Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Ubiquitin). Showing records 1 – 30 of 682 total matches.

[1] [2] [3] [4] [5] … [23]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Minnesota

1. Randles, Leah Ann. Defining how the 26S proteasome recognizes ubiquitinated substrates.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2012, University of Minnesota

 Regulated protein degradation in eukaryotes is performed predominantly by the ubiquitin-proteasome pathway. Prior to their degradation by the 26S proteasome, protein substrates become covalently modified… (more)

Subjects/Keywords: Proteasome; Ubiquitin; Ubiquitin Receptor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Randles, L. A. (2012). Defining how the 26S proteasome recognizes ubiquitinated substrates. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/162241

Chicago Manual of Style (16th Edition):

Randles, Leah Ann. “Defining how the 26S proteasome recognizes ubiquitinated substrates.” 2012. Doctoral Dissertation, University of Minnesota. Accessed June 16, 2019. http://hdl.handle.net/11299/162241.

MLA Handbook (7th Edition):

Randles, Leah Ann. “Defining how the 26S proteasome recognizes ubiquitinated substrates.” 2012. Web. 16 Jun 2019.

Vancouver:

Randles LA. Defining how the 26S proteasome recognizes ubiquitinated substrates. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/11299/162241.

Council of Science Editors:

Randles LA. Defining how the 26S proteasome recognizes ubiquitinated substrates. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://hdl.handle.net/11299/162241


University of Dundee

2. Leidecker, Orsolya. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.

Degree: PhD, 2012, University of Dundee

 NEDD8 modification of proteins is extensively studied in the recent years, and the ubiquitin-like molecule has been shown to be involved in numerous signalling pathways.… (more)

Subjects/Keywords: nedd8; ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leidecker, O. (2012). Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf

Chicago Manual of Style (16th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Doctoral Dissertation, University of Dundee. Accessed June 16, 2019. http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf.

MLA Handbook (7th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Web. 16 Jun 2019.

Vancouver:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf.

Council of Science Editors:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Doctoral Dissertation]. University of Dundee; 2012. Available from: http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf


Penn State University

3. Davidshofer, Kristine C. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.

Degree: PhD, Physiology, 2008, Penn State University

Ubiquitin is a highly conserved eukaryotic protein of 76 amino acids that is added post-translationally to other proteins or to itself by a hierarchical cascade… (more)

Subjects/Keywords: RING domain; ubiquitin; ubiquitin conjugating enzyme

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Davidshofer, K. C. (2008). ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8500

Chicago Manual of Style (16th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Doctoral Dissertation, Penn State University. Accessed June 16, 2019. https://etda.libraries.psu.edu/catalog/8500.

MLA Handbook (7th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Web. 16 Jun 2019.

Vancouver:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2019 Jun 16]. Available from: https://etda.libraries.psu.edu/catalog/8500.

Council of Science Editors:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8500


University of Hong Kong

4. 王琳; Wang, Lin. The role of MDM2 in hepatic lipid metabolism.

Degree: Master of Medical Sciences, 2016, University of Hong Kong

 Background and objective: Liver is a core organ in regulation of lipid homeostasis, which is vital for life and health. Obesity is closely associated with… (more)

Subjects/Keywords: Ubiquitin; Lipids - Metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

王琳; Wang, L. (2016). The role of MDM2 in hepatic lipid metabolism. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/236279

Chicago Manual of Style (16th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Masters Thesis, University of Hong Kong. Accessed June 16, 2019. http://hdl.handle.net/10722/236279.

MLA Handbook (7th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Web. 16 Jun 2019.

Vancouver:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10722/236279.

Council of Science Editors:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Masters Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/236279


University of Hong Kong

5. 成云; Cheng, Yun. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.

Degree: PhD, 2016, University of Hong Kong

 CREB-H is an endoplasmic reticulum-tethered bZIP transcription factor, which critically regulates lipid homeostasis and gluconeogenesis in the liver. CREB-H is proteolytically activated by regulated intramembrane… (more)

Subjects/Keywords: Transcription factors; Ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

成云; Cheng, Y. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862

Chicago Manual of Style (16th Edition):

成云; Cheng, Yun. “β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed June 16, 2019. Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862.

MLA Handbook (7th Edition):

成云; Cheng, Yun. “β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.” 2016. Web. 16 Jun 2019.

Vancouver:

成云; Cheng Y. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Jun 16]. Available from: Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862.

Council of Science Editors:

成云; Cheng Y. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862


University of Hong Kong

6. Li, Tao. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.

Degree: M. Phil., 2016, University of Hong Kong

DNA methylation is a crucial epigenetic modification and functions as a key factor in controlling gene expression and mammalian development. The DNA methyltransferase DNMT1 is… (more)

Subjects/Keywords: Ubiquitin; DNA - Methylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, T. (2016). Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/240653

Chicago Manual of Style (16th Edition):

Li, Tao. “Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.” 2016. Masters Thesis, University of Hong Kong. Accessed June 16, 2019. http://hdl.handle.net/10722/240653.

MLA Handbook (7th Edition):

Li, Tao. “Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.” 2016. Web. 16 Jun 2019.

Vancouver:

Li T. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10722/240653.

Council of Science Editors:

Li T. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. [Masters Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/240653


University of Hong Kong

7. Ruan, Yafei. Investigating the regulation of UHRF1 in cell cycle.

Degree: Master of Medical Sciences, 2015, University of Hong Kong

UHRF1 is a multi-domain protein with multiple functions in mammalian cells including maintenance of DNA methylation, histone modification, DNA damage et al. UHRF1 itself also… (more)

Subjects/Keywords: Cell cycle; Ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ruan, Y. (2015). Investigating the regulation of UHRF1 in cell cycle. (Masters Thesis). University of Hong Kong. Retrieved from Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484

Chicago Manual of Style (16th Edition):

Ruan, Yafei. “Investigating the regulation of UHRF1 in cell cycle.” 2015. Masters Thesis, University of Hong Kong. Accessed June 16, 2019. Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484.

MLA Handbook (7th Edition):

Ruan, Yafei. “Investigating the regulation of UHRF1 in cell cycle.” 2015. Web. 16 Jun 2019.

Vancouver:

Ruan Y. Investigating the regulation of UHRF1 in cell cycle. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Jun 16]. Available from: Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484.

Council of Science Editors:

Ruan Y. Investigating the regulation of UHRF1 in cell cycle. [Masters Thesis]. University of Hong Kong; 2015. Available from: Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484


University of Hong Kong

8. Tucker, Wesley Owen. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.

Degree: PhD, 2013, University of Hong Kong

DNA aptamers have been studied since their inception in 1990, but have only targeted membrane and serum proteins in therapeutics. Their potential as inhibitors of… (more)

Subjects/Keywords: Ubiquitin; Osteoblasts; Oligonucleotides

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tucker, W. O. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640

Chicago Manual of Style (16th Edition):

Tucker, Wesley Owen. “Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed June 16, 2019. Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640.

MLA Handbook (7th Edition):

Tucker, Wesley Owen. “Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.” 2013. Web. 16 Jun 2019.

Vancouver:

Tucker WO. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Jun 16]. Available from: Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640.

Council of Science Editors:

Tucker WO. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640


Penn State University

9. Ellis, Christopher Ross. Simulation studies of the glycosylation code.

Degree: PhD, Chemistry, 2014, Penn State University

 This thesis reports computational studies that investigate the impact of protein- carbohydrate and protein-protein interactions upon protein structure and folding. The core of this thesis,… (more)

Subjects/Keywords: glycosylation; toluene; ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ellis, C. R. (2014). Simulation studies of the glycosylation code. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/22562

Chicago Manual of Style (16th Edition):

Ellis, Christopher Ross. “Simulation studies of the glycosylation code.” 2014. Doctoral Dissertation, Penn State University. Accessed June 16, 2019. https://etda.libraries.psu.edu/catalog/22562.

MLA Handbook (7th Edition):

Ellis, Christopher Ross. “Simulation studies of the glycosylation code.” 2014. Web. 16 Jun 2019.

Vancouver:

Ellis CR. Simulation studies of the glycosylation code. [Internet] [Doctoral dissertation]. Penn State University; 2014. [cited 2019 Jun 16]. Available from: https://etda.libraries.psu.edu/catalog/22562.

Council of Science Editors:

Ellis CR. Simulation studies of the glycosylation code. [Doctoral Dissertation]. Penn State University; 2014. Available from: https://etda.libraries.psu.edu/catalog/22562


University of California – San Diego

10. Gonzales, Frankie Robert. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.

Degree: Chemistry, 2017, University of California – San Diego

 Protein homeostasis in is critical to maintain cell health and viability. Protein homeostasis can be divided into two major categories: protein synthesis, and protein degradation.… (more)

Subjects/Keywords: Biochemistry; Proteasome; Ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gonzales, F. R. (2017). Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/6958j596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonzales, Frankie Robert. “Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.” 2017. Thesis, University of California – San Diego. Accessed June 16, 2019. http://www.escholarship.org/uc/item/6958j596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonzales, Frankie Robert. “Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.” 2017. Web. 16 Jun 2019.

Vancouver:

Gonzales FR. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Jun 16]. Available from: http://www.escholarship.org/uc/item/6958j596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonzales FR. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/6958j596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

11. Leidecker, Orsolya. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.

Degree: PhD, 2012, University of Dundee

 NEDD8 modification of proteins is extensively studied in the recent years, and the ubiquitin-like molecule has been shown to be involved in numerous signalling pathways.… (more)

Subjects/Keywords: 572; nedd8; ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leidecker, O. (2012). Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873

Chicago Manual of Style (16th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Doctoral Dissertation, University of Dundee. Accessed June 16, 2019. https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873.

MLA Handbook (7th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Web. 16 Jun 2019.

Vancouver:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Jun 16]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873.

Council of Science Editors:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Doctoral Dissertation]. University of Dundee; 2012. Available from: https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873


University of Alberta

12. Mottet, Kelly. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The significance of poxvirus manipulation of the host ubiquitin proteasome system has become increasingly apparent. Ubiquitin is post-translationally added to target proteins by a highly… (more)

Subjects/Keywords: ligase; ubiquitination; proteasome; ubiquitin; poxvirus

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mottet, K. (2010). The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/zp38wf105

Chicago Manual of Style (16th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Masters Thesis, University of Alberta. Accessed June 16, 2019. https://era.library.ualberta.ca/files/zp38wf105.

MLA Handbook (7th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Web. 16 Jun 2019.

Vancouver:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2019 Jun 16]. Available from: https://era.library.ualberta.ca/files/zp38wf105.

Council of Science Editors:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/zp38wf105


Ruhr Universität Bochum

13. Nöpel-Dünnebacke, Stefanie. Identifikation und Lokalisation peroxisomaler Proteine des Menschen.

Degree: 2010, Ruhr Universität Bochum

 Problem Weitere Aufklärung der in Teilschritten ungeklärten Peroxisomenbiogenese durch Lokalisation von vierputativ peroxisomalen Proteinen in humanen Fibroblasten. Methode Immunfluoreszenzanalysen der GF-Fusionsproteine in humanen Fibroblasten und… (more)

Subjects/Keywords: Peroxisom; Peroxisom / Biogenese; Ubiquitin; Fibroblast

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nöpel-Dünnebacke, S. (2010). Identifikation und Lokalisation peroxisomaler Proteine des Menschen. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nöpel-Dünnebacke, Stefanie. “Identifikation und Lokalisation peroxisomaler Proteine des Menschen.” 2010. Thesis, Ruhr Universität Bochum. Accessed June 16, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nöpel-Dünnebacke, Stefanie. “Identifikation und Lokalisation peroxisomaler Proteine des Menschen.” 2010. Web. 16 Jun 2019.

Vancouver:

Nöpel-Dünnebacke S. Identifikation und Lokalisation peroxisomaler Proteine des Menschen. [Internet] [Thesis]. Ruhr Universität Bochum; 2010. [cited 2019 Jun 16]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nöpel-Dünnebacke S. Identifikation und Lokalisation peroxisomaler Proteine des Menschen. [Thesis]. Ruhr Universität Bochum; 2010. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Nakamura, Kasumi. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.

Degree: 博士(医学), 2016, Hamamatsu University School of Medicine / 浜松医科大学

The ubiquitin–proteasome pathway plays an important role in regulating apoptosis and the cell cycle. Recently, proteasome inhibitors have been shown to have antitumor effects and… (more)

Subjects/Keywords: ubiquitin; flavonoid; proteasome; inhibitor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nakamura, K. (2016). Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. (Thesis). Hamamatsu University School of Medicine / 浜松医科大学. Retrieved from http://hdl.handle.net/10271/3143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakamura, Kasumi. “Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.” 2016. Thesis, Hamamatsu University School of Medicine / 浜松医科大学. Accessed June 16, 2019. http://hdl.handle.net/10271/3143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakamura, Kasumi. “Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.” 2016. Web. 16 Jun 2019.

Vancouver:

Nakamura K. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. [Internet] [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2016. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10271/3143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakamura K. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2016. Available from: http://hdl.handle.net/10271/3143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

15. 胡晓斌; Hu, Xiaobin. The role of Uhrf1 in development and tumorigenesis.

Degree: PhD, 2014, University of Hong Kong

published_or_final_version

Biochemistry

Doctoral

Doctor of Philosophy

Subjects/Keywords: Ubiquitin; Carcinogenesis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

胡晓斌; Hu, X. (2014). The role of Uhrf1 in development and tumorigenesis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205

Chicago Manual of Style (16th Edition):

胡晓斌; Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed June 16, 2019. Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205.

MLA Handbook (7th Edition):

胡晓斌; Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Web. 16 Jun 2019.

Vancouver:

胡晓斌; Hu X. The role of Uhrf1 in development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Jun 16]. Available from: Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205.

Council of Science Editors:

胡晓斌; Hu X. The role of Uhrf1 in development and tumorigenesis. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205


University of Oxford

16. Tan, Ka-Liong. Translational relevance of AIPL1 and NUB1 in cancer.

Degree: PhD, 2017, University of Oxford

 <b>Background:</b> Aryl Hydrocarbon Receptor Interacting Protein-Like 1 (AIPL1) interacts with NUB1 and restricts the entry of NUB1 protein into the nucleus. The interferon-induced NEDD8 ultimate… (more)

Subjects/Keywords: Breast – Cancer; ubiquitin; Neddylation; chaperone

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tan, K. (2017). Translational relevance of AIPL1 and NUB1 in cancer. (Doctoral Dissertation). University of Oxford. Retrieved from https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085

Chicago Manual of Style (16th Edition):

Tan, Ka-Liong. “Translational relevance of AIPL1 and NUB1 in cancer.” 2017. Doctoral Dissertation, University of Oxford. Accessed June 16, 2019. https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085.

MLA Handbook (7th Edition):

Tan, Ka-Liong. “Translational relevance of AIPL1 and NUB1 in cancer.” 2017. Web. 16 Jun 2019.

Vancouver:

Tan K. Translational relevance of AIPL1 and NUB1 in cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2019 Jun 16]. Available from: https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085.

Council of Science Editors:

Tan K. Translational relevance of AIPL1 and NUB1 in cancer. [Doctoral Dissertation]. University of Oxford; 2017. Available from: https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085


University of Hong Kong

17. Tse, Ho-sum. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.

Degree: PhD, 2013, University of Hong Kong

Ubiquitin carboxy-terminal hydrolase L1 (UCH-L1), a protein of 223 amino acids, is a member of deubiquitinating enzymes and it is one of the most abundant… (more)

Subjects/Keywords: Parkinson's disease.; Ubiquitin.; Hydrolases.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tse, H. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505

Chicago Manual of Style (16th Edition):

Tse, Ho-sum. “Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed June 16, 2019. Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505.

MLA Handbook (7th Edition):

Tse, Ho-sum. “Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.” 2013. Web. 16 Jun 2019.

Vancouver:

Tse H. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Jun 16]. Available from: Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505.

Council of Science Editors:

Tse H. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505


University of Toronto

18. Gabrielli, Lisa Marie. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.

Degree: 2009, University of Toronto

3BP2 has been previously described as the protein mutated in the osteoporotic disorder, Cherubism. The gain of function mutation that characterizes Cherubism is the result… (more)

Subjects/Keywords: 3BP2; Ubiquitin; Nedd4; HECT; 0760

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gabrielli, L. M. (2009). Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18292

Chicago Manual of Style (16th Edition):

Gabrielli, Lisa Marie. “Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.” 2009. Masters Thesis, University of Toronto. Accessed June 16, 2019. http://hdl.handle.net/1807/18292.

MLA Handbook (7th Edition):

Gabrielli, Lisa Marie. “Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.” 2009. Web. 16 Jun 2019.

Vancouver:

Gabrielli LM. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/1807/18292.

Council of Science Editors:

Gabrielli LM. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18292


University of Toronto

19. Wu, Edwin. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.

Degree: 2013, University of Toronto

The ubiquitin-proteasome system regulates protein degradation. Although proteasomes localize in the nucleus of proliferating Saccharomyces cerevisiae, they are sequestered into cytoplasmic proteasome storage granules (PSG)… (more)

Subjects/Keywords: proteasome; ubiquitin; quiescence; 0487

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, E. (2013). The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43342

Chicago Manual of Style (16th Edition):

Wu, Edwin. “The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.” 2013. Masters Thesis, University of Toronto. Accessed June 16, 2019. http://hdl.handle.net/1807/43342.

MLA Handbook (7th Edition):

Wu, Edwin. “The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.” 2013. Web. 16 Jun 2019.

Vancouver:

Wu E. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/1807/43342.

Council of Science Editors:

Wu E. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43342


University of California – Berkeley

20. Worden, Evan Josiah. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.

Degree: Molecular & Cell Biology, 2016, University of California – Berkeley

 The 26S proteasome is responsible for selective protein degradation in eukaryotic cells. Polyubiquitin chains mark proteins for degradation by the proteasome, but before degradation can… (more)

Subjects/Keywords: Biochemistry; deubiquitinase; proteasome; ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Worden, E. J. (2016). Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Thesis, University of California – Berkeley. Accessed June 16, 2019. http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Web. 16 Jun 2019.

Vancouver:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Internet] [Thesis]. University of California – Berkeley; 2016. [cited 2019 Jun 16]. Available from: http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Thesis]. University of California – Berkeley; 2016. Available from: http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

21. Jolly, Lachlan. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.

Degree: 2010, University of Adelaide

 Neural Progenitor Cells (NPCs) are the primordial cells of central nervous system (CNS). Understanding how they are regulated benefits our knowledge of normal development, the… (more)

Subjects/Keywords: neurogenesis; ubiquitin; embryonic stem cell

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jolly, L. (2010). The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/65477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jolly, Lachlan. “The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.” 2010. Thesis, University of Adelaide. Accessed June 16, 2019. http://hdl.handle.net/2440/65477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jolly, Lachlan. “The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.” 2010. Web. 16 Jun 2019.

Vancouver:

Jolly L. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/2440/65477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jolly L. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/65477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

22. Staren, Daniel M. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.

Degree: MS, Molecular Biology, 2012, Loyola University Chicago

Ubiquitin has previously been identified as another natural agonist of CXC chemokine receptor 4 (CXCR4). In addition, recent evidence suggests that ubiquitin may activate… (more)

Subjects/Keywords: CXCR4; Ubiquitin; Molecular Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Staren, D. M. (2012). Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staren, Daniel M. “Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.” 2012. Thesis, Loyola University Chicago. Accessed June 16, 2019. http://ecommons.luc.edu/luc_theses/840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staren, Daniel M. “Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.” 2012. Web. 16 Jun 2019.

Vancouver:

Staren DM. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2019 Jun 16]. Available from: http://ecommons.luc.edu/luc_theses/840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staren DM. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. [Thesis]. Loyola University Chicago; 2012. Available from: http://ecommons.luc.edu/luc_theses/840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

23. Shrestha, Amit. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .

Degree: 2017, University of Ottawa

 In addition to apoptosis, metacapases function to regulate various other processes that promote and sustain life. For example, the Saccharomyces cerevisiae metacaspase Yca1 promotes cellular… (more)

Subjects/Keywords: Proteostasis; Metacaspase; Ubiquitin; Rsp5; Yca1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shrestha, A. (2017). The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/36661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shrestha, Amit. “The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .” 2017. Thesis, University of Ottawa. Accessed June 16, 2019. http://hdl.handle.net/10393/36661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shrestha, Amit. “The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .” 2017. Web. 16 Jun 2019.

Vancouver:

Shrestha A. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10393/36661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shrestha A. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/36661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

24. Parvatiyar, Kislay. Ubiquitin Dependent Regulation of Innate Antiviral Signaling.

Degree: PhD, Microbiology and Immunology (Medicine), 2010, University of Miami

 Induction of type I interferons by the transcription factors IRF3 and IRF7 is essential in the initiation of antiviral innate immunity. Activation of IRF3/7 requires… (more)

Subjects/Keywords: Ubiquitin Editing Complex; Signal Transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parvatiyar, K. (2010). Ubiquitin Dependent Regulation of Innate Antiviral Signaling. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/651

Chicago Manual of Style (16th Edition):

Parvatiyar, Kislay. “Ubiquitin Dependent Regulation of Innate Antiviral Signaling.” 2010. Doctoral Dissertation, University of Miami. Accessed June 16, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/651.

MLA Handbook (7th Edition):

Parvatiyar, Kislay. “Ubiquitin Dependent Regulation of Innate Antiviral Signaling.” 2010. Web. 16 Jun 2019.

Vancouver:

Parvatiyar K. Ubiquitin Dependent Regulation of Innate Antiviral Signaling. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 Jun 16]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/651.

Council of Science Editors:

Parvatiyar K. Ubiquitin Dependent Regulation of Innate Antiviral Signaling. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/651


University of Toronto

25. Heir, Pardeep. Regulation of Cellular Oxygen Sensing Pathways by VHL.

Degree: PhD, 2015, University of Toronto

Erythropoiesis represents a vital physiologic process that can be adjusted to combat compromised oxygen availability, otherwise known as hypoxia. The canonical response to adapt to… (more)

Subjects/Keywords: Erythropoiesis; Hypoxia; Ubiquitin; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Heir, P. (2015). Regulation of Cellular Oxygen Sensing Pathways by VHL. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76677

Chicago Manual of Style (16th Edition):

Heir, Pardeep. “Regulation of Cellular Oxygen Sensing Pathways by VHL.” 2015. Doctoral Dissertation, University of Toronto. Accessed June 16, 2019. http://hdl.handle.net/1807/76677.

MLA Handbook (7th Edition):

Heir, Pardeep. “Regulation of Cellular Oxygen Sensing Pathways by VHL.” 2015. Web. 16 Jun 2019.

Vancouver:

Heir P. Regulation of Cellular Oxygen Sensing Pathways by VHL. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/1807/76677.

Council of Science Editors:

Heir P. Regulation of Cellular Oxygen Sensing Pathways by VHL. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/76677


University of Toronto

26. Pascoe, Natasha. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.

Degree: PhD, 2018, University of Toronto

 Deubiquitinating enzymes represent attractive therapeutic targets that can be leveraged towards the treatment of a variety of devastating human health disorders. This doctoral thesis recounts… (more)

Subjects/Keywords: Biologics; DUBs; Ubiquitin; Yeast; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pascoe, N. (2018). Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89877

Chicago Manual of Style (16th Edition):

Pascoe, Natasha. “Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.” 2018. Doctoral Dissertation, University of Toronto. Accessed June 16, 2019. http://hdl.handle.net/1807/89877.

MLA Handbook (7th Edition):

Pascoe, Natasha. “Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.” 2018. Web. 16 Jun 2019.

Vancouver:

Pascoe N. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/1807/89877.

Council of Science Editors:

Pascoe N. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89877


Purdue University

27. Wade, Cameron. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.

Degree: MS, PULSe, 2016, Purdue University

 ElaD is a cysteine protease found in Escherichia coli (E. coli) and has been shown to function as a deubiquitinating enzyme (DUB). However, ubiquitin and… (more)

Subjects/Keywords: Bacteria; Deubiquitinase; Escherichia Coli; Ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wade, C. (2016). Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. (Thesis). Purdue University. Retrieved from https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Thesis, Purdue University. Accessed June 16, 2019. https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Web. 16 Jun 2019.

Vancouver:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Internet] [Thesis]. Purdue University; 2016. [cited 2019 Jun 16]. Available from: https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Thesis]. Purdue University; 2016. Available from: https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

28. Hart, Matthew Robert. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .

Degree: 2013, University of Arizona

 Much of what is known about the role of the ERBB family in cellular biology and in cancer has to do with canonical downstream signaling… (more)

Subjects/Keywords: EGFR; Peptide; Ubiquitin; Genetics; Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hart, M. R. (2013). Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293476

Chicago Manual of Style (16th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed June 16, 2019. http://hdl.handle.net/10150/293476.

MLA Handbook (7th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Web. 16 Jun 2019.

Vancouver:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/10150/293476.

Council of Science Editors:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293476


University of Rochester

29. Shen, Run; Chen, Di (1955 - ). Regulation of Runx2 protein stability in chondrocyte development.

Degree: PhD, 2009, University of Rochester

 During endochondral bone formation, cartilages provide the template for vascular invasion, which brings in osteoblasts to deposit bone matrix on the debris of apoptotic chondrocytes.… (more)

Subjects/Keywords: Ubiquitin; Runx2; Chondrocyte; PTHrP; Cartilage

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shen, Run; Chen, D. (. -. ). (2009). Regulation of Runx2 protein stability in chondrocyte development. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6536

Chicago Manual of Style (16th Edition):

Shen, Run; Chen, Di (1955 - ). “Regulation of Runx2 protein stability in chondrocyte development.” 2009. Doctoral Dissertation, University of Rochester. Accessed June 16, 2019. http://hdl.handle.net/1802/6536.

MLA Handbook (7th Edition):

Shen, Run; Chen, Di (1955 - ). “Regulation of Runx2 protein stability in chondrocyte development.” 2009. Web. 16 Jun 2019.

Vancouver:

Shen, Run; Chen D(-). Regulation of Runx2 protein stability in chondrocyte development. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2019 Jun 16]. Available from: http://hdl.handle.net/1802/6536.

Council of Science Editors:

Shen, Run; Chen D(-). Regulation of Runx2 protein stability in chondrocyte development. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6536


University of California – San Francisco

30. Hadjivassiliou, Haralambos Antonis. Mechanisms of pre-mRNA splicing in yeast.

Degree: Biochemistry and Molecular Biology, 2014, University of California – San Francisco

 The spliceosome is one of the cells largest and important molecular machines and yet it remains as one the least understood systems. This is a… (more)

Subjects/Keywords: Biochemistry; Biophysics; Sad1; spliceosome; ubiquitin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hadjivassiliou, H. A. (2014). Mechanisms of pre-mRNA splicing in yeast. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2wj5816h

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hadjivassiliou, Haralambos Antonis. “Mechanisms of pre-mRNA splicing in yeast.” 2014. Thesis, University of California – San Francisco. Accessed June 16, 2019. http://www.escholarship.org/uc/item/2wj5816h.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hadjivassiliou, Haralambos Antonis. “Mechanisms of pre-mRNA splicing in yeast.” 2014. Web. 16 Jun 2019.

Vancouver:

Hadjivassiliou HA. Mechanisms of pre-mRNA splicing in yeast. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2019 Jun 16]. Available from: http://www.escholarship.org/uc/item/2wj5816h.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hadjivassiliou HA. Mechanisms of pre-mRNA splicing in yeast. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/2wj5816h

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [23]

.