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You searched for subject:(Ubiquitination). Showing records 1 – 30 of 263 total matches.

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University of Alberta

1. Abou Zeinab, Rami M. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.

Degree: PhD, Medical Sciences-Laboratory Medicine and Pathology, 2015, University of Alberta

 Cancer and tumor suppressors have been highly associated with many cancer researches. The main function of these proteins is to detect any error in DNA… (more)

Subjects/Keywords: ubiquitination; Pirh2; p73

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APA (6th Edition):

Abou Zeinab, R. M. (2015). The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cjq085k005

Chicago Manual of Style (16th Edition):

Abou Zeinab, Rami M. “The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.” 2015. Doctoral Dissertation, University of Alberta. Accessed August 25, 2019. https://era.library.ualberta.ca/files/cjq085k005.

MLA Handbook (7th Edition):

Abou Zeinab, Rami M. “The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation.” 2015. Web. 25 Aug 2019.

Vancouver:

Abou Zeinab RM. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2019 Aug 25]. Available from: https://era.library.ualberta.ca/files/cjq085k005.

Council of Science Editors:

Abou Zeinab RM. The Role of Pirh2 E3 Ligases in Ubiquitination and p73 Regulation. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/cjq085k005


University of Miami

2. Catoe, Heath Wesley. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2010, University of Miami

 The Estrogen Receptor alpha (ER alpha) is a multi-domain transcription factor that has been extensively studied due to its known involvement in breast cancer treatment… (more)

Subjects/Keywords: Degradation; Ubiquitination; Transcription

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APA (6th Edition):

Catoe, H. W. (2010). The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/464

Chicago Manual of Style (16th Edition):

Catoe, Heath Wesley. “The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.” 2010. Doctoral Dissertation, University of Miami. Accessed August 25, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/464.

MLA Handbook (7th Edition):

Catoe, Heath Wesley. “The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation.” 2010. Web. 25 Aug 2019.

Vancouver:

Catoe HW. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 Aug 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/464.

Council of Science Editors:

Catoe HW. The Role of E6-Associated Protein in Estrogen Receptor Alpha Regulation. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/464


University of Alberta

3. Mottet, Kelly. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The significance of poxvirus manipulation of the host ubiquitin proteasome system has become increasingly apparent. Ubiquitin is post-translationally added to target proteins by a highly… (more)

Subjects/Keywords: ligase; ubiquitination; proteasome; ubiquitin; poxvirus

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APA (6th Edition):

Mottet, K. (2010). The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/zp38wf105

Chicago Manual of Style (16th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Masters Thesis, University of Alberta. Accessed August 25, 2019. https://era.library.ualberta.ca/files/zp38wf105.

MLA Handbook (7th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Web. 25 Aug 2019.

Vancouver:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2019 Aug 25]. Available from: https://era.library.ualberta.ca/files/zp38wf105.

Council of Science Editors:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/zp38wf105


Princeton University

4. Arlow, Tim. Ubiquitination of the DNA Mismatch Repair Protein Msh2 .

Degree: PhD, 2012, Princeton University

 MSH2 is required for DNA mismatch repair recognition in eukaryotes. Deleterious mutations in human MSH2 account for approximately half of the alleles associated with a… (more)

Subjects/Keywords: Bortezomib; DNA Mismatch Repair; Ubiquitination

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APA (6th Edition):

Arlow, T. (2012). Ubiquitination of the DNA Mismatch Repair Protein Msh2 . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp012n49t172r

Chicago Manual of Style (16th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Doctoral Dissertation, Princeton University. Accessed August 25, 2019. http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

MLA Handbook (7th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Web. 25 Aug 2019.

Vancouver:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Internet] [Doctoral dissertation]. Princeton University; 2012. [cited 2019 Aug 25]. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

Council of Science Editors:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Doctoral Dissertation]. Princeton University; 2012. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r


Wayne State University

5. Alharbi, Majed Abdullah. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.

Degree: MS, Pharmaceutical Sciences, 2015, Wayne State University

  Ubiquitin proteasome system is a relatively newly discovered pathway for protein degradation. Many studies have successfully pointed out the critical functions that UPS plays… (more)

Subjects/Keywords: Ubiquitination; Medicinal Chemistry and Pharmaceutics

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APA (6th Edition):

Alharbi, M. A. (2015). Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/435

Chicago Manual of Style (16th Edition):

Alharbi, Majed Abdullah. “Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.” 2015. Masters Thesis, Wayne State University. Accessed August 25, 2019. https://digitalcommons.wayne.edu/oa_theses/435.

MLA Handbook (7th Edition):

Alharbi, Majed Abdullah. “Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions.” 2015. Web. 25 Aug 2019.

Vancouver:

Alharbi MA. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. [Internet] [Masters thesis]. Wayne State University; 2015. [cited 2019 Aug 25]. Available from: https://digitalcommons.wayne.edu/oa_theses/435.

Council of Science Editors:

Alharbi MA. Protein Ubiquitination In Primary Human Skeletal Muscle Cells Under Hyperinsulinemic Hyperglycemic Conditions. [Masters Thesis]. Wayne State University; 2015. Available from: https://digitalcommons.wayne.edu/oa_theses/435


University of Edinburgh

6. Furniss, James John. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.

Degree: PhD, 2016, University of Edinburgh

 Accurate regulation of gene transcription is essential for organismal survival, and is orchestrated by myriad transcription factors and cofactors (TFs). Little is known about how… (more)

Subjects/Keywords: 571.6; ubiquitination; transcription; Arabidopsis; immunity

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APA (6th Edition):

Furniss, J. J. (2016). Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/22004

Chicago Manual of Style (16th Edition):

Furniss, James John. “Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/22004.

MLA Handbook (7th Edition):

Furniss, James John. “Ubiquitin-ligase-mediated transcription initiation in cellular stress defences.” 2016. Web. 25 Aug 2019.

Vancouver:

Furniss JJ. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/22004.

Council of Science Editors:

Furniss JJ. Ubiquitin-ligase-mediated transcription initiation in cellular stress defences. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/22004


University of Texas – Austin

7. Yeh, Ting-Chun. IAP antagonist Grim mediates cell death through divergent pathways.

Degree: Cellular and Molecular Biology, 2014, University of Texas – Austin

 Apoptosis is an evolutionarily conserved process, carried out through proteolytic cascades that lead to activation of cysteinyl aspartate-specific proteases (caspases), which in turn cause cell… (more)

Subjects/Keywords: IAP; Grim; Ubiquitination; Caspase

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APA (6th Edition):

Yeh, T. (2014). IAP antagonist Grim mediates cell death through divergent pathways. (Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/44080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeh, Ting-Chun. “IAP antagonist Grim mediates cell death through divergent pathways.” 2014. Thesis, University of Texas – Austin. Accessed August 25, 2019. http://hdl.handle.net/2152/44080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeh, Ting-Chun. “IAP antagonist Grim mediates cell death through divergent pathways.” 2014. Web. 25 Aug 2019.

Vancouver:

Yeh T. IAP antagonist Grim mediates cell death through divergent pathways. [Internet] [Thesis]. University of Texas – Austin; 2014. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2152/44080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeh T. IAP antagonist Grim mediates cell death through divergent pathways. [Thesis]. University of Texas – Austin; 2014. Available from: http://hdl.handle.net/2152/44080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

8. Marks, Helen Margaret. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.

Degree: PhD, 2014, Queen Mary, University of London

Ubiquitination is an extremely important post-translational modification, regulating a wide variety of cellular processes including proteasomal degradation, subcellular targeting, endocytosis and DNA repair. The HECT… (more)

Subjects/Keywords: 616.99; Cancer; Prostate; Ubiquitination

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APA (6th Edition):

Marks, H. M. (2014). Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265

Chicago Manual of Style (16th Edition):

Marks, Helen Margaret. “Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.” 2014. Doctoral Dissertation, Queen Mary, University of London. Accessed August 25, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265.

MLA Handbook (7th Edition):

Marks, Helen Margaret. “Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer.” 2014. Web. 25 Aug 2019.

Vancouver:

Marks HM. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2014. [cited 2019 Aug 25]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265.

Council of Science Editors:

Marks HM. Investigating the Nedd4-mediated ubiquitination of PMEPA1, and its potential role in the regulation of the androgen receptor as part of the steroid response pathway in prostatic cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2014. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8578 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667265

9. Jagline, Hélène. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

Les hétérotopies nodulaires périventriculaires (HNP) sont des malformations du cortex cérébral caractérisées par la formation d’amas de neurones dans des parties inappropriées du cerveau. Elles… (more)

Subjects/Keywords: Hétérotopies; Ubiquitination; Cortex; Heterotopia; Ubiquitination; Cortex; 572.8; 573.8

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APA (6th Edition):

Jagline, H. (2017). Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ095

Chicago Manual of Style (16th Edition):

Jagline, Hélène. “Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed August 25, 2019. http://www.theses.fr/2017STRAJ095.

MLA Handbook (7th Edition):

Jagline, Hélène. “Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene.” 2017. Web. 25 Aug 2019.

Vancouver:

Jagline H. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2019 Aug 25]. Available from: http://www.theses.fr/2017STRAJ095.

Council of Science Editors:

Jagline H. Compréhension des mécanismes physiopathologiques des hétérotopies nodulaires périventriculaires associées à des mutations dans le gène NEDD4L : Understanding of the pathophysiological mechanisms of periventricular nodular heterotopias associated with mutations in the NEDD4L gene. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ095

10. El Hachem, Najla. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2017, Côte d'Azur

L’incidence du mélanome a augmenté de façon considérable lors des trente dernières années avec un doublement tous les dix ans. Le mélanome ne représente que… (more)

Subjects/Keywords: Ubiquitination; E3 ligase; Intégrines; Adhésion; Ubiquitination; E3 ligase; Integrins; Adhesion

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APA (6th Edition):

El Hachem, N. (2017). Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. (Doctoral Dissertation). Côte d'Azur. Retrieved from http://www.theses.fr/2017AZUR4035

Chicago Manual of Style (16th Edition):

El Hachem, Najla. “Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.” 2017. Doctoral Dissertation, Côte d'Azur. Accessed August 25, 2019. http://www.theses.fr/2017AZUR4035.

MLA Handbook (7th Edition):

El Hachem, Najla. “Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma.” 2017. Web. 25 Aug 2019.

Vancouver:

El Hachem N. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. [Internet] [Doctoral dissertation]. Côte d'Azur; 2017. [cited 2019 Aug 25]. Available from: http://www.theses.fr/2017AZUR4035.

Council of Science Editors:

El Hachem N. Rôle pro-tumorigénique de HACE1 dans le mélanome : Pro-tumorigenic role of HACE1 in melanoma. [Doctoral Dissertation]. Côte d'Azur; 2017. Available from: http://www.theses.fr/2017AZUR4035

11. Courivaud, Thomas. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.

Degree: Docteur es, Biologie, 2015, Université Pierre et Marie Curie – Paris VI

La voie de signalisation TGF-β joue un rôle biphasique durant la cancérogenèse. Mon laboratoire a identifié une nouvelle protéine inhibitrice de la voie TGF-β, WWP1.… (more)

Subjects/Keywords: WWP1; Tgf-β; Ubiquitination; RhoA; Stard13; Cancer; Cancer; Ubiquitination; 570

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APA (6th Edition):

Courivaud, T. (2015). Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066300

Chicago Manual of Style (16th Edition):

Courivaud, Thomas. “Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed August 25, 2019. http://www.theses.fr/2015PA066300.

MLA Handbook (7th Edition):

Courivaud, Thomas. “Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis.” 2015. Web. 25 Aug 2019.

Vancouver:

Courivaud T. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Aug 25]. Available from: http://www.theses.fr/2015PA066300.

Council of Science Editors:

Courivaud T. Caractérisation d'un nouveau mécanisme d'action de la E3 ubiquitine ligase WWP1 et régulation de son activité dans la cancérogenèse : Characterization of a new mecanism of the E3 ubiquitin ligase WWP1 and regulation of its activity during cancerogenesis. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066300

12. Martins, Sara. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.

Degree: Docteur es, Biologie, 2016, Paris Saclay

 Les brassinostéroïdes (BR) sont des hormones stéroïdes végétales qui jouent un rôle dans la croissance et le développement des plantes. Ils sont perçus à la… (more)

Subjects/Keywords: Brassinostéroïdes; Endocytose; Ubiquitination; BRI1; Température; Brassinosteroids; Endocytosis; Ubiquitination; BRI1; Temperature

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APA (6th Edition):

Martins, S. (2016). Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2016SACLS394

Chicago Manual of Style (16th Edition):

Martins, Sara. “Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.” 2016. Doctoral Dissertation, Paris Saclay. Accessed August 25, 2019. http://www.theses.fr/2016SACLS394.

MLA Handbook (7th Edition):

Martins, Sara. “Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température.” 2016. Web. 25 Aug 2019.

Vancouver:

Martins S. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. [Internet] [Doctoral dissertation]. Paris Saclay; 2016. [cited 2019 Aug 25]. Available from: http://www.theses.fr/2016SACLS394.

Council of Science Editors:

Martins S. Ubiquitin-mediated endocytosis of the plant steroid hormone receptor BRI1 and regulation by elevated ambient temperature : Étude de l'endocytose dépendante de l'ubiquitine du récepteur aux hormones stéroïdes végétales BRI1 et de sa régulation par la température. [Doctoral Dissertation]. Paris Saclay; 2016. Available from: http://www.theses.fr/2016SACLS394


Université Paris-Sud – Paris XI

13. Robin, Charlotte. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.

Degree: Docteur es, Structure, fonction et ingénierie des protéines, 2011, Université Paris-Sud – Paris XI

Le virus de la mosaïque jaune du navet (TYMV) est un excellent modèle pour la réplication des virus à ARN simple brin de polarité positive.… (more)

Subjects/Keywords: Tymovirus; Réplication; Polymérase; Ubiquitination; Tymovirus; Replication; Polymerase; Proteinase; Ubiquitination; Crystallography

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APA (6th Edition):

Robin, C. (2011). Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114853

Chicago Manual of Style (16th Edition):

Robin, Charlotte. “Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed August 25, 2019. http://www.theses.fr/2011PA114853.

MLA Handbook (7th Edition):

Robin, Charlotte. “Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus.” 2011. Web. 25 Aug 2019.

Vancouver:

Robin C. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Aug 25]. Available from: http://www.theses.fr/2011PA114853.

Council of Science Editors:

Robin C. Etude structurale de protéines virales impliquées dans la réplication et régulation du cycle de multiplication d'un virus de plante : Structural studies of viral proteins involved in replication and regulation of the multiplication cycle in a plant virus. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114853


Texas Medical Center

14. Su, Chun-Hui. 14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1.

Degree: PhD, 2010, Texas Medical Center

 Mammalian constitutive photomorphogenic 1 (COP1), a p53 E3 ubiquitin ligase, is a key negative regulator for p53. DNA damage leads to the translocation of COP1… (more)

Subjects/Keywords: COP1; p53; 14-3-3sigma; ubiquitination; Biology

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APA (6th Edition):

Su, C. (2010). 14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/101

Chicago Manual of Style (16th Edition):

Su, Chun-Hui. “14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed August 25, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/101.

MLA Handbook (7th Edition):

Su, Chun-Hui. “14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1.” 2010. Web. 25 Aug 2019.

Vancouver:

Su C. 14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2019 Aug 25]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/101.

Council of Science Editors:

Su C. 14-3-3SIGMA NEGATIVELY REGULATES THE STABILITY AND SUBCELLULAR LOCALIZATION OF COP1. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/101


Texas A&M University

15. Avila Pacheco, Julian Ricardo, 1983-. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.

Degree: 2012, Texas A&M University

 Programmed cell death (PCD) is an active process by which organisms coordinate the controlled destruction of cells. In tomato, the protein kinase Adi3 (AvrPto-dependent Pto-interacting… (more)

Subjects/Keywords: Ubiquitination; Phosphorylation; Tomato; Programmed cell death

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APA (6th Edition):

Avila Pacheco, Julian Ricardo, 1. (2012). Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Thesis, Texas A&M University. Accessed August 25, 2019. http://hdl.handle.net/1969.1/148132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Web. 25 Aug 2019.

Vancouver:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1969.1/148132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

16. Sanada, Masashi. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

This paper was published in Nature 2009 Aug 13;460(7257):904-8. doi: 10.1038/nature08240. http://www.nature.com/nature/journal/v460/n7257/full/nature08240.html

新制・論文博士

乙第12855号

論医博第2085号

Subjects/Keywords: myelodysplasia; LOH; tumor suppressor; ubiquitination; cytokine sensitivity

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APA (6th Edition):

Sanada, M. (2014). Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Thesis, Kyoto University / 京都大学. Accessed August 25, 2019. http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Web. 25 Aug 2019.

Vancouver:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Δασκαλάκη, Αικατερίνη. Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

The Notch signaling pathway is an evolutionary conserved mechanism which participates in a variety of cell fate decisions. In D. melanogaster, key-players in this signaling… (more)

Subjects/Keywords: Δροσόφιλα; Ενδοκύττωση; Ουβικουιτινυλίωση; Endocytosis; Mindbomb1; Neuralized; Ubiquitination

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APA (6th Edition):

Δασκαλάκη, . . (2012). Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/28060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Δασκαλάκη, Αικατερίνη. “Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed August 25, 2019. http://hdl.handle.net/10442/hedi/28060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Δασκαλάκη, Αικατερίνη. “Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch.” 2012. Web. 25 Aug 2019.

Vancouver:

Δασκαλάκη . Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/10442/hedi/28060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Δασκαλάκη . Ο ρόλος των ενδοκυττάριων μοτίβων της πρωτεΐνης Delta στη σηματοδότηση Notch. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/28060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

18. Wang, Shao-Fang. Biochemical and biophysical studies of MDM2-ligand interactions.

Degree: PhD, 2012, University of Edinburgh

 MDM2, murine double minute 2, is a RING type-E3 ligase protein and also an oncogene. MDM2 plays a critical role in determining the steady levels… (more)

Subjects/Keywords: MDM2; virtual screening; ligand; E3 ligase; ubiquitination

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, S. (2012). Biochemical and biophysical studies of MDM2-ligand interactions. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9527

Chicago Manual of Style (16th Edition):

Wang, Shao-Fang. “Biochemical and biophysical studies of MDM2-ligand interactions.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/9527.

MLA Handbook (7th Edition):

Wang, Shao-Fang. “Biochemical and biophysical studies of MDM2-ligand interactions.” 2012. Web. 25 Aug 2019.

Vancouver:

Wang S. Biochemical and biophysical studies of MDM2-ligand interactions. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/9527.

Council of Science Editors:

Wang S. Biochemical and biophysical studies of MDM2-ligand interactions. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/9527


University of California – San Diego

19. Gallo, Leandro. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.

Degree: Chemistry, 2016, University of California – San Diego

 Inhibitor of kappaB kinase beta (IKKbeta) is the master regulatory kinase that modulates canonical nuclear factor kappaB (NFkappaB) inflammatory activation. Under inflammatory conditions, IKKbeta is… (more)

Subjects/Keywords: Molecular biology; Biology; Biochemistry; cancer; inflammation; ubiquitination

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APA (6th Edition):

Gallo, L. (2016). Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/60j494r4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gallo, Leandro. “Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.” 2016. Thesis, University of California – San Diego. Accessed August 25, 2019. http://www.escholarship.org/uc/item/60j494r4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gallo, Leandro. “Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling.” 2016. Web. 25 Aug 2019.

Vancouver:

Gallo L. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Aug 25]. Available from: http://www.escholarship.org/uc/item/60j494r4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gallo L. Activating mutations of Lys171 in the kinase domain of IKKbeta unleash a novel mechanism of oncogenic signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/60j494r4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

20. Aleidi, Shereen Mohammad Suleimann. Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases .

Degree: 2016, University of Sydney

 This thesis present a series of experimental approaches that aim to characterize the roles of three E3 ubiquitin ligase enzymes in the post-translational regulation of… (more)

Subjects/Keywords: ubiquitination; ABC transporters; lipid; post-translational; regulation

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APA (6th Edition):

Aleidi, S. M. S. (2016). Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/15477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aleidi, Shereen Mohammad Suleimann. “Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases .” 2016. Thesis, University of Sydney. Accessed August 25, 2019. http://hdl.handle.net/2123/15477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aleidi, Shereen Mohammad Suleimann. “Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases .” 2016. Web. 25 Aug 2019.

Vancouver:

Aleidi SMS. Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases . [Internet] [Thesis]. University of Sydney; 2016. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2123/15477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aleidi SMS. Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/15477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

21. Procter, Dean Joseph. Genetic contribution to cytopathic effect in Vaccinia virus .

Degree: 2014, University of Sydney

 Vaccinia virus (VACV) infection induces cell migration, the formation of cytoplasmic extensions, actin polymerisation, cell rounding, detachment and lysis—changes collectively known as cytopathic effects (CPEs).… (more)

Subjects/Keywords: Poxvirus; Vaccinia; Cullin-3; Ubiquitination; BTB; Kelch

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APA (6th Edition):

Procter, D. J. (2014). Genetic contribution to cytopathic effect in Vaccinia virus . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/12502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Procter, Dean Joseph. “Genetic contribution to cytopathic effect in Vaccinia virus .” 2014. Thesis, University of Sydney. Accessed August 25, 2019. http://hdl.handle.net/2123/12502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Procter, Dean Joseph. “Genetic contribution to cytopathic effect in Vaccinia virus .” 2014. Web. 25 Aug 2019.

Vancouver:

Procter DJ. Genetic contribution to cytopathic effect in Vaccinia virus . [Internet] [Thesis]. University of Sydney; 2014. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2123/12502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Procter DJ. Genetic contribution to cytopathic effect in Vaccinia virus . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/12502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

22. Landré, Vivien. Regulation and effects of IRF-1 and p53 ubiquitination.

Degree: PhD, 2013, University of Edinburgh

 Protein ubiquitination is a key regulator of both protein stability and activity, and is involved in the regulation of a vast variety of cellular pathways.… (more)

Subjects/Keywords: 612; ubiquitination; transcription; E3 ligases; allostery

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APA (6th Edition):

Landré, V. (2013). Regulation and effects of IRF-1 and p53 ubiquitination. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/10639

Chicago Manual of Style (16th Edition):

Landré, Vivien. “Regulation and effects of IRF-1 and p53 ubiquitination.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/10639.

MLA Handbook (7th Edition):

Landré, Vivien. “Regulation and effects of IRF-1 and p53 ubiquitination.” 2013. Web. 25 Aug 2019.

Vancouver:

Landré V. Regulation and effects of IRF-1 and p53 ubiquitination. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/10639.

Council of Science Editors:

Landré V. Regulation and effects of IRF-1 and p53 ubiquitination. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/10639


Harvard University

23. Zhou, Alicia. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.

Degree: PhD, Biological and Biomedical Sciences, 2012, Harvard University

 The IkappaB kinase epsilon (IKKepsilon, IKKi, IKBKE) is both a regulator of innate immunity and a breast cancer oncogene that is amplified and overexpressed in… (more)

Subjects/Keywords: IKKepsilon; NF-kappaB; ubiquitination; biology; pathology; biochemistry

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APA (6th Edition):

Zhou, A. (2012). Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028

Chicago Manual of Style (16th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Doctoral Dissertation, Harvard University. Accessed August 25, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

MLA Handbook (7th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Web. 25 Aug 2019.

Vancouver:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2019 Aug 25]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

Council of Science Editors:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028


The Ohio State University

24. Shirsekar, Gautam Shashikant. Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>).

Degree: PhD, Plant Pathology, 2013, The Ohio State University

Ubiquitination is a vital cellular biochemical process that has been shown to be involved in growth and development of eukaryotic organisms, including plants. The… (more)

Subjects/Keywords: Plant Pathology; ubiquitination, rice, innate immunity, plant

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APA (6th Edition):

Shirsekar, G. S. (2013). Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>). (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1383664921

Chicago Manual of Style (16th Edition):

Shirsekar, Gautam Shashikant. “Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>).” 2013. Doctoral Dissertation, The Ohio State University. Accessed August 25, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1383664921.

MLA Handbook (7th Edition):

Shirsekar, Gautam Shashikant. “Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>).” 2013. Web. 25 Aug 2019.

Vancouver:

Shirsekar GS. Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>). [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Aug 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1383664921.

Council of Science Editors:

Shirsekar GS. Ubiquitination in Innate Immunity of Rice (<i>Oryza sativa</i>). [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1383664921


Université Catholique de Louvain

25. Gualdron Lopez, Melisa. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.

Degree: 2012, Université Catholique de Louvain

 Trypanosoma brucei is the parasitic protist that is responsible for human sleeping sickness in Africa, a disease for which no adequate, affordable and harmless treatment… (more)

Subjects/Keywords: Trypanosoma brucei; Glycosome; Peroxin; PEX5; Ubiquitination.

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APA (6th Edition):

Gualdron Lopez, M. (2012). Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/112195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gualdron Lopez, Melisa. “Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.” 2012. Thesis, Université Catholique de Louvain. Accessed August 25, 2019. http://hdl.handle.net/2078.1/112195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gualdron Lopez, Melisa. “Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei.” 2012. Web. 25 Aug 2019.

Vancouver:

Gualdron Lopez M. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. [Internet] [Thesis]. Université Catholique de Louvain; 2012. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2078.1/112195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gualdron Lopez M. Study of the molecular mechanism involved in recycling of matrix protein receptor, PEX5, during glycosome biogenesis in Trypanosoma brucei. [Thesis]. Université Catholique de Louvain; 2012. Available from: http://hdl.handle.net/2078.1/112195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

26. Guo, Ouyang. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.

Degree: MS, Anatomy & Neurobiology, 2016, Boston University

 Ubiquilin (UBQLN) is a member of type2 ubiquitin-like (UBL) protein family characterized by an UBL domain at the N-terminus and an ubiquitin associated (UBA) domain… (more)

Subjects/Keywords: Neurosciences; AMPAR; Neurodegenerative disease; Proteasome; Ubiquilin; Ubiquitination

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APA (6th Edition):

Guo, O. (2016). The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17126

Chicago Manual of Style (16th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Masters Thesis, Boston University. Accessed August 25, 2019. http://hdl.handle.net/2144/17126.

MLA Handbook (7th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Web. 25 Aug 2019.

Vancouver:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Internet] [Masters thesis]. Boston University; 2016. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/2144/17126.

Council of Science Editors:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/17126


University of Edinburgh

27. Koszela, Joanna. Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes.

Degree: PhD, 2014, University of Edinburgh

 The ubiquitin-proteasome system (UPS) controls the stability, activity and localisation of most of the proteome and regulates virtually all cellular processes through modification of proteins… (more)

Subjects/Keywords: 572; ubiquitination; E2 enzymes; high-throughput screening

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APA (6th Edition):

Koszela, J. (2014). Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17893

Chicago Manual of Style (16th Edition):

Koszela, Joanna. “Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/17893.

MLA Handbook (7th Edition):

Koszela, Joanna. “Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes.” 2014. Web. 25 Aug 2019.

Vancouver:

Koszela J. Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/17893.

Council of Science Editors:

Koszela J. Novel screening methods for inhibitors of the human ubiquitin-conjugating enzymes. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/17893


University of Edinburgh

28. Yang, Lanlan. The role of ubiquitination of ERCC1 in DNA repair in melanoma.

Degree: PhD, 2015, University of Edinburgh

 Melanoma is one of the most common cancers in the world. For primary melanoma, early diagnosis and surgical excision are effective treatments but, despite the… (more)

Subjects/Keywords: 616.99; ERCC1; ubiquitination; DNA repair; melanoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, L. (2015). The role of ubiquitination of ERCC1 in DNA repair in melanoma. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18739

Chicago Manual of Style (16th Edition):

Yang, Lanlan. “The role of ubiquitination of ERCC1 in DNA repair in melanoma.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/18739.

MLA Handbook (7th Edition):

Yang, Lanlan. “The role of ubiquitination of ERCC1 in DNA repair in melanoma.” 2015. Web. 25 Aug 2019.

Vancouver:

Yang L. The role of ubiquitination of ERCC1 in DNA repair in melanoma. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/18739.

Council of Science Editors:

Yang L. The role of ubiquitination of ERCC1 in DNA repair in melanoma. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/18739


University of Edinburgh

29. Gil Mir, Maria Eugenia. Expanding the MDM2 interactome.

Degree: PhD, 2016, University of Edinburgh

 p53 is a key component of the protein network that regulates cell cycle progression and prevents cancer. Under non-stressed conditions, its activity is controlled by… (more)

Subjects/Keywords: MDM2; interactome; ubiquitination; p53; OTUB2; TRIM25

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APA (6th Edition):

Gil Mir, M. E. (2016). Expanding the MDM2 interactome. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25781

Chicago Manual of Style (16th Edition):

Gil Mir, Maria Eugenia. “Expanding the MDM2 interactome.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed August 25, 2019. http://hdl.handle.net/1842/25781.

MLA Handbook (7th Edition):

Gil Mir, Maria Eugenia. “Expanding the MDM2 interactome.” 2016. Web. 25 Aug 2019.

Vancouver:

Gil Mir ME. Expanding the MDM2 interactome. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1842/25781.

Council of Science Editors:

Gil Mir ME. Expanding the MDM2 interactome. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25781


University of the Western Cape

30. Jooste, Lauren Sarah. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .

Degree: 2015, University of the Western Cape

 P53 is one of the most important tumour suppressor proteins in the body which protects the cell against the tumourigenic effects of DNA damage by… (more)

Subjects/Keywords: Cancer; Tumour suppressor; Protein; In vitro; Ubiquitination

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APA (6th Edition):

Jooste, L. S. (2015). In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5051

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jooste, Lauren Sarah. “In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .” 2015. Thesis, University of the Western Cape. Accessed August 25, 2019. http://hdl.handle.net/11394/5051.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jooste, Lauren Sarah. “In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) .” 2015. Web. 25 Aug 2019.

Vancouver:

Jooste LS. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . [Internet] [Thesis]. University of the Western Cape; 2015. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/11394/5051.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jooste LS. In vitro investigation of the ubiquitination and degradation of p53 by Murine Double Minute 2 (MDM2) and Retinoblastoma Binding Protein 6 (RBBP6) . [Thesis]. University of the Western Cape; 2015. Available from: http://hdl.handle.net/11394/5051

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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