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University: University of Kentucky

You searched for subject:(Toxicology). Showing records 1 – 30 of 52 total matches.

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University of Kentucky

1. Li, Jieran. IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER.

Degree: 2018, University of Kentucky

 Large quantities of manufactured nanomaterials (MNM) are released into the environment by human activity each year. The entry of MNM into the terrestrial food webs,… (more)

Subjects/Keywords: Nanotechnology; Plant; Environment; Agriculture; Toxicology; Insect; Toxicology

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APA (6th Edition):

Li, J. (2018). IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/24

Chicago Manual of Style (16th Edition):

Li, Jieran. “IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER.” 2018. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/24.

MLA Handbook (7th Edition):

Li, Jieran. “IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER.” 2018. Web. 23 Jul 2019.

Vancouver:

Li J. IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/24.

Council of Science Editors:

Li J. IMPACT OF PHYSICO-CHEMICAL PROPERTIES OF MANUFACTURED NANOMATERIALS ON PLANT UPTAKE AND TROPHIC TRANSFER. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/toxicology_etds/24


University of Kentucky

2. Chan, Nelson Lap Shun. IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY.

Degree: 2011, University of Kentucky

 CAG/CTG repeat instability is associated with at least 14 neurological disorders, including Huntington’s disease and Myotonic dystrophy type 1. In vitro and in vivo studies… (more)

Subjects/Keywords: CTG; Repeat Instability; Hairpin; Helicase; Polymerase; Medical Toxicology; Toxicology

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APA (6th Edition):

Chan, N. L. S. (2011). IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/832

Chicago Manual of Style (16th Edition):

Chan, Nelson Lap Shun. “IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY.” 2011. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/832.

MLA Handbook (7th Edition):

Chan, Nelson Lap Shun. “IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY.” 2011. Web. 23 Jul 2019.

Vancouver:

Chan NLS. IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY. [Internet] [Doctoral dissertation]. University of Kentucky; 2011. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/832.

Council of Science Editors:

Chan NLS. IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY. [Doctoral Dissertation]. University of Kentucky; 2011. Available from: https://uknowledge.uky.edu/gradschool_diss/832


University of Kentucky

3. Petriello, Michael C. ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY.

Degree: 2015, University of Kentucky

 Cardiovascular disease is the leading cause of mortality in Western societies and is linked to multiple modifiable risk factors including lifestyle choices. Emerging evidence implicates… (more)

Subjects/Keywords: Cardiovascular Disease; PCBs; Nrf2; Caveolae; Nutrition; Nitro-fatty acids; Environmental Health; Life Sciences; Pharmacology, Toxicology and Environmental Health; Toxicology

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APA (6th Edition):

Petriello, M. C. (2015). ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/11

Chicago Manual of Style (16th Edition):

Petriello, Michael C. “ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY.” 2015. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/11.

MLA Handbook (7th Edition):

Petriello, Michael C. “ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY.” 2015. Web. 23 Jul 2019.

Vancouver:

Petriello MC. ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY. [Internet] [Doctoral dissertation]. University of Kentucky; 2015. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/11.

Council of Science Editors:

Petriello MC. ROLE OF CAVEOLIN-1 AND NRF2 IN NUTRITIONAL MODULATION OF PCB TOXICITY. [Doctoral Dissertation]. University of Kentucky; 2015. Available from: https://uknowledge.uky.edu/toxicology_etds/11


University of Kentucky

4. Jung, Kyung Sik. EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS.

Degree: 2013, University of Kentucky

 A previous thoracic aortic aneurysm time course study from our laboratory determined that ascending aortic dilation was significantly increased by day 5, and reached a… (more)

Subjects/Keywords: Ascending Aortic Aneurysms; Hemorrhage; Angiotensin II; Neutrophil; Cellular Heterogeneity; Medical Toxicology

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APA (6th Edition):

Jung, K. S. (2013). EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/toxicology_etds/6

Chicago Manual of Style (16th Edition):

Jung, Kyung Sik. “EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS.” 2013. Masters Thesis, University of Kentucky. Accessed July 23, 2019. http://uknowledge.uky.edu/toxicology_etds/6.

MLA Handbook (7th Edition):

Jung, Kyung Sik. “EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS.” 2013. Web. 23 Jul 2019.

Vancouver:

Jung KS. EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS. [Internet] [Masters thesis]. University of Kentucky; 2013. [cited 2019 Jul 23]. Available from: http://uknowledge.uky.edu/toxicology_etds/6.

Council of Science Editors:

Jung KS. EFFECTS OF CELLULAR HETEROGENEITY AND IMMUNE CELLS IN ANGIOTENSIN II-INFUSED HEMORRHAGED ASCENDING AORTAS. [Masters Thesis]. University of Kentucky; 2013. Available from: http://uknowledge.uky.edu/toxicology_etds/6


University of Kentucky

5. Kudupoje, Manoj B. MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY.

Degree: 2017, University of Kentucky

 Alkaloid toxicities negatively impact livestock health and production and are of serious economic concern to animal industries. To date, few strategies have been developed to… (more)

Subjects/Keywords: ergotamine; adsorbent; myograph; molecularly imprinted; isotherms; fescue; Animal Sciences; Biotechnology; Toxicology

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APA (6th Edition):

Kudupoje, M. B. (2017). MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/animalsci_etds/76

Chicago Manual of Style (16th Edition):

Kudupoje, Manoj B. “MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY.” 2017. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/animalsci_etds/76.

MLA Handbook (7th Edition):

Kudupoje, Manoj B. “MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY.” 2017. Web. 23 Jul 2019.

Vancouver:

Kudupoje MB. MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY. [Internet] [Doctoral dissertation]. University of Kentucky; 2017. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/animalsci_etds/76.

Council of Science Editors:

Kudupoje MB. MOLECULARLY IMPRINTED POLYMERS SYNTHESIZED AS ADSORBENTS FOR ERGOT ALKALOIDS: CHARACTERIZATION AND IN VITRO AND EX VIVO ASSESSMENT OF EFFECTS ON ERGOT ALKALOID BIOAVAILABILITY. [Doctoral Dissertation]. University of Kentucky; 2017. Available from: https://uknowledge.uky.edu/animalsci_etds/76


University of Kentucky

6. Lozada Santiago, Enerlyn Meliza. GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION.

Degree: 2011, University of Kentucky

 Loss of function of the WRN protein causes the genetic disorder Werner Syndrome that is characterized by increased cancer and premature aging. WRN belongs to… (more)

Subjects/Keywords: Werner Protein; Acetylation; Genotoxins; DNA damage; Replication; Medical Biochemistry; Toxicology

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APA (6th Edition):

Lozada Santiago, E. M. (2011). GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/817

Chicago Manual of Style (16th Edition):

Lozada Santiago, Enerlyn Meliza. “GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION.” 2011. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/817.

MLA Handbook (7th Edition):

Lozada Santiago, Enerlyn Meliza. “GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION.” 2011. Web. 23 Jul 2019.

Vancouver:

Lozada Santiago EM. GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION. [Internet] [Doctoral dissertation]. University of Kentucky; 2011. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/817.

Council of Science Editors:

Lozada Santiago EM. GENOTOXIN-INDUCED ACETYLATION OF THE WERNER SYNDROME PROTEIN (WRN) AND EFFECT ON ITS DNA METABOLIC FUNCTION. [Doctoral Dissertation]. University of Kentucky; 2011. Available from: https://uknowledge.uky.edu/gradschool_diss/817


University of Kentucky

7. Sun, Yulan. THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE.

Degree: 2010, University of Kentucky

 Parthenolide (PN), a major active component of the traditional herbal medicine feverfew, has been shown to have anti-inflammatory and anti-tumor properties. More remarkably, the cytotoxicity… (more)

Subjects/Keywords: Parthenolide|radiation|prostate cancer|oxidative stress|redox signaling; Medical Toxicology

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APA (6th Edition):

Sun, Y. (2010). THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/768

Chicago Manual of Style (16th Edition):

Sun, Yulan. “THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE.” 2010. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/768.

MLA Handbook (7th Edition):

Sun, Yulan. “THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE.” 2010. Web. 23 Jul 2019.

Vancouver:

Sun Y. THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/768.

Council of Science Editors:

Sun Y. THE RADIOSENSITIZATION EFFECT OF PARTHENOLIDE IN PROSTATE CANCER: IMPLICATIONS FOR SELECTIVE CANCER KILLING BY MODULATION OF INTRACELLULAR REDOX STATE. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/768


University of Kentucky

8. Zhang, Wei. LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE.

Degree: 2015, University of Kentucky

 Doxorubicin (DOX) is a broad-spectrum and effective chemotherapeutic agent, but its use in oncologic practice is limited by dose-dependent cumulative cardiotoxicity. DOX-induced cardiotoxicity is in… (more)

Subjects/Keywords: Mrp1; doxorubicin; cardiotoxicity; glutathione; GS-HNE; Medical Toxicology

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APA (6th Edition):

Zhang, W. (2015). LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/12

Chicago Manual of Style (16th Edition):

Zhang, Wei. “LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE.” 2015. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/12.

MLA Handbook (7th Edition):

Zhang, Wei. “LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE.” 2015. Web. 23 Jul 2019.

Vancouver:

Zhang W. LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kentucky; 2015. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/12.

Council of Science Editors:

Zhang W. LOSS OF MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 1 (MRP1/ABCC1) POTENTIATES DOXORUBICIN-INDUCED CARDIOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kentucky; 2015. Available from: https://uknowledge.uky.edu/toxicology_etds/12


University of Kentucky

9. Hitron, John Andrew. AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY.

Degree: 2018, University of Kentucky

 The reversible oxidation of cysteine by reactive oxygen species (ROS) is both a mechanism for cellular protein signaling as well as a cause of cellular… (more)

Subjects/Keywords: Resin-Assisted Capture; Reversible Cysteine Oxidation; Immobilized Reductants; Cysteine Redox Proteomics; Heavy Metals; Cell Biology; Medical Cell Biology; Medical Toxicology; Research Methods in Life Sciences; Toxicology

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APA (6th Edition):

Hitron, J. A. (2018). AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/22

Chicago Manual of Style (16th Edition):

Hitron, John Andrew. “AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY.” 2018. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/22.

MLA Handbook (7th Edition):

Hitron, John Andrew. “AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY.” 2018. Web. 23 Jul 2019.

Vancouver:

Hitron JA. AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/22.

Council of Science Editors:

Hitron JA. AN OPTIMIZED SOLID-PHASE REDUCTION AND CAPTURE STRATEGY FOR THE STUDY OF REVERSIBLY-OXIDIZED CYSTEINES AND ITS APPLICATION TO METAL TOXICITY. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/toxicology_etds/22


University of Kentucky

10. Tian, Lei. BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ.

Degree: 2010, University of Kentucky

 The integrity of an organism's genome depends on the fidelity of DNA replication and the efficiency of DNA repair. The DNA mismatch repair (MMR) system,… (more)

Subjects/Keywords: Genome instability; mismatch repair; MutSα; MutSβ; Trinucleotide repeat; Biochemistry; Chemistry; Medical Toxicology

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APA (6th Edition):

Tian, L. (2010). BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/43

Chicago Manual of Style (16th Edition):

Tian, Lei. “BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ.” 2010. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/43.

MLA Handbook (7th Edition):

Tian, Lei. “BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ.” 2010. Web. 23 Jul 2019.

Vancouver:

Tian L. BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/43.

Council of Science Editors:

Tian L. BIOCHEMICAL CHARACTERIZATION OF HUMAN MISMATCH RECOGNITION PROTEINS MUTSα AND MUTSβ. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/43


University of Kentucky

11. Stults, Dawn Michelle. STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS.

Degree: 2010, University of Kentucky

 The human ribosomal RNA genes are critically important for cell metabolism and viability. They code for the catalytic RNAs which, encased in a housing of… (more)

Subjects/Keywords: Non‐allelic homologous recombination (NAHR); genomic instability; cancer; DNA repair; ribosomal RNA; Medical Toxicology

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APA (6th Edition):

Stults, D. M. (2010). STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/68

Chicago Manual of Style (16th Edition):

Stults, Dawn Michelle. “STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS.” 2010. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/68.

MLA Handbook (7th Edition):

Stults, Dawn Michelle. “STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS.” 2010. Web. 23 Jul 2019.

Vancouver:

Stults DM. STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/68.

Council of Science Editors:

Stults DM. STRUCTURAL INSTABILITY OF HUMAN RIBOSOMAL RNA GENE CLUSTERS. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/68


University of Kentucky

12. Zhu, Menglei. FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING.

Degree: 2010, University of Kentucky

 Prostate cancer is the most frequently diagnosed non-skin cancer and the third leading cause of cancer mortality among men in the US. Androgens are functionally… (more)

Subjects/Keywords: prostate cancer; androgen receptor; EMT; taxol; chemotherapy; Medical Toxicology; Medicine and Health Sciences

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APA (6th Edition):

Zhu, M. (2010). FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/109

Chicago Manual of Style (16th Edition):

Zhu, Menglei. “FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING.” 2010. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/109.

MLA Handbook (7th Edition):

Zhu, Menglei. “FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING.” 2010. Web. 23 Jul 2019.

Vancouver:

Zhu M. FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/109.

Council of Science Editors:

Zhu M. FUNCTION OF ANDROGEN RECEPTOR IN PROSTATE CANCER EPITHELIAL MESENCHYMAL TRANSITION AND MICROTUBULE TARGETING. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/109


University of Kentucky

13. Wamucho, Anye. MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS.

Degree: 2019, University of Kentucky

 There has been an increase in the incorporation of silver nanomaterials into consumer products due to their antimicrobial properties. Therefore there is potential for silver… (more)

Subjects/Keywords: DNA methylation; Histone methylation; Multigenerational toxicity; Mutation; Nematode; Silver Nanomaterials; Genetics; Genomics; Nanotechnology; Toxicology

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APA (6th Edition):

Wamucho, A. (2019). MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/26

Chicago Manual of Style (16th Edition):

Wamucho, Anye. “MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS.” 2019. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/26.

MLA Handbook (7th Edition):

Wamucho, Anye. “MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS.” 2019. Web. 23 Jul 2019.

Vancouver:

Wamucho A. MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS. [Internet] [Doctoral dissertation]. University of Kentucky; 2019. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/26.

Council of Science Editors:

Wamucho A. MULTIGENERATIONAL GENOMIC AND EPIGENETIC EFFECTS OF MANUFACTURED SILVER NANOMATERIALS IN CAENORHABDITIS ELEGANS. [Doctoral Dissertation]. University of Kentucky; 2019. Available from: https://uknowledge.uky.edu/toxicology_etds/26


University of Kentucky

14. Holcomb, Nathaniel C. NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY.

Degree: 2017, University of Kentucky

 Lung cancer is a particularly devastating disease, accounting for the most deaths among all cancer types in the United States. Despite a reduction in the… (more)

Subjects/Keywords: Nucleotide Excision Repair; Cigarette Smoking; Arsenic; Lung Cancer; Appalachian Kentucky; Medical Toxicology

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APA (6th Edition):

Holcomb, N. C. (2017). NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/17

Chicago Manual of Style (16th Edition):

Holcomb, Nathaniel C. “NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY.” 2017. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/17.

MLA Handbook (7th Edition):

Holcomb, Nathaniel C. “NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY.” 2017. Web. 23 Jul 2019.

Vancouver:

Holcomb NC. NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY. [Internet] [Doctoral dissertation]. University of Kentucky; 2017. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/17.

Council of Science Editors:

Holcomb NC. NUCLEOTIDE EXCISION REPAIR: IMPACTS OF ENVIRONMENTAL CARCINOGENS AND ITS ROLE IN CANCER SUSCEPTIBILITY IN APPALACHIAN KENTUCKY. [Doctoral Dissertation]. University of Kentucky; 2017. Available from: https://uknowledge.uky.edu/toxicology_etds/17


University of Kentucky

15. Zhang, Xuan. ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS.

Degree: 2011, University of Kentucky

 The purpose of this study was to determine whether androgen promotes AT1aR expression on smooth muscle to confer high prevalence of AngII-induced AAAs in hyperlipidemic… (more)

Subjects/Keywords: Androgen; Angiotensin receptor; Abdominal aortic aneurysm; Sexual dimorphism; Smooth muscle; Medical Pathology; Medical Toxicology

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APA (6th Edition):

Zhang, X. (2011). ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/140

Chicago Manual of Style (16th Edition):

Zhang, Xuan. “ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS.” 2011. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/140.

MLA Handbook (7th Edition):

Zhang, Xuan. “ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS.” 2011. Web. 23 Jul 2019.

Vancouver:

Zhang X. ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS. [Internet] [Doctoral dissertation]. University of Kentucky; 2011. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/140.

Council of Science Editors:

Zhang X. ANDROGEN INCREASES ANGIOTENSIN RECEPTOR TYPE 1A ON SMOOTH MUSCLE CELLS TO PROMOTE ANGIOTENSIN II-INDUCED ABDOMINAL AORTIC ANEURYSMS. [Doctoral Dissertation]. University of Kentucky; 2011. Available from: https://uknowledge.uky.edu/gradschool_diss/140


University of Kentucky

16. Oostveen, Emily Kay. THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS.

Degree: 2014, University of Kentucky

 Manufactured CeO2 nanomaterials (CeO2-MNMs) are used for a wide variety of applications including diesel fuel additives and chemical/mechanical planarization media. To test the effects of… (more)

Subjects/Keywords: ceria nanoparticle; nanotoxicology; ecotoxicology; model organism; redox chemistry; Agriculture; Biochemistry; Environmental Health; Environmental Microbiology and Microbial Ecology; Toxicology

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APA (6th Edition):

Oostveen, E. K. (2014). THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/pss_etds/46

Chicago Manual of Style (16th Edition):

Oostveen, Emily Kay. “THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS.” 2014. Masters Thesis, University of Kentucky. Accessed July 23, 2019. http://uknowledge.uky.edu/pss_etds/46.

MLA Handbook (7th Edition):

Oostveen, Emily Kay. “THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS.” 2014. Web. 23 Jul 2019.

Vancouver:

Oostveen EK. THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS. [Internet] [Masters thesis]. University of Kentucky; 2014. [cited 2019 Jul 23]. Available from: http://uknowledge.uky.edu/pss_etds/46.

Council of Science Editors:

Oostveen EK. THE ROLE OF SURFACE CHEMISTRY IN THE TOXICITY OF MANUFACTURED CERIUM DIOXIDE NANOMATERIALS TO CAENORHABDITIS ELEGANS. [Masters Thesis]. University of Kentucky; 2014. Available from: http://uknowledge.uky.edu/pss_etds/46


University of Kentucky

17. Majkova, Zuzana. POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS.

Degree: 2010, University of Kentucky

 Cardiovascular diseases are the number one cause of death in Western societies. Endothelial dysfunction is an early event in the pathology of atherosclerosis, which is… (more)

Subjects/Keywords: Endothelium; monocyte chemoattractant protein-1; cardiovascular disease; polychlorinated biphenyls; docosahexaenoic acid; Nutrition; Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Majkova, Z. (2010). POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/24

Chicago Manual of Style (16th Edition):

Majkova, Zuzana. “POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS.” 2010. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/24.

MLA Handbook (7th Edition):

Majkova, Zuzana. “POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS.” 2010. Web. 23 Jul 2019.

Vancouver:

Majkova Z. POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/24.

Council of Science Editors:

Majkova Z. POLYCHLORINATED BIPHENYL-INDUCED ENDOTHELIAL CELL DYSFUNCTION AND ITS MODULATION BY DIETARY LIPIDS. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/24


University of Kentucky

18. Ding, Kai. DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES.

Degree: 2018, University of Kentucky

 As the principal pro-inflammatory prostanoid, prostaglandin E2 (PGE2) serves as mediator of pain and fever in inflammatory reactions. The biosynthesis of PGE2 starts from arachidonic… (more)

Subjects/Keywords: anti-inflammatory drugs; mPGES-1 inhibitors; selectivity; isatin derivatives; benzylidenebarbituric acid; carrageenan air-pouch; Chemicals and Drugs; Organic Chemicals; Pharmacology; Toxicology

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APA (6th Edition):

Ding, K. (2018). DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/chemistry_etds/102

Chicago Manual of Style (16th Edition):

Ding, Kai. “DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES.” 2018. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/chemistry_etds/102.

MLA Handbook (7th Edition):

Ding, Kai. “DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES.” 2018. Web. 23 Jul 2019.

Vancouver:

Ding K. DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/chemistry_etds/102.

Council of Science Editors:

Ding K. DESIGN, SYNTHESIS AND BIOLOGICAL EVALUATION OF INHIBITORS AGAINST BOTH HUMAN AND MOUSE MICROSOMAL PROSTAGLANDIN E2 SYNTHASE-1 ENZYMES. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/chemistry_etds/102


University of Kentucky

19. Hoffman, Jessie Baldwin. PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION.

Degree: 2018, University of Kentucky

 Exposure to environmental pollutants poses numerous risk factors for human health, including increasing incidence of cardiovascular disease and diabetes. Persistent organic pollutants, such as polychlorinated… (more)

Subjects/Keywords: Gut Microbiota; Cardiometabolic disease; PCBs; Inulin; Prebiotic; Nutrition; Environmental Health; Microbiology; Molecular, Genetic, and Biochemical Nutrition; Toxicology

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APA (6th Edition):

Hoffman, J. B. (2018). PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/25

Chicago Manual of Style (16th Edition):

Hoffman, Jessie Baldwin. “PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION.” 2018. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pharmacol_etds/25.

MLA Handbook (7th Edition):

Hoffman, Jessie Baldwin. “PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION.” 2018. Web. 23 Jul 2019.

Vancouver:

Hoffman JB. PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pharmacol_etds/25.

Council of Science Editors:

Hoffman JB. PCB DISRUPTION OF GUT AND HOST HEALTH: IMPLICATIONS OF PREBIOTIC NUTRITIONAL INTERVENTION. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/pharmacol_etds/25


University of Kentucky

20. Wise, James Tate Fortin. METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS.

Degree: 2019, University of Kentucky

 Hexavalent chromium, Cr(VI), is an established human carcinogen that is a worldwide environmental health concern. It is well understood that reactive oxygen species, genomic instability,… (more)

Subjects/Keywords: Cellular Energetics; Hexavalent Chromium; Lung Cancer; Lipogenesis; “Warburg effect”; Medical Biochemistry; Medical Cell Biology; Medical Nutrition; Medical Toxicology

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APA (6th Edition):

Wise, J. T. F. (2019). METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/27

Chicago Manual of Style (16th Edition):

Wise, James Tate Fortin. “METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS.” 2019. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pharmacol_etds/27.

MLA Handbook (7th Edition):

Wise, James Tate Fortin. “METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS.” 2019. Web. 23 Jul 2019.

Vancouver:

Wise JTF. METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS. [Internet] [Doctoral dissertation]. University of Kentucky; 2019. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pharmacol_etds/27.

Council of Science Editors:

Wise JTF. METABOLISM REPROGRAMMING IN HEXAVALENT CHROMIUM-INDUCED HUMAN LUNG CARCINOGENESIS. [Doctoral Dissertation]. University of Kentucky; 2019. Available from: https://uknowledge.uky.edu/pharmacol_etds/27

21. Rodríguez, Janice Ortega. NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE.

Degree: 2012, University of Kentucky

 DNA mismatch repair (MMR) is a critical genome-maintenance system. It ensures genome stability by correcting mismatches generated during DNA replication, suppressing homologous recombination, and inducing… (more)

Subjects/Keywords: PCNA; MutLα; Phosphorylation; HNPCC; AML; Medical Toxicology

…process easier. I am deeply thankful to the Chair of Toxicology, Dr. Mary Vore, who provided me… 

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APA (6th Edition):

Rodríguez, J. O. (2012). NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/1

Chicago Manual of Style (16th Edition):

Rodríguez, Janice Ortega. “NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE.” 2012. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/toxicology_etds/1.

MLA Handbook (7th Edition):

Rodríguez, Janice Ortega. “NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE.” 2012. Web. 23 Jul 2019.

Vancouver:

Rodríguez JO. NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE. [Internet] [Doctoral dissertation]. University of Kentucky; 2012. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/toxicology_etds/1.

Council of Science Editors:

Rodríguez JO. NOVEL MECHANISM LEADING TO MISMATCH REPAIR DEFICIENCY AND MUTATOR PHENOTYPE. [Doctoral Dissertation]. University of Kentucky; 2012. Available from: https://uknowledge.uky.edu/toxicology_etds/1


University of Kentucky

22. Shen, Huiyun. ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION.

Degree: 2008, University of Kentucky

 Atherosclerosis is a chronic inflammatory disease thought to be initiated by endothelial cell dysfunction. Research described in this dissertation is focused on the role of… (more)

Subjects/Keywords: atherosclerosis; zinc deficiency; NF-êB; PPAR; AhR; Medical Toxicology

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APA (6th Edition):

Shen, H. (2008). ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/610

Chicago Manual of Style (16th Edition):

Shen, Huiyun. “ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION.” 2008. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/610.

MLA Handbook (7th Edition):

Shen, Huiyun. “ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION.” 2008. Web. 23 Jul 2019.

Vancouver:

Shen H. ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION. [Internet] [Doctoral dissertation]. University of Kentucky; 2008. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/610.

Council of Science Editors:

Shen H. ZINC DEFICIENCY AND MECHANISMS OF ENDOTHELIAL CELL DYSFUNCTION. [Doctoral Dissertation]. University of Kentucky; 2008. Available from: https://uknowledge.uky.edu/gradschool_diss/610


University of Kentucky

23. Murphy, Lynea Alene. IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS.

Degree: 2008, University of Kentucky

 At the beginning of mitosis, chromosomes are condensed and segregated to facilitate correct alignment later in cytokinesis. Condensin is the pentameric enzyme responsible for this… (more)

Subjects/Keywords: PRC1; Condensin phosphorylation; cytokinesis; HSF2; bookmarking; Medical Toxicology

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APA (6th Edition):

Murphy, L. A. (2008). IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/645

Chicago Manual of Style (16th Edition):

Murphy, Lynea Alene. “IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS.” 2008. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/645.

MLA Handbook (7th Edition):

Murphy, Lynea Alene. “IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS.” 2008. Web. 23 Jul 2019.

Vancouver:

Murphy LA. IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS. [Internet] [Doctoral dissertation]. University of Kentucky; 2008. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/645.

Council of Science Editors:

Murphy LA. IMPLICATIONS FOR THE HSF2/PRC1 INTERACTION AND REGULATION OF CONDENSIN BY PHOSPHORYLATION DURING MITOSIS. [Doctoral Dissertation]. University of Kentucky; 2008. Available from: https://uknowledge.uky.edu/gradschool_diss/645


University of Kentucky

24. Anantharaman, Muthuswamy. NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH.

Degree: 2008, University of Kentucky

 Alzheimer’s disease (AD) is a multifactorial, progressive, age-related neurodegenerative disease. Oxidative stress hypothesis is most prevalent and is gaining significant support. Inspite of the progress… (more)

Subjects/Keywords: Alzheimer's disease; Mitochondria; MnSOD; Nitration; Energy Metabolism; Medical Toxicology

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APA (6th Edition):

Anantharaman, M. (2008). NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/665

Chicago Manual of Style (16th Edition):

Anantharaman, Muthuswamy. “NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH.” 2008. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gradschool_diss/665.

MLA Handbook (7th Edition):

Anantharaman, Muthuswamy. “NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH.” 2008. Web. 23 Jul 2019.

Vancouver:

Anantharaman M. NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH. [Internet] [Doctoral dissertation]. University of Kentucky; 2008. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gradschool_diss/665.

Council of Science Editors:

Anantharaman M. NITRATION AND INACTIVATION OF MANGANESE SUPEROXIDE DISMUTASE PLAYS A CRITICAL ROLE IN METABOLIC SWITCH. [Doctoral Dissertation]. University of Kentucky; 2008. Available from: https://uknowledge.uky.edu/gradschool_diss/665


University of Kentucky

25. Starnes, Daniel L. THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS.

Degree: 2016, University of Kentucky

 In recent decades, there has been a rapid expansion in the use of manufactured nanoparticles (MNPs). Experimental evidence and material flow models predict that MNPs… (more)

Subjects/Keywords: Silver Nanomaterials; Zinc oxide nanomaterials; Bioaccumulation; Nanomaterial transformations; Wastewater treatment; Agriculture; Agricultural Science; Agriculture; Genomics; Toxicology

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APA (6th Edition):

Starnes, D. L. (2016). THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pss_etds/74

Chicago Manual of Style (16th Edition):

Starnes, Daniel L. “THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS.” 2016. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pss_etds/74.

MLA Handbook (7th Edition):

Starnes, Daniel L. “THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS.” 2016. Web. 23 Jul 2019.

Vancouver:

Starnes DL. THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS. [Internet] [Doctoral dissertation]. University of Kentucky; 2016. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pss_etds/74.

Council of Science Editors:

Starnes DL. THE EFFECTS OF MANUFACTURED NANOMATERIAL TRANSFORMATIONS ON BIOAVAILABILITY, TOXICITY AND TRANSCRIPTOMIC RESPONSES OF CAENORHABDITIS ELEGANS. [Doctoral Dissertation]. University of Kentucky; 2016. Available from: https://uknowledge.uky.edu/pss_etds/74


University of Kentucky

26. Al-Darraji, Ahmed Hamish Neamah. AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION.

Degree: 2019, University of Kentucky

 Introduction: Myocardial infarction (MI) remains the leading cause of morbidity and mortality worldwide. Induced by cardiomyocyte death, MI initiates a prolonged and uncontrolled inflammatory response… (more)

Subjects/Keywords: Myocardial infarction; Post-myocardial infarction inflammation; Macrophage; Macrophage polarization; Azithromycin; liposomal azithromycin; Medicinal Chemistry and Pharmaceutics; Pharmaceutics and Drug Design; Pharmacology; Pharmacology, Toxicology and Environmental Health; Pharmacy and Pharmaceutical Sciences; Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Al-Darraji, A. H. N. (2019). AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/30

Chicago Manual of Style (16th Edition):

Al-Darraji, Ahmed Hamish Neamah. “AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION.” 2019. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pharmacol_etds/30.

MLA Handbook (7th Edition):

Al-Darraji, Ahmed Hamish Neamah. “AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION.” 2019. Web. 23 Jul 2019.

Vancouver:

Al-Darraji AHN. AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION. [Internet] [Doctoral dissertation]. University of Kentucky; 2019. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pharmacol_etds/30.

Council of Science Editors:

Al-Darraji AHN. AZITHROMYCIN THERAPY REDUCES CARDIAC INFLAMMATION AND MITIGATES ADVERSE CARDIAC REMODELING AFTER MYOCARDIAL INFARCTION. [Doctoral Dissertation]. University of Kentucky; 2019. Available from: https://uknowledge.uky.edu/pharmacol_etds/30

27. Helsley, Robert N. THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE.

Degree: 2016, University of Kentucky

 Cardiovascular disease (CVD) is the leading cause of death worldwide and is partially attributed to perturbations in lipid metabolism. Xenobiotics, such as pharmaceutical drugs and… (more)

Subjects/Keywords: HIV drugs; Phthalates; Pregnane X Receptor; IκB Kinase β; Adipogenesis; Insulin Resistance; Cardiovascular Diseases; Hormones, Hormone Substitutes, and Hormone Antagonists; Lipids; Medical Molecular Biology; Medical Nutrition; Medical Pharmacology; Medical Sciences; Medical Toxicology; Molecular, Genetic, and Biochemical Nutrition; Nutritional and Metabolic Diseases; Other Pharmacology, Toxicology and Environmental Health

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Helsley, R. N. (2016). THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/14

Chicago Manual of Style (16th Edition):

Helsley, Robert N. “THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE.” 2016. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pharmacol_etds/14.

MLA Handbook (7th Edition):

Helsley, Robert N. “THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE.” 2016. Web. 23 Jul 2019.

Vancouver:

Helsley RN. THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE. [Internet] [Doctoral dissertation]. University of Kentucky; 2016. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pharmacol_etds/14.

Council of Science Editors:

Helsley RN. THE ROLE OF PXR AND IKKβ SIGNALING IN CARDIOMETABOLIC DISEASE. [Doctoral Dissertation]. University of Kentucky; 2016. Available from: https://uknowledge.uky.edu/pharmacol_etds/14


University of Kentucky

28. Lewis, Ricky W. TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA.

Degree: 2016, University of Kentucky

 Engineered nanomaterials (ENMs) have become ubiquitous in consumer products and industrial applications, and consequently the environment. Much of the environmentally released ENMs are expected to… (more)

Subjects/Keywords: engineered nanomaterial; plant growth promoting rhizobacteria; metal toxicity; nanotoxicology; respiration stress; high-throughput respiration assay; Agriculture; Bacteriology; Chemistry; Environmental Microbiology and Microbial Ecology; Microbial Physiology; Microbiology; Organismal Biological Physiology; Pharmacology, Toxicology and Environmental Health; Toxicology

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APA (6th Edition):

Lewis, R. W. (2016). TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pss_etds/77

Chicago Manual of Style (16th Edition):

Lewis, Ricky W. “TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA.” 2016. Doctoral Dissertation, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/pss_etds/77.

MLA Handbook (7th Edition):

Lewis, Ricky W. “TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA.” 2016. Web. 23 Jul 2019.

Vancouver:

Lewis RW. TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA. [Internet] [Doctoral dissertation]. University of Kentucky; 2016. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/pss_etds/77.

Council of Science Editors:

Lewis RW. TOXICITY OF ENGINEERED NANOMATERIALS TO PLANT GROWTH PROMOTING RHIZOBACTERIA. [Doctoral Dissertation]. University of Kentucky; 2016. Available from: https://uknowledge.uky.edu/pss_etds/77


University of Kentucky

29. Monteiro Davolli, Gabriel. REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST.

Degree: 2015, University of Kentucky

 The objectives of this thesis were: (1) to evaluate the downregulation of the stallion hypothalamic-pituitary-gonadal (HPG) axis by a GnRH antagonist (acyline) based upon endocrine,… (more)

Subjects/Keywords: Stallion; Equine Arteritis Virus (EAV); Sexual Behavior; Testosterone Suppression; Large or Food Animal and Equine Medicine; Veterinary Infectious Diseases; Veterinary Preventive Medicine, Epidemiology, and Public Health; Veterinary Toxicology and Pharmacology

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APA (6th Edition):

Monteiro Davolli, G. (2015). REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/gluck_etds/22

Chicago Manual of Style (16th Edition):

Monteiro Davolli, Gabriel. “REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST.” 2015. Masters Thesis, University of Kentucky. Accessed July 23, 2019. http://uknowledge.uky.edu/gluck_etds/22.

MLA Handbook (7th Edition):

Monteiro Davolli, Gabriel. “REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST.” 2015. Web. 23 Jul 2019.

Vancouver:

Monteiro Davolli G. REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST. [Internet] [Masters thesis]. University of Kentucky; 2015. [cited 2019 Jul 23]. Available from: http://uknowledge.uky.edu/gluck_etds/22.

Council of Science Editors:

Monteiro Davolli G. REVERSIBLE DOWNREGULATION OF HYPOTHALAMIC-PITUITARY-GONADAL AXIS IN THE STALLION WITH A THIRD-GENERATION GNRH ANTAGONIST. [Masters Thesis]. University of Kentucky; 2015. Available from: http://uknowledge.uky.edu/gluck_etds/22


University of Kentucky

30. Taylor, Victoria A. PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584.

Degree: 2017, University of Kentucky

 Kentucky 31 tall fescue (KY31) infected with the common toxic endophyte strains of Epichloё coenophiala produces toxic alkaloids that improve plant vigor, but cause numerous… (more)

Subjects/Keywords: foal; hormones; mare; novel endophyte; tall fescue; vasoconstriction; Animal Sciences; Large or Food Animal and Equine Medicine; Life Sciences; Plant Sciences; Veterinary Toxicology and Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Taylor, V. A. (2017). PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584. (Masters Thesis). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gluck_etds/33

Chicago Manual of Style (16th Edition):

Taylor, Victoria A. “PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584.” 2017. Masters Thesis, University of Kentucky. Accessed July 23, 2019. https://uknowledge.uky.edu/gluck_etds/33.

MLA Handbook (7th Edition):

Taylor, Victoria A. “PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584.” 2017. Web. 23 Jul 2019.

Vancouver:

Taylor VA. PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584. [Internet] [Masters thesis]. University of Kentucky; 2017. [cited 2019 Jul 23]. Available from: https://uknowledge.uky.edu/gluck_etds/33.

Council of Science Editors:

Taylor VA. PHYSIOLOGICAL CHANGES ASSOCIATED WITH PREGNANT OR NONPREGNANT MARES GRAZING PASTURES OF ORCHARDGRASS-BLUEGRASS, KENTUCKY 31 TALL FESCUE INFECTED WITH EPICHLOË COENOPHIALA, OR KYFA9821 TALL FESCUE INFECTED WITH THE NOVEL ENDOPHYTE AR584. [Masters Thesis]. University of Kentucky; 2017. Available from: https://uknowledge.uky.edu/gluck_etds/33

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