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You searched for subject:(TGF 1). Showing records 1 – 30 of 73 total matches.

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NSYSU

1. Huang, Sz-yang. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 Pancreatic ductal adenocarcinoma (PDAC) is one of the most insidious forms of cancer whose incidence nearly equals its death rate. Despite extensive research studies, no… (more)

Subjects/Keywords: TGF; PDAC; HIF-1

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APA (6th Edition):

Huang, S. (2010). Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Thesis, NSYSU. Accessed August 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Web. 19 Aug 2019.

Vancouver:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Internet] [Thesis]. NSYSU; 2010. [cited 2019 Aug 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

2. Wu, Yi-Chen. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.

Degree: Master, Biological Sciences, 2010, NSYSU

 Hepatic injuries due to viral infection and alcoholic abuse frequently lead to activation and proliferation of hepatic stellate cells (HSCs), concomitantly with tissue repairing and… (more)

Subjects/Keywords: BMP-2; Hepatic; fibrogenesis; TGF-β1

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APA (6th Edition):

Wu, Y. (2010). Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Yi-Chen. “Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.” 2010. Thesis, NSYSU. Accessed August 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Yi-Chen. “Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.” 2010. Web. 19 Aug 2019.

Vancouver:

Wu Y. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. [Internet] [Thesis]. NSYSU; 2010. [cited 2019 Aug 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu Y. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Anastasi, Cyril. De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase.

Degree: Docteur es, Biochimie, 2016, Lyon

La Bone Morphogenetic Protein-1 (BMP-1) est une métalloprotéase impliquée dans la maturation et l'activation de nombreuses molécules extracellulaires. Parmi celles-ci, on trouve notamment les collagènes… (more)

Subjects/Keywords: BMP-1; Collagènes; TGF-ß; Bétaglycan; CD109; TSP-1; BMP-1; Collagens; TGF-ß; Betaglycan; CD109; TSP-1; 572

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APA (6th Edition):

Anastasi, C. (2016). De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1097

Chicago Manual of Style (16th Edition):

Anastasi, Cyril. “De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase.” 2016. Doctoral Dissertation, Lyon. Accessed August 19, 2019. http://www.theses.fr/2016LYSE1097.

MLA Handbook (7th Edition):

Anastasi, Cyril. “De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase.” 2016. Web. 19 Aug 2019.

Vancouver:

Anastasi C. De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2019 Aug 19]. Available from: http://www.theses.fr/2016LYSE1097.

Council of Science Editors:

Anastasi C. De la maturation des collagènes à la régulation de la signalisation TGF-ß : nouveaux rôles moléculaires et cellulaires de la métalloprotéase BMP-1 : From collagen maturation to regulation of TGF-ß signaling : novel molecular and cellular functions of the BMP-1 metalloproteinase. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1097


Universidade Federal da Bahia

4. Sandra Regina Villas Bôas de Freitas. Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano.

Degree: 2009, Universidade Federal da Bahia

 Fator de crescimento endotelial vascular (VEGF) e fator de crescimento e transformação beta (TGFβ-1) são citocinas multifuncionais que estimulam a migração, invasão e angiogênese tumoral.… (more)

Subjects/Keywords: angiogênese; flavonóides; glioblastoma; Croton betulaster; TGFβ; -1; VEGF; angiogenesis; flavonoide; glioblastoma; Croton betulaster; TGFβ; -1; VEGF; IMUNOLOGIA

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APA (6th Edition):

Freitas, S. R. V. B. d. (2009). Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano. (Thesis). Universidade Federal da Bahia. Retrieved from http://www.bibliotecadigital.ufba.br/tde_busca/arquivo.php?codArquivo=3543

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Freitas, Sandra Regina Villas Bôas de. “Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano.” 2009. Thesis, Universidade Federal da Bahia. Accessed August 19, 2019. http://www.bibliotecadigital.ufba.br/tde_busca/arquivo.php?codArquivo=3543.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Freitas, Sandra Regina Villas Bôas de. “Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano.” 2009. Web. 19 Aug 2019.

Vancouver:

Freitas SRVBd. Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano. [Internet] [Thesis]. Universidade Federal da Bahia; 2009. [cited 2019 Aug 19]. Available from: http://www.bibliotecadigital.ufba.br/tde_busca/arquivo.php?codArquivo=3543.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Freitas SRVBd. Efeito dos flavonoides de Croton betulaster Mull no controle do crescimento e regulação de citocinas pró-angiogênicas VEGF e TGFβ em célula de glioblastoma multiforme humano. [Thesis]. Universidade Federal da Bahia; 2009. Available from: http://www.bibliotecadigital.ufba.br/tde_busca/arquivo.php?codArquivo=3543

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Henaoui, Imène-Sarah. MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Nice

La Fibrose Pulmonaire idiopathique (FPI) est une maladie fibroproliférative pour laquelle il n’existe aucun traitement efficace. Les mécanismes à l’origine de cette pathologie sont méconnus… (more)

Subjects/Keywords: MicroARN; Fibrose pulmonaire idiopathique; TGF-β; Cavéoline-1; Myofibroblastes; MicroRNAs; Idiopathic pulmonary fibrosis; TGF-β; Caveolin-1; Myofibroblasts

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APA (6th Edition):

Henaoui, I. (2013). MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2013NICE4136

Chicago Manual of Style (16th Edition):

Henaoui, Imène-Sarah. “MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1.” 2013. Doctoral Dissertation, Nice. Accessed August 19, 2019. http://www.theses.fr/2013NICE4136.

MLA Handbook (7th Edition):

Henaoui, Imène-Sarah. “MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1.” 2013. Web. 19 Aug 2019.

Vancouver:

Henaoui I. MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1. [Internet] [Doctoral dissertation]. Nice; 2013. [cited 2019 Aug 19]. Available from: http://www.theses.fr/2013NICE4136.

Council of Science Editors:

Henaoui I. MiR-199a-5p, un « fibromiR » amplificateur de la voie du TGF-beta dans la fibrose pulmonaire idiopathique : MiR-199a-5p is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1. [Doctoral Dissertation]. Nice; 2013. Available from: http://www.theses.fr/2013NICE4136

6. Sandi vargas, Maria José. Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005).

Degree: Docteur es, Biologie, Spécialité Immunologie, 2011, Aix-Marseille 2

P8 est un gène lié au stress cellulaire qui a été identifié et caractérisé dans notre laboratoire. Il est surexprimé dans diverses pathologies, et plus… (more)

Subjects/Keywords: P8; Cancer du pancréas; Tgf-β; 1; Transition-épithélio-mésenchymateuse; P8; Pancreatic cancer; Tgf-β; 1; Epithelial to mesenchymal transition

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APA (6th Edition):

Sandi vargas, M. J. (2011). Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005). (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX22114

Chicago Manual of Style (16th Edition):

Sandi vargas, Maria José. “Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005).” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed August 19, 2019. http://www.theses.fr/2011AIX22114.

MLA Handbook (7th Edition):

Sandi vargas, Maria José. “Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005).” 2011. Web. 19 Aug 2019.

Vancouver:

Sandi vargas MJ. Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005). [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2019 Aug 19]. Available from: http://www.theses.fr/2011AIX22114.

Council of Science Editors:

Sandi vargas MJ. Etude du rôle de la protéine de stress p8 et son implication dans la progression tumorale et la formation de métastases dans le cancer du pancréas : Il fragile filo della vita : l’opera poetica di Mario Luzi (1914-2005). [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX22114

7. Mrad, Marguerite. Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages.

Degree: Docteur es, Biotechnologies, cancérologie, 2016, Université Toulouse III – Paul Sabatier

L'infiltration des mélanomes par les macrophages (TAM) est souvent corrélée à un mauvais pronostic. Cependant, les mécanismes qui régulent le recrutement et la fonction de… (more)

Subjects/Keywords: Inflammation; Mélanome; Macrophage; Polarisation; Sphingosine 1-phosphate; TGF beta; Inflammation; Melanoma; Macrophage; Polarization; Sphingosine 1-phosphate; TGF beta

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APA (6th Edition):

Mrad, M. (2016). Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30215

Chicago Manual of Style (16th Edition):

Mrad, Marguerite. “Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed August 19, 2019. http://www.theses.fr/2016TOU30215.

MLA Handbook (7th Edition):

Mrad, Marguerite. “Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages.” 2016. Web. 19 Aug 2019.

Vancouver:

Mrad M. Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2019 Aug 19]. Available from: http://www.theses.fr/2016TOU30215.

Council of Science Editors:

Mrad M. Etude des altérations du métabolisme de la sphingosine-1-phosphate dans le mélanome cutané : rôle sur l'infiltration et la polarisation des macrophages associés aux tumeurs : Study of the alterations of sphingosine-1-phosphate metabolism in cutaneous melanoma : role on the infiltration and polarization of tumor-associated macrophages. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30215


University of Bradford

8. Khaghani, Seyed Ali. Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins.

Degree: PhD, 2010, University of Bradford

 Articular cartilage is an avascular and flexible connective tissue found in joints. It produces a cushioning effect at the joints and provides low friction to… (more)

Subjects/Keywords: 611; Primary chondrocyte cell; Cell / Tissue engineering; Monolayer cell culture; Pellet culture; Cell marker; Wound closure assay; Integrin-?1 (CD29); TGF-?1; TGF-?2; TGF-?3; Articular cartilage; Growth factors

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APA (6th Edition):

Khaghani, S. A. (2010). Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF-β1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/5371

Chicago Manual of Style (16th Edition):

Khaghani, Seyed Ali. “Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF-β1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins.” 2010. Doctoral Dissertation, University of Bradford. Accessed August 19, 2019. http://hdl.handle.net/10454/5371.

MLA Handbook (7th Edition):

Khaghani, Seyed Ali. “Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF-β1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins.” 2010. Web. 19 Aug 2019.

Vancouver:

Khaghani SA. Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF-β1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins. [Internet] [Doctoral dissertation]. University of Bradford; 2010. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/10454/5371.

Council of Science Editors:

Khaghani SA. Cell and tissue engineering of articular cartilage via regulation and alignment of primary chondrocyte using manipulated transforming growth factors and ECM proteins : effect of transforming growth factor-beta (TGF-β1, 2 and 3) on the biological regulation and wound repair of chondrocyte monolayers with and without presence of ECM proteins. [Doctoral Dissertation]. University of Bradford; 2010. Available from: http://hdl.handle.net/10454/5371

9. Alberti, Silvia. "Study of novel pathogenic mechanism in Huntington disease".

Degree: 2014, Università degli Studi di Catania

 Huntington Disease (HD) is a neurodegenerative disorder resulting from the expansion of polyglutamine stretch in the huntingtin protein (Htt). Mutant Htt (mHtt) leads to progressive… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; TGF-beta 1, Huntington disease, macrophages

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APA (6th Edition):

Alberti, S. (2014). "Study of novel pathogenic mechanism in Huntington disease". (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alberti, Silvia. “"Study of novel pathogenic mechanism in Huntington disease".” 2014. Thesis, Università degli Studi di Catania. Accessed August 19, 2019. http://hdl.handle.net/10761/1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alberti, Silvia. “"Study of novel pathogenic mechanism in Huntington disease".” 2014. Web. 19 Aug 2019.

Vancouver:

Alberti S. "Study of novel pathogenic mechanism in Huntington disease". [Internet] [Thesis]. Università degli Studi di Catania; 2014. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/10761/1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alberti S. "Study of novel pathogenic mechanism in Huntington disease". [Thesis]. Università degli Studi di Catania; 2014. Available from: http://hdl.handle.net/10761/1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Donato, Tatiani Ayako Goto. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF1) nas vias de sinalização na biomineralização in vitro.

Degree: PhD, Biologia Celular e Tecidual, 2014, University of São Paulo

Este estudo in vitro teve como objetivo avaliar os efeitos moleculares do TGF1, com diferentes períodos de suplementação, sobre a formação do fenótipo osteogênico das… (more)

Subjects/Keywords: In vitro mineralization; Linhagem celular MC3T3-E1; MC3T3-E1 cell line; Mineralização in vitro; Neutralização de receptores de TGF-β 1; Neutralization of TGF-β 1 receptors; Noncollagenous proteins; Proteínas não colágenas; Smad3; Smad3; TGF-β 1; TGF-β 1

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APA (6th Edition):

Donato, T. A. G. (2014). Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;

Chicago Manual of Style (16th Edition):

Donato, Tatiani Ayako Goto. “Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro.” 2014. Doctoral Dissertation, University of São Paulo. Accessed August 19, 2019. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;.

MLA Handbook (7th Edition):

Donato, Tatiani Ayako Goto. “Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro.” 2014. Web. 19 Aug 2019.

Vancouver:

Donato TAG. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2019 Aug 19]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;.

Council of Science Editors:

Donato TAG. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;


NSYSU

11. SU, HUEI-YU. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 Early cancers of the pancreas often cause no symptoms leading to pancreatic cancer often can't be detected until the cancer already reach to advanced stage.… (more)

Subjects/Keywords: EMT; TGF-β1/Smad4; Nestin; Interleukin-6; PDAC

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SU, H. (2014). Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Thesis, NSYSU. Accessed August 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Web. 19 Aug 2019.

Vancouver:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2019 Aug 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo Health Science Campus

12. Nadour, Alaa M. MBG - Induced EMT.

Degree: MSBS, College of Graduate Studies, 2007, University of Toledo Health Science Campus

 Interest and research in Epithelial-mesenchymal transition are currently growing at a high level due the importance of this process in embryogenesis, organ development, tumor progression,… (more)

Subjects/Keywords: Biology, Molecular; MBG; EMT; TGF-¿¿¿¿; Epithelial; Mesenchymal; cells; LEF-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nadour, A. M. (2007). MBG - Induced EMT. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1194969320

Chicago Manual of Style (16th Edition):

Nadour, Alaa M. “MBG - Induced EMT.” 2007. Masters Thesis, University of Toledo Health Science Campus. Accessed August 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1194969320.

MLA Handbook (7th Edition):

Nadour, Alaa M. “MBG - Induced EMT.” 2007. Web. 19 Aug 2019.

Vancouver:

Nadour AM. MBG - Induced EMT. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2007. [cited 2019 Aug 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1194969320.

Council of Science Editors:

Nadour AM. MBG - Induced EMT. [Masters Thesis]. University of Toledo Health Science Campus; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1194969320


Case Western Reserve University

13. Yang, Jiayi. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.

Degree: PhD, Biochemistry, 2009, Case Western Reserve University

  Several reports from various laboratories, including ours, find that transforming growth factor-ß (TGF-ß) functions as a tumor suppressor of the prostate. Tumor suppression mediated… (more)

Subjects/Keywords: Biochemistry; Biomedical Research; Cellular Biology; Molecular Biology; TGF-¿¿¿¿/SMAD; Survivin; Smad2; Smads; PROSTATE; Smad3; TGF-¿¿¿¿1

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APA (6th Edition):

Yang, J. (2009). TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147

Chicago Manual of Style (16th Edition):

Yang, Jiayi. “TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.” 2009. Doctoral Dissertation, Case Western Reserve University. Accessed August 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147.

MLA Handbook (7th Edition):

Yang, Jiayi. “TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.” 2009. Web. 19 Aug 2019.

Vancouver:

Yang J. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2009. [cited 2019 Aug 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147.

Council of Science Editors:

Yang J. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. [Doctoral Dissertation]. Case Western Reserve University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147


The Ohio State University

14. Liu, Xiaoying. Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction.

Degree: PhD, Ohio State Biochemistry Program, 2009, The Ohio State University

  Fibrotic disorders are characterized by chronic myofibroblast activation and associated with excessive accumulation of contractile protein smooth muscle a-actin (SMaA), an early marker of… (more)

Subjects/Keywords: Biochemistry; Cellular Biology; Molecular Biology; myofibroblast, TGF&946; 1, TNF&945; , thrombin, YB-1, Egr-1, Smad, ERK

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APA (6th Edition):

Liu, X. (2009). Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1236711907

Chicago Manual of Style (16th Edition):

Liu, Xiaoying. “Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction.” 2009. Doctoral Dissertation, The Ohio State University. Accessed August 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1236711907.

MLA Handbook (7th Edition):

Liu, Xiaoying. “Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction.” 2009. Web. 19 Aug 2019.

Vancouver:

Liu X. Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2019 Aug 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236711907.

Council of Science Editors:

Liu X. Molecular mechanisms of myofibroblast differentiation and the role of TGF beta 1, TNF alpha, and thrombin signal transduction. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236711907

15. Gomes, Luciana Rodrigues. Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos.

Degree: PhD, Bioquímica, 2011, University of São Paulo

 A causa de morte da maioria das pacientes com câncer de mama se deve à doença metastática desenvolvida a partir do tumor primário. A degradação… (more)

Subjects/Keywords: Breast cancer; Câncer de mama; Cellular invasion; Expressão gênica; Gene expression; Invasão; MMPs; MMPs; RECK; RECK; TGFβ; -1; TGFβ; -1; TIMPs; TIMPs

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APA (6th Edition):

Gomes, L. R. (2011). Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/46/46131/tde-04092012-095106/ ;

Chicago Manual of Style (16th Edition):

Gomes, Luciana Rodrigues. “Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos.” 2011. Doctoral Dissertation, University of São Paulo. Accessed August 19, 2019. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-04092012-095106/ ;.

MLA Handbook (7th Edition):

Gomes, Luciana Rodrigues. “Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos.” 2011. Web. 19 Aug 2019.

Vancouver:

Gomes LR. Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2019 Aug 19]. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-04092012-095106/ ;.

Council of Science Editors:

Gomes LR. Papel de TGFβ-1 na regulação da expressão de MMPs seus inibidores (TIMPs e Reck) em modelo de carcinoma mamário humano: análise funcional de RECK e sua correlação com dados clínico-patológicos. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-04092012-095106/ ;


Universidade do Rio Grande do Norte

16. Santos, Pedro Paulo de Andrade. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .

Degree: 2012, Universidade do Rio Grande do Norte

 Myofibroblasts are cells that exhibit a hybrid phenotype, sharing the morphological characteristics of fibroblasts and smooth muscle cells, which is acquired during a process called… (more)

Subjects/Keywords: Ameloblastom, Tumor odontogênico adenomatóide; Ceratocisto odontogênico; Miofibroblasto; TGF-1; IFN-; MMP-13; EMMPRIN; imuno-histoquímica; Ameloblastoma; Adenomatoid odontogenic tumor; Odontogenic keratocyst; Myofibroblasts; TGF-1; IFN-; MMP-13; EMMPRIN; Immunohistochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Santos, P. P. d. A. (2012). Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . (Doctoral Dissertation). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/18671

Chicago Manual of Style (16th Edition):

Santos, Pedro Paulo de Andrade. “Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .” 2012. Doctoral Dissertation, Universidade do Rio Grande do Norte. Accessed August 19, 2019. http://repositorio.ufrn.br/handle/123456789/18671.

MLA Handbook (7th Edition):

Santos, Pedro Paulo de Andrade. “Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .” 2012. Web. 19 Aug 2019.

Vancouver:

Santos PPdA. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . [Internet] [Doctoral dissertation]. Universidade do Rio Grande do Norte; 2012. [cited 2019 Aug 19]. Available from: http://repositorio.ufrn.br/handle/123456789/18671.

Council of Science Editors:

Santos PPdA. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . [Doctoral Dissertation]. Universidade do Rio Grande do Norte; 2012. Available from: http://repositorio.ufrn.br/handle/123456789/18671


Universidade Federal de Santa Maria

17. Roberta Carneiro da Fontoura Pereira. AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS.

Degree: 2012, Universidade Federal de Santa Maria

O plasma rico em plaquetas (PRP) é obtido a partir do sangue total, através de uma ou duas centrifugações, resultando em um pequeno volume de… (more)

Subjects/Keywords: equinos; 1; TGF-β; concentração de plaquetas; centrifugações; Plasma rico em plaquetas; MEDICINA VETERINARIA; Platelet-rich plasma; centrifugations; pletelet concentration; TGF-β; 1; horse

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APA (6th Edition):

Pereira, R. C. d. F. (2012). AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4609

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pereira, Roberta Carneiro da Fontoura. “AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS.” 2012. Thesis, Universidade Federal de Santa Maria. Accessed August 19, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4609.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pereira, Roberta Carneiro da Fontoura. “AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS.” 2012. Web. 19 Aug 2019.

Vancouver:

Pereira RCdF. AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2012. [cited 2019 Aug 19]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4609.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pereira RCdF. AVALIAÇÃO DE SETE PROTOCOLOS DE OBTENÇÃO DE PLASMA RICO EM PLAQUETAS. [Thesis]. Universidade Federal de Santa Maria; 2012. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4609

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

18. Santos, Pedro Paulo de Andrade. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .

Degree: 2012, Universidade do Rio Grande do Norte

 Myofibroblasts are cells that exhibit a hybrid phenotype, sharing the morphological characteristics of fibroblasts and smooth muscle cells, which is acquired during a process called… (more)

Subjects/Keywords: Ameloblastom, Tumor odontogênico adenomatóide; Ceratocisto odontogênico; Miofibroblasto; TGF-1; IFN-; MMP-13; EMMPRIN; imuno-histoquímica; Ameloblastoma; Adenomatoid odontogenic tumor; Odontogenic keratocyst; Myofibroblasts; TGF-1; IFN-; MMP-13; EMMPRIN; Immunohistochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Santos, P. P. d. A. (2012). Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . (Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/18671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Pedro Paulo de Andrade. “Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .” 2012. Thesis, Universidade do Rio Grande do Norte. Accessed August 19, 2019. http://repositorio.ufrn.br/handle/123456789/18671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Pedro Paulo de Andrade. “Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais .” 2012. Web. 19 Aug 2019.

Vancouver:

Santos PPdA. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . [Internet] [Thesis]. Universidade do Rio Grande do Norte; 2012. [cited 2019 Aug 19]. Available from: http://repositorio.ufrn.br/handle/123456789/18671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos PPdA. Estudo imuno-histoquímico da presença de miofibroblastos e da expressão do fator transformador de crescimento-beta1, interferon gama, metaloproteinase de matriz 13 e indutor de metaloproteinases de matriz em lesões odontogênicas epiteliais . [Thesis]. Universidade do Rio Grande do Norte; 2012. Available from: http://repositorio.ufrn.br/handle/123456789/18671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Fisichella, Vincenzo. TGF-beta 1 pathway and age-related macular degenertion.

Degree: 2017, Università degli Studi di Catania

 Purpose: To set up a retinal degenerative model in rat that mimics pathologic conditions such as age-related macular degeneration (AMD) using amyloid-beta (Abeta) oligomers, and… (more)

Subjects/Keywords: Area 06 - Scienze mediche; amyloid-beta, age related macular degeneration, TGF beta-1, drug delivery

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APA (6th Edition):

Fisichella, V. (2017). TGF-beta 1 pathway and age-related macular degenertion. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/3870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fisichella, Vincenzo. “TGF-beta 1 pathway and age-related macular degenertion.” 2017. Thesis, Università degli Studi di Catania. Accessed August 19, 2019. http://hdl.handle.net/10761/3870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fisichella, Vincenzo. “TGF-beta 1 pathway and age-related macular degenertion.” 2017. Web. 19 Aug 2019.

Vancouver:

Fisichella V. TGF-beta 1 pathway and age-related macular degenertion. [Internet] [Thesis]. Università degli Studi di Catania; 2017. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/10761/3870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fisichella V. TGF-beta 1 pathway and age-related macular degenertion. [Thesis]. Università degli Studi di Catania; 2017. Available from: http://hdl.handle.net/10761/3870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

20. Kramer, Elizabeth L. Role of the EGFR Pathway in Lung Remodeling and Disease.

Degree: PhD, Medicine : Molecular and Developmental Biology, 2009, University of Cincinnati

 Lung remodeling occurs in many chronic lung diseases, including pulmonary fibrosis, bronchopulmonary dysplasia (BPD), and asthma. The signaling pathways that mediate lung remodeling are largely… (more)

Subjects/Keywords: Biology; EGFR; TGF-alpha; Egr-1; lung remodeling; house dust mite; airway smooth muscle remodeling

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APA (6th Edition):

Kramer, E. L. (2009). Role of the EGFR Pathway in Lung Remodeling and Disease. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250206890

Chicago Manual of Style (16th Edition):

Kramer, Elizabeth L. “Role of the EGFR Pathway in Lung Remodeling and Disease.” 2009. Doctoral Dissertation, University of Cincinnati. Accessed August 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250206890.

MLA Handbook (7th Edition):

Kramer, Elizabeth L. “Role of the EGFR Pathway in Lung Remodeling and Disease.” 2009. Web. 19 Aug 2019.

Vancouver:

Kramer EL. Role of the EGFR Pathway in Lung Remodeling and Disease. [Internet] [Doctoral dissertation]. University of Cincinnati; 2009. [cited 2019 Aug 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250206890.

Council of Science Editors:

Kramer EL. Role of the EGFR Pathway in Lung Remodeling and Disease. [Doctoral Dissertation]. University of Cincinnati; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250206890

21. Ferreira de Lima Júnior, Sérgio. Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano .

Degree: 2012, Universidade Federal de Pernambuco

 O câncer cervical (CC) é o segundo tipo de câncer mais comum a afetar mulheres em todo mundo. O Papillomavírus humano (HPV) é encontrado em… (more)

Subjects/Keywords: TGF-β; IL-6; HPV; 1; Neoplasia intraepitelial cervical; Sequenciamento de DNA

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APA (6th Edition):

Ferreira de Lima Júnior, S. (2012). Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/397

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ferreira de Lima Júnior, Sérgio. “Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano .” 2012. Thesis, Universidade Federal de Pernambuco. Accessed August 19, 2019. http://repositorio.ufpe.br/handle/123456789/397.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ferreira de Lima Júnior, Sérgio. “Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano .” 2012. Web. 19 Aug 2019.

Vancouver:

Ferreira de Lima Júnior S. Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2012. [cited 2019 Aug 19]. Available from: http://repositorio.ufpe.br/handle/123456789/397.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ferreira de Lima Júnior S. Avaliação dos polimorfismos nos genes das citocinas IL 6 (RS 1800795) e TGF- β (RS 1982073) e RS 1800471) e suas relações com o grau de lesão cervical em pacientes infectados pelo Papillomavírus humano . [Thesis]. Universidade Federal de Pernambuco; 2012. Available from: http://repositorio.ufpe.br/handle/123456789/397

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

22. Angenete, Eva. Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 BACKGROUND Preoperative radiotherapy reduces local recurrence due to rectal cancer, but increases postoperative morbidity. The aim of this thesis was to study the impact of… (more)

Subjects/Keywords: radiotherapy; rectal cancer; extracellular matrix; MMP-2; PAI-1; uPA; Calprotectin; TGF-beta1

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APA (6th Edition):

Angenete, E. (2009). Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/19389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Angenete, Eva. “Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed August 19, 2019. http://hdl.handle.net/2077/19389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Angenete, Eva. “Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy.” 2009. Web. 19 Aug 2019.

Vancouver:

Angenete E. Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/2077/19389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Angenete E. Remodelling of extracellular matrix in rectal cancer and preoperative radiotherapy. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/19389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

23. Hogan, Kelly A. MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY.

Degree: PhD, Integrative Biosciences, 2011, Penn State University

 Molecular crosstalk leading to the integration of signal transduction pathways—and the formation of a signaling network—is particularly important for maintaining cellular homeostasis. Stimuli received at… (more)

Subjects/Keywords: TGF beta-1; ultraviolet radiation; NF kappa B; keratinocytes; TNF alpha; skin

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APA (6th Edition):

Hogan, K. A. (2011). MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/12539

Chicago Manual of Style (16th Edition):

Hogan, Kelly A. “MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY.” 2011. Doctoral Dissertation, Penn State University. Accessed August 19, 2019. https://etda.libraries.psu.edu/catalog/12539.

MLA Handbook (7th Edition):

Hogan, Kelly A. “MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY.” 2011. Web. 19 Aug 2019.

Vancouver:

Hogan KA. MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY. [Internet] [Doctoral dissertation]. Penn State University; 2011. [cited 2019 Aug 19]. Available from: https://etda.libraries.psu.edu/catalog/12539.

Council of Science Editors:

Hogan KA. MODULATION OF NUCLEAR FACTOR KAPPA B (NFκB) TRANSACTIVATION BY. [Doctoral Dissertation]. Penn State University; 2011. Available from: https://etda.libraries.psu.edu/catalog/12539


Iowa State University

24. Song, Xiaoling. Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors.

Degree: 2010, Iowa State University

 As the major physiological inhibitor of plasminogen activator (PA), the type I plasminogen activator inhibitor (PAI-1) is important for controlling blood clotting and tissue remodeling… (more)

Subjects/Keywords: EGF; PAI-1; RNA accessible region; Synergy; TGF-β; Transcription; Biochemistry, Biophysics, and Structural Biology

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APA (6th Edition):

Song, X. (2010). Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/11817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Song, Xiaoling. “Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors.” 2010. Thesis, Iowa State University. Accessed August 19, 2019. https://lib.dr.iastate.edu/etd/11817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Song, Xiaoling. “Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors.” 2010. Web. 19 Aug 2019.

Vancouver:

Song X. Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors. [Internet] [Thesis]. Iowa State University; 2010. [cited 2019 Aug 19]. Available from: https://lib.dr.iastate.edu/etd/11817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Song X. Plasminogen activator inhibitor 1: mechanisms of its synergistic regulation by growth factors. [Thesis]. Iowa State University; 2010. Available from: https://lib.dr.iastate.edu/etd/11817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Chatzifrangkeskou, Maria. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.

Degree: Docteur es, Biologie, 2016, Université Pierre et Marie Curie – Paris VI

La cardiomyopathie dilatée est l'une des principales causes d'insuffisance cardiaque en Europe. Dans le cadre de la cardiomyopathie liée aux mutations du gène LMNA, en… (more)

Subjects/Keywords: Cardiomyopathie dilatée; LMNA; Cofiline; Actine; ERK 1/2; TGF-β; Dilated cardiomyopathy; LMNA; Cofilin; 573.1

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APA (6th Edition):

Chatzifrangkeskou, M. (2016). Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066380

Chicago Manual of Style (16th Edition):

Chatzifrangkeskou, Maria. “Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed August 19, 2019. http://www.theses.fr/2016PA066380.

MLA Handbook (7th Edition):

Chatzifrangkeskou, Maria. “Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA.” 2016. Web. 19 Aug 2019.

Vancouver:

Chatzifrangkeskou M. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2019 Aug 19]. Available from: http://www.theses.fr/2016PA066380.

Council of Science Editors:

Chatzifrangkeskou M. Roles of ERK1/2 signaling in LMNA-cardiomyopathy : Les rôles de la signalisation ERK1/2 dans la cardiomyopathie liée aux mutations du gène LMNA. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066380


Harvard University

26. Haitz, Karyn. Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors.

Degree: Doctor of Medicine, 2017, Harvard University

Part 1 Purpose: Basal cell carcinoma (BCC) vaccination could reduce treatment cost and improve quality of life. Hedgehog interacting protein 1 (HHIP1) is overexpressed in… (more)

Subjects/Keywords: Basal cell carcinoma; dendritic-cell targeted vaccine; immunotherapy; PD-1; TGF-beta

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APA (6th Edition):

Haitz, K. (2017). Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621377

Chicago Manual of Style (16th Edition):

Haitz, Karyn. “Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors.” 2017. Doctoral Dissertation, Harvard University. Accessed August 19, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621377.

MLA Handbook (7th Edition):

Haitz, Karyn. “Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors.” 2017. Web. 19 Aug 2019.

Vancouver:

Haitz K. Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2019 Aug 19]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621377.

Council of Science Editors:

Haitz K. Immunotherapy for Cutaneous Malignancies: Rationale for a Basal Cell Carcinoma Vaccine and Modulation of PD-1 in Tissue by Local Growth Factors. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40621377


University of Rochester

27. Farhat, Youssef. Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing.

Degree: PhD, 2015, University of Rochester

 Flexor tendon injuries, such as deep cuts to the hands, pose unique challenges due to their tendency to form debilitating adhesions, which are a type… (more)

Subjects/Keywords: PAI-1; Protease; Scar; Tendon; TGF-β1; Wound; Fibrosis; Flexor; Healing; Adhesion

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APA (6th Edition):

Farhat, Y. (2015). Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30055

Chicago Manual of Style (16th Edition):

Farhat, Youssef. “Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing.” 2015. Doctoral Dissertation, University of Rochester. Accessed August 19, 2019. http://hdl.handle.net/1802/30055.

MLA Handbook (7th Edition):

Farhat, Youssef. “Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing.” 2015. Web. 19 Aug 2019.

Vancouver:

Farhat Y. Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/1802/30055.

Council of Science Editors:

Farhat Y. Identification of PAI-1 as a biological target for the promotion of scarless flexor tendon healing. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30055

28. 三上,大輔. Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する.

Degree: 博士(医学), University of Fukui / 福井大学

以下に掲載:Hypertension Research 37 pp.422-431 2014. Nature Publishing Group. 共著者:Hideki Kimura, Kazuko Kamiyama, Kunio Torii, Kenji Kasuno, Naoki Takahashi, Haruyoshi Yoshida, Masayuki Iwano

Subjects/Keywords: ERK pathway; plasminogen activator inhibitor-1; TGF-beta 1

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APA (6th Edition):

三上,大輔. (n.d.). Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する. (Thesis). University of Fukui / 福井大学. Retrieved from http://hdl.handle.net/10098/8448

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

三上,大輔. “Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する.” Thesis, University of Fukui / 福井大学. Accessed August 19, 2019. http://hdl.handle.net/10098/8448.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

三上,大輔. “Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する.” Web. 19 Aug 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

Vancouver:

三上,大輔. Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する. [Internet] [Thesis]. University of Fukui / 福井大学; [cited 2019 Aug 19]. Available from: http://hdl.handle.net/10098/8448.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

三上,大輔. Telmisartan activates endogenous peroxisome proliferator-activated receptor-δ and may have anti-fibrotic effects in human mesangial cells : テルミサルタンはヒトメサンギウム細胞において,内因性peroxisome proliferator-activated receptor-δ(PPAR-δ)を活性化し,潜在的な抗線維化作用を有する. [Thesis]. University of Fukui / 福井大学; Available from: http://hdl.handle.net/10098/8448

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


McMaster University

29. KAFKA, JESSICA KATHERINE. THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION.

Degree: PhD, 2015, McMaster University

Thirty five million people are currently living with HIV-1 today with women accounting for half of infected individuals globally. Sexual transmission is the main route… (more)

Subjects/Keywords: Semen; TGF-Beta 1; HIV-1; Female Genital Tract; Male Genital Tract; Epithelial Cell; Mucosal Immunity

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APA (6th Edition):

KAFKA, J. K. (2015). THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/17273

Chicago Manual of Style (16th Edition):

KAFKA, JESSICA KATHERINE. “THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION.” 2015. Doctoral Dissertation, McMaster University. Accessed August 19, 2019. http://hdl.handle.net/11375/17273.

MLA Handbook (7th Edition):

KAFKA, JESSICA KATHERINE. “THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION.” 2015. Web. 19 Aug 2019.

Vancouver:

KAFKA JK. THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION. [Internet] [Doctoral dissertation]. McMaster University; 2015. [cited 2019 Aug 19]. Available from: http://hdl.handle.net/11375/17273.

Council of Science Editors:

KAFKA JK. THE ROLE OF SEMEN TRANSFORMING GROWTH FACTOR BETA 1 IN MODULATING IMMUNE RESPONSES DURING HIV-1 INFECTION. [Doctoral Dissertation]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/17273

30. Belchiolina Beatriz Fonseca. Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2.

Degree: 2011, Federal University of Uberlândia

A interação patógeno-hospedeiro de C. jejuni é um assunto ainda pouco explorado em aves e humanos. Assim esse trabalho objetivou avaliar a habilidade de C.… (more)

Subjects/Keywords: Explants; Caco-2; IL-8; IL-1β; TGF-β; 3; Explant; Bacteriologia; Campylobacter; Distension; Birds; IMUNOLOGIA APLICADA; Distensão; Aves

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APA (6th Edition):

Fonseca, B. B. (2011). Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2. (Thesis). Federal University of Uberlândia. Retrieved from http://www.bdtd.ufu.br//tde_busca/arquivo.php?codArquivo=3833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fonseca, Belchiolina Beatriz. “Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2.” 2011. Thesis, Federal University of Uberlândia. Accessed August 19, 2019. http://www.bdtd.ufu.br//tde_busca/arquivo.php?codArquivo=3833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fonseca, Belchiolina Beatriz. “Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2.” 2011. Web. 19 Aug 2019.

Vancouver:

Fonseca BB. Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2. [Internet] [Thesis]. Federal University of Uberlândia; 2011. [cited 2019 Aug 19]. Available from: http://www.bdtd.ufu.br//tde_busca/arquivo.php?codArquivo=3833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fonseca BB. Resposta celular à infecção por Campylobacter. jejuni em embriões e explants de íleo de aves e participação do citoesqueleto e lisossomos no processo de invasão em células CACO-2. [Thesis]. Federal University of Uberlândia; 2011. Available from: http://www.bdtd.ufu.br//tde_busca/arquivo.php?codArquivo=3833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

.