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You searched for subject:(TOR Serine Threonine Kinases). Showing records 1 – 30 of 1782 total matches.

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University of Texas Southwestern Medical Center

1. Zaman, Aubhishek. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.

Degree: 2017, University of Texas Southwestern Medical Center

 Monomeric RALGTPases, via direct binding to the exocyst (a.k.a Sec6/8 complex), help mount productive cell autonomous responses to trophic and immunogenic signals. However, RAL-exocyst downstream… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Signal Transduction; TOR Serine-Threonine Kinases

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APA (6th Edition):

Zaman, A. (2017). Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Web. 09 Jul 2020.

Vancouver:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Shang, Libin. Mechanistic Studies of Autophagy Initiation in Mammalian Cells.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 Macroautophagy (herein referred to as autophagy) is an evolutionarily conserved self-digestive process cells use to adapt to starvation and other stresses. During autophagy, portions of… (more)

Subjects/Keywords: TOR-Serine-Threonine Kinases; Protein-Serine-Threonine Kinases; Autophagy

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APA (6th Edition):

Shang, L. (2011). Mechanistic Studies of Autophagy Initiation in Mammalian Cells. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/886

Chicago Manual of Style (16th Edition):

Shang, Libin. “Mechanistic Studies of Autophagy Initiation in Mammalian Cells.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/886.

MLA Handbook (7th Edition):

Shang, Libin. “Mechanistic Studies of Autophagy Initiation in Mammalian Cells.” 2011. Web. 09 Jul 2020.

Vancouver:

Shang L. Mechanistic Studies of Autophagy Initiation in Mammalian Cells. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/886.

Council of Science Editors:

Shang L. Mechanistic Studies of Autophagy Initiation in Mammalian Cells. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/886


University of Texas Southwestern Medical Center

3. Britt, Rebecca. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.

Degree: 2014, University of Texas Southwestern Medical Center

 Lung cancer continues to be the leading cause of cancer related death in both men and women. Pre-clinical studies of targeted therapies are needed in… (more)

Subjects/Keywords: Autophagy; Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; TOR Serine-Threonine Kinases

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APA (6th Edition):

Britt, R. (2014). Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Britt, Rebecca. “Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Britt, Rebecca. “Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.” 2014. Web. 09 Jul 2020.

Vancouver:

Britt R. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Britt R. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Ghosh, Anwesha. Studies on Cellular Nutrient Responses and Protein Degradation.

Degree: 2015, University of Texas Southwestern Medical Center

 I have worked on two projects. The first project investigates mechanisms involved in cellular responses to amino acids. Amino-acid abundance promotes protein synthesis and cell… (more)

Subjects/Keywords: Autophagy; Intercellular Signaling Peptides and Proteins; TOR Serine-Threonine Kinases; Ubiquitin-Protein Ligases

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APA (6th Edition):

Ghosh, A. (2015). Studies on Cellular Nutrient Responses and Protein Degradation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghosh, Anwesha. “Studies on Cellular Nutrient Responses and Protein Degradation.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghosh, Anwesha. “Studies on Cellular Nutrient Responses and Protein Degradation.” 2015. Web. 09 Jul 2020.

Vancouver:

Ghosh A. Studies on Cellular Nutrient Responses and Protein Degradation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghosh A. Studies on Cellular Nutrient Responses and Protein Degradation. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

5. Nutter, Jennifer Makenzie. Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth.

Degree: MS, Biomedical Sciences, 2014, East Carolina University

 Prostate cancer is currently the second highest leading cause of cancer death in men. Notch is a transmembrane receptor protein that is part of a… (more)

Subjects/Keywords: Oncology;

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APA (6th Edition):

Nutter, J. M. (2014). Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4564

Chicago Manual of Style (16th Edition):

Nutter, Jennifer Makenzie. “Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth.” 2014. Masters Thesis, East Carolina University. Accessed July 09, 2020. http://hdl.handle.net/10342/4564.

MLA Handbook (7th Edition):

Nutter, Jennifer Makenzie. “Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth.” 2014. Web. 09 Jul 2020.

Vancouver:

Nutter JM. Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth. [Internet] [Masters thesis]. East Carolina University; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/10342/4564.

Council of Science Editors:

Nutter JM. Interrelated role of Notch signaling and mTORC pathways in prostate cancer cell survival and growth. [Masters Thesis]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4564

6. Lucas de Lima Carvalho. InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano.

Degree: Master, 2016, Universidade Federal do Ceará

 INTRODUÃÃO. O cÃncer colorretal (CCR) Ã uma das neoplasias mais prevalentes em todo o mundo, sendo uma das principais causas de Ãbito por cÃncer. No… (more)

Subjects/Keywords: FARMACOLOGIA; Mucosite; Serina-Treonina Quinases TOR; Sirolimo; Everolimo; Celulas de Paneth; Mucositis; TOR Serine-Threonine Kinases; Sirolimus; Everolimus; Paneth Cells

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APA (6th Edition):

Carvalho, L. d. L. (2016). InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=18610 ;

Chicago Manual of Style (16th Edition):

Carvalho, Lucas de Lima. “InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano.” 2016. Masters Thesis, Universidade Federal do Ceará. Accessed July 09, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=18610 ;.

MLA Handbook (7th Edition):

Carvalho, Lucas de Lima. “InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano.” 2016. Web. 09 Jul 2020.

Vancouver:

Carvalho LdL. InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano. [Internet] [Masters thesis]. Universidade Federal do Ceará 2016. [cited 2020 Jul 09]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=18610 ;.

Council of Science Editors:

Carvalho LdL. InibiÃÃo do mTOR agrava a mucosite intestinal induzida por irinotecano. [Masters Thesis]. Universidade Federal do Ceará 2016. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=18610 ;


University of Texas Southwestern Medical Center

7. Cooper, Jonathan Mark. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).

Degree: 2016, University of Texas Southwestern Medical Center

 Oncogenic mutation of Ras or Ras effector signaling characterizes roughly thirty percent of all cancers. Persistent obstacles to the treatment of these diseases by direct… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; Mesoderm; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; Small Molecule Libraries; TOR Serine-Threonine Kinases

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APA (6th Edition):

Cooper, J. M. (2016). Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cooper, Jonathan Mark. “Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cooper, Jonathan Mark. “Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).” 2016. Web. 09 Jul 2020.

Vancouver:

Cooper JM. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cooper JM. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Driscoll, David R. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular, Cell and Cancer Biology Department, 2016, U of Massachusetts : Med

  Pancreatic ductal adenocarcinoma (PDAC), the most common form of pancreatic cancer, develops through progression of premalignant pancreatic intraepithelial neoplasias (PanINs). In mouse-models, KRAS-activation in… (more)

Subjects/Keywords: Pancreatic Ductal Carcinoma; Multiprotein Complexes; TOR Serine-Threonine Kinases; Proto-Oncogene Proteins p21(ras); Cancer Biology; Neoplasms

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APA (6th Edition):

Driscoll, D. R. (2016). The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/821

Chicago Manual of Style (16th Edition):

Driscoll, David R. “The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 09, 2020. http://escholarship.umassmed.edu/gsbs_diss/821.

MLA Handbook (7th Edition):

Driscoll, David R. “The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.” 2016. Web. 09 Jul 2020.

Vancouver:

Driscoll DR. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Jul 09]. Available from: http://escholarship.umassmed.edu/gsbs_diss/821.

Council of Science Editors:

Driscoll DR. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/821


University of Texas Southwestern Medical Center

9. Wong, Sze. Regulation of Human Telomerase Alternative Splicing.

Degree: 2014, University of Texas Southwestern Medical Center

 Telomerase adds TTAGGG repeats onto chromosome ends (telomeres). Since telomerase is expressed in ~90% of all human cancer cells while being absent in most somatic… (more)

Subjects/Keywords: Alternative Splicing; Protein-Serine-Threonine Kinases; Telomerase

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APA (6th Edition):

Wong, S. (2014). Regulation of Human Telomerase Alternative Splicing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3574

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Sze. “Regulation of Human Telomerase Alternative Splicing.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3574.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Sze. “Regulation of Human Telomerase Alternative Splicing.” 2014. Web. 09 Jul 2020.

Vancouver:

Wong S. Regulation of Human Telomerase Alternative Splicing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3574.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong S. Regulation of Human Telomerase Alternative Splicing. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3574

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Estrada, Armando, III 1980-. Regulation of Cell Migration by WNK1.

Degree: 2012, University of Texas Southwestern Medical Center

 Cell motility is an immensely complex process that involves reorganization of the cytoskeleton, and consequent membrane deformation, triggered by a variety of motogenic stimuli, including… (more)

Subjects/Keywords: Cell Movement; Cytoskeleton; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Estrada, Armando, I. 1. (2012). Regulation of Cell Migration by WNK1. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Web. 09 Jul 2020.

Vancouver:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Wedin, Kyle Edward. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 With no lysine (WNK) kinases are a family of protein kinases characterized by unusual kinase domain architecture. These large proteins, divergent outside of a kinase… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Hypertension; Transcription Factors

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APA (6th Edition):

Wedin, K. E. (2011). With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/892

Chicago Manual of Style (16th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/892.

MLA Handbook (7th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Web. 09 Jul 2020.

Vancouver:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/892.

Council of Science Editors:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/892

12. Venkatesh, Aditya. Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation.

Degree: Interdisciplinary Graduate Program, Ophthalmology, 2016, U of Massachusetts : Med

  Retinitis Pigmentosa (RP) is an inherited photoreceptor degenerative disease that leads to blindness and affects about 1 in 4000 people worldwide. The disease is… (more)

Subjects/Keywords: Autophagy; Retinal Cone Photoreceptor Cells; Retinal Rod Photoreceptor Cells; Retinitis Pigmentosa; TOR Serine-Threonine Kinases; Cellular and Molecular Physiology; Eye Diseases; Ophthalmology

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APA (6th Edition):

Venkatesh, A. (2016). Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/822

Chicago Manual of Style (16th Edition):

Venkatesh, Aditya. “Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 09, 2020. http://escholarship.umassmed.edu/gsbs_diss/822.

MLA Handbook (7th Edition):

Venkatesh, Aditya. “Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation.” 2016. Web. 09 Jul 2020.

Vancouver:

Venkatesh A. Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Jul 09]. Available from: http://escholarship.umassmed.edu/gsbs_diss/822.

Council of Science Editors:

Venkatesh A. Activation of mTORC1 Improves Cone Cell Metabolism and Extends Vision in Retinitis Pigmentosa Mice: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/822

13. Hung, Chien-Min. mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2016, U of Massachusetts : Med

  Recent studies suggest adipose tissue plays a critical role in regulating whole body energy homeostasis in both animals and humans. In particular, activating brown… (more)

Subjects/Keywords: Brown Adipose Tissue; Multiprotein Complexes; TOR Serine-Threonine Kinases; Brown Adipocytes; Lipogenesis; Thermogenesis; Forkhead Box Protein O1; mTORC2; Biochemistry; Cellular and Molecular Physiology; Molecular Biology

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APA (6th Edition):

Hung, C. (2016). mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/845

Chicago Manual of Style (16th Edition):

Hung, Chien-Min. “mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 09, 2020. http://escholarship.umassmed.edu/gsbs_diss/845.

MLA Handbook (7th Edition):

Hung, Chien-Min. “mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation.” 2016. Web. 09 Jul 2020.

Vancouver:

Hung C. mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Jul 09]. Available from: http://escholarship.umassmed.edu/gsbs_diss/845.

Council of Science Editors:

Hung C. mTORC2 Promotes Lipid Storage and Suppresses Thermogenesis in Brown Adipose Tissue in Part Through AKT-Independent Regulation of FoxO1: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/845


University of Texas Southwestern Medical Center

14. Moon, Thomas Matthew. Autoinhibition and Chloride Sensing in the WNK1 Kinase.

Degree: PhD, Molecular Biophysics, 2012, University of Texas Southwestern Medical Center

 Protein kinases control diverse cellular pathways and have are the subject of intensive study regarding how they maintain specificity toward sub- strates. The research presented… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Crystallography, X-Ray; Protein Kinases Inhibitors

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APA (6th Edition):

Moon, T. M. (2012). Autoinhibition and Chloride Sensing in the WNK1 Kinase. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1001

Chicago Manual of Style (16th Edition):

Moon, Thomas Matthew. “Autoinhibition and Chloride Sensing in the WNK1 Kinase.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1001.

MLA Handbook (7th Edition):

Moon, Thomas Matthew. “Autoinhibition and Chloride Sensing in the WNK1 Kinase.” 2012. Web. 09 Jul 2020.

Vancouver:

Moon TM. Autoinhibition and Chloride Sensing in the WNK1 Kinase. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1001.

Council of Science Editors:

Moon TM. Autoinhibition and Chloride Sensing in the WNK1 Kinase. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1001


University of Texas Southwestern Medical Center

15. Tu, Szu-Wei. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.

Degree: 2013, University of Texas Southwestern Medical Center

The file named "TU-DISSERTATION-2013.pdf" is the primary dissertation file. Twelve (12) supplemental videos are also provided (in Audio Video Interleaved format).

With No Lysine (WNK)… (more)

Subjects/Keywords: Cytokinesis; Mitosis; Protein-Serine-Threonine Kinases; Spindle Apparatus

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APA (6th Edition):

Tu, S. (2013). Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tu, Szu-Wei. “Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tu, Szu-Wei. “Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.” 2013. Web. 09 Jul 2020.

Vancouver:

Tu S. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tu S. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Yoon, Jimok. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.

Degree: 2013, University of Texas Southwestern Medical Center

 The mechanisms that regulate the cellular behaviors including morphology, motility, navigation, and connectivity that are critical for normal human health are still incompletely understood. These… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Transcription Factors; DNA-Binding Proteins

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APA (6th Edition):

Yoon, J. (2013). Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoon, Jimok. “Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoon, Jimok. “Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.” 2013. Web. 09 Jul 2020.

Vancouver:

Yoon J. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoon J. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Shi, Qing. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 Hedgehog (Hh) signaling is essential for both embryonic development and adult tissue homeostasis. Malfunction of Hh signaling pathway causes many human disorders including birth defects… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Drosophila Proteins; Hedgehog Proteins

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APA (6th Edition):

Shi, Q. (2012). Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1037

Chicago Manual of Style (16th Edition):

Shi, Qing. “Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1037.

MLA Handbook (7th Edition):

Shi, Qing. “Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.” 2012. Web. 09 Jul 2020.

Vancouver:

Shi Q. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1037.

Council of Science Editors:

Shi Q. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1037


University of Texas Southwestern Medical Center

18. Jacob, Leni Susan. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 The Hedgehog (Hh) and Wnt signal transduction pathways are master regulators of embryogenesis and tissue renewal and represent anticancer therapeutic targets. Using genome-wide RNA interference… (more)

Subjects/Keywords: Signal Transduction; Protein-Serine-Threonine Kinases; Wnt Proteins

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APA (6th Edition):

Jacob, L. S. (2011). Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/884

Chicago Manual of Style (16th Edition):

Jacob, Leni Susan. “Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/884.

MLA Handbook (7th Edition):

Jacob, Leni Susan. “Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.” 2011. Web. 09 Jul 2020.

Vancouver:

Jacob LS. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/884.

Council of Science Editors:

Jacob LS. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/884


University of Texas Southwestern Medical Center

19. Yue, Tao. Studies of the Hippo Signaling Pathway.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 How multicellular organisms control their growth to reach proper organ size during development is a fascinating question. Recent studies, initially from Drosophila, have identified the… (more)

Subjects/Keywords: Tumor Suppressor Proteins; Protein-Serine-Threonine Kinases; Drosophila melanogaster

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APA (6th Edition):

Yue, T. (2012). Studies of the Hippo Signaling Pathway. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1104

Chicago Manual of Style (16th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1104.

MLA Handbook (7th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Web. 09 Jul 2020.

Vancouver:

Yue T. Studies of the Hippo Signaling Pathway. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1104.

Council of Science Editors:

Yue T. Studies of the Hippo Signaling Pathway. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1104


University of Texas Southwestern Medical Center

20. Cheng, Chih-Jen. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.

Degree: PhD, Integrative Biology, 2013, University of Texas Southwestern Medical Center

 Potassium homeostasis is one of the most sophisticated processes involving multiple organs in mammals. Many physiological functions, such as excitability of muscles and neurons, rely… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Potassium Channels, Inwardly Rectifying; Kidney

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APA (6th Edition):

Cheng, C. (2013). Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1234

Chicago Manual of Style (16th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1234.

MLA Handbook (7th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Web. 09 Jul 2020.

Vancouver:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1234.

Council of Science Editors:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1234


University of Texas Southwestern Medical Center

21. Taylor, Clinton A., IV. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

 Protein-protein interactions are essential for nearly every cellular process. Within signaling pathways, such interactions carry out numerous functions such as defining substrate specificity, inhibition of… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinase Kinases; Protein-Serine-Threonine Kinases; Signal Transduction; Transcription Factors

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APA (6th Edition):

Taylor, Clinton A., I. (2016). Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Web. 09 Jul 2020.

Vancouver:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Louis dit Picard, Hélène. Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis.

Degree: Docteur es, Génétique, 2014, Université Paris Descartes – Paris V

L’Hypertension Hyperkaliémique Familiale (HHF) est une forme rare d’hypertension associée à une hyperkaliémie et une acidose métabolique hyperchlorémique, très sensible aux diurétiques thiazidiques. Les premières… (more)

Subjects/Keywords: Hypertension; KLHL3; Séquençage d’exome entier; Transport ionique; Serine/thréonine kinases; Hypertension; KLHL3; Whole exome sequencing; Ion transport; Serine/threonine kinases; 576.5

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APA (6th Edition):

Louis dit Picard, H. (2014). Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T040

Chicago Manual of Style (16th Edition):

Louis dit Picard, Hélène. “Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed July 09, 2020. http://www.theses.fr/2014PA05T040.

MLA Handbook (7th Edition):

Louis dit Picard, Hélène. “Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis.” 2014. Web. 09 Jul 2020.

Vancouver:

Louis dit Picard H. Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2020 Jul 09]. Available from: http://www.theses.fr/2014PA05T040.

Council of Science Editors:

Louis dit Picard H. Hypertension hyperkaliémique familiale : découverte de nouveaux gènes et analyses physiopathologiques : Familial hyperkalemic hypertension : highlight of new genes and physiopathological analysis. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T040


Jawaharlal Nehru University

23. Tiwari, Divya. Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -.

Degree: Immunology, 2009, Jawaharlal Nehru University

None

Bibliography p.161-170 Appendicies p.157-159

Advisors/Committee Members: Nandicoori, Vinay Kumar.

Subjects/Keywords: pathogen; Elucidating; tuberculosis; serine; threonine kinases

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APA (6th Edition):

Tiwari, D. (2009). Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/14721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tiwari, Divya. “Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -.” 2009. Thesis, Jawaharlal Nehru University. Accessed July 09, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/14721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tiwari, Divya. “Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -.” 2009. Web. 09 Jul 2020.

Vancouver:

Tiwari D. Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -. [Internet] [Thesis]. Jawaharlal Nehru University; 2009. [cited 2020 Jul 09]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tiwari D. Elucidating the role of M tuberculosis serine/ threonine kinases in the pathogen and its survival in the host; -. [Thesis]. Jawaharlal Nehru University; 2009. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Canova, Marc. Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis.

Degree: Docteur es, Biochimie, 2009, Université Claude Bernard – Lyon I

Le séquençage intégral du génome de Mycobacterium tuberculosis a permis de mettre en évidence l’existence de onze Sérine/Thréonine Protéine-Kinases (STPKs) chez cette bactérie. Bien que… (more)

Subjects/Keywords: Mycobacterium tuberculosis; Phosphorylation; Sérine/thréonine protéine-kinase; Mycobacterium tuberculosis; Phosphorylation; Serine/threonine proteine-kinases

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APA (6th Edition):

Canova, M. (2009). Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2009LYO10130

Chicago Manual of Style (16th Edition):

Canova, Marc. “Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis.” 2009. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed July 09, 2020. http://www.theses.fr/2009LYO10130.

MLA Handbook (7th Edition):

Canova, Marc. “Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis.” 2009. Web. 09 Jul 2020.

Vancouver:

Canova M. Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2009. [cited 2020 Jul 09]. Available from: http://www.theses.fr/2009LYO10130.

Council of Science Editors:

Canova M. Caractéristion de nouveaux substrats des sérine - thréonine protéine-kinases de mycobacterium tuberculosis : Caracterisation of new substrats of serine - threonine proteine-kinases of mycobacterium tuberculosis. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2009. Available from: http://www.theses.fr/2009LYO10130


University of Texas Southwestern Medical Center

25. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 09 Jul 2020.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

26. Peña, Christopher George. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer of the uterus is a common malignancy in women with no adequate treatments for tumors that have progressed beyond the uterus. The serine-threonine kinase… (more)

Subjects/Keywords: Adenocarcinoma; Chemokine CCL2; Endometrial Neoplasms; Macrophages; Neoplasm Proteins; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Peña, C. G. (2015). LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peña, Christopher George. “LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peña, Christopher George. “LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.” 2015. Web. 09 Jul 2020.

Vancouver:

Peña CG. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peña CG. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

27. Flowers, Ebony Michelle. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.

Degree: 2018, University of Texas Southwestern Medical Center

 Polycystic kidney disease is a hereditary disorder characterized by the progressive manifestation of numerous fluid-filled sacs, known as cysts, within the renal epithelia. The enlargement… (more)

Subjects/Keywords: Glutamine; Kidney; Polycystic Kidney Diseases; Protein-Serine-Threonine Kinases; TRPP Cation Channels

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APA (6th Edition):

Flowers, E. M. (2018). Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Web. 09 Jul 2020.

Vancouver:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

28. Öncel, Dilhan. Analysis of Aurora B Regulation and Signaling.

Degree: 2006, University of Texas Southwestern Medical Center

 Aurora B is a serine/threonine kinase that functions in a complex with two other chromosomal passenger proteins called INCENP and Survivin. Its function is implicated… (more)

Subjects/Keywords: Mitosis; Protein-Serine-Threonine Kinases; Cytokinesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Öncel, D. (2006). Analysis of Aurora B Regulation and Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Öncel, Dilhan. “Analysis of Aurora B Regulation and Signaling.” 2006. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Öncel, Dilhan. “Analysis of Aurora B Regulation and Signaling.” 2006. Web. 09 Jul 2020.

Vancouver:

Öncel D. Analysis of Aurora B Regulation and Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2006. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Öncel D. Analysis of Aurora B Regulation and Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2006. Available from: http://hdl.handle.net/2152.5/591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. Ren, Fangfang. The Hippo Signaling Pathway in Organ Size Control and Regeneraton.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 The Hippo (Hpo) signaling pathway controls cell growth, proliferation and apoptosis in both Drosophila and vertebrates. Our lab has previously demonstrated that Hpo signaling regulates… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; 14-3-3 Proteins; Intracellular Signaling Peptides and Proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ren, F. (2012). The Hippo Signaling Pathway in Organ Size Control and Regeneraton. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1005

Chicago Manual of Style (16th Edition):

Ren, Fangfang. “The Hippo Signaling Pathway in Organ Size Control and Regeneraton.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1005.

MLA Handbook (7th Edition):

Ren, Fangfang. “The Hippo Signaling Pathway in Organ Size Control and Regeneraton.” 2012. Web. 09 Jul 2020.

Vancouver:

Ren F. The Hippo Signaling Pathway in Organ Size Control and Regeneraton. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1005.

Council of Science Editors:

Ren F. The Hippo Signaling Pathway in Organ Size Control and Regeneraton. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1005


University of Texas Southwestern Medical Center

30. Lee, Byung-Hoon. Identification of Substrates and Pathways Regulated by WNK1.

Degree: PhD, 2004, University of Texas Southwestern Medical Center

 WNK (With No lysine (K)), a serine/threonine protein kinase, is a unique molecule not belonging to any other canonical protein kinase family including mitogen-activated protein… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Mutagenesis; Hypertension

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, B. (2004). Identification of Substrates and Pathways Regulated by WNK1. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/599

Chicago Manual of Style (16th Edition):

Lee, Byung-Hoon. “Identification of Substrates and Pathways Regulated by WNK1.” 2004. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/599.

MLA Handbook (7th Edition):

Lee, Byung-Hoon. “Identification of Substrates and Pathways Regulated by WNK1.” 2004. Web. 09 Jul 2020.

Vancouver:

Lee B. Identification of Substrates and Pathways Regulated by WNK1. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2004. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/599.

Council of Science Editors:

Lee B. Identification of Substrates and Pathways Regulated by WNK1. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2004. Available from: http://hdl.handle.net/2152.5/599

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