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You searched for subject:(TrkB). Showing records 1 – 30 of 75 total matches.

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Duke University

1. Krishnamurthy, Kamesh. Disease Modification of Epilepsy by Disruption of TrkB Signaling .

Degree: 2019, Duke University

  Epilepsy is the most common acquired neurological disorder and is characterized by spontaneous, recurrent seizures. Of the various forms of epilepsy, Temporal Lobe Epilepsy… (more)

Subjects/Keywords: Neurosciences; Epilepsy; TrkB

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Krishnamurthy, K. (2019). Disease Modification of Epilepsy by Disruption of TrkB Signaling . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/18648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Krishnamurthy, Kamesh. “Disease Modification of Epilepsy by Disruption of TrkB Signaling .” 2019. Thesis, Duke University. Accessed January 22, 2020. http://hdl.handle.net/10161/18648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Krishnamurthy, Kamesh. “Disease Modification of Epilepsy by Disruption of TrkB Signaling .” 2019. Web. 22 Jan 2020.

Vancouver:

Krishnamurthy K. Disease Modification of Epilepsy by Disruption of TrkB Signaling . [Internet] [Thesis]. Duke University; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10161/18648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Krishnamurthy K. Disease Modification of Epilepsy by Disruption of TrkB Signaling . [Thesis]. Duke University; 2019. Available from: http://hdl.handle.net/10161/18648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

2. Babaei Bourojeni, Farin. The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons .

Degree: Neuroscience Studies, 2014, Queens University

 Although neurotrophins are conventionally associated with the proper growth and survival of developing neurons, there is increasing evidence that they play an equally significant role… (more)

Subjects/Keywords: Microscopy; TrkB; Motoneuron; Dendrite

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APA (6th Edition):

Babaei Bourojeni, F. (2014). The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8553

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Babaei Bourojeni, Farin. “The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons .” 2014. Thesis, Queens University. Accessed January 22, 2020. http://hdl.handle.net/1974/8553.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Babaei Bourojeni, Farin. “The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons .” 2014. Web. 22 Jan 2020.

Vancouver:

Babaei Bourojeni F. The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons . [Internet] [Thesis]. Queens University; 2014. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1974/8553.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Babaei Bourojeni F. The Distribution of Cytoplasmic and Membrane-Associated Tropomyosin-Related Kinase B (TrkB) Receptor in the Dendritic Tree of Adult Spinal Motoneurons . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8553

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. 尾立, 西市. TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

 Tropomyosin-related kinase B (TrkB) plays an important role in tumor progression in various kinds of cancers; however, little is known about biological significance of TrkB(more)

Subjects/Keywords: LCNEC; TrkB; BDNF; Invasion; Tumorigenicity; Lung cancer

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APA (6th Edition):

尾立, . (2013). TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

尾立, 西市. “TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.” 2013. Thesis, Kyushu University / 九州大学. Accessed January 22, 2020. http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

尾立, 西市. “TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.” 2013. Web. 22 Jan 2020.

Vancouver:

尾立 . TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

尾立 . TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Valverde Guevara, Yessenia Maria. Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響.

Degree: 博士(歯学), 2013, Niigata University / 新潟大学

学位の種類: 博士(歯学). 報告番号: 甲第3818号. 学位記番号: 新大院博(歯)甲第284号. 学位授与年月日: 平成25年9月20日

Peripheral nerve injury sometimes induces neuroma formation at the injury site. The inferior alveolar nerve (IAN) is… (more)

Subjects/Keywords: nerve injury; neuroma; IAN; BDNF; trkB

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APA (6th Edition):

Valverde Guevara, Y. M. (2013). Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/24561

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Valverde Guevara, Yessenia Maria. “Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響.” 2013. Thesis, Niigata University / 新潟大学. Accessed January 22, 2020. http://hdl.handle.net/10191/24561.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Valverde Guevara, Yessenia Maria. “Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響.” 2013. Web. 22 Jan 2020.

Vancouver:

Valverde Guevara YM. Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響. [Internet] [Thesis]. Niigata University / 新潟大学; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10191/24561.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Valverde Guevara YM. Effect of a local application of an antibody to BDNF on neuroma formation after transection of the inferior alveolar nerve in the rat : 下歯槽神経切断部に局所投与したBDNF抗体の神経腫形成に対する影響. [Thesis]. Niigata University / 新潟大学; 2013. Available from: http://hdl.handle.net/10191/24561

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

5. Nicolini, Chiara. DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM.

Degree: PhD, 2016, McMaster University

Autism is a neurodevelopmental disorder characterized by impairments in social communication and interaction and by repetitive patterns of behaviour, interests and activities. It is perhaps… (more)

Subjects/Keywords: Autism; LM22A-4; TrkB; VPA; Animal Model

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APA (6th Edition):

Nicolini, C. (2016). DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/20034

Chicago Manual of Style (16th Edition):

Nicolini, Chiara. “DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM.” 2016. Doctoral Dissertation, McMaster University. Accessed January 22, 2020. http://hdl.handle.net/11375/20034.

MLA Handbook (7th Edition):

Nicolini, Chiara. “DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM.” 2016. Web. 22 Jan 2020.

Vancouver:

Nicolini C. DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM. [Internet] [Doctoral dissertation]. McMaster University; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11375/20034.

Council of Science Editors:

Nicolini C. DEFECTIVE TrkB SIGNALING PATHWAYS IN IDIOPATHIC AUTISM. [Doctoral Dissertation]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20034


University of Tasmania

6. Zbela, AM. Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells.

Degree: 2019, University of Tasmania

 Over the last few decades, learning and memory processes have been extensively studied from a variety of perspectives. Long-term potentiation (LTP), the long lasting strengthening… (more)

Subjects/Keywords: TrkB; BNDF; optogenetics; LTP; synaptic plasticity

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APA (6th Edition):

Zbela, A. (2019). Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/31710/1/Zbela_whole_thesis.pdf ; Zbela, AM ORCID: 0000-0002-7306-1168 <https://orcid.org/0000-0002-7306-1168> 2019 , 'Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zbela, AM. “Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells.” 2019. Thesis, University of Tasmania. Accessed January 22, 2020. https://eprints.utas.edu.au/31710/1/Zbela_whole_thesis.pdf ; Zbela, AM ORCID: 0000-0002-7306-1168 <https://orcid.org/0000-0002-7306-1168> 2019 , 'Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zbela, AM. “Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells.” 2019. Web. 22 Jan 2020.

Vancouver:

Zbela A. Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells. [Internet] [Thesis]. University of Tasmania; 2019. [cited 2020 Jan 22]. Available from: https://eprints.utas.edu.au/31710/1/Zbela_whole_thesis.pdf ; Zbela, AM ORCID: 0000-0002-7306-1168 <https://orcid.org/0000-0002-7306-1168> 2019 , 'Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zbela A. Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells. [Thesis]. University of Tasmania; 2019. Available from: https://eprints.utas.edu.au/31710/1/Zbela_whole_thesis.pdf ; Zbela, AM ORCID: 0000-0002-7306-1168 <https://orcid.org/0000-0002-7306-1168> 2019 , 'Development of optogenetic approaches to modulate synaptic plasticity in the postsynaptic cells', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

7. Zamani, Akram. Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2).

Degree: 2017, University of Melbourne

 Suppressor of cytokine signalling 2 (SOCS2) negatively regulates cytokine signalling but it also has a positive effect on neurite outgrowth of cortical and dorsal root… (more)

Subjects/Keywords: BDNF; TrkB; SOCS2; Hippocampal neurons; Neurite growth

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APA (6th Edition):

Zamani, A. (2017). Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2). (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/192660

Chicago Manual of Style (16th Edition):

Zamani, Akram. “Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2).” 2017. Doctoral Dissertation, University of Melbourne. Accessed January 22, 2020. http://hdl.handle.net/11343/192660.

MLA Handbook (7th Edition):

Zamani, Akram. “Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2).” 2017. Web. 22 Jan 2020.

Vancouver:

Zamani A. Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2). [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11343/192660.

Council of Science Editors:

Zamani A. Tropomyosin related kinase B (TrkB) regulates neurite outgrowth via a novel interaction with suppressor of cytokine signalling 2 (SOCS2). [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/192660

8. Terol, Marie. Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus.

Degree: Docteur es, Biologie Santé, 2016, Montpellier

Le virus T lymphotropique humain de type 1 (HTLV-1) est l’agent étiologique d’une forme rare et très agressive de leucémie de l’adulte (ATL). Le processus… (more)

Subjects/Keywords: Htlv-1; Atl; Hbz; BDNF/TrkB; JunD; Htlv-1; Atl; Hbz; BDNF/TrkB; JunD

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APA (6th Edition):

Terol, M. (2016). Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2016MONTT012

Chicago Manual of Style (16th Edition):

Terol, Marie. “Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus.” 2016. Doctoral Dissertation, Montpellier. Accessed January 22, 2020. http://www.theses.fr/2016MONTT012.

MLA Handbook (7th Edition):

Terol, Marie. “Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus.” 2016. Web. 22 Jan 2020.

Vancouver:

Terol M. Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus. [Internet] [Doctoral dissertation]. Montpellier; 2016. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2016MONTT012.

Council of Science Editors:

Terol M. Caractérisation des mécanismes moléculaires impliqués dans la prolifération cellulaire induite par la protéine HBZ du rétrovirus HTLV-1 : Deciphering the molecular mechanisms responsible for the cellular proliferation induced by the HBZ oncoprotein of the HTLV-1 retrovirus. [Doctoral Dissertation]. Montpellier; 2016. Available from: http://www.theses.fr/2016MONTT012


Universidade do Rio Grande do Sul

9. Thomaz, Amanda Cristina Godot. Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano.

Degree: 2015, Universidade do Rio Grande do Sul

Meduloblastoma é o tumor maligno intracranial mais comum em crianças. A desregulação da sinalização BDNF/TrkB tem sido associada a aumento da proliferação, invasão e resistência… (more)

Subjects/Keywords: TrkB; Fator neurotrófico derivado do encéfalo; BDNF; Receptor trkB; Meduloblastoma; Medulloblastoma; Brain tumor; Childhood cancer

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APA (6th Edition):

Thomaz, A. C. G. (2015). Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/131184

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thomaz, Amanda Cristina Godot. “Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed January 22, 2020. http://hdl.handle.net/10183/131184.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thomaz, Amanda Cristina Godot. “Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano.” 2015. Web. 22 Jan 2020.

Vancouver:

Thomaz ACG. Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10183/131184.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thomaz ACG. Avaliação do papel da sinalização por BDNF/TRKB na viabilidade e sobrevivência de células de meduloblastoma humano. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/131184

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

10. Souza, Cassia Sallaberry de. Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo.

Degree: 2012, Universidade do Rio Grande do Sul

Os efeitos benéficos da administração crônica de cafeína sobre a memória têm sido observados em diferentes condições e modelos animais, mas os mecanismos subjacentes aos… (more)

Subjects/Keywords: Envelhecimento; Aging; Caffeine; Cafeína; Memory; Memória; BDNF; Fator neurotrófico derivado do encéfalo; Receptor trkB; TrkB

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APA (6th Edition):

Souza, C. S. d. (2012). Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/60964

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Souza, Cassia Sallaberry de. “Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed January 22, 2020. http://hdl.handle.net/10183/60964.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Souza, Cassia Sallaberry de. “Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo.” 2012. Web. 22 Jan 2020.

Vancouver:

Souza CSd. Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10183/60964.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Souza CSd. Cafeína reverte prejuízo da memória decorrente da idade com modificações no fator neurotrófico derivado do encéfalo. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/60964

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Abbaci, Amazigh. Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells.

Degree: Docteur es, Immunologie - Oncologie, 2017, Limoges

La leucémie lymphoïde chronique (LLC) est caractérisée par une accumulation anormale de lymphocytes B matures. Les thérapies actuelles reposent sur l'utilisation d'inhibiteurs ciblant les kinases… (more)

Subjects/Keywords: Lymphocytes B leucémiques; RCPG; NTSR2; TrkB; Apoptose; B-CLL; GPCR; NTSR2; TrkB; Apoptosis; 616.99; 615.37

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APA (6th Edition):

Abbaci, A. (2017). Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2017LIMO0029

Chicago Manual of Style (16th Edition):

Abbaci, Amazigh. “Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells.” 2017. Doctoral Dissertation, Limoges. Accessed January 22, 2020. http://www.theses.fr/2017LIMO0029.

MLA Handbook (7th Edition):

Abbaci, Amazigh. “Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells.” 2017. Web. 22 Jan 2020.

Vancouver:

Abbaci A. Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells. [Internet] [Doctoral dissertation]. Limoges; 2017. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2017LIMO0029.

Council of Science Editors:

Abbaci A. Caractérisation fonctionnelle du récepteur de type 2 de la neurotensine dans la résistance à la mort cellulaire des lymphocytes B au cours de la Leucémie Lymphoïde Chronique : Functional characterization of neurotensin type 2 receptor in cell death-resistance of chronic lymphocytic leukemia B cells. [Doctoral Dissertation]. Limoges; 2017. Available from: http://www.theses.fr/2017LIMO0029

12. Pedard, Martin. Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF.

Degree: Docteur es, Neurosciences, 2018, Bourgogne Franche-Comté

Le BDNF (brain-derived neurotrophic factor) a été découvert dans le cerveau et est largement impliqué dans la neuroplasticité, la mémoire et la cognition via l’activation… (more)

Subjects/Keywords: Bdnf; Fonction endothéliale; No; Cognition; TrkB; Inflammation; Bdnf; Endothelial fonction; No; Cognition; TrkB; Inflammation; 612.8

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APA (6th Edition):

Pedard, M. (2018). Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI008

Chicago Manual of Style (16th Edition):

Pedard, Martin. “Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed January 22, 2020. http://www.theses.fr/2018UBFCI008.

MLA Handbook (7th Edition):

Pedard, Martin. “Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF.” 2018. Web. 22 Jan 2020.

Vancouver:

Pedard M. Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2018UBFCI008.

Council of Science Editors:

Pedard M. Endothélium, inflammation et cognition : focus sur le BDNF : Endothelium, inflammation and cognition : focus on BDNF. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI008


Universidade do Rio Grande do Sul

13. Torres, Carolina Machado. Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal.

Degree: 2015, Universidade do Rio Grande do Sul

 Introdução O gene NTRK2 codifica um receptor pertencente a família de neurotrofinas Tirosina Kinase, conhecido como TrkB. O TrkB é um receptor de membrana com… (more)

Subjects/Keywords: Epilepsia do lobo temporal; Temporal lobe epilepsy; NTRK2; Receptor trkB; Fatores de crescimento neural; TrkB; Diagnóstico duplo (Psiquiatria); Polymorphisms; Polimorfismo genético

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Torres, C. M. (2015). Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/118289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Torres, Carolina Machado. “Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed January 22, 2020. http://hdl.handle.net/10183/118289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Torres, Carolina Machado. “Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal.” 2015. Web. 22 Jan 2020.

Vancouver:

Torres CM. Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10183/118289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Torres CM. Estudo de alelos variantes do gene da tirosina kinase B (NTRK2) na epilepsia do lobo temporal. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/118289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Suavinha, Angélica Caroline Dutra Romano. Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA.

Degree: Mestrado, Produtos Naturais e Sintéticos, 2014, University of São Paulo

 Recentemente suspeitas de que mecanismos epigenéticos poderiam estar relacionados à fisiopatologia da depressão foram levantadas. Estudos recentes indicam que as alterações na transcrição gênica, induzidas… (more)

Subjects/Keywords: antidepressant; antidepressivo; BDNF; BDNF; forced swimming test.; inhibitors of methylation; inibidores de metilação do DNA; mTOR; mTOR; nado forçado.; TRkB; TRkB

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APA (6th Edition):

Suavinha, A. C. D. R. (2014). Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60138/tde-24062014-092102/ ;

Chicago Manual of Style (16th Edition):

Suavinha, Angélica Caroline Dutra Romano. “Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA.” 2014. Masters Thesis, University of São Paulo. Accessed January 22, 2020. http://www.teses.usp.br/teses/disponiveis/60/60138/tde-24062014-092102/ ;.

MLA Handbook (7th Edition):

Suavinha, Angélica Caroline Dutra Romano. “Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA.” 2014. Web. 22 Jan 2020.

Vancouver:

Suavinha ACDR. Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60138/tde-24062014-092102/ ;.

Council of Science Editors:

Suavinha ACDR. Participação da via BDNF-TRkB-mTor do córtex pré-frontal medial ventral no efeito tipo antidepressivo induzido por inibidores da metilação do DNA. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/60/60138/tde-24062014-092102/ ;


University of Helsinki

15. Antila, Hanna. Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä.

Degree: Farmaceutiska fakulteten, 2012, University of Helsinki

 Tissue plasminogen activator (tPA) is a serine protease that cleaves the inactive plasminogen to a broad-spectrum protease plasmin. Plasmin is involved in the degradation of… (more)

Subjects/Keywords: tPA; BDNF; TrkB; Plasticity; Fluoxetine; muovautuvuus; fluoksetiini; Farmakologi; Pharmacology; Farmakologia; tPA; BDNF; TrkB; Plasticity; Fluoxetine; muovautuvuus; fluoksetiini

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APA (6th Edition):

Antila, H. (2012). Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/34785

Chicago Manual of Style (16th Edition):

Antila, Hanna. “Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä.” 2012. Masters Thesis, University of Helsinki. Accessed January 22, 2020. http://hdl.handle.net/10138/34785.

MLA Handbook (7th Edition):

Antila, Hanna. “Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä.” 2012. Web. 22 Jan 2020.

Vancouver:

Antila H. Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä. [Internet] [Masters thesis]. University of Helsinki; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10138/34785.

Council of Science Editors:

Antila H. Kudoksen plasminogeeniaktivaattori mahdollisena masennuslääkkeiden hermokasvutekijävaikutusten välittäjänä. [Masters Thesis]. University of Helsinki; 2012. Available from: http://hdl.handle.net/10138/34785


University of Edinburgh

16. Koudelka, Juraj. Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development.

Degree: 2013, University of Edinburgh

 Neurotrophins BDNF and NT4 influence the development of the rodent gustatory system. Despite binding to the same receptor, TrkB, they have different roles. BDNF is… (more)

Subjects/Keywords: 612.8; TrkB signalling; Gustatory development; Geniculate ganglion; Innervation

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APA (6th Edition):

Koudelka, J. (2013). Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8830

Chicago Manual of Style (16th Edition):

Koudelka, Juraj. “Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed January 22, 2020. http://hdl.handle.net/1842/8830.

MLA Handbook (7th Edition):

Koudelka, Juraj. “Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development.” 2013. Web. 22 Jan 2020.

Vancouver:

Koudelka J. Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1842/8830.

Council of Science Editors:

Koudelka J. Determining TrkB intracellular signalling pathways required for specific aspects of gustatory development. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/8830


Texas Medical Center

17. Graham, Timothy C. Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma.

Degree: MS, 2011, Texas Medical Center

  Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma  Timothy Christopher Graham, B.S. Supervisory Professor: Patrick Zweidler-McKay, MD/PhD  Neuroblastoma is the most common extra-cranial… (more)

Subjects/Keywords: neuroblastoma; TrkB; Fyn; proliferation; Cancer Biology; Molecular and Cellular Neuroscience

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APA (6th Edition):

Graham, T. C. (2011). Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/123

Chicago Manual of Style (16th Edition):

Graham, Timothy C. “Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma.” 2011. Masters Thesis, Texas Medical Center. Accessed January 22, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/123.

MLA Handbook (7th Edition):

Graham, Timothy C. “Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma.” 2011. Web. 22 Jan 2020.

Vancouver:

Graham TC. Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma. [Internet] [Masters thesis]. Texas Medical Center; 2011. [cited 2020 Jan 22]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/123.

Council of Science Editors:

Graham TC. Delineating the mechanism(s) of BDNF/TrkB mediated proliferation in Neuroblastoma. [Masters Thesis]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/123


University of Louisville

18. Tang, Tao. BDNF-TRKB signaling regulates adult taste bud innervation.

Degree: PhD, 2017, University of Louisville

  Taste receptor cells transduce stimuli transmitting information to gustatory neurons that carry it to the brain. They turn-over continuously in adulthood, and must be… (more)

Subjects/Keywords: BDNF; TrkB; taste bud; nerve fiber; Medicine and Health Sciences

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APA (6th Edition):

Tang, T. (2017). BDNF-TRKB signaling regulates adult taste bud innervation. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2762 ; https://ir.library.louisville.edu/etd/2762

Chicago Manual of Style (16th Edition):

Tang, Tao. “BDNF-TRKB signaling regulates adult taste bud innervation.” 2017. Doctoral Dissertation, University of Louisville. Accessed January 22, 2020. 10.18297/etd/2762 ; https://ir.library.louisville.edu/etd/2762.

MLA Handbook (7th Edition):

Tang, Tao. “BDNF-TRKB signaling regulates adult taste bud innervation.” 2017. Web. 22 Jan 2020.

Vancouver:

Tang T. BDNF-TRKB signaling regulates adult taste bud innervation. [Internet] [Doctoral dissertation]. University of Louisville; 2017. [cited 2020 Jan 22]. Available from: 10.18297/etd/2762 ; https://ir.library.louisville.edu/etd/2762.

Council of Science Editors:

Tang T. BDNF-TRKB signaling regulates adult taste bud innervation. [Doctoral Dissertation]. University of Louisville; 2017. Available from: 10.18297/etd/2762 ; https://ir.library.louisville.edu/etd/2762


University of Helsinki

19. Anttila, Jenni. Mania ja BDNF-viestinvälitys.

Degree: Farmaceutiska fakulteten, 2013, University of Helsinki

 Aivoperäisellä hermokasvutekijällä (BDNF) ja sen vaikutuksia välittävällä hermokasvutekijäreseptori TrkB:llä vaikuttaa olevan rooli mielialahäiriöiden, kuten masennuksen ja manian, synnyssä ja lääkehoidossa. BDNF on neurotrofiinien perheeseen kuuluva… (more)

Subjects/Keywords: BDNF; TrkB; litium; valproaatti; mania; primaarihermosolu; Farmakologi; Pharmacology; Farmakologia

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APA (6th Edition):

Anttila, J. (2013). Mania ja BDNF-viestinvälitys. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/38112

Chicago Manual of Style (16th Edition):

Anttila, Jenni. “Mania ja BDNF-viestinvälitys.” 2013. Masters Thesis, University of Helsinki. Accessed January 22, 2020. http://hdl.handle.net/10138/38112.

MLA Handbook (7th Edition):

Anttila, Jenni. “Mania ja BDNF-viestinvälitys.” 2013. Web. 22 Jan 2020.

Vancouver:

Anttila J. Mania ja BDNF-viestinvälitys. [Internet] [Masters thesis]. University of Helsinki; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10138/38112.

Council of Science Editors:

Anttila J. Mania ja BDNF-viestinvälitys. [Masters Thesis]. University of Helsinki; 2013. Available from: http://hdl.handle.net/10138/38112


University of California – San Diego

20. Sheik, Daniel. Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression.

Degree: Chemistry, 2016, University of California – San Diego

 This dissertation focuses on targeting amyloid aggregates as a means to inhibit disease transmission, as with semen-derived enhancer of virus infection (SEVI), or disease progression,… (more)

Subjects/Keywords: Chemistry; Polymer chemistry; Organic chemistry; Amyloid; HIV; Polymer; SEVI; SSTR4; TrkB

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APA (6th Edition):

Sheik, D. (2016). Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/7hc0v4xp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sheik, Daniel. “Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression.” 2016. Thesis, University of California – San Diego. Accessed January 22, 2020. http://www.escholarship.org/uc/item/7hc0v4xp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sheik, Daniel. “Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression.” 2016. Web. 22 Jan 2020.

Vancouver:

Sheik D. Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Jan 22]. Available from: http://www.escholarship.org/uc/item/7hc0v4xp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sheik D. Direct and Indirect Targeting of Amyloid Fibrils with Small Molecules and Polymeric Nanoparticles to Inhibit Disease Transmission and Progression. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/7hc0v4xp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

21. Vandenberg, Angela. The role of BDNF/TrkB signaling in the maturation of the adolescent PFC.

Degree: Neuroscience, 2013, University of California – San Francisco

 Brain derived neurotrophic factor (BDNF) and its receptor TrkB are involved in developmental maturation of cell processes, synaptogenesis, synaptic plasticity, and neuronal differentiation and survival.… (more)

Subjects/Keywords: Neurosciences; BDNF; GABAergic inhibition; prefrontal cortex; reversal learning; TrkB; Val66Met polymorphism

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APA (6th Edition):

Vandenberg, A. (2013). The role of BDNF/TrkB signaling in the maturation of the adolescent PFC. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/51p12017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vandenberg, Angela. “The role of BDNF/TrkB signaling in the maturation of the adolescent PFC.” 2013. Thesis, University of California – San Francisco. Accessed January 22, 2020. http://www.escholarship.org/uc/item/51p12017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vandenberg, Angela. “The role of BDNF/TrkB signaling in the maturation of the adolescent PFC.” 2013. Web. 22 Jan 2020.

Vancouver:

Vandenberg A. The role of BDNF/TrkB signaling in the maturation of the adolescent PFC. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2020 Jan 22]. Available from: http://www.escholarship.org/uc/item/51p12017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vandenberg A. The role of BDNF/TrkB signaling in the maturation of the adolescent PFC. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/51p12017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

22. Allen, Katherine M. Postnatal hippocampal neurogenesis in schizophrenia and during adolescence.

Degree: Psychiatry, 2014, University of New South Wales

 Schizophrenia may result from aberrations in maturational neurodevelopmental processes. While many genetic and environmental factors increase the risk of schizophrenia, it is currently unclear how… (more)

Subjects/Keywords: Adolescence; Schizophrenia; Neurogenesis; Testosterone; Sex hormones; Hippocampus; BDNF; TrkB

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APA (6th Edition):

Allen, K. M. (2014). Postnatal hippocampal neurogenesis in schizophrenia and during adolescence. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/55653 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:38291/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Allen, Katherine M. “Postnatal hippocampal neurogenesis in schizophrenia and during adolescence.” 2014. Doctoral Dissertation, University of New South Wales. Accessed January 22, 2020. http://handle.unsw.edu.au/1959.4/55653 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:38291/SOURCE02?view=true.

MLA Handbook (7th Edition):

Allen, Katherine M. “Postnatal hippocampal neurogenesis in schizophrenia and during adolescence.” 2014. Web. 22 Jan 2020.

Vancouver:

Allen KM. Postnatal hippocampal neurogenesis in schizophrenia and during adolescence. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2020 Jan 22]. Available from: http://handle.unsw.edu.au/1959.4/55653 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:38291/SOURCE02?view=true.

Council of Science Editors:

Allen KM. Postnatal hippocampal neurogenesis in schizophrenia and during adolescence. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/55653 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:38291/SOURCE02?view=true


University of Texas Southwestern Medical Center

23. Buzin, Nicole Renee. Role of BDNF-TrkB Signaling in Cocaine Addiction.

Degree: 2014, University of Texas Southwestern Medical Center

 Cocaine addiction results in neuroadaptations and drug-induced neuroplasticity that promote changes in protein expression and neuron morphology. Cocaine-induced increases in dopamine ultimately alter dopamine signaling… (more)

Subjects/Keywords: Brain-Derived Neurotrophic Factor; Cocaine-Related Disorders; Nucleus Accumbens; Receptor, trkB

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APA (6th Edition):

Buzin, N. R. (2014). Role of BDNF-TrkB Signaling in Cocaine Addiction. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Buzin, Nicole Renee. “Role of BDNF-TrkB Signaling in Cocaine Addiction.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 22, 2020. http://hdl.handle.net/2152.5/3306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Buzin, Nicole Renee. “Role of BDNF-TrkB Signaling in Cocaine Addiction.” 2014. Web. 22 Jan 2020.

Vancouver:

Buzin NR. Role of BDNF-TrkB Signaling in Cocaine Addiction. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2152.5/3306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Buzin NR. Role of BDNF-TrkB Signaling in Cocaine Addiction. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Arizona State University

24. Warraich, Zuha. Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery.

Degree: PhD, Neuroscience, 2013, Arizona State University

 Stroke remains the leading cause of adult disability in developed countries. Most survivors live with residual motor impairments that severely diminish independence and quality of… (more)

Subjects/Keywords: Nanoscience; Molecular biology; motor recovery; neuroplasticity; stroke recovery; trkB receptor agonist

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APA (6th Edition):

Warraich, Z. (2013). Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/18735

Chicago Manual of Style (16th Edition):

Warraich, Zuha. “Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery.” 2013. Doctoral Dissertation, Arizona State University. Accessed January 22, 2020. http://repository.asu.edu/items/18735.

MLA Handbook (7th Edition):

Warraich, Zuha. “Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery.” 2013. Web. 22 Jan 2020.

Vancouver:

Warraich Z. Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery. [Internet] [Doctoral dissertation]. Arizona State University; 2013. [cited 2020 Jan 22]. Available from: http://repository.asu.edu/items/18735.

Council of Science Editors:

Warraich Z. Investigating the Efficacy of Novel TrkB Agonists to Augment Stroke Recovery. [Doctoral Dissertation]. Arizona State University; 2013. Available from: http://repository.asu.edu/items/18735


University of Ottawa

25. Lee, Heow Won. Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction .

Degree: 2019, University of Ottawa

 Myocardial infarction (MI) induced heart failure (HF) is a leading cause of morbidity and mortality over the world. Regular exercise improves quality of life and… (more)

Subjects/Keywords: Exercise; Heart failure; BDNF-TrkB signaling; LV; PVN; RVLM; ANA-12

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APA (6th Edition):

Lee, H. W. (2019). Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/39642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Heow Won. “Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction .” 2019. Thesis, University of Ottawa. Accessed January 22, 2020. http://hdl.handle.net/10393/39642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Heow Won. “Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction .” 2019. Web. 22 Jan 2020.

Vancouver:

Lee HW. Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10393/39642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee HW. Role of BDNF in Cardiac Remodeling and Dysfunction in Rats After Myocardial Infarction . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/39642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

26. Harward, Stephen Cannada. BDNF-TrkB Signaling in Single-Spine Structural Plasticity .

Degree: 2016, Duke University

  Multiple lines of evidence reveal that activation of the tropomyosin related kinase B (TrkB) receptor is a critical molecular mechanism underlying status epilepticus (SE)… (more)

Subjects/Keywords: Neurosciences; BDNF; Epilepsy; FRET; LTP; Structural plasticity; TrkB

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APA (6th Edition):

Harward, S. C. (2016). BDNF-TrkB Signaling in Single-Spine Structural Plasticity . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/12096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harward, Stephen Cannada. “BDNF-TrkB Signaling in Single-Spine Structural Plasticity .” 2016. Thesis, Duke University. Accessed January 22, 2020. http://hdl.handle.net/10161/12096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harward, Stephen Cannada. “BDNF-TrkB Signaling in Single-Spine Structural Plasticity .” 2016. Web. 22 Jan 2020.

Vancouver:

Harward SC. BDNF-TrkB Signaling in Single-Spine Structural Plasticity . [Internet] [Thesis]. Duke University; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10161/12096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harward SC. BDNF-TrkB Signaling in Single-Spine Structural Plasticity . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/12096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

27. Gu, Bin. A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder .

Degree: 2015, Duke University

  Temporal lobe epilepsy is a common and devastating disorder that features recurrent seizures and is often associated with pathologic anxiety and hippocampal sclerosis. An… (more)

Subjects/Keywords: Pharmacology; anxiety; phospholipase C gamma 1; temporal lobe epilepsy; TrkB

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APA (6th Edition):

Gu, B. (2015). A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gu, Bin. “A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder .” 2015. Thesis, Duke University. Accessed January 22, 2020. http://hdl.handle.net/10161/9860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gu, Bin. “A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder .” 2015. Web. 22 Jan 2020.

Vancouver:

Gu B. A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder . [Internet] [Thesis]. Duke University; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10161/9860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gu B. A Peptide Selectively Uncoupling BDNF Receptor TrkB from Phospholipase C gamma 1 Prevents Epilepsy and Anxiety-like Disorder . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Helsinki

28. Rosenholm, Marko. Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP.

Degree: Farmaceutiska fakulteten, 2016, University of Helsinki

 Lääkeindusoidulla neuroplastisiteetilla, eli hermoston muovautuvuuden lisäämisellä on tutkimatonta potentiaalia neurologisten sairauksien lääkehoidossa. Useiden masennusoireita lievittävien lääkkeiden on havaittu välittävän neuroplastisuutta lisääviä vaikutuksia aivoperäisen hermokasvutekijän (BDNF)… (more)

Subjects/Keywords: THIP; pharmaco-EEG; TrkB; BDNF; neuroplasticity; neuroplastisuus; Farmakologi; Pharmacology; Farmakologia

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APA (6th Edition):

Rosenholm, M. (2016). Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/174504

Chicago Manual of Style (16th Edition):

Rosenholm, Marko. “Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP.” 2016. Masters Thesis, University of Helsinki. Accessed January 22, 2020. http://hdl.handle.net/10138/174504.

MLA Handbook (7th Edition):

Rosenholm, Marko. “Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP.” 2016. Web. 22 Jan 2020.

Vancouver:

Rosenholm M. Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP. [Internet] [Masters thesis]. University of Helsinki; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10138/174504.

Council of Science Editors:

Rosenholm M. Linking neuroplastic signaling responses with neuronal inhibition in the adult and developing brain : Pharmaco-EEG studies using THIP. [Masters Thesis]. University of Helsinki; 2016. Available from: http://hdl.handle.net/10138/174504

29. Stragier, Emilien. Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice.

Degree: Docteur es, Neurosciences, 2014, Université Paris Descartes – Paris V

La consommation chronique et excessive d’éthanol provoque des modifications neurobiologiques adaptatives. Les mécanismes qui les contrôlent sont multiples et certains ont été reliés à des… (more)

Subjects/Keywords: Prise chronique d’éthanol; Épigénétique; Hippocampe; Neurogenèse; Capacités cognitives; BDNF; TrkB; Plasticité; Souris; Chronic ethanol intake; Epigenetics; Hippocampus; Neurogenesis; Cognitive performances; BDNF; TrkB; Plasticity; Mice; 571.95

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stragier, E. (2014). Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05P604

Chicago Manual of Style (16th Edition):

Stragier, Emilien. “Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed January 22, 2020. http://www.theses.fr/2014PA05P604.

MLA Handbook (7th Edition):

Stragier, Emilien. “Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice.” 2014. Web. 22 Jan 2020.

Vancouver:

Stragier E. Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2014PA05P604.

Council of Science Editors:

Stragier E. Ethanol et épigénétique : conséquences neuroplastiques et fonctionnelles chez la souris : Ethanol and epigenetic : neuroplastic and functional consequences in mice. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05P604

30. Pinet, Sandra. Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement.

Degree: Docteur es, Neuro-Oncologie, 2016, Limoges

Les glioblastomes (GBM) sont des tumeurs astrocytaires au pronostic défavorable. L’échec des thérapies actuelles (chimio et radiothérapies) est principalement lié à la résistance des cellules… (more)

Subjects/Keywords: Glioblastomes; Cellules souches cancéreuses; Neurotrophines; TrkB; Radiothérapie; Cellules souches mésenchymateuses; Exosomes; Glioblastoma; Cancer stem cells; Exosomes; TrkB; Neurotrophins; Radiotherapy; Mesenchymal stem cells; 616.994

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APA (6th Edition):

Pinet, S. (2016). Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2016LIMO0039

Chicago Manual of Style (16th Edition):

Pinet, Sandra. “Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement.” 2016. Doctoral Dissertation, Limoges. Accessed January 22, 2020. http://www.theses.fr/2016LIMO0039.

MLA Handbook (7th Edition):

Pinet, Sandra. “Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement.” 2016. Web. 22 Jan 2020.

Vancouver:

Pinet S. Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement. [Internet] [Doctoral dissertation]. Limoges; 2016. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2016LIMO0039.

Council of Science Editors:

Pinet S. Rôle du transfert des récepteurs des neurotrophines via les exosomes dans l'agressivité du glioblastome et le contrôle du microenvironnement : Neurotrophins-containing exosomes promote the transfer of glioblastoma aggressiveness and the control of microenvironnement. [Doctoral Dissertation]. Limoges; 2016. Available from: http://www.theses.fr/2016LIMO0039

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