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You searched for subject:(Vascular smooth muscle cell). Showing records 1 – 30 of 35495 total matches.

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University of Cambridge

1. Harman, Jennifer. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.

Degree: PhD, 2019, University of Cambridge

 Widespread changes in gene expression accompany vascular smooth muscle cell (VSMC) phenotypic switching, a hallmark of vascular disease. Upon insult, VSMCs downregulate contractile proteins and… (more)

Subjects/Keywords: Vascular smooth muscle cell; H3K9me2; Vascular smooth muscle cell phenotypic switch

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APA (6th Edition):

Harman, J. (2019). Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289975 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767866

Chicago Manual of Style (16th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Doctoral Dissertation, University of Cambridge. Accessed September 23, 2019. https://www.repository.cam.ac.uk/handle/1810/289975 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767866.

MLA Handbook (7th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Web. 23 Sep 2019.

Vancouver:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2019 Sep 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/289975 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767866.

Council of Science Editors:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289975 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767866


University of Cambridge

2. Harman, Jennifer. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression .

Degree: 2019, University of Cambridge

 Widespread changes in gene expression accompany vascular smooth muscle cell (VSMC) phenotypic switching, a hallmark of vascular disease. Upon insult, VSMCs downregulate contractile proteins and… (more)

Subjects/Keywords: Vascular smooth muscle cell; H3K9me2; Vascular smooth muscle cell phenotypic switch

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APA (6th Edition):

Harman, J. (2019). Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289975

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression .” 2019. Thesis, University of Cambridge. Accessed September 23, 2019. https://www.repository.cam.ac.uk/handle/1810/289975.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression .” 2019. Web. 23 Sep 2019.

Vancouver:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression . [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Sep 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/289975.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression . [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289975

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

3. Pessolano, Lawrence. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.

Degree: PhD, Biochemistry, 2016, Boston University

Vascular smooth muscle cells (SMCs) regulate vessel contraction but during diseases including atherosclerosis, SMCs undergo functional changes that contribute to pathology. Chronic inflammation in the… (more)

Subjects/Keywords: Molecular biology; Atherosclerosis; Cholesterol trafficking; Smooth muscle cell; Smooth muscle cell differentiation; Vascular biology

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APA (6th Edition):

Pessolano, L. (2016). Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14599

Chicago Manual of Style (16th Edition):

Pessolano, Lawrence. “Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.” 2016. Doctoral Dissertation, Boston University. Accessed September 23, 2019. http://hdl.handle.net/2144/14599.

MLA Handbook (7th Edition):

Pessolano, Lawrence. “Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.” 2016. Web. 23 Sep 2019.

Vancouver:

Pessolano L. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2144/14599.

Council of Science Editors:

Pessolano L. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14599


University of Toronto

4. Hui, Sonya. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.

Degree: 2010, University of Toronto

Increased intracellular calcium (Ca2+) is required for vascular smooth muscle cell (VSMC) proliferation through mechanisms that are not well-known. Preventing calmodulin (CaM)-cyclin E interaction with… (more)

Subjects/Keywords: Calcium; Cell Cycle; Vascular smooth muscle cell; 0719

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APA (6th Edition):

Hui, S. (2010). Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25625

Chicago Manual of Style (16th Edition):

Hui, Sonya. “Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.” 2010. Masters Thesis, University of Toronto. Accessed September 23, 2019. http://hdl.handle.net/1807/25625.

MLA Handbook (7th Edition):

Hui, Sonya. “Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.” 2010. Web. 23 Sep 2019.

Vancouver:

Hui S. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1807/25625.

Council of Science Editors:

Hui S. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25625


Queens University

5. Truong, Tammy. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 During the development of atherosclerosis, contractile vascular smooth muscle cells (VSMCs) change to cells capable of migrating and proliferating to mediate repair, where the responses… (more)

Subjects/Keywords: cell migration; vascular smooth muscle cell; cAMP; compartmentalization; phosphodiesterase 4D

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APA (6th Edition):

Truong, T. (2014). Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Truong, Tammy. “Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .” 2014. Thesis, Queens University. Accessed September 23, 2019. http://hdl.handle.net/1974/8658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Truong, Tammy. “Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .” 2014. Web. 23 Sep 2019.

Vancouver:

Truong T. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . [Internet] [Thesis]. Queens University; 2014. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1974/8658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Truong T. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

6. Robb, Tiffany Marie. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .

Degree: 2012, University of Arizona

 Intimal hyperplasia (IH) is characteristic of a cell population increase within the innermost layer of the arterial wall. It is hypothesized that extracellular matrix vascular(more)

Subjects/Keywords: Vascular endothleial cell; Vascular smooth muscle cell; Pharmacology & Toxicology; Intimal hyperplasia; Simvastatin

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APA (6th Edition):

Robb, T. M. (2012). Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/228464

Chicago Manual of Style (16th Edition):

Robb, Tiffany Marie. “Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .” 2012. Masters Thesis, University of Arizona. Accessed September 23, 2019. http://hdl.handle.net/10150/228464.

MLA Handbook (7th Edition):

Robb, Tiffany Marie. “Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .” 2012. Web. 23 Sep 2019.

Vancouver:

Robb TM. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . [Internet] [Masters thesis]. University of Arizona; 2012. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10150/228464.

Council of Science Editors:

Robb TM. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . [Masters Thesis]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/228464


University of Aberdeen

7. McKean, Jenny Susan. The role of the cAMP mediator Epac in vascular smooth muscle cell migration.

Degree: PhD, 2015, University of Aberdeen

 Surgical intervention can result in endothelial denudation, driving growth factor-stimulated vascular smooth muscle cell (VSMC) migration towards the intima, leading to luminal narrowing and restenosis.… (more)

Subjects/Keywords: Coronary arteries; Vascular smooth muscle; Cell migration; Cyclic adenylic acid

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APA (6th Edition):

McKean, J. S. (2015). The role of the cAMP mediator Epac in vascular smooth muscle cell migration. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069

Chicago Manual of Style (16th Edition):

McKean, Jenny Susan. “The role of the cAMP mediator Epac in vascular smooth muscle cell migration.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed September 23, 2019. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069.

MLA Handbook (7th Edition):

McKean, Jenny Susan. “The role of the cAMP mediator Epac in vascular smooth muscle cell migration.” 2015. Web. 23 Sep 2019.

Vancouver:

McKean JS. The role of the cAMP mediator Epac in vascular smooth muscle cell migration. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2019 Sep 23]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069.

Council of Science Editors:

McKean JS. The role of the cAMP mediator Epac in vascular smooth muscle cell migration. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069


University of Western Ontario

8. Leung, Sharon Z. The Role of Sirtuin 6 in Maintaining Vascular Integrity.

Degree: 2016, University of Western Ontario

 Oxidative stress is an underlying cause for vascular pathologies including inflammation, hypertension, and atherosclerosis. Sirtuins (SIRTs) are a family of NAD+ dependent deacetylases with pronounced… (more)

Subjects/Keywords: SIRT6; vascular smooth muscle cell; angiotensin II; inflammation; Biochemistry

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APA (6th Edition):

Leung, S. Z. (2016). The Role of Sirtuin 6 in Maintaining Vascular Integrity. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leung, Sharon Z. “The Role of Sirtuin 6 in Maintaining Vascular Integrity.” 2016. Thesis, University of Western Ontario. Accessed September 23, 2019. https://ir.lib.uwo.ca/etd/3676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leung, Sharon Z. “The Role of Sirtuin 6 in Maintaining Vascular Integrity.” 2016. Web. 23 Sep 2019.

Vancouver:

Leung SZ. The Role of Sirtuin 6 in Maintaining Vascular Integrity. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2019 Sep 23]. Available from: https://ir.lib.uwo.ca/etd/3676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leung SZ. The Role of Sirtuin 6 in Maintaining Vascular Integrity. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/3676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Clemson University

9. Bradley, Suzanne Nicole. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.

Degree: MS, Bioengineering, 2018, Clemson University

Vascular smooth muscle cells regulate blood flow by contracting and relaxing blood vessels. Vascular smooth muscle cells display one of two distinct phenotypes: contractile or… (more)

Subjects/Keywords: Cell Stretching; Micropatterning; Microstamping; Vascular Smooth Muscle Cells

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APA (6th Edition):

Bradley, S. N. (2018). Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/2957

Chicago Manual of Style (16th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Masters Thesis, Clemson University. Accessed September 23, 2019. https://tigerprints.clemson.edu/all_theses/2957.

MLA Handbook (7th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Web. 23 Sep 2019.

Vancouver:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Internet] [Masters thesis]. Clemson University; 2018. [cited 2019 Sep 23]. Available from: https://tigerprints.clemson.edu/all_theses/2957.

Council of Science Editors:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Masters Thesis]. Clemson University; 2018. Available from: https://tigerprints.clemson.edu/all_theses/2957


University of Toronto

10. Mantella, Laura-Eve. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.

Degree: 2016, University of Toronto

Long noncoding RNAs (lncRNAs) are a new class of noncoding RNA and emerging evidence suggests that they represent a cellular hub for coordination of various… (more)

Subjects/Keywords: aortic aneurysm; hypertension; mechanical stretch; vascular smooth muscle cell; 0379

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APA (6th Edition):

Mantella, L. (2016). Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75343

Chicago Manual of Style (16th Edition):

Mantella, Laura-Eve. “Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.” 2016. Masters Thesis, University of Toronto. Accessed September 23, 2019. http://hdl.handle.net/1807/75343.

MLA Handbook (7th Edition):

Mantella, Laura-Eve. “Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.” 2016. Web. 23 Sep 2019.

Vancouver:

Mantella L. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1807/75343.

Council of Science Editors:

Mantella L. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75343

11. E.M. Volpi. STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO.

Degree: 2012, Università degli Studi di Milano

Vascular Calcification (VC), induced by a high-phosphate, is characterized by a passive deposition of calcium-phosphate (Ca-P) and an active transformation of vascular smooth muscle cells… (more)

Subjects/Keywords: vascular calcification; vascular smooth muscle cell; phosphate; ascorbic acid; lanthanum; Settore MED/14 - Nefrologia

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APA (6th Edition):

Volpi, E. (2012). STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/171328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Volpi, E.M.. “STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO.” 2012. Thesis, Università degli Studi di Milano. Accessed September 23, 2019. http://hdl.handle.net/2434/171328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Volpi, E.M.. “STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO.” 2012. Web. 23 Sep 2019.

Vancouver:

Volpi E. STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2434/171328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Volpi E. STUDIO DEI MECCANISMI PATOGENETICI COINVOLTI NELLA PROGRESSIONE DELLE CALCIFICAZIONI VASCOLARI INDOTTE DA ELEVATI LIVELLI DI FOSFORO. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/171328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

12. Guo, Xia. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.

Degree: PhD, Physiology, 2014, University of Georgia

 The objective of this study was to develop a novel in vitro model for smooth muscle cell (SMC) differentiation from human embryonic stem cell-derived mesenchymal… (more)

Subjects/Keywords: Vascular smooth muscle cells

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APA (6th Edition):

Guo, X. (2014). Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd

Chicago Manual of Style (16th Edition):

Guo, Xia. “Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.” 2014. Doctoral Dissertation, University of Georgia. Accessed September 23, 2019. http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd.

MLA Handbook (7th Edition):

Guo, Xia. “Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.” 2014. Web. 23 Sep 2019.

Vancouver:

Guo X. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2019 Sep 23]. Available from: http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd.

Council of Science Editors:

Guo X. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd


University of Manchester

13. Gibbons, Claire. The cytoskeletal protein adducin and its role in vascular smooth muscle.

Degree: PhD, 2012, University of Manchester

 Actin dynamics are precisely regulated by a large number of actin binding proteins which collectively alter the rates of actin filament assembly and disassembly. Spectrin,… (more)

Subjects/Keywords: 612.7; Vascular smooth muscle; Adducin

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APA (6th Edition):

Gibbons, C. (2012). The cytoskeletal protein adducin and its role in vascular smooth muscle. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573

Chicago Manual of Style (16th Edition):

Gibbons, Claire. “The cytoskeletal protein adducin and its role in vascular smooth muscle.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573.

MLA Handbook (7th Edition):

Gibbons, Claire. “The cytoskeletal protein adducin and its role in vascular smooth muscle.” 2012. Web. 23 Sep 2019.

Vancouver:

Gibbons C. The cytoskeletal protein adducin and its role in vascular smooth muscle. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573.

Council of Science Editors:

Gibbons C. The cytoskeletal protein adducin and its role in vascular smooth muscle. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573


University of Lund

14. Shakirova, Yulia. Integration of signalling in smooth muscle caveolae.

Degree: 2010, University of Lund

 Caveolae and lipid rafts are plasma membrane microdomains that are considered to play a role in cellular signalling. Caveolins and cavins as well as cholesterol… (more)

Subjects/Keywords: Cell- och molekylärbiologi; Caveola; caveolin; detrusor; cavin; smooth muscle contractility; remodelling; NO; vascular dysfunction; smooth muscle; erectile dysfunction

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APA (6th Edition):

Shakirova, Y. (2010). Integration of signalling in smooth muscle caveolae. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/1716723 ; http://portal.research.lu.se/ws/files/3325600/1716728.pdf

Chicago Manual of Style (16th Edition):

Shakirova, Yulia. “Integration of signalling in smooth muscle caveolae.” 2010. Doctoral Dissertation, University of Lund. Accessed September 23, 2019. http://lup.lub.lu.se/record/1716723 ; http://portal.research.lu.se/ws/files/3325600/1716728.pdf.

MLA Handbook (7th Edition):

Shakirova, Yulia. “Integration of signalling in smooth muscle caveolae.” 2010. Web. 23 Sep 2019.

Vancouver:

Shakirova Y. Integration of signalling in smooth muscle caveolae. [Internet] [Doctoral dissertation]. University of Lund; 2010. [cited 2019 Sep 23]. Available from: http://lup.lub.lu.se/record/1716723 ; http://portal.research.lu.se/ws/files/3325600/1716728.pdf.

Council of Science Editors:

Shakirova Y. Integration of signalling in smooth muscle caveolae. [Doctoral Dissertation]. University of Lund; 2010. Available from: http://lup.lub.lu.se/record/1716723 ; http://portal.research.lu.se/ws/files/3325600/1716728.pdf


NSYSU

15. Chen, Yao-Chang. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.

Degree: PhD, Marine Biotechnology and Resources, 2015, NSYSU

 Arterial reconstruction procedures, including balloon angioplasty, stenting and coronary artery bypass, are used to restore blood flow in atherosclerotic arteries. Restenosis of these arteries is… (more)

Subjects/Keywords: vascular smooth muscle cell; dihydroaustrasulfone alcohol; proliferation; migration; restenosis; phenotypic modulation; cell cycle

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APA (6th Edition):

Chen, Y. (2015). The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046

Chicago Manual of Style (16th Edition):

Chen, Yao-Chang. “The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.” 2015. Doctoral Dissertation, NSYSU. Accessed September 23, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046.

MLA Handbook (7th Edition):

Chen, Yao-Chang. “The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.” 2015. Web. 23 Sep 2019.

Vancouver:

Chen Y. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2019 Sep 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046.

Council of Science Editors:

Chen Y. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046


University of Aberdeen

16. Whyte, Claire Susan. The effect of DHA and EPA on fibrosis-related factors in vascular cells.

Degree: 2009, University of Aberdeen

 Endothelial cells (ECs) and smooth muscle cell (SMC) play a key part during development of fibrosis in the intima being partly responsible for synthesis of… (more)

Subjects/Keywords: 616.1; Atherosclerosis : Vascular smooth muscle : Muscle, Smooth, Vascular

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APA (6th Edition):

Whyte, C. S. (2009). The effect of DHA and EPA on fibrosis-related factors in vascular cells. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25877 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499741

Chicago Manual of Style (16th Edition):

Whyte, Claire Susan. “The effect of DHA and EPA on fibrosis-related factors in vascular cells.” 2009. Doctoral Dissertation, University of Aberdeen. Accessed September 23, 2019. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25877 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499741.

MLA Handbook (7th Edition):

Whyte, Claire Susan. “The effect of DHA and EPA on fibrosis-related factors in vascular cells.” 2009. Web. 23 Sep 2019.

Vancouver:

Whyte CS. The effect of DHA and EPA on fibrosis-related factors in vascular cells. [Internet] [Doctoral dissertation]. University of Aberdeen; 2009. [cited 2019 Sep 23]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25877 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499741.

Council of Science Editors:

Whyte CS. The effect of DHA and EPA on fibrosis-related factors in vascular cells. [Doctoral Dissertation]. University of Aberdeen; 2009. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=25877 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499741


Dublin City University

17. Morrow, David. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.

Degree: School of Biotechnology, 2006, Dublin City University

Vascular Smooth Muscle Cell (SMC) fate decisions are fundamental features in the pathogenesis of vascular disease. We investigated the role of Notch 1 and 3… (more)

Subjects/Keywords: Biotechnology; Vascular Smooth Muscle Cell; Vascular Disease; Cell Growth

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APA (6th Edition):

Morrow, D. (2006). The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. (Thesis). Dublin City University. Retrieved from http://doras.dcu.ie/17705/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morrow, David. “The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.” 2006. Thesis, Dublin City University. Accessed September 23, 2019. http://doras.dcu.ie/17705/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morrow, David. “The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.” 2006. Web. 23 Sep 2019.

Vancouver:

Morrow D. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. [Internet] [Thesis]. Dublin City University; 2006. [cited 2019 Sep 23]. Available from: http://doras.dcu.ie/17705/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morrow D. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. [Thesis]. Dublin City University; 2006. Available from: http://doras.dcu.ie/17705/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

18. Bhattacharyya, Aparna. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.

Degree: 2012, University of Western Ontario

 One of the challenges in the designing of clinically-relevant vascular substitutes is our lack of understanding on how vascular smooth muscle cells (VSMCs) and vascular(more)

Subjects/Keywords: tissue engineering; vascular smooth muscle cell; vascular endothelial cell; phenotype modulation; polyurethane scaffolds; Notch signaling; Molecular, Cellular, and Tissue Engineering

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APA (6th Edition):

Bhattacharyya, A. (2012). Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhattacharyya, Aparna. “Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.” 2012. Thesis, University of Western Ontario. Accessed September 23, 2019. https://ir.lib.uwo.ca/etd/501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhattacharyya, Aparna. “Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.” 2012. Web. 23 Sep 2019.

Vancouver:

Bhattacharyya A. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2019 Sep 23]. Available from: https://ir.lib.uwo.ca/etd/501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhattacharyya A. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

19. Sommerville, Laura Jean. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.

Degree: PhD, 2010, Temple University

Molecular and Cellular Physiology

The underlying cause of all vascular proliferative diseases is injury-induced activation of vascular endothelium and vascular smooth muscle cells (VSMC). Activated… (more)

Subjects/Keywords: Biology, Physiology; Allograft Inflammatory Factor-1; Atherosclerosis; Smooth Muscle Cell Activation; Vascular Proliferative Disease

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APA (6th Edition):

Sommerville, L. J. (2010). The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,76756

Chicago Manual of Style (16th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Doctoral Dissertation, Temple University. Accessed September 23, 2019. http://digital.library.temple.edu/u?/p245801coll10,76756.

MLA Handbook (7th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Web. 23 Sep 2019.

Vancouver:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 23]. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756.

Council of Science Editors:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756

20. Κωστόπουλος, Χρήστος. Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου.

Degree: 2008, University of Patras

Τα αγγειακά λεία μυικά κύτταρα (ΑΛΜΚ) αποτελούν το κυρίαρχο στοιχείο του μέσου χιτώνα των αιμοφόρων αγγείων, ενώ συμμετέχουν ενεργά και στο σχηματισμό και την ωρίμανση… (more)

Subjects/Keywords: Αγγειακό λείο μυικό κύτταρο; Αθηροσκλήρωση; Καρδιαγγειακή νόσος; 616.13; Vascular smooth muscle cell; Atherosclerosis; Cardiovascular disease

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APA (6th Edition):

Κωστόπουλος, . (2008). Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου. (Masters Thesis). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/794

Chicago Manual of Style (16th Edition):

Κωστόπουλος, Χρήστος. “Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου.” 2008. Masters Thesis, University of Patras. Accessed September 23, 2019. http://nemertes.lis.upatras.gr/jspui/handle/10889/794.

MLA Handbook (7th Edition):

Κωστόπουλος, Χρήστος. “Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου.” 2008. Web. 23 Sep 2019.

Vancouver:

Κωστόπουλος . Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου. [Internet] [Masters thesis]. University of Patras; 2008. [cited 2019 Sep 23]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/794.

Council of Science Editors:

Κωστόπουλος . Το αγγειακό λείο μυϊκό κύτταρο : μοριακή δομή και ρόλος στην παθογένεια της καρδιαγγειακής νόσου. [Masters Thesis]. University of Patras; 2008. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/794


Clemson University

21. Mcallister, William. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.

Degree: MS, Bioengineering, 2011, Clemson University

 In this master's thesis we look at elucidating the interactions between nanoparticles and cells. Specifically, we looked at how the cell mechanics are affected, cytotoxicity… (more)

Subjects/Keywords: Atomic Force Microscopy; Cell Mechanics; Gold Nanoparticles; Vascular Smooth Muscle Cells; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Mcallister, W. (2011). Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/1101

Chicago Manual of Style (16th Edition):

Mcallister, William. “Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.” 2011. Masters Thesis, Clemson University. Accessed September 23, 2019. https://tigerprints.clemson.edu/all_theses/1101.

MLA Handbook (7th Edition):

Mcallister, William. “Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.” 2011. Web. 23 Sep 2019.

Vancouver:

Mcallister W. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. [Internet] [Masters thesis]. Clemson University; 2011. [cited 2019 Sep 23]. Available from: https://tigerprints.clemson.edu/all_theses/1101.

Council of Science Editors:

Mcallister W. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. [Masters Thesis]. Clemson University; 2011. Available from: https://tigerprints.clemson.edu/all_theses/1101


University of Toronto

22. Sattar, Joobin. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.

Degree: 2015, University of Toronto

RGS5 within vascular smooth muscle cells (VSMCs) is capable of inhibiting G-protein signaling involved in VSMC recruitment. Based on the suggested ability of statin to… (more)

Subjects/Keywords: Atherosclerosis; G-Protein Signaling; Neointimal Hyperplasia; Pleiotropic; Statins; Vascular Smooth Muscle Cell; 0719

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APA (6th Edition):

Sattar, J. (2015). Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70645

Chicago Manual of Style (16th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Masters Thesis, University of Toronto. Accessed September 23, 2019. http://hdl.handle.net/1807/70645.

MLA Handbook (7th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Web. 23 Sep 2019.

Vancouver:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1807/70645.

Council of Science Editors:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70645


Boston University

23. Rim, Nae Gyune. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.

Degree: PhD, Biomedical Engineering, 2018, Boston University

 To successfully develop a functional tissue-engineered vascular patch, recapitulating the hierarchical structure of vessel is critical to mimic mechanical properties. Here, we use a cell(more)

Subjects/Keywords: Biomedical engineering; Finite element modeling; Hydrogel; Tensile testing; Vascular smooth muscle cell

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APA (6th Edition):

Rim, N. G. (2018). Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/30707

Chicago Manual of Style (16th Edition):

Rim, Nae Gyune. “Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.” 2018. Doctoral Dissertation, Boston University. Accessed September 23, 2019. http://hdl.handle.net/2144/30707.

MLA Handbook (7th Edition):

Rim, Nae Gyune. “Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.” 2018. Web. 23 Sep 2019.

Vancouver:

Rim NG. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2144/30707.

Council of Science Editors:

Rim NG. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/30707


University of Hong Kong

24. 姚惠琪.; Yiu, Wai-ki. The effects of supercooling and re-warming on vascular cells survival and proliferation.

Degree: PhD, 2010, University of Hong Kong

published_or_final_version

Surgery

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Lui, VCH, Cheng, SWK.

Subjects/Keywords: Cell proliferation.; Vascular smooth muscle.; Vascular endothelium.

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APA (6th Edition):

姚惠琪.; Yiu, W. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312

Chicago Manual of Style (16th Edition):

姚惠琪.; Yiu, Wai-ki. “The effects of supercooling and re-warming on vascular cells survival and proliferation.” 2010. Doctoral Dissertation, University of Hong Kong. Accessed September 23, 2019. Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312.

MLA Handbook (7th Edition):

姚惠琪.; Yiu, Wai-ki. “The effects of supercooling and re-warming on vascular cells survival and proliferation.” 2010. Web. 23 Sep 2019.

Vancouver:

姚惠琪.; Yiu W. The effects of supercooling and re-warming on vascular cells survival and proliferation. [Internet] [Doctoral dissertation]. University of Hong Kong; 2010. [cited 2019 Sep 23]. Available from: Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312.

Council of Science Editors:

姚惠琪.; Yiu W. The effects of supercooling and re-warming on vascular cells survival and proliferation. [Doctoral Dissertation]. University of Hong Kong; 2010. Available from: Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312

25. Roszkowska, Monika. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.

Degree: Docteur es, Biochimie, 2018, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne)

Chez les patients atteints d'athérosclérose, les calcifications vasculaires sont une caracteristique des plaques d'athérome. Elles résultent de la trans-différenciation des cellules musculaires lisses (CMLs) en… (more)

Subjects/Keywords: Cellule musculaire lisse; Phosphatase alcaline; Calcification vasculaire; Chondrocyte; Vascular smooth muscle cell; Tissue-nonspecific alkaline phosphatase; Vascular calcification; Chondrocyte; 572

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APA (6th Edition):

Roszkowska, M. (2018). Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. (Doctoral Dissertation). Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Retrieved from http://www.theses.fr/2018LYSE1059

Chicago Manual of Style (16th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Doctoral Dissertation, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Accessed September 23, 2019. http://www.theses.fr/2018LYSE1059.

MLA Handbook (7th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Web. 23 Sep 2019.

Vancouver:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Internet] [Doctoral dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2018LYSE1059.

Council of Science Editors:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Doctoral Dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. Available from: http://www.theses.fr/2018LYSE1059


University of Pennsylvania

26. Yu, Christopher. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.

Degree: 2017, University of Pennsylvania

 Arterial stiffness is a risk factor for several cardiometabolic diseases and is caused by pathological remodeling of the vascular extracellular matrix (ECM). Vascular smooth muscle(more)

Subjects/Keywords: Extracellular matrix stiffness; Long non-coding RNAs; Transcriptome; Vascular smooth muscle cells; Vascular stiffness; Cell Biology; Genetics

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APA (6th Edition):

Yu, C. (2017). Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Christopher. “Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.” 2017. Thesis, University of Pennsylvania. Accessed September 23, 2019. https://repository.upenn.edu/edissertations/2710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Christopher. “Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.” 2017. Web. 23 Sep 2019.

Vancouver:

Yu C. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Sep 23]. Available from: https://repository.upenn.edu/edissertations/2710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu C. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Giot, Jean-Philippe. Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Poitiers

L'angiodermite nécrotique ou "Hypertensive Leg Ulcer" (HLU) est un ulcère de jambe inflammatoire de traitement difficile, associé à une hypertension artérielle chronique. La douleur est… (more)

Subjects/Keywords: Cytokines; Angiodermite nécrotique; Kératinocytes; Muscle lisse vasculaire; Douleur; Nécrose; Cytokines; Hypertensive leg ulcer; Keratinocytes; Vascular smooth muscle cell; Pain; Necrosis; 574.29

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APA (6th Edition):

Giot, J. (2013). Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2013POIT1406

Chicago Manual of Style (16th Edition):

Giot, Jean-Philippe. “Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer.” 2013. Doctoral Dissertation, Poitiers. Accessed September 23, 2019. http://www.theses.fr/2013POIT1406.

MLA Handbook (7th Edition):

Giot, Jean-Philippe. “Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer.” 2013. Web. 23 Sep 2019.

Vancouver:

Giot J. Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer. [Internet] [Doctoral dissertation]. Poitiers; 2013. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2013POIT1406.

Council of Science Editors:

Giot J. Implication des cytokines inflammatoires dans l'angiodermite nécrotique : Implication of inflammatory cytokines in hypertensive leg ulcer. [Doctoral Dissertation]. Poitiers; 2013. Available from: http://www.theses.fr/2013POIT1406


University of Vermont

28. Nystoriak, Matthew. Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles.

Degree: PhD, Pharmacology, 2010, University of Vermont

 Subarachnoid hemorrhage (SAH) following cerebral aneurysm rupture is associated with substantial morbidity and mortality. The ability of SAH to induce vasospasm in large diameter pial… (more)

Subjects/Keywords: Vascular Smooth Muscle; Endothelium; Cav1.2; Artery

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APA (6th Edition):

Nystoriak, M. (2010). Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/168

Chicago Manual of Style (16th Edition):

Nystoriak, Matthew. “Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles.” 2010. Doctoral Dissertation, University of Vermont. Accessed September 23, 2019. https://scholarworks.uvm.edu/graddis/168.

MLA Handbook (7th Edition):

Nystoriak, Matthew. “Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles.” 2010. Web. 23 Sep 2019.

Vancouver:

Nystoriak M. Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles. [Internet] [Doctoral dissertation]. University of Vermont; 2010. [cited 2019 Sep 23]. Available from: https://scholarworks.uvm.edu/graddis/168.

Council of Science Editors:

Nystoriak M. Role of Voltage-Dependent Calcium Channels in Subarachnoid Hemorrhage-Induced Constriction of Intracerebral Arterioles. [Doctoral Dissertation]. University of Vermont; 2010. Available from: https://scholarworks.uvm.edu/graddis/168


University of Aberdeen

29. Wirrig, Christiane. Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells.

Degree: 2012, University of Aberdeen

 Cardiovascular diseases are a major cause of death worldwide. Aneurysmal rupture in cerebral arteries or loss of endothelial integrity in the course of atherosclerosis or… (more)

Subjects/Keywords: 616.1; Vascular smooth muscle; Sphingolipids; Atherosclerosis

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APA (6th Edition):

Wirrig, C. (2012). Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=185829 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558636

Chicago Manual of Style (16th Edition):

Wirrig, Christiane. “Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells.” 2012. Doctoral Dissertation, University of Aberdeen. Accessed September 23, 2019. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=185829 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558636.

MLA Handbook (7th Edition):

Wirrig, Christiane. “Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells.” 2012. Web. 23 Sep 2019.

Vancouver:

Wirrig C. Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells. [Internet] [Doctoral dissertation]. University of Aberdeen; 2012. [cited 2019 Sep 23]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=185829 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558636.

Council of Science Editors:

Wirrig C. Effects of sphingolipids on the inflammatory reactivity of vascular smooth muscle cells. [Doctoral Dissertation]. University of Aberdeen; 2012. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=185829 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558636


Youngstown State University

30. Reed, Andraele N. Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats.

Degree: MSin Biology, Department of Biological Sciences, 2014, Youngstown State University

 Hypertension may be caused by excessive vasoconstriction that can occur through a variety of dysfunctions in cellular mechanisms. Estrogen has been postulated to have protective… (more)

Subjects/Keywords: Biology; hypertension; estrogen; vascular smooth muscle; relaxation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reed, A. N. (2014). Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats. (Masters Thesis). Youngstown State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ysu1407342968

Chicago Manual of Style (16th Edition):

Reed, Andraele N. “Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats.” 2014. Masters Thesis, Youngstown State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ysu1407342968.

MLA Handbook (7th Edition):

Reed, Andraele N. “Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats.” 2014. Web. 23 Sep 2019.

Vancouver:

Reed AN. Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats. [Internet] [Masters thesis]. Youngstown State University; 2014. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1407342968.

Council of Science Editors:

Reed AN. Regulation of Aortic Smooth Muscle Relaxation in Spontaneously Hypertensive Rats. [Masters Thesis]. Youngstown State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1407342968

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