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You searched for subject:(VEGFA). Showing records 1 – 12 of 12 total matches.

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Kansas State University

1. McFee, Renee Marie. The role of vascular endothelial growth factor isoforms in early follicle development.

Degree: MS, Department of Animal Sciences and Industry, 2012, Kansas State University

 Since vascularization of the theca layer increases as follicles progress in size through preantral and antral stages, the principal angiogenic factor, vascular endothelial growth factor… (more)

Subjects/Keywords: Ovary; Follicle; VEGFA; Isoforms; Physiology (0719)

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APA (6th Edition):

McFee, R. M. (2012). The role of vascular endothelial growth factor isoforms in early follicle development. (Masters Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/13525

Chicago Manual of Style (16th Edition):

McFee, Renee Marie. “The role of vascular endothelial growth factor isoforms in early follicle development.” 2012. Masters Thesis, Kansas State University. Accessed November 15, 2019. http://hdl.handle.net/2097/13525.

MLA Handbook (7th Edition):

McFee, Renee Marie. “The role of vascular endothelial growth factor isoforms in early follicle development.” 2012. Web. 15 Nov 2019.

Vancouver:

McFee RM. The role of vascular endothelial growth factor isoforms in early follicle development. [Internet] [Masters thesis]. Kansas State University; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2097/13525.

Council of Science Editors:

McFee RM. The role of vascular endothelial growth factor isoforms in early follicle development. [Masters Thesis]. Kansas State University; 2012. Available from: http://hdl.handle.net/2097/13525


Universidade Estadual de Campinas

2. Palodetto, Bruna, 1987-. Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas .

Degree: 2016, Universidade Estadual de Campinas

 Resumo: A interação entre as células-tronco hematopoéticas (HSCs) e o microambiente da medula óssea (MO) é essencial para a regulação da hematopoese; e a interação… (more)

Subjects/Keywords: Leucemia; Proteína VEGFA humana; Proteína SEMA3A humana

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APA (6th Edition):

Palodetto, Bruna, 1. (2016). Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas . (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/313056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Palodetto, Bruna, 1987-. “Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas .” 2016. Thesis, Universidade Estadual de Campinas. Accessed November 15, 2019. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Palodetto, Bruna, 1987-. “Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas .” 2016. Web. 15 Nov 2019.

Vancouver:

Palodetto, Bruna 1. Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas . [Internet] [Thesis]. Universidade Estadual de Campinas; 2016. [cited 2019 Nov 15]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Palodetto, Bruna 1. Influência de VEGFA e SEMA3A na patogênese de síndromes mielodisplásicas e leucemias agudas . [Thesis]. Universidade Estadual de Campinas; 2016. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

3. Li, Han. Role of Spongiotrophoblast VEGFA during Pregnancy.

Degree: 2012, University of Toronto

The role of placental VEGFA in placental development and maternal pregnancy adaptations is unknown. In this thesis, one or both copies of Vegfa was knocked… (more)

Subjects/Keywords: VEGFA; spongiotrophoblast; placenta; pregnancy; cardiovascular; 0719

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APA (6th Edition):

Li, H. (2012). Role of Spongiotrophoblast VEGFA during Pregnancy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42892

Chicago Manual of Style (16th Edition):

Li, Han. “Role of Spongiotrophoblast VEGFA during Pregnancy.” 2012. Masters Thesis, University of Toronto. Accessed November 15, 2019. http://hdl.handle.net/1807/42892.

MLA Handbook (7th Edition):

Li, Han. “Role of Spongiotrophoblast VEGFA during Pregnancy.” 2012. Web. 15 Nov 2019.

Vancouver:

Li H. Role of Spongiotrophoblast VEGFA during Pregnancy. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1807/42892.

Council of Science Editors:

Li H. Role of Spongiotrophoblast VEGFA during Pregnancy. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/42892

4. Fraineau, Sylvain. Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Poitiers

L'angiogenèse est un processus physiologique qui correspond à la formation de nouveaux vaisseaux sanguins à partir d'un réseau vasculaire préexistant et est régulée par l'équilibre… (more)

Subjects/Keywords: Protéine S; Angiogenèse; Prolifération; Migration; Voies de signalisation; Vegfa; Protein S; Angiogenesis; Proliferation; Migration; Signaling pathways; Vegfa; 571.5

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APA (6th Edition):

Fraineau, S. (2012). Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2012POIT2307

Chicago Manual of Style (16th Edition):

Fraineau, Sylvain. “Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process.” 2012. Doctoral Dissertation, Poitiers. Accessed November 15, 2019. http://www.theses.fr/2012POIT2307.

MLA Handbook (7th Edition):

Fraineau, Sylvain. “Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process.” 2012. Web. 15 Nov 2019.

Vancouver:

Fraineau S. Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process. [Internet] [Doctoral dissertation]. Poitiers; 2012. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2012POIT2307.

Council of Science Editors:

Fraineau S. Effets cellulaires et voies de signalisation activés par le facteur anticoagulant, la protéine S, sur les cellules endothéliales : implication lors de l'angiogenèse : Cellular effects and signaling pathways activated by the anticoagulant factor, protein S, on endothelial cells in the angiogenic process. [Doctoral Dissertation]. Poitiers; 2012. Available from: http://www.theses.fr/2012POIT2307


University of Minnesota

5. Metivier, Kristy Stacy. Gene and protein expression in canine follicular thyroid carcinoma.

Degree: PhD, Veterinary Medicine, 2011, University of Minnesota

 The major goal of this study was to investigate the molecular characteristics of canine follicular thyroid carcinoma (FTC). This is a rapidly growing and highly… (more)

Subjects/Keywords: Canine; Follicular thyroid carcinoma; Gene expression; Osteopontin; Plasma; VEGFA; Veterinary Medicine

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APA (6th Edition):

Metivier, K. S. (2011). Gene and protein expression in canine follicular thyroid carcinoma. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/116265

Chicago Manual of Style (16th Edition):

Metivier, Kristy Stacy. “Gene and protein expression in canine follicular thyroid carcinoma.” 2011. Doctoral Dissertation, University of Minnesota. Accessed November 15, 2019. http://purl.umn.edu/116265.

MLA Handbook (7th Edition):

Metivier, Kristy Stacy. “Gene and protein expression in canine follicular thyroid carcinoma.” 2011. Web. 15 Nov 2019.

Vancouver:

Metivier KS. Gene and protein expression in canine follicular thyroid carcinoma. [Internet] [Doctoral dissertation]. University of Minnesota; 2011. [cited 2019 Nov 15]. Available from: http://purl.umn.edu/116265.

Council of Science Editors:

Metivier KS. Gene and protein expression in canine follicular thyroid carcinoma. [Doctoral Dissertation]. University of Minnesota; 2011. Available from: http://purl.umn.edu/116265


Université de Montréal

6. Ciarlillo, Domenic. A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice .

Degree: 2017, Université de Montréal

 L’hypoxie contribue à la guérison cutanée via l’induction de HIF-1 (hypoxia-inducible factor-1). HIF-1 gère l’expression de VEGFA (vascular endothelial growth factor A) en se liant… (more)

Subjects/Keywords: HIF-1α; VEGFA; Peau; Guérison; Tissu de granulation; HRE; Skin; Wound healing; Granulation tissue

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APA (6th Edition):

Ciarlillo, D. (2017). A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/19161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ciarlillo, Domenic. “A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice .” 2017. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/19161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ciarlillo, Domenic. “A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice .” 2017. Web. 15 Nov 2019.

Vancouver:

Ciarlillo D. A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/19161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ciarlillo D. A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/19161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. 한, 송미. LDHB deficiency ameliorates ischemic neuronal cell death.

Degree: 2017, Ajou University

Although brain occupies only 2% of body weight, it consumes approximately 25% of the body's total energy and neuronal activity comprise 80% of brain energy… (more)

Subjects/Keywords: Lactate dehydrogenase; Ischemic stress; Neuronal cell death; VEGFA; 젖산탈수소효소; 허혈성 스트레스; 신경세포 사멸; 혈관 표피성장인자

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APA (6th Edition):

한, . (2017). LDHB deficiency ameliorates ischemic neuronal cell death. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16423 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000024174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

한, 송미. “LDHB deficiency ameliorates ischemic neuronal cell death.” 2017. Thesis, Ajou University. Accessed November 15, 2019. http://repository.ajou.ac.kr/handle/201003/16423 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000024174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

한, 송미. “LDHB deficiency ameliorates ischemic neuronal cell death.” 2017. Web. 15 Nov 2019.

Vancouver:

한 . LDHB deficiency ameliorates ischemic neuronal cell death. [Internet] [Thesis]. Ajou University; 2017. [cited 2019 Nov 15]. Available from: http://repository.ajou.ac.kr/handle/201003/16423 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000024174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

한 . LDHB deficiency ameliorates ischemic neuronal cell death. [Thesis]. Ajou University; 2017. Available from: http://repository.ajou.ac.kr/handle/201003/16423 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000024174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Le Hecho, Sara. Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis.

Degree: Docteur es, Physiologie Physiopathologie, 2014, Université Pierre et Marie Curie – Paris VI

Dans les maladies chroniques du foie, l’angiogenèse et la fibrose sont étroitement liées. Nosprécédents travaux ont permis de montrer que les myofibroblastes portaux (MFP)contribuaient de… (more)

Subjects/Keywords: Myofibroblastes portaux; Angiogenèse; Fibrose hépatique; Collagène XV; Cellule endothéliale sinusoïdale; VEGFA; Portal myofibroblasts; Angiogenesis; 616.362

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APA (6th Edition):

Le Hecho, S. (2014). Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066252

Chicago Manual of Style (16th Edition):

Le Hecho, Sara. “Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed November 15, 2019. http://www.theses.fr/2014PA066252.

MLA Handbook (7th Edition):

Le Hecho, Sara. “Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis.” 2014. Web. 15 Nov 2019.

Vancouver:

Le Hecho S. Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2014PA066252.

Council of Science Editors:

Le Hecho S. Les myofibroblastes portaux : fonction angiogénique et implication dans la progression de la fibrose hépatique : Portal myofibroblasts : angiogenic function and involvement in progression of liver fibrosis. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066252


University of Lund

9. Rehn, Matilda. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.

Degree: 2011, University of Lund

 Hematopoiesis is the process of blood formation that originates with the hematopoietic stem cell (HSC), a cell type that is responsible for the life-long supply… (more)

Subjects/Keywords: Hematologi; Hematopoietic Stem Cells; Stem Cell Niche; Hypoxia; Hypoxia-Inducible Factor; Vegfa.

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APA (6th Edition):

Rehn, M. (2011). THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf

Chicago Manual of Style (16th Edition):

Rehn, Matilda. “THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.” 2011. Doctoral Dissertation, University of Lund. Accessed November 15, 2019. http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf.

MLA Handbook (7th Edition):

Rehn, Matilda. “THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.” 2011. Web. 15 Nov 2019.

Vancouver:

Rehn M. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. [Internet] [Doctoral dissertation]. University of Lund; 2011. [cited 2019 Nov 15]. Available from: http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf.

Council of Science Editors:

Rehn M. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. [Doctoral Dissertation]. University of Lund; 2011. Available from: http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf


University of Toronto

10. Rahman, Muhammad Nafeesur. Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells.

Degree: 2010, University of Toronto

Embryonic stem cells (ESCs) are derived from the inner cell mass (ICM) of the embryo that have great potential for regenerative therapies because of their… (more)

Subjects/Keywords: Agarose; Hydrogel; Embryoid Bodies; VEGFA; Blood Progenitor Cells; 3D; Differentiation; Serum-Free; 0541; 0542

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APA (6th Edition):

Rahman, M. N. (2010). Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24626

Chicago Manual of Style (16th Edition):

Rahman, Muhammad Nafeesur. “Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells.” 2010. Masters Thesis, University of Toronto. Accessed November 15, 2019. http://hdl.handle.net/1807/24626.

MLA Handbook (7th Edition):

Rahman, Muhammad Nafeesur. “Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells.” 2010. Web. 15 Nov 2019.

Vancouver:

Rahman MN. Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1807/24626.

Council of Science Editors:

Rahman MN. Vascular Endothelial Growth Factor Functionalized Agarose Can Efficiently Guide Pluripotent Stem Cell Aggregates Toward Blood Progenitor Cells. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24626


Université de Montréal

11. Tsoi, Mayra. Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor .

Degree: 2013, Université de Montréal

 Les tumeurs des cellules de la granulosa (GCTs) sont des tumeurs avec un potentiel malin ayant tendance à récidiver, provoquant ainsi la mort dans 80%… (more)

Subjects/Keywords: Tumeur des cellules de la granulosa; Thérapie anti-VEGFA; Angiogenèse; Traitement adjuvant; Modèle murin préclinique; Signalisation AKT; Signalisation MAPK; Granulosa cell tumor; Anti-VEGFA therapy; Angiogenesis; Adjuvant therapy; Preclinical mouse model; AKT signaling; MAPK signaling

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APA (6th Edition):

Tsoi, M. (2013). Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsoi, Mayra. “Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor .” 2013. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/9608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsoi, Mayra. “Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor .” 2013. Web. 15 Nov 2019.

Vancouver:

Tsoi M. Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/9608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsoi M. Anti-VEGFA therapy reduces tumor growth and extends survival in a murine model of ovarian granulosa cell tumor . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Fan, Jieqing. Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2014, University of Cincinnati

 Angiogenesis defines the process in which new vessels grow from existing vessels. Revealing the molecular mechanisms involved in this process will help deepen our understanding… (more)

Subjects/Keywords: Developmental Biology; Angiogenesis; Crim1; Endothelial cells; Vegfa; Vessel stability

…VEGF, or VEGFA) is the limiting step in almost all angiogenesis events, no matter in… …normal development or in pathogenesis (Carmeliet, 2003). VEGFA is secreted growth… …and some neurons, the VEGFA signaling is regarded as the mere most important pathway… …et al., 1999); loss of only one allele of Vegfa gene in mice cause embryonic lethality… …and blood-island formation. VEGFA has three major isoforms VEGFA121, VEGFA165 and VEGFA189… 

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APA (6th Edition):

Fan, J. (2014). Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1397734750

Chicago Manual of Style (16th Edition):

Fan, Jieqing. “Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling.” 2014. Doctoral Dissertation, University of Cincinnati. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1397734750.

MLA Handbook (7th Edition):

Fan, Jieqing. “Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling.” 2014. Web. 15 Nov 2019.

Vancouver:

Fan J. Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling. [Internet] [Doctoral dissertation]. University of Cincinnati; 2014. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1397734750.

Council of Science Editors:

Fan J. Crim1 Maintains Retinal Vascular Stability during Development by Regulating Endothelial Cell Vegfa Autocrine Signaling. [Doctoral Dissertation]. University of Cincinnati; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1397734750

.