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You searched for subject:(Tau protein). Showing records 1 – 30 of 90 total matches.

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University of Manchester

1. Noble, Elizabeth. The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease.

Degree: 2017, University of Manchester

 Several lines of evidence suggest that the expression of the cellular prion protein (PrPC) is altered with age and in sporadic Alzheimer’s disease, however, published… (more)

Subjects/Keywords: Alzheimers; Amyloid; Prion protein; Tau

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Noble, E. (2017). The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306712

Chicago Manual of Style (16th Edition):

Noble, Elizabeth. “The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease.” 2017. Doctoral Dissertation, University of Manchester. Accessed November 13, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306712.

MLA Handbook (7th Edition):

Noble, Elizabeth. “The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease.” 2017. Web. 13 Nov 2019.

Vancouver:

Noble E. The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2019 Nov 13]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306712.

Council of Science Editors:

Noble E. The molecular mechanisms linking amyloid-beta, the prion protein and tau in Alzheimer's disease. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306712

2. Laurent, Cyril. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.

Degree: Docteur es, Neurosciences, 2013, Université Lille II – Droit et Santé

 Le vieillissement de la population est à l’origine de l’augmentation du nombre de personnes souffrant de démences, dont la plus fréquente est la maladie d’Alzheimer… (more)

Subjects/Keywords: Neuro-inflammation; Alzheimer's Disease; Tau Protein

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APA (6th Edition):

Laurent, C. (2013). Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2013LIL2S041

Chicago Manual of Style (16th Edition):

Laurent, Cyril. “Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.” 2013. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2013LIL2S041.

MLA Handbook (7th Edition):

Laurent, Cyril. “Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.” 2013. Web. 13 Nov 2019.

Vancouver:

Laurent C. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2013. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2013LIL2S041.

Council of Science Editors:

Laurent C. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2013. Available from: http://www.theses.fr/2013LIL2S041

3. Violet, Marie. Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition.

Degree: Docteur es, Neurosciences, 2014, Université Lille II – Droit et Santé

 La protéine Tau est impliquée dans de nombreuses maladies neurodégénératives regroupées sous le terme de Tauopathies dont la plus fréquente est la maladie d’Alzheimer (MA),… (more)

Subjects/Keywords: Protéines tau; Membrane nucléaire; Maladie d'Alzheimer; Membrane cellulaire; Stress oxydatif; Tau protein; Alzheimer's disease

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APA (6th Edition):

Violet, M. (2014). Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2014LIL2S008

Chicago Manual of Style (16th Edition):

Violet, Marie. “Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition.” 2014. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2014LIL2S008.

MLA Handbook (7th Edition):

Violet, Marie. “Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition.” 2014. Web. 13 Nov 2019.

Vancouver:

Violet M. Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2014. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2014LIL2S008.

Council of Science Editors:

Violet M. Fonctions atypiques de Tau en conditions physiologique et pathologique : Atypical functions of Tau in physiological and pathological condition. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2014. Available from: http://www.theses.fr/2014LIL2S008

4. Talmat-Amar, Yasmina. Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model.

Degree: Docteur es, Neurosciences, 2012, Université Montpellier I

 La protéine Tau est une protéine associée aux microtubules, localisée principalement dans les axones. Elle joue un rôle important dans la polymérisation et la stabilisation… (more)

Subjects/Keywords: Protéine Tau; Microtubules; Transport axonal; Maladie d'Alzheimer; Tau protein; Microtubules; Axonal transport; Alzheimer disease

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APA (6th Edition):

Talmat-Amar, Y. (2012). Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2012MON1T005

Chicago Manual of Style (16th Edition):

Talmat-Amar, Yasmina. “Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model.” 2012. Doctoral Dissertation, Université Montpellier I. Accessed November 13, 2019. http://www.theses.fr/2012MON1T005.

MLA Handbook (7th Edition):

Talmat-Amar, Yasmina. “Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model.” 2012. Web. 13 Nov 2019.

Vancouver:

Talmat-Amar Y. Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model. [Internet] [Doctoral dissertation]. Université Montpellier I; 2012. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2012MON1T005.

Council of Science Editors:

Talmat-Amar Y. Étude de la toxicité neuronale induite par la protéine Tau dans la maladie d’Alzheimer, sur un modèle Invertébré : Drosophila melanogaster : Toward understanding the mechanisms of Tau induced neurotoxicity in Alzheimer disease using Drosophila melanogaster model. [Doctoral Dissertation]. Université Montpellier I; 2012. Available from: http://www.theses.fr/2012MON1T005

5. Hervy, Jordan. Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules.

Degree: Docteur es, Physique pour les sciences du vivant, 2018, Grenoble Alpes

La maladie d’Alzheimer, de nombreux syndromes parkinsoniens, certaines démences fronto-temporales telle que la maladie de Pick sont des exemples de maladies neurodégénératives appelées « tauopathies… (more)

Subjects/Keywords: Tauopathies; Microtubule; Protéine Tau; Axone; Modélisation; Tauopathies; Microtubule; Tau protein; Axon; Modeling; 530

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APA (6th Edition):

Hervy, J. (2018). Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules. (Doctoral Dissertation). Grenoble Alpes. Retrieved from http://www.theses.fr/2018GREAY062

Chicago Manual of Style (16th Edition):

Hervy, Jordan. “Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules.” 2018. Doctoral Dissertation, Grenoble Alpes. Accessed November 13, 2019. http://www.theses.fr/2018GREAY062.

MLA Handbook (7th Edition):

Hervy, Jordan. “Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules.” 2018. Web. 13 Nov 2019.

Vancouver:

Hervy J. Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules. [Internet] [Doctoral dissertation]. Grenoble Alpes; 2018. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2018GREAY062.

Council of Science Editors:

Hervy J. Modélisation de l'interaction dynamique protéines Tau - microtubules : Modeling the dynamical interaction Tau Proteins - microtubules. [Doctoral Dissertation]. Grenoble Alpes; 2018. Available from: http://www.theses.fr/2018GREAY062

6. Refenes, Nikolaos. Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης.

Degree: 2017, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The interaction between the proteins α-synuclein and tau has been proposed as a possible mechanism to develop Parkinson’s disease (PD). The utility of the Drosophila… (more)

Subjects/Keywords: Νόσος Πάρκινσον; TAU πρωτεΐνη; Α-συνουκλεΐνη; Δροσόφιλα; Parkinson's disease; TAU protein; A-synuclein; Drosophila

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APA (6th Edition):

Refenes, N. (2017). Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/40983

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Refenes, Nikolaos. “Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης.” 2017. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed November 13, 2019. http://hdl.handle.net/10442/hedi/40983.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Refenes, Nikolaos. “Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης.” 2017. Web. 13 Nov 2019.

Vancouver:

Refenes N. Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10442/hedi/40983.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Refenes N. Ανεξάρτητη και συνεργική επίδραση των γονιδίων MAPT και SNCA σε έναν πληθυσμό της Νότιας Ευρώπης με νόσο του Πάρκινσον και έλεγχος χημικών ενώσεων για την αναστολή των νευροτοξικών δράσεων της tau πρωτεΐνης. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. Available from: http://hdl.handle.net/10442/hedi/40983

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Thomaidou, Evanthia. Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς.

Degree: 2018, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

BACKGROUND: Postoperative cognitive decline and co-existing brain damage usually complicate cardiothoracic patients after surgery. This study investigated if patients could benefit from the perioperative administration… (more)

Subjects/Keywords: Ερυθρομυκίνη; Καρδιοχειρουργικές επεμβάσεις; Γνωσιακή έκπτωση; TAU πρωτεΐνη; Erythromycin; CABG; POCD; TAU protein

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APA (6th Edition):

Thomaidou, E. (2018). Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/43151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thomaidou, Evanthia. “Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς.” 2018. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed November 13, 2019. http://hdl.handle.net/10442/hedi/43151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thomaidou, Evanthia. “Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς.” 2018. Web. 13 Nov 2019.

Vancouver:

Thomaidou E. Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2018. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10442/hedi/43151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thomaidou E. Διερεύνηση της επίδρασης της προεγχειρητικής χορήγησης ερυθρομυκίνης στη γνωσιακή λειτουργία, στη διεγχειρητική εγκεφαλική οξυμετρία και σε βιοχημικούς δείκτες ισχαιμίας και φλεγμονής σε καρδιοχειρουργικούς ασθενείς. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2018. Available from: http://hdl.handle.net/10442/hedi/43151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Schirmer, Claire. Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide.

Degree: Docteur es, Biologie, 2014, Rennes 1

Les maladies dites ''conformationnelles'' sont caractérisées par un mauvais repliement des protéines qui, de ce fait, ne peuvent plus assurer leur fonction biologique. C'est le… (more)

Subjects/Keywords: Maladie d’Alzheimer; Protéine tau; Agrégation protéique; Fibres amyloides; Hsp90; Alzheimer's disease; Tau protein; Protein aggregation; Amyloid fibers; Hsp90

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schirmer, C. (2014). Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2014REN1S162

Chicago Manual of Style (16th Edition):

Schirmer, Claire. “Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide.” 2014. Doctoral Dissertation, Rennes 1. Accessed November 13, 2019. http://www.theses.fr/2014REN1S162.

MLA Handbook (7th Edition):

Schirmer, Claire. “Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide.” 2014. Web. 13 Nov 2019.

Vancouver:

Schirmer C. Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide. [Internet] [Doctoral dissertation]. Rennes 1; 2014. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2014REN1S162.

Council of Science Editors:

Schirmer C. Chaperons moléculaires et tauopathies : effets de Hsp90 sur la fibrillation in vitro du peptide VQIVYK issu de la protéine tau : Molecular chaperones and tauopathies : Hsp90's effect on fibrillation in vitro of VQIVYK the tau-derived peptide. [Doctoral Dissertation]. Rennes 1; 2014. Available from: http://www.theses.fr/2014REN1S162

9. Di Battista, Maria Elena. Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects.

Degree: 2015, Università degli Studi di Catania

 Il deficit cognitivo è uno degli aspetti più comuni e importanti della malattia di Parkinson (PD), influisce notevolmente la qualità e l aspettativa di vita… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; Parkinson Dementia, MAPT gene, tau protein

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APA (6th Edition):

Di Battista, M. E. (2015). Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/3836

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Di Battista, Maria Elena. “Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects.” 2015. Thesis, Università degli Studi di Catania. Accessed November 13, 2019. http://hdl.handle.net/10761/3836.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Di Battista, Maria Elena. “Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects.” 2015. Web. 13 Nov 2019.

Vancouver:

Di Battista ME. Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects. [Internet] [Thesis]. Università degli Studi di Catania; 2015. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10761/3836.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Di Battista ME. Dementia in Parkinson s Disease: relationship between clinical and neurobiological aspects. [Thesis]. Università degli Studi di Catania; 2015. Available from: http://hdl.handle.net/10761/3836

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Dinkel, Paul David. Seeded Propagation of Tau Fibrils.

Degree: PhD, Chemistry and Biochemistry, 2013, U of Denver

  In various neurodegenerative diseases, including Alzheimer's disease, progressive supranuclear palsy, Pick's disease, and corticobasal degeneration, the deposition of fibrils composed of misfolded tau protein(more)

Subjects/Keywords: Alzheimer's disease; amyloid; fibrils; filaments; protein misfolding; tau

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APA (6th Edition):

Dinkel, P. D. (2013). Seeded Propagation of Tau Fibrils. (Doctoral Dissertation). U of Denver. Retrieved from https://digitalcommons.du.edu/etd/799

Chicago Manual of Style (16th Edition):

Dinkel, Paul David. “Seeded Propagation of Tau Fibrils.” 2013. Doctoral Dissertation, U of Denver. Accessed November 13, 2019. https://digitalcommons.du.edu/etd/799.

MLA Handbook (7th Edition):

Dinkel, Paul David. “Seeded Propagation of Tau Fibrils.” 2013. Web. 13 Nov 2019.

Vancouver:

Dinkel PD. Seeded Propagation of Tau Fibrils. [Internet] [Doctoral dissertation]. U of Denver; 2013. [cited 2019 Nov 13]. Available from: https://digitalcommons.du.edu/etd/799.

Council of Science Editors:

Dinkel PD. Seeded Propagation of Tau Fibrils. [Doctoral Dissertation]. U of Denver; 2013. Available from: https://digitalcommons.du.edu/etd/799


The Ohio State University

11. Schafer, Nicole D. Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease.

Degree: PhD, Biophysics, 2013, The Ohio State University

 The aggregation of tau protein in the brain is a defining pathological characteristic of several diseases, known as tauopathies, the most common of which is… (more)

Subjects/Keywords: Biophysics; Alzheimer's disease; tau protein aggregation; small molecule binding and inhibition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schafer, N. D. (2013). Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1366219959

Chicago Manual of Style (16th Edition):

Schafer, Nicole D. “Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease.” 2013. Doctoral Dissertation, The Ohio State University. Accessed November 13, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1366219959.

MLA Handbook (7th Edition):

Schafer, Nicole D. “Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease.” 2013. Web. 13 Nov 2019.

Vancouver:

Schafer ND. Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Nov 13]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1366219959.

Council of Science Editors:

Schafer ND. Targeting Tau Aggregation for the Diagnosis and Treatment of Alzheimer’s Disease. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1366219959


NSYSU

12. Yen, Yi-Chen. Molecular Interaction of Tau and Microtubule.

Degree: Master, Institute of Biomedical Sciences, 2002, NSYSU

Tau protein is one of the microtubule-associated proteins (MAPs) and mainly expressed in neuronal cells. It hasbeen demonstrated that Tau may play an important role… (more)

Subjects/Keywords: phosphorylation; tau protein; microtubule

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APA (6th Edition):

Yen, Y. (2002). Molecular Interaction of Tau and Microtubule. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yen, Yi-Chen. “Molecular Interaction of Tau and Microtubule.” 2002. Thesis, NSYSU. Accessed November 13, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yen, Yi-Chen. “Molecular Interaction of Tau and Microtubule.” 2002. Web. 13 Nov 2019.

Vancouver:

Yen Y. Molecular Interaction of Tau and Microtubule. [Internet] [Thesis]. NSYSU; 2002. [cited 2019 Nov 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yen Y. Molecular Interaction of Tau and Microtubule. [Thesis]. NSYSU; 2002. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

13. Apicco, Daniel. Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia.

Degree: PhD, Pharmacology, 2017, Boston University

 Abnormal aggregation of microtubule associated protein tau is the defining pathological hallmark of tauopathies, which include Alzheimer’s disease (AD) and related frontotemporal dementias (FTLD-tau). However,… (more)

Subjects/Keywords: Neurosciences; RNA binding protein; Tau; TIA1; Dementia; Neurodegeneration; Stress granule

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APA (6th Edition):

Apicco, D. (2017). Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/23406

Chicago Manual of Style (16th Edition):

Apicco, Daniel. “Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia.” 2017. Doctoral Dissertation, Boston University. Accessed November 13, 2019. http://hdl.handle.net/2144/23406.

MLA Handbook (7th Edition):

Apicco, Daniel. “Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia.” 2017. Web. 13 Nov 2019.

Vancouver:

Apicco D. Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia. [Internet] [Doctoral dissertation]. Boston University; 2017. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2144/23406.

Council of Science Editors:

Apicco D. Emerging roles for RNA binding proteins in the pathogenesis of Alzheimer's disease and frontotemporal dementia. [Doctoral Dissertation]. Boston University; 2017. Available from: http://hdl.handle.net/2144/23406


University of New Mexico

14. Jones, Emmalee. Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes.

Degree: Nanoscience and Microsystems, 2015, University of New Mexico

 A protein's sequence of amino acids determines how it folds. That folded structure is linked to protein function, and misfolding to dysfunction. Protein misfolding and… (more)

Subjects/Keywords: lipid membrane; tau protein; Alzheimer's disease; neutron reflectivity; X-ray scattering

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APA (6th Edition):

Jones, E. (2015). Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/25786

Chicago Manual of Style (16th Edition):

Jones, Emmalee. “Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes.” 2015. Doctoral Dissertation, University of New Mexico. Accessed November 13, 2019. http://hdl.handle.net/1928/25786.

MLA Handbook (7th Edition):

Jones, Emmalee. “Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes.” 2015. Web. 13 Nov 2019.

Vancouver:

Jones E. Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/1928/25786.

Council of Science Editors:

Jones E. Structural Elucidation of the Interaction Between Neurodegenerative Disease-Related Tau Protein with Model Lipid Membranes. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: http://hdl.handle.net/1928/25786


University of Michigan

15. Ismail, Tania. Role of Biomolecule Cues in Alzheimer’s disease.

Degree: MSin Engineering, Bioengineering, College of Engineering & Computer Science, 2017, University of Michigan

 The master’s thesis study investigates the role of biomolecules in Alzheimer’s disease. Alzheimer’s disease is caused by amyloid plaques and neurofibrillary tangles. The major cause… (more)

Subjects/Keywords: Protein; Alzheimers; Aggregation; Amyloid; Tau; Glycosaminoglycans; Biomedical engineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ismail, T. (2017). Role of Biomolecule Cues in Alzheimer’s disease. (Masters Thesis). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/140773

Chicago Manual of Style (16th Edition):

Ismail, Tania. “Role of Biomolecule Cues in Alzheimer’s disease.” 2017. Masters Thesis, University of Michigan. Accessed November 13, 2019. http://hdl.handle.net/2027.42/140773.

MLA Handbook (7th Edition):

Ismail, Tania. “Role of Biomolecule Cues in Alzheimer’s disease.” 2017. Web. 13 Nov 2019.

Vancouver:

Ismail T. Role of Biomolecule Cues in Alzheimer’s disease. [Internet] [Masters thesis]. University of Michigan; 2017. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2027.42/140773.

Council of Science Editors:

Ismail T. Role of Biomolecule Cues in Alzheimer’s disease. [Masters Thesis]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/140773

16. Papegaey, Anthony. Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers.

Degree: Docteur es, Neurosciences, 2016, Université Lille II – Droit et Santé

 Le terme dégénérescence lobaire frontotemporale ou FTLD définit un groupe hétérogène de maladies neurodégénératives caractérisé par des troubles du langage, du comportement et/ou moteurs qui… (more)

Subjects/Keywords: Astrogliose; Progranuline; Protéine Tau; Démence; Dégénérescence lobaire frontotemporale; Marqueurs biologiques; Transmission synaptique; Astrogliosis; Progranulin; Synaptic impairment; Tau protein; Frontotemporal lobar degeneration

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APA (6th Edition):

Papegaey, A. (2016). Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2016LIL2S048

Chicago Manual of Style (16th Edition):

Papegaey, Anthony. “Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers.” 2016. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2016LIL2S048.

MLA Handbook (7th Edition):

Papegaey, Anthony. “Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers.” 2016. Web. 13 Nov 2019.

Vancouver:

Papegaey A. Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2016. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2016LIL2S048.

Council of Science Editors:

Papegaey A. Dégénérescences lobaires frontotemporales : vers une nouvelle classification, vers de nouveaux marqueurs : Frontotemporal lobar degeneration : to a new classification, to new markers. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2016. Available from: http://www.theses.fr/2016LIL2S048

17. Sigala, Julien. Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility.

Degree: Docteur es, Biologie et médecine du développement et de la reproduction, 2016, Université Lille II – Droit et Santé

La mobilité est une fonction clé de la qualité fécondante et de sélection des spermatozoïdes des techniques d’assistance médicale à la procréation (AMP). Nous manquons… (more)

Subjects/Keywords: Protéine Tau; Protélyse; Protéome; Marqueurs biologiques; PMA; Infertilité; Stérilité; Spermatozoïdes; Tau protein; Sperm; Proteolysis; Assisted reproductive technology; ART; Biomarker; Spermatocyte

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APA (6th Edition):

Sigala, J. (2016). Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2016LIL2S039

Chicago Manual of Style (16th Edition):

Sigala, Julien. “Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility.” 2016. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2016LIL2S039.

MLA Handbook (7th Edition):

Sigala, Julien. “Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility.” 2016. Web. 13 Nov 2019.

Vancouver:

Sigala J. Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2016. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2016LIL2S039.

Council of Science Editors:

Sigala J. Qualité du protéome du spermatozoïde humain et infertilité : Human sperm proteome quality and infertility. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2016. Available from: http://www.theses.fr/2016LIL2S039

18. Marciniak, Elodie. Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease.

Degree: Docteur es, Neurosciences, 2015, Université Lille II – Droit et Santé

 La maladie d’Alzheimer (MA) est une maladie neurodégénérative caractérisée d’un point de vue anatomopathologique par une accumulation extracellulaire de peptides amyloïdes et d’une dégénérescence neurofibrillaire… (more)

Subjects/Keywords: Maladie d'Alzheimer; Insulinorésistance; Métabolisme; Mémoire; Protéine Tau; Plasticité neuronale; Chimiokine CCL3; Neuroinflammation; Alzheimer's disease; Chemokines; Neuroinflammation; Tau protein

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APA (6th Edition):

Marciniak, E. (2015). Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2015LIL2S036

Chicago Manual of Style (16th Edition):

Marciniak, Elodie. “Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease.” 2015. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2015LIL2S036.

MLA Handbook (7th Edition):

Marciniak, Elodie. “Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease.” 2015. Web. 13 Nov 2019.

Vancouver:

Marciniak E. Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2015. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2015LIL2S036.

Council of Science Editors:

Marciniak E. Neuroinflammation & Insulinorésistance : contribution au développement physiopathologique de la maladie d’Alzheimer : Neuroinflammation & Insulinoresistance : involvement in pathophysiological development of Alzheimer Disease. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2015. Available from: http://www.theses.fr/2015LIL2S036


University of Gothenburg / Göteborgs Universitet

19. Jessen Krut, Jan. On cerebrospinal fluid biomarkers of HIV-1 infection.

Degree: 2015, University of Gothenburg / Göteborgs Universitet

 HIV invades the central nervous system (CNS) shortly after transmission and is present throughout the course of infection, causing immune activation and neuroinflammation. If left… (more)

Subjects/Keywords: HIV-1; HIV-associated neurocognitive disorders; amyloid protein; tau protein; neurofilament protein; central nervous system; cerebrospinal fluid

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APA (6th Edition):

Jessen Krut, J. (2015). On cerebrospinal fluid biomarkers of HIV-1 infection. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/40449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jessen Krut, Jan. “On cerebrospinal fluid biomarkers of HIV-1 infection.” 2015. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed November 13, 2019. http://hdl.handle.net/2077/40449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jessen Krut, Jan. “On cerebrospinal fluid biomarkers of HIV-1 infection.” 2015. Web. 13 Nov 2019.

Vancouver:

Jessen Krut J. On cerebrospinal fluid biomarkers of HIV-1 infection. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2077/40449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jessen Krut J. On cerebrospinal fluid biomarkers of HIV-1 infection. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. Available from: http://hdl.handle.net/2077/40449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Melo, Thaiany Quevedo. Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração.

Degree: Mestrado, Fisiopatologia Experimental, 2012, University of São Paulo

A neurodegeneração é um processo onde ocorre morte celular progressiva. O tráfego neuronal anterógrado e retrógado, e entre os compartimentos é essencial para a viabilidade… (more)

Subjects/Keywords: Alfa-sinucleína; Alfa-sinucleína; Amyloid beta-peptides; Doenças neurodegenerativas; Neurodegenerative diseases; Peptídeos beta-amiloides; Protein transport; Proteína Rab; Proteínas tau; Rab protein; Tau proteins; Transporte proteico

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APA (6th Edition):

Melo, T. Q. (2012). Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-10082012-122654/ ;

Chicago Manual of Style (16th Edition):

Melo, Thaiany Quevedo. “Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração.” 2012. Masters Thesis, University of São Paulo. Accessed November 13, 2019. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-10082012-122654/ ;.

MLA Handbook (7th Edition):

Melo, Thaiany Quevedo. “Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração.” 2012. Web. 13 Nov 2019.

Vancouver:

Melo TQ. Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2019 Nov 13]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-10082012-122654/ ;.

Council of Science Editors:

Melo TQ. Análise da expressão das proteínas Rab anterior à agregação proteica associada a neurodegeneração. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-10082012-122654/ ;


Université de Grenoble

21. Gallat, Francois-Xavier. Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques.

Degree: Docteur es, Physique, 2011, Université de Grenoble

Ce travail de cette thèse porte sur la dynamique des protéines, accompagnées de leur eau d'hydratation, une couche de solvant autour des protéines vitale pour… (more)

Subjects/Keywords: Protéine Tau; Diffusion incohérente de neutrons; Spectroscopie terahertz; Deutération de protéines; Transition dynamique; Protein Tau; Incoherent neutron scattering -TeraHertz Spectroscopy; Terahertz spectroscopy; Protein deuteration; Dynamic transition; 530

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APA (6th Edition):

Gallat, F. (2011). Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2011GRENY061

Chicago Manual of Style (16th Edition):

Gallat, Francois-Xavier. “Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques.” 2011. Doctoral Dissertation, Université de Grenoble. Accessed November 13, 2019. http://www.theses.fr/2011GRENY061.

MLA Handbook (7th Edition):

Gallat, Francois-Xavier. “Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques.” 2011. Web. 13 Nov 2019.

Vancouver:

Gallat F. Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques. [Internet] [Doctoral dissertation]. Université de Grenoble; 2011. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2011GRENY061.

Council of Science Editors:

Gallat F. Dynamique des protéines et de la couche d'hydratation étudiée par diffusion de neutrons et méthodes biophysiques complémentaires : Dynamics of proteins and the hydration shell's, explored by neutron scattering and other biophysical techniques. [Doctoral Dissertation]. Université de Grenoble; 2011. Available from: http://www.theses.fr/2011GRENY061


University of Vermont

22. Ali, Rehan. An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion.

Degree: MS, Neuroscience, 2018, University of Vermont

  Neurons are polarized cells with dendrites and an axon projecting from their cell body. Due to this polarized structure a major challenge for neurons… (more)

Subjects/Keywords: Data Analysis; Kinesin Motors; Matlab; Particle Motion; Single Particle Tracking; Tau Protein; Neuroscience and Neurobiology

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APA (6th Edition):

Ali, R. (2018). An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ali, Rehan. “An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion.” 2018. Thesis, University of Vermont. Accessed November 13, 2019. https://scholarworks.uvm.edu/graddis/870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ali, Rehan. “An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion.” 2018. Web. 13 Nov 2019.

Vancouver:

Ali R. An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion. [Internet] [Thesis]. University of Vermont; 2018. [cited 2019 Nov 13]. Available from: https://scholarworks.uvm.edu/graddis/870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ali R. An Automated Analysis Of Single Particle Tracking Data For Proteins That Exhibit Multi Component Motion. [Thesis]. University of Vermont; 2018. Available from: https://scholarworks.uvm.edu/graddis/870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

23. Wang, Yin-Hsuan. The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells.

Degree: Master, Biological Sciences, 2017, NSYSU

 We previously described that PKA/GSKIP/GSK3β complex serves as a platform to anchor and phosphorylate Drp1 affecting mitochondria dynamics to provide neuroprotection. Recently, a known PKA/GSK3β… (more)

Subjects/Keywords: PKA; Tau; Alzheimer disease; Human induced pluripotent stem (iPS) cells; Aβ precursor protein (; GSK3β; GSKIP

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APA (6th Edition):

Wang, Y. (2017). The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0404117-114837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Yin-Hsuan. “The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells.” 2017. Thesis, NSYSU. Accessed November 13, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0404117-114837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Yin-Hsuan. “The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells.” 2017. Web. 13 Nov 2019.

Vancouver:

Wang Y. The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Nov 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0404117-114837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang Y. The cAMP/PKA-mediated Tau S409 phosphorylation through GSKIP/GSK3 axis in SH-SY5Y and iPS cells. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0404117-114837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

24. Wobst, Heike Julia. Mouse models for the investigation of MAPT and its role in neurodegenerative disease.

Degree: PhD, 2013, University of Oxford

 Microtubule-associated protein tau plays an important role in many neurodegenerative diseases. By using and creating various novel mouse models, the role of tau and its… (more)

Subjects/Keywords: 612.8; Life Sciences; Biochemistry; Biology (medical sciences); Neuroscience; neurodegeneration; frontotemporal dementia; tau protein

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APA (6th Edition):

Wobst, H. J. (2013). Mouse models for the investigation of MAPT and its role in neurodegenerative disease. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:d8777b25-18f2-4919-9af0-36b23fcebeab ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647530

Chicago Manual of Style (16th Edition):

Wobst, Heike Julia. “Mouse models for the investigation of MAPT and its role in neurodegenerative disease.” 2013. Doctoral Dissertation, University of Oxford. Accessed November 13, 2019. http://ora.ox.ac.uk/objects/uuid:d8777b25-18f2-4919-9af0-36b23fcebeab ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647530.

MLA Handbook (7th Edition):

Wobst, Heike Julia. “Mouse models for the investigation of MAPT and its role in neurodegenerative disease.” 2013. Web. 13 Nov 2019.

Vancouver:

Wobst HJ. Mouse models for the investigation of MAPT and its role in neurodegenerative disease. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Nov 13]. Available from: http://ora.ox.ac.uk/objects/uuid:d8777b25-18f2-4919-9af0-36b23fcebeab ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647530.

Council of Science Editors:

Wobst HJ. Mouse models for the investigation of MAPT and its role in neurodegenerative disease. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:d8777b25-18f2-4919-9af0-36b23fcebeab ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647530


University of Kentucky

25. Meier, Shelby. PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION.

Degree: 2019, University of Kentucky

 Tauopathies are a group of neurodegenerative diseases characterized by the abnormal deposition of the protein tau, a microtubule stabilizing protein. Under normal physiological conditions tau(more)

Subjects/Keywords: tau; protein translation; Alzheimer's disease; traumatic brain injury; neurofibrillary tangles; chronic traumatic encephalopathy; Neurosciences

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APA (6th Edition):

Meier, S. (2019). PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/physiology_etds/44

Chicago Manual of Style (16th Edition):

Meier, Shelby. “PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION.” 2019. Doctoral Dissertation, University of Kentucky. Accessed November 13, 2019. https://uknowledge.uky.edu/physiology_etds/44.

MLA Handbook (7th Edition):

Meier, Shelby. “PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION.” 2019. Web. 13 Nov 2019.

Vancouver:

Meier S. PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION. [Internet] [Doctoral dissertation]. University of Kentucky; 2019. [cited 2019 Nov 13]. Available from: https://uknowledge.uky.edu/physiology_etds/44.

Council of Science Editors:

Meier S. PATHOLOGICAL TAU AS A CAUSE, AND CONSEQUENCE, OF CELLULAR DYSFUNCTION. [Doctoral Dissertation]. University of Kentucky; 2019. Available from: https://uknowledge.uky.edu/physiology_etds/44

26. Gulisano, Walter. A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology.

Degree: 2018, Università degli Studi di Catania

 The aim of this thesis was to study the pathogenetic mechanisms underlying Alzheimer s disease (AD), a neurodegenerative disorder affecting the elderly and characterized by… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; Amyloid beta peptide, tau protein, Alzheimer's disease, synaptic palsticity

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APA (6th Edition):

Gulisano, W. (2018). A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/4152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gulisano, Walter. “A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology.” 2018. Thesis, Università degli Studi di Catania. Accessed November 13, 2019. http://hdl.handle.net/10761/4152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gulisano, Walter. “A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology.” 2018. Web. 13 Nov 2019.

Vancouver:

Gulisano W. A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology. [Internet] [Thesis]. Università degli Studi di Catania; 2018. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10761/4152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gulisano W. A renewed vision for Amyloid beta and tau in Alzheimer s disease pathophysiology. [Thesis]. Università degli Studi di Catania; 2018. Available from: http://hdl.handle.net/10761/4152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

27. Krüger, J. (Johanna). Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration.

Degree: 2010, University of Oulu

 Abstract Alzheimer's disease (AD) and frontotemporal lobar degeneration (FTLD) are the two most common neurodegenerative diseases leading to early onset dementia (< 65 years). Mutations in… (more)

Subjects/Keywords: Alzheimer’s disease; amyloid precursor protein; frontotemporal lobar degeneration; genetics; haplogroup; microtubule associated protein tau; mitochondrial DNA; polymorphism; presenilin; progranulin

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APA (6th Edition):

Krüger, J. (. (2010). Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514263156

Chicago Manual of Style (16th Edition):

Krüger, J (Johanna). “Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration.” 2010. Doctoral Dissertation, University of Oulu. Accessed November 13, 2019. http://urn.fi/urn:isbn:9789514263156.

MLA Handbook (7th Edition):

Krüger, J (Johanna). “Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration.” 2010. Web. 13 Nov 2019.

Vancouver:

Krüger J(. Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration. [Internet] [Doctoral dissertation]. University of Oulu; 2010. [cited 2019 Nov 13]. Available from: http://urn.fi/urn:isbn:9789514263156.

Council of Science Editors:

Krüger J(. Molecular genetics of early-onset Alzheimer's disease and frontotemporal lobar degeneration. [Doctoral Dissertation]. University of Oulu; 2010. Available from: http://urn.fi/urn:isbn:9789514263156


Freie Universität Berlin

28. Reimann, Oliver. Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau.

Degree: 2017, Freie Universität Berlin

 Der Fokus dieser Dissertation liegt auf Tau, einem Schlüsselprotein in der Alzheimer Erkrankung. Im Fortlauf von Alzheimer wird Tau hyperphosphoryliert, wobei der hohe Grad der… (more)

Subjects/Keywords: tau protein; alzheimer's disease; NCL; protein chemistry; peptides; 500 Naturwissenschaften und Mathematik::540 Chemie::547 Organische Chemie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reimann, O. (2017). Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12120

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reimann, Oliver. “Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau.” 2017. Thesis, Freie Universität Berlin. Accessed November 13, 2019. http://dx.doi.org/10.17169/refubium-12120.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reimann, Oliver. “Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau.” 2017. Web. 13 Nov 2019.

Vancouver:

Reimann O. Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2019 Nov 13]. Available from: http://dx.doi.org/10.17169/refubium-12120.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reimann O. Tag-freie semisynthetische Tau Proteine und neue Antikörper gegen Phospho-Tau. [Thesis]. Freie Universität Berlin; 2017. Available from: http://dx.doi.org/10.17169/refubium-12120

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

29. Broncel, Malgorzata. Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen.

Degree: 2011, Freie Universität Berlin

 Die Aggregation löslicher Proteine zu proteolytisch stabilen Fibrillen ist ein Kennzeichen neurodegenerativer Krankheiten. Dieses Phänomen wurde bei Proteinen beobachtet, die unter bestimmten Bedingungen strukturelle Übergänge… (more)

Subjects/Keywords: Coiled-coil; Microtububle-assisted Tau protein; Phosphorylation; Post-translational modification; Protein aggregation; 500 Naturwissenschaften und Mathematik::540 Chemie

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APA (6th Edition):

Broncel, M. (2011). Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Broncel, Malgorzata. “Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen.” 2011. Thesis, Freie Universität Berlin. Accessed November 13, 2019. http://dx.doi.org/10.17169/refubium-9797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Broncel, Malgorzata. “Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen.” 2011. Web. 13 Nov 2019.

Vancouver:

Broncel M. Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2019 Nov 13]. Available from: http://dx.doi.org/10.17169/refubium-9797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Broncel M. Synthetische Phosphopeptide und Phosphoproteine als Werkzeuge zum Studium der Aggregation von Peptiden und Proteinen. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-9797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Chauderlier, Alban. Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism.

Degree: Docteur es, Neurosciences, 2016, Université Lille II – Droit et Santé

 Les tauopathies, dont la maladie d’Alzheimer (MA) est l’exemple le plus connu, sont des maladies neurodégénératives caractérisées par une agrégation intra-neuronale progressive de protéine Tau(more)

Subjects/Keywords: DDX6; Protéines Tau; Maladie d'Alzheimer; Régulation de la stabilité de l'ARNm; Dommages à l'ARN; Tau protein; Alzheimer’s disease (AD); Hyperthermia; DNA damage; RNA damage

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APA (6th Edition):

Chauderlier, A. (2016). Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2016LIL2S054

Chicago Manual of Style (16th Edition):

Chauderlier, Alban. “Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism.” 2016. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed November 13, 2019. http://www.theses.fr/2016LIL2S054.

MLA Handbook (7th Edition):

Chauderlier, Alban. “Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism.” 2016. Web. 13 Nov 2019.

Vancouver:

Chauderlier A. Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2016. [cited 2019 Nov 13]. Available from: http://www.theses.fr/2016LIL2S054.

Council of Science Editors:

Chauderlier A. Fonctions atypiques de la protéine Tau : rôles dans la protection des acides nucléiques et le métabolisme des ARN : Atypical functions of Tau protein : roles in nucleic acid protection and RNA metabolism. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2016. Available from: http://www.theses.fr/2016LIL2S054

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