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You searched for subject:(VEGF Vascular endothelium growth factor ). Showing records 1 – 30 of 530 total matches.

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1. 手塚, 里奈. Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究.

Degree: 博士(歯学), 2016, The Nippon Dental University / 日本歯科大学

2015

Subjects/Keywords: Zoledoronic acid (ZOL); Dexamethasone (DEX); Vascular endothelial growth factor (VEGF); Osteoprotegerin (OPG); Receptor activator of nuclear factor kappa-B ligand (RANKL)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

手塚, . (2016). Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究. (Thesis). The Nippon Dental University / 日本歯科大学. Retrieved from http://id.nii.ac.jp/1102/00000770/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

手塚, 里奈. “Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究.” 2016. Thesis, The Nippon Dental University / 日本歯科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1102/00000770/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

手塚, 里奈. “Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究.” 2016. Web. 23 Aug 2019.

Vancouver:

手塚 . Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究. [Internet] [Thesis]. The Nippon Dental University / 日本歯科大学; 2016. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1102/00000770/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

手塚 . Jawbone changes in sodium zoledronic acid- and dexamethasone-treated rats : ゾレドロン酸ナトリウムおよびデキサメタゾン投与ラットの顎骨変化に関する研究. [Thesis]. The Nippon Dental University / 日本歯科大学; 2016. Available from: http://id.nii.ac.jp/1102/00000770/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura, Mitsutoshi. Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与.

Degree: 博士(医学), 2013, Nara Medical University / 奈良県立医科大学

Cerebral venous ischemia can result in severe brain edema. Inhibition of vascular endothelial growth factor (VEGF) activity by a neutralizing antibody can completely block the… (more)

Subjects/Keywords: cerebral venous infarction; vascular endothelial growth factor; antagonist; brain edema

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APA (6th Edition):

Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura, M. (2013). Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura, Mitsutoshi. “Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与.” 2013. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 23, 2019. http://hdl.handle.net/10564/2676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura, Mitsutoshi. “Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与.” 2013. Web. 23 Aug 2019.

Vancouver:

Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura M. Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2013. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10564/2676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miyake, Hitoshi; Nakagawa, Ichiro; Takeshima, Yasuhiro; Nishimura, Fumihiko; Park, Young-Su; Nakamura M. Post-ischemic administration of vascular endothelial growth factor inhibitor in a rat model of cerebral venous infarction. : ラット脳静脈モデルにおける血管内皮増殖因子桔抗薬の虚血後投与. [Thesis]. Nara Medical University / 奈良県立医科大学; 2013. Available from: http://hdl.handle.net/10564/2676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa, Shin. Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する.

Degree: 博士(医学), 2015, Nara Medical University / 奈良県立医科大学

Although recent research showed that advanced glycation endproduct (AGE) and hydroquinone (HQ) are related to the pathogenesis of age-related macular degeneration (AMD), the mechanism how… (more)

Subjects/Keywords: Age-related macular degeneration; Advanced glycation endproduct(s); Hydroquinone; Retinal pigment epithelial cells; Vascular endothelial growth factor

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APA (6th Edition):

Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa, S. (2015). Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3101

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa, Shin. “Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する.” 2015. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 23, 2019. http://hdl.handle.net/10564/3101.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa, Shin. “Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する.” 2015. Web. 23 Aug 2019.

Vancouver:

Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa S. Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10564/3101.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsujinaka, Hiroki; Itaya-Hironaka, Asako; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Ota, Hiroyo; Takeda, Maiko; Fujimura, Takanori; Takasawa S. Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of VEGF gene. : ヒト網膜色素上皮はヒドロキノンと最終糖化産物の共刺激により、VEGF遺伝子の発現上昇を伴って増殖する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. Available from: http://hdl.handle.net/10564/3101

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. 五十嵐, 靖之. VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される.

Degree: 歯学博士, 2016, Iwate Medical University / 岩手医科大学

2015

Subjects/Keywords: mesenchymal stem cells; lymphatic endothelial cells; vascular endothelial growth factor receptor 3; vascular endothelial growth factor C; p44/42 mitogen-activated protein kinase; mesenchymal stem cells; lymphatic endothelial cells; vascular endothelial growth factor receptor 3; vascular endothelial growth factor C; p44/42 mitogen-activated protein kinase

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APA (6th Edition):

五十嵐, . (2016). VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される. (Thesis). Iwate Medical University / 岩手医科大学. Retrieved from http://id.nii.ac.jp/1181/00005667/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

五十嵐, 靖之. “VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される.” 2016. Thesis, Iwate Medical University / 岩手医科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1181/00005667/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

五十嵐, 靖之. “VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される.” 2016. Web. 23 Aug 2019.

Vancouver:

五十嵐 . VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される. [Internet] [Thesis]. Iwate Medical University / 岩手医科大学; 2016. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1181/00005667/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

五十嵐 . VEGF-C and TGF-β reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes. : 間葉系幹細胞の骨芽細胞ならびにリンパ管内皮細胞への分化はVEGF-CとTGF-βによって相互に調節される. [Thesis]. Iwate Medical University / 岩手医科大学; 2016. Available from: http://id.nii.ac.jp/1181/00005667/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. 三木, 厚. Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持.

Degree: 博士(医学), 2007, Okayama University / 岡山大学

 Development of a subcutaneously implantable bioartificial pancreas (BAP) with immunoisolatory function could have a great impact on the treatment of diabetes mellitus. We have developed… (more)

Subjects/Keywords: bioartificial pancreas; human vascular endothelial cells; basic fibroblast growth factor; neovascularization

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APA (6th Edition):

三木, . (2007). Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持. (Thesis). Okayama University / 岡山大学. Retrieved from http://ousar.lib.okayama-u.ac.jp/11831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

三木, 厚. “Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持.” 2007. Thesis, Okayama University / 岡山大学. Accessed August 23, 2019. http://ousar.lib.okayama-u.ac.jp/11831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

三木, 厚. “Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持.” 2007. Web. 23 Aug 2019.

Vancouver:

三木 . Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持. [Internet] [Thesis]. Okayama University / 岡山大学; 2007. [cited 2019 Aug 23]. Available from: http://ousar.lib.okayama-u.ac.jp/11831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

三木 . Maintenance of Neovascularization at the Implantation Site of an Artificial Device by bFGF and Endothelial Cell Transplant : 塩基性線維芽細胞増殖因子と内皮細胞による人工臓器デバイス移植部位における血管維持. [Thesis]. Okayama University / 岡山大学; 2007. Available from: http://ousar.lib.okayama-u.ac.jp/11831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. 韦, 嘉章. Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する.

Degree: 博士(医学), 2014, Hamamatsu University School of Medicine / 浜松医科大学

Background: Linoleic acid (LA) promotes monocyte chemotaxis and cell adhesion molecules such as MCP-1 and VCAM-1, which contribute to atherosclerogenesis. These molecules are restrained by… (more)

Subjects/Keywords: Cyclic AMP; Endothelial cells; Endothelium-derived relaxing factor; Linoleic acid; Phosphdiesterase

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APA (6th Edition):

韦, . (2014). Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する. (Thesis). Hamamatsu University School of Medicine / 浜松医科大学. Retrieved from http://hdl.handle.net/10271/2877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

韦, 嘉章. “Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する.” 2014. Thesis, Hamamatsu University School of Medicine / 浜松医科大学. Accessed August 23, 2019. http://hdl.handle.net/10271/2877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

韦, 嘉章. “Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する.” 2014. Web. 23 Aug 2019.

Vancouver:

韦 . Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する. [Internet] [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10271/2877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

韦 . Linoleic acid attenuates endothelium-derived relaxing factor production by suppressing cAMP-hydrolyzing phosphodiesterase activity : リノール酸はcAMP加水分解型ホスホジエステラーゼ阻害を介して内皮由来血管弛緩因子産生を抑制する. [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2014. Available from: http://hdl.handle.net/10271/2877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

7. Iida, Yuto. Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study .

Degree: 2018, Kyoto University

Subjects/Keywords: branch retinal vein occlusion; optical coherence tomography angiography; fluorescein angiography; non perfusion area; anti-vascular endothelial growth factor

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APA (6th Edition):

Iida, Y. (2018). Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iida, Yuto. “Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study .” 2018. Thesis, Kyoto University. Accessed August 23, 2019. http://hdl.handle.net/2433/232104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iida, Yuto. “Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study .” 2018. Web. 23 Aug 2019.

Vancouver:

Iida Y. Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/2433/232104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iida Y. Morphological and Functional Retinal Vessel Changes in Branch Retinal Vein Occlusion: An Optical Coherence Tomography Angiography Study . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Kato, Manabu. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.

Degree: 博士(医学), 2017, Mie University / 三重大学

The prostate gland is unique in that it undergoes rapid regression following castration but regenerates completely once androgens are replaced. Residual ductal components play an… (more)

Subjects/Keywords: basal epithelial cells; castration; fibroblast growth factor 2; fibroblast growth factor receptor; growth factor; organ culture; prostate

Page 1 Page 2 Page 3

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APA (6th Edition):

Kato, M. (2017). Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00016972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kato, Manabu. “Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.” 2017. Thesis, Mie University / 三重大学. Accessed August 23, 2019. http://hdl.handle.net/10076/00016972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kato, Manabu. “Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.” 2017. Web. 23 Aug 2019.

Vancouver:

Kato M. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10076/00016972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kato M. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00016972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki, Toshiro. Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment.

Degree: 博士(理学), 2016, Tokyo Metropolitan University / 首都大学東京

Titanium and stainless steel were modified with dopamine for the immobilization of biomolecules, epidermal growth factor (EGF). First, the treatment of metal surfaces with a… (more)

Subjects/Keywords: Titanium; Stainless steel; Dopamine; Epidermal growth factor

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APA (6th Edition):

Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki, T. (2016). Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment. (Thesis). Tokyo Metropolitan University / 首都大学東京. Retrieved from http://hdl.handle.net/10748/6081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki, Toshiro. “Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment.” 2016. Thesis, Tokyo Metropolitan University / 首都大学東京. Accessed August 23, 2019. http://hdl.handle.net/10748/6081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki, Toshiro. “Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment.” 2016. Web. 23 Aug 2019.

Vancouver:

Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki T. Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment. [Internet] [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10748/6081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kang, Jeonghwa; Sakuragi, Makoto; Shibata, Aya; Abe, Hiroshi; Kitajima, Takashi; Tada, Seiichi; Mizutani, Masayoshi; Ohmori, Hitoshi; Ayame, Hirohito; Son, Tae Il; Aigaki T. Immobilization of epidermal growth factor on titanium and stainless steel surfaces via dopamine treatment. [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2016. Available from: http://hdl.handle.net/10748/6081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. 横山, 宗昴. 血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ.

Degree: 博士(医学), 2017, Tokai University Repository / 東海大学

Subjects/Keywords: 多血小板血漿, 軟骨再生, 変形性関節症, 細胞増殖因子; Platelet Rich Plasma, Cartilage regeneration, Osteoarthritis, Growth factor, Vascular endothelial growth factor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

横山, . (2017). 血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ. (Thesis). Tokai University Repository / 東海大学. Retrieved from https://opac.time.u-tokai.ac.jp/webopac/TD00000254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

横山, 宗昴. “血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ.” 2017. Thesis, Tokai University Repository / 東海大学. Accessed August 23, 2019. https://opac.time.u-tokai.ac.jp/webopac/TD00000254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

横山, 宗昴. “血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ.” 2017. Web. 23 Aug 2019.

Vancouver:

横山 . 血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ. [Internet] [Thesis]. Tokai University Repository / 東海大学; 2017. [cited 2019 Aug 23]. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

横山 . 血小板活性化血清関節内投与による軟骨治療効果の検討 : ケッショウバン カッセイカ ケッセイ カンセツナイ トウヨ ニヨル ナンコツ チリョウ コウカ ノ ケントウ. [Thesis]. Tokai University Repository / 東海大学; 2017. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. 中村, 普彦. Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う.

Degree: 博士(医学), 2018, Kurume University / 久留米大学

2015年度

Subjects/Keywords: Arteriovenous malformation; alternatively activated macrophage; perinidal dilated capillary network; vascular remodeling; VEGF

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APA (6th Edition):

中村, . (2018). Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う. (Thesis). Kurume University / 久留米大学. Retrieved from http://hdl.handle.net/11316/492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

中村, 普彦. “Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う.” 2018. Thesis, Kurume University / 久留米大学. Accessed August 23, 2019. http://hdl.handle.net/11316/492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

中村, 普彦. “Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う.” 2018. Web. 23 Aug 2019.

Vancouver:

中村 . Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う. [Internet] [Thesis]. Kurume University / 久留米大学; 2018. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/11316/492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

中村 . Alternatively Activated Macrophages Play an Important Role in Vascular Remodeling and Hemorrhaging in Patients with Brain Arteriovenous Malformation : 代替活性化マクロファージは脳動静脈奇形における血管再構築と出血に重要な役割を担う. [Thesis]. Kurume University / 久留米大学; 2018. Available from: http://hdl.handle.net/11316/492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. 田島, 久嗣. ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: VEGF

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APA (6th Edition):

田島, . (n.d.). ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/6292

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

田島, 久嗣. “ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed August 23, 2019. http://hdl.handle.net/10061/6292.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

田島, 久嗣. “ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ.” Web. 23 Aug 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

田島 . ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10061/6292.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

田島 . ピリジルメチル基を有するVEGF阻害剤の創製に関する研究 : The discovery and preparation of pyridylmethyl derivatives as VEGF inhibitors; ピリジル メチルキ オ ユウスル VEGF ソガイザイ ノ ソウセイ ニ カンスル ケンキュウ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/6292

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

13. 持永, 浩史. High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について.

Degree: 博士(医学), 2014, Nagasaki University / 長崎大学

 Background It has been shown that 5-fluorouracil (5-FU) sensitivity in patients with non–small-cell lung cancer (NSCLC) is associated with epidermal growth factor receptor (EGFR) mutation… (more)

Subjects/Keywords: Adenocarcinoma in situ; Dihydropyrimidine dehydrogenase; epidermal growth factor receptor mutation; epidermal growth factor receptor tyrosine kinase inhibitor

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APA (6th Edition):

持永, . (2014). High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

持永, 浩史. “High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について.” 2014. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/35414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

持永, 浩史. “High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について.” 2014. Web. 23 Aug 2019.

Vancouver:

持永 . High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/35414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

持永 . High Expression of Dihydropyrimidine Dehydrogenase in Lung Adenocarcinoma is Associated With Mutations in Epidermal Growth Factor Receptor: Implications for the Treatment of Non–Small-Cell Lung Cancer Using 5-Fluorouracil : 肺腺癌におけるDPD高発現はEGFR遺伝子変異と関連する:5-FU系抗癌剤を用いた非小細胞肺癌治療への影響について. [Thesis]. Nagasaki University / 長崎大学; 2014. Available from: http://hdl.handle.net/10069/35414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Ueno, Toshinori. Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる.

Degree: 博士(医学), 2014, Hiroshima University / 広島大学

 Mesenchymal stem cells (MSCs) are multipotent adult stem cells that have regenerative capability and exert paracrine actions on damaged tissues. Since peritoneal fibrosis is a… (more)

Subjects/Keywords: epithelial-to-mesenchymal transition; hepatocyte growth factor; mesenchymal stem cells; peritoneal fibrosis; transforming growth factor-β1

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APA (6th Edition):

Ueno, T. (2014). Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる. (Thesis). Hiroshima University / 広島大学. Retrieved from http://ir.lib.hiroshima-u.ac.jp/00036259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ueno, Toshinori. “Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる.” 2014. Thesis, Hiroshima University / 広島大学. Accessed August 23, 2019. http://ir.lib.hiroshima-u.ac.jp/00036259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ueno, Toshinori. “Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる.” 2014. Web. 23 Aug 2019.

Vancouver:

Ueno T. Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる. [Internet] [Thesis]. Hiroshima University / 広島大学; 2014. [cited 2019 Aug 23]. Available from: http://ir.lib.hiroshima-u.ac.jp/00036259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ueno T. Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signaling : 間葉系幹細胞は炎症抑制およびTGF-β1シグナルの抑制を介し腹膜線維化を改善させる. [Thesis]. Hiroshima University / 広島大学; 2014. Available from: http://ir.lib.hiroshima-u.ac.jp/00036259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Transforming growth factor-β1 (TGF-β1) reportedly causes the differentiation of fibroblasts to myofibroblasts during wound healing. We investigated the mechanism underlying the activation of latent TGF-β1… (more)

Subjects/Keywords: keratinocyte; fibroblast; transforming growth factor-β1; αv-integrin; matrix metalloproteinase

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APA (6th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, H. (2014). Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Web. 23 Aug 2019.

Vancouver:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. 小林, 信博. Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.

Degree: 博士(歯学), 2017, Osaka Dental University / 大阪歯科大学

The effect of porous alpha-tricalcium phosphate (-TCP) with immobilized basic fibroblast growth factor (bFGF) on bone regeneration was evaluated in a canine mandibular bone defect… (more)

Subjects/Keywords: alpha-tricalcium phosphate |basic fibroblast growth factor | bone regeneration

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APA (6th Edition):

小林, . (2017). Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Thesis, Osaka Dental University / 大阪歯科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Web. 23 Aug 2019.

Vancouver:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. Available from: http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. 岡, 宏亮. Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts.

Degree: 博士(医学), 2016, Oita University / 大分大学

AIMS: Idiopathic pulmonary fibrosis continues to be a devastating clinical disorder for which there are few therapeutic options, and the pathogenesis of this disease remains… (more)

Subjects/Keywords: Pulmonary fibrosis; Statin; Mevalonate; Transforming growth factor-β

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APA (6th Edition):

岡, . (2016). Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

岡, 宏亮. “Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts.” 2016. Thesis, Oita University / 大分大学. Accessed August 23, 2019. http://hdl.handle.net/10559/15611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

岡, 宏亮. “Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts.” 2016. Web. 23 Aug 2019.

Vancouver:

岡 . Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10559/15611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

岡 . Inhibitory effects of pitavastatin on fibrogenic mediator production by human lung fibroblasts. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto, Kunio. Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

BACKGROUND: Continuous administration of prostacyclin has improved the survival of patients with pulmonary arterial hypertension (PAH). However, this treatment has some problems, including its short… (more)

Subjects/Keywords: cAMP; Hepatocyte growth factor; Monocrotaline; Pulmonary arterial hypertension; Thromboxane synthase

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APA (6th Edition):

Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto, K. (2014). Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto, Kunio. “Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 23, 2019. http://hdl.handle.net/10564/2711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto, Kunio. “Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与.” 2014. Web. 23 Aug 2019.

Vancouver:

Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto K. Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10564/2711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakamura, Atsuhiro; Nagaya, Noritoshi; Obata, Hiroaki; Sakai, Katsuya; Sakai, Yoshiki; Yoshikawa, Masanori; Hamada, Kaoru; Matsumoto K. Oral administration of a novel long-acting prostacyclin agonist with thromboxane synthase inhibitory activity for pulmonary arterial hypertension. : 肺高血圧症に対するトロンボキサン合成阻害作用をもった新規長期作用型プロスタサイクリンアゴニストの経口投与. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. 後藤, 響. Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―.

Degree: 博士(医学), 2017, Nagasaki University / 長崎大学

 Purpose: The purpose of this study was to investigate the pathology and mechanism of immobilization-induced skin fibrosis in a rat joint contracture model. Methods: Rats… (more)

Subjects/Keywords: Hypoxia; immobilization; collagen; myofibroblast; skin fibrosis; transforming growth factor-β1

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APA (6th Edition):

後藤, . (2017). Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/37569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

後藤, 響. “Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―.” 2017. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/37569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

後藤, 響. “Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―.” 2017. Web. 23 Aug 2019.

Vancouver:

後藤 . Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/37569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

後藤 . Development and progression of immobilization-induced skin fibrosis through overexpression of transforming growth factor-ß1 and hypoxic conditions in a rat knee joint contracture model. : TGF-ß1の過剰発現と低酸素状態は不動に伴う皮膚の線維化の発生と進行に関与する―膝関節屈曲拘縮モデルラットにおける検討―. [Thesis]. Nagasaki University / 長崎大学; 2017. Available from: http://hdl.handle.net/10069/37569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 後藤,俊平. マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells.

Degree: 博士(医学), 2017, Nihon University / 日本大学

Subjects/Keywords: 脱分化脂肪細胞; dedifferentiated fat cell; ペリサイト; pericyte; 血管新生; angiogenesis; TGF-β; transforming growth factor-β; 血管内皮細胞; vascular endothelial cell

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APA (6th Edition):

後藤,俊平. (2017). マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/1198 ; http://dx.doi.org/10.15006/32665A5182

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

後藤,俊平. “マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells.” 2017. Thesis, Nihon University / 日本大学. Accessed August 23, 2019. http://repository.nihon-u.ac.jp/xmlui/handle/11263/1198 ; http://dx.doi.org/10.15006/32665A5182.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

後藤,俊平. “マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells.” 2017. Web. 23 Aug 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

後藤,俊平. マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells. [Internet] [Thesis]. Nihon University / 日本大学; 2017. [cited 2019 Aug 23]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/1198 ; http://dx.doi.org/10.15006/32665A5182.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

後藤,俊平. マウス脱分化脂肪細胞のペリサイト分化メカニズム : Mechanisms of pericyte differentiation in mouse dedifferentiated fat cells. [Thesis]. Nihon University / 日本大学; 2017. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/1198 ; http://dx.doi.org/10.15006/32665A5182

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

21. Ozasa, Ryuki; Ohno, Jun; Iwahashi, Teruaki. Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice.

Degree: 博士(歯学), 2013, Fukuoka Dental College / 福岡歯科大学

2013年度

Subjects/Keywords: センチネルリンパ節; 担がんリンパ節; 口腔メラノーマ; リンパ管増生新生; リンパ行性転移; Sentinel lymph node; Tumor-bearing lymph node; Oral melanoma; Lymphangiogenesis; Lympatic metastasis; Animals; Female; Gene Expression Regulation, Neoplastic; Humans; Lymph Nodes; Lymphangiogenesis; Lymphatic Metastasis; Melanoma; Melanoma, Experimental; Mice; Prognosis; Uterine Cervical Neoplasms; Vascular Endothelial Growth Factor C; Vascular Endothelial Growth Factor Receptor-3

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APA (6th Edition):

Ozasa, Ryuki; Ohno, Jun; Iwahashi, T. (2013). Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000001/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ozasa, Ryuki; Ohno, Jun; Iwahashi, Teruaki. “Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice.” 2013. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1167/00000001/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ozasa, Ryuki; Ohno, Jun; Iwahashi, Teruaki. “Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice.” 2013. Web. 23 Aug 2019.

Vancouver:

Ozasa, Ryuki; Ohno, Jun; Iwahashi T. Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2013. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1167/00000001/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ozasa, Ryuki; Ohno, Jun; Iwahashi T. Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. : Tumor-induced lymphangiogenesis in cervical lymph nodes in oral melanoma-bearing mice. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2013. Available from: http://id.nii.ac.jp/1167/00000001/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. 福島, 直文; フクシマ, ナオブミ. 新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk.

Degree: 博士(医学), 1993, Gunma University / 群馬大学

学位記番号:医博乙943

Subjects/Keywords: Fibronectin; Neonate; Human milk; Epidermal growth factor; Fibroblast growth factor

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APA (6th Edition):

福島, 直文; フクシマ, . (1993). 新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk. (Thesis). Gunma University / 群馬大学. Retrieved from http://hdl.handle.net/10087/8284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

福島, 直文; フクシマ, ナオブミ. “新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk.” 1993. Thesis, Gunma University / 群馬大学. Accessed August 23, 2019. http://hdl.handle.net/10087/8284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

福島, 直文; フクシマ, ナオブミ. “新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk.” 1993. Web. 23 Aug 2019.

Vancouver:

福島, 直文; フクシマ . 新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk. [Internet] [Thesis]. Gunma University / 群馬大学; 1993. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10087/8284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

福島, 直文; フクシマ . 新生児血中フィブロネクチン濃度と人乳中フィブロネクチン産生刺激活性に関する検討 : Blood Levels of Fibronectin in Neonates and Fibronectin Synthesis Stimulating Activity in Human Breast Milk. [Thesis]. Gunma University / 群馬大学; 1993. Available from: http://hdl.handle.net/10087/8284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. 松瀬, 和也. Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生.

Degree: 博士(歯学), 2018, Osaka Dental University / 大阪歯科大学

We evaluated the effect of porous alpha-tricalcium phosphate (α-TCP) with immobilized basic fibroblast growth factor (bFGF) on periodontal regeneration in a canine model of 2-wall… (more)

Subjects/Keywords: Alpha-tricalcium phosphate; Basic fibroblast growth factor; Canine2-wall periodontal defect model; Immobilization; Periodontal regeneration

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APA (6th Edition):

松瀬, . (2018). Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000157/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

松瀬, 和也. “Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生.” 2018. Thesis, Osaka Dental University / 大阪歯科大学. Accessed August 23, 2019. http://id.nii.ac.jp/1392/00000157/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

松瀬, 和也. “Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生.” 2018. Web. 23 Aug 2019.

Vancouver:

松瀬 . Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2018. [cited 2019 Aug 23]. Available from: http://id.nii.ac.jp/1392/00000157/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

松瀬 . Periodontal regeneration induced by porous alpha-tricalcium phosphate with immobilized basic fibroblast growth factor in a canine model of 2-wall periodontal defects : イヌ下顎骨二壁性骨欠損モデルにおける塩基性線維芽細胞増殖因子を固定化したα-リン酸三カルシウムの歯周組織再生. [Thesis]. Osaka Dental University / 大阪歯科大学; 2018. Available from: http://id.nii.ac.jp/1392/00000157/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

24. Hiwatashi, Nao. The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

Final publication is available at http://onlinelibrary.wiley.com/doi/10.1002/term.2060/abstract;jsessionid=F0849D98381EEF9E83401A02B9042F4D.f04t02

新制・課程博士

甲第19602号

医博第4109号

Subjects/Keywords: vocal fold scar; collagen-gelatin sponge; basic fibroblast growth factor; wound healing; regeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hiwatashi, N. (2016). The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215428 ; http://dx.doi.org/10.14989/doctor.k19602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hiwatashi, Nao. “The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療.” 2016. Thesis, Kyoto University / 京都大学. Accessed August 23, 2019. http://hdl.handle.net/2433/215428 ; http://dx.doi.org/10.14989/doctor.k19602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hiwatashi, Nao. “The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療.” 2016. Web. 23 Aug 2019.

Vancouver:

Hiwatashi N. The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/2433/215428 ; http://dx.doi.org/10.14989/doctor.k19602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hiwatashi N. The efficacy of a novel collagen-gelatin scaffold with basic fibroblast growth factor for the treatment of vocal fold scar : 塩基性線維芽細胞増殖因子徐放性コラーゲンゼラチンスポンジを用いた声帯瘢痕の再生治療. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215428 ; http://dx.doi.org/10.14989/doctor.k19602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. 豊田, 啓介. Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。.

Degree: 博士(医学), 2013, Nagasaki University / 長崎大学

 Glioblastoma multiforme (GBM) cells invade along the existing normal capillaries in brain. Normal capillary endothelial cells function as the blood-brain barrier (BBB) that limits permeability… (more)

Subjects/Keywords: Blood-brain barrier; Fibroblast growth factor 2; Glioblastoma; Rat brain endothelial cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

豊田, . (2013). Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

豊田, 啓介. “Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。.” 2013. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/35391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

豊田, 啓介. “Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。.” 2013. Web. 23 Aug 2019.

Vancouver:

豊田 . Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2013. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/35391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

豊田 . Initial Contact of Glioblastoma Cells with Existing Normal Brain Endothelial Cells Strengthen the Barrier Function via Fibroblast Growth Factor 2 Secretion: A New In Vitro Blood-Brain Barrier Model. : 新規血液脳関門in vitro再構成モデルにおいて、膠芽腫細胞が正常脳毛細血管内皮細胞に接触した初期反応として、膠芽腫細胞が分泌するFibroblast Growth Factor 2を介し正常脳毛細血管内皮細胞の血液脳関門機能は強化される。. [Thesis]. Nagasaki University / 長崎大学; 2013. Available from: http://hdl.handle.net/10069/35391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. 江口, 正倫. Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明.

Degree: 博士(医学), 2015, Nagasaki University / 長崎大学

 Aims Functional and structural changes in pulmonary vasculature characterize pulmonary arterial hypertension (PAH) and the prognosis of advanced PAH remains poor despite progress in pharmacotherapy.… (more)

Subjects/Keywords: Endothelin-1; Transforming growth factor beta; Pulmonary hypertension; Adipose derived regenerative cell (ADRC)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

江口, . (2015). Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

江口, 正倫. “Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/35495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

江口, 正倫. “Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明.” 2015. Web. 23 Aug 2019.

Vancouver:

江口 . Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/35495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

江口 . Adipose-derived regenerative cell therapy inhibits the progression of monocrotaline-induced pulmonary hypertension in rats : 脂肪由来幹細胞(ADRCs)を用いた細胞移植療法の肺動脈性肺高血圧症モデルラットへの有効性の検討とそのメカニズムの解明. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/35495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. 鈴木, 貴久. Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される.

Degree: 博士(医学), 2015, Nagasaki University / 長崎大学

 Objective: To investigate the role of thrombospondin 1 (TSP-1) in RA. Methods: Expression of TSP-1 in synovial tissues was determined by immunohistochemistry. Expression of TSP-1… (more)

Subjects/Keywords: rheumatoid arthritis; fibroblast-like synovial cell; synovitis; thrombospondin 1; transforming growth factor-β1; biomarker

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

鈴木, . (2015). Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

鈴木, 貴久. “Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/36188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

鈴木, 貴久. “Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される.” 2015. Web. 23 Aug 2019.

Vancouver:

鈴木 . Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/36188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

鈴木 . Upregulation of thrombospondin-1(TSP-1) expression in synovial tissues and plasma of rheumatoid arthritis (RA): Role of transforming growth factor-β1 toward fibroblast-like synovial cells : Thrombospondin-1は関節リウマチにおいて滑膜組織及び血漿レベルで過剰発現しており、 滑膜線維芽細胞ではtransforming growth factor-β1により誘導される. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/36188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. 富永, 哲郎. Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.

Degree: 博士(医学), 2016, Nagasaki University / 長崎大学

 Background: It has been shown that epidermal growth factor receptor (EGFR) mutation status is associated with 5-fluorouracil (5-FU) sensitivity in non-small-cell lung cancer (NSCLC). However,… (more)

Subjects/Keywords: Non-small-cell lung cancer; Sp1; Dihydropyrimidine dehydrogenase; Epidermal growth factor receptor mutation; 5-fluorouracil

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APA (6th Edition):

富永, . (2016). Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed August 23, 2019. http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Web. 23 Aug 2019.

Vancouver:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano, Masayuki. Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

Patients with chronic kidney disease (CKD) die of cardiovascular diseases for unknown reasons. Blood vessel formation in plaques and its relationship with plaque stability could… (more)

Subjects/Keywords: atherosclerosis; chronic kidney disease; placental growth factor; soluble fms-like; tyrosine kinase-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano, M. (2014). Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano, Masayuki. “Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 23, 2019. http://hdl.handle.net/10564/2699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano, Masayuki. “Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる.” 2014. Web. 23 Aug 2019.

Vancouver:

Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano M. Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10564/2699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matsui, Masaru; Takeda, Yukiji; Uemura, Shiro; Matsumoto, Takaki; Seno, Ayako; Onoue, Kenji; Tsushima, Hideo; Morimoto, Katsuhiko; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Samejima, Ken-ichi; Kawata, Hiroyuki; Kawakami, Rika; Nakatani, Kimihiko; Iwano M. Suppressed soluble Fms-like tyrosine kinase-1 production aggravates atherosclerosis in chronic kidney disease. : 慢性腎臓病では血中可溶型fms様チロシンキナーゼ-1産生の減少が動脈硬化症を悪化させる. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii, Manabu. An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

OBJECTIVE: To investigate the predictive values of placental growth factor (PlGF) and its endogenous antagonist, soluble fms-like tyrosine kinase-1 (sFlt-1), for the long-term prognosis of… (more)

Subjects/Keywords: stable coronary artery disease; prognosis; biomarker; placental growth factor; soluble Flt-1

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APA (6th Edition):

Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii, M. (2014). An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii, Manabu. “An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 23, 2019. http://hdl.handle.net/10564/2700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii, Manabu. “An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である.” 2014. Web. 23 Aug 2019.

Vancouver:

Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii M. An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10564/2700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matsumoto, Takaki; Uemura, Shiro; Takeda, Yukiji; Matsui, Masaru; Okada, Sadanori; Nishida, Taku; Soeda, Tsunenari; Okayama, Satoshi; Somekawa, Satoshi; Ishigami, Ken-ichi; Onoue, Kenji; Kawata, Hiroyuki; Kawakami, Rika; Horii M. An elevated ratio of placental growth factor to soluble fms-like tyrosine kinase-1 predicts adverse outcomes in patients with stable coronary artery disease. : 胎盤増殖因子の可溶性Fms様チロシンキナーゼ-1に対する血中濃度比の上昇は安定冠動脈疾患患者における有害事象発症の予測因子である. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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