakhras, Raghda Said H. “Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4.” 2011. Doctoral Dissertation, University of Patras. Accessed September 20, 2020. http://nemertes.lis.upatras.gr/jspui/handle/10889/4762.

MLA Handbook (7th Edition):

Alakhras, Raghda Said H. “Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4.” 2011. Web. 20 Sep 2020.

Vancouver:

Alakhras RSH. Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4. [Internet] [Doctoral dissertation]. University of Patras; 2011. [cited 2020 Sep 20]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/4762.

Council of Science Editors:

Alakhras RSH. Study of the genotoxicity mechanisms of all-trans retinoic acid and its analogue EA-4. [Doctoral Dissertation]. University of Patras; 2011. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/4762


University of Toledo Health Science Campus

24. Qi, Huiling. Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia.

Degree: PhD, College of Graduate Studies, 2005, University of Toledo Health Science Campus

 The folate receptor (FR) type Beta, which is expressed in about 70% of acute myelogenous leukemia (AML) and can be selectively up-regulated by all-trans retinoic(more)

Subjects/Keywords: Biology, Molecular; Folate Receptor; Acute Myelogenous Leukemia; All-trans Retinoic Acid; Histone Deacetylase Inhibitor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qi, H. (2005). Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1147304406

Chicago Manual of Style (16th Edition):

Qi, Huiling. “Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia.” 2005. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed September 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1147304406.

MLA Handbook (7th Edition):

Qi, Huiling. “Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia.” 2005. Web. 20 Sep 2020.

Vancouver:

Qi H. Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2005. [cited 2020 Sep 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1147304406.

Council of Science Editors:

Qi H. Modulation of Folate Receptor Beta for Drug Targeting in Acute Myelogenous Leukemia. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1147304406

25. 佐飛, 真知子. P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析.

Degree: Japan Advanced Institute of Science and Technology / 北陸先端科学技術大学院大学

Supervisor:塚原 俊文

材料科学研究科

修士

Subjects/Keywords: 分化誘導, P19胚性腫瘍細胞; all-trans Retinoic acid, Microarray

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APA (6th Edition):

佐飛, . (n.d.). P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析. (Thesis). Japan Advanced Institute of Science and Technology / 北陸先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10119/3171

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐飛, 真知子. “P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析.” Thesis, Japan Advanced Institute of Science and Technology / 北陸先端科学技術大学院大学. Accessed September 20, 2020. http://hdl.handle.net/10119/3171.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐飛, 真知子. “P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析.” Web. 20 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

佐飛 . P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析. [Internet] [Thesis]. Japan Advanced Institute of Science and Technology / 北陸先端科学技術大学院大学; [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10119/3171.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

佐飛 . P19胚性腫瘍細胞の分化誘導初期における遺伝子発現解析. [Thesis]. Japan Advanced Institute of Science and Technology / 北陸先端科学技術大学院大学; Available from: http://hdl.handle.net/10119/3171

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

26. Imran, Muhammad. Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions.

Degree: 2014, Ajou University

 Mouse TIS21 (12-O-tetradecanoyl phorbol-13-acetate inducible sequence 21), an ortholog of human BTG2 (B-cell translocation gene 2) and rat PC3, is a tumor suppressor that belongs… (more)

Subjects/Keywords: ATRA, all-trans-retinoic acid; Btg2, B-Cell translocation gene 2; GSK3, glycogen synthase kinase 3; DCF, dichlorofluorescin diacetate; LPS, Lipopolysaccharide; NAC, N-acetyl cysteine; PC3, pheochromocytoma cell-3; ROS, reactive oxygen species; TIS21(12-O-tetradecanoyl phorbol-13-acetate inducible sequence 21)

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APA (6th Edition):

Imran, M. (2014). Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/10859 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Imran, Muhammad. “Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions.” 2014. Thesis, Ajou University. Accessed September 20, 2020. http://repository.ajou.ac.kr/handle/201003/10859 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Imran, Muhammad. “Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions.” 2014. Web. 20 Sep 2020.

Vancouver:

Imran M. Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions. [Internet] [Thesis]. Ajou University; 2014. [cited 2020 Sep 20]. Available from: http://repository.ajou.ac.kr/handle/201003/10859 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Imran M. Role of BTG2/TIS21/PC3 in differentiation of myeloid leukemia cells and its regulation under stress conditions. [Thesis]. Ajou University; 2014. Available from: http://repository.ajou.ac.kr/handle/201003/10859 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

27. Bengtsson, Astrid. Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer.

Degree: 2009, University of Lund

 Colorectal cancer is the third most common type of cancer, and the second leading cause of cancer-related deaths in the Western world. Chronic inflammation, such… (more)

Subjects/Keywords: Cancer and Oncology; TNF alpha; COX-2; interferon alpha; all-trans retinoic acid; EGF; inflammation; colorectal cancer; CysLT2R; Leukotriene; CysLT1R

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APA (6th Edition):

Bengtsson, A. (2009). Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/1390695 ; https://portal.research.lu.se/ws/files/3889894/1390698.pdf

Chicago Manual of Style (16th Edition):

Bengtsson, Astrid. “Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer.” 2009. Doctoral Dissertation, University of Lund. Accessed September 20, 2020. https://lup.lub.lu.se/record/1390695 ; https://portal.research.lu.se/ws/files/3889894/1390698.pdf.

MLA Handbook (7th Edition):

Bengtsson, Astrid. “Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer.” 2009. Web. 20 Sep 2020.

Vancouver:

Bengtsson A. Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer. [Internet] [Doctoral dissertation]. University of Lund; 2009. [cited 2020 Sep 20]. Available from: https://lup.lub.lu.se/record/1390695 ; https://portal.research.lu.se/ws/files/3889894/1390698.pdf.

Council of Science Editors:

Bengtsson A. Signaling and regulation of cysteinyl leukotriene receptors in intestinal epithelial cells and colon cancer. [Doctoral Dissertation]. University of Lund; 2009. Available from: https://lup.lub.lu.se/record/1390695 ; https://portal.research.lu.se/ws/files/3889894/1390698.pdf


Penn State University

28. Ma, Yifan. ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE .

Degree: 2008, Penn State University

 min A (VA) is characterized as an anti-infective vitamin that plays an essential role in both innate and adaptive immunity. Polyriboinosinic: polyribocytidylic acid (PIC), a… (more)

Subjects/Keywords: all-trans retinoic acid; polyriboinosinic: polyribocytidylic acid (poly IC); vaccine response; type 1/ type 2 response; neonatal immunity; antigen-presenting cells; germinal center reaction

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APA (6th Edition):

Ma, Y. (2008). ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ma, Yifan. “ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE .” 2008. Thesis, Penn State University. Accessed September 20, 2020. https://submit-etda.libraries.psu.edu/catalog/6881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ma, Yifan. “ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE .” 2008. Web. 20 Sep 2020.

Vancouver:

Ma Y. ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE . [Internet] [Thesis]. Penn State University; 2008. [cited 2020 Sep 20]. Available from: https://submit-etda.libraries.psu.edu/catalog/6881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ma Y. ALL-TRANS RETINOIC ACID COMBINED WITH POLYRIBOINOSINIC:POLYRIBOCYTIDYLIC ACID PROMOTES TETANUS TOXOID-INDUCED VACCINE RESPONSES IN ADULT AND NEONATAL MICE . [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cal Poly

29. Brown, David A. THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION.

Degree: MS, Food Science and Nutrition, 2009, Cal Poly

 Vitamin A metabolites and retinoids may slow the progression of breast cancer and elicit anti-neoplastic properties similar to those of omega-3 fatty acids. Studies using… (more)

Subjects/Keywords: All-trans retinoic acid; breast cancer; fatty acid; MCF-7 cell culture; Peroxisome Proliferator; ATRA; Hormones, Hormone Substitutes, and Hormone Antagonists; Lipids; Pharmaceutical Preparations

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APA (6th Edition):

Brown, D. A. (2009). THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION. (Masters Thesis). Cal Poly. Retrieved from https://digitalcommons.calpoly.edu/theses/180 ; 10.15368/theses.2009.151

Chicago Manual of Style (16th Edition):

Brown, David A. “THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION.” 2009. Masters Thesis, Cal Poly. Accessed September 20, 2020. https://digitalcommons.calpoly.edu/theses/180 ; 10.15368/theses.2009.151.

MLA Handbook (7th Edition):

Brown, David A. “THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION.” 2009. Web. 20 Sep 2020.

Vancouver:

Brown DA. THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION. [Internet] [Masters thesis]. Cal Poly; 2009. [cited 2020 Sep 20]. Available from: https://digitalcommons.calpoly.edu/theses/180 ; 10.15368/theses.2009.151.

Council of Science Editors:

Brown DA. THE EFFECT OF ALL-TRANS RETINOIC ACID AND FATTY ACIDS ON MCF-7 BREAST CANCER CELL PROGRESSION. [Masters Thesis]. Cal Poly; 2009. Available from: https://digitalcommons.calpoly.edu/theses/180 ; 10.15368/theses.2009.151

30. 入山, 規良. 急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる.

Degree: 博士( 医学 ), 2013, Nihon University / 日本大学

Subjects/Keywords: 全トランスレチノイン酸; 亜ヒ酸; HT93A; aquaporin 9; 顆粒球刺激因子; all-trans retinoic acid; arsenic trioxide; granulocyte colony-stimulating factor

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APA (6th Edition):

入山, . (2013). 急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/27 ; http://dx.doi.org/10.15006/32665B7022

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

入山, 規良. “急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる.” 2013. Thesis, Nihon University / 日本大学. Accessed September 20, 2020. http://repository.nihon-u.ac.jp/xmlui/handle/11263/27 ; http://dx.doi.org/10.15006/32665B7022.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

入山, 規良. “急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる.” 2013. Web. 20 Sep 2020.

Vancouver:

入山 . 急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる. [Internet] [Thesis]. Nihon University / 日本大学; 2013. [cited 2020 Sep 20]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/27 ; http://dx.doi.org/10.15006/32665B7022.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

入山 . 急性前骨髄性白血病細胞株HT93A においてG-CSF の併用はATRA 及び亜ヒ酸の分化誘導と亜ヒ酸の細胞内取り込みを増強させる. [Thesis]. Nihon University / 日本大学; 2013. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/27 ; http://dx.doi.org/10.15006/32665B7022

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [524]

.