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You searched for subject:(YAP1). Showing records 1 – 16 of 16 total matches.

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Penn State University

1. Hegde, Shailaja N. The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal.

Degree: PhD, Pathobiology, 2013, Penn State University

 Leukemia can be viewed as a newly formed, abnormal hematopoietic tissue initiated by a few leukemic stem cells (LSCs) that undergo a poorly regulated differentiation… (more)

Subjects/Keywords: CML; Hedgehog; p53; Yap1; BCR-ABL

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APA (6th Edition):

Hegde, S. N. (2013). The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/20210

Chicago Manual of Style (16th Edition):

Hegde, Shailaja N. “The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal.” 2013. Doctoral Dissertation, Penn State University. Accessed August 24, 2019. https://etda.libraries.psu.edu/catalog/20210.

MLA Handbook (7th Edition):

Hegde, Shailaja N. “The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal.” 2013. Web. 24 Aug 2019.

Vancouver:

Hegde SN. The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal. [Internet] [Doctoral dissertation]. Penn State University; 2013. [cited 2019 Aug 24]. Available from: https://etda.libraries.psu.edu/catalog/20210.

Council of Science Editors:

Hegde SN. The Role of Hedgehog,p53 and Yap1 in chronic myelogenous leukemia and Friend vrus erythroleukemia stem cell self renewal. [Doctoral Dissertation]. Penn State University; 2013. Available from: https://etda.libraries.psu.edu/catalog/20210


Boston University

2. Tsai, Michelle. RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression.

Degree: MS, Medical Sciences, 2014, Boston University

 Oxidative stress mechanism in yeast presents an innovative pathway to understand in creating the next generation of antifungal drugs. Rgc1 and Rgc2 are paralogous proteins… (more)

Subjects/Keywords: Cellular biology; Rgc1; Rgc2; Yap1; Oxidative stress

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APA (6th Edition):

Tsai, M. (2014). RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15092

Chicago Manual of Style (16th Edition):

Tsai, Michelle. “RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression.” 2014. Masters Thesis, Boston University. Accessed August 24, 2019. http://hdl.handle.net/2144/15092.

MLA Handbook (7th Edition):

Tsai, Michelle. “RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression.” 2014. Web. 24 Aug 2019.

Vancouver:

Tsai M. RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression. [Internet] [Masters thesis]. Boston University; 2014. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2144/15092.

Council of Science Editors:

Tsai M. RGC1/RGC2 deletions cause increased sensitivity to oxidative stress in Saccharomyces cerevisiae, which can be overcome by constitutive nuclear Yap1 expression. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15092


NSYSU

3. Li, Yu-wei. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

 Glioblastoma multiforme (GBM) is the most aggressive and malignant primary brain tumor. Primary glioblastomas have a worse prognosis with a median survival of less than… (more)

Subjects/Keywords: Mouse model; Glioma; YAP1; MST1/2; Hippo pathway; Glioblastoma; Apoptosis; Verteporfin

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APA (6th Edition):

Li, Y. (2016). The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yu-wei. “The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.” 2016. Thesis, NSYSU. Accessed August 24, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yu-wei. “The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.” 2016. Web. 24 Aug 2019.

Vancouver:

Li Y. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. [Internet] [Thesis]. NSYSU; 2016. [cited 2019 Aug 24]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. ELSZTEIN, Carolina. Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae .

Degree: 2011, Universidade Federal de Pernambuco

 Quando submetidas a condições de estresse as células respondem a partir da indução de genes e ativação de proteínas em mecanismos regulados por cascatas de… (more)

Subjects/Keywords: Expressão gênica; MAP quinase; Parede celular; PHMB; Saccharomyces cerevisiae; YAP1

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APA (6th Edition):

ELSZTEIN, C. (2011). Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/6098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ELSZTEIN, Carolina. “Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae .” 2011. Thesis, Universidade Federal de Pernambuco. Accessed August 24, 2019. http://repositorio.ufpe.br/handle/123456789/6098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ELSZTEIN, Carolina. “Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae .” 2011. Web. 24 Aug 2019.

Vancouver:

ELSZTEIN C. Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2011. [cited 2019 Aug 24]. Available from: http://repositorio.ufpe.br/handle/123456789/6098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ELSZTEIN C. Identificação dos mecanismos moleculares de resistência ao polhexametileno biguanida na levedura Saccharomyces cerevisiae . [Thesis]. Universidade Federal de Pernambuco; 2011. Available from: http://repositorio.ufpe.br/handle/123456789/6098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

5. Sun, Tianyanxin. The Roles of Hippo Signaling Pathway in Mouse Ovarian Function.

Degree: PhD, Physiology, 2015, Penn State University

 Mammalian follicular development is regulated by various secreted and gap-junction-mediated intercellular communication signals. This complicated network of regulatory and metabolic mechanisms is not completely known.… (more)

Subjects/Keywords: Hippo Signaling; Follicular Development; Ovulation; YAP1; LATS1; Mouse

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APA (6th Edition):

Sun, T. (2015). The Roles of Hippo Signaling Pathway in Mouse Ovarian Function. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/28915

Chicago Manual of Style (16th Edition):

Sun, Tianyanxin. “The Roles of Hippo Signaling Pathway in Mouse Ovarian Function.” 2015. Doctoral Dissertation, Penn State University. Accessed August 24, 2019. https://etda.libraries.psu.edu/catalog/28915.

MLA Handbook (7th Edition):

Sun, Tianyanxin. “The Roles of Hippo Signaling Pathway in Mouse Ovarian Function.” 2015. Web. 24 Aug 2019.

Vancouver:

Sun T. The Roles of Hippo Signaling Pathway in Mouse Ovarian Function. [Internet] [Doctoral dissertation]. Penn State University; 2015. [cited 2019 Aug 24]. Available from: https://etda.libraries.psu.edu/catalog/28915.

Council of Science Editors:

Sun T. The Roles of Hippo Signaling Pathway in Mouse Ovarian Function. [Doctoral Dissertation]. Penn State University; 2015. Available from: https://etda.libraries.psu.edu/catalog/28915

6. Masliantsev, Konstantin. Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets.

Degree: Docteur es, Aspects Moléculaires et Cellulaires de la Biologie, 2018, Poitiers

Les gliomes malins sont les tumeurs les plus fréquentes du système nerveux central. Les glioblastomes représentant plus de 50% des gliomes, constituent la forme la… (more)

Subjects/Keywords: Gliome; Glioblastome; Cellules souches de glioblastomes; Radiorésistance; Stat3; Signalisation Hippo; Yap1; Tead3.; Glioma; Glioblastoma; Glioblastoma stem cells; Radioresistance; Stat3; Hippo signaling; Yap1; Tead3.; 612.82; 616.994

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APA (6th Edition):

Masliantsev, K. (2018). Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2018POIT1407

Chicago Manual of Style (16th Edition):

Masliantsev, Konstantin. “Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets.” 2018. Doctoral Dissertation, Poitiers. Accessed August 24, 2019. http://www.theses.fr/2018POIT1407.

MLA Handbook (7th Edition):

Masliantsev, Konstantin. “Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets.” 2018. Web. 24 Aug 2019.

Vancouver:

Masliantsev K. Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets. [Internet] [Doctoral dissertation]. Poitiers; 2018. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2018POIT1407.

Council of Science Editors:

Masliantsev K. Rôle des signalisations STAT3 et Hippo dans les gliomes : Identification de nouveaux biomarqueurs pronostiques et cibles thérapeutiques : Role of STAT3 and Hippo signaling pathways in glioma : Identification of new prognostic biomarkers and therapeutic targets. [Doctoral Dissertation]. Poitiers; 2018. Available from: http://www.theses.fr/2018POIT1407

7. 甲斐, 友喜. Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway.

Degree: 博士(医学), 2017, Oita University / 大分大学

 We have previously reported that Salvador homologue 1 (SAV1), a component of the Hippo pathway, is significantly down-regulated in high-grade clear cell renal cell carcinoma… (more)

Subjects/Keywords: SAV1; Hippo pathway; YAP1; clear cell renal cell carcinoma; knockout mouse; atypical renal tubule

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APA (6th Edition):

甲斐, . (2017). Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

甲斐, 友喜. “Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway.” 2017. Thesis, Oita University / 大分大学. Accessed August 24, 2019. http://hdl.handle.net/10559/15764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

甲斐, 友喜. “Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway.” 2017. Web. 24 Aug 2019.

Vancouver:

甲斐 . Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway. [Internet] [Thesis]. Oita University / 大分大学; 2017. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10559/15764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

甲斐 . Kidney-specific knockout of Sav1 in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway. [Thesis]. Oita University / 大分大学; 2017. Available from: http://hdl.handle.net/10559/15764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Pascoal, Rita Fernandes. Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas.

Degree: 2014, Instituto Politécnico de Leiria

 No âmbito da saúde e farmacologia a procura de compostos para a prevenção e tratamento de doenças é constante, tendo sido valorizada atualmente a medicina… (more)

Subjects/Keywords: Screening de drogas; stresse oxidativo,; Yap1; Msn2.; Domínio/Área Científica::Engenharia e Tecnologia

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APA (6th Edition):

Pascoal, R. F. (2014). Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas. (Thesis). Instituto Politécnico de Leiria. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:iconline.ipleiria.pt:10400.8/1934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pascoal, Rita Fernandes. “Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas.” 2014. Thesis, Instituto Politécnico de Leiria. Accessed August 24, 2019. http://www.rcaap.pt/detail.jsp?id=oai:iconline.ipleiria.pt:10400.8/1934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pascoal, Rita Fernandes. “Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas.” 2014. Web. 24 Aug 2019.

Vancouver:

Pascoal RF. Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas. [Internet] [Thesis]. Instituto Politécnico de Leiria; 2014. [cited 2019 Aug 24]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:iconline.ipleiria.pt:10400.8/1934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pascoal RF. Melhoramento genético de estirpes Saccharomyces cerevisiae YAP1-GFP e MSN2-GFP para a determinação de sensibilidade a drogas. [Thesis]. Instituto Politécnico de Leiria; 2014. Available from: http://www.rcaap.pt/detail.jsp?id=oai:iconline.ipleiria.pt:10400.8/1934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

9. Schaal, Courtney. Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC.

Degree: 2016, University of South Florida

 Lung cancer is the leading cause of cancer-related death in both men and women, nationally and internationally and kills more people each year than breast,… (more)

Subjects/Keywords: Smoking-related lung cancer; Sox2; alpha7; E2F1; Yap1; Cell Biology; Molecular Biology; Oncology

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APA (6th Edition):

Schaal, C. (2016). Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schaal, Courtney. “Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC.” 2016. Thesis, University of South Florida. Accessed August 24, 2019. https://scholarcommons.usf.edu/etd/6582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schaal, Courtney. “Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC.” 2016. Web. 24 Aug 2019.

Vancouver:

Schaal C. Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC. [Internet] [Thesis]. University of South Florida; 2016. [cited 2019 Aug 24]. Available from: https://scholarcommons.usf.edu/etd/6582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schaal C. Regulation of nAChRs and Stemness by Nicotine and E-cigarettes in NSCLC. [Thesis]. University of South Florida; 2016. Available from: https://scholarcommons.usf.edu/etd/6582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

10. Xu, Jia. 14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER.

Degree: PhD, 2012, Texas Medical Center

  TGF-β plays an important role in differentiation and tissue morphogenesis as well as cancer progression. However, the role of TGF-β in cancer is complicate.… (more)

Subjects/Keywords: 14-3-3 zeta; TGF-beta; molecular switch; 14-3-3 sigma; breast cancer; bone metastases; Gli2; p53; YAP1; Cancer Biology

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APA (6th Edition):

Xu, J. (2012). 14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/268

Chicago Manual of Style (16th Edition):

Xu, Jia. “14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed August 24, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/268.

MLA Handbook (7th Edition):

Xu, Jia. “14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER.” 2012. Web. 24 Aug 2019.

Vancouver:

Xu J. 14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Aug 24]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/268.

Council of Science Editors:

Xu J. 14-3-3 ZETA OVEREXPRESSION SERVES AS A NOVEL MOLECULAR SWITCH TURNING TGF-BETA FROM TUMOR SUPPRESSOR TO TUMOR PROMOTER. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/268


Texas Medical Center

11. Previs, Rebecca A. YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies.

Degree: MS, 2015, Texas Medical Center

  Purpose: The PI3K/AKT/P70S6K pathway is an attractive therapeutic target in ovarian and uterine malignancies due to its high rate of dysregulation and key roles… (more)

Subjects/Keywords: ovarian cancer; uterine cancer; AKT inhibitors; YAP1; P70S6K; angiogenesis; bevacizumab; adaptive resistance; Medicine and Health Sciences; Neoplasms

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APA (6th Edition):

Previs, R. A. (2015). YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/595

Chicago Manual of Style (16th Edition):

Previs, Rebecca A. “YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies.” 2015. Masters Thesis, Texas Medical Center. Accessed August 24, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/595.

MLA Handbook (7th Edition):

Previs, Rebecca A. “YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies.” 2015. Web. 24 Aug 2019.

Vancouver:

Previs RA. YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies. [Internet] [Masters thesis]. Texas Medical Center; 2015. [cited 2019 Aug 24]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/595.

Council of Science Editors:

Previs RA. YAP1 expression predicts sensitivity to dual AKT/P70S6K inhibition in ovarian and uterine malignancies. [Masters Thesis]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/595


Virginia Tech

12. Sha, Wei. Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress.

Degree: PhD, Genetics, Bioinformatics, and Computational Biology, 2006, Virginia Tech

 Oxidative stress is a harmful condition in a cell, tissue, or organ, caused by an imbalance between reactive oxygen species or other oxidants and the… (more)

Subjects/Keywords: oxidative stress; microarray data analysis; cumeme hydroperoxide; Yap1

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APA (6th Edition):

Sha, W. (2006). Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/27840

Chicago Manual of Style (16th Edition):

Sha, Wei. “Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress.” 2006. Doctoral Dissertation, Virginia Tech. Accessed August 24, 2019. http://hdl.handle.net/10919/27840.

MLA Handbook (7th Edition):

Sha, Wei. “Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress.” 2006. Web. 24 Aug 2019.

Vancouver:

Sha W. Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress. [Internet] [Doctoral dissertation]. Virginia Tech; 2006. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10919/27840.

Council of Science Editors:

Sha W. Microarray data analysis methods and their applications to gene expression data analysis for Saccharomyces cerevisiae under oxidative stress. [Doctoral Dissertation]. Virginia Tech; 2006. Available from: http://hdl.handle.net/10919/27840


IUPUI

13. Qin, Li. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.

Degree: 2014, IUPUI

Indiana University-Purdue University (IUPUI)

Most pancreatic cancer patients receiving gemcitabine chemotherapy eventually develop resistance to gemcitabine. To improve survival and prognosis of pancreatic cancer patients,… (more)

Subjects/Keywords: 14-3-3sigma; PDGFD; YAP1; Uhrf1; DNMT1; Apoptosis; Chemotherapy; Drugs  – Side effects; Pancreas  – Diseases; Cancer  – Genetic aspects; Cancer  – Molecular aspects; Genomics

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APA (6th Edition):

Qin, L. (2014). Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qin, Li. “Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.” 2014. Thesis, IUPUI. Accessed August 24, 2019. http://hdl.handle.net/1805/6295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qin, Li. “Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.” 2014. Web. 24 Aug 2019.

Vancouver:

Qin L. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. [Internet] [Thesis]. IUPUI; 2014. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/1805/6295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qin L. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

14. Nakagawa, Chad Isamu. Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy.

Degree: MS, Molecular Microbiology and Immunology, 2015, University of Southern California

 Hepatocellular carcinoma (HCC) is the fifth most common type of cancer. The mortality rate continues to rise every year. Hepatitis B virus (HBV) is a… (more)

Subjects/Keywords: hepatocellular carcinoma; HCC; hepatitis B virus X protein; HBx; NANOG; YAP1; β -catenin; tumor initiating cells; TICs

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nakagawa, C. I. (2015). Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615884/rec/3149

Chicago Manual of Style (16th Edition):

Nakagawa, Chad Isamu. “Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy.” 2015. Masters Thesis, University of Southern California. Accessed August 24, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615884/rec/3149.

MLA Handbook (7th Edition):

Nakagawa, Chad Isamu. “Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy.” 2015. Web. 24 Aug 2019.

Vancouver:

Nakagawa CI. Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Aug 24]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615884/rec/3149.

Council of Science Editors:

Nakagawa CI. Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615884/rec/3149


University of Guelph

15. Sharma, Jyoti. Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development .

Degree: 2016, University of Guelph

 Blastocyst formation is an important milestone in preimplantation embryo development. During murine preimplantation embryogenesis, the Hippo signaling pathway is known to play a significant role… (more)

Subjects/Keywords: Preimplantation embryo development; Blastocyst; Trophectoderm; Inner Cell Mass; Hippo signaling pathway; MST1/2 Kinase; LATS1/2 Kinase; YAP1; TAZ

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sharma, J. (2016). Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Jyoti. “Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development .” 2016. Thesis, University of Guelph. Accessed August 24, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Jyoti. “Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development .” 2016. Web. 24 Aug 2019.

Vancouver:

Sharma J. Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development . [Internet] [Thesis]. University of Guelph; 2016. [cited 2019 Aug 24]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma J. Role Of Hippo Signaling Pathway In Bovine Preimplantation Embryo Development . [Thesis]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

16. Berra, Siham. The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae .

Degree: 2012, Université de Montréal

Subjects/Keywords: bléomycine; Bleomycin; cancer testiculaire; testicular cancer; Imp2; Yap1; Tpo1; résistance à la drogue; Drug resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Berra, S. (2012). The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/8495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Berra, Siham. “The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae .” 2012. Thesis, Université de Montréal. Accessed August 24, 2019. http://hdl.handle.net/1866/8495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Berra, Siham. “The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae .” 2012. Web. 24 Aug 2019.

Vancouver:

Berra S. The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/1866/8495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Berra S. The overexpression of the efflux pump Tpo1 leads to the bleomycin resistance in Saccharomyces cerevisiae . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/8495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.