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You searched for subject:(TNF). Showing records 1 – 30 of 736 total matches.

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1. 藤原, 範雄. 腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: TNF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

藤原, . (n.d.). 腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/5235

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

藤原, 範雄. “腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed April 09, 2020. http://hdl.handle.net/10061/5235.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

藤原, 範雄. “腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ.” Web. 09 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

藤原 . 腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10061/5235.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

藤原 . 腫瘍壊死因子産生阻害剤の合成と構造活性相関および薬理作用に関する研究 : Synthesis, pharmacological evaluation and SAR study of novel tumor necrosis factor-alpha inhibitors; シュヨウ エシ インシ サンセイ ソガイザイ ノ ゴウセイ ト コウゾウ カッセイ ソウカン オヨビ ヤクリ サヨウ ニ カンスル ケンキュウ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/5235

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

2. Sousa, Cláudia Emanuele Carvalho de. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.

Degree: Mestrado, Fisiologia Geral, 2011, University of São Paulo

O TNF atua diretamente sobre a pineal inibindo a via biossintética da melatonina. De forma semelhante o LPS, potente ativador da resposta imune inata, tem… (more)

Subjects/Keywords: Glândula pineal; NFKB; NFKB; Pineal gland; TNF; TNF; TNF-R1; TNF-R1

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APA (6th Edition):

Sousa, C. E. C. d. (2011). Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;

Chicago Manual of Style (16th Edition):

Sousa, Cláudia Emanuele Carvalho de. “Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.” 2011. Masters Thesis, University of São Paulo. Accessed April 09, 2020. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;.

MLA Handbook (7th Edition):

Sousa, Cláudia Emanuele Carvalho de. “Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.” 2011. Web. 09 Apr 2020.

Vancouver:

Sousa CECd. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2020 Apr 09]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;.

Council of Science Editors:

Sousa CECd. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;


University of Louisville

3. Lewis, Robert K., 1977-. Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling.

Degree: PhD, 2010, University of Louisville

 The fundamental question of the exact nature of the role played by TNF in the failing myocardium remains one of contention. Many preclinical studies have… (more)

Subjects/Keywords: TNF; Heart failure

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lewis, Robert K., 1. (2010). Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/820 ; https://ir.library.louisville.edu/etd/820

Chicago Manual of Style (16th Edition):

Lewis, Robert K., 1977-. “Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling.” 2010. Doctoral Dissertation, University of Louisville. Accessed April 09, 2020. 10.18297/etd/820 ; https://ir.library.louisville.edu/etd/820.

MLA Handbook (7th Edition):

Lewis, Robert K., 1977-. “Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling.” 2010. Web. 09 Apr 2020.

Vancouver:

Lewis, Robert K. 1. Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling. [Internet] [Doctoral dissertation]. University of Louisville; 2010. [cited 2020 Apr 09]. Available from: 10.18297/etd/820 ; https://ir.library.louisville.edu/etd/820.

Council of Science Editors:

Lewis, Robert K. 1. Inflammatory cell tumor necrosis factor signaling modulates post-infarction left ventricular remodeling. [Doctoral Dissertation]. University of Louisville; 2010. Available from: 10.18297/etd/820 ; https://ir.library.louisville.edu/etd/820


University of Illinois – Chicago

4. Lee, Wan-Ju. Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults.

Degree: 2016, University of Illinois – Chicago

 This dissertation examined use of tumor necrosis factor-alpha inhibitors (TNFI) and risk of TNFI-associated serious infection compared to oral immunosuppressants in children and young adults… (more)

Subjects/Keywords: TNF inhibitors; infections

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APA (6th Edition):

Lee, W. (2016). Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Wan-Ju. “Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults.” 2016. Thesis, University of Illinois – Chicago. Accessed April 09, 2020. http://hdl.handle.net/10027/21614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Wan-Ju. “Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults.” 2016. Web. 09 Apr 2020.

Vancouver:

Lee W. Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10027/21614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee W. Utilization and Risk of Serious Infection Associated with TNF-α Inhibitors in Children and Young Adults. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

5. Srivastava, Nutan. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.

Degree: MS, Department of Medicine, 2015, University of Alberta

 Neutrophils are highly abundant innate immune cells that are important in immediate responses to injury and infection, and secrete the proinflammatory cytokine tumor necrosis factor… (more)

Subjects/Keywords: Neutrophil; Recycling endosomes; TNF-alpha

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APA (6th Edition):

Srivastava, N. (2015). NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/mp48sg31c

Chicago Manual of Style (16th Edition):

Srivastava, Nutan. “NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.” 2015. Masters Thesis, University of Alberta. Accessed April 09, 2020. https://era.library.ualberta.ca/files/mp48sg31c.

MLA Handbook (7th Edition):

Srivastava, Nutan. “NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.” 2015. Web. 09 Apr 2020.

Vancouver:

Srivastava N. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. [Internet] [Masters thesis]. University of Alberta; 2015. [cited 2020 Apr 09]. Available from: https://era.library.ualberta.ca/files/mp48sg31c.

Council of Science Editors:

Srivastava N. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. [Masters Thesis]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/mp48sg31c


Universidade do Rio Grande do Sul

6. Jardim, Fernanda Rafaela. Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B.

Degree: 2008, Universidade do Rio Grande do Sul

 Fibrose hepática é caracterizada pelo acúmulo de matriz extracelular fibrótica, cuja presença danifica as funções do fígado. Células estreladas hepáticas (HSCs) participam ativamente deste processo,… (more)

Subjects/Keywords: Adenosina; TNF-alfa; Cirrose hepática

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APA (6th Edition):

Jardim, F. R. (2008). Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/14831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jardim, Fernanda Rafaela. “Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B.” 2008. Thesis, Universidade do Rio Grande do Sul. Accessed April 09, 2020. http://hdl.handle.net/10183/14831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jardim, Fernanda Rafaela. “Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B.” 2008. Web. 09 Apr 2020.

Vancouver:

Jardim FR. Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2008. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10183/14831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jardim FR. Ação da adenosina extracelular sobre uma linhagem de célula estrelada hepática tratada com TNF-[alfa] : papel do receptor A2B. [Thesis]. Universidade do Rio Grande do Sul; 2008. Available from: http://hdl.handle.net/10183/14831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

7. Chen, Tony (1981 - ). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.

Degree: PhD, 2011, University of Rochester

 Injuries to articular cartilage are debilitating and typically require surgical intervention due to the tissue’s inability to self-repair. Current surgical options for focal cartilage defects… (more)

Subjects/Keywords: Collagen alignment; Chondrocyte; TNF-&alpha

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APA (6th Edition):

Chen, T. (. -. ). (2011). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14143

Chicago Manual of Style (16th Edition):

Chen, Tony (1981 - ). “The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.” 2011. Doctoral Dissertation, University of Rochester. Accessed April 09, 2020. http://hdl.handle.net/1802/14143.

MLA Handbook (7th Edition):

Chen, Tony (1981 - ). “The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.” 2011. Web. 09 Apr 2020.

Vancouver:

Chen T(-). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1802/14143.

Council of Science Editors:

Chen T(-). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14143


North Carolina State University

8. Harris, Virginia Gail. The intracellular targeting of cPLA2.

Degree: MS, Microbiology, 2002, North Carolina State University

 Tumor necrosis factor-α (TNF) is an inflammatory cytokine that can induce apoptosis in virus-infected cells, susceptible tumor cells, and cells whose transcriptional or translational processes… (more)

Subjects/Keywords: cPLA2; TNF

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APA (6th Edition):

Harris, V. G. (2002). The intracellular targeting of cPLA2. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/1243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harris, Virginia Gail. “The intracellular targeting of cPLA2.” 2002. Thesis, North Carolina State University. Accessed April 09, 2020. http://www.lib.ncsu.edu/resolver/1840.16/1243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harris, Virginia Gail. “The intracellular targeting of cPLA2.” 2002. Web. 09 Apr 2020.

Vancouver:

Harris VG. The intracellular targeting of cPLA2. [Internet] [Thesis]. North Carolina State University; 2002. [cited 2020 Apr 09]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/1243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harris VG. The intracellular targeting of cPLA2. [Thesis]. North Carolina State University; 2002. Available from: http://www.lib.ncsu.edu/resolver/1840.16/1243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. 조, 영숙. 악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화.

Degree: 2009, Ajou University

"cytokine은 신호 전달 단백의 하나로 이 분자들은 호르몬처럼 세포 간 정보 전달에 광범위하게 이용된다. TNF-α는 염증과 관련되어 급성 반응을 나타내며 IL-6은 특정 미생물 물질에 반응하여… (more)

Subjects/Keywords: 악관절장애; TNF-a; IL-6

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APA (6th Edition):

조, . (2009). 악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1782 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

조, 영숙. “악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화.” 2009. Thesis, Ajou University. Accessed April 09, 2020. http://repository.ajou.ac.kr/handle/201003/1782 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

조, 영숙. “악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화.” 2009. Web. 09 Apr 2020.

Vancouver:

조 . 악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화. [Internet] [Thesis]. Ajou University; 2009. [cited 2020 Apr 09]. Available from: http://repository.ajou.ac.kr/handle/201003/1782 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

조 . 악관절 질환에게서 추출한 관절낭액의 proinflammatory cytokine의 농도변화. [Thesis]. Ajou University; 2009. Available from: http://repository.ajou.ac.kr/handle/201003/1782 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo Health Science Campus

10. Saunders, Rudel Anton. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.

Degree: PhD, College of Medicine, 2010, University of Toledo Health Science Campus

  Phospholipid transfer protein (PLTP) is critically important for reverse cholesteroltransport (RCT), and its expression level and activity increase when mice are fed a highfat… (more)

Subjects/Keywords: Biology; PLTP; NFkB; TNF; Prieurianin

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APA (6th Edition):

Saunders, R. A. (2010). Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805

Chicago Manual of Style (16th Edition):

Saunders, Rudel Anton. “Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.” 2010. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed April 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805.

MLA Handbook (7th Edition):

Saunders, Rudel Anton. “Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.” 2010. Web. 09 Apr 2020.

Vancouver:

Saunders RA. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2010. [cited 2020 Apr 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805.

Council of Science Editors:

Saunders RA. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805


University of Debrecen

11. Nemes, Olívia. Tumor nekrózis faktor-alfa (TNF-α) szerepe .

Degree: DE – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, University of Debrecen

 A TNF-α egy gyulladást keltő citokin, amelynek kettő formája ismert, egy szolubilis és egy membrán kötött. Részt vesznek a fertőzés és a daganatok elleni védekezésben,… (more)

Subjects/Keywords: TNF; TNF-R1; TNF-R2; TNF-R signaling

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APA (6th Edition):

Nemes, O. (n.d.). Tumor nekrózis faktor-alfa (TNF-α) szerepe . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/211769

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nemes, Olívia. “Tumor nekrózis faktor-alfa (TNF-α) szerepe .” Thesis, University of Debrecen. Accessed April 09, 2020. http://hdl.handle.net/2437/211769.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nemes, Olívia. “Tumor nekrózis faktor-alfa (TNF-α) szerepe .” Web. 09 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Nemes O. Tumor nekrózis faktor-alfa (TNF-α) szerepe . [Internet] [Thesis]. University of Debrecen; [cited 2020 Apr 09]. Available from: http://hdl.handle.net/2437/211769.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Nemes O. Tumor nekrózis faktor-alfa (TNF-α) szerepe . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/211769

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Debrecen

12. Hajdu, Dorottya Zsuzsanna. Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása .

Degree: DE – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2014, University of Debrecen

 Célom volt az atípusos antipszichotikumok hatásának vizsgálata humán monocita-makrofág differenciálódásra. Kísérleteim során használt gyógyszerek a Quetiapin, Aripiprasol, Ziprasidon, Risperidon, Clozapin és Olanzapin, a Haloperidol antipszichotikumok… (more)

Subjects/Keywords: monocita; differenciálódás; TNF-alfa

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hajdu, D. Z. (2014). Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/191161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hajdu, Dorottya Zsuzsanna. “Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása .” 2014. Thesis, University of Debrecen. Accessed April 09, 2020. http://hdl.handle.net/2437/191161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hajdu, Dorottya Zsuzsanna. “Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása .” 2014. Web. 09 Apr 2020.

Vancouver:

Hajdu DZ. Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása . [Internet] [Thesis]. University of Debrecen; 2014. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/2437/191161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hajdu DZ. Az antipszichotikumok makrofágok funkcionális aktivitására gyakorolt hatása . [Thesis]. University of Debrecen; 2014. Available from: http://hdl.handle.net/2437/191161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Silva, Mariana Machado da. Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1.

Degree: Mestrado, Enfermagem Fundamental, 2008, University of São Paulo

Apesar de causar um enorme impacto na história da evolução e prognóstico a partir da infecção pelo HIV, a terapia anti-retroviral altamente potente prolongada apresenta… (more)

Subjects/Keywords: HIV; HIV; Lipodystrophy Syndrome; Microsatellites; Microssatélites; Síndrome da Lipodistrofia; TNF; TNF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva, M. M. d. (2008). Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/22/22132/tde-27052008-144002/ ;

Chicago Manual of Style (16th Edition):

Silva, Mariana Machado da. “Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1.” 2008. Masters Thesis, University of São Paulo. Accessed April 09, 2020. http://www.teses.usp.br/teses/disponiveis/22/22132/tde-27052008-144002/ ;.

MLA Handbook (7th Edition):

Silva, Mariana Machado da. “Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1.” 2008. Web. 09 Apr 2020.

Vancouver:

Silva MMd. Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Apr 09]. Available from: http://www.teses.usp.br/teses/disponiveis/22/22132/tde-27052008-144002/ ;.

Council of Science Editors:

Silva MMd. Polimorfismo da região do fator de necrose tumoral (TNF) na síndrome da lipodistrofia associada à terapia anti-retroviral em portadores do HIV-1. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/22/22132/tde-27052008-144002/ ;

14. Kouris, Anargyros. Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Psoriasis is one of the most common immune-mediated chronic inflammatory skin diseases, in which proinflammatory cytokines, such as, TNF-α, IL-17, IL-1β, IL-6 and IL-22 play… (more)

Subjects/Keywords: Ψωράση; TNF-α; Μονοκύτταρα; Psoriasis; Monocytes; TNF-α

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APA (6th Edition):

Kouris, A. (2014). Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kouris, Anargyros. “Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 09, 2020. http://hdl.handle.net/10442/hedi/42257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kouris, Anargyros. “Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση.” 2014. Web. 09 Apr 2020.

Vancouver:

Kouris A. Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10442/hedi/42257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kouris A. Ψωρίαση: διερεύνηση μεταβολών των Τ-λεμφοκυττάρων στην ανοσολογική διαταραχή των ασθενών με ψωρίαση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/42257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Γεωργιάδου, Μαρία. Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Tumor necrosis factor (TNF), a pleiotropic cytokine involved in a broad spectrum of inflammatory and immune responses, exerts its biological effects by engaging two distinct… (more)

Subjects/Keywords: Ηπατοκυτταρικό καρκίνωμα; Οκτρεοτίδη; Υποδοχείς TNF; Απόπτωση; Hepatocellular carcinoma; Kupffer cells; Octreotide; TNF; TNF receptors; p65; Apoptosis

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APA (6th Edition):

Γεωργιάδου, . . (2011). Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed April 09, 2020. http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Web. 09 Apr 2020.

Vancouver:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

16. Ilić, Branislav B., 1981-. Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje.

Degree: Stomatološki fakultet, 2016, Univerzitet u Beogradu

oralna hirurgija - onkogenetika / oral surgery - oncogenetic

Uvod: Odontogeni keratocistični tumor (OKT) i oralni planocelularni karcinom (OPK) pripadaju grupi oralnih epitelnih tumora. Iako… (more)

Subjects/Keywords: oral epithelial tumors; KCOT; OSCC; gene polymorphisms; MTHFR; GST; TNF α; TNF α R1; TNF α R2

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APA (6th Edition):

Ilić, Branislav B., 1. (2016). Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:13271/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ilić, Branislav B., 1981-. “Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje.” 2016. Thesis, Univerzitet u Beogradu. Accessed April 09, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:13271/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ilić, Branislav B., 1981-. “Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje.” 2016. Web. 09 Apr 2020.

Vancouver:

Ilić, Branislav B. 1. Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2020 Apr 09]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13271/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ilić, Branislav B. 1. Ispitivanje polimorfizama gena za metilentetrahidrofolat reduktazu, glutation transferazu, faktor nekroze tumora alfa i njegove receptore u epitelnim tumorima usne duplje. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13271/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

17. Pozniak, Paul Daniel. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.

Degree: PhD, 2016, Temple University

Biomedical Neuroscience

The use of highly active antiretroviral therapy (HAART) has significantly decreased the mortality rate of HIV-1 patients, however the increased survival has led… (more)

Subjects/Keywords: Neurosciences;

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APA (6th Edition):

Pozniak, P. D. (2016). TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,386002

Chicago Manual of Style (16th Edition):

Pozniak, Paul Daniel. “TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.” 2016. Doctoral Dissertation, Temple University. Accessed April 09, 2020. http://digital.library.temple.edu/u?/p245801coll10,386002.

MLA Handbook (7th Edition):

Pozniak, Paul Daniel. “TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.” 2016. Web. 09 Apr 2020.

Vancouver:

Pozniak PD. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Apr 09]. Available from: http://digital.library.temple.edu/u?/p245801coll10,386002.

Council of Science Editors:

Pozniak PD. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,386002


Universidade do Rio Grande do Norte

18. Carvalho, Fabiana Maria Coimbra de. Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica .

Degree: 2016, Universidade do Rio Grande do Norte

 Trypsin inhibitors are studied in a variety of models for their anti-obesity and anti-inflammatory bioactive properties. Our group has previously demonstrated the satietogenic effect of… (more)

Subjects/Keywords: Obesidade; Dieta de cafeteria; TNF-α; Glicemia

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APA (6th Edition):

Carvalho, F. M. C. d. (2016). Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica . (Masters Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/21674

Chicago Manual of Style (16th Edition):

Carvalho, Fabiana Maria Coimbra de. “Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica .” 2016. Masters Thesis, Universidade do Rio Grande do Norte. Accessed April 09, 2020. http://repositorio.ufrn.br/handle/123456789/21674.

MLA Handbook (7th Edition):

Carvalho, Fabiana Maria Coimbra de. “Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica .” 2016. Web. 09 Apr 2020.

Vancouver:

Carvalho FMCd. Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica . [Internet] [Masters thesis]. Universidade do Rio Grande do Norte; 2016. [cited 2020 Apr 09]. Available from: http://repositorio.ufrn.br/handle/123456789/21674.

Council of Science Editors:

Carvalho FMCd. Efeito anti-inflamatório de proteínas sacietogênicas da semente de tamarindo em modelo experimental de síndrome metabólica . [Masters Thesis]. Universidade do Rio Grande do Norte; 2016. Available from: http://repositorio.ufrn.br/handle/123456789/21674


University of Rochester

19. Janelsins, Michelle Christine; Bowers, William J. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.

Degree: PhD, 2009, University of Rochester

 Alzheimer’s disease (AD) is a complex neurodegenerative disorder that progressively impairs intellectual and emotional functioning in afflicted individuals. The disease is characterized pathologically by a… (more)

Subjects/Keywords: AD mouse model; Neuroinflammation; Alzheimer's TNF-α

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APA (6th Edition):

Janelsins, Michelle Christine; Bowers, W. J. (2009). Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6616

Chicago Manual of Style (16th Edition):

Janelsins, Michelle Christine; Bowers, William J. “Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 09, 2020. http://hdl.handle.net/1802/6616.

MLA Handbook (7th Edition):

Janelsins, Michelle Christine; Bowers, William J. “Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.” 2009. Web. 09 Apr 2020.

Vancouver:

Janelsins, Michelle Christine; Bowers WJ. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1802/6616.

Council of Science Editors:

Janelsins, Michelle Christine; Bowers WJ. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6616


Boston University

20. Bhambhani, Vijeta. Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells.

Degree: MS, Medical Sciences, 2015, Boston University

 Two types mechanisms that control gene expression involve cis-regulatory factors and trans-regulatory factors. Cis-acting regulatory RNAs include targeted messenger RNA (mRNA) specificity and AU-rich elements… (more)

Subjects/Keywords: Biology; eIF5B; FXR1; miRNA; TNF-alpha; Translation

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APA (6th Edition):

Bhambhani, V. (2015). Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16241

Chicago Manual of Style (16th Edition):

Bhambhani, Vijeta. “Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells.” 2015. Masters Thesis, Boston University. Accessed April 09, 2020. http://hdl.handle.net/2144/16241.

MLA Handbook (7th Edition):

Bhambhani, Vijeta. “Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells.” 2015. Web. 09 Apr 2020.

Vancouver:

Bhambhani V. Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/2144/16241.

Council of Science Editors:

Bhambhani V. Micro-RNA mediated regulation of a cytokine factor: TNF-alpha: an exploration of gene expression control in proliferating and quiescent cells. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16241


University of Manchester

21. Mercer, Louise. Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis.

Degree: PhD, 2013, University of Manchester

 AimThe broad aim of this thesis was to explore the risk of malignancy in people with rheumatoid arthritis (RA), treated with anti-tumour necrosis factor (TNF)… (more)

Subjects/Keywords: 616.7; Rheumatoid arthritis; Anti-TNF; Malignancy

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APA (6th Edition):

Mercer, L. (2013). Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/safety-of-longterm-antitnf-use-with-respect-to-malignancy-in-a-national-cohort-of-people-with-rheumatoid-arthritis(be2ba2c3-a9a2-4a23-a3fb-c5688e990f93).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.568614

Chicago Manual of Style (16th Edition):

Mercer, Louise. “Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis.” 2013. Doctoral Dissertation, University of Manchester. Accessed April 09, 2020. https://www.research.manchester.ac.uk/portal/en/theses/safety-of-longterm-antitnf-use-with-respect-to-malignancy-in-a-national-cohort-of-people-with-rheumatoid-arthritis(be2ba2c3-a9a2-4a23-a3fb-c5688e990f93).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.568614.

MLA Handbook (7th Edition):

Mercer, Louise. “Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis.” 2013. Web. 09 Apr 2020.

Vancouver:

Mercer L. Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2020 Apr 09]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/safety-of-longterm-antitnf-use-with-respect-to-malignancy-in-a-national-cohort-of-people-with-rheumatoid-arthritis(be2ba2c3-a9a2-4a23-a3fb-c5688e990f93).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.568614.

Council of Science Editors:

Mercer L. Safety of long-term anti-TNF use, with respect to malignancy, in a national cohort of people with rheumatoid arthritis. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/safety-of-longterm-antitnf-use-with-respect-to-malignancy-in-a-national-cohort-of-people-with-rheumatoid-arthritis(be2ba2c3-a9a2-4a23-a3fb-c5688e990f93).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.568614


Louisiana State University

22. Wang, Yanning. TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes.

Degree: MS, Human Ecology, 2009, Louisiana State University

 Chronic inflammation is involved in the adipose tissue dysfunction through regulation of endocrine and storage function of adipocytes. As a representative proinflammatory cytokine, TNF-α was… (more)

Subjects/Keywords: C/EBP; PPAR-gamma; TNF-alpha; Adiponectin

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APA (6th Edition):

Wang, Y. (2009). TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes. (Masters Thesis). Louisiana State University. Retrieved from etd-07012009-181133 ; https://digitalcommons.lsu.edu/gradschool_theses/3684

Chicago Manual of Style (16th Edition):

Wang, Yanning. “TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes.” 2009. Masters Thesis, Louisiana State University. Accessed April 09, 2020. etd-07012009-181133 ; https://digitalcommons.lsu.edu/gradschool_theses/3684.

MLA Handbook (7th Edition):

Wang, Yanning. “TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes.” 2009. Web. 09 Apr 2020.

Vancouver:

Wang Y. TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes. [Internet] [Masters thesis]. Louisiana State University; 2009. [cited 2020 Apr 09]. Available from: etd-07012009-181133 ; https://digitalcommons.lsu.edu/gradschool_theses/3684.

Council of Science Editors:

Wang Y. TNF-alpha inhibition of adiponectin expression by targeting PPAR-gamma and C/EBP in adipocytes. [Masters Thesis]. Louisiana State University; 2009. Available from: etd-07012009-181133 ; https://digitalcommons.lsu.edu/gradschool_theses/3684


Universidade Nova

23. Raposeiro, Rita Maria Mendes. Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy.

Degree: 2014, Universidade Nova

 Ankylosing spondylitis (AS) is a chronic inflammatory rheumatic disease of the axial skeleton. The major outcome of this disease is defined by new bone formation,… (more)

Subjects/Keywords: Ankylosing spondylitis; Inflammation; Osteoclastogenesis; TNF-blocking therapy

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APA (6th Edition):

Raposeiro, R. M. M. (2014). Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raposeiro, Rita Maria Mendes. “Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy.” 2014. Thesis, Universidade Nova. Accessed April 09, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raposeiro, Rita Maria Mendes. “Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy.” 2014. Web. 09 Apr 2020.

Vancouver:

Raposeiro RMM. Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy. [Internet] [Thesis]. Universidade Nova; 2014. [cited 2020 Apr 09]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raposeiro RMM. Osteoclast activity in ankylosing spondylitis patients before and after TNF-blocking therapy. [Thesis]. Universidade Nova; 2014. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori, Yukio. Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat.

Degree: 2018, Nagoya University / 名古屋大学

Objectives: The aim of this study was to identify the molecular mechanisms underlying high-fat and high-cholesterol (HFC) diet-induced steatohepatitis and associated liver fibrosis progression in… (more)

Subjects/Keywords: Steatohepatitis; Inflammation; TNF-α; NF-κb; Fibrosis

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APA (6th Edition):

Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori, Y. (2018). Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/16803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori, Yukio. “Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed April 09, 2020. http://hdl.handle.net/2237/16803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori, Yukio. “Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat.” 2018. Web. 09 Apr 2020.

Vancouver:

Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori Y. Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/2237/16803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moriya, Takashi; Kitamori, Kazuya; Naito, Hisao; Yanagiba, Yukie; Ito, Yuki; Yamagishi, Nozomi; Tamada, Hazuki; Jia, Xiaofang; Tsuchikura, Satoru; Ikeda, Katsumi; Yamori Y. Simultaneous changes in high-fat and high-cholesterol diet-induced steatohepatitis and severe fibrosis and those underlying molecular mechanisms in novel SHRSP5/Dmcr rat. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/16803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Bazin, Thomas. Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα.

Degree: Docteur es, Microbiologie-immunologie, 2018, Bordeaux

L’interface hôte/microbiote intestinal est un système d’interactions complexes dont le déséquilibre est associé au développement des maladies inflammatoires chroniques. Les traitements anti-TNFα sont très efficaces… (more)

Subjects/Keywords: Microbiote intestinal; Maladies inflammatoire chronique intestinale; Spondyloarthrite; Inflammation; Anti TNF-Alpha; TNF-Alpha; Intestinal microbiota; Inflammatory bowel disease; Spondyloarthritis; Inflammation; TNF-Alpha inhibitor; TNF-Alpha

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bazin, T. (2018). Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2018BORD0285

Chicago Manual of Style (16th Edition):

Bazin, Thomas. “Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα.” 2018. Doctoral Dissertation, Bordeaux. Accessed April 09, 2020. http://www.theses.fr/2018BORD0285.

MLA Handbook (7th Edition):

Bazin, Thomas. “Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα.” 2018. Web. 09 Apr 2020.

Vancouver:

Bazin T. Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα. [Internet] [Doctoral dissertation]. Bordeaux; 2018. [cited 2020 Apr 09]. Available from: http://www.theses.fr/2018BORD0285.

Council of Science Editors:

Bazin T. Microbiote intestinal et inflammation : prédiction de la réponse aux anti-TNFα dans les maladies inflammatoires chroniques et modulation de la croissance bactérienne in vitro en réponse au TNFα : Gut microbiota and inflammation : prediction of anti-TNFα response in chronic inflammatory diseases and modulation of bacterial growth in vitro in response to TNFα. [Doctoral Dissertation]. Bordeaux; 2018. Available from: http://www.theses.fr/2018BORD0285

26. Teixeira, Chantelle Simões Leite. TNF-alfa e metabolismo do adipócito.

Degree: 2015, RCAAP

Dissertação para obtenção do grau de Mestre no Instituto Superior de Ciências da Saúde Egas Moniz

A prevalência da obesidade tem vindo a aumentar ao… (more)

Subjects/Keywords: Obesidade; Tecido adiposo; TNF-α; Adipócito

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APA (6th Edition):

Teixeira, C. S. L. (2015). TNF-alfa e metabolismo do adipócito. (Thesis). RCAAP. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:comum.rcaap.pt:10400.26/10984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teixeira, Chantelle Simões Leite. “TNF-alfa e metabolismo do adipócito.” 2015. Thesis, RCAAP. Accessed April 09, 2020. https://www.rcaap.pt/detail.jsp?id=oai:comum.rcaap.pt:10400.26/10984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teixeira, Chantelle Simões Leite. “TNF-alfa e metabolismo do adipócito.” 2015. Web. 09 Apr 2020.

Vancouver:

Teixeira CSL. TNF-alfa e metabolismo do adipócito. [Internet] [Thesis]. RCAAP; 2015. [cited 2020 Apr 09]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:comum.rcaap.pt:10400.26/10984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teixeira CSL. TNF-alfa e metabolismo do adipócito. [Thesis]. RCAAP; 2015. Available from: https://www.rcaap.pt/detail.jsp?id=oai:comum.rcaap.pt:10400.26/10984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oslo

27. Ben Omar, Sakina. Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk.

Degree: 2008, University of Oslo

 Aterosklerose er en inflammatorisk sykdom der immuncellenes aktivitet er sentral i patogenesen. I blodåreveggen utvikles aterosklerotiske plakk som består av ekstracellulært materiale og lipidrike makrofag-skumceller,… (more)

Subjects/Keywords: oksidert LDL OXLDL TNF-aTNFalpha; VDP::568

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APA (6th Edition):

Ben Omar, S. (2008). Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk. (Thesis). University of Oslo. Retrieved from http://urn.nb.no/URN:NBN:no-21459 ; https://www.duo.uio.no/handle/10852/12236 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/12236/1/BenOmar_masteroppgave.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ben Omar, Sakina. “Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk.” 2008. Thesis, University of Oslo. Accessed April 09, 2020. http://urn.nb.no/URN:NBN:no-21459 ; https://www.duo.uio.no/handle/10852/12236 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/12236/1/BenOmar_masteroppgave.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ben Omar, Sakina. “Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk.” 2008. Web. 09 Apr 2020.

Vancouver:

Ben Omar S. Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk. [Internet] [Thesis]. University of Oslo; 2008. [cited 2020 Apr 09]. Available from: http://urn.nb.no/URN:NBN:no-21459 ; https://www.duo.uio.no/handle/10852/12236 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/12236/1/BenOmar_masteroppgave.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ben Omar S. Cysteinproteasene legumain og cathepsin B i monocytter/makrofager og i aterosklerotiske plakk. [Thesis]. University of Oslo; 2008. Available from: http://urn.nb.no/URN:NBN:no-21459 ; https://www.duo.uio.no/handle/10852/12236 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/12236/1/BenOmar_masteroppgave.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Bumdelger, Batmunkh. Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導.

Degree: 博士(医学), 2014, Hiroshima University / 広島大学

 Abdominal aortic aneurysm (AAA) is known to develop mainly by the increased diameter of aorta through metalloproteinases (MMPs). Although activities of MMPs are tightly regulated… (more)

Subjects/Keywords: Abdominal aortic aneurysm; Mmp9; Timp1; TNF-α

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APA (6th Edition):

Bumdelger, B. (2014). Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導. (Thesis). Hiroshima University / 広島大学. Retrieved from http://ir.lib.hiroshima-u.ac.jp/00036323

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bumdelger, Batmunkh. “Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導.” 2014. Thesis, Hiroshima University / 広島大学. Accessed April 09, 2020. http://ir.lib.hiroshima-u.ac.jp/00036323.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bumdelger, Batmunkh. “Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導.” 2014. Web. 09 Apr 2020.

Vancouver:

Bumdelger B. Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導. [Internet] [Thesis]. Hiroshima University / 広島大学; 2014. [cited 2020 Apr 09]. Available from: http://ir.lib.hiroshima-u.ac.jp/00036323.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bumdelger B. Induction of Timp1 in Smooth Muscle Cells during Development of Abdominal Aortic Aneurysms : 腹部大動脈瘤形成における、平滑筋細胞でのTimp1遺伝子誘導. [Thesis]. Hiroshima University / 広島大学; 2014. Available from: http://ir.lib.hiroshima-u.ac.jp/00036323

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

29. Poluga, Jasmina L., 1963-. Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije.

Degree: Medicinski fakultet, 2013, Univerzitet u Beogradu

Infektivne bolesti - malarija / Infectious diseases - malaria

REZIME: Malarija je multisistemska, potencijalno letalna bolest uzrokovana parazitima roda Plasmodium. Dokazano je da kod malarije… (more)

Subjects/Keywords: malaria; cytokines; parasitemia; thrombocytopenia; TNF- ; IL-6

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APA (6th Edition):

Poluga, Jasmina L., 1. (2013). Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:5550/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Poluga, Jasmina L., 1963-. “Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije.” 2013. Thesis, Univerzitet u Beogradu. Accessed April 09, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:5550/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Poluga, Jasmina L., 1963-. “Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije.” 2013. Web. 09 Apr 2020.

Vancouver:

Poluga, Jasmina L. 1. Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije. [Internet] [Thesis]. Univerzitet u Beogradu; 2013. [cited 2020 Apr 09]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:5550/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Poluga, Jasmina L. 1. Odnos koncentracije proinflamatornih citokina u serumu i kliničkih, parazitoloških i hematoloških promena u toku importovane malarije. [Thesis]. Univerzitet u Beogradu; 2013. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:5550/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

30. Aurand, Thomas. Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers .

Degree: 2016, Cornell University

 The US Centers for Disease Control and Prevention (CDC) estimates chronic diseases account for 86% of the annual health care costs in the US. Monitoring… (more)

Subjects/Keywords: Synthetic Biology; IFN-gamma; TNF-alpha

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APA (6th Edition):

Aurand, T. (2016). Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/44390

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aurand, Thomas. “Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers .” 2016. Thesis, Cornell University. Accessed April 09, 2020. http://hdl.handle.net/1813/44390.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aurand, Thomas. “Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers .” 2016. Web. 09 Apr 2020.

Vancouver:

Aurand T. Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers . [Internet] [Thesis]. Cornell University; 2016. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1813/44390.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aurand T. Development Of Novel Genetic Circuits For The Detection Of Disease Biomarkers . [Thesis]. Cornell University; 2016. Available from: http://hdl.handle.net/1813/44390

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [25]

.