Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Ubiquitination). Showing records 1 – 30 of 321 total matches.

[1] [2] [3] [4] [5] … [11]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Saskatchewan

1. Wang, Sheng. Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana.

Degree: 2018, University of Saskatchewan

 Protein ubiquitination, a fundamental protein modification cascade, is catalyzed by three types of enzymes referred to as E1 for ubiquitin (Ub) activation, E2 for Ub… (more)

Subjects/Keywords: Ubiquitination; gametogenesis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, S. (2018). Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/11670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Sheng. “Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana.” 2018. Thesis, University of Saskatchewan. Accessed March 04, 2021. http://hdl.handle.net/10388/11670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Sheng. “Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana.” 2018. Web. 04 Mar 2021.

Vancouver:

Wang S. Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana. [Internet] [Thesis]. University of Saskatchewan; 2018. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10388/11670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang S. Studies of E2s Related to Unconventional Ubiquitination in Arabidopsis thaliana. [Thesis]. University of Saskatchewan; 2018. Available from: http://hdl.handle.net/10388/11670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

2. Abou Zeinab, Rami M. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.

Degree: PhD, Medical Sciences-Laboratory Medicine and Pathology, 2015, University of Alberta

 Cancer and tumor suppressors have been highly associated with many cancer researches. The main function of these proteins is to detect any error in DNA… (more)

Subjects/Keywords: ubiquitination; Pirh2; p73

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abou Zeinab, R. M. (2015). The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cjq085k005

Chicago Manual of Style (16th Edition):

Abou Zeinab, Rami M. “The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.” 2015. Doctoral Dissertation, University of Alberta. Accessed March 04, 2021. https://era.library.ualberta.ca/files/cjq085k005.

MLA Handbook (7th Edition):

Abou Zeinab, Rami M. “The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.” 2015. Web. 04 Mar 2021.

Vancouver:

Abou Zeinab RM. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2021 Mar 04]. Available from: https://era.library.ualberta.ca/files/cjq085k005.

Council of Science Editors:

Abou Zeinab RM. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/cjq085k005


University of Miami

3. Catoe, Heath W. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2010, University of Miami

  The Estrogen Receptor alpha (ER alpha) is a multi-domain transcription factor that has been extensively studied due to its known involvement in breast cancer… (more)

Subjects/Keywords: Degradation; Ubiquitination; Transcription

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Catoe, H. W. (2010). The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/464

Chicago Manual of Style (16th Edition):

Catoe, Heath W. “The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.” 2010. Doctoral Dissertation, University of Miami. Accessed March 04, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/464.

MLA Handbook (7th Edition):

Catoe, Heath W. “The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.” 2010. Web. 04 Mar 2021.

Vancouver:

Catoe HW. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2021 Mar 04]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/464.

Council of Science Editors:

Catoe HW. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/464


University of Rochester

4. Lin, Xi. Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis.

Degree: PhD, 2020, University of Rochester

 Osteoarthritis (OA) is a whole joint disease associated with cartilage destruction and chronic inflammation. Catabolic factors, including arthritogenic enzymes are produced by various cell types… (more)

Subjects/Keywords: Lymphatics; Macrophage; Ubiquitination; Posttraumatic osteoarthritis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, X. (2020). Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/35729

Chicago Manual of Style (16th Edition):

Lin, Xi. “Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis.” 2020. Doctoral Dissertation, University of Rochester. Accessed March 04, 2021. http://hdl.handle.net/1802/35729.

MLA Handbook (7th Edition):

Lin, Xi. “Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis.” 2020. Web. 04 Mar 2021.

Vancouver:

Lin X. Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis. [Internet] [Doctoral dissertation]. University of Rochester; 2020. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1802/35729.

Council of Science Editors:

Lin X. Impact of Macrophages on the Synovial Lymphatic System in Mice with Posttraumatic Osteoarthritis. [Doctoral Dissertation]. University of Rochester; 2020. Available from: http://hdl.handle.net/1802/35729


University of Alberta

5. Mottet, Kelly. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The significance of poxvirus manipulation of the host ubiquitin proteasome system has become increasingly apparent. Ubiquitin is post-translationally added to target proteins by a highly… (more)

Subjects/Keywords: ligase; ubiquitination; proteasome; ubiquitin; poxvirus

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mottet, K. (2010). The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/zp38wf105

Chicago Manual of Style (16th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Masters Thesis, University of Alberta. Accessed March 04, 2021. https://era.library.ualberta.ca/files/zp38wf105.

MLA Handbook (7th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Web. 04 Mar 2021.

Vancouver:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2021 Mar 04]. Available from: https://era.library.ualberta.ca/files/zp38wf105.

Council of Science Editors:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/zp38wf105


Wayne State University

6. Alharbi, Majed Abdullah. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.

Degree: MS, Pharmaceutical Sciences, 2015, Wayne State University

  Ubiquitin proteasome system is a relatively newly discovered pathway for protein degradation. Many studies have successfully pointed out the critical functions that UPS plays… (more)

Subjects/Keywords: Ubiquitination; Medicinal Chemistry and Pharmaceutics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alharbi, M. A. (2015). Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/435

Chicago Manual of Style (16th Edition):

Alharbi, Majed Abdullah. “Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.” 2015. Masters Thesis, Wayne State University. Accessed March 04, 2021. https://digitalcommons.wayne.edu/oa_theses/435.

MLA Handbook (7th Edition):

Alharbi, Majed Abdullah. “Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.” 2015. Web. 04 Mar 2021.

Vancouver:

Alharbi MA. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. [Internet] [Masters thesis]. Wayne State University; 2015. [cited 2021 Mar 04]. Available from: https://digitalcommons.wayne.edu/oa_theses/435.

Council of Science Editors:

Alharbi MA. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. [Masters Thesis]. Wayne State University; 2015. Available from: https://digitalcommons.wayne.edu/oa_theses/435


Queen Mary, University of London

7. Marks, Helen Margaret. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.

Degree: PhD, 2014, Queen Mary, University of London

Ubiquitination is an extremely important post-translational modification, regulating a wide variety of cellular processes including proteasomal degradation, subcellular targeting, endocytosis and DNA repair. The HECT… (more)

Subjects/Keywords: 616.99; Cancer; Prostate; Ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marks, H. M. (2014). Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265

Chicago Manual of Style (16th Edition):

Marks, Helen Margaret. “Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.” 2014. Doctoral Dissertation, Queen Mary, University of London. Accessed March 04, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265.

MLA Handbook (7th Edition):

Marks, Helen Margaret. “Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.” 2014. Web. 04 Mar 2021.

Vancouver:

Marks HM. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2014. [cited 2021 Mar 04]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265.

Council of Science Editors:

Marks HM. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2014. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265


Princeton University

8. Arlow, Tim. Ubiquitination of the DNA Mismatch Repair Protein Msh2 .

Degree: PhD, 2012, Princeton University

 MSH2 is required for DNA mismatch repair recognition in eukaryotes. Deleterious mutations in human MSH2 account for approximately half of the alleles associated with a… (more)

Subjects/Keywords: Bortezomib; DNA Mismatch Repair; Ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arlow, T. (2012). Ubiquitination of the DNA Mismatch Repair Protein Msh2 . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp012n49t172r

Chicago Manual of Style (16th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Doctoral Dissertation, Princeton University. Accessed March 04, 2021. http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

MLA Handbook (7th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Web. 04 Mar 2021.

Vancouver:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Internet] [Doctoral dissertation]. Princeton University; 2012. [cited 2021 Mar 04]. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

Council of Science Editors:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Doctoral Dissertation]. Princeton University; 2012. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r


University of Melbourne

9. Low, Ley Hian. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.

Degree: 2012, University of Melbourne

 The Nedd4 family interacting protein 1 (Ndfip1) is a neuroprotective protein, highly up-regulated in the neuron following brain injury. Many fundamental questions regarding the functions… (more)

Subjects/Keywords: Ndfip1; Nedd4; ubiquitination; PTEN

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Low, L. H. (2012). The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37781

Chicago Manual of Style (16th Edition):

Low, Ley Hian. “The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.” 2012. Doctoral Dissertation, University of Melbourne. Accessed March 04, 2021. http://hdl.handle.net/11343/37781.

MLA Handbook (7th Edition):

Low, Ley Hian. “The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.” 2012. Web. 04 Mar 2021.

Vancouver:

Low LH. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/11343/37781.

Council of Science Editors:

Low LH. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37781


University of Ottawa

10. Blinder, Anna. Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 .

Degree: 2020, University of Ottawa

 Pannexin 1 (PANX1) and Pannexin 3 (PANX3) are single-membrane channel glycoproteins that allow for communication between the cell and its environment to regulate cellular differentiation,… (more)

Subjects/Keywords: Pannexin; Ubiquitination; Proteasome; Degradation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blinder, A. (2020). Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blinder, Anna. “Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 .” 2020. Thesis, University of Ottawa. Accessed March 04, 2021. http://hdl.handle.net/10393/40278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blinder, Anna. “Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 .” 2020. Web. 04 Mar 2021.

Vancouver:

Blinder A. Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10393/40278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blinder A. Ubiquitination and Proteasomal Regulation of Pannexin 1 and Pannexin 3 . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

11. Furniss, James John. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.

Degree: PhD, 2016, University of Edinburgh

 Accurate regulation of gene transcription is essential for organismal survival, and is orchestrated by myriad transcription factors and cofactors (TFs). Little is known about how… (more)

Subjects/Keywords: 571.6; ubiquitination; transcription; Arabidopsis; immunity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Furniss, J. J. (2016). Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/22004

Chicago Manual of Style (16th Edition):

Furniss, James John. “Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed March 04, 2021. http://hdl.handle.net/1842/22004.

MLA Handbook (7th Edition):

Furniss, James John. “Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.” 2016. Web. 04 Mar 2021.

Vancouver:

Furniss JJ. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1842/22004.

Council of Science Editors:

Furniss JJ. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/22004


Vanderbilt University

12. Hang, Brian I. Kinase Regulation of XIAP in Wnt Signaling.

Degree: PhD, Cell and Developmental Biology, 2016, Vanderbilt University

 The Wnt signaling pathway plays essential roles in a wide variety of biological processes including early animal development, cell fate determination, cell proliferation, organogenesis, and… (more)

Subjects/Keywords: Wnt; XIAP; phosphorylation; ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hang, B. I. (2016). Kinase Regulation of XIAP in Wnt Signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12849

Chicago Manual of Style (16th Edition):

Hang, Brian I. “Kinase Regulation of XIAP in Wnt Signaling.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 04, 2021. http://hdl.handle.net/1803/12849.

MLA Handbook (7th Edition):

Hang, Brian I. “Kinase Regulation of XIAP in Wnt Signaling.” 2016. Web. 04 Mar 2021.

Vancouver:

Hang BI. Kinase Regulation of XIAP in Wnt Signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1803/12849.

Council of Science Editors:

Hang BI. Kinase Regulation of XIAP in Wnt Signaling. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/12849


University of Texas – Austin

13. Yeh, Ting-Chun. IAP antagonist Grim mediates cell death through divergent pathways.

Degree: PhD, Cell and Molecular Biology, 2014, University of Texas – Austin

 Apoptosis is an evolutionarily conserved process, carried out through proteolytic cascades that lead to activation of cysteinyl aspartate-specific proteases (caspases), which in turn cause cell… (more)

Subjects/Keywords: IAP; Grim; Ubiquitination; Caspase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yeh, T. (2014). IAP antagonist Grim mediates cell death through divergent pathways. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/44080

Chicago Manual of Style (16th Edition):

Yeh, Ting-Chun. “IAP antagonist Grim mediates cell death through divergent pathways.” 2014. Doctoral Dissertation, University of Texas – Austin. Accessed March 04, 2021. http://hdl.handle.net/2152/44080.

MLA Handbook (7th Edition):

Yeh, Ting-Chun. “IAP antagonist Grim mediates cell death through divergent pathways.” 2014. Web. 04 Mar 2021.

Vancouver:

Yeh T. IAP antagonist Grim mediates cell death through divergent pathways. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2014. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/2152/44080.

Council of Science Editors:

Yeh T. IAP antagonist Grim mediates cell death through divergent pathways. [Doctoral Dissertation]. University of Texas – Austin; 2014. Available from: http://hdl.handle.net/2152/44080


Université Paris-Sud – Paris XI

14. Robin, Charlotte. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.

Degree: Docteur es, Structure, fonction et ingénierie des protéines, 2011, Université Paris-Sud – Paris XI

Le virus de la mosaïque jaune du navet (TYMV) est un excellent modèle pour la réplication des virus à ARN simple brin de polarité positive.… (more)

Subjects/Keywords: Tymovirus; Réplication; Polymérase; Ubiquitination; Tymovirus; Replication; Polymerase; Proteinase; Ubiquitination; Crystallography

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robin, C. (2011). Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114853

Chicago Manual of Style (16th Edition):

Robin, Charlotte. “Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed March 04, 2021. http://www.theses.fr/2011PA114853.

MLA Handbook (7th Edition):

Robin, Charlotte. “Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.” 2011. Web. 04 Mar 2021.

Vancouver:

Robin C. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2011PA114853.

Council of Science Editors:

Robin C. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114853

15. Groslambert, Marine. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.

Degree: Docteur es, Sciences de la Vie, 2019, Lyon

L'inflammation est un processus déclenché suite à la détection de pathogènes et de dommages tissulaires. Elle conduit à la sécrétion de cytokines pro-inflammatoires par les… (more)

Subjects/Keywords: NLRP3; Inflammasome; Ubiquitination; Phosphorylation; NLRP3; Inflammasome; Ubiquitination; Phosphorylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Groslambert, M. (2019). Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSEN022

Chicago Manual of Style (16th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Doctoral Dissertation, Lyon. Accessed March 04, 2021. http://www.theses.fr/2019LYSEN022.

MLA Handbook (7th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Web. 04 Mar 2021.

Vancouver:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2019LYSEN022.

Council of Science Editors:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSEN022

16. El Hachem, Najla. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2017, Université Côte d'Azur (ComUE)

L’incidence du mélanome a augmenté de façon considérable lors des trente dernières années avec un doublement tous les dix ans. Le mélanome ne représente que… (more)

Subjects/Keywords: Ubiquitination; E3 ligase; Intégrines; Adhésion; Ubiquitination; E3 ligase; Integrins; Adhesion

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

El Hachem, N. (2017). Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2017AZUR4035

Chicago Manual of Style (16th Edition):

El Hachem, Najla. “Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.” 2017. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed March 04, 2021. http://www.theses.fr/2017AZUR4035.

MLA Handbook (7th Edition):

El Hachem, Najla. “Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.” 2017. Web. 04 Mar 2021.

Vancouver:

El Hachem N. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2017. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2017AZUR4035.

Council of Science Editors:

El Hachem N. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2017. Available from: http://www.theses.fr/2017AZUR4035

17. Martins, Sara. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.

Degree: Docteur es, Biologie, 2016, Université Paris-Saclay (ComUE)

 Les brassinostéroïdes (BR) sont des hormones stéroïdes végétales qui jouent un rôle dans la croissance et le développement des plantes. Ils sont perçus à la… (more)

Subjects/Keywords: Brassinostéroïdes; Endocytose; Ubiquitination; BRI1; Température; Brassinosteroids; Endocytosis; Ubiquitination; BRI1; Temperature

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martins, S. (2016). Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2016SACLS394

Chicago Manual of Style (16th Edition):

Martins, Sara. “Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.” 2016. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed March 04, 2021. http://www.theses.fr/2016SACLS394.

MLA Handbook (7th Edition):

Martins, Sara. “Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.” 2016. Web. 04 Mar 2021.

Vancouver:

Martins S. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2016. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2016SACLS394.

Council of Science Editors:

Martins S. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2016. Available from: http://www.theses.fr/2016SACLS394

18. Courivaud, Thomas. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.

Degree: Docteur es, Biologie, 2015, Université Pierre et Marie Curie – Paris VI

La voie de signalisation TGF-β joue un rôle biphasique durant la cancérogenèse. Mon laboratoire a identifié une nouvelle protéine inhibitrice de la voie TGF-β, WWP1.… (more)

Subjects/Keywords: WWP1; Tgf-β; Ubiquitination; RhoA; Stard13; Cancer; Cancer; Ubiquitination; 570

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Courivaud, T. (2015). Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066300

Chicago Manual of Style (16th Edition):

Courivaud, Thomas. “Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed March 04, 2021. http://www.theses.fr/2015PA066300.

MLA Handbook (7th Edition):

Courivaud, Thomas. “Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.” 2015. Web. 04 Mar 2021.

Vancouver:

Courivaud T. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2015PA066300.

Council of Science Editors:

Courivaud T. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066300

19. Jagline, Hélène. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

Les hétérotopies nodulaires périventriculaires (HNP) sont des malformations du cortex cérébral caractérisées par la formation d’amas de neurones dans des parties inappropriées du cerveau. Elles… (more)

Subjects/Keywords: Hétérotopies; Ubiquitination; Cortex; Heterotopia; Ubiquitination; Cortex; 572.8; 573.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jagline, H. (2017). Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ095

Chicago Manual of Style (16th Edition):

Jagline, Hélène. “Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed March 04, 2021. http://www.theses.fr/2017STRAJ095.

MLA Handbook (7th Edition):

Jagline, Hélène. “Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.” 2017. Web. 04 Mar 2021.

Vancouver:

Jagline H. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2021 Mar 04]. Available from: http://www.theses.fr/2017STRAJ095.

Council of Science Editors:

Jagline H. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ095


University of California – San Diego

20. Gallo, Leandro. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.

Degree: Chemistry, 2016, University of California – San Diego

 Inhibitor of kappaB kinase beta (IKKbeta) is the master regulatory kinase that modulates canonical nuclear factor kappaB (NFkappaB) inflammatory activation. Under inflammatory conditions, IKKbeta is… (more)

Subjects/Keywords: Molecular biology; Biology; Biochemistry; cancer; inflammation; ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gallo, L. (2016). Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/60j494r4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gallo, Leandro. “Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.” 2016. Thesis, University of California – San Diego. Accessed March 04, 2021. http://www.escholarship.org/uc/item/60j494r4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gallo, Leandro. “Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.” 2016. Web. 04 Mar 2021.

Vancouver:

Gallo L. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 Mar 04]. Available from: http://www.escholarship.org/uc/item/60j494r4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gallo L. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/60j494r4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

21. Sanada, Masashi. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

This paper was published in Nature 2009 Aug 13;460(7257):904-8. doi: 10.1038/nature08240. http://www.nature.com/nature/journal/v460/n7257/full/nature08240.html

新制・論文博士

乙第12855号

論医博第2085号

Subjects/Keywords: myelodysplasia; LOH; tumor suppressor; ubiquitination; cytokine sensitivity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sanada, M. (2014). Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Thesis, Kyoto University / 京都大学. Accessed March 04, 2021. http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Web. 04 Mar 2021.

Vancouver:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

22. Jones, Christine Marie. Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications.

Degree: PhD, Cell and Developmental Biology, 2018, Vanderbilt University

 Proper cell division to yield two daughter cells with identical complements of genomic material requires coordination between mitosis and cytokinesis. In the event of a… (more)

Subjects/Keywords: SIN; Dma1; auto-ubiquitination; S. pombe

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jones, C. M. (2018). Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10579

Chicago Manual of Style (16th Edition):

Jones, Christine Marie. “Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed March 04, 2021. http://hdl.handle.net/1803/10579.

MLA Handbook (7th Edition):

Jones, Christine Marie. “Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications.” 2018. Web. 04 Mar 2021.

Vancouver:

Jones CM. Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1803/10579.

Council of Science Editors:

Jones CM. Regulation of the key mitotic checkpoint protein Dma1 through post-translational modifications. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/10579


Texas A&M University

23. Avila Pacheco, Julian Ricardo, 1983-. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.

Degree: PhD, Biochemistry, 2012, Texas A&M University

 Programmed cell death (PCD) is an active process by which organisms coordinate the controlled destruction of cells. In tomato, the protein kinase Adi3 (AvrPto-dependent Pto-interacting… (more)

Subjects/Keywords: Ubiquitination; Phosphorylation; Tomato; Programmed cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Avila Pacheco, Julian Ricardo, 1. (2012). Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148132

Chicago Manual of Style (16th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Doctoral Dissertation, Texas A&M University. Accessed March 04, 2021. http://hdl.handle.net/1969.1/148132.

MLA Handbook (7th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Web. 04 Mar 2021.

Vancouver:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1969.1/148132.

Council of Science Editors:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148132


Texas A&M University

24. Zhou, Jinggeng. Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development.

Degree: PhD, Biochemistry, 2015, Texas A&M University

 Arabidopsis BRI1-associated receptor kinase1 (BAK1) is a signaling partner of multiple receptors involved in plant immunity, growth and cell death control. BAK1 contains an extracellular… (more)

Subjects/Keywords: Receptor; Ubiquitination; Phosphorylation; Cleavage; Immunity; Development

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, J. (2015). Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/187461

Chicago Manual of Style (16th Edition):

Zhou, Jinggeng. “Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development.” 2015. Doctoral Dissertation, Texas A&M University. Accessed March 04, 2021. http://hdl.handle.net/1969.1/187461.

MLA Handbook (7th Edition):

Zhou, Jinggeng. “Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development.” 2015. Web. 04 Mar 2021.

Vancouver:

Zhou J. Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development. [Internet] [Doctoral dissertation]. Texas A&M University; 2015. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1969.1/187461.

Council of Science Editors:

Zhou J. Post-translational Modifications of Arabidopsis Receptor-Like Kinases in Plant Immunity and Development. [Doctoral Dissertation]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/187461


Harvard University

25. Zhou, Alicia. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.

Degree: PhD, Biological and Biomedical Sciences, 2012, Harvard University

 The IkappaB kinase epsilon (IKKepsilon, IKKi, IKBKE) is both a regulator of innate immunity and a breast cancer oncogene that is amplified and overexpressed in… (more)

Subjects/Keywords: IKKepsilon; NF-kappaB; ubiquitination; biology; pathology; biochemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, A. (2012). Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028

Chicago Manual of Style (16th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Doctoral Dissertation, Harvard University. Accessed March 04, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

MLA Handbook (7th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Web. 04 Mar 2021.

Vancouver:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2021 Mar 04]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

Council of Science Editors:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028


Boston University

26. Guo, Ouyang. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.

Degree: MS, Anatomy & Neurobiology, 2016, Boston University

 Ubiquilin (UBQLN) is a member of type2 ubiquitin-like (UBL) protein family characterized by an UBL domain at the N-terminus and an ubiquitin associated (UBA) domain… (more)

Subjects/Keywords: Neurosciences; AMPAR; Neurodegenerative disease; Proteasome; Ubiquilin; Ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guo, O. (2016). The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17126

Chicago Manual of Style (16th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Masters Thesis, Boston University. Accessed March 04, 2021. http://hdl.handle.net/2144/17126.

MLA Handbook (7th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Web. 04 Mar 2021.

Vancouver:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/2144/17126.

Council of Science Editors:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/17126


Université Catholique de Louvain

27. Gualdron Lopez, Melisa. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.

Degree: 2012, Université Catholique de Louvain

 Trypanosoma brucei is the parasitic protist that is responsible for human sleeping sickness in Africa, a disease for which no adequate, affordable and harmless treatment… (more)

Subjects/Keywords: Trypanosoma brucei; Glycosome; Peroxin; PEX5; Ubiquitination.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gualdron Lopez, M. (2012). Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/112195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gualdron Lopez, Melisa. “Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.” 2012. Thesis, Université Catholique de Louvain. Accessed March 04, 2021. http://hdl.handle.net/2078.1/112195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gualdron Lopez, Melisa. “Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.” 2012. Web. 04 Mar 2021.

Vancouver:

Gualdron Lopez M. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. [Internet] [Thesis]. Université Catholique de Louvain; 2012. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/2078.1/112195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gualdron Lopez M. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. [Thesis]. Université Catholique de Louvain; 2012. Available from: http://hdl.handle.net/2078.1/112195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

28. Gil Mir, Maria Eugenia. Expanding the MDM2 interactome.

Degree: PhD, 2016, University of Edinburgh

 p53 is a key component of the protein network that regulates cell cycle progression and prevents cancer. Under non-stressed conditions, its activity is controlled by… (more)

Subjects/Keywords: 616.99; MDM2; interactome; ubiquitination; p53; OTUB2; TRIM25

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gil Mir, M. E. (2016). Expanding the MDM2 interactome. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25781

Chicago Manual of Style (16th Edition):

Gil Mir, Maria Eugenia. “Expanding the MDM2 interactome.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed March 04, 2021. http://hdl.handle.net/1842/25781.

MLA Handbook (7th Edition):

Gil Mir, Maria Eugenia. “Expanding the MDM2 interactome.” 2016. Web. 04 Mar 2021.

Vancouver:

Gil Mir ME. Expanding the MDM2 interactome. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1842/25781.

Council of Science Editors:

Gil Mir ME. Expanding the MDM2 interactome. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25781


University of Edinburgh

29. Yang, Lanlan. The role of ubiquitination of ERCC1 in DNA repair in melanoma.

Degree: PhD, 2015, University of Edinburgh

 Melanoma is one of the most common cancers in the world. For primary melanoma, early diagnosis and surgical excision are effective treatments but, despite the… (more)

Subjects/Keywords: 616.99; ERCC1; ubiquitination; DNA repair; melanoma

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, L. (2015). The role of ubiquitination of ERCC1 in DNA repair in melanoma. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18739

Chicago Manual of Style (16th Edition):

Yang, Lanlan. “The role of ubiquitination of ERCC1 in DNA repair in melanoma.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed March 04, 2021. http://hdl.handle.net/1842/18739.

MLA Handbook (7th Edition):

Yang, Lanlan. “The role of ubiquitination of ERCC1 in DNA repair in melanoma.” 2015. Web. 04 Mar 2021.

Vancouver:

Yang L. The role of ubiquitination of ERCC1 in DNA repair in melanoma. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/1842/18739.

Council of Science Editors:

Yang L. The role of ubiquitination of ERCC1 in DNA repair in melanoma. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/18739


University of the Western Cape

30. Jooste, Lauren Sarah. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .

Degree: 2015, University of the Western Cape

 P53 is one of the most important tumour suppressor proteins in the body which protects the cell against the tumourigenic effects of DNA damage by… (more)

Subjects/Keywords: Cancer; Tumour suppressor; Protein; In vitro; Ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jooste, L. S. (2015). In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5051

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jooste, Lauren Sarah. “In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .” 2015. Thesis, University of the Western Cape. Accessed March 04, 2021. http://hdl.handle.net/11394/5051.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jooste, Lauren Sarah. “In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .” 2015. Web. 04 Mar 2021.

Vancouver:

Jooste LS. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . [Internet] [Thesis]. University of the Western Cape; 2015. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/11394/5051.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jooste LS. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . [Thesis]. University of the Western Cape; 2015. Available from: http://hdl.handle.net/11394/5051

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [11]

.