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You searched for subject:(Transgenic mice). Showing records 1 – 30 of 374 total matches.

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Rutgers University

1. Anderson, Jeremy, 1993-. Role of notch signaling after traumatic brain injury in a transgenic mouse model.

Degree: PhD, Biomedical Engineering, 2019, Rutgers University

Traumatic brain injury (TBI) is a leading cause of disability and death in the United States and worldwide. Endogenous neural stem/progenitor cells (NSPCs) in the… (more)

Subjects/Keywords: Neural stem cells; Transgenic mice

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APA (6th Edition):

Anderson, Jeremy, 1. (2019). Role of notch signaling after traumatic brain injury in a transgenic mouse model. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/61683/

Chicago Manual of Style (16th Edition):

Anderson, Jeremy, 1993-. “Role of notch signaling after traumatic brain injury in a transgenic mouse model.” 2019. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/61683/.

MLA Handbook (7th Edition):

Anderson, Jeremy, 1993-. “Role of notch signaling after traumatic brain injury in a transgenic mouse model.” 2019. Web. 18 Jan 2021.

Vancouver:

Anderson, Jeremy 1. Role of notch signaling after traumatic brain injury in a transgenic mouse model. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61683/.

Council of Science Editors:

Anderson, Jeremy 1. Role of notch signaling after traumatic brain injury in a transgenic mouse model. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61683/


University of Hawaii – Manoa

2. Suzuki, Shana T.N. Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ.

Degree: MS, 2011, University of Hawaii – Manoa

vii, 86 leaves, bound ill. (some col.) 29 cm

Myostatin, a member of the transforming growth factor-β superfamily, is a highly conserved negative regulator of… (more)

Subjects/Keywords: Transgenic mice  – Feeding and feeds; Transgenic mice  – Endocrinology

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APA (6th Edition):

Suzuki, S. T. N. (2011). Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ. (Masters Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/20774

Chicago Manual of Style (16th Edition):

Suzuki, Shana T N. “Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ.” 2011. Masters Thesis, University of Hawaii – Manoa. Accessed January 18, 2021. http://hdl.handle.net/10125/20774.

MLA Handbook (7th Edition):

Suzuki, Shana T N. “Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ.” 2011. Web. 18 Jan 2021.

Vancouver:

Suzuki STN. Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ. [Internet] [Masters thesis]. University of Hawaii – Manoa; 2011. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10125/20774.

Council of Science Editors:

Suzuki STN. Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ. [Masters Thesis]. University of Hawaii – Manoa; 2011. Available from: http://hdl.handle.net/10125/20774

3. Bertin, Eléonore. Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis.

Degree: Docteur es, Neurosciences, 2019, Bordeaux

 La signalisation purinergique ainsi que l'augmentation en surface des récepteurs ionotropiques P2X4 activé par l'ATP sont exacerbés dans divers troubles du SNC dont la sclérose… (more)

Subjects/Keywords: Souris transgéniques; Sla; Als; Transgenic mice

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APA (6th Edition):

Bertin, E. (2019). Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2019BORD0341

Chicago Manual of Style (16th Edition):

Bertin, Eléonore. “Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis.” 2019. Doctoral Dissertation, Bordeaux. Accessed January 18, 2021. http://www.theses.fr/2019BORD0341.

MLA Handbook (7th Edition):

Bertin, Eléonore. “Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis.” 2019. Web. 18 Jan 2021.

Vancouver:

Bertin E. Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis. [Internet] [Doctoral dissertation]. Bordeaux; 2019. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2019BORD0341.

Council of Science Editors:

Bertin E. Étude de l'augmentation du trafic en surface des récepteurs P2X4 de l’ATP à l’aide de nouveaux modèles murins transgéniques : implications dans les processus mnésiques et la sclérose latérale amyotrophique : Study of the increase of P2X4 ATP-gated receptor surface trafficking using novel transgenic murine models : role in memory processes and amyotrophic lateral sclerosis. [Doctoral Dissertation]. Bordeaux; 2019. Available from: http://www.theses.fr/2019BORD0341


East Carolina University

4. Kirsanov, Oleksandr. ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA.

Degree: MS, MS-Biomedical Science, 2018, East Carolina University

 Spermatogenesis in the mammalian testis results in the daily production of millions of spermatozoa. This developmental process is founded upon the actions of a small… (more)

Subjects/Keywords: Spermatogonia; Animals; Mice, Transgenic; Mammals; Spermatogenesis

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APA (6th Edition):

Kirsanov, O. (2018). ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/6969

Chicago Manual of Style (16th Edition):

Kirsanov, Oleksandr. “ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA.” 2018. Masters Thesis, East Carolina University. Accessed January 18, 2021. http://hdl.handle.net/10342/6969.

MLA Handbook (7th Edition):

Kirsanov, Oleksandr. “ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA.” 2018. Web. 18 Jan 2021.

Vancouver:

Kirsanov O. ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA. [Internet] [Masters thesis]. East Carolina University; 2018. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10342/6969.

Council of Science Editors:

Kirsanov O. ANALYSIS OF TRANSGENIC MOUSE MODELS TO STUDY MAMMALIAN SPERMATOGONIA. [Masters Thesis]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/6969


University of Manitoba

5. Yeganeh, Behzad. Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter.

Degree: Biochemistry and Medical Genetics, 2010, University of Manitoba

 Calreticulin (CRT) is a multifunctional Ca2+ dependent chaperone protein, which is localized to the endoplasmic reticulum and plays many important biological roles. In addition to… (more)

Subjects/Keywords: Molecular Biology; Lung Cancer; Transgenic mice; Calreticulin

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APA (6th Edition):

Yeganeh, B. (2010). Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4239

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeganeh, Behzad. “Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter.” 2010. Thesis, University of Manitoba. Accessed January 18, 2021. http://hdl.handle.net/1993/4239.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeganeh, Behzad. “Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter.” 2010. Web. 18 Jan 2021.

Vancouver:

Yeganeh B. Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1993/4239.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeganeh B. Characterization of lung adenocarcinoma in transgenic mice overexpressing calreticulin under control of the Tie-2 promoter. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4239

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

6. Guo, Su Tang. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.

Degree: PhD, 2018, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Colon cancer is one of the most common and deadly malignancies. Despite recent advances in early diagnosis and… (more)

Subjects/Keywords: INPP4B; colon cancer; pten; transgenic mice

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APA (6th Edition):

Guo, S. T. (2018). The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1386333

Chicago Manual of Style (16th Edition):

Guo, Su Tang. “The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.” 2018. Doctoral Dissertation, University of Newcastle. Accessed January 18, 2021. http://hdl.handle.net/1959.13/1386333.

MLA Handbook (7th Edition):

Guo, Su Tang. “The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.” 2018. Web. 18 Jan 2021.

Vancouver:

Guo ST. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. [Internet] [Doctoral dissertation]. University of Newcastle; 2018. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1959.13/1386333.

Council of Science Editors:

Guo ST. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. [Doctoral Dissertation]. University of Newcastle; 2018. Available from: http://hdl.handle.net/1959.13/1386333


Columbia University

7. Abrams, Jeffrey. Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice.

Degree: 2017, Columbia University

 Proper functioning of the voltage gated sodium channel, NaV1.5, is essential for maintenance of normal cardiac electrophysiological properties. Changes to the biophysical properties of sodium… (more)

Subjects/Keywords: Biophysics; Sodium channels; Transgenic mice; Electrophysiology

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APA (6th Edition):

Abrams, J. (2017). Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D86Q28NQ

Chicago Manual of Style (16th Edition):

Abrams, Jeffrey. “Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice.” 2017. Doctoral Dissertation, Columbia University. Accessed January 18, 2021. https://doi.org/10.7916/D86Q28NQ.

MLA Handbook (7th Edition):

Abrams, Jeffrey. “Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice.” 2017. Web. 18 Jan 2021.

Vancouver:

Abrams J. Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2021 Jan 18]. Available from: https://doi.org/10.7916/D86Q28NQ.

Council of Science Editors:

Abrams J. Dysfunctional Sodium Channels and Arrhythmogenesis: Insights into the Molecular Regulation of Cardiac Sodium Channels Using Transgenic Mice. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D86Q28NQ


George Mason University

8. Bozzelli, P. Lorenzo. The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease .

Degree: 2014, George Mason University

 The role of essential trace elements, such as zinc and copper, in Alzheimer’s disease (AD) has received much attention in the last decade. Zinc (Zn)… (more)

Subjects/Keywords: ZnT3; transgenic mice; biometals; Alzheimer's disease

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APA (6th Edition):

Bozzelli, P. L. (2014). The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease . (Thesis). George Mason University. Retrieved from http://hdl.handle.net/1920/9077

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bozzelli, P Lorenzo. “The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease .” 2014. Thesis, George Mason University. Accessed January 18, 2021. http://hdl.handle.net/1920/9077.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bozzelli, P Lorenzo. “The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease .” 2014. Web. 18 Jan 2021.

Vancouver:

Bozzelli PL. The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease . [Internet] [Thesis]. George Mason University; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1920/9077.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bozzelli PL. The Effects That Dietary Manipulations of Zinc and Copper Have on Nest Building, Zinc Transporter and Glial Fibrillary Acidic Protein Expression in a Transgenic Mouse Model of Alzheimer’s Disease . [Thesis]. George Mason University; 2014. Available from: http://hdl.handle.net/1920/9077

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Rojas, José-Manuel. Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens.

Degree: PhD, 2003, Nottingham Trent University

 CD4⁺ T cells play a central role in antitumour immunity; not only do they provide help for the development of CTL recognising tumour antigens but… (more)

Subjects/Keywords: 616; Transgenic mice

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APA (6th Edition):

Rojas, J. (2003). Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens. (Doctoral Dissertation). Nottingham Trent University. Retrieved from http://irep.ntu.ac.uk/id/eprint/40993/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273771

Chicago Manual of Style (16th Edition):

Rojas, José-Manuel. “Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens.” 2003. Doctoral Dissertation, Nottingham Trent University. Accessed January 18, 2021. http://irep.ntu.ac.uk/id/eprint/40993/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273771.

MLA Handbook (7th Edition):

Rojas, José-Manuel. “Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens.” 2003. Web. 18 Jan 2021.

Vancouver:

Rojas J. Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens. [Internet] [Doctoral dissertation]. Nottingham Trent University; 2003. [cited 2021 Jan 18]. Available from: http://irep.ntu.ac.uk/id/eprint/40993/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273771.

Council of Science Editors:

Rojas J. Identification of novel immunogenic HLA-DR-restricted peptides from tumour-associated antigens. [Doctoral Dissertation]. Nottingham Trent University; 2003. Available from: http://irep.ntu.ac.uk/id/eprint/40993/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273771


Montana State University

10. Orr, Miranda Ethel. Mouse and stem cell models of frontotemporal dementia.

Degree: PhD, College of Letters & Science, 2012, Montana State University

 Alzheimer's disease (AD) is the most prevalent brain disease in the United States, and an escalating health concern. AD patient brains acquire hallmark protein aggregates,… (more)

Subjects/Keywords: Dementia.; Alzheimer's disease.; Stem cells.; Transgenic mice.

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APA (6th Edition):

Orr, M. E. (2012). Mouse and stem cell models of frontotemporal dementia. (Doctoral Dissertation). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/1994

Chicago Manual of Style (16th Edition):

Orr, Miranda Ethel. “Mouse and stem cell models of frontotemporal dementia.” 2012. Doctoral Dissertation, Montana State University. Accessed January 18, 2021. https://scholarworks.montana.edu/xmlui/handle/1/1994.

MLA Handbook (7th Edition):

Orr, Miranda Ethel. “Mouse and stem cell models of frontotemporal dementia.” 2012. Web. 18 Jan 2021.

Vancouver:

Orr ME. Mouse and stem cell models of frontotemporal dementia. [Internet] [Doctoral dissertation]. Montana State University; 2012. [cited 2021 Jan 18]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/1994.

Council of Science Editors:

Orr ME. Mouse and stem cell models of frontotemporal dementia. [Doctoral Dissertation]. Montana State University; 2012. Available from: https://scholarworks.montana.edu/xmlui/handle/1/1994


University of Hong Kong

11. Ng, Chun-pong. The role of Densin-180 on mouse behavior and synaptic protein distribution.

Degree: 2013, University of Hong Kong

 Lrrc7 (Leucine Rich Repeat Containing 7) encodes Densin-180, a scaffold protein located in the postsynaptic density of excitatory synapses. A transgenic mouse line with disruption… (more)

Subjects/Keywords: Transgenic mice - Behavior

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APA (6th Edition):

Ng, C. (2013). The role of Densin-180 on mouse behavior and synaptic protein distribution. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/249861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ng, Chun-pong. “The role of Densin-180 on mouse behavior and synaptic protein distribution.” 2013. Thesis, University of Hong Kong. Accessed January 18, 2021. http://hdl.handle.net/10722/249861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ng, Chun-pong. “The role of Densin-180 on mouse behavior and synaptic protein distribution.” 2013. Web. 18 Jan 2021.

Vancouver:

Ng C. The role of Densin-180 on mouse behavior and synaptic protein distribution. [Internet] [Thesis]. University of Hong Kong; 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10722/249861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ng C. The role of Densin-180 on mouse behavior and synaptic protein distribution. [Thesis]. University of Hong Kong; 2013. Available from: http://hdl.handle.net/10722/249861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

12. Chan, Sing-kwok. Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation.

Degree: 1998, University of Hong Kong

Subjects/Keywords: Transgenic mice.; Endothelins.

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APA (6th Edition):

Chan, S. (1998). Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/35179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Sing-kwok. “Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation.” 1998. Thesis, University of Hong Kong. Accessed January 18, 2021. http://hdl.handle.net/10722/35179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Sing-kwok. “Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation.” 1998. Web. 18 Jan 2021.

Vancouver:

Chan S. Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation. [Internet] [Thesis]. University of Hong Kong; 1998. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10722/35179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan S. Mouse preproendothelin-1 gene: transgenic mouse models to study tissue-specific and developmental expression andregulation. [Thesis]. University of Hong Kong; 1998. Available from: http://hdl.handle.net/10722/35179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

13. Myelnikov, Dmitriy. Transforming mice : technique and communication in the making of transgenic animals, 1974-1988.

Degree: PhD, 2015, University of Cambridge

Subjects/Keywords: 500; transgenic animals; transgenic mice

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APA (6th Edition):

Myelnikov, D. (2015). Transforming mice : technique and communication in the making of transgenic animals, 1974-1988. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.16164 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678957

Chicago Manual of Style (16th Edition):

Myelnikov, Dmitriy. “Transforming mice : technique and communication in the making of transgenic animals, 1974-1988.” 2015. Doctoral Dissertation, University of Cambridge. Accessed January 18, 2021. https://doi.org/10.17863/CAM.16164 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678957.

MLA Handbook (7th Edition):

Myelnikov, Dmitriy. “Transforming mice : technique and communication in the making of transgenic animals, 1974-1988.” 2015. Web. 18 Jan 2021.

Vancouver:

Myelnikov D. Transforming mice : technique and communication in the making of transgenic animals, 1974-1988. [Internet] [Doctoral dissertation]. University of Cambridge; 2015. [cited 2021 Jan 18]. Available from: https://doi.org/10.17863/CAM.16164 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678957.

Council of Science Editors:

Myelnikov D. Transforming mice : technique and communication in the making of transgenic animals, 1974-1988. [Doctoral Dissertation]. University of Cambridge; 2015. Available from: https://doi.org/10.17863/CAM.16164 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678957


Central Connecticut State University

14. Lanza, Janna R., 1976-. Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza.

Degree: Department of Biological Sciences, 2004, Central Connecticut State University

 Infertility is a common problem, affecting approximately 10% of all couples attempting to conceive (Watson et al., 1998). Although numerous potential causes of human infertility… (more)

Subjects/Keywords: Transgenic mice; Mice  – Infertility

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APA (6th Edition):

Lanza, Janna R., 1. (2004). Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lanza, Janna R., 1976-. “Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza.” 2004. Thesis, Central Connecticut State University. Accessed January 18, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lanza, Janna R., 1976-. “Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza.” 2004. Web. 18 Jan 2021.

Vancouver:

Lanza, Janna R. 1. Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza. [Internet] [Thesis]. Central Connecticut State University; 2004. [cited 2021 Jan 18]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lanza, Janna R. 1. Abnormal apoptosis in sterile mshi/mshi mutant mice / Janna R. Lanza. [Thesis]. Central Connecticut State University; 2004. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

15. Ye, Naiqing. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.

Degree: Department of Biomolecular Sciences, 2006, Central Connecticut State University

 The male sterility and histoincompatibility mutant, mshi, results from a recessive mutation that arose spontaneously in the BALB/cBy inbred mouse strain. Using a 402-member BALB/cBy-mshi/mshi… (more)

Subjects/Keywords: Mice  – Infertility; Transgenic mice

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APA (6th Edition):

Ye, N. (2006). Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ye, Naiqing. “Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.” 2006. Thesis, Central Connecticut State University. Accessed January 18, 2021. http://content.library.ccsu.edu/u?/ccsutheses,976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ye, Naiqing. “Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.” 2006. Web. 18 Jan 2021.

Vancouver:

Ye N. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. [Internet] [Thesis]. Central Connecticut State University; 2006. [cited 2021 Jan 18]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ye N. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. [Thesis]. Central Connecticut State University; 2006. Available from: http://content.library.ccsu.edu/u?/ccsutheses,976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

16. Lu, Yi-Chien. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.

Degree: Department of Biomolecular Sciences, 2004, Central Connecticut State University

 The recessive male sterility and histoincompatibility mutation (mshi) in the mouse generates pleiotropic effects on graft transplantation and male reproduction. Previous analysis of backcross mice(more)

Subjects/Keywords: Mice  – Infertility; Transgenic mice

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APA (6th Edition):

Lu, Y. (2004). Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Yi-Chien. “Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.” 2004. Thesis, Central Connecticut State University. Accessed January 18, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Yi-Chien. “Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.” 2004. Web. 18 Jan 2021.

Vancouver:

Lu Y. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. [Internet] [Thesis]. Central Connecticut State University; 2004. [cited 2021 Jan 18]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu Y. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. [Thesis]. Central Connecticut State University; 2004. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

17. Yeung, Rigil Kent LIFS. Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation.

Degree: 2018, Hong Kong University of Science and Technology

 Intronic polymorphisms of the GABAA receptor β2 subunit gene (GABRB2) were earlier associated with schizophrenia, deficit of gene expression, different electrophysiological properties of the long… (more)

Subjects/Keywords: GABA ; Receptors ; Neurobehavioral disorders ; Transgenic mice ; Messenger RNA ; Mice as laboratory animals

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APA (6th Edition):

Yeung, R. K. L. (2018). Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-92991 ; https://doi.org/10.14711/thesis-991012615863503412 ; http://repository.ust.hk/ir/bitstream/1783.1-92991/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeung, Rigil Kent LIFS. “Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation.” 2018. Thesis, Hong Kong University of Science and Technology. Accessed January 18, 2021. http://repository.ust.hk/ir/Record/1783.1-92991 ; https://doi.org/10.14711/thesis-991012615863503412 ; http://repository.ust.hk/ir/bitstream/1783.1-92991/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeung, Rigil Kent LIFS. “Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation.” 2018. Web. 18 Jan 2021.

Vancouver:

Yeung RKL. Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2018. [cited 2021 Jan 18]. Available from: http://repository.ust.hk/ir/Record/1783.1-92991 ; https://doi.org/10.14711/thesis-991012615863503412 ; http://repository.ust.hk/ir/bitstream/1783.1-92991/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeung RKL. Gabrb2 knockout mice displayed schizophrenia-like and comorbid phenotypes with GABAergic interneuron-astrocyte-microglia dysregulation and neuroinflammation. [Thesis]. Hong Kong University of Science and Technology; 2018. Available from: http://repository.ust.hk/ir/Record/1783.1-92991 ; https://doi.org/10.14711/thesis-991012615863503412 ; http://repository.ust.hk/ir/bitstream/1783.1-92991/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Κατσιμπούλας, Μιχαήλ. Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Introduction: The newly discovered endocannabinoid system contributes tothe physiological regulation of energy balance, food intake and lipid and glucosemetabolism through both central and peripheral effects.… (more)

Subjects/Keywords: Αθηροσκλήρωση; Ενδοκανναβινοειδή; Άσκηση; Μύες; Διαγονιδιακοί μύες; Αναστολέας; Atherosclerosis; Endocannabinoids; Exercise; Mice; Transgenic mice; Inhibitors

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APA (6th Edition):

Κατσιμπούλας, . . (2013). Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/38033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κατσιμπούλας, Μιχαήλ. “Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 18, 2021. http://hdl.handle.net/10442/hedi/38033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κατσιμπούλας, Μιχαήλ. “Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη.” 2013. Web. 18 Jan 2021.

Vancouver:

Κατσιμπούλας . Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10442/hedi/38033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κατσιμπούλας . Ο ρόλος των ενδοκανναβινοειδών στην αθηροσκλήρωση: πειραματική μελέτη. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/38033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Dalhousie University

19. Fraser, Leanne M. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.

Degree: PhD, Department of Psychology and Neuroscience, 2013, Dalhousie University

 The triple transgenic (3xTg-AD) mouse model of Alzheimer’s disease (AD) possesses three transgenes that lead to the development of amyloid-beta (A?) plaques (APPswe, PS1M146V) and… (more)

Subjects/Keywords: "Transgenic mice"; "Alzheimer's disease"; "Mouse models"; "Behaviour"; "Memory"; "Motor coordination"

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APA (6th Edition):

Fraser, L. M. (2013). Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/28080

Chicago Manual of Style (16th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Doctoral Dissertation, Dalhousie University. Accessed January 18, 2021. http://hdl.handle.net/10222/28080.

MLA Handbook (7th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Web. 18 Jan 2021.

Vancouver:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10222/28080.

Council of Science Editors:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/28080


University of Hong Kong

20. Siu, Kwan-yin. The development and characterization of a knockout model for secretin.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Secretin.; Transgenic mice - Physiology.

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APA (6th Edition):

Siu, K. (2008). The development and characterization of a knockout model for secretin. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/55588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Siu, Kwan-yin. “The development and characterization of a knockout model for secretin.” 2008. Thesis, University of Hong Kong. Accessed January 18, 2021. http://hdl.handle.net/10722/55588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Siu, Kwan-yin. “The development and characterization of a knockout model for secretin.” 2008. Web. 18 Jan 2021.

Vancouver:

Siu K. The development and characterization of a knockout model for secretin. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10722/55588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Siu K. The development and characterization of a knockout model for secretin. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/55588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

21. 鍾志堅. The development and characterization of a gene-knockout mouse model for secretin receptor.

Degree: 2005, University of Hong Kong

Subjects/Keywords: Transgenic mice - Physiology.; Secretin - Receptors.

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APA (6th Edition):

鍾志堅.. (2005). The development and characterization of a gene-knockout mouse model for secretin receptor. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/134065

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

鍾志堅.. “The development and characterization of a gene-knockout mouse model for secretin receptor.” 2005. Thesis, University of Hong Kong. Accessed January 18, 2021. http://hdl.handle.net/10722/134065.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

鍾志堅.. “The development and characterization of a gene-knockout mouse model for secretin receptor.” 2005. Web. 18 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

鍾志堅.. The development and characterization of a gene-knockout mouse model for secretin receptor. [Internet] [Thesis]. University of Hong Kong; 2005. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10722/134065.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

鍾志堅.. The development and characterization of a gene-knockout mouse model for secretin receptor. [Thesis]. University of Hong Kong; 2005. Available from: http://hdl.handle.net/10722/134065

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

22. Moshkani, Safiehkhatoon. B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity.

Degree: PhD, 2012, University of Rochester

 TNF- transgenic (TNFtg) mice develop a spontaneous inflammatory joint disease resembling rheumatoid arthritis (RA) in patients. Bin cells phenotypically defined as CD23+CD21highCD1dhigh B cells have… (more)

Subjects/Keywords: Autoimmunity; B Cells; Inflammation; Lymph Node; Rheumatoid Arthritis; TNF-Transgenic Mice

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APA (6th Edition):

Moshkani, S. (2012). B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25517

Chicago Manual of Style (16th Edition):

Moshkani, Safiehkhatoon. “B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity.” 2012. Doctoral Dissertation, University of Rochester. Accessed January 18, 2021. http://hdl.handle.net/1802/25517.

MLA Handbook (7th Edition):

Moshkani, Safiehkhatoon. “B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity.” 2012. Web. 18 Jan 2021.

Vancouver:

Moshkani S. B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1802/25517.

Council of Science Editors:

Moshkani S. B Cells in Inflamed Lymph Nodes: A Link between Inflammation and Autoimmunity. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25517


University of Alberta

23. Hsi Dickie, Belinda. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.

Degree: PhD, Department of Surgery, 2009, University of Alberta

 The feasibility of the Alb-uPA/SCID/Bg chimeric mouse as a model for Hepatitis C Virus (HCV) infection was assessed experimentally by (1) the infection and treatment… (more)

Subjects/Keywords: Hepatitis C; Mouse Model; Transgenic mice; Alb-uPA/SCID/Beige mouse

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APA (6th Edition):

Hsi Dickie, B. (2009). Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/rr171x54d

Chicago Manual of Style (16th Edition):

Hsi Dickie, Belinda. “Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.” 2009. Doctoral Dissertation, University of Alberta. Accessed January 18, 2021. https://era.library.ualberta.ca/files/rr171x54d.

MLA Handbook (7th Edition):

Hsi Dickie, Belinda. “Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.” 2009. Web. 18 Jan 2021.

Vancouver:

Hsi Dickie B. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2021 Jan 18]. Available from: https://era.library.ualberta.ca/files/rr171x54d.

Council of Science Editors:

Hsi Dickie B. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/rr171x54d


University of Oulu

24. Chen, Z. (Zhijun). Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals.

Degree: 2008, University of Oulu

 Abstract A data base search using the amino acid sequence of Saccharomyces cerevisiae Etr1p, the last enzyme of mitochondrial fatty acid synthesis type II (FAS… (more)

Subjects/Keywords: MECR/ETR1; biochemistry; lipid; mitochondria; structural enzymology; transgenic mice

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APA (6th Edition):

Chen, Z. (. (2008). Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514289804

Chicago Manual of Style (16th Edition):

Chen, Z (Zhijun). “Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals.” 2008. Doctoral Dissertation, University of Oulu. Accessed January 18, 2021. http://urn.fi/urn:isbn:9789514289804.

MLA Handbook (7th Edition):

Chen, Z (Zhijun). “Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals.” 2008. Web. 18 Jan 2021.

Vancouver:

Chen Z(. Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals. [Internet] [Doctoral dissertation]. University of Oulu; 2008. [cited 2021 Jan 18]. Available from: http://urn.fi/urn:isbn:9789514289804.

Council of Science Editors:

Chen Z(. Characterization of the 2-enoyl thioester reductase of mitochondrial fatty acid synthesis type II in mammals. [Doctoral Dissertation]. University of Oulu; 2008. Available from: http://urn.fi/urn:isbn:9789514289804


University of Michigan

25. Osborn, Laurelee. Expression Of Amylase Genes In Transgenic Mice.

Degree: PhD, Molecular biology, 1987, University of Michigan

 The amylase multigene family is a useful model system for the study of evolution and regulation of gene expression. I have characterized two nonallelic murine… (more)

Subjects/Keywords: Amylase; Expression; Genes; Mice; Transgenic

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APA (6th Edition):

Osborn, L. (1987). Expression Of Amylase Genes In Transgenic Mice. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/128119

Chicago Manual of Style (16th Edition):

Osborn, Laurelee. “Expression Of Amylase Genes In Transgenic Mice.” 1987. Doctoral Dissertation, University of Michigan. Accessed January 18, 2021. http://hdl.handle.net/2027.42/128119.

MLA Handbook (7th Edition):

Osborn, Laurelee. “Expression Of Amylase Genes In Transgenic Mice.” 1987. Web. 18 Jan 2021.

Vancouver:

Osborn L. Expression Of Amylase Genes In Transgenic Mice. [Internet] [Doctoral dissertation]. University of Michigan; 1987. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2027.42/128119.

Council of Science Editors:

Osborn L. Expression Of Amylase Genes In Transgenic Mice. [Doctoral Dissertation]. University of Michigan; 1987. Available from: http://hdl.handle.net/2027.42/128119


Penn State University

26. Shi, Chenxu. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .

Degree: 2011, Penn State University

 Polyamines, including putrescine, spermidine, and spermine, are ubiquitous polycationic compounds that are essential for cell growth and differentiation. To better understand cellular function of polyamines,… (more)

Subjects/Keywords: Polyamines; S-adenosylmethionine decarboxylase; Spermidine synthase; Transgenic mice; Nonmelanoma skin cancer

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APA (6th Edition):

Shi, C. (2011). INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/12128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shi, Chenxu. “INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .” 2011. Thesis, Penn State University. Accessed January 18, 2021. https://submit-etda.libraries.psu.edu/catalog/12128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shi, Chenxu. “INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .” 2011. Web. 18 Jan 2021.

Vancouver:

Shi C. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Jan 18]. Available from: https://submit-etda.libraries.psu.edu/catalog/12128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shi C. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/12128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

27. Lee, Rebecca Arwyn. L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice.

Degree: MS, Medical Sciences, 2015, Boston University

 As we learn more about immune cell subpopulations, we find an increasingly complex system of cells with diverse functions. L2pB1 cells are a PD-L2 positive… (more)

Subjects/Keywords: Immunology; B1a cells; B cells; Depletion; Diabetes; Diphtheria toxin; Transgenic mice

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, R. A. (2015). L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16050

Chicago Manual of Style (16th Edition):

Lee, Rebecca Arwyn. “L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice.” 2015. Masters Thesis, Boston University. Accessed January 18, 2021. http://hdl.handle.net/2144/16050.

MLA Handbook (7th Edition):

Lee, Rebecca Arwyn. “L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice.” 2015. Web. 18 Jan 2021.

Vancouver:

Lee RA. L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice. [Internet] [Masters thesis]. Boston University; 2015. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2144/16050.

Council of Science Editors:

Lee RA. L2PB1 cell depletion with diphtheria toxin in PD-L2 KIKO mice. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16050


Brigham Young University

28. Jimenez Rondan, Felix Ruben. The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung.

Degree: PhD, 2015, Brigham Young University

 The tight junctions (TJ), which are located in the apical region between epithelial and endothelial cells, regulate the paracellular diffusion of ions and small molecules… (more)

Subjects/Keywords: claudin 6; mice; transgenic; murine; lung; Cell and Developmental Biology; Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jimenez Rondan, F. R. (2015). The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5413&context=etd

Chicago Manual of Style (16th Edition):

Jimenez Rondan, Felix Ruben. “The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung.” 2015. Doctoral Dissertation, Brigham Young University. Accessed January 18, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5413&context=etd.

MLA Handbook (7th Edition):

Jimenez Rondan, Felix Ruben. “The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung.” 2015. Web. 18 Jan 2021.

Vancouver:

Jimenez Rondan FR. The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung. [Internet] [Doctoral dissertation]. Brigham Young University; 2015. [cited 2021 Jan 18]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5413&context=etd.

Council of Science Editors:

Jimenez Rondan FR. The Biology of Claudin 6 (Cldn6) in the Developing Mouse Lung. [Doctoral Dissertation]. Brigham Young University; 2015. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5413&context=etd


NSYSU

29. Huang, Chi-Yuan. Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme.

Degree: Master, Institute of Biomedical Sciences, 2018, NSYSU

 Glioblastoma multiforme (GBM) is the most common, deadly and aggressive brain tumor arising from glial cells. In contrast to other solid cancers, clinical trials in… (more)

Subjects/Keywords: high recurrence; Malignant tumor; transgenic mice; Glioblastoma multiforme; knockdown

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, C. (2018). Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0710118-094036

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Chi-Yuan. “Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme.” 2018. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0710118-094036.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Chi-Yuan. “Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme.” 2018. Web. 18 Jan 2021.

Vancouver:

Huang C. Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme. [Internet] [Thesis]. NSYSU; 2018. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0710118-094036.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang C. Inhibition of GDNF Signaling Reduces Tumorigenic Potential and Metastasis in Glioblastoma Multiforme. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0710118-094036

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

30. Chang, Alexander S. Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line.

Degree: 2010, University of Texas Southwestern Medical Center

 The neurons that make up the suprachiasmatic nucleus (SCN) temporally organize behavior into circadian cycles of activity and rest. When dissociated, these neurons individually oscillate… (more)

Subjects/Keywords: Circadian Rhythm; Neuropeptides; Mice, Transgenic

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chang, A. S. (2010). Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/247

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Alexander S. “Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed January 18, 2021. http://hdl.handle.net/2152.5/247.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Alexander S. “Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line.” 2010. Web. 18 Jan 2021.

Vancouver:

Chang AS. Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2152.5/247.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang AS. Analysis of Circadian Rhythm Using a Novel SCN-Specific Cre Transgenic Mouse Line. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/247

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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