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You searched for subject:(TRPV1). Showing records 1 – 30 of 92 total matches.

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University of Debrecen

1. Csató, Viktória. A vaszkuláris vanilloid receptor farmakológiai vizsgálata .

Degree: DE – TEK – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2010, University of Debrecen

 A vanilloid receptor (TRPV1) egy kalcium ionokat is átengedő nem specifikus kation csatorna. A TRPV1 elsősorban a fájdalmat közvetítő nociceptorokon, a primer szenzoros neuronokon expresszálódik.… (more)

Subjects/Keywords: TRPV1; rezisztenciaér

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APA (6th Edition):

Csató, V. (2010). A vaszkuláris vanilloid receptor farmakológiai vizsgálata . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/96083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Csató, Viktória. “A vaszkuláris vanilloid receptor farmakológiai vizsgálata .” 2010. Thesis, University of Debrecen. Accessed June 25, 2019. http://hdl.handle.net/2437/96083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Csató, Viktória. “A vaszkuláris vanilloid receptor farmakológiai vizsgálata .” 2010. Web. 25 Jun 2019.

Vancouver:

Csató V. A vaszkuláris vanilloid receptor farmakológiai vizsgálata . [Internet] [Thesis]. University of Debrecen; 2010. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/2437/96083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Csató V. A vaszkuláris vanilloid receptor farmakológiai vizsgálata . [Thesis]. University of Debrecen; 2010. Available from: http://hdl.handle.net/2437/96083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Ho, Karen W. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.

Degree: PhD, Pharmacology, 2014, Vanderbilt University

 Astrocytes are one type of glia in the central nervous system, and they provide metabolic and structural support to neurons. Following injury astrocytes undergo an… (more)

Subjects/Keywords: astrocyte; migration; TRPV1

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APA (6th Edition):

Ho, K. W. (2014). The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;

Chicago Manual of Style (16th Edition):

Ho, Karen W. “The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed June 25, 2019. http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;.

MLA Handbook (7th Edition):

Ho, Karen W. “The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.” 2014. Web. 25 Jun 2019.

Vancouver:

Ho KW. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Jun 25]. Available from: http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;.

Council of Science Editors:

Ho KW. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;


University of Debrecen

3. Chandan, Harman Paul Singh. Possible pharmacological exploitations of Capsaicin and TRPV1 receptor .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Capsaicinoids are found to exert multiple pharmacological and physiological effects including the activities of analgesia, anticancer, anti-inflammation, antioxidant and anti-obesity. Therefore, capsaicinoids may have the… (more)

Subjects/Keywords: Capsaicin; TRPV1

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APA (6th Edition):

Chandan, H. P. S. (n.d.). Possible pharmacological exploitations of Capsaicin and TRPV1 receptor . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/230479

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chandan, Harman Paul Singh. “Possible pharmacological exploitations of Capsaicin and TRPV1 receptor .” Thesis, University of Debrecen. Accessed June 25, 2019. http://hdl.handle.net/2437/230479.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chandan, Harman Paul Singh. “Possible pharmacological exploitations of Capsaicin and TRPV1 receptor .” Web. 25 Jun 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Chandan HPS. Possible pharmacological exploitations of Capsaicin and TRPV1 receptor . [Internet] [Thesis]. University of Debrecen; [cited 2019 Jun 25]. Available from: http://hdl.handle.net/2437/230479.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Chandan HPS. Possible pharmacological exploitations of Capsaicin and TRPV1 receptor . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/230479

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Vermont

4. Manuelyan, Inessa. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.

Degree: Pharmacology, 2014, University of Vermont

  Research in the past on transient receptor potential cation channel subfamily V member 1 (TRPV1) has been limited to mainly nervous tissue TRPV1 because… (more)

Subjects/Keywords: Middle Meningeal Artery; Vascular Smooth Muscle TRPV1; TRPV1 mediated vasoconstriction; TRPV1 in smooth muscle cells; TRPV1-mediated mechanism of vasoconstriction

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APA (6th Edition):

Manuelyan, I. (2014). Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manuelyan, Inessa. “Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.” 2014. Thesis, University of Vermont. Accessed June 25, 2019. https://scholarworks.uvm.edu/hcoltheses/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manuelyan, Inessa. “Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery.” 2014. Web. 25 Jun 2019.

Vancouver:

Manuelyan I. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. [Internet] [Thesis]. University of Vermont; 2014. [cited 2019 Jun 25]. Available from: https://scholarworks.uvm.edu/hcoltheses/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manuelyan I. Capsaicin-Induced Ca2+ Influx and Constriction of the Middle Meningeal Artery. [Thesis]. University of Vermont; 2014. Available from: https://scholarworks.uvm.edu/hcoltheses/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

5. Ward, Nicholas John. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Neuronal responses to stress are an important component of neurodegenerative disease and may represent targets for therapeutic intervention. In normal and pathogenic physiological conditions, members… (more)

Subjects/Keywords: neurodegeneration; glaucoma; TRPV1; TRP channels

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APA (6th Edition):

Ward, N. J. (2014). The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;

Chicago Manual of Style (16th Edition):

Ward, Nicholas John. “The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed June 25, 2019. http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;.

MLA Handbook (7th Edition):

Ward, Nicholas John. “The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.” 2014. Web. 25 Jun 2019.

Vancouver:

Ward NJ. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Jun 25]. Available from: http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;.

Council of Science Editors:

Ward NJ. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;


University of Kentucky

6. Zhang, Guangfan. EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS.

Degree: 2008, University of Kentucky

 Airway hyperresponsivness (AHR) is one of most prominent pathophysiological features of asthma. Increasing evidence suggests that vagal bronchopulmonary afferents may be involved in the development… (more)

Subjects/Keywords: asthma; allergen; vagus; TRPV1; capsaicin; Medical Physiology

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APA (6th Edition):

Zhang, G. (2008). EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS. (Doctoral Dissertation). University of Kentucky. Retrieved from http://uknowledge.uky.edu/gradschool_diss/688

Chicago Manual of Style (16th Edition):

Zhang, Guangfan. “EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS.” 2008. Doctoral Dissertation, University of Kentucky. Accessed June 25, 2019. http://uknowledge.uky.edu/gradschool_diss/688.

MLA Handbook (7th Edition):

Zhang, Guangfan. “EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS.” 2008. Web. 25 Jun 2019.

Vancouver:

Zhang G. EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS. [Internet] [Doctoral dissertation]. University of Kentucky; 2008. [cited 2019 Jun 25]. Available from: http://uknowledge.uky.edu/gradschool_diss/688.

Council of Science Editors:

Zhang G. EFFECT OF CHRONIC AIRWAY INFLAMMATION INDUCED BY ALLERGEN SENSITIZATION ON VAGAL BRONCHOPULMONARY SENSORY NERVES IN RATS. [Doctoral Dissertation]. University of Kentucky; 2008. Available from: http://uknowledge.uky.edu/gradschool_diss/688

7. Buckley, Cadie L. Ion channel determinants of cardiac hypertrophy and cardiomyopathy.

Degree: 2016, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2011.

The work presented in this dissertation address the role of two ion channels; TRPV1 and CRACM1, and the… (more)

Subjects/Keywords: TRPV1; CRACM1

…2.4.1. Mice lacking functional TRPV1 42 2.4.2. Pressure overload model 43 2.4.3… …lacking functional TRPV1 during pressure overload cardiac hypertrophy 45 2.4.5. Mice lacking… …functional TRPV1 are protected from hypertrophy and apoptosis after modeled pressure overload… …cardiac hypertrophy 47 2.4.6. Mice lacking functional TRPV1 have less fibrosis, tissue… …Treatment of WT mice with a TRPV1 antagonist recapitulates findings from transgenic experiments… 

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APA (6th Edition):

Buckley, C. L. (2016). Ion channel determinants of cardiac hypertrophy and cardiomyopathy. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/101222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Buckley, Cadie L. “Ion channel determinants of cardiac hypertrophy and cardiomyopathy.” 2016. Thesis, University of Hawaii – Manoa. Accessed June 25, 2019. http://hdl.handle.net/10125/101222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Buckley, Cadie L. “Ion channel determinants of cardiac hypertrophy and cardiomyopathy.” 2016. Web. 25 Jun 2019.

Vancouver:

Buckley CL. Ion channel determinants of cardiac hypertrophy and cardiomyopathy. [Internet] [Thesis]. University of Hawaii – Manoa; 2016. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/10125/101222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Buckley CL. Ion channel determinants of cardiac hypertrophy and cardiomyopathy. [Thesis]. University of Hawaii – Manoa; 2016. Available from: http://hdl.handle.net/10125/101222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

8. Nolden, Alissa Allen. Biological factors involved in sensory responses to chemesthetic stimuli.

Degree: PhD, Food Science, 2016, Penn State University

 Chemesthetic agents consist of chemical compounds known to activate receptors involved with pain, touch and thermal perception, such as warming, cooling, stinging, pricking, burning and… (more)

Subjects/Keywords: capsaicin; ethanol; TRPV1; bitter; burn; psychophysics; TAS2Rs

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APA (6th Edition):

Nolden, A. A. (2016). Biological factors involved in sensory responses to chemesthetic stimuli. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/13185aua223

Chicago Manual of Style (16th Edition):

Nolden, Alissa Allen. “Biological factors involved in sensory responses to chemesthetic stimuli.” 2016. Doctoral Dissertation, Penn State University. Accessed June 25, 2019. https://etda.libraries.psu.edu/catalog/13185aua223.

MLA Handbook (7th Edition):

Nolden, Alissa Allen. “Biological factors involved in sensory responses to chemesthetic stimuli.” 2016. Web. 25 Jun 2019.

Vancouver:

Nolden AA. Biological factors involved in sensory responses to chemesthetic stimuli. [Internet] [Doctoral dissertation]. Penn State University; 2016. [cited 2019 Jun 25]. Available from: https://etda.libraries.psu.edu/catalog/13185aua223.

Council of Science Editors:

Nolden AA. Biological factors involved in sensory responses to chemesthetic stimuli. [Doctoral Dissertation]. Penn State University; 2016. Available from: https://etda.libraries.psu.edu/catalog/13185aua223


University of Saskatchewan

9. Lam, Doris. Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors.

Degree: 2012, University of Saskatchewan

 Diabetes can have a devastating effect on sensory nerves often leading to clinical symptoms of pain abnormalities termed diabetic sensory neuropathy (DSN). Converging evidence demonstrate… (more)

Subjects/Keywords: oxidative stress; hyperglycemia; TRPV1; P2X; antioxidant

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APA (6th Edition):

Lam, D. (2012). Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2012-04-420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lam, Doris. “Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors.” 2012. Thesis, University of Saskatchewan. Accessed June 25, 2019. http://hdl.handle.net/10388/ETD-2012-04-420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lam, Doris. “Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors.” 2012. Web. 25 Jun 2019.

Vancouver:

Lam D. Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors. [Internet] [Thesis]. University of Saskatchewan; 2012. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/10388/ETD-2012-04-420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lam D. Modulation of sensory function by high glucose-induced oxidative stress: Role of TRPV1 receptors. [Thesis]. University of Saskatchewan; 2012. Available from: http://hdl.handle.net/10388/ETD-2012-04-420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

10. Lin, Xiaohui. Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels .

Degree: 2018, University of Sydney

 The endocannabinoid (eCB) system was found to modulate synaptic transmission in the central nervous system (CNS). The retina carries out complex neural computations that involve… (more)

Subjects/Keywords: Endocannabinoid; Electrophysiology; Retinal Ganglion Cells; Retina; TRPV1

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APA (6th Edition):

Lin, X. (2018). Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/18946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Xiaohui. “Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels .” 2018. Thesis, University of Sydney. Accessed June 25, 2019. http://hdl.handle.net/2123/18946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Xiaohui. “Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels .” 2018. Web. 25 Jun 2019.

Vancouver:

Lin X. Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels . [Internet] [Thesis]. University of Sydney; 2018. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/2123/18946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin X. Modulation of retinal ganglion cell responses by the endocannabinoid system and the involvement of TRPV1 channels . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/18946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

11. Sprague, Jared Michael. TRPV1 Sensitization in Primary Sensory Neurons.

Degree: PhD, Biological Sciences in Dental Medicine, 2014, Harvard University

 Pain is a major personal and community burden throughout the world with currently limited treatment options for persistent pain due to unacceptable side effects, dependence… (more)

Subjects/Keywords: Neurosciences; Bortezomib; Optovin; Sensitization; TRPA1; TRPV1

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APA (6th Edition):

Sprague, J. M. (2014). TRPV1 Sensitization in Primary Sensory Neurons. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115

Chicago Manual of Style (16th Edition):

Sprague, Jared Michael. “TRPV1 Sensitization in Primary Sensory Neurons.” 2014. Doctoral Dissertation, Harvard University. Accessed June 25, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115.

MLA Handbook (7th Edition):

Sprague, Jared Michael. “TRPV1 Sensitization in Primary Sensory Neurons.” 2014. Web. 25 Jun 2019.

Vancouver:

Sprague JM. TRPV1 Sensitization in Primary Sensory Neurons. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2019 Jun 25]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115.

Council of Science Editors:

Sprague JM. TRPV1 Sensitization in Primary Sensory Neurons. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115

12. Hori, Kiyomi; Ozaki, Noriyuki; Suzuki, Shigeyuki. Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats.

Degree: 博士(医学), 2018, Nagoya University / 名古屋大学

The role of ion channels expressed in sensory neurons on mechanical and thermal hyperalgesia was examined in a rat model of cisplatin-induced peripheral neuropathy. The… (more)

Subjects/Keywords: Cisplatin; TRPV1; TRPV2; P2X3; ASIC3; Hyperalgesia

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APA (6th Edition):

Hori, Kiyomi; Ozaki, Noriyuki; Suzuki, S. (2018). Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/23011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hori, Kiyomi; Ozaki, Noriyuki; Suzuki, Shigeyuki. “Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed June 25, 2019. http://hdl.handle.net/2237/23011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hori, Kiyomi; Ozaki, Noriyuki; Suzuki, Shigeyuki. “Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats.” 2018. Web. 25 Jun 2019.

Vancouver:

Hori, Kiyomi; Ozaki, Noriyuki; Suzuki S. Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/2237/23011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hori, Kiyomi; Ozaki, Noriyuki; Suzuki S. Upregulations of P2X3 and ASIC3 involve in hyperalgesia induced by cisplatin administration in rats. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/23011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Medical Branch – Galveston

13. [No author]. Group III mGluR8 negatively modulates TRP channels .

Degree: University of Texas Medical Branch – Galveston

 Several lines of evidence indicate group III metabotropic glutamate receptors (mGluRs) have systemic anti-hyperalgesic effects. We hypothesized this could occur through modulation of TRP channels… (more)

Subjects/Keywords: mGluR8; TRPV1; TRPA1

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APA (6th Edition):

author], [. (n.d.). Group III mGluR8 negatively modulates TRP channels . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11203

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Group III mGluR8 negatively modulates TRP channels .” Thesis, University of Texas Medical Branch – Galveston. Accessed June 25, 2019. http://hdl.handle.net/2152.3/11203.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Group III mGluR8 negatively modulates TRP channels .” Web. 25 Jun 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. Group III mGluR8 negatively modulates TRP channels . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2019 Jun 25]. Available from: http://hdl.handle.net/2152.3/11203.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. Group III mGluR8 negatively modulates TRP channels . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11203

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


McMaster University

14. Balenko, Matthew. CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE.

Degree: MSc, 2014, McMaster University

Breast cancer has the highest incidence rate in women, accounting for more than 22% of all cancers and possessing a strong disposition to metastasize to… (more)

Subjects/Keywords: Glutamate; Cancer; Pain; TRPV1; Capsazepine; in vivo

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APA (6th Edition):

Balenko, M. (2014). CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/15970

Chicago Manual of Style (16th Edition):

Balenko, Matthew. “CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE.” 2014. Masters Thesis, McMaster University. Accessed June 25, 2019. http://hdl.handle.net/11375/15970.

MLA Handbook (7th Edition):

Balenko, Matthew. “CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE.” 2014. Web. 25 Jun 2019.

Vancouver:

Balenko M. CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE. [Internet] [Masters thesis]. McMaster University; 2014. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/11375/15970.

Council of Science Editors:

Balenko M. CAPSAZEPINE ATTENUATES CANCER-INDUCED BONE PAIN BY INHIBITING GLUTAMATE RELEASE. [Masters Thesis]. McMaster University; 2014. Available from: http://hdl.handle.net/11375/15970


University of Minnesota

15. Potenzieri, Carl Robert. Cannabinoid modulation of nociception and nociceptor activity during inflammation.

Degree: PhD, Neuroscience, 2009, University of Minnesota

 Previous studies have demonstrated that peripherally-administered cannabinoids at the site of injury produce antinociception in animal models of acute and persistent pain. Peripheral cannabinoid one… (more)

Subjects/Keywords: Cannabinoids; Hyperlagesia; Nociceptors; Pain; TRPV1 receptors; Neuroscience

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APA (6th Edition):

Potenzieri, C. R. (2009). Cannabinoid modulation of nociception and nociceptor activity during inflammation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/54666

Chicago Manual of Style (16th Edition):

Potenzieri, Carl Robert. “Cannabinoid modulation of nociception and nociceptor activity during inflammation.” 2009. Doctoral Dissertation, University of Minnesota. Accessed June 25, 2019. http://purl.umn.edu/54666.

MLA Handbook (7th Edition):

Potenzieri, Carl Robert. “Cannabinoid modulation of nociception and nociceptor activity during inflammation.” 2009. Web. 25 Jun 2019.

Vancouver:

Potenzieri CR. Cannabinoid modulation of nociception and nociceptor activity during inflammation. [Internet] [Doctoral dissertation]. University of Minnesota; 2009. [cited 2019 Jun 25]. Available from: http://purl.umn.edu/54666.

Council of Science Editors:

Potenzieri CR. Cannabinoid modulation of nociception and nociceptor activity during inflammation. [Doctoral Dissertation]. University of Minnesota; 2009. Available from: http://purl.umn.edu/54666


Duquesne University

16. Zeyzus Johns, Bree. TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.

Degree: MS, Biological Sciences, 2009, Duquesne University

 Transient Receptor Potential Vanilloid 1 plays an important role in the pain pathway. TRPV1 is expressed in primary afferent nociceptors and acts as a transducer… (more)

Subjects/Keywords: TRPV1; DRG; mRNA; CCI; pain; nociception

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APA (6th Edition):

Zeyzus Johns, B. (2009). TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. (Masters Thesis). Duquesne University. Retrieved from https://dsc.duq.edu/etd/1407

Chicago Manual of Style (16th Edition):

Zeyzus Johns, Bree. “TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.” 2009. Masters Thesis, Duquesne University. Accessed June 25, 2019. https://dsc.duq.edu/etd/1407.

MLA Handbook (7th Edition):

Zeyzus Johns, Bree. “TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.” 2009. Web. 25 Jun 2019.

Vancouver:

Zeyzus Johns B. TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. [Internet] [Masters thesis]. Duquesne University; 2009. [cited 2019 Jun 25]. Available from: https://dsc.duq.edu/etd/1407.

Council of Science Editors:

Zeyzus Johns B. TRPV1 mRNA is Differentially Expressed in Different Vertebral Levels of Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. [Masters Thesis]. Duquesne University; 2009. Available from: https://dsc.duq.edu/etd/1407


Duquesne University

17. Andersen, Karl A. Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.

Degree: MS, Biological Sciences, 2010, Duquesne University

 Transient Receptor Potential Vanilloid 1 (TRPV1) is ligand-gated ion channel that plays an important role in the pain signaling pathway. It is predominantly expressed by… (more)

Subjects/Keywords: CCI; DRG; QPCR; Rat; Splice Variant; TRPV1

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APA (6th Edition):

Andersen, K. A. (2010). Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. (Masters Thesis). Duquesne University. Retrieved from https://dsc.duq.edu/etd/263

Chicago Manual of Style (16th Edition):

Andersen, Karl A. “Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.” 2010. Masters Thesis, Duquesne University. Accessed June 25, 2019. https://dsc.duq.edu/etd/263.

MLA Handbook (7th Edition):

Andersen, Karl A. “Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury.” 2010. Web. 25 Jun 2019.

Vancouver:

Andersen KA. Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. [Internet] [Masters thesis]. Duquesne University; 2010. [cited 2019 Jun 25]. Available from: https://dsc.duq.edu/etd/263.

Council of Science Editors:

Andersen KA. Exploration of TRPV1 Splice Variant Expression in Rat Dorsal Root Ganglia Following Sciatic Nerve Injury. [Masters Thesis]. Duquesne University; 2010. Available from: https://dsc.duq.edu/etd/263


University of Vermont

18. Mathews, Morgan E. Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling.

Degree: Neurological Sciences, 2016, University of Vermont

  Stress can cause or contribute to bladder dysfunction though specific effects remain unclear. Individuals with interstitial cystitis (IC)/bladder pain syndrome (BPS) experience increased symptom… (more)

Subjects/Keywords: PACAP; RVS; urinary bladder; DRG; TRPV1; TRPV4

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APA (6th Edition):

Mathews, M. E. (2016). Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mathews, Morgan E. “Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling.” 2016. Thesis, University of Vermont. Accessed June 25, 2019. https://scholarworks.uvm.edu/hcoltheses/201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mathews, Morgan E. “Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling.” 2016. Web. 25 Jun 2019.

Vancouver:

Mathews ME. Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling. [Internet] [Thesis]. University of Vermont; 2016. [cited 2019 Jun 25]. Available from: https://scholarworks.uvm.edu/hcoltheses/201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mathews ME. Impacts of chronic stress on urinary bladder function & recovery through modification of PACAP signaling. [Thesis]. University of Vermont; 2016. Available from: https://scholarworks.uvm.edu/hcoltheses/201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

19. Bohlen, Christopher. Venom-derived toxins as biochemical and pharmacological probes of pain pathway.

Degree: Neuroscience, 2012, University of California – San Francisco

 Venoms often target vital processes to cause paralysis or death, but many types of venom also elicit notoriously intense pain. While these pain-producing effects can… (more)

Subjects/Keywords: Neurosciences; ASIC; pain; toxin; TRPV1; venom

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APA (6th Edition):

Bohlen, C. (2012). Venom-derived toxins as biochemical and pharmacological probes of pain pathway. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2wx6v5w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bohlen, Christopher. “Venom-derived toxins as biochemical and pharmacological probes of pain pathway.” 2012. Thesis, University of California – San Francisco. Accessed June 25, 2019. http://www.escholarship.org/uc/item/2wx6v5w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bohlen, Christopher. “Venom-derived toxins as biochemical and pharmacological probes of pain pathway.” 2012. Web. 25 Jun 2019.

Vancouver:

Bohlen C. Venom-derived toxins as biochemical and pharmacological probes of pain pathway. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Jun 25]. Available from: http://www.escholarship.org/uc/item/2wx6v5w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bohlen C. Venom-derived toxins as biochemical and pharmacological probes of pain pathway. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/2wx6v5w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

20. Warwick, Charles A. The role of complement component C5a in nociceptive sensitization.

Degree: PhD, Pharmacology, 2017, University of Iowa

  The complement system is a principal component of innate immunity. Recent studies have underscored the importance of C5a and other complement components in inflammatory… (more)

Subjects/Keywords: C5a; Complement; Inflammation; Macrophage; Pain; TRPV1; Pharmacology

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APA (6th Edition):

Warwick, C. A. (2017). The role of complement component C5a in nociceptive sensitization. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5676

Chicago Manual of Style (16th Edition):

Warwick, Charles A. “The role of complement component C5a in nociceptive sensitization.” 2017. Doctoral Dissertation, University of Iowa. Accessed June 25, 2019. https://ir.uiowa.edu/etd/5676.

MLA Handbook (7th Edition):

Warwick, Charles A. “The role of complement component C5a in nociceptive sensitization.” 2017. Web. 25 Jun 2019.

Vancouver:

Warwick CA. The role of complement component C5a in nociceptive sensitization. [Internet] [Doctoral dissertation]. University of Iowa; 2017. [cited 2019 Jun 25]. Available from: https://ir.uiowa.edu/etd/5676.

Council of Science Editors:

Warwick CA. The role of complement component C5a in nociceptive sensitization. [Doctoral Dissertation]. University of Iowa; 2017. Available from: https://ir.uiowa.edu/etd/5676

21. Santos, Fabio Martinez dos. Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática.

Degree: PhD, Ciências Morfofuncionais, 2015, University of São Paulo

A técnica de Mobilização Neural (MOB) é um método não invasivo que demonstrou tanto na pesquisa básica, como na pesquisa clinica ser eficaz na redução… (more)

Subjects/Keywords: DRG; DRG; Fisioterapia; Opióides; Opioids; PAG; PAG; Physiotherapy; Substance P; Substância P; TRPV1; TRPV1

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APA (6th Edition):

Santos, F. M. d. (2015). Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42131/tde-05102015-150313/ ;

Chicago Manual of Style (16th Edition):

Santos, Fabio Martinez dos. “Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática.” 2015. Doctoral Dissertation, University of São Paulo. Accessed June 25, 2019. http://www.teses.usp.br/teses/disponiveis/42/42131/tde-05102015-150313/ ;.

MLA Handbook (7th Edition):

Santos, Fabio Martinez dos. “Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática.” 2015. Web. 25 Jun 2019.

Vancouver:

Santos FMd. Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2019 Jun 25]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-05102015-150313/ ;.

Council of Science Editors:

Santos FMd. Mobilização neural: avaliação molecular e comportamental em ratos Wistar após indução de dor neuropática. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-05102015-150313/ ;


Universidade Federal de Santa Maria

22. Gabriela Trevisan dos Santos. CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO.

Degree: 2011, Universidade Federal de Santa Maria

O receptor de potencial transitório vanilóide 1 (TRPV1) é relevante para a percepção de estímulos nocivos e tem sido estudado como um alvo terapêutico para… (more)

Subjects/Keywords: capsaicina; TRPV1; Esteróide; vanilóide; antinocicepção; Vernonia; CIENCIAS BIOLOGICAS; Steroid; TRPV1; capsaicin; vanilloid; antinociception; Vernonia

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APA (6th Edition):

Santos, G. T. d. (2011). CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3985

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Gabriela Trevisan dos. “CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO.” 2011. Thesis, Universidade Federal de Santa Maria. Accessed June 25, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3985.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Gabriela Trevisan dos. “CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO.” 2011. Web. 25 Jun 2019.

Vancouver:

Santos GTd. CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2011. [cited 2019 Jun 25]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3985.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos GTd. CARACTERIZAÇÃO DO ESTERÓIDE α-ESPINASTEROL COMO UM NOVO ANTAGONISTA DO RECEPTOR TRPV1 COM EFEITO ANTINOCICEPTIVO. [Thesis]. Universidade Federal de Santa Maria; 2011. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3985

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Païta, Lucille. Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion.

Degree: Docteur es, Cardiologie, 2016, Lyon

L'infarctus du myocarde, une des causes majeures de mortalité à travers le monde, engendre une mort irréversible du muscle cardiaque suite à une ischémie. Cette… (more)

Subjects/Keywords: Coeur; TRPV1; Infarctus; Isoflurane; Cardiomyocytes; Calcium; Heart; TRPV1; Myocardial infarction; Isoflurane; Cardiomyocytes; Calcium; 571

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APA (6th Edition):

Païta, L. (2016). Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1329

Chicago Manual of Style (16th Edition):

Païta, Lucille. “Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion.” 2016. Doctoral Dissertation, Lyon. Accessed June 25, 2019. http://www.theses.fr/2016LYSE1329.

MLA Handbook (7th Edition):

Païta, Lucille. “Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion.” 2016. Web. 25 Jun 2019.

Vancouver:

Païta L. Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2019 Jun 25]. Available from: http://www.theses.fr/2016LYSE1329.

Council of Science Editors:

Païta L. Caractérisation du canal cationique TRPV1 dans les cardiomyocytes : Implication of TRPV1 in cardioprotection during ischemia reperfusion. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1329

24. MarÃlia Leite Dias. Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico.

Degree: Master, 2012, Universidade Federal do Ceará

A Riparina IV, uma alcamida sintetizada de Aniba riparia, foi testada em modelos animais padronizados de dor, bem como os possÃveis mecanismos de aÃÃo envolvidos.… (more)

Subjects/Keywords: FARMACOLOGIA; Riparina IV; Nociceptividade; TRPV1; TRPM8; Glutamate; Ãxido NÃtrico; Riparin IV; Nociception; TRPV1; TRPM8; Glutamate; Nitric oxide

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APA (6th Edition):

Dias, M. L. (2012). Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8632 ;

Chicago Manual of Style (16th Edition):

Dias, MarÃlia Leite. “Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico.” 2012. Masters Thesis, Universidade Federal do Ceará. Accessed June 25, 2019. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8632 ;.

MLA Handbook (7th Edition):

Dias, MarÃlia Leite. “Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico.” 2012. Web. 25 Jun 2019.

Vancouver:

Dias ML. Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico. [Internet] [Masters thesis]. Universidade Federal do Ceará 2012. [cited 2019 Jun 25]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8632 ;.

Council of Science Editors:

Dias ML. Atividade antinociceptiva da riparina IV: participaÃÃo dos receptores TRPV1, TRPM8, receptores glutamatÃrgicos e do Ãxido nÃtrico. [Masters Thesis]. Universidade Federal do Ceará 2012. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8632 ;


Université de Montréal

25. Zhao, Shangang. Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion .

Degree: 2011, Université de Montréal

 Les cellules beta pancréatiques sécrètent l’insuline lors d’une augmentation post-prandiale du glucose dans le sang. Ce processus essentiel est contrôlé par des facteurs physiologiques, nutritionnels… (more)

Subjects/Keywords: Diabète; Sécrétion d’insuline stimulée par le glucose; MAG; ABHD6; TRPV1; Diabetes; Glucose-stimulated insulin secretion; Monoacylglycerol; ABHD6; TRPV1

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APA (6th Edition):

Zhao, S. (2011). Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/7136

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhao, Shangang. “Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion .” 2011. Thesis, Université de Montréal. Accessed June 25, 2019. http://hdl.handle.net/1866/7136.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhao, Shangang. “Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion .” 2011. Web. 25 Jun 2019.

Vancouver:

Zhao S. Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/1866/7136.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhao S. Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/7136

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Megat, Salim. Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors.

Degree: Docteur es, Neurosciences, 2014, Université de Strasbourg

Les antidépresseurs ont un effet antiallodynique qui dépend de la stimulation des récepteurs β2-adrénergiques. Ceux-ci stimulent la production d’adénosine monophosphate cyclique (AMPc) régulé par les… (more)

Subjects/Keywords: Douleur neuropathique; PDE4; PDE5; TNFα; Ganglion rachidien; TRPV1; Neuropathic pain; PDE4; PDE5; TNFα; Dorsal root ganglia; TRPV1; 572.4; 612.88

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APA (6th Edition):

Megat, S. (2014). Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2014STRAJ085

Chicago Manual of Style (16th Edition):

Megat, Salim. “Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors.” 2014. Doctoral Dissertation, Université de Strasbourg. Accessed June 25, 2019. http://www.theses.fr/2014STRAJ085.

MLA Handbook (7th Edition):

Megat, Salim. “Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors.” 2014. Web. 25 Jun 2019.

Vancouver:

Megat S. Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2014. [cited 2019 Jun 25]. Available from: http://www.theses.fr/2014STRAJ085.

Council of Science Editors:

Megat S. Traitement de la douleur neuropathique : des antidépresseurs aux inhibiteurs de phosphodiestérases : Treatment of neuropathic pain : from antidepressants to phosphodiesterases inhibitors. [Doctoral Dissertation]. Université de Strasbourg; 2014. Available from: http://www.theses.fr/2014STRAJ085

27. Lotteau, Sabine. Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle.

Degree: Docteur es, Physiologie, 2013, Université Claude Bernard – Lyon I

Le canal cationique TRPV1 (Transient Receptor Potential Vanilloid 1) est activé par la capsaïcine, une acidose, de fortes températures ainsi que par les anesthésiques volatils… (more)

Subjects/Keywords: Canaux calciques; Muscle squelettique; Canaux TRPV1; Hyperthermie maligne; Calcium intracellulaire; Calcium channels; Skeletal muscle; TRPV1 channel; Malignant hyperthermia; Intracellular calcium; 573.75

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lotteau, S. (2013). Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2013LYO10167

Chicago Manual of Style (16th Edition):

Lotteau, Sabine. “Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle.” 2013. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed June 25, 2019. http://www.theses.fr/2013LYO10167.

MLA Handbook (7th Edition):

Lotteau, Sabine. “Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle.” 2013. Web. 25 Jun 2019.

Vancouver:

Lotteau S. Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2013. [cited 2019 Jun 25]. Available from: http://www.theses.fr/2013LYO10167.

Council of Science Editors:

Lotteau S. Caractérisation et implication du canal cationique TRPV1 dans la physiopathologie du muscle strié squelettique : Characterisation and implication of TRPV1 cationic channel in physiopathology of skeletal muscle. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2013. Available from: http://www.theses.fr/2013LYO10167


National University of Ireland – Galway

28. Madasu, Manish Kumar. Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey .

Degree: 2016, National University of Ireland – Galway

 The ability to experience pain is essential for survival, and to prevent potential tissue damage upon exposure to noxious stimuli. The periaqueductal grey - rostral… (more)

Subjects/Keywords: Pain; Negative affect; TRPV1; Medicine; Pharmacology and Therapeutics

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APA (6th Edition):

Madasu, M. K. (2016). Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/6034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Madasu, Manish Kumar. “Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey .” 2016. Thesis, National University of Ireland – Galway. Accessed June 25, 2019. http://hdl.handle.net/10379/6034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Madasu, Manish Kumar. “Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey .” 2016. Web. 25 Jun 2019.

Vancouver:

Madasu MK. Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey . [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/10379/6034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Madasu MK. Genotype-dependent responsivity to inflammatory pain: the role of TRPV1 in the periaqueductal grey . [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/6034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

29. Yuen, Nathaniel. Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia .

Degree: Physiology, 2012, Queens University

 The environment in which animals are maintained is a factor that has the potential to alter the physiologic phenotype. We addressed the hypothesis that the… (more)

Subjects/Keywords: Animal Care Environment; Murine; Cage Change; Hypoxia; TRPV1

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APA (6th Edition):

Yuen, N. (2012). Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yuen, Nathaniel. “Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia .” 2012. Thesis, Queens University. Accessed June 25, 2019. http://hdl.handle.net/1974/7433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yuen, Nathaniel. “Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia .” 2012. Web. 25 Jun 2019.

Vancouver:

Yuen N. Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia . [Internet] [Thesis]. Queens University; 2012. [cited 2019 Jun 25]. Available from: http://hdl.handle.net/1974/7433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yuen N. Murine Phenotype Analyses and the Role of TRPV1 in Hypoxia . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. DelloStritto, Daniel Justin. TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease.

Degree: PhD, Integrated Pharmaceutical Medicine, 2016, NEOMED Integrated Pharmaceutical Medicine

 Diabetes is a growing epidemic worldwide leading to an increase in cardiovascular morbidity and mortality. The maintenance of coronary blood flow (CBF) is crucial to… (more)

Subjects/Keywords: Biology; Physiology; TRPV1, Diabetes, ROS, 4-HNE, Hydrogen Peroxide, Differential Regulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DelloStritto, D. J. (2016). TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease. (Doctoral Dissertation). NEOMED Integrated Pharmaceutical Medicine. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1469567446

Chicago Manual of Style (16th Edition):

DelloStritto, Daniel Justin. “TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease.” 2016. Doctoral Dissertation, NEOMED Integrated Pharmaceutical Medicine. Accessed June 25, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1469567446.

MLA Handbook (7th Edition):

DelloStritto, Daniel Justin. “TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease.” 2016. Web. 25 Jun 2019.

Vancouver:

DelloStritto DJ. TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease. [Internet] [Doctoral dissertation]. NEOMED Integrated Pharmaceutical Medicine; 2016. [cited 2019 Jun 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1469567446.

Council of Science Editors:

DelloStritto DJ. TRPing up the Balance of Oxidative Stress - Transient Receptor Potential Vanilloid 1’s Role in Diabetic Microvascular Disease. [Doctoral Dissertation]. NEOMED Integrated Pharmaceutical Medicine; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1469567446

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