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You searched for subject:(TLR4). Showing records 1 – 30 of 125 total matches.

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NSYSU

1. Chen, Pei-hsuan. Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4.

Degree: Master, Biological Sciences, 2008, NSYSU

 Tumor necrosis factor-alpha (TNF-α) is a potent proinflammatory cytokine, inducing the acute-phase response that leads to physiological changes that serve to eliminate the infecting organisms.… (more)

Subjects/Keywords: iNOS; TLR4

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APA (6th Edition):

Chen, P. (2008). Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902108-125407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Pei-hsuan. “Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4.” 2008. Thesis, NSYSU. Accessed August 24, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902108-125407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Pei-hsuan. “Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4.” 2008. Web. 24 Aug 2019.

Vancouver:

Chen P. Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4. [Internet] [Thesis]. NSYSU; 2008. [cited 2019 Aug 24]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902108-125407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen P. Lack of TNF-α Receptor 1 Decreases Pseudomonas aeruginosa-induced Mortality in Burned Mice through Negative Regulation of Toll-like Receptor 4. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902108-125407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Mississippi State University

2. Jan, Basit Latief. INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING.

Degree: MS, Veterinary Medicine, College of, 2010, Mississippi State University

  Alcohol consumption is a significant risk-factor for mortality in patients with sepsis. This study was carried to investigate the mechanisms by which acute ethanol… (more)

Subjects/Keywords: TLR4; Ethanol

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APA (6th Edition):

Jan, B. L. (2010). INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-11122010-170231/ ;

Chicago Manual of Style (16th Edition):

Jan, Basit Latief. “INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING.” 2010. Masters Thesis, Mississippi State University. Accessed August 24, 2019. http://sun.library.msstate.edu/ETD-db/theses/available/etd-11122010-170231/ ;.

MLA Handbook (7th Edition):

Jan, Basit Latief. “INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING.” 2010. Web. 24 Aug 2019.

Vancouver:

Jan BL. INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING. [Internet] [Masters thesis]. Mississippi State University; 2010. [cited 2019 Aug 24]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-11122010-170231/ ;.

Council of Science Editors:

Jan BL. INNATE IMMUNITY AND INFLAMMATION IN SEPSIS IN A MOUSE MODEL FOR BINGE DRINKING. [Masters Thesis]. Mississippi State University; 2010. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-11122010-170231/ ;


Oklahoma State University

3. Aravind, Shruthishree. Novel Effects of Opioids on Toll-like Receptor 4.

Degree: Department of Biochemistry and Molecular Biology, 2011, Oklahoma State University

 Opioid analgesics are now known to activate both classic opioid receptors and TLR4 on glial cells. Ongoing research in this field shows the involvement of… (more)

Subjects/Keywords: opioids; tlr4

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APA (6th Edition):

Aravind, S. (2011). Novel Effects of Opioids on Toll-like Receptor 4. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/8526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aravind, Shruthishree. “Novel Effects of Opioids on Toll-like Receptor 4.” 2011. Thesis, Oklahoma State University. Accessed August 24, 2019. http://hdl.handle.net/11244/8526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aravind, Shruthishree. “Novel Effects of Opioids on Toll-like Receptor 4.” 2011. Web. 24 Aug 2019.

Vancouver:

Aravind S. Novel Effects of Opioids on Toll-like Receptor 4. [Internet] [Thesis]. Oklahoma State University; 2011. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11244/8526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aravind S. Novel Effects of Opioids on Toll-like Receptor 4. [Thesis]. Oklahoma State University; 2011. Available from: http://hdl.handle.net/11244/8526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

4. Mallard, Beth. The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis .

Degree: 2011, University of Otago

 Background: Con A administration leads to T cell-mediated hepatitis in mice, the mechanism of which involves the cytokines interferon (IFN)-γ and tumour necrosis factor (TNF)-α.… (more)

Subjects/Keywords: TLR4; hepatitis; Con A

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APA (6th Edition):

Mallard, B. (2011). The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/606

Chicago Manual of Style (16th Edition):

Mallard, Beth. “The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis .” 2011. Doctoral Dissertation, University of Otago. Accessed August 24, 2019. http://hdl.handle.net/10523/606.

MLA Handbook (7th Edition):

Mallard, Beth. “The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis .” 2011. Web. 24 Aug 2019.

Vancouver:

Mallard B. The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis . [Internet] [Doctoral dissertation]. University of Otago; 2011. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10523/606.

Council of Science Editors:

Mallard B. The role of Toll-like receptor 4 in Concanavalin A-induced immune-mediated hepatitis . [Doctoral Dissertation]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/606


University of Otago

5. Muhamad, Marlini. The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice .

Degree: 2011, University of Otago

 The process of liver regeneration after partial hepatectomy is very complex and is associated with signalling cascades involving initiation signals, transcription factors, cytokines, growth factors,… (more)

Subjects/Keywords: TLR4; LPS; liver regeneration

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APA (6th Edition):

Muhamad, M. (2011). The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/1695

Chicago Manual of Style (16th Edition):

Muhamad, Marlini. “The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice .” 2011. Doctoral Dissertation, University of Otago. Accessed August 24, 2019. http://hdl.handle.net/10523/1695.

MLA Handbook (7th Edition):

Muhamad, Marlini. “The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice .” 2011. Web. 24 Aug 2019.

Vancouver:

Muhamad M. The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice . [Internet] [Doctoral dissertation]. University of Otago; 2011. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10523/1695.

Council of Science Editors:

Muhamad M. The role of lipopolysaccharide/Toll-like Receptor 4 signalling pathway during liver regeneration after partial hepatectomy in mice . [Doctoral Dissertation]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/1695

6. 이, 태구. BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절.

Degree: 2010, Ajou University

"미세아교세포 (microglia) 는 중추신경계 (CNS) 에 상주하는 면역세포로 외부의 자극에 의해 활성화되어 면역반응과 염증반응을 유발하는 것으로 알려져 있다. 면역 반응 시 생성이 증가되는IFN-β 는 항… (more)

Subjects/Keywords: PGE2; IFN-β; LPS; TLR4

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APA (6th Edition):

이, . (2010). BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/2239 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

이, 태구. “BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절.” 2010. Thesis, Ajou University. Accessed August 24, 2019. http://repository.ajou.ac.kr/handle/201003/2239 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

이, 태구. “BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절.” 2010. Web. 24 Aug 2019.

Vancouver:

이 . BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절. [Internet] [Thesis]. Ajou University; 2010. [cited 2019 Aug 24]. Available from: http://repository.ajou.ac.kr/handle/201003/2239 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

이 . BV-2 미세아교세포에서 PGE2 에 의한 LPS 유도 IFN-β생성 조절. [Thesis]. Ajou University; 2010. Available from: http://repository.ajou.ac.kr/handle/201003/2239 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

7. Henry, Rebecca Jane. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection.

Degree: 2015, National University of Ireland – Galway

Toll like receptors (TLRs) are key players in host defence, homeostasis and response to injury. However, uncontrolled and aberrant TLR activation has been proposed to… (more)

Subjects/Keywords: Endocannabinoid; Neuroinflammation; TLR3; TLR4; Physiology

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APA (6th Edition):

Henry, R. J. (2015). Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection. (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection.” 2015. Thesis, National University of Ireland – Galway. Accessed August 24, 2019. http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection.” 2015. Web. 24 Aug 2019.

Vancouver:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection. [Internet] [Thesis]. National University of Ireland – Galway; 2015. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection. [Thesis]. National University of Ireland – Galway; 2015. Available from: http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. 稲村, 幸雄. Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走.

Degree: 博士(医学), 2017, Nagasaki University / 長崎大学

 The toll-like receptor 4 (TLR4)-mediated immune response is considered as one of the triggers of acute respiratory distress syndrome. The agonistic monoclonal antibody UT12 specific… (more)

Subjects/Keywords: lung; TLR4; neutrophils; dendritic cells

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APA (6th Edition):

稲村, . (2017). Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/37068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

稲村, 幸雄. “Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走.” 2017. Thesis, Nagasaki University / 長崎大学. Accessed August 24, 2019. http://hdl.handle.net/10069/37068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

稲村, 幸雄. “Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走.” 2017. Web. 24 Aug 2019.

Vancouver:

稲村 . Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2017. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10069/37068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

稲村 . Recruitment of distinct immune cell populations to the lung after intratracheal TLR4 signaling activation by two different stimulations : 異なる二種類の刺激による気管内Toll様受容体4(TLR4)シグナル伝達活性化に伴う、特徴的な免疫細胞集団の肺への遊走. [Thesis]. Nagasaki University / 長崎大学; 2017. Available from: http://hdl.handle.net/10069/37068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

9. Denko, Laura Michelle. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.

Degree: MS, Human Nutrition, Foods, and Exercise, 2012, Virginia Tech

 Obesity-related metabolic derangements have been linked to toll-like receptor 4 (TLR4), an innate immune system receptor, due to its role in proinflammatory pathways. Lipopolysaccharide (LPS),… (more)

Subjects/Keywords: TLR4; endotoxin; LPS; skeletal muscle

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APA (6th Edition):

Denko, L. M. (2012). Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/76808

Chicago Manual of Style (16th Edition):

Denko, Laura Michelle. “Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.” 2012. Masters Thesis, Virginia Tech. Accessed August 24, 2019. http://hdl.handle.net/10919/76808.

MLA Handbook (7th Edition):

Denko, Laura Michelle. “Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.” 2012. Web. 24 Aug 2019.

Vancouver:

Denko LM. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. [Internet] [Masters thesis]. Virginia Tech; 2012. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10919/76808.

Council of Science Editors:

Denko LM. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. [Masters Thesis]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/76808


Baylor University

10. [No author]. Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes.

Degree: 2018, Baylor University

 Islet transplantation has emerged as an important treatment option for brittle type 1 diabetes and as an adjunct procedure after total pancreatectomy to prevent brittle… (more)

Subjects/Keywords: Islet. Transplantation. Diabetes. TLR4. Drugs.

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APA (6th Edition):

author], [. (2018). Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes. (Thesis). Baylor University. Retrieved from http://hdl.handle.net/2104/10442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes. ” 2018. Thesis, Baylor University. Accessed August 24, 2019. http://hdl.handle.net/2104/10442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes. ” 2018. Web. 24 Aug 2019.

Vancouver:

author] [. Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes. [Internet] [Thesis]. Baylor University; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2104/10442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Inhibition of TLR4 minimizes islet damage due to sterile inflammation and improves islet transplant outcomes. [Thesis]. Baylor University; 2018. Available from: http://hdl.handle.net/2104/10442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Uniwersytet im. Adama Mickiewicza w Poznaniu

11. Sikorski, Krzysztof. STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej .

Degree: 2013, Uniwersytet im. Adama Mickiewicza w Poznaniu

 Cytokina pro-zapalna interferon-gamma (IFNγ) oraz receptor Toll-podobny 4 (TLR4) współdziałają w leukocytach powodując zwiększoną odpowiedź zapalną: znaczny wzrost ekspresji chemokin i cytokin. Stawiamy hipotezę, że… (more)

Subjects/Keywords: miażdżyca; atherosclerosis; IFN-gamma; Receptor TLR4; TLR4; JAK-STAT; śródbłonek; endothelium

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APA (6th Edition):

Sikorski, K. (2013). STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej . (Doctoral Dissertation). Uniwersytet im. Adama Mickiewicza w Poznaniu. Retrieved from http://hdl.handle.net/10593/6444

Chicago Manual of Style (16th Edition):

Sikorski, Krzysztof. “STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej .” 2013. Doctoral Dissertation, Uniwersytet im. Adama Mickiewicza w Poznaniu. Accessed August 24, 2019. http://hdl.handle.net/10593/6444.

MLA Handbook (7th Edition):

Sikorski, Krzysztof. “STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej .” 2013. Web. 24 Aug 2019.

Vancouver:

Sikorski K. STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej . [Internet] [Doctoral dissertation]. Uniwersytet im. Adama Mickiewicza w Poznaniu; 2013. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10593/6444.

Council of Science Editors:

Sikorski K. STAT1 – główny czynnik regulatorowy w zapaleniu i dysfunkcji naczyniowej . [Doctoral Dissertation]. Uniwersytet im. Adama Mickiewicza w Poznaniu; 2013. Available from: http://hdl.handle.net/10593/6444

12. Oldenburg, Reid. Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens.

Degree: Docteur es, Sciences de la vie et de la santé. Immunologie, 2016, Sorbonne Paris Cité

La biosynthèse de phénol-glycolipides (PGL) par Mycobacterium tuberculosis et M. leprae favorise l’invasion des macrophages via l'interaction de la partie saccharidique des PGL avec le… (more)

Subjects/Keywords: Phénol-glycolipides mycobactériens; TLR4; TRIF; Mycobacterial phenolic glycolipids; TLR4; TRIF

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APA (6th Edition):

Oldenburg, R. (2016). Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCC234

Chicago Manual of Style (16th Edition):

Oldenburg, Reid. “Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed August 24, 2019. http://www.theses.fr/2016USPCC234.

MLA Handbook (7th Edition):

Oldenburg, Reid. “Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens.” 2016. Web. 24 Aug 2019.

Vancouver:

Oldenburg R. Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2016USPCC234.

Council of Science Editors:

Oldenburg R. Immunomodulatory properties of mycobacterial phenolic glycolipids : Propriétés immunomodulatrices des phénol-glycolipides mycobactériens. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCC234


University of Cincinnati

13. Allen, Jessica L. Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105.

Degree: PhD, Medicine : Immunobiology, 2010, University of Cincinnati

 Consistent with their important roles in both adaptive and innate immune responses, B lymphocytes express both somatically-recombined, antigen-specific receptors of the adaptive immune system and… (more)

Subjects/Keywords: Immunology; TLR4; RP105; B cells; BAFF

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APA (6th Edition):

Allen, J. L. (2010). Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275658195

Chicago Manual of Style (16th Edition):

Allen, Jessica L. “Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105.” 2010. Doctoral Dissertation, University of Cincinnati. Accessed August 24, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275658195.

MLA Handbook (7th Edition):

Allen, Jessica L. “Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105.” 2010. Web. 24 Aug 2019.

Vancouver:

Allen JL. Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105. [Internet] [Doctoral dissertation]. University of Cincinnati; 2010. [cited 2019 Aug 24]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275658195.

Council of Science Editors:

Allen JL. Inhibition by Proxy: Modulation of TLR4-driven B cell responses by RP105. [Doctoral Dissertation]. University of Cincinnati; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275658195


Universitat de Valencia

14. Alfonso Loeches, Silvia. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .

Degree: 2012, Universitat de Valencia

 El abuso de alcohol puede causar daño cerebral y en ciertos casos cursa con neurodegeneración, aunque los mecanismos moleculares de estos efectos se desconocen. El… (more)

Subjects/Keywords: ALCOHOL; DAÑO CEREBRAL; RECEPTORES TLR4; INFLAMASOMA NLRP3

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APA (6th Edition):

Alfonso Loeches, S. (2012). Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/24273

Chicago Manual of Style (16th Edition):

Alfonso Loeches, Silvia. “Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .” 2012. Doctoral Dissertation, Universitat de Valencia. Accessed August 24, 2019. http://hdl.handle.net/10550/24273.

MLA Handbook (7th Edition):

Alfonso Loeches, Silvia. “Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .” 2012. Web. 24 Aug 2019.

Vancouver:

Alfonso Loeches S. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2012. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10550/24273.

Council of Science Editors:

Alfonso Loeches S. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . [Doctoral Dissertation]. Universitat de Valencia; 2012. Available from: http://hdl.handle.net/10550/24273

15. Faiyaz, Rahiman Sharief. Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy.

Degree: 2018, NUI Galway

 Liver regeneration after partial hepatectomy (PH) is a complex process involving an inflammatory response that is followed by the proliferation of both parenchymal and nonparenchymal… (more)

Subjects/Keywords: Regeneration; Liver; TLR4; Complement; Medicine; Physiology

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APA (6th Edition):

Faiyaz, R. S. (2018). Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy. (Thesis). NUI Galway. Retrieved from http://hdl.handle.net/10379/14621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Faiyaz, Rahiman Sharief. “Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy.” 2018. Thesis, NUI Galway. Accessed August 24, 2019. http://hdl.handle.net/10379/14621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Faiyaz, Rahiman Sharief. “Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy.” 2018. Web. 24 Aug 2019.

Vancouver:

Faiyaz RS. Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy. [Internet] [Thesis]. NUI Galway; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10379/14621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Faiyaz RS. Role of LPS/TLR4 and complement signaling in liver regeneration after partial hepatectomy. [Thesis]. NUI Galway; 2018. Available from: http://hdl.handle.net/10379/14621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Uniwersytet im. Adama Mickiewicza w Poznaniu

16. Chmielewski, Stefan. Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF .

Degree: 2014, Uniwersytet im. Adama Mickiewicza w Poznaniu

 Hipoteza zakładała, że w komórkach nienależących do układu immunologicznego, takich jak komórki śródbłonka oraz mięśni gładkich, integracja szlaków sygnalizacyjnych JAK/STAT i TLR4 za pośrednictwem czynnika… (more)

Subjects/Keywords: STAT1; TLR4; IRF; miażdżyca; nadciśnienie; atherosclerosis; hypertension

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APA (6th Edition):

Chmielewski, S. (2014). Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF . (Doctoral Dissertation). Uniwersytet im. Adama Mickiewicza w Poznaniu. Retrieved from http://hdl.handle.net/10593/12307

Chicago Manual of Style (16th Edition):

Chmielewski, Stefan. “Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF .” 2014. Doctoral Dissertation, Uniwersytet im. Adama Mickiewicza w Poznaniu. Accessed August 24, 2019. http://hdl.handle.net/10593/12307.

MLA Handbook (7th Edition):

Chmielewski, Stefan. “Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF .” 2014. Web. 24 Aug 2019.

Vancouver:

Chmielewski S. Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF . [Internet] [Doctoral dissertation]. Uniwersytet im. Adama Mickiewicza w Poznaniu; 2014. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10593/12307.

Council of Science Editors:

Chmielewski S. Integracja szlaków sygnalizacyjnych interferonu gamma i receptora TLR4 w procesie zapalnym naczyń krwionośnych, warunkowana czynnikami transkrypcyjnymi STAT1 i IRF . [Doctoral Dissertation]. Uniwersytet im. Adama Mickiewicza w Poznaniu; 2014. Available from: http://hdl.handle.net/10593/12307


University of Ottawa

17. Ariana, Ardeshir. Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK .

Degree: 2017, University of Ottawa

 Necroptosis is a pathway of inflammatory cell death that is associated with several pathologies and is induced by ligation of surface TLR or cytokine receptors… (more)

Subjects/Keywords: Necroptosis; TLR4; Endocytosis; Dynamin; P38MAPK; mouse macrophages

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APA (6th Edition):

Ariana, A. (2017). Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/35968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ariana, Ardeshir. “Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK .” 2017. Thesis, University of Ottawa. Accessed August 24, 2019. http://hdl.handle.net/10393/35968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ariana, Ardeshir. “Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK .” 2017. Web. 24 Aug 2019.

Vancouver:

Ariana A. Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10393/35968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ariana A. Dissection of TLR4-Induced Necroptosis Using Specific Inhibitors of Endocytosis and P38 MAPK . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/35968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Chile

18. Rossel Salas, Eduardo David. Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll .

Degree: 2012, Universidad de Chile

 Enfermedades como la sepsis, artritis reumatoide, tendinitis, entre otras, están ligadas a respuestas pro-inflamatorias anómalas. En ellas participan citoquinas pro-inflamatorias y factores de transcripción activados… (more)

Subjects/Keywords: Receptores tipo Toll; Proteínas adaptadoras; TLR4

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APA (6th Edition):

Rossel Salas, E. D. (2012). Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll . (Thesis). Universidad de Chile. Retrieved from http://www.repositorio.uchile.cl/handle/2250/112353

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossel Salas, Eduardo David. “Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll .” 2012. Thesis, Universidad de Chile. Accessed August 24, 2019. http://www.repositorio.uchile.cl/handle/2250/112353.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossel Salas, Eduardo David. “Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll .” 2012. Web. 24 Aug 2019.

Vancouver:

Rossel Salas ED. Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll . [Internet] [Thesis]. Universidad de Chile; 2012. [cited 2019 Aug 24]. Available from: http://www.repositorio.uchile.cl/handle/2250/112353.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossel Salas ED. Modelamiento de sitios de acoplamiento e interacciones proteína-proteína en receptores tipo toll . [Thesis]. Universidad de Chile; 2012. Available from: http://www.repositorio.uchile.cl/handle/2250/112353

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Australian National University

19. Menon, Deepthi. The biological roles of glutathione transferase Omega 1 .

Degree: 2015, Australian National University

 Glutathionylation is the reversible redox modification of protein thiols by disulphide formation with glutathione. Glutathionylation can alter protein structure and activity in response to changes… (more)

Subjects/Keywords: GSTO1-1; TLR4; glutathionylation; LPS; inflammation

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APA (6th Edition):

Menon, D. (2015). The biological roles of glutathione transferase Omega 1 . (Thesis). Australian National University. Retrieved from http://hdl.handle.net/1885/14281

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Menon, Deepthi. “The biological roles of glutathione transferase Omega 1 .” 2015. Thesis, Australian National University. Accessed August 24, 2019. http://hdl.handle.net/1885/14281.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Menon, Deepthi. “The biological roles of glutathione transferase Omega 1 .” 2015. Web. 24 Aug 2019.

Vancouver:

Menon D. The biological roles of glutathione transferase Omega 1 . [Internet] [Thesis]. Australian National University; 2015. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/1885/14281.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Menon D. The biological roles of glutathione transferase Omega 1 . [Thesis]. Australian National University; 2015. Available from: http://hdl.handle.net/1885/14281

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Machado, Sanseray da Silveira Cruz. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).

Degree: Mestrado, Fisiologia Geral, 2010, University of São Paulo

O fator de transcrição nuclear kappa B (NFKB), central na resposta inflamatória, é constitutivamente expresso em glândulas pineais de rato. A inibição da translocação nuclear… (more)

Subjects/Keywords: Glândula pineal; Lipopolissacarídeo; Lipopolysaccharide; Melatonin; Melatonina; NFKB; NFKB; Pineal gland; TLR4; TLR4

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APA (6th Edition):

Machado, S. d. S. C. (2010). Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;

Chicago Manual of Style (16th Edition):

Machado, Sanseray da Silveira Cruz. “Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).” 2010. Masters Thesis, University of São Paulo. Accessed August 24, 2019. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;.

MLA Handbook (7th Edition):

Machado, Sanseray da Silveira Cruz. “Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).” 2010. Web. 24 Aug 2019.

Vancouver:

Machado SdSC. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). [Internet] [Masters thesis]. University of São Paulo; 2010. [cited 2019 Aug 24]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;.

Council of Science Editors:

Machado SdSC. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). [Masters Thesis]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;

21. Dubois, Natasha. Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models.

Degree: Docteur es, Immunologie, 2016, Paris Saclay

En 2014 la Tuberculose (TB) à dépassé le VIH comme la principale cause de décès par maladie infectieuse dans le monde soulignant le besoin urgent… (more)

Subjects/Keywords: Adjuvant; Tlr4; Th1; Lymphocytes B; Innée; Adaptative; Adjuvant; Tlr4; Th1; B cells; Innate; Adaptative

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APA (6th Edition):

Dubois, N. (2016). Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2016SACLS131

Chicago Manual of Style (16th Edition):

Dubois, Natasha. “Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models.” 2016. Doctoral Dissertation, Paris Saclay. Accessed August 24, 2019. http://www.theses.fr/2016SACLS131.

MLA Handbook (7th Edition):

Dubois, Natasha. “Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models.” 2016. Web. 24 Aug 2019.

Vancouver:

Dubois N. Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models. [Internet] [Doctoral dissertation]. Paris Saclay; 2016. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2016SACLS131.

Council of Science Editors:

Dubois N. Caractérisation de la réponse immune induite par un adjuvant comprenant un agoniste du TLR4 dans des modèles murins : Characterization of the immune response to a TLR4-based adjuvant in murine models. [Doctoral Dissertation]. Paris Saclay; 2016. Available from: http://www.theses.fr/2016SACLS131

22. Κρανιδιώτη, Χαρίκλεια. Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο.

Degree: 2010, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

We aimed to investigate if angiopoietin-2 (Ang 2) participates in the septic process and what may be the role of monocytes as a site of… (more)

Subjects/Keywords: Αγγειοποιητίνη-2; Σήψη; Σηπτικό σύνδρομο; Μονοκύτταρα; Υποδοχέας TLR4; Angiopoietin-2; Sepsis; Septic shock; Monocytes; TLR4 receptor

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APA (6th Edition):

Κρανιδιώτη, . . (2010). Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/24264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κρανιδιώτη, Χαρίκλεια. “Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο.” 2010. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed August 24, 2019. http://hdl.handle.net/10442/hedi/24264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κρανιδιώτη, Χαρίκλεια. “Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο.” 2010. Web. 24 Aug 2019.

Vancouver:

Κρανιδιώτη . Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2010. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10442/hedi/24264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κρανιδιώτη . Η σημασία της αγγειοποιητίνης-2 στο σηπτικό σύνδρομο. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2010. Available from: http://hdl.handle.net/10442/hedi/24264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Helsinki

23. Galeev, Alibek. Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective.

Degree: Department of Food and Environmental Sciences; Helsingfors universitet, Agrikultur- och forstvetenskapliga fakulteten, Institutionen för livsmedels- och miljövetenskaper, 2016, University of Helsinki

 Campylobacter species, particularly C. jejuni and C. coli, are the most common cause of human gastroenteritis worldwide. Lipooligosaccharides (LOS) are heat-stable amphiphilic glycolipids essential for… (more)

Subjects/Keywords: Campylobacter coli; TLR4; growth conditions; lipooligosaccharides; immune response; Mikrobiologi; Microbiology; Mikrobiologia; Campylobacter coli; TLR4; growth conditions; lipooligosaccharides; immune response

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APA (6th Edition):

Galeev, A. (2016). Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/163719

Chicago Manual of Style (16th Edition):

Galeev, Alibek. “Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective.” 2016. Masters Thesis, University of Helsinki. Accessed August 24, 2019. http://hdl.handle.net/10138/163719.

MLA Handbook (7th Edition):

Galeev, Alibek. “Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective.” 2016. Web. 24 Aug 2019.

Vancouver:

Galeev A. Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective. [Internet] [Masters thesis]. University of Helsinki; 2016. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10138/163719.

Council of Science Editors:

Galeev A. Evaluating the effect of different growth conditions on glycoconjugate expression in Campylobacter coli: a methodological perspective. [Masters Thesis]. University of Helsinki; 2016. Available from: http://hdl.handle.net/10138/163719

24. Merle, Nicolas. Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques.

Degree: Docteur es, Immunologie, 2017, Sorbonne Paris Cité

Le système du complément est une cascade de défense immunitaire complexe et étroitement régulée, conduisant à des dommages tissulaires lorsqu’il est suractivé. L’hème, un motif… (more)

Subjects/Keywords: Maladies hémolytiques; Hème; Système du complément; TLR4; Cellule endothéliale; Néphropathie; Hemolytic diseases; Heme; Complement system; TLR4; Endothelial cells; Nephropathy; 616.97

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APA (6th Edition):

Merle, N. (2017). Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB076

Chicago Manual of Style (16th Edition):

Merle, Nicolas. “Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed August 24, 2019. http://www.theses.fr/2017USPCB076.

MLA Handbook (7th Edition):

Merle, Nicolas. “Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques.” 2017. Web. 24 Aug 2019.

Vancouver:

Merle N. Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2017USPCB076.

Council of Science Editors:

Merle N. Mechanisms of complement activation under hemolytic conditions : Mécanismes d’activation du système du complément dans des conditions hémolytiques. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB076


Texas Medical Center

25. Park, Hyun J. EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES.

Degree: PhD, 2010, Texas Medical Center

 Vaccines which use the strategy of fusing adjuvant murine â-defensin2 (mBD2) to an antigen in order to elicit stronger anti-antigen immune responses are referred to… (more)

Subjects/Keywords: cancer vaccine; defensin beta 2; TLR4; Immunity; Immunoprophylaxis and Therapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Park, H. J. (2010). EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/15

Chicago Manual of Style (16th Edition):

Park, Hyun J. “EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed August 24, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/15.

MLA Handbook (7th Edition):

Park, Hyun J. “EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES.” 2010. Web. 24 Aug 2019.

Vancouver:

Park HJ. EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2019 Aug 24]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/15.

Council of Science Editors:

Park HJ. EFFICACY AND MECHANISM OF â-DEFENSIN2 FUSED ANTIGEN PROTEIN VACCINES. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/15


University of Colorado

26. Slivka, Peter F. A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays.

Degree: PhD, Chemistry & Biochemistry, 2012, University of Colorado

  Pathological pain is a serious health problem that is initiated and perpetuated by Toll-like Receptors (TLRs) on glial cells. Among the TLRs, Toll-like Receptor… (more)

Subjects/Keywords: Bacterial Chemotaxis; Chemosensory Array; MD2; TLR4; Biochemistry; Cell Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Slivka, P. F. (2012). A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays. (Doctoral Dissertation). University of Colorado. Retrieved from http://scholar.colorado.edu/chem_gradetds/64

Chicago Manual of Style (16th Edition):

Slivka, Peter F. “A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays.” 2012. Doctoral Dissertation, University of Colorado. Accessed August 24, 2019. http://scholar.colorado.edu/chem_gradetds/64.

MLA Handbook (7th Edition):

Slivka, Peter F. “A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays.” 2012. Web. 24 Aug 2019.

Vancouver:

Slivka PF. A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays. [Internet] [Doctoral dissertation]. University of Colorado; 2012. [cited 2019 Aug 24]. Available from: http://scholar.colorado.edu/chem_gradetds/64.

Council of Science Editors:

Slivka PF. A Constrained Peptide That Targets the TLR4/MD2 Interaction & Investigating the Mechanism of Ultra-stability in Bacterial Chemosensory Arrays. [Doctoral Dissertation]. University of Colorado; 2012. Available from: http://scholar.colorado.edu/chem_gradetds/64

27. João Felipe Mota. Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro.

Degree: 2011, Universidade Federal de São Paulo

A alta ingestão de dieta hiperlipídica induz obesidade e resistência à insulina, aspectos relacionados à inflamação crônica. A ativação do receptor toll like 4 (TLR4)… (more)

Subjects/Keywords: TLR4; Dieta hiperlipídica; Obesidade; Estresse oxidativo; NUTRICAO; Estresse inflamatório

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APA (6th Edition):

Mota, J. F. (2011). Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=2096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mota, João Felipe. “Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro.” 2011. Thesis, Universidade Federal de São Paulo. Accessed August 24, 2019. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=2096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mota, João Felipe. “Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro.” 2011. Web. 24 Aug 2019.

Vancouver:

Mota JF. Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro. [Internet] [Thesis]. Universidade Federal de São Paulo; 2011. [cited 2019 Aug 24]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=2096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mota JF. Efeito da suplementação da vitamina E (a-tocoferol) ou D3 na expressão gênica de TLR4 e adipocinas. Estudos in vivo e in vitro. [Thesis]. Universidade Federal de São Paulo; 2011. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=2096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

28. Jotić, Ana D., 1979-. Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama.

Degree: Medicinski fakultet, 2015, Univerzitet u Beogradu

Medicina / Medicine

Uvod: Receptori slični toll-u (eng. Toll like receptors, TLR) imaju bitnu ulogu u aktivaciji početnog imunskog odgovora. Svaki opisani TLR prepoznaje određene… (more)

Subjects/Keywords: Chronic otitis media; Toll-like receptor (TLR) 2; TLR4; polymorphism

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APA (6th Edition):

Jotić, Ana D., 1. (2015). Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:9763/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jotić, Ana D., 1979-. “Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama.” 2015. Thesis, Univerzitet u Beogradu. Accessed August 24, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:9763/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jotić, Ana D., 1979-. “Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama.” 2015. Web. 24 Aug 2019.

Vancouver:

Jotić, Ana D. 1. Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama. [Internet] [Thesis]. Univerzitet u Beogradu; 2015. [cited 2019 Aug 24]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:9763/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jotić, Ana D. 1. Značaj ekspresije i polimorfizama receptora sličnih Toll-u 2 i 4 u zapaljenskim oboljenjima srednjeg uva i njihovim komplikacijama. [Thesis]. Univerzitet u Beogradu; 2015. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:9763/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

29. van den Borne, P. Changing blood flow in peripheral artery disease.

Degree: 2014, Universiteit Utrecht

 Cardiovascular disease (CVD) is the leading cause of death globally and it is predicted this will remain to increase throughout 2030 to an estimated 23,3… (more)

Subjects/Keywords: Arteriogenesis; inflammation; CXCL10; CD200-CD200R; TLR4; exosomes; LTB4; atherosclerosis; AAA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

van den Borne, P. (2014). Changing blood flow in peripheral artery disease. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/294660

Chicago Manual of Style (16th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl:8080/handle/1874/294660.

MLA Handbook (7th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Web. 24 Aug 2019.

Vancouver:

van den Borne P. Changing blood flow in peripheral artery disease. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/294660.

Council of Science Editors:

van den Borne P. Changing blood flow in peripheral artery disease. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/294660

30. 蔡, 君柔. A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント.

Degree: 博士(医学), 2013, Nagasaki University / 長崎大学

 IRF family members are important transcription factors involved in TLR and RLR signalling and type I IFN expression. Many cellular factors are reportedly associated with… (more)

Subjects/Keywords: BTBD2; Interferon regulatory factors(IRFs); Toll-like Receptor 4(TLR4)

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APA (6th Edition):

蔡, . (2013). A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

蔡, 君柔. “A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント.” 2013. Thesis, Nagasaki University / 長崎大学. Accessed August 24, 2019. http://hdl.handle.net/10069/35432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

蔡, 君柔. “A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント.” 2013. Web. 24 Aug 2019.

Vancouver:

蔡 . A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2013. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10069/35432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

蔡 . A shorter variant of BTBD2 as a novel negative regulator of IRF-associated signalling : IRF-依存性シグナル伝達の抑制因子としてのBTBD2(新規)バリアント. [Thesis]. Nagasaki University / 長崎大学; 2013. Available from: http://hdl.handle.net/10069/35432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5]

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