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You searched for subject:(UPR). Showing records 1 – 30 of 102 total matches.

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Cornell University

1. He, Yin. Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling.

Degree: PhD, Genetics, 2013, Cornell University

 The endoplasmic reticulum (ER) serves as the site of protein synthesis, folding, maturation, modification, secretion, and degradation for approximately one-third of the proteome. Disruptions in… (more)

Subjects/Keywords: ER stress; UPR; IRE1alpha; XBP1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

He, Y. (2013). Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/34097

Chicago Manual of Style (16th Edition):

He, Yin. “Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling.” 2013. Doctoral Dissertation, Cornell University. Accessed October 20, 2020. http://hdl.handle.net/1813/34097.

MLA Handbook (7th Edition):

He, Yin. “Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling.” 2013. Web. 20 Oct 2020.

Vancouver:

He Y. Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1813/34097.

Council of Science Editors:

He Y. Investigating Mammalian Unfolded Protein Response: The Physiology And Regulatory Mechanisms Of Ire1Alpha-Xbp1 Signaling. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/34097

2. Bodeau, Sandra. Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma.

Degree: Docteur es, Biologie-santé. Physiopathologie Humaine - Approches pharmacologiques des cancers, 2017, Amiens

Au cours de ce travail, nous nous sommes intéressés à l'impact de l'exposition à la ciclosporine A (CsA) sur la signalisation angiogénique dans le carcinome… (more)

Subjects/Keywords: VEGF; UPR; Post-transplant RCC

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APA (6th Edition):

Bodeau, S. (2017). Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma. (Doctoral Dissertation). Amiens. Retrieved from http://www.theses.fr/2017AMIE0021

Chicago Manual of Style (16th Edition):

Bodeau, Sandra. “Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma.” 2017. Doctoral Dissertation, Amiens. Accessed October 20, 2020. http://www.theses.fr/2017AMIE0021.

MLA Handbook (7th Edition):

Bodeau, Sandra. “Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma.” 2017. Web. 20 Oct 2020.

Vancouver:

Bodeau S. Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma. [Internet] [Doctoral dissertation]. Amiens; 2017. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2017AMIE0021.

Council of Science Editors:

Bodeau S. Modulation et ciblage du facteur de croissance de l'endothélium vasculaire (VEGF) dans le carcinome à cellules rénales post-transplantation : Modulation and targeting of the vascular endothelial growth factor (VEGF) in post-transplant renal cell carcinoma. [Doctoral Dissertation]. Amiens; 2017. Available from: http://www.theses.fr/2017AMIE0021


University of Manchester

3. Mularczyk, Ewa. Understanding molecular pathology of chondrodysplasias : the role of ER stress.

Degree: PhD, 2012, University of Manchester

 MCDS is an autosomal dominant disorder, with a mild dwarfed phenotype and is caused by mutations in collagen X. The majority of the mutations identified… (more)

Subjects/Keywords: 616.7; UPR; ER stress; chondrodysplasia

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APA (6th Edition):

Mularczyk, E. (2012). Understanding molecular pathology of chondrodysplasias : the role of ER stress. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/understanding-molecular-pathology-of-chondrodysplasias-the-role-of-er-stress(86ad2dcd-fcb6-4860-90d8-74f17996ac0d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.724631

Chicago Manual of Style (16th Edition):

Mularczyk, Ewa. “Understanding molecular pathology of chondrodysplasias : the role of ER stress.” 2012. Doctoral Dissertation, University of Manchester. Accessed October 20, 2020. https://www.research.manchester.ac.uk/portal/en/theses/understanding-molecular-pathology-of-chondrodysplasias-the-role-of-er-stress(86ad2dcd-fcb6-4860-90d8-74f17996ac0d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.724631.

MLA Handbook (7th Edition):

Mularczyk, Ewa. “Understanding molecular pathology of chondrodysplasias : the role of ER stress.” 2012. Web. 20 Oct 2020.

Vancouver:

Mularczyk E. Understanding molecular pathology of chondrodysplasias : the role of ER stress. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Oct 20]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-molecular-pathology-of-chondrodysplasias-the-role-of-er-stress(86ad2dcd-fcb6-4860-90d8-74f17996ac0d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.724631.

Council of Science Editors:

Mularczyk E. Understanding molecular pathology of chondrodysplasias : the role of ER stress. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-molecular-pathology-of-chondrodysplasias-the-role-of-er-stress(86ad2dcd-fcb6-4860-90d8-74f17996ac0d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.724631


University of Florida

4. Kim, Min Hyun. Regulation and Function of Zinc and Zinc Transporters during ER Stress.

Degree: PhD, Nutritional Sciences, 2017, University of Florida

 Extensive endoplasmic reticulum (ER) stress damages the liver causing apoptosis and steatosis, despite the activation of the unfolded protein response (UPR). Restriction of zinc from… (more)

Subjects/Keywords: apoptosis  – steatosis  – upr  – zinc  – zip14

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, M. H. (2017). Regulation and Function of Zinc and Zinc Transporters during ER Stress. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0051263

Chicago Manual of Style (16th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Doctoral Dissertation, University of Florida. Accessed October 20, 2020. https://ufdc.ufl.edu/UFE0051263.

MLA Handbook (7th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Web. 20 Oct 2020.

Vancouver:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2020 Oct 20]. Available from: https://ufdc.ufl.edu/UFE0051263.

Council of Science Editors:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Doctoral Dissertation]. University of Florida; 2017. Available from: https://ufdc.ufl.edu/UFE0051263


Universitat de Valencia

5. Navarro Tapia, Elisabet. Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae .

Degree: 2016, Universitat de Valencia

 Los mecanismos de tolerancia a etanol han sido ampliamente estudiados en la levadura Saccharomyces cerevisiae debido a su importancia en el sector industrial de bebidas… (more)

Subjects/Keywords: levaduras; etanol; UPR; Saccharomyces; microarrays

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APA (6th Edition):

Navarro Tapia, E. (2016). Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/50921

Chicago Manual of Style (16th Edition):

Navarro Tapia, Elisabet. “Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae .” 2016. Doctoral Dissertation, Universitat de Valencia. Accessed October 20, 2020. http://hdl.handle.net/10550/50921.

MLA Handbook (7th Edition):

Navarro Tapia, Elisabet. “Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae .” 2016. Web. 20 Oct 2020.

Vancouver:

Navarro Tapia E. Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2016. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10550/50921.

Council of Science Editors:

Navarro Tapia E. Estudio de la adaptación a estrés por etanol en cepas de Saccharomyces cerevisiae . [Doctoral Dissertation]. Universitat de Valencia; 2016. Available from: http://hdl.handle.net/10550/50921


University of Manchester

6. Mularczyk, Ewa. Understanding molecular pathology of chondrodysplasias: the role of ER stress.

Degree: 2011, University of Manchester

 MCDS is an autosomal dominant disorder, with a mild dwarfed phenotype and is caused by mutations in collagen X. The majority of the mutations identified… (more)

Subjects/Keywords: UPR; ER stress; chondrodysplasia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mularczyk, E. (2011). Understanding molecular pathology of chondrodysplasias: the role of ER stress. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:140302

Chicago Manual of Style (16th Edition):

Mularczyk, Ewa. “Understanding molecular pathology of chondrodysplasias: the role of ER stress.” 2011. Doctoral Dissertation, University of Manchester. Accessed October 20, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:140302.

MLA Handbook (7th Edition):

Mularczyk, Ewa. “Understanding molecular pathology of chondrodysplasias: the role of ER stress.” 2011. Web. 20 Oct 2020.

Vancouver:

Mularczyk E. Understanding molecular pathology of chondrodysplasias: the role of ER stress. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Oct 20]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:140302.

Council of Science Editors:

Mularczyk E. Understanding molecular pathology of chondrodysplasias: the role of ER stress. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:140302

7. Fattouh, Nour. Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia.

Degree: Docteur es, Biologie Santé, 2018, Montpellier

Les bactéries intracellulaires Wolbachia ont développé une vaste gamme d’interactions symbiotiques, du parasitisme reproductif au mutualisme chez les arthropodes terrestres et les nématodes filaires, devenant… (more)

Subjects/Keywords: Wolbachia; Endosymbiote; Réticulum endoplasmique; Upr; Wolbachia; Endosymbiont; Endoplasmic reticulum; Upr

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APA (6th Edition):

Fattouh, N. (2018). Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2018MONTT079

Chicago Manual of Style (16th Edition):

Fattouh, Nour. “Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia.” 2018. Doctoral Dissertation, Montpellier. Accessed October 20, 2020. http://www.theses.fr/2018MONTT079.

MLA Handbook (7th Edition):

Fattouh, Nour. “Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia.” 2018. Web. 20 Oct 2020.

Vancouver:

Fattouh N. Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia. [Internet] [Doctoral dissertation]. Montpellier; 2018. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2018MONTT079.

Council of Science Editors:

Fattouh N. Caractérisation du mode de vie intracellulaire des endosymbiotes Wolbachia : Characterization of the intracellular lifestyle of the endosymbionts Wolbachia. [Doctoral Dissertation]. Montpellier; 2018. Available from: http://www.theses.fr/2018MONTT079


University of California – San Diego

8. Rodvold, Jeffrey James. Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation.

Degree: Biomedical Sciences, 2017, University of California – San Diego

 The tumor microenvironment is harbor to a variety of insults that privilege tumor cells to co-opt host immunity. Many of these insults, including nutrient deprivation… (more)

Subjects/Keywords: Biology; ER stress; Tumor immunology; UPR

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APA (6th Edition):

Rodvold, J. J. (2017). Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/49z0t0b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodvold, Jeffrey James. “Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation.” 2017. Thesis, University of California – San Diego. Accessed October 20, 2020. http://www.escholarship.org/uc/item/49z0t0b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodvold, Jeffrey James. “Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation.” 2017. Web. 20 Oct 2020.

Vancouver:

Rodvold JJ. Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2020 Oct 20]. Available from: http://www.escholarship.org/uc/item/49z0t0b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodvold JJ. Anatomy of the cell extrinsic effects of endoplasmic reticulum stress in tumor facilitation. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/49z0t0b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Mihailidou, Chrysovalantou. Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 The accumulation of unfolded and misfolded proteins in the endoplasmic reticulum (ER) causes stress that has been designated as ER stress. Cells respond to ER… (more)

Subjects/Keywords: Τουνικαμυκίνη; Διαβήτης; p21; CHOP; Tunicamycin; UPR; Diabetes

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APA (6th Edition):

Mihailidou, C. (2014). Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mihailidou, Chrysovalantou. “Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 20, 2020. http://hdl.handle.net/10442/hedi/41739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mihailidou, Chrysovalantou. “Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου.” 2014. Web. 20 Oct 2020.

Vancouver:

Mihailidou C. Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10442/hedi/41739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mihailidou C. Ρύθμιση του ογκοκατασταλτικού γονιδίου p21 από το προ-αποπτωτικό μόριο CHOP και το στρές του ενδοπλασματικού δικτύου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/41739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

10. Burman, Ankita. Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF).

Degree: PhD, Cell and Developmental Biology, 2019, Vanderbilt University

 Endoplasmic reticulum (ER) stress in type II AECs has been associated with pathogenesis of IPF; however, factors inducing ER stress and downstream mechanisms through which… (more)

Subjects/Keywords: UPR; IPF; ER stress; Pulmonary. Fibrosis; Lung

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Burman, A. (2019). Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF). (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10419

Chicago Manual of Style (16th Edition):

Burman, Ankita. “Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF).” 2019. Doctoral Dissertation, Vanderbilt University. Accessed October 20, 2020. http://hdl.handle.net/1803/10419.

MLA Handbook (7th Edition):

Burman, Ankita. “Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF).” 2019. Web. 20 Oct 2020.

Vancouver:

Burman A. Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF). [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1803/10419.

Council of Science Editors:

Burman A. Unraveling the Role of Endoplasmic Reticulum Stress in Idiopathic Pulmonary Fibrosis (IPF). [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/10419


Texas A&M University

11. Guo, Jinbai. Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae.

Degree: PhD, Genetics, 2007, Texas A&M University

 Cell cycle progression of Saccharomyces cerevisiae cells was monitored in continuous cultures limited for glucose or nitrogen. The G1 cell cycle phase, before initiation of… (more)

Subjects/Keywords: Cell division; UPR

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APA (6th Edition):

Guo, J. (2007). Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/5912

Chicago Manual of Style (16th Edition):

Guo, Jinbai. “Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae.” 2007. Doctoral Dissertation, Texas A&M University. Accessed October 20, 2020. http://hdl.handle.net/1969.1/5912.

MLA Handbook (7th Edition):

Guo, Jinbai. “Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae.” 2007. Web. 20 Oct 2020.

Vancouver:

Guo J. Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. Texas A&M University; 2007. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1969.1/5912.

Council of Science Editors:

Guo J. Control of cell division by nutrients, and ER stress signaling in Saccharomyces cerevisiae. [Doctoral Dissertation]. Texas A&M University; 2007. Available from: http://hdl.handle.net/1969.1/5912


National University of Ireland – Galway

12. Hossain, Muhammad Mosaraf. Role of MicroRNAs and unfolded protein response in breast cancer .

Degree: 2017, National University of Ireland – Galway

 Stressful conditions of the tumour microenvironment which overwhelm the folding capacity of the endoplasmic reticulum (EnR) activate an evolutionary conserved signalling pathway known as the… (more)

Subjects/Keywords: MicroRNA; UPR; Breast cancer; Medicine; Pathology

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APA (6th Edition):

Hossain, M. M. (2017). Role of MicroRNAs and unfolded protein response in breast cancer . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/7138

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hossain, Muhammad Mosaraf. “Role of MicroRNAs and unfolded protein response in breast cancer .” 2017. Thesis, National University of Ireland – Galway. Accessed October 20, 2020. http://hdl.handle.net/10379/7138.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hossain, Muhammad Mosaraf. “Role of MicroRNAs and unfolded protein response in breast cancer .” 2017. Web. 20 Oct 2020.

Vancouver:

Hossain MM. Role of MicroRNAs and unfolded protein response in breast cancer . [Internet] [Thesis]. National University of Ireland – Galway; 2017. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10379/7138.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hossain MM. Role of MicroRNAs and unfolded protein response in breast cancer . [Thesis]. National University of Ireland – Galway; 2017. Available from: http://hdl.handle.net/10379/7138

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kansas State University

13. Balthazor, James. Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response.

Degree: PhD, Biochemistry and Molecular Biophysics Interdepartmental Program, 2015, Kansas State University

 Armet is a bifunctional protein that is apparently universally distributed among multicellular animal species, vertebrate and invertebrate alike. A member of the Unfolded Protein Response,… (more)

Subjects/Keywords: UPR; RNA-seq; Pea aphid; Armet; Saliva

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APA (6th Edition):

Balthazor, J. (2015). Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response. (Doctoral Dissertation). Kansas State University. Retrieved from http://hdl.handle.net/2097/35218

Chicago Manual of Style (16th Edition):

Balthazor, James. “Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response.” 2015. Doctoral Dissertation, Kansas State University. Accessed October 20, 2020. http://hdl.handle.net/2097/35218.

MLA Handbook (7th Edition):

Balthazor, James. “Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response.” 2015. Web. 20 Oct 2020.

Vancouver:

Balthazor J. Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response. [Internet] [Doctoral dissertation]. Kansas State University; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/2097/35218.

Council of Science Editors:

Balthazor J. Studies of human Armet and of pea aphid transcripts of saliva proteins and the Unfolded Protein Response. [Doctoral Dissertation]. Kansas State University; 2015. Available from: http://hdl.handle.net/2097/35218


University of Texas – Austin

14. Park, Jihyun. Regulation and function of two membrane-associated protein kinases.

Degree: PhD, Cell and Molecular Biology, 2014, University of Texas – Austin

 Protein kinases, many of which are regulated by autophosphorylation, modify their substrates by phosphorylation. Due to their roles in cellular signaling cascades controlling processes such… (more)

Subjects/Keywords: Kinase; TRPM7; Autophosphorylation; UPR; PERK; Cancer

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APA (6th Edition):

Park, J. (2014). Regulation and function of two membrane-associated protein kinases. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/47189

Chicago Manual of Style (16th Edition):

Park, Jihyun. “Regulation and function of two membrane-associated protein kinases.” 2014. Doctoral Dissertation, University of Texas – Austin. Accessed October 20, 2020. http://hdl.handle.net/2152/47189.

MLA Handbook (7th Edition):

Park, Jihyun. “Regulation and function of two membrane-associated protein kinases.” 2014. Web. 20 Oct 2020.

Vancouver:

Park J. Regulation and function of two membrane-associated protein kinases. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2014. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/2152/47189.

Council of Science Editors:

Park J. Regulation and function of two membrane-associated protein kinases. [Doctoral Dissertation]. University of Texas – Austin; 2014. Available from: http://hdl.handle.net/2152/47189

15. Poncet, Anaïs. Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection.

Degree: Docteur es, Cancérologie, génétique, hématologie, immunologie, 2019, Université Lille II – Droit et Santé

Toxoplasma gondii (T. gondii) est un parasite intracellulaire obligatoire responsable d’une zoonose cosmopolite, la toxoplasmose. Chez les sujets sains, les parasites de type II persistent… (more)

Subjects/Keywords: Cellules dendritiques; Toxoplasma gondii; Réponse UPR; Dendritic cells; Toxoplasma gondii; UPR response

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poncet, A. (2019). Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2019LIL2S038

Chicago Manual of Style (16th Edition):

Poncet, Anaïs. “Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection.” 2019. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed October 20, 2020. http://www.theses.fr/2019LIL2S038.

MLA Handbook (7th Edition):

Poncet, Anaïs. “Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection.” 2019. Web. 20 Oct 2020.

Vancouver:

Poncet A. Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2019. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2019LIL2S038.

Council of Science Editors:

Poncet A. Modulation de l’activité des cellules dendritiques par la réponse UPR induite lors de l’infection à Toxoplasma gondii : UPR-mediated modulation of dendritic cell responses during Toxoplasma gondii infection. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2019. Available from: http://www.theses.fr/2019LIL2S038

16. Gerakis, Yannis. Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2016, Université Côte d'Azur (ComUE)

La maladie d'Alzheimer est une pathologie neurodégénérative progressive liée à l'âge qui détériore premièrement les fonctions liées aux mémoires de travail et épisodiques, avant de… (more)

Subjects/Keywords: Alzheimer; Neurodégénérescence; UPR; Protéostasie; Peptide amyloïde; Alzheimer; Neurodegeneration; UPR; Proteostasis; Amyloid peptide

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gerakis, Y. (2016). Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2016AZUR4097

Chicago Manual of Style (16th Edition):

Gerakis, Yannis. “Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor.” 2016. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed October 20, 2020. http://www.theses.fr/2016AZUR4097.

MLA Handbook (7th Edition):

Gerakis, Yannis. “Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor.” 2016. Web. 20 Oct 2020.

Vancouver:

Gerakis Y. Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2016. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2016AZUR4097.

Council of Science Editors:

Gerakis Y. Stress réticulaire et maladie d'Alzheimer : contribution du facteur de transcription XBP-1s : Reticular stress in the Alzheimer's disease : role of the XBP-1s transcription factor. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2016. Available from: http://www.theses.fr/2016AZUR4097


Universidade Federal de Viçosa

17. Maximiller Dal-Bianco Lamas Costa. Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs.

Degree: 2007, Universidade Federal de Viçosa

A identificação de vias de sinalização de resposta em células afetadas por diferentes estresses, bem como das interações entres estas vias, constitui um dos interesses… (more)

Subjects/Keywords: ER-stress; NRP; PCD; NRP; Osmotic stress; UPR; BIOLOGIA MOLECULAR; Estresse osmótico; Estresse no retículo; UPR; PCD

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APA (6th Edition):

Costa, M. D. L. (2007). Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs. (Thesis). Universidade Federal de Viçosa. Retrieved from http://www.tede.ufv.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Costa, Maximiller Dal-Bianco Lamas. “Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs.” 2007. Thesis, Universidade Federal de Viçosa. Accessed October 20, 2020. http://www.tede.ufv.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Costa, Maximiller Dal-Bianco Lamas. “Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs.” 2007. Web. 20 Oct 2020.

Vancouver:

Costa MDL. Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs. [Internet] [Thesis]. Universidade Federal de Viçosa; 2007. [cited 2020 Oct 20]. Available from: http://www.tede.ufv.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Costa MDL. Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs. [Thesis]. Universidade Federal de Viçosa; 2007. Available from: http://www.tede.ufv.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Brun, Agnès. Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Université de Strasbourg

Les rétinopathies pigmentaires sont la première cause de dégénérescence rétinienne héréditaire, affectant 1,5 millions de personnes dans le monde. A ce jour, aucun traitement n’est… (more)

Subjects/Keywords: Rétinopathies pigmentaires; VPA; Inhibiteur GADD34/PP1c; Thérapie pharmacologique; UPR; Retinitis pigmentosa; VPA; GADD34/PP1c inhibitor; Pharmacologic therapy; UPR; 572.8; 615.7

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APA (6th Edition):

Brun, A. (2019). Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2019STRAJ076

Chicago Manual of Style (16th Edition):

Brun, Agnès. “Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations.” 2019. Doctoral Dissertation, Université de Strasbourg. Accessed October 20, 2020. http://www.theses.fr/2019STRAJ076.

MLA Handbook (7th Edition):

Brun, Agnès. “Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations.” 2019. Web. 20 Oct 2020.

Vancouver:

Brun A. Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2019. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2019STRAJ076.

Council of Science Editors:

Brun A. Développement de thérapies pharmacologiques innovantes dans le traitement des dégénérescences rétiniennes héréditaires : Development of innovative pharmacological therapies for the treatment of hereditary retinal degenerations. [Doctoral Dissertation]. Université de Strasbourg; 2019. Available from: http://www.theses.fr/2019STRAJ076

19. Pires da silva, Julie. Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart.

Degree: Docteur es, Physiologie, physiopathologie, 2018, Université Paris-Saclay (ComUE)

Le réticulum endoplasmique rugueux (RE), assure la synthèse, le repliement et la maturation des protéines de la voie de sécrétion. Les altérations des fonctions physiologiques… (more)

Subjects/Keywords: Stress RE; Sirt1; Apoptose; Autophagie; Réponse UPR; Coeur; ER Stress; Sirt1; Apoptosis; Autophagy; Upr; Heart; 570

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APA (6th Edition):

Pires da silva, J. (2018). Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS211

Chicago Manual of Style (16th Edition):

Pires da silva, Julie. “Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 20, 2020. http://www.theses.fr/2018SACLS211.

MLA Handbook (7th Edition):

Pires da silva, Julie. “Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart.” 2018. Web. 20 Oct 2020.

Vancouver:

Pires da silva J. Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2018SACLS211.

Council of Science Editors:

Pires da silva J. Rôle de la sirtuine 1 dans la modulation des réponses apoptotique et autophagique du coeur au stress du réticulum endoplasmique : Role of Sirtuin 1 in the modulation of ER stress-induced apoptosis and autophagy in heart. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS211

20. Perochon, Jessica. Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster.

Degree: Docteur es, Sciences de la vie et de la santé, 2015, Université Paris-Saclay (ComUE)

Le réticulum endoplasmique (RE) est un organite assurant de nombreuses fonctionscellulaires telles que la conformation et des modifications post-traductionnelles des protéines ou lemaintien de l’homéostasie… (more)

Subjects/Keywords: PERK; Upr; Homéostasie; Apoptose; Drosophila; Disque imaginal d’aile; PERK; Upr; Homeostasis; Apoptosis; Drosophila; Wing imaginal disc; 571.6

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APA (6th Edition):

Perochon, J. (2015). Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2015SACLV036

Chicago Manual of Style (16th Edition):

Perochon, Jessica. “Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster.” 2015. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 20, 2020. http://www.theses.fr/2015SACLV036.

MLA Handbook (7th Edition):

Perochon, Jessica. “Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster.” 2015. Web. 20 Oct 2020.

Vancouver:

Perochon J. Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2015. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2015SACLV036.

Council of Science Editors:

Perochon J. Étude des conséquences d’un stress chronique du Réticulum Endoplasmique (RE) chez Drosophila melanogaster : Study of the consequences of a chronic ER stress in Drosophila melanogaster. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2015. Available from: http://www.theses.fr/2015SACLV036

21. Lebeau, Justine. Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis.

Degree: Docteur es, Biologie cellulaire, 2014, Université Claude Bernard – Lyon I

Les signalisations oncogéniques induisent une consommation accrue de glucose qui n'est que partiellement satisfaite par le microenvironnement. Pour s'adapter et survivre à ce stress métabolique,… (more)

Subjects/Keywords: Stress du réticulum endoplasmique; UPR; Carence en glucose; Cancer; Endoplasmic Reticulum Stress; UPR; Glucose deprivation; Cancer; 571.6

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APA (6th Edition):

Lebeau, J. (2014). Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10175

Chicago Manual of Style (16th Edition):

Lebeau, Justine. “Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed October 20, 2020. http://www.theses.fr/2014LYO10175.

MLA Handbook (7th Edition):

Lebeau, Justine. “Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis.” 2014. Web. 20 Oct 2020.

Vancouver:

Lebeau J. Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2014LYO10175.

Council of Science Editors:

Lebeau J. Stress du réticulum endoplasmique et tumorigenèse : Endoplasmic Reticulum Stress in tumorigenesis. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10175

22. Demay, Yohan. Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress.

Degree: Docteur es, Biologie cellulaire, 2014, Versailles-St Quentin en Yvelines

Le réticulum endoplasmique (RE) joue un rôle majeur dans la conformation des protéines. L’accumulation de protéines non- ou mal-conformées dans le RE induit un stress… (more)

Subjects/Keywords: RE; Stress; UPR; Préséniline; Homéostasie; Développement; Retard; Drosophile; ER; Stress; UPR; Presenilin; Homeostasis; Development; Delay; Drosophila

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APA (6th Edition):

Demay, Y. (2014). Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress. (Doctoral Dissertation). Versailles-St Quentin en Yvelines. Retrieved from http://www.theses.fr/2014VERS0024

Chicago Manual of Style (16th Edition):

Demay, Yohan. “Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress.” 2014. Doctoral Dissertation, Versailles-St Quentin en Yvelines. Accessed October 20, 2020. http://www.theses.fr/2014VERS0024.

MLA Handbook (7th Edition):

Demay, Yohan. “Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress.” 2014. Web. 20 Oct 2020.

Vancouver:

Demay Y. Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress. [Internet] [Doctoral dissertation]. Versailles-St Quentin en Yvelines; 2014. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2014VERS0024.

Council of Science Editors:

Demay Y. Etude du maintien de l'homéostasie tissulaire après induction d'un stress chronique du RE : Study of tissue homeostasis after a chronic ER stress. [Doctoral Dissertation]. Versailles-St Quentin en Yvelines; 2014. Available from: http://www.theses.fr/2014VERS0024


Virginia Tech

23. Catanzaro, Nicholas Jr. Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection.

Degree: PhD, Biomedical and Veterinary Sciences, 2020, Virginia Tech

 Porcine reproductive and respiratory syndrome virus (PRRSV) causes an economically-devastating disease in the global swine industry. Annually, PRRSV is estimated to cause more than $600… (more)

Subjects/Keywords: Porcine reproductive and respiratory syndrome virus (PRRSV); stress granules (SGs); unfolded protein response (UPR); virus replication; unfolded protein response (UPR)

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APA (6th Edition):

Catanzaro, N. J. (2020). Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/97907

Chicago Manual of Style (16th Edition):

Catanzaro, Nicholas Jr. “Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection.” 2020. Doctoral Dissertation, Virginia Tech. Accessed October 20, 2020. http://hdl.handle.net/10919/97907.

MLA Handbook (7th Edition):

Catanzaro, Nicholas Jr. “Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection.” 2020. Web. 20 Oct 2020.

Vancouver:

Catanzaro NJ. Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection. [Internet] [Doctoral dissertation]. Virginia Tech; 2020. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10919/97907.

Council of Science Editors:

Catanzaro NJ. Molecular Mechanisms of Host Responses to Porcine Reproductive and Respiratory Syndrome Virus (PRRSV) Infection. [Doctoral Dissertation]. Virginia Tech; 2020. Available from: http://hdl.handle.net/10919/97907


University of Helsinki

24. Montonen, Ella. CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa.

Degree: Farmaceutiska fakulteten, 2015, University of Helsinki

 Solulimakalvoston stressi (ER-stressi) syntyy, kun laskostumattomia ja väärinlaskostuneita proteiineja kerääntyy solulimakalvostoon. Laskostumattomat proteiinit käynnistävät solussa laskostumattomien proteiinien vasteen (UPR-reitti), joka pyrkii vähentämään solulimakalvoston proteiinikuormaa ja… (more)

Subjects/Keywords: CDNF; ALS; UPR; ER-stress; unfolded proteins; ER-stressi; UPR-reitti; laskostumattomat proteiinit; Farmakologi; Pharmacology; Farmakologia

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APA (6th Edition):

Montonen, E. (2015). CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/158359

Chicago Manual of Style (16th Edition):

Montonen, Ella. “CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa.” 2015. Masters Thesis, University of Helsinki. Accessed October 20, 2020. http://hdl.handle.net/10138/158359.

MLA Handbook (7th Edition):

Montonen, Ella. “CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa.” 2015. Web. 20 Oct 2020.

Vancouver:

Montonen E. CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa. [Internet] [Masters thesis]. University of Helsinki; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10138/158359.

Council of Science Editors:

Montonen E. CDNF:n vaikutukset solulimakalvoston stressiin ja kliinisiin oireisiin amyotrofisen lateraaliskleroosin hiirimallissa. [Masters Thesis]. University of Helsinki; 2015. Available from: http://hdl.handle.net/10138/158359

25. 小林, 克史. 小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: 小胞体ストレス; UPR

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APA (6th Edition):

小林, . (n.d.). 小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/1578

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小林, 克史. “小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed October 20, 2020. http://hdl.handle.net/10061/1578.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小林, 克史. “小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ.” Web. 20 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

小林 . 小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10061/1578.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

小林 . 小胞体ストレス応答タンパク質IRE1の構造研究 : Structural study of the endoplasmic reticulum stress protein IRE1; ショウホウタイ ストレス オウトウ タンパクシツ IRE1 ノ コウゾウ ケンキュウ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/1578

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of California – San Diego

26. Chan, Priscilla. Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration.

Degree: Biology, 2016, University of California – San Diego

 The endoplasmic reticulum is an organelle that responsible for the folding and processing of newly synthesized proteins. When unfolded or misfolded proteins accumulate in the… (more)

Subjects/Keywords: Biology; Pathology; Achromatopsia; ER Stress; PS19; Retinal Degeneration; Retinitis Pigmentosa; UPR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chan, P. (2016). Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/50737722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Priscilla. “Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration.” 2016. Thesis, University of California – San Diego. Accessed October 20, 2020. http://www.escholarship.org/uc/item/50737722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Priscilla. “Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration.” 2016. Web. 20 Oct 2020.

Vancouver:

Chan P. Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Oct 20]. Available from: http://www.escholarship.org/uc/item/50737722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan P. Endoplasmic Reticulum Stress in Mouse Models of Neurodegeneration. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/50737722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

27. Peña, Salvador. Hypoxia and the Mitochondrial Unfolded Protein Response.

Degree: PhD, 2017, University of Rochester

 Mitochondria are central to cellular metabolism, generating ATP through oxidative phosphorylation. Additionally, mitochondria are signaling organelles that regulate stress-induced cell death and adaptation. Altered production… (more)

Subjects/Keywords: ATFS-1; Ischemia reperfusion; Unfolded protein response; UPR; MosSCI

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Peña, S. (2017). Hypoxia and the Mitochondrial Unfolded Protein Response. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/32922

Chicago Manual of Style (16th Edition):

Peña, Salvador. “Hypoxia and the Mitochondrial Unfolded Protein Response.” 2017. Doctoral Dissertation, University of Rochester. Accessed October 20, 2020. http://hdl.handle.net/1802/32922.

MLA Handbook (7th Edition):

Peña, Salvador. “Hypoxia and the Mitochondrial Unfolded Protein Response.” 2017. Web. 20 Oct 2020.

Vancouver:

Peña S. Hypoxia and the Mitochondrial Unfolded Protein Response. [Internet] [Doctoral dissertation]. University of Rochester; 2017. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1802/32922.

Council of Science Editors:

Peña S. Hypoxia and the Mitochondrial Unfolded Protein Response. [Doctoral Dissertation]. University of Rochester; 2017. Available from: http://hdl.handle.net/1802/32922


NSYSU

28. Guo, Bo-Lin. The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway.

Degree: Master, Marine Biotechnology and Resources, 2017, NSYSU

 Parkinson's disease (PD) is a degenerative disorder of the central nervous system. Symptoms of PD include resting tremor, rigidity, bradykinesia, and posture instability. Motor symptoms… (more)

Subjects/Keywords: Parkinson's disease; ER stress; autophagy; UPR; 6-OHDA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guo, B. (2017). The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631117-233150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Bo-Lin. “The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway.” 2017. Thesis, NSYSU. Accessed October 20, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631117-233150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Bo-Lin. “The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway.” 2017. Web. 20 Oct 2020.

Vancouver:

Guo B. The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway. [Internet] [Thesis]. NSYSU; 2017. [cited 2020 Oct 20]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631117-233150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo B. The neuroprotective effects of marine-derived compound via the regulation of unfolded protein response and autophagy pathway. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631117-233150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

29. Chakrabarti, Anirikh. Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes.

Degree: PhD, Chemical Engineering, 2012, Cornell University

 Eukaryotes have developed evolutionarily conserved mechanisms to respond to diverse ranges of internal and external perturbations e.g., changes in oxygen/nutrient levels, temperature oscillations, protein folding… (more)

Subjects/Keywords: Hypoxia; upr; emt; Multiscale Modeling; Sensitivity Analysis; Robustness; Cellular Stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chakrabarti, A. (2012). Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29190

Chicago Manual of Style (16th Edition):

Chakrabarti, Anirikh. “Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes.” 2012. Doctoral Dissertation, Cornell University. Accessed October 20, 2020. http://hdl.handle.net/1813/29190.

MLA Handbook (7th Edition):

Chakrabarti, Anirikh. “Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes.” 2012. Web. 20 Oct 2020.

Vancouver:

Chakrabarti A. Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1813/29190.

Council of Science Editors:

Chakrabarti A. Systems Analysis Of Core Architectures Regulating Cellular Responses Under Stress In Eukaryotes. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29190


Cornell University

30. Singh, Rajni. The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis.

Degree: PhD, Physiology, 2013, Cornell University

 Type 2 diabetes is a growing epidemic that has been accompanied by a similarly drastic increase in obesity. Its occurrence is associated with [beta]-cell dysfunction,… (more)

Subjects/Keywords: Type 2 Diabetes; ER stress; UPR; Sel1L; Glucose; Energy Balance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Singh, R. (2013). The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33927

Chicago Manual of Style (16th Edition):

Singh, Rajni. “The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis.” 2013. Doctoral Dissertation, Cornell University. Accessed October 20, 2020. http://hdl.handle.net/1813/33927.

MLA Handbook (7th Edition):

Singh, Rajni. “The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis.” 2013. Web. 20 Oct 2020.

Vancouver:

Singh R. The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1813/33927.

Council of Science Editors:

Singh R. The Genetic Basis Of Type 2 Diabetes: The Role Of Sel1L In Glucose And Energy Homeostasis. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33927

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