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You searched for subject:(Transforming Growth Factor beta1 pharmacology 60). Showing records 1 – 30 of 36928 total matches.

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1. Moore, Lakisha Dionne. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.

Degree: PhD, 2008, University of Alabama – Birmingham

Overexpresssion of transforming growth factor (TGF)-[beta] has been implicated in promoting immune suppression, tumor angiogenesis, tumor cell migration, and invasion in many cancers including carcinoma… (more)

Subjects/Keywords: Breast Neoplasms  – genetics <; br>; Neoplasm Metastasis <; br>; RNA Interference <; br>; Transforming Growth Factor beta1  – genetics <; br>; Transforming Growth Factor beta1  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moore, L. D. (2008). Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,233

Chicago Manual of Style (16th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,233.

MLA Handbook (7th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Web. 12 Dec 2019.

Vancouver:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233.

Council of Science Editors:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233

2. Roarty, Kevin Patrick. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Breast cancer is the second most common cancer worldwide behind lung cancer, affecting women of all ages, races, ethnicities, and socioeconomic strata. Concerted efforts have… (more)

Subjects/Keywords: Gene Expression Regulation, Developmental <; br>; Mammary Glands, Animal  – growth & development <; br>; Mammary Glands, Animal  – metabolism <; br>; Transforming Growth Factor beta1  – pharmacology <; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Roarty, K. P. (2008). The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,337

Chicago Manual of Style (16th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,337.

MLA Handbook (7th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Web. 12 Dec 2019.

Vancouver:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337.

Council of Science Editors:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337

3. Seo, Hwa-Seon. The role of TGF[beta] signaling in skeletal development.

Degree: PhD, 2008, University of Alabama – Birmingham

Each skeletal element is essential for body movement along with the joints, muscles, tendons and ligaments. They also protect internal organs and serve as a… (more)

Subjects/Keywords: Bone and Bones  – abnormalities <; br>; Homeodomain Proteins  – genetics <; br>; Joints  – abnormalities <; br>; Mesoderm  – embryology <; br>; Protein-Serine-Threonine Kinases  – Metabolism <; br>; Receptors, Transforming Growth Factor beta  – metabolism <; br>; Transforming Growth Factor beta1  – physiology

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APA (6th Edition):

Seo, H. (2008). The role of TGF[beta] signaling in skeletal development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,338

Chicago Manual of Style (16th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,338.

MLA Handbook (7th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Web. 12 Dec 2019.

Vancouver:

Seo H. The role of TGF[beta] signaling in skeletal development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338.

Council of Science Editors:

Seo H. The role of TGF[beta] signaling in skeletal development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338


University of Illinois – Urbana-Champaign

4. Xu, Jing. Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system.

Degree: MS, 0408, 2010, University of Illinois – Urbana-Champaign

 In this work, we explored a thick in vitro 3-D cell culture model with layered structure and applied transforming growth factor-beta1 (TGF-??1) on the top… (more)

Subjects/Keywords: myofibroblasts; transforming growth factor-beta1; alpha-smooth muscle actin; diffusion; confocal microscopy imaging; immunofluorescence staining

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APA (6th Edition):

Xu, J. (2010). Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/16963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Jing. “Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system.” 2010. Thesis, University of Illinois – Urbana-Champaign. Accessed December 12, 2019. http://hdl.handle.net/2142/16963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Jing. “Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system.” 2010. Web. 12 Dec 2019.

Vancouver:

Xu J. Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2010. [cited 2019 Dec 12]. Available from: http://hdl.handle.net/2142/16963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu J. Characterization and modeling of cell activation upon TGF-??1 stimulation in a 3-D culture system. [Thesis]. University of Illinois – Urbana-Champaign; 2010. Available from: http://hdl.handle.net/2142/16963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North-West University

5. Campbell, Berenice. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .

Degree: 2010, North-West University

 Pigmentation disorders occur in multiple conditions (Hakozaki et al., 2006:105). Although many modalities of treatments are available, none are completely satisfactory (Briganti et al., 2003:101).… (more)

Subjects/Keywords: Pigmentation; Topical delivery; Pheroid; Transforming growth factor-beta1; Tumour necrosis factor-alpha; Bestatin; Tape stripping; Pigmentasie; Topikale aflewering; Transformerende groei faktor-beta1; Tumor nekrosis faktor-alpha; Bestatien; Bandstroping

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Campbell, B. (2010). Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/4739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Campbell, Berenice. “Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .” 2010. Thesis, North-West University. Accessed December 12, 2019. http://hdl.handle.net/10394/4739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Campbell, Berenice. “Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .” 2010. Web. 12 Dec 2019.

Vancouver:

Campbell B. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . [Internet] [Thesis]. North-West University; 2010. [cited 2019 Dec 12]. Available from: http://hdl.handle.net/10394/4739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campbell B. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . [Thesis]. North-West University; 2010. Available from: http://hdl.handle.net/10394/4739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McGill University

6. Rafiei, Shahrzad. Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction.

Degree: MS, Division of Experimental Medicine., 2007, McGill University

The survival of breast cancer patients declines when tumors are invasive and have an increased possibility of metastasizing to distal sites. Transforming Growth Factor-beta (TGF-beta)… (more)

Subjects/Keywords: Talin.; Transforming Growth Factor beta1  – antagonists & inhibitors.

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APA (6th Edition):

Rafiei, S. (2007). Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile100203.pdf

Chicago Manual of Style (16th Edition):

Rafiei, Shahrzad. “Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction.” 2007. Masters Thesis, McGill University. Accessed December 12, 2019. http://digitool.library.mcgill.ca/thesisfile100203.pdf.

MLA Handbook (7th Edition):

Rafiei, Shahrzad. “Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction.” 2007. Web. 12 Dec 2019.

Vancouver:

Rafiei S. Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction. [Internet] [Masters thesis]. McGill University; 2007. [cited 2019 Dec 12]. Available from: http://digitool.library.mcgill.ca/thesisfile100203.pdf.

Council of Science Editors:

Rafiei S. Talin : a novel inducible antagonist of transforming growth factor-beta 1 (TGF-[beta]1) signal transduction. [Masters Thesis]. McGill University; 2007. Available from: http://digitool.library.mcgill.ca/thesisfile100203.pdf

7. Rafael Braga Petito. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.

Degree: 2008, Fundação Oswaldo Cruz

Neural fibrosis is one of the causes of deformities during leprosy, which remains a major global health problem. Neural degeneration progresses after the patient healing… (more)

Subjects/Keywords: BIOLOGIA MOLECULAR; Mycobacterium leprae; Fibrose/complicações; Hanseníase; Saúde Pública; Fator Transformador de Crescimento beta1/análise; Mycobacterium leprae; Fibrosis/Complications; Leprosy; Public Health; Transforming Growth Factor beta1/analysis

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APA (6th Edition):

Petito, R. B. (2008). Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. (Thesis). Fundação Oswaldo Cruz. Retrieved from http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petito, Rafael Braga. “Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.” 2008. Thesis, Fundação Oswaldo Cruz. Accessed December 12, 2019. http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petito, Rafael Braga. “Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.” 2008. Web. 12 Dec 2019.

Vancouver:

Petito RB. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. [Internet] [Thesis]. Fundação Oswaldo Cruz; 2008. [cited 2019 Dec 12]. Available from: http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petito RB. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. [Thesis]. Fundação Oswaldo Cruz; 2008. Available from: http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo Health Science Campus

8. Elkareh, Jihad Victor. Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy.

Degree: PhD, College of Graduate Studies, 2008, University of Toledo Health Science Campus

  It has been recognized that patients with chronic renal failure eventually develop diastolic dysfunction, cardiac hypertrophy and systemic oxidant stress along with increases in… (more)

Subjects/Keywords: Pharmacology; Surgery; cardiomyopathy; cardiotonic steroids; renal failure; marinobufagenin; fibrosis; transforming growth factor TGF-&946

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APA (6th Edition):

Elkareh, J. V. (2008). Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1213115467

Chicago Manual of Style (16th Edition):

Elkareh, Jihad Victor. “Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy.” 2008. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed December 12, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1213115467.

MLA Handbook (7th Edition):

Elkareh, Jihad Victor. “Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy.” 2008. Web. 12 Dec 2019.

Vancouver:

Elkareh JV. Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2008. [cited 2019 Dec 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1213115467.

Council of Science Editors:

Elkareh JV. Molecular Mechanism of Fibrosis and Central Role of Cardiotonic Steroids in Uremic Cardiomyopathy. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1213115467

9. Lopes, Roberto Iglesias. Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos.

Degree: PhD, Urologia, 2014, University of São Paulo

Introdução: A alteração dos níveis normais de marcadores séricos e urinários ocorre na presença de dano renal associado à uropatia obstrutiva. Valores séricos e e… (more)

Subjects/Keywords: Antígeno CA 19-9; CA 19-9 antigen; Fator de crescimento transformador beta 1; Obstrução ureteral; Ratos Wistar; Transforming growth factor beta1; Ureter; Ureter; Ureteral obstruction; Wistar rats

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APA (6th Edition):

Lopes, R. I. (2014). Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5153/tde-29042014-101952/ ;

Chicago Manual of Style (16th Edition):

Lopes, Roberto Iglesias. “Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos.” 2014. Doctoral Dissertation, University of São Paulo. Accessed December 12, 2019. http://www.teses.usp.br/teses/disponiveis/5/5153/tde-29042014-101952/ ;.

MLA Handbook (7th Edition):

Lopes, Roberto Iglesias. “Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos.” 2014. Web. 12 Dec 2019.

Vancouver:

Lopes RI. Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2019 Dec 12]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5153/tde-29042014-101952/ ;.

Council of Science Editors:

Lopes RI. Avaliação dos valores séricos e urinários de CA 19-9 e TGFbeta1 na obstrução parcial e completa de ureteres em ratos. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5153/tde-29042014-101952/ ;

10. Gilbert, Rebekah (Rebekah Sims). The role of TGF-β receptors in the induction of mucosal tolerance .

Degree: PhD, 2011, University of Alabama – Birmingham

Oral tolerance, defined as the immunologic state of systemic and mucosal unresponsiveness to an antigen, is an essential function of the mucosal immune system. Although… (more)

Subjects/Keywords: Antigens  – immunology<; br>; Immune Tolerance<; br>; Immunity, Mucosal  – immunology<; br>; Receptors, Transforming Growth Factor beta<; br>; T-Lymphocytes, Regulatory<; br>; Transforming Growth Factor beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gilbert, R. (. S. (2011). The role of TGF-β receptors in the induction of mucosal tolerance . (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,945

Chicago Manual of Style (16th Edition):

Gilbert, Rebekah (Rebekah Sims). “The role of TGF-β receptors in the induction of mucosal tolerance .” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,945.

MLA Handbook (7th Edition):

Gilbert, Rebekah (Rebekah Sims). “The role of TGF-β receptors in the induction of mucosal tolerance .” 2011. Web. 12 Dec 2019.

Vancouver:

Gilbert R(S. The role of TGF-β receptors in the induction of mucosal tolerance . [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,945.

Council of Science Editors:

Gilbert R(S. The role of TGF-β receptors in the induction of mucosal tolerance . [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,945

11. Lucas, Jason Anthony. Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Pressure overload stress (eg. hypertension or aortic stenosis) results in excessive cardiac fibrosis, changes in left ventricular (LV) geometry, and disruption of LV contractility, ultimately… (more)

Subjects/Keywords: Atrial Natriuretic Factor  – metabolism<; br>; Cardiomyopathy, Dilated  – physiopathology<; br>; Heart  – physiopathology<; br>; Signal Transduction  – physiology<; br>; Transforming Growth Factor beta  – antagonists & inhibitors Vasodilation  – physiology<; br>; Ventricular Dysfunction, Left  – physiopathology

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APA (6th Edition):

Lucas, J. A. (2009). Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,570

Chicago Manual of Style (16th Edition):

Lucas, Jason Anthony. “Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,570.

MLA Handbook (7th Edition):

Lucas, Jason Anthony. “Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis.” 2009. Web. 12 Dec 2019.

Vancouver:

Lucas JA. Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,570.

Council of Science Editors:

Lucas JA. Counterregulatory roles of transforming growth factor (TGF)-[beta] and a trial natruiretic peptide (ANP) in pressure overload-induced cardiac remodeling and fibrosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,570


Texas A&M University

12. McMillin, Matthew Alexander. The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis.

Degree: 2013, Texas A&M University

 Hepatic encephalopathy (HE) is a neurological complication that arises due to liver dysfunction and is associated with increased circulating ammonia and a systemic immune response.… (more)

Subjects/Keywords: Hepatic Encephalopathy; Transforming Growth Factor Beta

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APA (6th Edition):

McMillin, M. A. (2013). The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McMillin, Matthew Alexander. “The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis.” 2013. Thesis, Texas A&M University. Accessed December 12, 2019. http://hdl.handle.net/1969.1/151843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McMillin, Matthew Alexander. “The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis.” 2013. Web. 12 Dec 2019.

Vancouver:

McMillin MA. The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2019 Dec 12]. Available from: http://hdl.handle.net/1969.1/151843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McMillin MA. The Role of Transforming Growth Factor Beta in Hepatic Encephalopathy Pathogeneis. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

13. Rhodes, Steven David. Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Skeletal manifestations including short stature, osteoporosis, kyphoscoliosis, and tibial dysplasia cumulatively affect approximately 70% of patients with neurofibromatosis type 1… (more)

Subjects/Keywords: Neurofibromatosis type 1; Bone; Osteoporosis; Pseudarthrosis; Mouse model; Osteoclasts; Transforming growth factor-beta1; Neurofibromatosis  – Research; Neurofibromatosis  – Genetic aspects; Osteoporosis  – Alternative treatment  – Research  – Methodology; Osteoporosis  – Prevention; Pseudarthrosis  – Research  – Analysis; Mice  – Diseases  – Pathophysiology  – Research; Osteoclasts  – Research; Hematopoiesis  – Research; Bone cells  – Research; Bones  – Growth; Spine  – Abnormalities; Nervous system  – Diseases; Bone marrow  – Transplantation  – Research; Transforming growth factors-beta  – Research

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rhodes, S. D. (2014). Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3793

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rhodes, Steven David. “Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects.” 2014. Thesis, IUPUI. Accessed December 12, 2019. http://hdl.handle.net/1805/3793.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rhodes, Steven David. “Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects.” 2014. Web. 12 Dec 2019.

Vancouver:

Rhodes SD. Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects. [Internet] [Thesis]. IUPUI; 2014. [cited 2019 Dec 12]. Available from: http://hdl.handle.net/1805/3793.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rhodes SD. Dissecting the cellular and molecular mechanisms mediating neurofibromatosis type 1 related bone defects. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/3793

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

14. Siraj, Sadia. A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity.

Degree: 2017, University of Western Ontario

 Chronic musculoskeletal pain results in significant personal, economic, and social burden. Early identification and intervention in those people with acute pain that are likely to… (more)

Subjects/Keywords: Chronic pain; acute musculoskeletal trauma; Hypothalamic-Pituitary-Adrenal axis (HPA axis); stress biomarkers; Cortisol; Brain-derived neurotrophic factor (BDNF); Transforming growth factor beta1 (TGFB1); early life adversity.; Physical Therapy; Preventive Medicine; Rehabilitation and Therapy; Trauma

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APA (6th Edition):

Siraj, S. (2017). A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4856

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Siraj, Sadia. “A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity.” 2017. Thesis, University of Western Ontario. Accessed December 12, 2019. https://ir.lib.uwo.ca/etd/4856.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Siraj, Sadia. “A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity.” 2017. Web. 12 Dec 2019.

Vancouver:

Siraj S. A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Dec 12]. Available from: https://ir.lib.uwo.ca/etd/4856.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Siraj S. A cross- sectional study of stress biomarkers and their associations with post-trauma complaints, and how those associations are moderated by early life adversity. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4856

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Tang, Yi, Ph.D. The role of transforming growth factor beta-1 in bone remodeling.

Degree: PhD, 2009, University of Alabama – Birmingham

Bone remodeling depends on the precise coordination of bone resorption by the osteoclasts and bone formation by the osteoblasts. It is proposed that osteoclastic bone… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Bone Regeneration  – drug effects<; br>; Cadherins  – metabolism<; br>; Cell Membrane  – metabolism<; br>; Multiprotein Complexes  – metabolism<; br>; Smad7 Protein  – metabolism<; br>; Transforming Growth Factor beta  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, Yi, P. D. (2009). The role of transforming growth factor beta-1 in bone remodeling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,405

Chicago Manual of Style (16th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,405.

MLA Handbook (7th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Web. 12 Dec 2019.

Vancouver:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405.

Council of Science Editors:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405

16. Katre, Ashwini A. Ozone and lung fibrosis.

Degree: M.S.P.H., 2009, University of Alabama – Birmingham

Ozone is an extremely reactive gas molecule composed of three oxygen atoms. The ground-level ozone is formed when nitrogen oxides and volatile organic compounds, released… (more)

Subjects/Keywords: Actins<; br>; Collagen  – metabolism<; br>; Ozone  – toxicity<; br>; Plasminogen Activator Inhibitor 1<; br>; Pulmonary Fibrosis  – chemically induced<; br>; Pulmonary Fibrosis  – pathology<; br>; Transforming Growth Factor beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Katre, A. A. (2009). Ozone and lung fibrosis. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,485

Chicago Manual of Style (16th Edition):

Katre, Ashwini A. “Ozone and lung fibrosis.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,485.

MLA Handbook (7th Edition):

Katre, Ashwini A. “Ozone and lung fibrosis.” 2009. Web. 12 Dec 2019.

Vancouver:

Katre AA. Ozone and lung fibrosis. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,485.

Council of Science Editors:

Katre AA. Ozone and lung fibrosis. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,485

17. Mukherjee, Sudipto. Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy.

Degree: PhD, 2007, University of Alabama – Birmingham

The goal of this study was to determine the influence, if any, of the insulin-like growth factors (IGFs) on retinal pigment epithelial (RPE) cell tractional… (more)

Subjects/Keywords: Insulin-Like Growth Factor Binding Proteins <; br>; Insulin-Like Growth Factor I  – pharmacology <; br>; Insulin-Like Growth Factor II  – pharmacology <; br>; Pigment Epithelium of Eye  – drug effects

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APA (6th Edition):

Mukherjee, S. (2007). Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,257

Chicago Manual of Style (16th Edition):

Mukherjee, Sudipto. “Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,257.

MLA Handbook (7th Edition):

Mukherjee, Sudipto. “Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy.” 2007. Web. 12 Dec 2019.

Vancouver:

Mukherjee S. Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,257.

Council of Science Editors:

Mukherjee S. Retinal pigment epithelial cells and the insulin-like growth factor system in proliferative vitreoretinopathy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,257


NSYSU

18. Kao, Ying-hsien. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.

Degree: PhD, Biological Sciences, 2009, NSYSU

 Liver fibrosis, a major medical problem with significant morbidity and mortality, is considered as a wound-healing response to a variety of chronic stimuli. It is… (more)

Subjects/Keywords: hepatoma-derived growth factor; liver fibrosis; hepatocytes; transforming growth factor-beta

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APA (6th Edition):

Kao, Y. (2009). Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053

Chicago Manual of Style (16th Edition):

Kao, Ying-hsien. “Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.” 2009. Doctoral Dissertation, NSYSU. Accessed December 12, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053.

MLA Handbook (7th Edition):

Kao, Ying-hsien. “Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.” 2009. Web. 12 Dec 2019.

Vancouver:

Kao Y. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. [Internet] [Doctoral dissertation]. NSYSU; 2009. [cited 2019 Dec 12]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053.

Council of Science Editors:

Kao Y. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. [Doctoral Dissertation]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053

19. Maynard, Craig Lueland. IL-10-competent regulatory T cells: development, phenotype and function.

Degree: PhD, 2007, University of Alabama – Birmingham

In order to avoid chronic cell activation and inflammation the immune system has developed numerous mechanisms that cooperate to induce and/or maintain what is known… (more)

Subjects/Keywords: Cell Differentiation  – immunology<; br>; Forkhead Transcription Factors  – immunology<; br>; Inflammation  – immunology<; br>; Interleukin-10  – metabolism<; br>; T-Lymphocyte Subsets  – cytology<; br>; Transforming Growth Factor beta

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APA (6th Edition):

Maynard, C. L. (2007). IL-10-competent regulatory T cells: development, phenotype and function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,529

Chicago Manual of Style (16th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,529.

MLA Handbook (7th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Web. 12 Dec 2019.

Vancouver:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529.

Council of Science Editors:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529

20. Lee, Yun Kyung. Late developmental plasticity in the T helper 17 lineage.

Degree: PhD, 2009, University of Alabama – Birmingham

CD4 T cells are a principal component of the adaptive immune system that is required for the efficient elimination of foreign antigens, but dysregulated CD4… (more)

Subjects/Keywords: Cell Lineage  – immunology<; br>; Interleukin-17  – metabolism<; br>; T-Lymphocyte Subsets  – immunology<; br>; Th1 Cells  – immunology<; br>; Th2 Cells  – immunology<; br>; Transforming Growth Factor beta  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, Y. K. (2009). Late developmental plasticity in the T helper 17 lineage. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,916

Chicago Manual of Style (16th Edition):

Lee, Yun Kyung. “Late developmental plasticity in the T helper 17 lineage.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,916.

MLA Handbook (7th Edition):

Lee, Yun Kyung. “Late developmental plasticity in the T helper 17 lineage.” 2009. Web. 12 Dec 2019.

Vancouver:

Lee YK. Late developmental plasticity in the T helper 17 lineage. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,916.

Council of Science Editors:

Lee YK. Late developmental plasticity in the T helper 17 lineage. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,916

21. 윤, 영실. Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구.

Degree: 2005, Ajou University

"목적: 모든 정상 세포는 제한된 수의 분열을 거친 후 더 이상 분열하지 않는 “replicative senescence”라는 노화과정을 겪게 된다. 세포노화의 원인으로 “free radical theory”가 잘 알려져… (more)

Subjects/Keywords: Transforming Growth Factor beta1 (TGF β1); senescence-like growth arrest; mitochondrial dysfunction; Reactive Oxygen Species (ROS); cellular respiration; Hepatocyte Growth Factor (HGF)

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APA (6th Edition):

윤, . (2005). Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/2293 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

윤, 영실. “Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구.” 2005. Thesis, Ajou University. Accessed December 12, 2019. http://repository.ajou.ac.kr/handle/201003/2293 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

윤, 영실. “Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구.” 2005. Web. 12 Dec 2019.

Vancouver:

윤 . Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구. [Internet] [Thesis]. Ajou University; 2005. [cited 2019 Dec 12]. Available from: http://repository.ajou.ac.kr/handle/201003/2293 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

윤 . Transforming Growth Factor β1(TGF-β1)에 의해 유도된 세포노화 과정에서 미토콘드리아 기능의 중요성 연구. [Thesis]. Ajou University; 2005. Available from: http://repository.ajou.ac.kr/handle/201003/2293 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

22. Grégoire, Catherine-Alexandra. Mechanisms underlying activation of neural stem cells in the adult central nervous system .

Degree: 2016, Université de Montréal

 À la fin du 19e siècle, Dr. Ramón y Cajal, un pionnier scientifique, a découvert les éléments cellulaires individuels, appelés neurones, composant le système nerveux.… (more)

Subjects/Keywords: cellules souches neurales; neurogenèse adulte; hippocampe; moelle épinière; environnement enrichi; exercice; comportement; neuroinflammation; transforming growth factor-beta1; cellules initiatrices de neurosphères; neural stem cells; adult neurogenesis; hippocampus; spinal cord; environmental enrichment; exercise; behaviour; neurosphere-initiating cells

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APA (6th Edition):

Grégoire, C. (2016). Mechanisms underlying activation of neural stem cells in the adult central nervous system . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grégoire, Catherine-Alexandra. “Mechanisms underlying activation of neural stem cells in the adult central nervous system .” 2016. Thesis, Université de Montréal. Accessed December 12, 2019. http://hdl.handle.net/1866/15972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grégoire, Catherine-Alexandra. “Mechanisms underlying activation of neural stem cells in the adult central nervous system .” 2016. Web. 12 Dec 2019.

Vancouver:

Grégoire C. Mechanisms underlying activation of neural stem cells in the adult central nervous system . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2019 Dec 12]. Available from: http://hdl.handle.net/1866/15972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grégoire C. Mechanisms underlying activation of neural stem cells in the adult central nervous system . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/15972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Tanner, Scott. Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis.

Degree: 2012, University of Alabama – Birmingham

Regulatory T cells (Treg) are necessary for the maintenance of immune homeostasis, and have been implicated in several autoimmune diseases, including inflammatory bowel disease (IBD).… (more)

Subjects/Keywords: Colitis – immunology.<; br>; Intestines – immunology<; br>; P-Glycoproteins<; br>; T-Lymphocytes, Regulatory – immunology<; br>; Transforming Growth Factor beta – pharmacology

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APA (6th Edition):

Tanner, S. (2012). Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tanner, Scott. “Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis.” 2012. Thesis, University of Alabama – Birmingham. Accessed December 12, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tanner, Scott. “Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis.” 2012. Web. 12 Dec 2019.

Vancouver:

Tanner S. Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 12]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tanner S. Regulatory T Cells In The Fvb.mdr1a-/- Model Of Colitis. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. 정, 수령. hepatocyte growth factor inhibits glial scar formation and.

Degree: 2009, Ajou University

Formation of glial scars physically impedes growth of regenerating axons following CNS injuries such as spinal cord trauma. Glial scars also produce various species of… (more)

Subjects/Keywords: spinal cord injury; hepatocyte growth factor; glial scars; chondroitin sulfate proteoglycans; transforming growth factor

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APA (6th Edition):

정, . (2009). hepatocyte growth factor inhibits glial scar formation and. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/2272 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 수령. “hepatocyte growth factor inhibits glial scar formation and.” 2009. Thesis, Ajou University. Accessed December 12, 2019. http://repository.ajou.ac.kr/handle/201003/2272 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 수령. “hepatocyte growth factor inhibits glial scar formation and.” 2009. Web. 12 Dec 2019.

Vancouver:

정 . hepatocyte growth factor inhibits glial scar formation and. [Internet] [Thesis]. Ajou University; 2009. [cited 2019 Dec 12]. Available from: http://repository.ajou.ac.kr/handle/201003/2272 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . hepatocyte growth factor inhibits glial scar formation and. [Thesis]. Ajou University; 2009. Available from: http://repository.ajou.ac.kr/handle/201003/2272 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida International University

25. Stewart, Tiffanie S. Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population.

Degree: PhD, Dietetics and Nutrition, 2016, Florida International University

  Liver disease is now a leading cause of non-AIDS related morbidity and mortality in people living with HIV (PLWH). The present study investigated the… (more)

Subjects/Keywords: HIV infection; HIV/HCV co-infection; liver fibrosis; alcohol; AUDIT; cocaine; body mass index; FIB-4 index; oxidative stress; hepatocyte apoptosis; transforming growth factor-beta1; microbial endotoxin; malondialdehyde; glutathione; MDA; GSH; TGF-β1; CK-18; LPS; Biochemical Phenomena, Metabolism, and Nutrition; Community Health and Preventive Medicine; Dietetics and Clinical Nutrition; Hepatology; Medical Biochemistry; Medical Immunology; Medical Nutrition; Other Medical Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stewart, T. S. (2016). Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population. (Doctoral Dissertation). Florida International University. Retrieved from http://digitalcommons.fiu.edu/etd/2459 ; 10.25148/etd.FIDC000286 ; FIDC000286

Chicago Manual of Style (16th Edition):

Stewart, Tiffanie S. “Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population.” 2016. Doctoral Dissertation, Florida International University. Accessed December 12, 2019. http://digitalcommons.fiu.edu/etd/2459 ; 10.25148/etd.FIDC000286 ; FIDC000286.

MLA Handbook (7th Edition):

Stewart, Tiffanie S. “Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population.” 2016. Web. 12 Dec 2019.

Vancouver:

Stewart TS. Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population. [Internet] [Doctoral dissertation]. Florida International University; 2016. [cited 2019 Dec 12]. Available from: http://digitalcommons.fiu.edu/etd/2459 ; 10.25148/etd.FIDC000286 ; FIDC000286.

Council of Science Editors:

Stewart TS. Lifestyle and Biological Risk Factors for Liver Fibrosis in the Miami Adult Studies on HIV (MASH) Cohort: An HIV Infected and HIV/HCV Co-infected Population. [Doctoral Dissertation]. Florida International University; 2016. Available from: http://digitalcommons.fiu.edu/etd/2459 ; 10.25148/etd.FIDC000286 ; FIDC000286

26. Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Transforming growth factor-β1 (TGF-β1) reportedly causes the differentiation of fibroblasts to myofibroblasts during wound healing. We investigated the mechanism underlying the activation of latent TGF-β1… (more)

Subjects/Keywords: keratinocyte; fibroblast; transforming growth factor-β1; αv-integrin; matrix metalloproteinase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, H. (2014). Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed December 12, 2019. http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Web. 12 Dec 2019.

Vancouver:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2019 Dec 12]. Available from: http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

27. Wu, Wei-Hao. The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats.

Degree: Master, Marine Biotechnology and Resources, 2013, NSYSU

 Diabetes mellitus is a metabolic disease that can have long-term complications, including diabetic neuropathy pain. Base on preliminary screening, we had found that a marine-derived… (more)

Subjects/Keywords: neuroinflammation; immunohistochemistry; transforming growth factor-β; antinociceptive effects

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APA (6th Edition):

Wu, W. (2013). The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816113-115809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Wei-Hao. “The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats.” 2013. Thesis, NSYSU. Accessed December 12, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816113-115809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Wei-Hao. “The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats.” 2013. Web. 12 Dec 2019.

Vancouver:

Wu W. The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats. [Internet] [Thesis]. NSYSU; 2013. [cited 2019 Dec 12]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816113-115809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu W. The anti-neuroinflammatory effects of a synthetic marine-derived compound on STZ induced diabetic rats. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816113-115809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

28. Chen, Zhi-Cheng. The effects of skeleton extract obtained from marine organisms on wound healing.

Degree: PhD, Marine Biotechnology and Resources, 2017, NSYSU

 Shijueming (SJM) is a traditional Chinese medicine. Records from 1757, in the Qing dynasty, detail the use of SJM for treating skin injuries, particularly poorly… (more)

Subjects/Keywords: wound healing; Shijueming; os sepiae; macrophage; transforming growth factor-beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Z. (2017). The effects of skeleton extract obtained from marine organisms on wound healing. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0120117-104748

Chicago Manual of Style (16th Edition):

Chen, Zhi-Cheng. “The effects of skeleton extract obtained from marine organisms on wound healing.” 2017. Doctoral Dissertation, NSYSU. Accessed December 12, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0120117-104748.

MLA Handbook (7th Edition):

Chen, Zhi-Cheng. “The effects of skeleton extract obtained from marine organisms on wound healing.” 2017. Web. 12 Dec 2019.

Vancouver:

Chen Z. The effects of skeleton extract obtained from marine organisms on wound healing. [Internet] [Doctoral dissertation]. NSYSU; 2017. [cited 2019 Dec 12]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0120117-104748.

Council of Science Editors:

Chen Z. The effects of skeleton extract obtained from marine organisms on wound healing. [Doctoral Dissertation]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0120117-104748


Ruhr Universität Bochum

29. Beck, Lars Hendrik. Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen.

Degree: 2009, Ruhr Universität Bochum

 Problem: Um detailliertere Einblicke in die regelrechte Entwicklung regulatorisch bedeutsamer Immunkomponenten gewinnen zu können, wurde hier erstmals die Frequenz und der Charakter TGF-β-produzierender T-Lymphozyten gesunder… (more)

Subjects/Keywords: Transforming Growth Factor beta; T-Lymphozyt; Helferzelle; Immunologie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beck, L. H. (2009). Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-25629

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Beck, Lars Hendrik. “Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen.” 2009. Thesis, Ruhr Universität Bochum. Accessed December 12, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-25629.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Beck, Lars Hendrik. “Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen.” 2009. Web. 12 Dec 2019.

Vancouver:

Beck LH. Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen. [Internet] [Thesis]. Ruhr Universität Bochum; 2009. [cited 2019 Dec 12]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-25629.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Beck LH. Bestimmung der Frequenz TGF-β-produzierender T-Lymphozyten gesunder Kinder verschiedener Altersklassen. [Thesis]. Ruhr Universität Bochum; 2009. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-25629

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ruhr Universität Bochum

30. Pennartz, Christian. Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor.

Degree: 2009, Ruhr Universität Bochum

 Chronisch entzündliche Darmerkrankungen sind geprägt durch destruierende Entzündungs-reaktionen der Darmschleimhaut. Bei intakter Basalmembran werden kleinere Epitheldefekte mittels der Epithelzellrestitution unabhängig von der Proliferation repariert. Ziel… (more)

Subjects/Keywords: Migration (Biologie); Proliferation; Transforming Growth Factor beta; Chronische Darmentzündung

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APA (6th Edition):

Pennartz, C. (2009). Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-27034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pennartz, Christian. “Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor.” 2009. Thesis, Ruhr Universität Bochum. Accessed December 12, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-27034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pennartz, Christian. “Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor.” 2009. Web. 12 Dec 2019.

Vancouver:

Pennartz C. Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor. [Internet] [Thesis]. Ruhr Universität Bochum; 2009. [cited 2019 Dec 12]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-27034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pennartz C. Untersuchungen zur Modulation der intestinalen Epithelrestitution in vitro durch Vascular endothelian growth factor. [Thesis]. Ruhr Universität Bochum; 2009. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-27034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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