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You searched for subject:(Vascular Smooth Muscle Cells VSMC ). Showing records 1 – 30 of 27319 total matches.

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1. Baeten, Jeremy T. The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells.

Degree: PhD, Biomedical Sciences, 2016, The Ohio State University

 The Notch signaling pathway has long been intricately linked with the development and function of the vasculature. In vascular smooth muscle cells (VSMCs), Notch signaling… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; Developmental Biology; Molecular Biology; Notch signaling; vascular smooth muscle cells; PDA; VSMC differentiation; miR145

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APA (6th Edition):

Baeten, J. T. (2016). The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1480513055823902

Chicago Manual of Style (16th Edition):

Baeten, Jeremy T. “The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells.” 2016. Doctoral Dissertation, The Ohio State University. Accessed August 07, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1480513055823902.

MLA Handbook (7th Edition):

Baeten, Jeremy T. “The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells.” 2016. Web. 07 Aug 2020.

Vancouver:

Baeten JT. The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2020 Aug 07]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1480513055823902.

Council of Science Editors:

Baeten JT. The Roles of the Notch2 and Notch3 Receptors in Vascular Smooth Muscle Cells. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1480513055823902


University of Georgia

2. Guo, Xia. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.

Degree: PhD, Physiology, 2014, University of Georgia

 The objective of this study was to develop a novel in vitro model for smooth muscle cell (SMC) differentiation from human embryonic stem cell-derived mesenchymal… (more)

Subjects/Keywords: Vascular smooth muscle cells

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APA (6th Edition):

Guo, X. (2014). Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd

Chicago Manual of Style (16th Edition):

Guo, Xia. “Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.” 2014. Doctoral Dissertation, University of Georgia. Accessed August 07, 2020. http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd.

MLA Handbook (7th Edition):

Guo, Xia. “Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling.” 2014. Web. 07 Aug 2020.

Vancouver:

Guo X. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2020 Aug 07]. Available from: http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd.

Council of Science Editors:

Guo X. Characterization of a novel in vitro model for smooth muscle differentiation and the role of dedicator of cytokinesis 2 in smooth muscle differentiation, phenotypic modulation, and vascular remodeling. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/guo_xia_201408_phd


Temple University

3. Cuneo, Anthony. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.

Degree: PhD, 2012, Temple University

Molecular and Cellular Physiology

Cardiovascular disease is the leading cause of mortality in the western world. The pro-inflammatory and pro-proliferative etiology of vascular proliferative diseases… (more)

Subjects/Keywords: Biology, Molecular; Biology, General; Biology, Cell; Atherosclerosis; Human antigen R (HuR); Interleukin-19 (IL-19); oxidized LDL (ox-LDL); Vascular Proliferative Diseases; Vascular Smooth Muscle Cells (VSMC)

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APA (6th Edition):

Cuneo, A. (2012). THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78032

Chicago Manual of Style (16th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Doctoral Dissertation, Temple University. Accessed August 07, 2020. http://digital.library.temple.edu/u?/p245801coll10,78032.

MLA Handbook (7th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Web. 07 Aug 2020.

Vancouver:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 07]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032.

Council of Science Editors:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032


University of Lund

4. Alajbegovic, Azra. Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states.

Degree: 2019, University of Lund

 As the world population is pushing toward 8 billion, cardiovascular diseases (CVD) remain the leading cause of death worldwide, representing 30% of all global deaths.… (more)

Subjects/Keywords: Cell and Molecular Biology; Cell Biology; Vascular smooth muscle cells (VSMC); cell phenotype; microRNA; Aneurysm; hypertension; Actin polymerization; GATA6; MRTFA; Vascular disease

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APA (6th Edition):

Alajbegovic, A. (2019). Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/c2560278-7d82-4418-81e5-6a43a3abf803 ; https://portal.research.lu.se/ws/files/69816700/Azra_Alajbegovic_WEBB.pdf

Chicago Manual of Style (16th Edition):

Alajbegovic, Azra. “Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states.” 2019. Doctoral Dissertation, University of Lund. Accessed August 07, 2020. https://lup.lub.lu.se/record/c2560278-7d82-4418-81e5-6a43a3abf803 ; https://portal.research.lu.se/ws/files/69816700/Azra_Alajbegovic_WEBB.pdf.

MLA Handbook (7th Edition):

Alajbegovic, Azra. “Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states.” 2019. Web. 07 Aug 2020.

Vancouver:

Alajbegovic A. Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states. [Internet] [Doctoral dissertation]. University of Lund; 2019. [cited 2020 Aug 07]. Available from: https://lup.lub.lu.se/record/c2560278-7d82-4418-81e5-6a43a3abf803 ; https://portal.research.lu.se/ws/files/69816700/Azra_Alajbegovic_WEBB.pdf.

Council of Science Editors:

Alajbegovic A. Transcriptional and post-transcriptional regulation of vascular smooth muscle cell phenotype - Implications for vascular disease states. [Doctoral Dissertation]. University of Lund; 2019. Available from: https://lup.lub.lu.se/record/c2560278-7d82-4418-81e5-6a43a3abf803 ; https://portal.research.lu.se/ws/files/69816700/Azra_Alajbegovic_WEBB.pdf

5. Willems, Brecht Andy Ghislaine. Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms.

Degree: 2017, Maastricht University

 This dissertation examines the role of smooth muscle cells (VSMC) in the development of vascular calcification and atherosclerosis. Smoking causes a significant increase in microcalcification,… (more)

Subjects/Keywords: smooth muscle cells (VSMC); vascular calcification; atherosclerosis; Ucma/GRP; vitamin K

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APA (6th Edition):

Willems, B. A. G. (2017). Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms. (Doctoral Dissertation). Maastricht University. Retrieved from https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; b75a00f0-3765-4453-9f5a-84441bd46e83 ; 10.26481/dis.20171201bw ; urn:isbn:9789462957671 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83

Chicago Manual of Style (16th Edition):

Willems, Brecht Andy Ghislaine. “Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms.” 2017. Doctoral Dissertation, Maastricht University. Accessed August 07, 2020. https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; b75a00f0-3765-4453-9f5a-84441bd46e83 ; 10.26481/dis.20171201bw ; urn:isbn:9789462957671 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83.

MLA Handbook (7th Edition):

Willems, Brecht Andy Ghislaine. “Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms.” 2017. Web. 07 Aug 2020.

Vancouver:

Willems BAG. Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms. [Internet] [Doctoral dissertation]. Maastricht University; 2017. [cited 2020 Aug 07]. Available from: https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; b75a00f0-3765-4453-9f5a-84441bd46e83 ; 10.26481/dis.20171201bw ; urn:isbn:9789462957671 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83.

Council of Science Editors:

Willems BAG. Vascular smooth muscle cell phenotype switching governs vascular calcification: Role of harnessing endogenous protective mechanisms. [Doctoral Dissertation]. Maastricht University; 2017. Available from: https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; b75a00f0-3765-4453-9f5a-84441bd46e83 ; 10.26481/dis.20171201bw ; urn:isbn:9789462957671 ; urn:nbn:nl:ui:27-b75a00f0-3765-4453-9f5a-84441bd46e83 ; https://cris.maastrichtuniversity.nl/en/publications/b75a00f0-3765-4453-9f5a-84441bd46e83


University of New South Wales

6. Li, Yue. Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening.

Degree: Centre for Vascular Research, 2013, University of New South Wales

 Coronary artery disease (CAD), underpinned by atherosclerosis, remains a leading cause of morbidity and mortality, particularly in the Western World. Although the advent of percutaneous… (more)

Subjects/Keywords: Vascular smooth muscle cells; Neointimal hyperplasia

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APA (6th Edition):

Li, Y. (2013). Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52997 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11675/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Li, Yue. “Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening.” 2013. Doctoral Dissertation, University of New South Wales. Accessed August 07, 2020. http://handle.unsw.edu.au/1959.4/52997 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11675/SOURCE01?view=true.

MLA Handbook (7th Edition):

Li, Yue. “Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening.” 2013. Web. 07 Aug 2020.

Vancouver:

Li Y. Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening. [Internet] [Doctoral dissertation]. University of New South Wales; 2013. [cited 2020 Aug 07]. Available from: http://handle.unsw.edu.au/1959.4/52997 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11675/SOURCE01?view=true.

Council of Science Editors:

Li Y. Novel strategies to inhibit smooth muscle cell hyperplasia and intimal thickening. [Doctoral Dissertation]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52997 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11675/SOURCE01?view=true


University of Cambridge

7. Reinhold, Johannes. Mitochondrial function in atherosclerosis and vascular smooth muscle cells.

Degree: PhD, 2019, University of Cambridge

 Atherosclerosis is the leading cause of death in the Western world. Although mitochondrial DNA (mtDNA) damage has been implicated in atherosclerosis, it is unclear whether… (more)

Subjects/Keywords: Atherosclerosis; Mitochondria; vascular smooth muscle cells

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APA (6th Edition):

Reinhold, J. (2019). Mitochondrial function in atherosclerosis and vascular smooth muscle cells. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/288352 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763838

Chicago Manual of Style (16th Edition):

Reinhold, Johannes. “Mitochondrial function in atherosclerosis and vascular smooth muscle cells.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/288352 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763838.

MLA Handbook (7th Edition):

Reinhold, Johannes. “Mitochondrial function in atherosclerosis and vascular smooth muscle cells.” 2019. Web. 07 Aug 2020.

Vancouver:

Reinhold J. Mitochondrial function in atherosclerosis and vascular smooth muscle cells. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/288352 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763838.

Council of Science Editors:

Reinhold J. Mitochondrial function in atherosclerosis and vascular smooth muscle cells. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/288352 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763838


University of Manchester

8. Losa Llabata, Marta. Gene regulation in embryonic development.

Degree: 2016, University of Manchester

 Branchial arches (BAs) are a series of transient structures that develop on the ventro-lateral surface of the head in vertebrate embryos. BAs initially appear as… (more)

Subjects/Keywords: branchial arch; neural crest cells; aortic arch; vascular smooth muscle cells

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APA (6th Edition):

Losa Llabata, M. (2016). Gene regulation in embryonic development. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:295949

Chicago Manual of Style (16th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Doctoral Dissertation, University of Manchester. Accessed August 07, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:295949.

MLA Handbook (7th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Web. 07 Aug 2020.

Vancouver:

Losa Llabata M. Gene regulation in embryonic development. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Aug 07]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:295949.

Council of Science Editors:

Losa Llabata M. Gene regulation in embryonic development. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:295949

9. Nakagaki Silva, Erick Eidy. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.

Degree: PhD, 2020, University of Cambridge

 Alternative splicing (AS) is primarily regulated by regulatory RNA-binding proteins (RBPs). It has been suggested that a small number of master splicing regulators might control… (more)

Subjects/Keywords: RBPMS; Vascular smooth muscle cells; Alternative splicing; PAC1 cells

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APA (6th Edition):

Nakagaki Silva, E. E. (2020). Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/297676

Chicago Manual of Style (16th Edition):

Nakagaki Silva, Erick Eidy. “Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.” 2020. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/297676.

MLA Handbook (7th Edition):

Nakagaki Silva, Erick Eidy. “Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.” 2020. Web. 07 Aug 2020.

Vancouver:

Nakagaki Silva EE. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/297676.

Council of Science Editors:

Nakagaki Silva EE. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/297676


University of Cambridge

10. Nakagaki Silva, Erick Eidy. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.

Degree: PhD, 2019, University of Cambridge

 Alternative splicing (AS) is primarily regulated by regulatory RNA-binding proteins (RBPs). It has been suggested that a small number of master splicing regulators might control… (more)

Subjects/Keywords: RBPMS; Vascular smooth muscle cells; Alternative splicing; PAC1 cells

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APA (6th Edition):

Nakagaki Silva, E. E. (2019). Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/297676 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.787875

Chicago Manual of Style (16th Edition):

Nakagaki Silva, Erick Eidy. “Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/297676 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.787875.

MLA Handbook (7th Edition):

Nakagaki Silva, Erick Eidy. “Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers.” 2019. Web. 07 Aug 2020.

Vancouver:

Nakagaki Silva EE. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/297676 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.787875.

Council of Science Editors:

Nakagaki Silva EE. Identification of RBPMS as a smooth muscle master splicing regulator via association of its gene with super-enhancers. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/297676 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.787875


University of Vienna

11. Czaloun, Christa. Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration.

Degree: 2010, University of Vienna

Atherosklerose und Restenose gehören zu den vasoproliferativen Erkrankungen, die sich durch eine Anhäufung von glatten Gefäßmuskelzellen in der Intima auszeichnen. Außerdem produzieren diese glatten Gefäßmuskelzellen… (more)

Subjects/Keywords: 44.38 Pharmakologie; 30.30 Naturwissenschaften in Beziehung zu anderen Fachgebieten; Indirubin-3´-monoxime / I3MO / Platelet derived growth factor / PDGF / Migration / glatte Gefäßmuskelzellen; Indirubin-3´-monoxime / I3MO / platelet derived growth factor / PDGF / migration / vascular smooth muscle cells / VSMC

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APA (6th Edition):

Czaloun, C. (2010). Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/10372/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Czaloun, Christa. “Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration.” 2010. Thesis, University of Vienna. Accessed August 07, 2020. http://othes.univie.ac.at/10372/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Czaloun, Christa. “Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration.” 2010. Web. 07 Aug 2020.

Vancouver:

Czaloun C. Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration. [Internet] [Thesis]. University of Vienna; 2010. [cited 2020 Aug 07]. Available from: http://othes.univie.ac.at/10372/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Czaloun C. Effect of Indirubin-3´-monoxime on platelet derived growth factor-induced vascular smooth muscle cell migration. [Thesis]. University of Vienna; 2010. Available from: http://othes.univie.ac.at/10372/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. MATHIEU, PATTIE SMITH. Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations.

Degree: School of Engineering. Discipline of Mechanical & Manuf. Eng, 2020, Trinity College Dublin

 Atherosclerosis is one of the leading causes of mortality worldwide, and presents as a narrowing or occlusion of an artery. Interventions to re-open the arterial… (more)

Subjects/Keywords: Multipotent Vascular Stem Cells; Vascular Smooth Muscle Cells; Tensile Strain; Extracellular Matrix; In-Stent Restenosis

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APA (6th Edition):

MATHIEU, P. S. (2020). Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/92298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MATHIEU, PATTIE SMITH. “Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations.” 2020. Thesis, Trinity College Dublin. Accessed August 07, 2020. http://hdl.handle.net/2262/92298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MATHIEU, PATTIE SMITH. “Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations.” 2020. Web. 07 Aug 2020.

Vancouver:

MATHIEU PS. Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations. [Internet] [Thesis]. Trinity College Dublin; 2020. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/2262/92298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MATHIEU PS. Multipotent vascular stem cells and the effects of cyclic tensile strain, collagen structure and stenting on medial vascular cell populations. [Thesis]. Trinity College Dublin; 2020. Available from: http://hdl.handle.net/2262/92298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

13. Bulick, Allen. Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering.

Degree: 2010, Texas A&M University

 The research presented here deals with synthetic materials for application in tissue engineering, primarily poly(ethylene glycol) (PEG) and poly(dimethyl siloxane)star (PDMS)star. Tissue engineering seeks to… (more)

Subjects/Keywords: Tissue Engineering; Smooth Muscle Cells; Vocal Fold; Vascular Grafts

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APA (6th Edition):

Bulick, A. (2010). Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2009-08-3249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bulick, Allen. “Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering.” 2010. Thesis, Texas A&M University. Accessed August 07, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2009-08-3249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bulick, Allen. “Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering.” 2010. Web. 07 Aug 2020.

Vancouver:

Bulick A. Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering. [Internet] [Thesis]. Texas A&M University; 2010. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2009-08-3249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bulick A. Systematic Investigation of Hydrogel Material Properties on Cell Responses for Vocal Fold and Vascular Graft Tissue Engineering. [Thesis]. Texas A&M University; 2010. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2009-08-3249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

14. Hedley, Thomas Elliot. The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development.

Degree: Physiology and Pathophysiology, 2017, University of Manitoba

 This study investigated whether heat shock protein-60 (Hsp60) modulates nuclear protein import (NPI) to promote vascular smooth muscle cell (VSMC) proliferation and atherosclerotic development. Rat… (more)

Subjects/Keywords: Heat shock protein-60; Atherosclerosis; Vascular smooth muscle cells

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APA (6th Edition):

Hedley, T. E. (2017). The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32376

Chicago Manual of Style (16th Edition):

Hedley, Thomas Elliot. “The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development.” 2017. Masters Thesis, University of Manitoba. Accessed August 07, 2020. http://hdl.handle.net/1993/32376.

MLA Handbook (7th Edition):

Hedley, Thomas Elliot. “The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development.” 2017. Web. 07 Aug 2020.

Vancouver:

Hedley TE. The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1993/32376.

Council of Science Editors:

Hedley TE. The role of heat shock protein-60 in vascular smooth muscle cell proliferation and atherosclerotic development. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32376


University of Sydney

15. Manuneedhi Cholan, Pradeep. TRAIL: A novel atheroprotective mechanism in the vasculature .

Degree: 2019, University of Sydney

 The vasculature is critical for the maintenance of cardiovascular homeostasis. Cardiovascular disease (CVD) is characterised by endothelial cell (EC) and vascular smooth muscle cell (VSMC)… (more)

Subjects/Keywords: TRAIL; CVD; endothelium; vascular smooth muscle cells; Angiotensin II

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APA (6th Edition):

Manuneedhi Cholan, P. (2019). TRAIL: A novel atheroprotective mechanism in the vasculature . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manuneedhi Cholan, Pradeep. “TRAIL: A novel atheroprotective mechanism in the vasculature .” 2019. Thesis, University of Sydney. Accessed August 07, 2020. http://hdl.handle.net/2123/20949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manuneedhi Cholan, Pradeep. “TRAIL: A novel atheroprotective mechanism in the vasculature .” 2019. Web. 07 Aug 2020.

Vancouver:

Manuneedhi Cholan P. TRAIL: A novel atheroprotective mechanism in the vasculature . [Internet] [Thesis]. University of Sydney; 2019. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/2123/20949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manuneedhi Cholan P. TRAIL: A novel atheroprotective mechanism in the vasculature . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

16. Dobnikar, Lina. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.

Degree: PhD, 2020, University of Cambridge

Vascular smooth muscle cells (VSMCs) possess a remarkable capacity to change phenotype in response to injury or inflammation. In healthy arteries, VSMCs exist in a… (more)

Subjects/Keywords: single-cell RNA-seq; vascular smooth muscle cells; cardiovascular

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APA (6th Edition):

Dobnikar, L. (2020). A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301990

Chicago Manual of Style (16th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/301990.

MLA Handbook (7th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Web. 07 Aug 2020.

Vancouver:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/301990.

Council of Science Editors:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301990


University of Minnesota

17. Steucke, Kerianne. Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation.

Degree: PhD, Biomedical Engineering, 2017, University of Minnesota

 Patient-specific vascular modeling seeks to provide physicians with predictive data that will enable them to make better treatment decisions and improve patient outcomes. To achieve… (more)

Subjects/Keywords: Biomechanics; Cardiovascular Disease; Mechano-Adaptation; Vascular Smooth Muscle Cells

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APA (6th Edition):

Steucke, K. (2017). Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/201104

Chicago Manual of Style (16th Edition):

Steucke, Kerianne. “Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation.” 2017. Doctoral Dissertation, University of Minnesota. Accessed August 07, 2020. http://hdl.handle.net/11299/201104.

MLA Handbook (7th Edition):

Steucke, Kerianne. “Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation.” 2017. Web. 07 Aug 2020.

Vancouver:

Steucke K. Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/11299/201104.

Council of Science Editors:

Steucke K. Empirical Determination of Vascular Smooth Muscle Cell Mechano-Adaptation. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/201104


Clemson University

18. Bradley, Suzanne Nicole. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.

Degree: MS, Bioengineering, 2018, Clemson University

Vascular smooth muscle cells regulate blood flow by contracting and relaxing blood vessels. Vascular smooth muscle cells display one of two distinct phenotypes: contractile or… (more)

Subjects/Keywords: Cell Stretching; Micropatterning; Microstamping; Vascular Smooth Muscle Cells

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APA (6th Edition):

Bradley, S. N. (2018). Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/2957

Chicago Manual of Style (16th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Masters Thesis, Clemson University. Accessed August 07, 2020. https://tigerprints.clemson.edu/all_theses/2957.

MLA Handbook (7th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Web. 07 Aug 2020.

Vancouver:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Internet] [Masters thesis]. Clemson University; 2018. [cited 2020 Aug 07]. Available from: https://tigerprints.clemson.edu/all_theses/2957.

Council of Science Editors:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Masters Thesis]. Clemson University; 2018. Available from: https://tigerprints.clemson.edu/all_theses/2957


University of Cambridge

19. Dobnikar, Lina. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.

Degree: PhD, 2020, University of Cambridge

Vascular smooth muscle cells (VSMCs) possess a remarkable capacity to change phenotype in response to injury or inflammation. In healthy arteries, VSMCs exist in a… (more)

Subjects/Keywords: single-cell RNA-seq; vascular smooth muscle cells; cardiovascular

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APA (6th Edition):

Dobnikar, L. (2020). A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760

Chicago Manual of Style (16th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760.

MLA Handbook (7th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Web. 07 Aug 2020.

Vancouver:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760.

Council of Science Editors:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760

20. F. Elli. MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT.

Degree: 2014, Università degli Studi di Milano

Vascular calcification is a significant contributor to cardiovascular risk in chronic kidney disease (CKD) patients, and its extent and severity has been correlated with mortality… (more)

Subjects/Keywords: Vascular calcification, Chronic Kidney Disease, Vascular Smooth Muscle Cells; Settore MED/14 - Nefrologia

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APA (6th Edition):

Elli, F. (2014). MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/245812

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elli, F.. “MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT.” 2014. Thesis, Università degli Studi di Milano. Accessed August 07, 2020. http://hdl.handle.net/2434/245812.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elli, F.. “MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT.” 2014. Web. 07 Aug 2020.

Vancouver:

Elli F. MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/2434/245812.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elli F. MOLECULAR AND CELLULAR MECHANISMS OF VASCULAR CALCIFICATION: PATHOGENESIS AND TREATMENT. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/245812

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

21. Gibbons, Claire. The cytoskeletal protein adducin and its role in vascular smooth muscle.

Degree: PhD, 2012, University of Manchester

 Actin dynamics are precisely regulated by a large number of actin binding proteins which collectively alter the rates of actin filament assembly and disassembly. Spectrin,… (more)

Subjects/Keywords: 612.7; Vascular smooth muscle; Adducin

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APA (6th Edition):

Gibbons, C. (2012). The cytoskeletal protein adducin and its role in vascular smooth muscle. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573

Chicago Manual of Style (16th Edition):

Gibbons, Claire. “The cytoskeletal protein adducin and its role in vascular smooth muscle.” 2012. Doctoral Dissertation, University of Manchester. Accessed August 07, 2020. https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573.

MLA Handbook (7th Edition):

Gibbons, Claire. “The cytoskeletal protein adducin and its role in vascular smooth muscle.” 2012. Web. 07 Aug 2020.

Vancouver:

Gibbons C. The cytoskeletal protein adducin and its role in vascular smooth muscle. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Aug 07]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573.

Council of Science Editors:

Gibbons C. The cytoskeletal protein adducin and its role in vascular smooth muscle. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-cytoskeletal-protein-adducin-and-its-role-in-vascular-smooth-muscle(66d8e7a0-68fa-41d4-91fc-a249c9a4331e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555573


University of Western Ontario

22. Wallace, Drew C. Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division.

Degree: 2016, University of Western Ontario

 Components of G protein-mediated signaling are associated with positioning and orienting the mitotic spindle in the process of cell division. However, a functional role for… (more)

Subjects/Keywords: G protein-mediated signaling; G protein signaling modulator 3 (GPSM3); cell division; proliferation; mitotic spindle; vascular smooth muscle cell (VSMC); Cell Biology; Developmental Biology

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APA (6th Edition):

Wallace, D. C. (2016). Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wallace, Drew C. “Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division.” 2016. Thesis, University of Western Ontario. Accessed August 07, 2020. https://ir.lib.uwo.ca/etd/4279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wallace, Drew C. “Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division.” 2016. Web. 07 Aug 2020.

Vancouver:

Wallace DC. Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Aug 07]. Available from: https://ir.lib.uwo.ca/etd/4279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wallace DC. Evidence of a Role for G Protein Signaling Modulator 3 in Cell Division. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

23. Losa Llabata, Marta. Gene regulation in embryonic development.

Degree: PhD, 2016, University of Manchester

 Branchial arches (BAs) are a series of transient structures that develop on the ventro-lateral surface of the head in vertebrate embryos. BAs initially appear as… (more)

Subjects/Keywords: 612.6; branchial arch; neural crest cells; aortic arch; vascular smooth muscle cells

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APA (6th Edition):

Losa Llabata, M. (2016). Gene regulation in embryonic development. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005

Chicago Manual of Style (16th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Doctoral Dissertation, University of Manchester. Accessed August 07, 2020. https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005.

MLA Handbook (7th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Web. 07 Aug 2020.

Vancouver:

Losa Llabata M. Gene regulation in embryonic development. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Aug 07]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005.

Council of Science Editors:

Losa Llabata M. Gene regulation in embryonic development. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005


University of Vermont

24. Manuelyan, Inessa. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.

Degree: Pharmacology, 2014, University of Vermont

  Research in the past on transient receptor potential cation channel subfamily V member 1 (TRPV1) has been limited to mainly nervous tissue TRPV1 because… (more)

Subjects/Keywords: Middle Meningeal Artery; Vascular Smooth Muscle TRPV1; TRPV1 mediated vasoconstriction; TRPV1 in smooth muscle cells; TRPV1-mediated mechanism of vasoconstriction

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APA (6th Edition):

Manuelyan, I. (2014). Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manuelyan, Inessa. “Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.” 2014. Thesis, University of Vermont. Accessed August 07, 2020. https://scholarworks.uvm.edu/hcoltheses/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manuelyan, Inessa. “Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.” 2014. Web. 07 Aug 2020.

Vancouver:

Manuelyan I. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. [Internet] [Thesis]. University of Vermont; 2014. [cited 2020 Aug 07]. Available from: https://scholarworks.uvm.edu/hcoltheses/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manuelyan I. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. [Thesis]. University of Vermont; 2014. Available from: https://scholarworks.uvm.edu/hcoltheses/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

25. Schwarz, Margarethe. Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen.

Degree: 2006, Johannes Gutenberg Universität Mainz

Apolipoprotein J (ApoJ) ist ein heterodimeres sekretiertes Glycoprotein, welchem sowohl antiapoptotische als auch antiinflammatorische Eigenschaften zugeschrieben werden. Es wird unter vielen pathophysiologischen Zuständen verstärkt exprimiert.… (more)

Subjects/Keywords: Apolipoprotein J (ApoJ), Atherosklerose, Toll-like Rezeptor 3 (TLR3), glatte Gefäßmuskelzellen (VSMC); Apolipoprotein J (ApoJ), atherosclerosis, Toll-like receptor 3 (TLR3), vascular smooth muscle cells (VSMC); Life sciences

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APA (6th Edition):

Schwarz, M. (2006). Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2007/1273/

Chicago Manual of Style (16th Edition):

Schwarz, Margarethe. “Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen.” 2006. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed August 07, 2020. http://ubm.opus.hbz-nrw.de/volltexte/2007/1273/.

MLA Handbook (7th Edition):

Schwarz, Margarethe. “Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen.” 2006. Web. 07 Aug 2020.

Vancouver:

Schwarz M. Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2006. [cited 2020 Aug 07]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1273/.

Council of Science Editors:

Schwarz M. Bedeutung und Regulation von Apolipoprotein J in atherosklerotischen Läsionen. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2006. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1273/


University of Cambridge

26. Harman, Jennifer. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.

Degree: PhD, 2019, University of Cambridge

 Widespread changes in gene expression accompany vascular smooth muscle cell (VSMC) phenotypic switching, a hallmark of vascular disease. Upon insult, VSMCs downregulate contractile proteins and… (more)

Subjects/Keywords: Vascular smooth muscle cell; H3K9me2; Vascular smooth muscle cell phenotypic switch

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harman, J. (2019). Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289975

Chicago Manual of Style (16th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 07, 2020. https://www.repository.cam.ac.uk/handle/1810/289975.

MLA Handbook (7th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Web. 07 Aug 2020.

Vancouver:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Aug 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/289975.

Council of Science Editors:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289975


University of Toronto

27. Xu, Songyi. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.

Degree: PhD, 2020, University of Toronto

 N-cadherin mediates cell-cell contacts in vascular smooth muscle cells (VSMCs) and regulates VSMC behaviours. Discoidin domain receptor 1 (DDR1) is a collagen binding receptor also… (more)

Subjects/Keywords: Cell-cell interaction; DDR1; N-cadherin; Rho GTPases; Vascular smooth muscle cells; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xu, S. (2020). N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/101328

Chicago Manual of Style (16th Edition):

Xu, Songyi. “N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.” 2020. Doctoral Dissertation, University of Toronto. Accessed August 07, 2020. http://hdl.handle.net/1807/101328.

MLA Handbook (7th Edition):

Xu, Songyi. “N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.” 2020. Web. 07 Aug 2020.

Vancouver:

Xu S. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. [Internet] [Doctoral dissertation]. University of Toronto; 2020. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1807/101328.

Council of Science Editors:

Xu S. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. [Doctoral Dissertation]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/101328

28. Μίχας, Γεώργιος. Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 The actions of glucocorticoids are mediated, in part, by 11β-hydroxysteroid dehydrogenase 1 (11β-HSD1), which amplifies their effects at the pre-receptor level by converting cortisone to… (more)

Subjects/Keywords: 11β - HSDI; Γλυκοκορτικοειδή; Λεία μυϊκά κύτταρα; Αγγεία; Glycocorticoids; Vascular smooth muscle cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Μίχας, . . (2011). Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/26673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Μίχας, Γεώργιος. “Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed August 07, 2020. http://hdl.handle.net/10442/hedi/26673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Μίχας, Γεώργιος. “Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells.” 2011. Web. 07 Aug 2020.

Vancouver:

Μίχας . Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/10442/hedi/26673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Μίχας . Effects of glucocorticoids and of 11β - hydroxysteroid dehydrogenase type I on vascular smooth muscle cells. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/26673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Delft University of Technology

29. Korevaar, Iris (author). Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels.

Degree: 2019, Delft University of Technology

 Worldwide 8-16% of the people are affected by chronic kidney disease (CKD). If the CKD is undetected, people have the risk to develop progressive loss… (more)

Subjects/Keywords: Vascular Smooth Muscle Cells; Arteriovenous fistula; Shear Stress; Flow experiments; IBIDI flow culturing system

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APA (6th Edition):

Korevaar, I. (. (2019). Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels. (Masters Thesis). Delft University of Technology. Retrieved from http://resolver.tudelft.nl/uuid:66a907eb-64de-41ed-a86d-9fbae24704ca

Chicago Manual of Style (16th Edition):

Korevaar, Iris (author). “Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels.” 2019. Masters Thesis, Delft University of Technology. Accessed August 07, 2020. http://resolver.tudelft.nl/uuid:66a907eb-64de-41ed-a86d-9fbae24704ca.

MLA Handbook (7th Edition):

Korevaar, Iris (author). “Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels.” 2019. Web. 07 Aug 2020.

Vancouver:

Korevaar I(. Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels. [Internet] [Masters thesis]. Delft University of Technology; 2019. [cited 2020 Aug 07]. Available from: http://resolver.tudelft.nl/uuid:66a907eb-64de-41ed-a86d-9fbae24704ca.

Council of Science Editors:

Korevaar I(. Assesing the response of arterial and venous smooth muscle cells to varying shear stress levels. [Masters Thesis]. Delft University of Technology; 2019. Available from: http://resolver.tudelft.nl/uuid:66a907eb-64de-41ed-a86d-9fbae24704ca


Clemson University

30. Kieu, Tri. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.

Degree: MS, Bioengineering, 2013, Clemson University

  The field of nanotechnology research has seen a large growth in the past few decades due to the great potential of novel nano-size material… (more)

Subjects/Keywords: Cytotoxicity; Gold Nanoparticle; Mechanic Properties; Vascular Smooth Muscle Cells; Biomedical Engineering and Bioengineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kieu, T. (2013). The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/1821

Chicago Manual of Style (16th Edition):

Kieu, Tri. “The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.” 2013. Masters Thesis, Clemson University. Accessed August 07, 2020. https://tigerprints.clemson.edu/all_theses/1821.

MLA Handbook (7th Edition):

Kieu, Tri. “The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.” 2013. Web. 07 Aug 2020.

Vancouver:

Kieu T. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. [Internet] [Masters thesis]. Clemson University; 2013. [cited 2020 Aug 07]. Available from: https://tigerprints.clemson.edu/all_theses/1821.

Council of Science Editors:

Kieu T. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. [Masters Thesis]. Clemson University; 2013. Available from: https://tigerprints.clemson.edu/all_theses/1821

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